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Wyszukujesz frazę ""Intestinal Mucosa metabolism"" wg kryterium: Temat


Tytuł :
Structural assembly of the megadalton-sized receptor for intestinal vitamin B12 uptake and kidney protein reabsorption
Autorzy :
Larsen, Casper
Etzerodt, Anders
Madsen, Mette
Skj��dt, Karsten
Moestrup, S��ren Kragh
Andersen, Christian Brix Folsted
Pokaż więcej
Temat :
Science
Article
ddc:500
lcsh:Science
lcsh:Q
Albumins/metabolism
Amino Acid Sequence
Anemia, Megaloblastic/genetics
Animals
CHO Cells
Cricetulus
Crystallography, X-Ray
Humans
Intestinal Mucosa/metabolism
Kidney/metabolism
Malabsorption Syndromes/genetics
Mutation
Protein Binding
Protein Conformation
Proteins/chemistry
Proteinuria/genetics
Receptors, Cell Surface/chemistry
Sequence Homology, Amino Acid
Vitamin B 12 Deficiency/genetics
Vitamin B 12/metabolism
General Physics and Astronomy
General Biochemistry, Genetics and Molecular Biology
General Chemistry
Amnionless
Transmembrane protein
Chemistry
Cubilin
Protein structure
Cell biology
Endocytic cycle
Transport protein
Cubam
Kidney metabolism
Źródło :
Nature Communications, Vol 9, Iss 1, Pp 1-11 (2018)
Nature Communications
Nature Communications 9(1), 5204 (2018). doi:10.1038/s41467-018-07468-4
Larsen, C, Etzerodt, A, Madsen, M, Skjødt, K, Moestrup, S K & Andersen, C B F 2018, ' Structural assembly of the megadalton-sized receptor for intestinal vitamin B 12 uptake and kidney protein reabsorption ', Nature Communications, vol. 9, 5204 . https://doi.org/10.1038/s41467-018-07468-4
Larsen, C, Etzerodt, A, Madsen, M, Skjødt, K, Moestrup, S K & Andersen, C B F 2018, ' Structural assembly of the megadalton-sized receptor for intestinal vitamin B 12 uptake and kidney protein reabsorption ', Nature Communications, vol. 9, no. 1, 5204 . https://doi.org/10.1038/s41467-018-07468-4
'Nature Communications ', vol: 9, pages: 5204-1-5204-11 (2018)
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0a72414698d05d7307c1845d044d2257
https://doaj.org/article/80fcc40c89f8413ba5eb477b1dfa5777
Tytuł :
Modulation of the fungal mycobiome is regulated by the chitin-binding receptor FIBCD1
Autorzy :
Moeller, Jesper B.
Leonardi, Irina
Schlosser, Anders
Flamar, Anne-Laure
Bessman, Nicholas J.
Putzel, Gregory Garbès
Thomsen, Theresa
Hammond, Mark
Jepsen, Christine S.
Skjødt, Karsten
Füchtbauer, Ernst-Martin
Farber, Donna L.
Sorensen, Grith L.
Iliev, Iliyan D.
Holmskov, Uffe
Artis, David
Pokaż więcej
Temat :
Research Articles
Brief Definitive Report
Immunology
Immunology and Allergy
Microbiology
Dysbiosis
medicine.disease
medicine
Chitin binding
Biology
Cell wall
Inflammation
medicine.symptom
Receptor
Pattern recognition receptor
Chitin
chemistry.chemical_compound
chemistry
Immune system
COLITIS
DOMAIN-CONTAINING 1
DYSBIOSIS
EXPRESSION
IMMUNITY
INFLAMMATORY RESPONSES
INNATE LYMPHOID-CELLS
PCR
SUSCEPTIBILITY
TRICHURIS-MURIS
Metagenomics
Humans
RNA, Ribosomal, 16S
Microbial Interactions
Disease Models, Animal
Gene Expression
DNA, Ribosomal Spacer
Mycobiome
Gastrointestinal Microbiome
Mice, Transgenic
Animals
Enteritis/etiology
Fungi/physiology
Intestinal Mucosa/metabolism
Protein Binding
Receptors, Cell Surface/metabolism
Mice
Chitin/metabolism
fungi
Źródło :
Moeller, J B, Leonardi, I, Schlosser, A, Flamar, A-L, Bessman, N J, Putzel, G G, Thomsen, T, Hammond, M, Jepsen, C S, Skjødt, K, Füchtbauer, E-M, Farber, D L, Sorensen, G L, Iliev, I D, Holmskov, U & Artis, D 2019, ' Modulation of the fungal mycobiome is regulated by the chitin-binding receptor FIBCD1 ', The Journal of Experimental Medicine, vol. 216, no. 12, pp. 2689-2700 . https://doi.org/10.1084/jem.20182244
The Journal of Experimental Medicine
Moeller, J B, Leonardi, I, Schlosser, A, Flamar, A L, Bessman, N J, Putzel, G G, Thomsen, T, Hammond, M, Jepsen, C S, Skjødt, K, Füchtbauer, E M, Farber, D L, Sorensen, G L, Iliev, I D, Holmskov, U & Artis, D 2019, ' Modulation of the fungal mycobiome is regulated by the chitin-binding receptor FIBCD1 ', The Journal of Experimental Medicine, vol. 216, no. 12, pp. 2689-2700 . https://doi.org/10.1084/jem.20182244
Opis pliku :
application/octet-stream
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::85339131815355f437b5bc445395583b
https://findresearcher.sdu.dk:8443/ws/files/169995901/Open_Access_version
Tytuł :
NLRP1 restricts butyrate producing commensals to exacerbate inflammatory bowel disease
Autorzy :
Tye, Hazel
Yu, Chien-Hsiung
Simms, Lisa A.
de Zoete, Marcel R.
Kim, Man Lyang
Zakrzewski, Martha
Penington, Jocelyn S.
Harapas, Cassandra R.
Souza-Fonseca-Guimaraes, Fernando
Wockner, Leesa F.
Preaudet, Adele
Mielke, Lisa A.
Wilcox, Stephen A.
Ogura, Yasunori
Corr, Sinead C.
Kanojia, Komal
Kouremenos, Konstantinos A.
De Souza, David P.
McConville, Malcolm J.
Flavell, Richard A.
Gerlic, Motti
Kile, Benjamin T.
Papenfuss, Anthony T.
Putoczki, Tracy L.
Radford-Smith, Graham L.
Masters, Seth L.
Pokaż więcej
Temat :
Adaptor Proteins, Signal Transducing/genetics
Animals
Apoptosis Regulatory Proteins/genetics
Butyrates/metabolism
Clostridiales
Colitis/metabolism
Colon/pathology
Female
Gastrointestinal Microbiome
Gene Deletion
Humans
Inflammasomes
Inflammatory Bowel Diseases/drug therapy
Interferon-gamma/metabolism
Interleukin-18/metabolism
Intestinal Mucosa/metabolism
Male
Mice
Mice, Inbred C57BL
Mice, Transgenic
Rectum/metabolism
Signal Transduction
T-Lymphocytes/cytology
Vancomycin/pharmacology
lcsh:Science
lcsh:Q
Adaptor Proteins
Signal Transducing/genetics
Inbred C57BL
Transgenic
General Physics and Astronomy
General Biochemistry, Genetics and Molecular Biology
General Chemistry
Inflammasome
medicine.drug
medicine
Colitis
medicine.disease
business.industry
business
Ulcerative colitis
Pyroptosis
Inflammatory bowel disease
Intestinal mucosa
Innate immune system
Immunology
Butyrate
Science
Źródło :
Nature Communications, 9(1)
Nature Communications, Vol 9, Iss 1, Pp 1-11 (2018)
Opis pliku :
image/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::289d7d1c6f61238bb26bb3d2ff10b06a
https://dspace.library.uu.nl/handle/1874/376771
Tytuł :
Firewalls prevent systemic dissemination of vectors derived from human adenovirus type 5 and suppress production of transgene-encoded antigen in a murine model of oral vaccination
Autorzy :
Julien Revaud
Yves Unterfinger
Nicolas Rol
Muhammad Suleman
Julia Shaw
Sandra Galea
Françoise Gavard
Sandrine A. Lacour
Muriel Coulpier
Nicolas Versillé
Menzo Havenga
Bernard Klonjkowski
Gina Zanella
Stéphane Biacchesi
Nathalie Cordonnier
Blaise Corthésy
Juliette Ben Arous
Jennifer P. Richardson
Pokaż więcej
Temat :
oral vaccination
viral vector
adenovirus
M cell
Peyer's patch
Microbiology
Original Research
Adenoviruses, Human/genetics
Adenoviruses, Human/immunology
Administration, Oral
Animals
Female
Gene Expression
Genes, Reporter
Genetic Vectors/administration & dosage
Genetic Vectors/genetics
Genetic Vectors/immunology
Humans
Immunization
Intestinal Mucosa/immunology
Intestinal Mucosa/metabolism
Mice
Organ Specificity
Peyer's Patches/immunology
Peyer's Patches/metabolism
Phagocytes/metabolism
Protein Transport
Transgenes/genetics
Transgenes/immunology
Vaccination
lcsh:Microbiology
lcsh:QR1-502
[SDV]Life Sciences [q-bio]
Infectious Diseases
Microbiology (medical)
Immunology
Intestinal epithelium
Microfold cell
Viral vector
Biology
Intestinal mucosa
Epithelium
medicine.anatomical_structure
medicine
Luciferase
Cell biology
Antigen
Źródło :
Frontiers in Cellular and Infection Microbiology (8), 1-15. (2018)
Frontiers in Cellular and Infection Microbiology
Frontiers in cellular and infection microbiology, vol. 8, pp. 6
Frontiers in Cellular and Infection Microbiology, Vol 8 (2018)
Frontiers in Cellular and Infection Microbiology, Frontiers, 2018, 8, pp.1-15. ⟨10.3389/fcimb.2018.00006⟩
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1f353300351326b915fbe3cd0e13a784
http://prodinra.inra.fr/record/425674
Tytuł :
Myeloid differentiation primary response gene (MyD) 88 signalling is not essential for intestinal fibrosis development
Autorzy :
Lutz, C.
Weder, B.
Hünerwadel, A.
Fagagnini, S.
Lang, B.
Beerenwinkel, N.
Rossel, J. B.
Rogler, G.
Misselwitz, B.
Hausmann, M.
Pokaż więcej
Temat :
Multidisciplinary
Green fluorescent protein
Inflammation
medicine.symptom
medicine
Immune system
business.industry
business
Transplantation
Fibrosis
medicine.disease
Matrix metalloproteinase
Sirius Red
chemistry.chemical_compound
chemistry
Inflammatory bowel disease
Pathology
medicine.medical_specialty
Gastrointestinal models
Intestinal diseases
lcsh:Medicine
lcsh:R
lcsh:Science
lcsh:Q
Article
Clinic for Gastroenterology and Hepatology
610 Medicine & health
Animals
Collagen/metabolism
Disease Models, Animal
Fibrosis/metabolism
Fibrosis/pathology
Inflammation/metabolism
Inflammation/pathology
Intestinal Mucosa/metabolism
Intestines/pathology
Matrix Metalloproteinase 9/metabolism
Mice
Mice, Inbred C57BL
Myeloid Differentiation Factor 88/metabolism
Neutrophils/metabolism
Neutrophils/pathology
Signal Transduction/physiology
Transforming Growth Factor beta1/metabolism
Źródło :
Scientific Reports, Vol 7, Iss 1, Pp 1-10 (2017)
Scientific Reports
Lutz, C; Weder, B; Hünerwadel, A; Fagagnini, S; Lang, B; Beerenwinkel, N; Rossel, J B; Rogler, G; Misselwitz, B; Hausmann, M (2017). Myeloid differentiation primary response gene (MyD) 88 signalling is not essential for intestinal fibrosis development. Scientific Reports, 7:17678.
Scientific Reports, 7 (1)
Scientific reports, vol. 7, no. 1, pp. 17678
Opis pliku :
application/pdf; application/application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d8e45e447b5914428033cacd0dd485ba
Tytuł :
PPARγ Modulates Long Chain Fatty Acid Processing in the Intestinal Epithelium
Autorzy :
Walter Wahli
Jürgen König
Cédric Le May
Matej Orešič
Kalina Duszka
Pokaż więcej
Temat :
intestine
lipid metabolism
PPARγ
[SDV]Life Sciences [q-bio]
ta1182
Article
[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]
[SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterology
[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism
[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular system
Animals
Fatty Acids/metabolism
Intestinal Mucosa/metabolism
Lipid Metabolism/genetics
Lipid Metabolism/physiology
Male
Mice
Mice, Knockout
PPAR gamma/genetics
PPAR gamma/metabolism
lcsh:Biology (General)
lcsh:QH301-705.5
lcsh:Chemistry
lcsh:QD1-999
Inorganic Chemistry
Organic Chemistry
Physical and Theoretical Chemistry
Computer Science Applications
Spectroscopy
Molecular Biology
General Medicine
Catalysis
Intestinal epithelium
Lipid metabolism
Knockout mouse
Endocrinology
medicine.medical_specialty
medicine
Gastric inhibitory polypeptide
Unsaturated fatty acid
Stomach emptying
Biology
Long chain fatty acid
Incretin
Internal medicine
Źródło :
International Journal of Molecular Sciences
International Journal of Molecular Sciences, MDPI, 2017, Equipe IV, 18 (12), ⟨10.3390/ijms18122559⟩
International Journal of Molecular Sciences 12 (18), . (2017)
International Journal of Molecular Sciences, MDPI, 2017, 18 (12), pp.2559. ⟨10.3390/ijms18122559⟩
International journal of molecular sciences, vol. 18, no. 12, pp. E2559
International Journal of Molecular Sciences, Vol 18, Iss 12, p 2559 (2017)
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c350ee02cfab0141fe0b273400b50f9c
https://hal.archives-ouvertes.fr/hal-01832934
Tytuł :
Combined bioavailable isoflavones and probiotics improve bone status and estrogen metabolism in postmenopausal osteopenic women: a randomized controlled trial
Autorzy :
Lambert, Max Norman Tandrup
Thybo, Catrine Bundgaard
Lykkeboe, Simon
Rasmussen, Lars Melholt
Frette, Xavier
Christensen, Lars Porskjær
Jeppesen, Per Bendix
Pokaż więcej
Temat :
Bioavailability
Bone density
Bone turnover
Equol
Isoflavone
Osteopenia
Postmenopause
Probiotics
Osteoporosis, Postmenopausal/metabolism
Bone Density Conservation Agents/pharmacology
Humans
Middle Aged
Biological Availability
Peptides/blood
Trifolium/chemistry
Bone Diseases, Metabolic/drug therapy
Female
Collagen Type I/blood
Phytotherapy
Plant Extracts/pharmacology
Bone and Bones/drug effects
Isoflavones/blood
Double-Blind Method
Probiotics/therapeutic use
Gastrointestinal Microbiome
Intestines/drug effects
Estrogens/deficiency
Selective Estrogen Receptor Modulators/pharmacology
Bone Density/drug effects
Intestinal Mucosa/metabolism
Bone Remodeling/drug effects
Drug Combinations
Journal Article
Nutrition and Dietetics
Medicine (miscellaneous)
Bone remodeling
Osteoporosis
medicine.disease
medicine
Estrogen receptor
chemistry.chemical_compound
chemistry
Endocrinology
medicine.medical_specialty
Estrogen
medicine.drug_class
Internal medicine
business.industry
business
Calcitriol
medicine.drug
Źródło :
Lambert, M N T, Thybo, C B, Lykkeboe, S, Rasmussen, L M, Fretté, X, Porskjær Christensen, L & Jeppesen, P B 2017, ' Combined bioavailable isoflavones and probiotics improve bone status and estrogen metabolism in postmenopausal osteopenic women: a randomized controlled trial ', American Journal of Clinical Nutrition . https://doi.org/10.3945/ajcn.117.153353
Lambert, M N T, Thybo, C B, Lykkeboe, S, Rasmussen, L M, Fretté, X, Christensen, L P & Jeppesen, P B 2017, ' Combined bioavailable isoflavones and probiotics improve bone status and estrogen metabolism in post-menopausal osteopenic women: a randomized controlled trial ', American Journal of Clinical Nutrition, vol. 106, no. 3, pp. 909-920 . https://doi.org/10.3945/ajcn.117.153353
Lambert, M N T, Thybo, C B, Lykkeboe, S, Rasmussen, L M, Frette, X, Christensen, L P & Jeppesen, P B 2017, ' Combined bioavailable isoflavones and probiotics improve bone status and estrogen metabolism in postmenopausal osteopenic women : a randomized controlled trial ', The American Journal of Clinical Nutrition, vol. 106, no. 3, pp. 909-920 . https://doi.org/10.3945/ajcn.117.153353
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b986b8e618f81e2d874a8ac70951e57a
https://pure.au.dk/portal/da/publications/combined-bioavailable-isoflavones-and-probiotics-improve-bone-status-and-estrogen-metabolism-in-postmenopausal-osteopenic-women-a-randomized-controlled-trial(59972fa3-fb79-4ac4-b81d-b292d6db5dee).html
Tytuł :
Oxygen-sensing neurons reciprocally regulate peripheral lipid metabolism via neuropeptide signaling in Caenorhabditis elegans
Autorzy :
Harkaranveer Ratanpal
Rosalind Hussey
Erik Vanstrum
Supriya Srinivasan
Emily Witham
Jaleh Mesgarzadeh
Pokaż więcej
Temat :
Lipid metabolism
Biology
Peripheral
Genetic screen
Neuropeptide
Caenorhabditis elegans
biology.organism_classification
Cell biology
Sensory system
Receptor
Regulator
Biochemistry
Research Article
Physical Sciences
Chemistry
Chemical Elements
Oxygen
Biology and Life Sciences
Cell Biology
Cellular Types
Animal Cells
Neurons
Neuroscience
Cellular Neuroscience
Lipids
Fats
Anatomy
Biological Tissue
Adipose Tissue
Medicine and Health Sciences
Metabolism
Oxygen Metabolism
Research and Analysis Methods
Experimental Organism Systems
Model Organisms
Caenorhabditis Elegans
Animal Models
Organisms
Eukaryota
Animals
Invertebrates
Nematoda
Caenorhabditis
Biotechnology
Genetic Engineering
Genetically Modified Organisms
Genetically Modified Animals
Molecular Biology
Molecular Biology Techniques
Genotyping
Automated Fluorescent Genotyping
lcsh:Genetics
lcsh:QH426-470
Caenorhabditis elegans/metabolism
Cell Communication
Guanylate Cyclase/metabolism
Intestinal Mucosa/metabolism
Lipid Metabolism
Neuropeptides/metabolism
Oxygen/metabolism
Sensory Receptor Cells/metabolism
Signal Transduction
Cancer Research
Genetics(clinical)
Genetics
Ecology, Evolution, Behavior and Systematics
Sensory Receptor Cells
Signal transduction
Cell signaling
Homeostasis
nervous system
Źródło :
PLoS Genetics
PLoS Genetics, Vol 14, Iss 3, p e1007305 (2018)
Hussey, R, Littlejohn, N K, Witham, E, Vanstrum, E, Mesgarzadeh, J, Ratanpal, H & Srinivasan, S 2018, ' Oxygen-sensing neurons reciprocally regulate peripheral lipid metabolism via neuropeptide signaling in Caenorhabditis elegans ', PLoS Genetics, vol. 14, no. 3, e1007305 . https://doi.org/10.1371/journal.pgen.1007305
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ab53a415685e15d5af84d08d59621622
https://syndication.highwire.org/content/doi/10.1101/142422
Tytuł :
Pheromone-sensing neurons regulate peripheral lipid metabolism in Caenorhabditis elegans
Autorzy :
Hussey, Rosalind
Stieglitz, Jon
Mesgarzadeh, Jaleh
Locke, Tiffany T.
Zhang, Ying K.
Schroeder, Frank C.
Srinivasan, Supriya
Pokaż więcej
Temat :
Acetylcholine/metabolism
Animals
Caenorhabditis elegans/genetics
Caenorhabditis elegans Proteins/genetics
Cyclic AMP/metabolism
GTP-Binding Protein alpha Subunits, Gi-Go/genetics
Intestinal Mucosa/metabolism
Lipid Metabolism
Pheromones/metabolism
Second Messenger Systems
Sensory Receptor Cells/metabolism
Research Article
Biology and Life Sciences
Anatomy
Biological Tissue
Adipose Tissue
Medicine and Health Sciences
Cell Biology
Cellular Types
Animal Cells
Neurons
Neuroscience
Cellular Neuroscience
Biochemistry
Lipids
Fats
Digestive System
Gastrointestinal Tract
Research and Analysis Methods
Experimental Organism Systems
Model Organisms
Caenorhabditis Elegans
Animal Models
Organisms
Invertebrates
Nematoda
Caenorhabditis
Oils
Biotechnology
Genetic Engineering
Genetically Modified Organisms
Genetically Modified Animals
Pheromones
Cancer Research
Genetics(clinical)
Genetics
Molecular Biology
Ecology, Evolution, Behavior and Systematics
Caenorhabditis elegans
biology.organism_classification
biology
Acetylcholine
medicine.drug
medicine
Cell biology
Lipid metabolism
Adipose tissue
Second messenger system
Regulator
Population
education.field_of_study
education
Genetic screen
lcsh:Genetics
lcsh:QH426-470
Źródło :
Hussey, R, Stieglitz, J, Mesgarzadeh, J, Locke, T T, Zhang, Y K, Schroeder, F C & Srinivasan, S 2017, ' Pheromone-sensing neurons regulate peripheral lipid metabolism in Caenorhabditis elegans ', PLoS Genetics, vol. 13, no. 5, e1006806 . https://doi.org/10.1371/journal.pgen.1006806
PLoS Genetics
PLoS Genetics, Vol 13, Iss 5, p e1006806 (2017)
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b30e4478e3938cd045e9fcbd810a88ce
https://www.pure.ed.ac.uk/ws/files/171740405/Heron_R_Pheromone_sensing_neurons....pdf
Tytuł :
All-Trans Retinoic Acid Modulates TLR4/NF-κB Signaling Pathway Targeting TNF-αand Nitric Oxide Synthase 2 Expression in Colonic Mucosa during Ulcerative Colitis and Colitis Associated Cancer
Autorzy :
Rafa, Hayet
Benkhelifa, Sarra
AitYounes, Sonia
Saoula, Houria
Belhadef, Said
Belkhelfa, Mourad
Boukercha, Aziza
Toumi, Ryma
Soufli, Imene
Moralès, Olivier
de Launoit, Yvan
Mahfouf, Hassen
Nakmouche, M’hamed
Delhem, Nadira
Touil-Boukoffa, Chafia
Pokaż więcej
Temat :
[SDV.CAN]Life Sciences [q-bio]/Cancer
MESH: Aged
MESH: Blotting, Western
MESH: Colitis/blood
MESH: Colitis, Ulcerative/blood
MESH: Colitis, Ulcerative/metabolism
MESH: Colorectal Neoplasms/blood
MESH: Colorectal Neoplasms/metabolism
MESH: Enzyme-Linked Immunosorbent Assay
MESH: Female
MESH: Humans
MESH: Immunohistochemistry
MESH: Intestinal Mucosa/metabolism
MESH: Male
MESH: Middle Aged
MESH: Nitric Oxide/blood
MESH: Nitric Oxide Synthase Type II/metabolism
MESH: Real-Time Polymerase Chain Reaction
MESH: Tumor Necrosis Factor-alpha/blood
[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry, Molecular Biology/Genomics [q-bio.GN]
[SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterology
Research Article
lcsh:Pathology
lcsh:RB1-214
Article Subject
Cell Biology
Immunology
Cancer research
Signal transduction
Colorectal cancer
medicine.disease
medicine
Tumor necrosis factor alpha
Ulcerative colitis
Biology
Retinoic acid
chemistry.chemical_compound
chemistry
Ex vivo
TLR4
Nitric oxide synthase 2
biology.protein
Źródło :
Mediators of Inflammation
Mediators of Inflammation, Hindawi Publishing Corporation, 2017, 2017, pp.1-16. ⟨10.1155/2017/7353252⟩
Mediators of Inflammation, Hindawi Publishing Corporation, 2017, 2017, pp.7353252. ⟨10.1155/2017/7353252⟩
Mediators of Inflammation, Vol 2017 (2017)
Opis pliku :
text/xhtml
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::61aabb23e503ebb00e3a943766bdaf55
https://hal.archives-ouvertes.fr/hal-03059784
Tytuł :
Milk Diets Influence Doxorubicin-Induced Intestinal Toxicity in Piglets
Autorzy :
Shen, R. L.
Pontoppidan, P. E.
Rathe, M.
Jiang, P.
Hansen, C. F.
Buddington, R. K.
Heegaard, Peter M. H.
Müller, K.
Sangild, P. T.
Pokaż więcej
Temat :
Bovine colostrum
Chemotherapy-induced mucositis
Enteral nutrition
Intestinal toxicity
Milk
Colostrum/metabolism
Humans
Male
Mucositis/chemically induced
Doxorubicin
Interleukin-8/metabolism
Cattle
Female
Intestine, Small/metabolism
Nutritional Status
Weight Gain
Infant, Newborn
Microvilli/enzymology
Disease Models, Animal
Animals, Newborn
Antibiotics, Antineoplastic
Permeability
Inflammation Mediators/blood
Animals
Intestinal Mucosa/metabolism
C-Reactive Protein/metabolism
Enteral Nutrition/adverse effects
Infant Formula/toxicity
Sus scrofa
Physiology (medical)
Gastroenterology
Hepatology
Physiology
C-reactive protein
biology.protein
biology
Medicine
business.industry
business
Colostrum
medicine.medical_specialty
Chemotherapy
medicine.medical_treatment
Internal medicine
Mucositis
medicine.disease
medicine.drug
Infant formula
Parenteral nutrition
Immunology
Intestinal mucosa
SDG 3 - Good Health and Well-being
polycyclic compounds
carbohydrates (lipids)
Źródło :
Shen, R L, Pontoppidan, P E L, Rathe, M, Jiang, P, Hansen, C F, Buddington, R K, Heegaard, P M H, Müller, K & Sangild, P T 2016, ' Milk Diets Influence Doxorubicin-Induced Intestinal Toxicity in Piglets ', American Journal of Physiology: Gastrointestinal and Liver Physiology, vol. 311, no. 2, pp. G324-G333 . https://doi.org/10.1152/ajpgi.00373.2015
Shen, R L, Pontoppidan, P E, Rathe, M, Jiang, P, Hansen, C F, Buddington, R K, Heegaard, P M H, Müller, K & Sangild, P T 2016, ' Milk diets influence doxorubicin-induced intestinal toxicity in piglets ', American Journal of Physiology: Gastrointestinal and Liver Physiology, vol. 311, no. 2, pp. G324-G333 . https://doi.org/10.1152/ajpgi.00373.2015
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b091c377600f81b4808e116a790a0fc6
https://findresearcher.sdu.dk:8443/ws/files/129616711/ajpgi.00373.2015.pdf

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