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Wyszukujesz frazę ""Interferon Type I"" wg kryterium: Temat


Tytuł:
Type I interferon induces cancer stem cells-mediated chemotherapy resistance.
Autorzy:
De Martino M; Department of Radiation Oncology, Weill Cornell Medical College, New York, NY, USA.
Vanpouille-Box C; Department of Radiation Oncology, Weill Cornell Medical College, New York, NY, USA.; Sandra and Edward Meyer Cancer Center, New York, NY, USA.
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Źródło:
Oncoimmunology [Oncoimmunology] 2022 Sep 22; Vol. 11 (1), pp. 2127274. Date of Electronic Publication: 2022 Sep 22 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Interferon Type I*/metabolism
Neoplasms*/drug therapy
Lysine/metabolism ; Neoplastic Stem Cells/metabolism
Czasopismo naukowe
Tytuł:
Lanthanide-Nucleotide Coordination Nanoparticles for STING Activation.
Autorzy:
Luo Z; Department of Chemistry, National University of Singapore, Singapore 117543, Singapore.
Liang X; State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University, Chengdu 610041, P. R. China.
He T; State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University, Chengdu 610041, P. R. China.
Qin X; Department of Chemistry, National University of Singapore, Singapore 117543, Singapore.
Li X; State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University, Chengdu 610041, P. R. China.
Li Y; State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University, Chengdu 610041, P. R. China.
Li L; State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University, Chengdu 610041, P. R. China.
Loh XJ; Agency for Science, Technology and Research, Institute of Materials Research and Engineering, Singapore 138634, Singapore.
Gong C; State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University, Chengdu 610041, P. R. China.
Liu X; Department of Chemistry, National University of Singapore, Singapore 117543, Singapore.; Agency for Science, Technology and Research, Institute of Materials Research and Engineering, Singapore 138634, Singapore.; The N.1 Institute for Health, National University of Singapore, Singapore 117456, Singapore.
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Źródło:
Journal of the American Chemical Society [J Am Chem Soc] 2022 Sep 14; Vol. 144 (36), pp. 16366-16377. Date of Electronic Publication: 2022 Aug 29.
Typ publikacji:
Journal Article
MeSH Terms:
Interferon Type I*/genetics
Interferon Type I*/metabolism
Lanthanoid Series Elements*
Nanoparticles*
Neoplasms*/therapy
Membrane Proteins/*metabolism
Adenosine Monophosphate ; Animals ; Europium ; Humans ; Interferon-beta ; Mice ; Molecular Docking Simulation ; Nucleotides ; Nucleotides, Cyclic/pharmacology ; Ovalbumin
Czasopismo naukowe
Tytuł:
Understanding Host-Virus Interactions: Assessment of Innate Immune Responses in Mastomys natalensis Cells after Arenavirus Infection.
Autorzy:
Brinkmann NM; Department of Virology, Bernhard Nocht Institute for Tropical Medicine, 20359 Hamburg, Germany.
Hoffmann C; Department of Virology, Bernhard Nocht Institute for Tropical Medicine, 20359 Hamburg, Germany.
Wurr S; Department of Virology, Bernhard Nocht Institute for Tropical Medicine, 20359 Hamburg, Germany.
Pallasch E; Department of Virology, Bernhard Nocht Institute for Tropical Medicine, 20359 Hamburg, Germany.; German Center for Infectious Disease, Partner Site Hamburg-Lübeck-Borstel-Riems, 20359 Hamburg, Germany.
Hinzmann J; Department of Virology, Bernhard Nocht Institute for Tropical Medicine, 20359 Hamburg, Germany.; German Center for Infectious Disease, Partner Site Hamburg-Lübeck-Borstel-Riems, 20359 Hamburg, Germany.
Ostermann E; Leibniz Institute of Virology, 20359 Hamburg, Germany.
Brune W; Leibniz Institute of Virology, 20359 Hamburg, Germany.
Eskes ME; Department of Virology, Bernhard Nocht Institute for Tropical Medicine, 20359 Hamburg, Germany.
Jungblut L; Department of Virology, Bernhard Nocht Institute for Tropical Medicine, 20359 Hamburg, Germany.
Günther S; Department of Virology, Bernhard Nocht Institute for Tropical Medicine, 20359 Hamburg, Germany.; German Center for Infectious Disease, Partner Site Hamburg-Lübeck-Borstel-Riems, 20359 Hamburg, Germany.
Unrau L; Department of Virology, Bernhard Nocht Institute for Tropical Medicine, 20359 Hamburg, Germany.
Oestereich L; Department of Virology, Bernhard Nocht Institute for Tropical Medicine, 20359 Hamburg, Germany.; German Center for Infectious Disease, Partner Site Hamburg-Lübeck-Borstel-Riems, 20359 Hamburg, Germany.
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Źródło:
Viruses [Viruses] 2022 Sep 08; Vol. 14 (9). Date of Electronic Publication: 2022 Sep 08.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms:
Arenaviridae Infections*
Arenavirus*/physiology
Interferon Type I*
Animals ; Humans ; Immunity, Innate ; Murinae ; Tanzania
Czasopismo naukowe
Tytuł:
Sarcoid-like lesions obfuscating the diagnosis of disseminated Mycobacterium genavense infection in a patient with IL-12Rβ1-associated immunodeficiency.
Autorzy:
Denicolò S; Department of Internal Medicine II (Infectious Diseases, Pneumology and Rheumatology), Medical University Innsbruck, Anichstr. 35, 6020, Innsbruck, Austria.; Department of Internal Medicine IV (Nephrology and Hypertensiology), Medical University Innsbruck, 6020, Innsbruck, Austria.
Laydevant S; Department of Internal Medicine II (Infectious Diseases, Pneumology and Rheumatology), Medical University Innsbruck, Anichstr. 35, 6020, Innsbruck, Austria.
Fink J; Department of Internal Medicine II (Infectious Diseases, Pneumology and Rheumatology), Medical University Innsbruck, Anichstr. 35, 6020, Innsbruck, Austria.
Geiger C; Department of Internal Medicine II (Infectious Diseases, Pneumology and Rheumatology), Medical University Innsbruck, Anichstr. 35, 6020, Innsbruck, Austria.
Pizzini A; Department of Internal Medicine II (Infectious Diseases, Pneumology and Rheumatology), Medical University Innsbruck, Anichstr. 35, 6020, Innsbruck, Austria.
Sarcletti M; Department of Dermatology, Venereology and Allergology, Medical University Innsbruck, 6020, Innsbruck, Austria.
Zschocke J; Institute of Human Genetics, Medical University Innsbruck, 6020, Innsbruck, Austria.
Bellmann-Weiler R; Department of Internal Medicine II (Infectious Diseases, Pneumology and Rheumatology), Medical University Innsbruck, Anichstr. 35, 6020, Innsbruck, Austria.
Weiss G; Department of Internal Medicine II (Infectious Diseases, Pneumology and Rheumatology), Medical University Innsbruck, Anichstr. 35, 6020, Innsbruck, Austria. guenter.weiss@i-med.ac.at.
Tancevski I; Department of Internal Medicine II (Infectious Diseases, Pneumology and Rheumatology), Medical University Innsbruck, Anichstr. 35, 6020, Innsbruck, Austria. ivan.tancevski@i-med.ac.at.
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Źródło:
BMC infectious diseases [BMC Infect Dis] 2022 Oct 04; Vol. 22 (1), pp. 770. Date of Electronic Publication: 2022 Oct 04.
Typ publikacji:
Case Reports; Journal Article
MeSH Terms:
Immunologic Deficiency Syndromes*/complications
Interferon Type I*
Lymphadenopathy*/complications
Mycobacterium Infections*/complications
Mycobacterium Infections*/diagnosis
Mycobacterium Infections*/drug therapy
Mycobacterium Infections, Nontuberculous*/complications
Mycobacterium Infections, Nontuberculous*/diagnosis
Mycobacterium Infections, Nontuberculous*/drug therapy
Sarcoidosis*/diagnosis
Adult ; Female ; Humans ; Male ; Mycobacterium ; Nontuberculous Mycobacteria/genetics ; Receptors, Interleukin-12/genetics
SCR Organism:
Mycobacterium genavense
Czasopismo naukowe
Tytuł:
Novel Poxin Stable cGAMP-Derivatives Are Remarkable STING Agonists.
Autorzy:
Stazzoni S; New address: Monoclonal Antibody Discovery (MAD) Lab, Fondazione Toscana Life Sciences, 53100, Siena, Italy.; Department of Chemistry, Ludwig-Maximilians-Universität München, Butenandtstr. 5-13, 81377, Munich, Germany.
Böhmer DFR; Division of Clinical Pharmacology, University Hospital, Ludwig-Maximilians-Universität München, Lindwurmstr. 2a, 80337, Munich, Germany.
Hernichel F; Department of Chemistry, Ludwig-Maximilians-Universität München, Butenandtstr. 5-13, 81377, Munich, Germany.
Özdemir D; Department of Chemistry, Ludwig-Maximilians-Universität München, Butenandtstr. 5-13, 81377, Munich, Germany.
Pappa A; Department of Chemistry, Ludwig-Maximilians-Universität München, Butenandtstr. 5-13, 81377, Munich, Germany.
Drexler D; Gene Center and Department of Biochemistry, Ludwig-Maximilians-Universität München, Feodor-Lynen-Str. 25, 81377, Munich, Germany.
Bauernfried S; Gene Center and Department of Biochemistry, Ludwig-Maximilians-Universität München, Feodor-Lynen-Str. 25, 81377, Munich, Germany.
Witte G; Gene Center and Department of Biochemistry, Ludwig-Maximilians-Universität München, Feodor-Lynen-Str. 25, 81377, Munich, Germany.
Wagner M; Department of Chemistry, Ludwig-Maximilians-Universität München, Butenandtstr. 5-13, 81377, Munich, Germany.
Veth S; Department of Chemistry, Ludwig-Maximilians-Universität München, Butenandtstr. 5-13, 81377, Munich, Germany.
Hopfner KP; Gene Center and Department of Biochemistry, Ludwig-Maximilians-Universität München, Feodor-Lynen-Str. 25, 81377, Munich, Germany.
Hornung V; Gene Center and Department of Biochemistry, Ludwig-Maximilians-Universität München, Feodor-Lynen-Str. 25, 81377, Munich, Germany.
König LM; Division of Clinical Pharmacology, University Hospital, Ludwig-Maximilians-Universität München, Lindwurmstr. 2a, 80337, Munich, Germany.
Carell T; Department of Chemistry, Ludwig-Maximilians-Universität München, Butenandtstr. 5-13, 81377, Munich, Germany.
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Źródło:
Angewandte Chemie (International ed. in English) [Angew Chem Int Ed Engl] 2022 Oct 04; Vol. 61 (40), pp. e202207175. Date of Electronic Publication: 2022 Aug 31.
Typ publikacji:
Journal Article
MeSH Terms:
Interferon Type I*
Nucleosides*
Membrane Proteins/*genetics
Animals ; Antiviral Agents ; Cytokines ; DNA ; Endonucleases ; Immunity, Innate ; Membrane Proteins/metabolism ; Mice ; Nucleotides, Cyclic ; Nucleotidyltransferases/metabolism ; Ribose
Czasopismo naukowe
Tytuł:
African swine fever virus M1249L protein antagonizes type I interferon production via suppressing phosphorylation of TBK1 and degrading IRF3.
Autorzy:
Cui S; Institute of Animal Sciences, Chinese Academy of Agricultural Sciences, Beijing, China.
Wang Y; Institute of Animal Sciences, Chinese Academy of Agricultural Sciences, Beijing, China.
Gao X; Biotechnology Research Institute, Chinese Academy of Agricultural Sciences, Beijing, China.
Xin T; Institute of Animal Sciences, Chinese Academy of Agricultural Sciences, Beijing, China.
Wang X; Institute of Microbiology, Chinese Academy of Sciences, Beijing, China.
Yu H; Institute of Animal Sciences, Chinese Academy of Agricultural Sciences, Beijing, China.
Chen S; Institute of Animal Sciences, Chinese Academy of Agricultural Sciences, Beijing, China.
Jiang Y; Institute of Animal Sciences, Chinese Academy of Agricultural Sciences, Beijing, China.
Chen Q; Beijing University of Agriculture, Beijing, China.
Jiang F; China Animal Disease Control Center, Beijing, China.
Wang D; China Animal Disease Control Center, Beijing, China.
Guo X; Institute of Animal Sciences, Chinese Academy of Agricultural Sciences, Beijing, China. Electronic address: .
Jia H; Institute of Animal Sciences, Chinese Academy of Agricultural Sciences, Beijing, China. Electronic address: .
Zhu H; Institute of Animal Sciences, Chinese Academy of Agricultural Sciences, Beijing, China. Electronic address: .
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Źródło:
Virus research [Virus Res] 2022 Oct 02; Vol. 319, pp. 198872. Date of Electronic Publication: 2022 Jul 16.
Typ publikacji:
Journal Article
MeSH Terms:
African Swine Fever*
African Swine Fever Virus*/genetics
Interferon Type I*/metabolism
Animals ; DNA/metabolism ; Immunity, Innate/genetics ; Interferon Regulatory Factor-3/genetics ; Interferon Regulatory Factor-3/metabolism ; Interferon-beta/genetics ; Interferon-beta/metabolism ; Membrane Proteins/metabolism ; Nucleotidyltransferases/genetics ; Nucleotidyltransferases/metabolism ; Phosphorylation/genetics ; Protein Serine-Threonine Kinases/genetics ; Swine
Czasopismo naukowe
Tytuł:
Increased Type I interferon signaling and brain endothelial barrier dysfunction in an experimental model of Alzheimer's disease.
Autorzy:
Jana A; Division of Cardiology, Department of Medicine, College of Medicine, University of Illinois, Chicago, IL, 60612, USA.
Wang X; Department of Biomedical Engineering, University of Illinois at Chicago, Chicago, IL, 60612, USA.; Department of Biochemistry and Molecular Genetics, University of Illinois, College of Medicine, Chicago, IL, 60607, USA.
Leasure JW; Department of Biochemistry and Molecular Genetics, University of Illinois, College of Medicine, Chicago, IL, 60607, USA.
Magana L; Department of Biochemistry and Molecular Genetics, University of Illinois, College of Medicine, Chicago, IL, 60607, USA.
Wang L; Division of Cardiology, Department of Medicine, College of Medicine, University of Illinois, Chicago, IL, 60612, USA.; Department of Biochemistry and Molecular Genetics, University of Illinois, College of Medicine, Chicago, IL, 60607, USA.
Kim YM; Division of Cardiology, Department of Medicine, College of Medicine, University of Illinois, Chicago, IL, 60612, USA.; Department of Biochemistry and Molecular Genetics, University of Illinois, College of Medicine, Chicago, IL, 60607, USA.
Dodiya H; Department of Neurobiology, University of Chicago, Chicago, IL, 60637, USA.; The Microbiome Center, University of Chicago, Chicago, IL, 60637, USA.
Toth PT; Research Resources Center, University of Chicago, Chicago, IL, 60612, USA.; Department of Pharmacology and Regenerative Medicine, University of Chicago, Chicago, IL, 60612, USA.
Sisodia SS; Department of Neurobiology, University of Chicago, Chicago, IL, 60637, USA.; The Microbiome Center, University of Chicago, Chicago, IL, 60637, USA.
Rehman J; Division of Cardiology, Department of Medicine, College of Medicine, University of Illinois, Chicago, IL, 60612, USA. .; Department of Biomedical Engineering, University of Illinois at Chicago, Chicago, IL, 60612, USA. .; Department of Biochemistry and Molecular Genetics, University of Illinois, College of Medicine, Chicago, IL, 60607, USA. .; Department of Pharmacology and Regenerative Medicine, University of Chicago, Chicago, IL, 60612, USA. .
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Źródło:
Scientific reports [Sci Rep] 2022 Oct 01; Vol. 12 (1), pp. 16488. Date of Electronic Publication: 2022 Oct 01.
Typ publikacji:
Journal Article
MeSH Terms:
Alzheimer Disease*/metabolism
Interferon Type I*/metabolism
Amyloid beta-Peptides/metabolism ; Animals ; Blood-Brain Barrier/metabolism ; Brain/metabolism ; Claudin-5/metabolism ; Disease Models, Animal ; Endothelial Cells/metabolism ; Endothelium/metabolism ; Female ; Humans ; Interferon-beta/metabolism ; Mice ; Mice, Transgenic ; Occludin/metabolism ; Plaque, Amyloid/pathology ; RNA/metabolism ; Receptor, Interferon alpha-beta/metabolism ; Tight Junction Proteins/metabolism
Czasopismo naukowe
Tytuł:
Chicken TAX1BP1 suppresses type I interferon production via degrading chicken MAVS and facilitates infectious bursal diseases virus replication.
Autorzy:
Hu X; Department of Veterinary Preventive Medicine, College of Animal Science and Technology, Jiangxi Agricultural University, Zhimin Street, Qingshan Lake, Nanchang, 330045, PR China; Jiangxi Provincial Key Laboratory for Animal Science and Technology, College of Animal Science and Technology, Jiangxi Agricultural University, Nanchang, 330045, PR China.
Wu X; Department of Veterinary Preventive Medicine, College of Animal Science and Technology, Jiangxi Agricultural University, Zhimin Street, Qingshan Lake, Nanchang, 330045, PR China; Jiangxi Provincial Key Laboratory for Animal Science and Technology, College of Animal Science and Technology, Jiangxi Agricultural University, Nanchang, 330045, PR China.
Xue M; Department of Veterinary Preventive Medicine, College of Animal Science and Technology, Jiangxi Agricultural University, Zhimin Street, Qingshan Lake, Nanchang, 330045, PR China; Jiangxi Provincial Key Laboratory for Animal Science and Technology, College of Animal Science and Technology, Jiangxi Agricultural University, Nanchang, 330045, PR China.
Chen Y; Department of Veterinary Preventive Medicine, College of Animal Science and Technology, Jiangxi Agricultural University, Zhimin Street, Qingshan Lake, Nanchang, 330045, PR China; Jiangxi Provincial Key Laboratory for Animal Science and Technology, College of Animal Science and Technology, Jiangxi Agricultural University, Nanchang, 330045, PR China.
Zhou B; Department of Veterinary Preventive Medicine, College of Animal Science and Technology, Jiangxi Agricultural University, Zhimin Street, Qingshan Lake, Nanchang, 330045, PR China; Jiangxi Provincial Key Laboratory for Animal Science and Technology, College of Animal Science and Technology, Jiangxi Agricultural University, Nanchang, 330045, PR China.
Wan T; College of Engineering, Jiangxi Agricultural University, Zhimin Street, Qingshan Lake, Nanchang, 330045, PR China.
You H; College of Foreign Languages, Jiangxi Agricultural University, Zhimin Street, Qingshan Lake, Nanchang, 330045, PR China.
Wu H; Department of Veterinary Preventive Medicine, College of Animal Science and Technology, Jiangxi Agricultural University, Zhimin Street, Qingshan Lake, Nanchang, 330045, PR China; Jiangxi Provincial Key Laboratory for Animal Science and Technology, College of Animal Science and Technology, Jiangxi Agricultural University, Nanchang, 330045, PR China. Electronic address: .
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Źródło:
Developmental and comparative immunology [Dev Comp Immunol] 2022 Oct; Vol. 135, pp. 104490. Date of Electronic Publication: 2022 Jul 03.
Typ publikacji:
Journal Article
MeSH Terms:
Birnaviridae Infections*
Infectious bursal disease virus*
Interferon Type I*
Animals ; Chickens ; Immunity, Innate ; Mammals ; Virus Replication
Czasopismo naukowe
Tytuł:
Mechanisms of host type I interferon response modulation by the nucleocapsid proteins of alpha- and betacoronaviruses.
Autorzy:
Yelemali P; Vaccine and Infectious Disease Organization (VIDO), University of Saskatchewan, 120 Veterinary Road, Saskatoon, SK, S7N 5E3, Canada.; Neuroscience Major, Rhodes College, Memphis, TN, USA.
Hao L; Vaccine and Infectious Disease Organization (VIDO), University of Saskatchewan, 120 Veterinary Road, Saskatoon, SK, S7N 5E3, Canada.; Vaccinology and Immunotherapeutics, School of Public Health, University of Saskatchewan, Saskatoon, SK, Canada.
Liu Q; Vaccine and Infectious Disease Organization (VIDO), University of Saskatchewan, 120 Veterinary Road, Saskatoon, SK, S7N 5E3, Canada. .; Vaccinology and Immunotherapeutics, School of Public Health, University of Saskatchewan, Saskatoon, SK, Canada. .; Department of Veterinary Microbiology, Western College of Veterinary Medicine, University of Saskatchewan, Saskatoon, SK, Canada. .
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Źródło:
Archives of virology [Arch Virol] 2022 Oct; Vol. 167 (10), pp. 1925-1930. Date of Electronic Publication: 2022 Jun 28.
Typ publikacji:
Journal Article; Review
MeSH Terms:
COVID-19*
Interferon Type I*
Porcine epidemic diarrhea virus*/genetics
Animals ; Antiviral Agents ; Humans ; Nucleocapsid Proteins/genetics ; Nucleocapsid Proteins/metabolism ; Pandemics ; SARS-CoV-2 ; Swine
Czasopismo naukowe
Tytuł:
Sorafenib suppresses the activation of type I interferon pathway induced by RLR-MAVS and cGAS-STING signaling.
Autorzy:
Huang Y; Biotherapy Center, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China; Cell-gene Therapy Translational Medicine Research Center, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China; Guangdong Provincial Key Laboratory of Liver Disease Research, Guangzhou, China.
Liang W; Biotherapy Center, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Li K; Department of Hepatic Surgery and Liver Transplantation Center, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Liao X; Biotherapy Center, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Chen J; Biotherapy Center, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Qiu X; Vaccine Research Institute, The Third Affiliated Hospital of Sun Yat-sen University, Sun Yat-sen University, Guangzhou, China.
Liu K; Biotherapy Center, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China. Electronic address: .
Qiu D; Vaccine Research Institute, The Third Affiliated Hospital of Sun Yat-sen University, Sun Yat-sen University, Guangzhou, China. Electronic address: .
Qin Y; Biotherapy Center, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China; Cell-gene Therapy Translational Medicine Research Center, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China; Guangdong Provincial Key Laboratory of Liver Disease Research, Guangzhou, China. Electronic address: .
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Źródło:
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2022 Oct 01; Vol. 623, pp. 181-188. Date of Electronic Publication: 2022 Jul 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Carcinoma, Hepatocellular*
Interferon Type I*/metabolism
Liver Neoplasms*
Humans ; Immunity, Innate ; Interferon Regulatory Factor-3/metabolism ; Nucleotidyltransferases/metabolism ; Protein Serine-Threonine Kinases ; Sorafenib/pharmacology
Czasopismo naukowe
Tytuł:
NBR1 mediates autophagic degradation of IRF3 to negatively regulate type I interferon production.
Autorzy:
Cai Y; Institute of Immunology, Zhejiang University School of Medicine, Hangzhou, 310058, People's Republic of China.
Zhu Y; Institute of Immunology, Zhejiang University School of Medicine, Hangzhou, 310058, People's Republic of China.
Zheng J; Institute of Immunology, Zhejiang University School of Medicine, Hangzhou, 310058, People's Republic of China.
Zhang Y; Institute of Immunology, Zhejiang University School of Medicine, Hangzhou, 310058, People's Republic of China.
Chen W; Institute of Immunology, Zhejiang University School of Medicine, Hangzhou, 310058, People's Republic of China. Electronic address: .
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Źródło:
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2022 Oct 01; Vol. 623, pp. 140-147. Date of Electronic Publication: 2022 Jul 14.
Typ publikacji:
Journal Article
MeSH Terms:
Interferon Type I*/metabolism
Virus Diseases*
Interferon Regulatory Factor-3/*metabolism
Intracellular Signaling Peptides and Proteins/*metabolism
Autophagy/physiology ; HEK293 Cells ; Humans ; Proteins/metabolism
Czasopismo naukowe
Tytuł:
Docetaxel resistance-derived LINC01085 contributes to the immunotherapy of hormone-independent prostate cancer by activating the STING/MAVS signaling pathway.
Autorzy:
Zhang J; Shanghai Key Laboratory of Compound Chinese Medicines, The MOE Key Laboratory for Standardization of Chinese Medicines and the SATCM Key Laboratory for New Resources and Quality Evaluation of Chinese Medicines, Institute of Chinese Materia Medica, Shanghai University of Traditional Chinese Medicine, Shanghai, 201203, China. Electronic address: .
Li S; Precision Research Center for Refractory Diseases, Institute for Clinical Research, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine (SJTU-SM), Shanghai, 201620, China.
Zhang J; Department of Clinical Laboratory Medicine, Shanghai Tenth People's Hospital of Tongji University, Shanghai, 200072, China.
Zhang W; School of Life Sciences, Shanghai University, Shanghai, 200444, China.
Jiang J; School of Life Sciences, Shanghai University, Shanghai, 200444, China.
Wu H; School of Life Sciences, Shanghai University, Shanghai, 200444, China.
Wu E; Shanghai Key Laboratory of Compound Chinese Medicines, The MOE Key Laboratory for Standardization of Chinese Medicines and the SATCM Key Laboratory for New Resources and Quality Evaluation of Chinese Medicines, Institute of Chinese Materia Medica, Shanghai University of Traditional Chinese Medicine, Shanghai, 201203, China.
Feng Y; Shanghai Key Laboratory of Compound Chinese Medicines, The MOE Key Laboratory for Standardization of Chinese Medicines and the SATCM Key Laboratory for New Resources and Quality Evaluation of Chinese Medicines, Institute of Chinese Materia Medica, Shanghai University of Traditional Chinese Medicine, Shanghai, 201203, China.
Yang L; Shanghai Key Laboratory of Compound Chinese Medicines, The MOE Key Laboratory for Standardization of Chinese Medicines and the SATCM Key Laboratory for New Resources and Quality Evaluation of Chinese Medicines, Institute of Chinese Materia Medica, Shanghai University of Traditional Chinese Medicine, Shanghai, 201203, China. Electronic address: .
Li Z; School of Life Sciences, Shanghai University, Shanghai, 200444, China. Electronic address: zhe_.
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Źródło:
Cancer letters [Cancer Lett] 2022 Oct 01; Vol. 545, pp. 215829. Date of Electronic Publication: 2022 Jul 19.
Typ publikacji:
Journal Article
MeSH Terms:
Interferon Type I*
Prostatic Neoplasms*/drug therapy
Prostatic Neoplasms*/genetics
Docetaxel/pharmacology ; Glycogen Synthase Kinase 3 beta ; Hormones ; Humans ; Immunotherapy ; Male ; RNA ; Signal Transduction
Czasopismo naukowe
Tytuł:
Friend or foe: RIG- I like receptors and diseases.
Autorzy:
Song J; Department of Rheumatology, Xiangya Hospital, Central South University, Changsha City, Hunan Province, China; Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha City, Hunan Province, China; Sepsis Translational Medicine Key Lab of Hunan Province, Central South University, Changsha City, Hunan Province, China.
Li M; Department of Rheumatology, Xiangya Hospital, Central South University, Changsha City, Hunan Province, China; Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha City, Hunan Province, China; Sepsis Translational Medicine Key Lab of Hunan Province, Central South University, Changsha City, Hunan Province, China; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Changsha City, Hunan Province, China.
Li C; Department of Rheumatology, Xiangya Hospital, Central South University, Changsha City, Hunan Province, China; Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha City, Hunan Province, China; Sepsis Translational Medicine Key Lab of Hunan Province, Central South University, Changsha City, Hunan Province, China.
Liu K; Department of Rheumatology, Xiangya Hospital, Central South University, Changsha City, Hunan Province, China; Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha City, Hunan Province, China; Sepsis Translational Medicine Key Lab of Hunan Province, Central South University, Changsha City, Hunan Province, China.
Zhu Y; Department of Rheumatology, Xiangya Hospital, Central South University, Changsha City, Hunan Province, China; Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha City, Hunan Province, China; Sepsis Translational Medicine Key Lab of Hunan Province, Central South University, Changsha City, Hunan Province, China. Electronic address: zhu_.
Zhang H; Department of Rheumatology, Xiangya Hospital, Central South University, Changsha City, Hunan Province, China; Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha City, Hunan Province, China; Sepsis Translational Medicine Key Lab of Hunan Province, Central South University, Changsha City, Hunan Province, China. Electronic address: .
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Źródło:
Autoimmunity reviews [Autoimmun Rev] 2022 Oct; Vol. 21 (10), pp. 103161. Date of Electronic Publication: 2022 Aug 01.
Typ publikacji:
Journal Article; Review
MeSH Terms:
COVID-19*/genetics
Interferon Type I*
MicroRNAs*
Antiviral Agents ; Cytokines ; Humans ; Immunity, Innate ; Nucleotides ; SARS-CoV-2 ; Tretinoin
Czasopismo naukowe
Tytuł:
Functional and expression analysis reveals the involvement of integrin αvβ3 in antiviral immunity of grass carp (Ctenopharyngodon idella).
Autorzy:
Xiong X; Hunan Engineering Technology Research Center of Featured Aquatic Resources Utilization, Hunan Agricultural University, Changsha, 410128, China; College of Animal Science and Technology, Hunan Agricultural University, Changsha, 410128, China.
Yang H; Hunan Engineering Technology Research Center of Featured Aquatic Resources Utilization, Hunan Agricultural University, Changsha, 410128, China; College of Animal Science and Technology, Hunan Agricultural University, Changsha, 410128, China.
Ding C; Hunan Engineering Technology Research Center of Featured Aquatic Resources Utilization, Hunan Agricultural University, Changsha, 410128, China; College of Animal Science and Technology, Hunan Agricultural University, Changsha, 410128, China.
Qin B; Hunan Engineering Technology Research Center of Featured Aquatic Resources Utilization, Hunan Agricultural University, Changsha, 410128, China; College of Animal Science and Technology, Hunan Agricultural University, Changsha, 410128, China.
Deng Y; Hunan Engineering Technology Research Center of Featured Aquatic Resources Utilization, Hunan Agricultural University, Changsha, 410128, China; College of Animal Science and Technology, Hunan Agricultural University, Changsha, 410128, China.
Xiong L; Hunan Engineering Technology Research Center of Featured Aquatic Resources Utilization, Hunan Agricultural University, Changsha, 410128, China; College of Animal Science and Technology, Hunan Agricultural University, Changsha, 410128, China.
Liu X; Hunan Engineering Technology Research Center of Featured Aquatic Resources Utilization, Hunan Agricultural University, Changsha, 410128, China.
Li Y; Hunan Engineering Technology Research Center of Featured Aquatic Resources Utilization, Hunan Agricultural University, Changsha, 410128, China.
Xiao T; Hunan Engineering Technology Research Center of Featured Aquatic Resources Utilization, Hunan Agricultural University, Changsha, 410128, China. Electronic address: .
Lv Z; Hunan Engineering Technology Research Center of Featured Aquatic Resources Utilization, Hunan Agricultural University, Changsha, 410128, China. Electronic address: lvzhao_.
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Źródło:
Fish & shellfish immunology [Fish Shellfish Immunol] 2022 Oct; Vol. 129, pp. 52-63. Date of Electronic Publication: 2022 Aug 19.
Typ publikacji:
Journal Article
MeSH Terms:
Carps*/genetics
Carps*/metabolism
Fish Diseases*
Interferon Type I*
Reoviridae*/physiology
Reoviridae Infections*
Animals ; Antiviral Agents ; Capsid Proteins ; Fish Proteins/chemistry ; Humans ; Integrin alphaVbeta3/genetics ; Mammals/metabolism ; Phylogeny
Czasopismo naukowe
Tytuł:
Nectin4 antagonises type I interferon production by targeting TRAF3 for autophagic degradation and disrupting TRAF3-TBK1 complex formation.
Autorzy:
Zhang W; School of Marine Sciences, Sun Yat-sen University, Guangzhou 510000, China; Guangdong Provincial Key Laboratory of Marine Resources and Coastal Engineering, Guangzhou 510000, China; Pearl River Estuary Marine Ecosystem Research Station, Ministry of Education, Zhuhai 519000, China; Southern Marine Science and Engineering Guangdong Laboratory (Zhuhai), Zhuhai 519000, China.
Weng J; School of Marine Sciences, Sun Yat-sen University, Guangzhou 510000, China; Guangdong Provincial Key Laboratory of Marine Resources and Coastal Engineering, Guangzhou 510000, China; Pearl River Estuary Marine Ecosystem Research Station, Ministry of Education, Zhuhai 519000, China; Southern Marine Science and Engineering Guangdong Laboratory (Zhuhai), Zhuhai 519000, China.
Yao L; School of Marine Sciences, Sun Yat-sen University, Guangzhou 510000, China; Guangdong Provincial Key Laboratory of Marine Resources and Coastal Engineering, Guangzhou 510000, China; Pearl River Estuary Marine Ecosystem Research Station, Ministry of Education, Zhuhai 519000, China; Southern Marine Science and Engineering Guangdong Laboratory (Zhuhai), Zhuhai 519000, China.
Jia P; School of Marine Sciences, Sun Yat-sen University, Guangzhou 510000, China; Fuzhou Medical University, Jiangxi, Fuzhou 344000, China.
Yi M; School of Marine Sciences, Sun Yat-sen University, Guangzhou 510000, China; Guangdong Provincial Key Laboratory of Marine Resources and Coastal Engineering, Guangzhou 510000, China; Pearl River Estuary Marine Ecosystem Research Station, Ministry of Education, Zhuhai 519000, China; Southern Marine Science and Engineering Guangdong Laboratory (Zhuhai), Zhuhai 519000, China.
Jia K; School of Marine Sciences, Sun Yat-sen University, Guangzhou 510000, China; Guangdong Provincial Key Laboratory of Marine Resources and Coastal Engineering, Guangzhou 510000, China; Pearl River Estuary Marine Ecosystem Research Station, Ministry of Education, Zhuhai 519000, China; Southern Marine Science and Engineering Guangdong Laboratory (Zhuhai), Zhuhai 519000, China. Electronic address: .
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Źródło:
International journal of biological macromolecules [Int J Biol Macromol] 2022 Oct 01; Vol. 218, pp. 654-664. Date of Electronic Publication: 2022 Jul 23.
Typ publikacji:
Journal Article
MeSH Terms:
Interferon Type I*
TNF Receptor-Associated Factor 3*/genetics
TNF Receptor-Associated Factor 3*/metabolism
Animals ; Autophagy ; Immunity, Innate ; Phosphorylation ; Signal Transduction
Czasopismo naukowe
Tytuł:
All-Atom Simulations Uncover Structural and Dynamical Properties of STING Proteins in the Membrane System.
Autorzy:
Payne RT; Boston University, Boston, Massachusetts02215, United States.
Crivelli S; Lawrence Berkeley National Laboratory, Berkeley, California94720, United States.
Watanabe M; Department of Chemistry, Fort Hays State University, Hays, Kansas67601, United States.
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Źródło:
Journal of chemical information and modeling [J Chem Inf Model] 2022 Sep 26; Vol. 62 (18), pp. 4486-4499. Date of Electronic Publication: 2022 Sep 14.
Typ publikacji:
Journal Article
MeSH Terms:
Interferon Type I*
Membrane Proteins/*metabolism
Disulfides ; Humans ; Ligands ; Membrane Proteins/chemistry ; Molecular Dynamics Simulation
Czasopismo naukowe
Tytuł:
Single-cell sequencing reveals activation of core transcription factors in PRC2-deficient malignant peripheral nerve sheath tumor.
Autorzy:
Zhang X; Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Lou HE; Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Gopalan V; Cancer Data Science Laboratory, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Liu Z; Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Jafarah HM; Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Lei H; Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Jones P; Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Sayers CM; Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Yohe ME; Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Chittiboina P; Neurosurgery Unit for Pituitary and Inheritable Diseases, National Institute of Neurological Diseases and Stroke, Bethesda, MD 20892, USA.
Widemann BC; Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Thiele CJ; Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Kelly MC; Center for Cancer Research Single Cell Analysis Facility, Cancer Research Technology Program, Frederick National Laboratory, Bethesda, MD 20892, USA.
Hannenhalli S; Cancer Data Science Laboratory, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Shern JF; Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA. Electronic address: .
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Źródło:
Cell reports [Cell Rep] 2022 Sep 20; Vol. 40 (12), pp. 111363.
Typ publikacji:
Journal Article
MeSH Terms:
Interferon Type I*/genetics
Neurofibrosarcoma*/genetics
Carcinogenesis ; Chromatin ; Humans ; Interferon Regulatory Factors/genetics ; Polycomb Repressive Complex 2/genetics ; Polycomb Repressive Complex 2/metabolism
Czasopismo naukowe
Tytuł:
Correlation between Type I Interferon Associated Factors and COVID-19 Severity.
Autorzy:
Bencze D; Department of Immunology, Faculty of Medicine, University of Debrecen, 1 Egyetem Square, H-4032 Debrecen, Hungary.; Doctoral School of Molecular Cell and Immune Biology, University of Debrecen, 1 Egyetem Square, H-4032 Debrecen, Hungary.
Fekete T; Department of Immunology, Faculty of Medicine, University of Debrecen, 1 Egyetem Square, H-4032 Debrecen, Hungary.
Pázmándi K; Department of Immunology, Faculty of Medicine, University of Debrecen, 1 Egyetem Square, H-4032 Debrecen, Hungary.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Sep 19; Vol. 23 (18). Date of Electronic Publication: 2022 Sep 19.
Typ publikacji:
Journal Article; Review
MeSH Terms:
COVID-19*
Interferon Type I*
Antiviral Agents/pharmacology ; Antiviral Agents/therapeutic use ; Humans ; Interferons/pharmacology ; SARS-CoV-2 ; Virus Replication
Czasopismo naukowe
Tytuł:
SARS-CoV-2 Infection of Airway Epithelium Triggers Pulmonary Endothelial Cell Activation and Senescence Associated with Type I IFN Production.
Autorzy:
Bordoni V; Cellular Immunology Laboratory, National Institute for Infectious Diseases 'L. Spallanzani' IRCCS, Via Portuense 292, 00149 Rome, Italy.
Mariotti D; Cellular Immunology Laboratory, National Institute for Infectious Diseases 'L. Spallanzani' IRCCS, Via Portuense 292, 00149 Rome, Italy.
Matusali G; Laboratory of Virology, National Institute for Infectious Diseases 'L. Spallanzani' IRCCS, Via Portuense 292, 00149 Rome, Italy.
Colavita F; Laboratory of Virology, National Institute for Infectious Diseases 'L. Spallanzani' IRCCS, Via Portuense 292, 00149 Rome, Italy.
Cimini E; Cellular Immunology Laboratory, National Institute for Infectious Diseases 'L. Spallanzani' IRCCS, Via Portuense 292, 00149 Rome, Italy.
Ippolito G; General Directorate for Research and Health Innovation, Italian Ministry of Health, 00144 Rome, Italy.
Agrati C; Cellular Immunology Laboratory, National Institute for Infectious Diseases 'L. Spallanzani' IRCCS, Via Portuense 292, 00149 Rome, Italy.
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Źródło:
Cells [Cells] 2022 Sep 17; Vol. 11 (18). Date of Electronic Publication: 2022 Sep 17.
Typ publikacji:
Journal Article
MeSH Terms:
COVID-19*/immunology
Endothelial Cells*/immunology
Interferon Type I*/immunology
Cellular Senescence ; Epithelium ; Humans ; Interleukin-6 ; Interleukin-8 ; Lung ; SARS-CoV-2
Czasopismo naukowe
Tytuł:
Polβ modulates the expression of type I interferon via STING pathway.
Autorzy:
Huang M; Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 Wen Yuan Road, Nanjing, 210023, China.
Wu T; Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 Wen Yuan Road, Nanjing, 210023, China.
Liu R; Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 Wen Yuan Road, Nanjing, 210023, China.
Wang M; Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 Wen Yuan Road, Nanjing, 210023, China; Department of Predictive Medicine, Institute of Biomedical Informatics, Cell Signal Transduction Laboratory, Bioinformatics Center, Henan Provincial Engineering Center for Tumor Molecular Medicine, School of Basic Medical Sciences, Henan University, Kaifeng, 475004, China.
Shi M; Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 Wen Yuan Road, Nanjing, 210023, China.
Xin J; Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 Wen Yuan Road, Nanjing, 210023, China.
Shao S; Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 Wen Yuan Road, Nanjing, 210023, China.
Zhao X; Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 Wen Yuan Road, Nanjing, 210023, China.
Ma Y; Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 Wen Yuan Road, Nanjing, 210023, China.
Gu L; Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 Wen Yuan Road, Nanjing, 210023, China.
Guo Z; Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 Wen Yuan Road, Nanjing, 210023, China.
Pan F; Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 Wen Yuan Road, Nanjing, 210023, China. Electronic address: .
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Źródło:
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2022 Sep 17; Vol. 621, pp. 137-143. Date of Electronic Publication: 2022 Jul 02.
Typ publikacji:
Journal Article
MeSH Terms:
Interferon Type I*
DNA Polymerase beta/*metabolism
Animals ; DNA Damage ; DNA Repair ; Membrane Proteins/metabolism ; Mice
Czasopismo naukowe

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