Temat: "Interleukin-6" - OpacWWW

Skocz do pozycji: 41.

Tytuł:: Weissella cibaria CMU exerts an anti‑inflammatory effect by inhibiting Aggregatibacter actinomycetemcomitans‑induced NF‑κB activation in macrophages.
Autorzy:: Kim MJ; Department of Convergence Technology for Food Industry and Dental Research Institute, School of Dentistry, Wonkwang University, Iksan, Jellabukdo 54538, Republic of Korea.
You YO; Department of Oral Biochemistry, School of Dentistry, Wonkwang University, Iksan, Jellabukdo 54538, Republic of Korea.
Kang JY; Department of Oral Biochemistry, School of Dentistry, Wonkwang University, Iksan, Jellabukdo 54538, Republic of Korea.
Kim HJ; Department of Oral Anatomy and Dental Research Institute, School of Dentistry, Wonkwang University, Iksan, Jellabukdo 54538, Republic of Korea.
Kang MS; Research and Development Department, Research Institute, OraPharm Inc., Seoul 04782, Republic of Korea.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2020 Nov; Vol. 22 (5), pp. 4143-4150. Date of Electronic Publication: 2020 Sep 15.
Typ publikacji:: Journal Article
MeSH Terms:: Aggregatibacter actinomycetemcomitans/*pathogenicity
Anti-Inflammatory Agents/*pharmacology
Cytokines/*genetics
Macrophages/*cytology
Probiotics/*pharmacology
Weissella/*physiology
Aggregatibacter actinomycetemcomitans/drug effects ; Aggregatibacter actinomycetemcomitans/immunology ; Animals ; Cell Survival ; Dose-Response Relationship, Drug ; Formaldehyde/adverse effects ; Gene Expression Regulation/drug effects ; Interleukin-1beta/genetics ; Interleukin-6/genetics ; Macrophages/immunology ; Mice ; NF-kappa B/metabolism ; Phosphorylation ; RAW 264.7 Cells ; Reactive Oxygen Species/metabolism ; Signal Transduction
SCR Organism:: Weissella cibaria

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Skocz do pozycji: 42.

Tytuł:: Complement 3 mediates periodontal destruction in patients with type 2 diabetes by regulating macrophage polarization in periodontal tissues.
Autorzy:: Li Y; Key Laboratory of Shaanxi Province for Craniofacial Precision Medicine Research, College of Stomatology, Xi'an Jiaotong University, Xi'an, China.; Department of Periodontology, College of Stomatology, Xi'an Jiaotong University, Xi'an, China.
Wang X; Key Laboratory of Shaanxi Province for Craniofacial Precision Medicine Research, College of Stomatology, Xi'an Jiaotong University, Xi'an, China.
Wang S; Department of Periodontology, College of Stomatology, Xi'an Jiaotong University, Xi'an, China.
Zhu C; Department of Periodontology, College of Stomatology, Xi'an Jiaotong University, Xi'an, China.
Guo J; Department of Periodontology, College of Stomatology, Xi'an Jiaotong University, Xi'an, China.
Li K; Core Research Laboratory, The Second Affiliated Hospital, School of Medicine, Xi'an Jiaotong University, Xi'an, China.
Li A; Key Laboratory of Shaanxi Province for Craniofacial Precision Medicine Research, College of Stomatology, Xi'an Jiaotong University, Xi'an, China.; Department of Periodontology, College of Stomatology, Xi'an Jiaotong University, Xi'an, China.; Pokaż więcej
Źródło:: Cell proliferation [Cell Prolif] 2020 Oct; Vol. 53 (10), pp. e12886. Date of Electronic Publication: 2020 Aug 14.
Typ publikacji:: Journal Article
MeSH Terms:: Complement C3/*metabolism
Diabetes Mellitus, Type 2/*pathology
Macrophages/*immunology
Periodontitis/*diagnosis
Adult ; Alveolar Process/diagnostic imaging ; Alveolar Process/pathology ; Alveolar Process/physiology ; Animals ; Bone Density ; Complement C3/analysis ; Complement C3/genetics ; Diabetes Mellitus, Experimental/metabolism ; Diabetes Mellitus, Experimental/pathology ; Diabetes Mellitus, Type 2/complications ; Female ; Gingival Crevicular Fluid/metabolism ; Humans ; Interleukin-6/genetics ; Interleukin-6/metabolism ; Macrophage Activation ; Macrophages/metabolism ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Middle Aged ; Periodontitis/etiology ; Tumor Necrosis Factor-alpha/genetics ; Tumor Necrosis Factor-alpha/metabolism

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Skocz do pozycji: 43.

Tytuł:: Lack of RAC1 in macrophages protects against atherosclerosis.
Autorzy:: Bandaru S; Department of Laboratory Medicine, Institute of Biomedicine, Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden.
Ala C; Department of Laboratory Medicine, Institute of Biomedicine, Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden.
Ekstrand M; Department of Molecular and Clinical Medicine, Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden.
Akula MK; Department of Molecular and Clinical Medicine, Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden.; Sahlgrenska Cancer Center, Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden.
Pedrelli M; Division of Clinical Chemistry, Department of Laboratory Medicine, Karolinska Institute, Stockholm, Sweden.
Liu X; Department of Laboratory Medicine, Institute of Biomedicine, Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden.
Bergström G; Department of Molecular and Clinical Medicine, Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden.; Department of Clinical Physiology, Västra Götalandregionen, Sahlgrenska University Hospital, Gothenburg, Sweden.
Håversen L; Department of Molecular and Clinical Medicine, Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden.
Borén J; Department of Molecular and Clinical Medicine, Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden.
Bergo MO; Sahlgrenska Cancer Center, Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden.; Department of Biosciences and Nutrition, Karolinska Institute, Stockholm, Sweden.
Akyürek LM; Department of Laboratory Medicine, Institute of Biomedicine, Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden.; Department of Clinical Pathology, Västra Götalandregionen, Sahlgrenska University Hospital, Gothenburg, Sweden.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2020 Sep 17; Vol. 15 (9), pp. e0239284. Date of Electronic Publication: 2020 Sep 17 (Print Publication: 2020).
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Atherosclerosis/*metabolism
Macrophages/*metabolism
Neuropeptides/*genetics
rac1 GTP-Binding Protein/*genetics
Animals ; Antigens, CD/genetics ; Antigens, CD/metabolism ; Antigens, Differentiation, Myelomonocytic/genetics ; Antigens, Differentiation, Myelomonocytic/metabolism ; Atherosclerosis/genetics ; Atherosclerosis/pathology ; Cells, Cultured ; Humans ; Interleukin-6/genetics ; Interleukin-6/metabolism ; Lipid Metabolism ; Mice ; Mice, Inbred C57BL ; Neuropeptides/metabolism ; Tumor Necrosis Factor-alpha/genetics ; Tumor Necrosis Factor-alpha/metabolism ; rac1 GTP-Binding Protein/metabolism

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Skocz do pozycji: 44.

Tytuł:: Cytokine Expression by Human Macrophage-Like Cells Derived from the Monocytic Cell Line THP-1 Differs Between Treatment With Milk from Preterm- and Term-Delivering Mothers and Pasteurized Donor Milk.
Autorzy:: Demers-Mathieu V; Nutrition Program, School of Biological and Population Health Sciences, College of Public Health and Human Sciences, Oregon State University, Corvallis, OR 97331, USA.
Huston RK; Department of Pediatrics, Randall Children's Hospital at Legacy Emanuel, Portland, OR 97227, USA.
Dallas DC; Department of Pediatrics, Randall Children's Hospital at Legacy Emanuel, Portland, OR 97227, USA.; Pokaż więcej
Źródło:: Molecules (Basel, Switzerland) [Molecules] 2020 May 20; Vol. 25 (10). Date of Electronic Publication: 2020 May 20.
Typ publikacji:: Journal Article
MeSH Terms:: Anti-Inflammatory Agents/*pharmacology
Cytokines/*genetics
Macrophages/*drug effects
Milk Proteins/*immunology
Milk, Human/*metabolism
Animals ; Anti-Inflammatory Agents/immunology ; Enterocolitis/immunology ; Enterocolitis/prevention & control ; Female ; Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating)/genetics ; Humans ; Infant ; Infant, Newborn ; Infant, Premature/immunology ; Infant, Premature/metabolism ; Inflammation/immunology ; Inflammation/prevention & control ; Interleukin-10/genetics ; Interleukin-12/genetics ; Interleukin-6/genetics ; Macrophages/immunology ; Milk Proteins/pharmacology ; Milk, Human/immunology ; Monocytes/drug effects ; Monocytes/metabolism ; Term Birth/metabolism ; Tumor Necrosis Factor-alpha/genetics

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Skocz do pozycji: 45.

Tytuł:: Isoorientin Inhibits Inflammation in Macrophages and Endotoxemia Mice by Regulating Glycogen Synthase Kinase 3 β .
Autorzy:: Li Y; Science and Technology Innovation Center, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong Province, China.
Zhao Y; Science and Technology Innovation Center, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong Province, China.
Tan X; Science and Technology Innovation Center, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong Province, China.; Department of Molecular Biosciences and Bioengineering, University of Hawaii at Manoa, Honolulu, Hawaii 96822, USA.
Liu J; Science and Technology Innovation Center, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong Province, China.
Zhi Y; Science and Technology Innovation Center, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong Province, China.
Yi L; Science and Technology Innovation Center, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong Province, China.
Bai S; Science and Technology Innovation Center, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong Province, China.
Du Q; Science and Technology Innovation Center, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong Province, China.
Li QX; Department of Molecular Biosciences and Bioengineering, University of Hawaii at Manoa, Honolulu, Hawaii 96822, USA.
Dong Y; Science and Technology Innovation Center, Guangzhou University of Chinese Medicine, Guangzhou, Guangdong Province, China.; Pokaż więcej
Źródło:: Mediators of inflammation [Mediators Inflamm] 2020 Oct 27; Vol. 2020, pp. 8704146. Date of Electronic Publication: 2020 Oct 27 (Print Publication: 2020).
Typ publikacji:: Journal Article
MeSH Terms:: Endotoxemia/*drug therapy
Glycogen Synthase Kinase 3 beta/*genetics
Inflammation/*drug therapy
Luteolin/*pharmacology
Macrophages/*drug effects
Animals ; Endotoxemia/metabolism ; Enzyme-Linked Immunosorbent Assay ; Extracellular Signal-Regulated MAP Kinases/metabolism ; Glycogen Synthase Kinase 3 beta/metabolism ; Heme Oxygenase-1/metabolism ; Heterocyclic Compounds, 3-Ring/pharmacology ; Interleukin-6/metabolism ; Lithium Chloride/pharmacology ; Macrophages/metabolism ; Male ; Membrane Proteins/metabolism ; Mice ; Mice, Inbred BALB C ; NF-E2-Related Factor 2/metabolism ; NF-kappa B p50 Subunit/metabolism ; Occludin/biosynthesis ; Phosphorylation ; RAW 264.7 Cells ; Real-Time Polymerase Chain Reaction ; Zonula Occludens-1 Protein/metabolism

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Skocz do pozycji: 46.

Tytuł:: Characterization of hepatic macrophages and evaluation of inflammatory response in heme oxygenase-1 deficient mice exposed to scAAV9 vectors.
Autorzy:: Tomczyk M; Department of Medical Biotechnology, Faculty of Biochemistry, Biophysics and Biotechnology, Jagiellonian University, Kraków, Poland.
Kraszewska I; Department of Medical Biotechnology, Faculty of Biochemistry, Biophysics and Biotechnology, Jagiellonian University, Kraków, Poland.
Mąka R; Department of Medical Biotechnology, Faculty of Biochemistry, Biophysics and Biotechnology, Jagiellonian University, Kraków, Poland.
Waligórska A; Department of Cell Biophysics, Faculty of Biochemistry, Biophysics and Biotechnology, Jagiellonian University, Kraków, Poland.
Dulak J; Department of Medical Biotechnology, Faculty of Biochemistry, Biophysics and Biotechnology, Jagiellonian University, Kraków, Poland.
Jaźwa-Kusior A; Department of Medical Biotechnology, Faculty of Biochemistry, Biophysics and Biotechnology, Jagiellonian University, Kraków, Poland.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2020 Oct 15; Vol. 15 (10), pp. e0240691. Date of Electronic Publication: 2020 Oct 15 (Print Publication: 2020).
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Genetic Vectors/*metabolism
Heme Oxygenase-1/*deficiency
Inflammation/*pathology
Liver/*pathology
Macrophages/*pathology
Animals ; Cells, Cultured ; Chemokine CCL2/metabolism ; Dependovirus/metabolism ; Gene Expression Regulation ; Green Fluorescent Proteins/metabolism ; HEK293 Cells ; Heme Oxygenase-1/metabolism ; Humans ; Inflammation/blood ; Inflammation/genetics ; Interleukin-6/blood ; Kupffer Cells/pathology ; Mice, Inbred C57BL ; Mice, Knockout ; Phenotype ; Signal Transduction/genetics ; Toll-Like Receptor 9/metabolism ; Transgenes

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Skocz do pozycji: 47.

Tytuł:: Adamantane Containing Peptidoglycan Fragments Enhance RANTES and IL-6 Production in Lipopolysaccharide-Induced Macrophages.
Autorzy:: Manček-Keber M; Department of Synthetic Biology and Immunology, National Institute of Chemistry, Hajdrihova 19, P.O. Box 660, SI-1001 Ljubljana, Slovenia.; Centre of Excelence EN-FIST, SI-1000 Ljubljana, Slovenia.
Ribić R; University Center Varaždin, University North, Jurja Križanića 31b, HR-42 000 Varaždin, Croatia.
Chain F; Department of Organic and Macromolecular Chemistry, Ghent University, Krijgslaan 281 S4, 9000 Ghent, Belgium.
Sinnaeve D; Department of Organic and Macromolecular Chemistry, Ghent University, Krijgslaan 281 S4, 9000 Ghent, Belgium.; Univ. Lille, Inserm, Institut Pasteur de Lille, CHU Lille, U1167-Labex DISTALZ-RID-AGE-Risk Factors and Molecular Determinants of Aging-Related Diseases, F-59000 Lille, France.; CNRS, ERL9002-Integrative Structural Biology, F-59000 Lille, France.
Martins JC; Department of Organic and Macromolecular Chemistry, Ghent University, Krijgslaan 281 S4, 9000 Ghent, Belgium.
Jerala R; Department of Synthetic Biology and Immunology, National Institute of Chemistry, Hajdrihova 19, P.O. Box 660, SI-1001 Ljubljana, Slovenia.; Centre of Excelence EN-FIST, SI-1000 Ljubljana, Slovenia.
Tomić S; Department of Chemistry, Faculty of Science, University of Zagreb, Horvatovac 102A, HR-10 000 Zagreb, Croatia.
Fehér K; Heidelberg Institute for Theoretical Studies, Schloss-Wolfsbrunnenweg 35, 69118 Heidelberg, Germany.; Molecular Recognition and Interaction Research Group, Hungarian Academy of Sciences, University of Debrecen, Egyetem tér 1, H-4032 Debrecen, Hungary.; Pokaż więcej
Źródło:: Molecules (Basel, Switzerland) [Molecules] 2020 Aug 14; Vol. 25 (16). Date of Electronic Publication: 2020 Aug 14.
Typ publikacji:: Journal Article
MeSH Terms:: Adamantane/*chemistry
Chemokine CCL5/*metabolism
Interleukin-6/*metabolism
Lipopolysaccharides/*pharmacology
Macrophages/*metabolism
Peptidoglycan/*pharmacology
Animals ; Cells, Cultured ; Macrophages/drug effects ; Macrophages/immunology ; Mice ; Mice, Inbred C57BL ; Peptidoglycan/chemistry

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Skocz do pozycji: 48.

Tytuł:: Acotiamide attenuates central urocortin 2-induced intestinal inflammatory responses, and urocortin 2 treatment reduces TNF-α productions in LPS-stimulated macrophage cell lines.
Autorzy:: Yamawaki H; Division of Gastroenterology, Nippon Medical School, Tokyo, Japan.
Futagami S; Division of Gastroenterology, Nippon Medical School, Tokyo, Japan.
Sakasegawa N; Division of Gastroenterology, Nippon Medical School, Tokyo, Japan.
Murakami M; Division of Gastroenterology, Nippon Medical School, Tokyo, Japan.
Agawa S; Division of Gastroenterology, Nippon Medical School, Tokyo, Japan.
Ikeda G; Division of Gastroenterology, Nippon Medical School, Tokyo, Japan.
Noda H; Division of Gastroenterology, Nippon Medical School, Tokyo, Japan.
Kirita K; Division of Gastroenterology, Nippon Medical School, Tokyo, Japan.
Gudis K; Division of Gastroenterology, Nippon Medical School, Tokyo, Japan.
Higuchi K; Division of Gastroenterology, Nippon Medical School, Tokyo, Japan.
Kodaka Y; Division of Gastroenterology, Nippon Medical School, Tokyo, Japan.
Ueki N; Division of Gastroenterology, Nippon Medical School, Tokyo, Japan.
Iwakiri K; Division of Gastroenterology, Nippon Medical School, Tokyo, Japan.; Pokaż więcej
Źródło:: Neurogastroenterology and motility [Neurogastroenterol Motil] 2020 Aug; Vol. 32 (8), pp. e13813. Date of Electronic Publication: 2020 Feb 07.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Urocortins*
Benzamides/*pharmacology
Gastrointestinal Agents/*pharmacology
Inflammation/*drug therapy
Intestines/*drug effects
Macrophages/*drug effects
Thiazoles/*pharmacology
Tumor Necrosis Factor-alpha/*metabolism
Animals ; Benzamides/therapeutic use ; Cell Line ; Gastrointestinal Agents/therapeutic use ; Inflammation/chemically induced ; Inflammation/metabolism ; Interleukin-6/metabolism ; Lipopolysaccharides ; Macrophages/metabolism ; Male ; Phosphorylation/drug effects ; Rats ; Rats, Sprague-Dawley ; Signal Transduction/drug effects ; Thiazoles/therapeutic use

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Skocz do pozycji: 49.

Tytuł:: The membrane-type estrogen receptor G-protein-coupled estrogen receptor suppresses lipopolysaccharide-induced interleukin 6 via inhibition of nuclear factor-kappa B pathway in murine macrophage cells.
Autorzy:: Okamoto M; Laboratory of Veterinary Immunology, Department of Veterinary Medicine, School of Veterinary Medicine, Azabu University, Sagamihara, Japan.
Suzuki T; Laboratory of Veterinary Immunology, Department of Veterinary Medicine, School of Veterinary Medicine, Azabu University, Sagamihara, Japan.
Mizukami Y; Center for Gene Research, Yamaguchi University, Yamaguchi, Japan.
Ikeda T; Laboratory of Veterinary Immunology, Department of Veterinary Medicine, School of Veterinary Medicine, Azabu University, Sagamihara, Japan.; Pokaż więcej
Źródło:: Animal science journal = Nihon chikusan Gakkaiho [Anim Sci J] 2017 Nov; Vol. 88 (11), pp. 1870-1879. Date of Electronic Publication: 2017 Jul 18.
Typ publikacji:: Journal Article
MeSH Terms:: Interleukin-6/*genetics
Interleukin-6/*metabolism
Lipopolysaccharides/*adverse effects
Macrophages/*metabolism
NF-kappa B/*metabolism
Receptors, Estrogen/*physiology
Receptors, G-Protein-Coupled/*physiology
Signal Transduction/*genetics
Signal Transduction/*physiology
Animals ; Female ; Gene Expression/genetics ; Interleukin-6/physiology ; JNK Mitogen-Activated Protein Kinases/metabolism ; JNK Mitogen-Activated Protein Kinases/physiology ; Mice ; NF-kappa B/physiology ; RAW 264.7 Cells

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Skocz do pozycji: 50.

Tytuł:: IL-17-differentiated macrophages secrete pro-inflammatory cytokines in response to oxidized low-density lipoprotein.
Autorzy:: de la Paz Sánchez-Martínez M; Unidad de Investigación Médica en Inmunología, Hospital de Pediatría, Centro Médico Nacional Siglo XXI, Instituto Mexicano del Seguro Social, Av. Cuauhtémoc 330 Col. Doctores, 06720, Ciudad de México, Mexico.
Blanco-Favela F; Unidad de Investigación Médica en Inmunología, Hospital de Pediatría, Centro Médico Nacional Siglo XXI, Instituto Mexicano del Seguro Social, Av. Cuauhtémoc 330 Col. Doctores, 06720, Ciudad de México, Mexico.
Mora-Ruiz MD; Unidad de Investigación Médica en Inmunología, Hospital de Pediatría, Centro Médico Nacional Siglo XXI, Instituto Mexicano del Seguro Social, Av. Cuauhtémoc 330 Col. Doctores, 06720, Ciudad de México, Mexico.
Chávez-Rueda AK; Unidad de Investigación Médica en Inmunología, Hospital de Pediatría, Centro Médico Nacional Siglo XXI, Instituto Mexicano del Seguro Social, Av. Cuauhtémoc 330 Col. Doctores, 06720, Ciudad de México, Mexico.
Bernabe-García M; Unidad de Investigación Médica en Nutrición, Hospital de Pediatría, Centro Médico Nacional Siglo XXI, Instituto Mexicano del Seguro Social, Av. Cuauhtémoc 330 Col. Doctores, 06720, Ciudad de México, Mexico.
Chávez-Sánchez L; Unidad de Investigación Médica en Inmunología, Hospital de Pediatría, Centro Médico Nacional Siglo XXI, Instituto Mexicano del Seguro Social, Av. Cuauhtémoc 330 Col. Doctores, 06720, Ciudad de México, Mexico. .; Pokaż więcej
Źródło:: Lipids in health and disease [Lipids Health Dis] 2017 Oct 10; Vol. 16 (1), pp. 196. Date of Electronic Publication: 2017 Oct 10.
Typ publikacji:: Journal Article
MeSH Terms:: Gene Expression/*drug effects
Interleukin-17/*pharmacology
Interleukin-6/*metabolism
Lipoproteins, LDL/*pharmacology
Macrophages/*drug effects
Tumor Necrosis Factor-alpha/*metabolism
B7-1 Antigen/genetics ; B7-1 Antigen/immunology ; B7-2 Antigen/genetics ; B7-2 Antigen/immunology ; Cell Differentiation ; Humans ; Interleukin-6/genetics ; Interleukin-6/immunology ; Macrophages/cytology ; Macrophages/immunology ; Primary Cell Culture ; Toll-Like Receptor 2/genetics ; Toll-Like Receptor 2/immunology ; Toll-Like Receptor 4/genetics ; Toll-Like Receptor 4/immunology ; Tumor Necrosis Factor-alpha/genetics ; Tumor Necrosis Factor-alpha/immunology

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Skocz do pozycji: 51.

Tytuł:: Role of Oxygen Supply in Macrophages in a Model of Simulated Orthodontic Tooth Movement.
Autorzy:: Schröder A; Department of Orthodontics, University Hospital Regensburg, 93053 Regensburg, Germany.
Barschkies L; Department of Orthodontics, University Hospital Regensburg, 93053 Regensburg, Germany.
Jantsch J; Institute of Clinical Microbiology and Hygiene, University Hospital Regensburg, 93053 Regensburg, Germany.
Proff P; Department of Orthodontics, University Hospital Regensburg, 93053 Regensburg, Germany.
Gölz L; Department of Orthodontics, University of Erlangen-Nuremberg, 91054 Erlangen, Germany.
Deschner J; Department of Periodontology and Operative Dentistry, University of Mainz, 55131 Mainz, Germany.
Kirschneck C; Department of Orthodontics, University Hospital Regensburg, 93053 Regensburg, Germany.; Pokaż więcej
Źródło:: Mediators of inflammation [Mediators Inflamm] 2020 Jul 29; Vol. 2020, pp. 5802435. Date of Electronic Publication: 2020 Jul 29 (Print Publication: 2020).
Typ publikacji:: Journal Article
MeSH Terms:: Cell Hypoxia/*physiology
Macrophages/*metabolism
Animals ; DNA, Complementary/metabolism ; Enzyme-Linked Immunosorbent Assay ; Immunoblotting ; Interleukin-6/blood ; Mice ; RAW 264.7 Cells ; Real-Time Polymerase Chain Reaction ; Tumor Necrosis Factor-alpha/blood ; Vascular Endothelial Growth Factor A/blood

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Skocz do pozycji: 52.

Tytuł:: Beneficial effects of IL-4 and IL-6 on rat neonatal target cardiac cells.
Autorzy:: Zogbi C; Lab Genetics & Mol Cardiology/LIM 13, Heart Institute (InCor), University of São Paulo Medical School, Av Dr Eneas C Aguiar 44, Sao Paulo, SP, 05403-000, Brazil.
Oliveira NC; Lab Genetics & Mol Cardiology/LIM 13, Heart Institute (InCor), University of São Paulo Medical School, Av Dr Eneas C Aguiar 44, Sao Paulo, SP, 05403-000, Brazil.
Levy D; Lab Genetics & Mol Cardiology/LIM 13, Heart Institute (InCor), University of São Paulo Medical School, Av Dr Eneas C Aguiar 44, Sao Paulo, SP, 05403-000, Brazil.
Bydlowski SP; Lab Genetics & Mol Cardiology/LIM 13, Heart Institute (InCor), University of São Paulo Medical School, Av Dr Eneas C Aguiar 44, Sao Paulo, SP, 05403-000, Brazil.
Bassaneze V; Lab Genetics & Mol Cardiology/LIM 13, Heart Institute (InCor), University of São Paulo Medical School, Av Dr Eneas C Aguiar 44, Sao Paulo, SP, 05403-000, Brazil.
Neri EA; Lab Genetics & Mol Cardiology/LIM 13, Heart Institute (InCor), University of São Paulo Medical School, Av Dr Eneas C Aguiar 44, Sao Paulo, SP, 05403-000, Brazil.
Krieger JE; Lab Genetics & Mol Cardiology/LIM 13, Heart Institute (InCor), University of São Paulo Medical School, Av Dr Eneas C Aguiar 44, Sao Paulo, SP, 05403-000, Brazil. .; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2020 Jul 23; Vol. 10 (1), pp. 12350. Date of Electronic Publication: 2020 Jul 23.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Paracrine Communication*
Endothelial Cells/*metabolism
Interleukin-4/*metabolism
Interleukin-6/*metabolism
Macrophages/*metabolism
Myocardium/*metabolism
Animals ; Animals, Newborn ; Culture Media, Conditioned ; Endothelial Cells/cytology ; Macrophages/cytology ; Myocardium/cytology ; Myocytes, Cardiac ; Rats

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Skocz do pozycji: 53.

Tytuł:: Resolution-Associated Lactoferrin Peptides Limit LPS Signaling and Cytokine Secretion from Human Macrophages.
Autorzy:: Lutaty A; The Laboratory for Molecular Pathways in the Resolution of Inflammation, The Department of Human Biology, University of Haifa, Haifa 3498838, Israel.
Soboh S; The Laboratory for Molecular Pathways in the Resolution of Inflammation, The Department of Human Biology, University of Haifa, Haifa 3498838, Israel.
Schif-Zuck S; The Laboratory for Molecular Pathways in the Resolution of Inflammation, The Department of Human Biology, University of Haifa, Haifa 3498838, Israel.
Ariel A; The Laboratory for Molecular Pathways in the Resolution of Inflammation, The Department of Human Biology, University of Haifa, Haifa 3498838, Israel.; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2020 Jul 21; Vol. 21 (14). Date of Electronic Publication: 2020 Jul 21.
Typ publikacji:: Journal Article
MeSH Terms:: Cytokines/*metabolism
Extracellular Signal-Regulated MAP Kinases/*antagonists & inhibitors
Inflammation Mediators/*metabolism
Lactoferrin/*pharmacology
MAP Kinase Signaling System/*drug effects
Macrophages/*metabolism
Peptides/*metabolism
Animals ; Cell Line ; Humans ; Inflammation/metabolism ; Interleukin-10/metabolism ; Interleukin-6/metabolism ; JNK Mitogen-Activated Protein Kinases/metabolism ; Lipopolysaccharides/pharmacology ; Macrophage Activation/drug effects ; Mice ; Tumor Necrosis Factor-alpha/metabolism ; p38 Mitogen-Activated Protein Kinases/metabolism

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Skocz do pozycji: 54.

Tytuł:: Early modulation of macrophage ROS-PPARγ-NF-κB signalling by sonodynamic therapy attenuates neointimal hyperplasia in rabbits.
Autorzy:: Yao J; Department of Cardiology, The First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, 23 Youzheng Street, Harbin, 150001, China.
Zhao X; Department of Cardiology, The First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, 23 Youzheng Street, Harbin, 150001, China.
Tan F; Department of Pathophysiology and Key Laboratory of Cardiovascular Pathophysiology, Harbin Medical University, Key Laboratory of Cardiovascular Medicine Research (Harbin Medical University), Ministry of Education, Harbin, China.
Cao X; Department of Pathophysiology and Key Laboratory of Cardiovascular Pathophysiology, Harbin Medical University, Key Laboratory of Cardiovascular Medicine Research (Harbin Medical University), Ministry of Education, Harbin, China.
Guo S; Department of Cardiology, The First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, 23 Youzheng Street, Harbin, 150001, China.
Li X; Department of Pathophysiology and Key Laboratory of Cardiovascular Pathophysiology, Harbin Medical University, Key Laboratory of Cardiovascular Medicine Research (Harbin Medical University), Ministry of Education, Harbin, China.
Huang Z; Department of Cardiology, The First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, 23 Youzheng Street, Harbin, 150001, China.
Diabakte K; Department of Cardiology, The First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, 23 Youzheng Street, Harbin, 150001, China.
Wang L; Department of Cardiology, The First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, 23 Youzheng Street, Harbin, 150001, China.
Liu M; Department of Cardiology, The First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, 23 Youzheng Street, Harbin, 150001, China.
Shen Z; Department of Cardiology, The First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, 23 Youzheng Street, Harbin, 150001, China.
Li B; Department of Cardiology, The First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, 23 Youzheng Street, Harbin, 150001, China.
Cao Z; Department of Cardiology, The First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, 23 Youzheng Street, Harbin, 150001, China.
Sheng S; Department of Cardiology, The First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, 23 Youzheng Street, Harbin, 150001, China.
Lu M; Department of Pathophysiology and Key Laboratory of Cardiovascular Pathophysiology, Harbin Medical University, Key Laboratory of Cardiovascular Medicine Research (Harbin Medical University), Ministry of Education, Harbin, China.
Cao Y; Department of Cardiology, The First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, 23 Youzheng Street, Harbin, 150001, China.
Jin H; Medicine Department, Karolinska Institute, Stockholm, Sweden.
Zhang Z; Laboratory of Photo- and Sono-Theranostic Technologies and Condensed Matter Science and Technology Institute, Harbin Institute of Technology, Harbin, China.
Tian Y; Department of Cardiology, The First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, 23 Youzheng Street, Harbin, 150001, China. .; Department of Pathophysiology and Key Laboratory of Cardiovascular Pathophysiology, Harbin Medical University, Key Laboratory of Cardiovascular Medicine Research (Harbin Medical University), Ministry of Education, Harbin, China. .; Heilongjiang Academy of Medical Sciences, Harbin, China. .; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2020 Jul 15; Vol. 10 (1), pp. 11638. Date of Electronic Publication: 2020 Jul 15.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Hyperplasia/*drug therapy
Macrophages/*metabolism
NF-kappa B p50 Subunit/*metabolism
Neointima/*drug therapy
PPAR gamma/*metabolism
Reactive Oxygen Species/*metabolism
Animals ; Apoptosis ; Cell Line ; Cell Movement ; Cell Proliferation ; Cholesterol/blood ; Coculture Techniques ; Humans ; Inflammation/drug therapy ; Interleukin-6/metabolism ; Male ; Plaque, Atherosclerotic/drug therapy ; Rabbits ; Random Allocation ; Signal Transduction ; THP-1 Cells ; Tomography, Optical Coherence

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Skocz do pozycji: 55.

Tytuł:: All-Trans Retinoic Acid Enhances both the Signaling for Priming and the Glycolysis for Activation of NLRP3 Inflammasome in Human Macrophage.
Autorzy:: Alatshan A; Departments of Physiology, Faculty of Medicine, University of Debrecen, H-4012 Debrecen, Hungary.; Doctoral School of Molecular Cellular and Immune Biology, Faculty of Medicine, University of Debrecen, H-4012 Debrecen, Hungary.
Kovács GE; Departments of Physiology, Faculty of Medicine, University of Debrecen, H-4012 Debrecen, Hungary.; Doctoral School of Molecular Cellular and Immune Biology, Faculty of Medicine, University of Debrecen, H-4012 Debrecen, Hungary.
Aladdin A; Department of Medical Chemistry, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.; Doctoral School of Molecular Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
Czimmerer Z; Department of Biochemistry and Molecular Biology, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
Tar K; Department of Medical Chemistry, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
Benkő S; Departments of Physiology, Faculty of Medicine, University of Debrecen, H-4012 Debrecen, Hungary.; Doctoral School of Molecular Cellular and Immune Biology, Faculty of Medicine, University of Debrecen, H-4012 Debrecen, Hungary.; Pokaż więcej
Źródło:: Cells [Cells] 2020 Jul 01; Vol. 9 (7). Date of Electronic Publication: 2020 Jul 01.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Glycolysis/*drug effects
Inflammasomes/*immunology
Macrophages/*metabolism
NLR Family, Pyrin Domain-Containing 3 Protein/*metabolism
Signal Transduction/*drug effects
Tretinoin/*pharmacology
Hexokinase/metabolism ; Humans ; Interleukin-10/metabolism ; Interleukin-1beta/metabolism ; Interleukin-6/metabolism ; Lipopolysaccharides/pharmacology ; MAP Kinase Signaling System/immunology ; NF-kappa B/blood ; Proto-Oncogene Proteins c-akt/antagonists & inhibitors ; STAT3 Transcription Factor/antagonists & inhibitors ; Signal Transduction/immunology ; TOR Serine-Threonine Kinases/antagonists & inhibitors ; Tretinoin/metabolism

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Skocz do pozycji: 56.

Tytuł:: Epigallocatechin-3-Gallate (EGCG), an Active Compound of Green Tea Attenuates Acute Lung Injury Regulating Macrophage Polarization and Krüpple-Like-Factor 4 (KLF4) Expression.
Autorzy:: Almatroodi SA; Department of Medical Laboratories, College of Applied Medical Sciences, Qassim University, Buraydah 52571, Saudi Arabia.
Almatroudi A; Department of Medical Laboratories, College of Applied Medical Sciences, Qassim University, Buraydah 52571, Saudi Arabia.
Alsahli MA; Department of Medical Laboratories, College of Applied Medical Sciences, Qassim University, Buraydah 52571, Saudi Arabia.
Aljasir MA; Department of Medical Laboratories, College of Applied Medical Sciences, Qassim University, Buraydah 52571, Saudi Arabia.
Syed MA; Translational Research Lab, Department of Biotechnology, Faculty of Natural Sciences, Jamia Millia Islamia, New Delhi 110025, India.
Rahmani AH; Department of Medical Laboratories, College of Applied Medical Sciences, Qassim University, Buraydah 52571, Saudi Arabia.; Pokaż więcej
Źródło:: Molecules (Basel, Switzerland) [Molecules] 2020 Jun 20; Vol. 25 (12). Date of Electronic Publication: 2020 Jun 20.
Typ publikacji:: Journal Article
MeSH Terms:: Acute Lung Injury/*drug therapy
Anti-Inflammatory Agents/*pharmacology
Catechin/*analogs & derivatives
Kruppel-Like Transcription Factors/*metabolism
Macrophages/*metabolism
Tea/*chemistry
8-Hydroxy-2'-Deoxyguanosine/metabolism ; Acute Lung Injury/chemically induced ; Acute Lung Injury/enzymology ; Acute Lung Injury/physiopathology ; Animals ; Anti-Inflammatory Agents/administration & dosage ; Arginase/metabolism ; Catechin/administration & dosage ; Catechin/pharmacology ; Cell Proliferation/drug effects ; Interleukin-1beta/metabolism ; Interleukin-6/metabolism ; Ki-67 Antigen/metabolism ; Kruppel-Like Factor 4 ; Lectins/metabolism ; Lipopolysaccharides/pharmacology ; Mice ; Mice, Inbred C57BL ; Nitric Oxide Synthase Type II/genetics ; Nitric Oxide Synthase Type II/metabolism ; Peroxidase/metabolism ; Proliferating Cell Nuclear Antigen/metabolism ; Triggering Receptor Expressed on Myeloid Cells-1/metabolism ; Tumor Necrosis Factor-alpha/metabolism ; Tyrosine/analogs & derivatives ; Tyrosine/metabolism ; beta-N-Acetylhexosaminidases/metabolism

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Skocz do pozycji: 57.

Tytuł:: Immune activation of murine RAW264.7 macrophages by sonicated and alkalized paramylon from Euglena gracilis.
Autorzy:: Guo Q; Shenzhen Key Laboratory of Marine Bioresources and Eco-environmental Science, Shenzhen Engineering Laboratory for Marine Algal Biotechnology, Guangdong Provincial Key Laboratory for Plant Epigenetics, College of Life Sciences and Oceanography, Shenzhen University, Shenzhen, 518060, China.
Bi D; Shenzhen Key Laboratory of Marine Bioresources and Eco-environmental Science, Shenzhen Engineering Laboratory for Marine Algal Biotechnology, Guangdong Provincial Key Laboratory for Plant Epigenetics, College of Life Sciences and Oceanography, Shenzhen University, Shenzhen, 518060, China.
Wu M; Shenzhen Key Laboratory of Marine Bioresources and Eco-environmental Science, Shenzhen Engineering Laboratory for Marine Algal Biotechnology, Guangdong Provincial Key Laboratory for Plant Epigenetics, College of Life Sciences and Oceanography, Shenzhen University, Shenzhen, 518060, China.; College of Food Engineering and Biotechnology, Hanshan Normal University, Chaozhou, 521041, China.; College of Physics and Optoelectronic Engineering, Shenzhen University, Shenzhen, 518060, China.
Yu B; Shenzhen Key Laboratory of Marine Bioresources and Eco-environmental Science, Shenzhen Engineering Laboratory for Marine Algal Biotechnology, Guangdong Provincial Key Laboratory for Plant Epigenetics, College of Life Sciences and Oceanography, Shenzhen University, Shenzhen, 518060, China.
Hu L; Shenzhen Key Laboratory of Marine Bioresources and Eco-environmental Science, Shenzhen Engineering Laboratory for Marine Algal Biotechnology, Guangdong Provincial Key Laboratory for Plant Epigenetics, College of Life Sciences and Oceanography, Shenzhen University, Shenzhen, 518060, China.
Liu C; Shenzhen Key Laboratory of Marine Bioresources and Eco-environmental Science, Shenzhen Engineering Laboratory for Marine Algal Biotechnology, Guangdong Provincial Key Laboratory for Plant Epigenetics, College of Life Sciences and Oceanography, Shenzhen University, Shenzhen, 518060, China.
Gu L; Shenzhen Key Laboratory of Marine Bioresources and Eco-environmental Science, Shenzhen Engineering Laboratory for Marine Algal Biotechnology, Guangdong Provincial Key Laboratory for Plant Epigenetics, College of Life Sciences and Oceanography, Shenzhen University, Shenzhen, 518060, China.
Zhu H; College of Food Engineering and Biotechnology, Hanshan Normal University, Chaozhou, 521041, China.
Lei A; Shenzhen Key Laboratory of Marine Bioresources and Eco-environmental Science, Shenzhen Engineering Laboratory for Marine Algal Biotechnology, Guangdong Provincial Key Laboratory for Plant Epigenetics, College of Life Sciences and Oceanography, Shenzhen University, Shenzhen, 518060, China.
Xu X; Shenzhen Key Laboratory of Marine Bioresources and Eco-environmental Science, Shenzhen Engineering Laboratory for Marine Algal Biotechnology, Guangdong Provincial Key Laboratory for Plant Epigenetics, College of Life Sciences and Oceanography, Shenzhen University, Shenzhen, 518060, China. .
Wang J; Shenzhen Key Laboratory of Marine Bioresources and Eco-environmental Science, Shenzhen Engineering Laboratory for Marine Algal Biotechnology, Guangdong Provincial Key Laboratory for Plant Epigenetics, College of Life Sciences and Oceanography, Shenzhen University, Shenzhen, 518060, China. .; Pokaż więcej
Źródło:: BMC microbiology [BMC Microbiol] 2020 Jun 19; Vol. 20 (1), pp. 171. Date of Electronic Publication: 2020 Jun 19.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Euglena gracilis/*metabolism
Glucans/*pharmacology
Macrophages/*parasitology
Signal Transduction/*drug effects
Animals ; Dose-Response Relationship, Drug ; Glucans/immunology ; Interleukin-6/metabolism ; Macrophage Activation ; Macrophages/immunology ; Mice ; Mitogen-Activated Protein Kinases/metabolism ; NF-kappa B/metabolism ; Nitric Oxide Synthase Type II/metabolism ; RAW 264.7 Cells ; Sonication ; Tumor Necrosis Factor-alpha/metabolism

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Skocz do pozycji: 58.

Tytuł:: Bicarbonate enhances the inflammatory response by activating JAK/STAT signalling in LPS + IFN-γ-stimulated macrophages.
Autorzy:: Kawakami T; Department of Cardiovascular Pharmacotherapy and Toxicology.; Department of Pathobiochemistry, Osaka University of Pharmaceutical Sciences, 4-20-1 Nasahara, Takatsuki, Osaka 569-1094, Japan.
Koike A; Department of Pathobiochemistry, Osaka University of Pharmaceutical Sciences, 4-20-1 Nasahara, Takatsuki, Osaka 569-1094, Japan.
Maehara T; Department of Pathobiochemistry, Osaka University of Pharmaceutical Sciences, 4-20-1 Nasahara, Takatsuki, Osaka 569-1094, Japan.
Hayashi T; Department of Cardiovascular Pharmacotherapy and Toxicology.
Fujimori K; Department of Pathobiochemistry, Osaka University of Pharmaceutical Sciences, 4-20-1 Nasahara, Takatsuki, Osaka 569-1094, Japan.; Pokaż więcej
Źródło:: Journal of biochemistry [J Biochem] 2020 Jun 01; Vol. 167 (6), pp. 623-631.
Typ publikacji:: Journal Article
MeSH Terms:: Interferon-gamma/*pharmacology
Janus Kinases/*metabolism
Lipopolysaccharides/*pharmacology
Macrophages/*drug effects
Macrophages/*metabolism
STAT1 Transcription Factor/*metabolism
Signal Transduction/*drug effects
Sodium Bicarbonate/*pharmacology
Animals ; Cyclooxygenase 2/genetics ; Gene Expression/drug effects ; Inflammation/chemically induced ; Inflammation/genetics ; Interleukin-6/genetics ; Janus Kinase Inhibitors/pharmacology ; Janus Kinases/antagonists & inhibitors ; Macrophage Activation/drug effects ; Male ; Mice ; Mice, Inbred C57BL ; Nitric Oxide/genetics ; Piperidines/pharmacology ; Pyrimidines/pharmacology ; RAW 264.7 Cells ; Recombinant Proteins/pharmacology

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Skocz do pozycji: 59.

Tytuł:: Role of microRNA-21 and Its Underlying Mechanisms in Inflammatory Responses in Diabetic Wounds.
Autorzy:: Liechty C; Laboratory for Fetal and Regenerative Biology, Department of Surgery, University of Colorado Denver-Anschutz Medical Campus and Children's Hospital Colorado, Aurora, CO 80045, USA.
Hu J; Laboratory for Fetal and Regenerative Biology, Department of Surgery, University of Colorado Denver-Anschutz Medical Campus and Children's Hospital Colorado, Aurora, CO 80045, USA.
Zhang L; Laboratory for Fetal and Regenerative Biology, Department of Surgery, University of Colorado Denver-Anschutz Medical Campus and Children's Hospital Colorado, Aurora, CO 80045, USA.
Liechty KW; Laboratory for Fetal and Regenerative Biology, Department of Surgery, University of Colorado Denver-Anschutz Medical Campus and Children's Hospital Colorado, Aurora, CO 80045, USA.
Xu J; Laboratory for Fetal and Regenerative Biology, Department of Surgery, University of Colorado Denver-Anschutz Medical Campus and Children's Hospital Colorado, Aurora, CO 80045, USA.; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2020 May 08; Vol. 21 (9). Date of Electronic Publication: 2020 May 08.
Typ publikacji:: Journal Article
MeSH Terms:: Diabetes Mellitus, Experimental/*metabolism
Macrophage Activation/*immunology
Macrophages/*metabolism
MicroRNAs/*metabolism
Signal Transduction/*genetics
Wound Healing/*genetics
Animals ; Diabetes Mellitus, Experimental/genetics ; Female ; Gene Expression Regulation/genetics ; Humans ; Hyperglycemia/metabolism ; Inflammation/genetics ; Inflammation/immunology ; Inflammation/metabolism ; Interleukin-1beta/metabolism ; Interleukin-6/metabolism ; Interleukin-8/metabolism ; Mice ; Mice, Inbred C57BL ; MicroRNAs/genetics ; Middle Aged ; NADPH Oxidase 2/metabolism ; Nitric Oxide Synthase Type II/metabolism ; Phosphatidylinositol 3-Kinases/metabolism ; Reactive Oxygen Species/metabolism ; Tumor Necrosis Factor-alpha/metabolism ; Up-Regulation

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Skocz do pozycji: 60.

Tytuł:: Angiotensin II induces RAW264.7 macrophage polarization to the M1‑type through the connexin 43/NF‑κB pathway.
Autorzy:: Wu L; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Medicine School of Shihezi University, Shihezi, Xinjiang 832008, P.R. China.
Chen K; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Medicine School of Shihezi University, Shihezi, Xinjiang 832008, P.R. China.
Xiao J; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Medicine School of Shihezi University, Shihezi, Xinjiang 832008, P.R. China.
Xin J; The 3rd Department of Cardiology, The First Affiliated Hospital of The Medical College, Shihezi University, Shihezi, Xinjiang 832008, P.R. China.
Zhang L; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Medicine School of Shihezi University, Shihezi, Xinjiang 832008, P.R. China.
Li X; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Medicine School of Shihezi University, Shihezi, Xinjiang 832008, P.R. China.
Li L; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Medicine School of Shihezi University, Shihezi, Xinjiang 832008, P.R. China.
Si J; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Medicine School of Shihezi University, Shihezi, Xinjiang 832008, P.R. China.
Wang L; The 3rd Department of Cardiology, The First Affiliated Hospital of The Medical College, Shihezi University, Shihezi, Xinjiang 832008, P.R. China.
Ma K; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Medicine School of Shihezi University, Shihezi, Xinjiang 832008, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2020 May; Vol. 21 (5), pp. 2103-2112. Date of Electronic Publication: 2020 Mar 12.
Typ publikacji:: Journal Article
MeSH Terms:: Angiotensin II/*pharmacology
Connexin 43/*metabolism
Macrophage Activation/*drug effects
Macrophages/*metabolism
NF-kappa B/*metabolism
RAW 264.7 Cells/*drug effects
Animals ; Cell Polarity/drug effects ; Cytokines/metabolism ; Interleukin-1beta/metabolism ; Interleukin-6/metabolism ; Mice ; Nitric Oxide Synthase Type II/metabolism ; Signal Transduction/drug effects ; Transcription Factor RelA/metabolism ; Tumor Necrosis Factor-alpha/metabolism

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