Large conductance Ca2+-activated K+ channel (BKCa) α-subunit splice variants in resistance arteries from rat cerebral and skeletal muscle vasculature.
Autorzy:
Nourian Z; Dalton Cardiovascular Research Center, University of Missouri, Columbia, Missouri, United States of America. Li M; Dalton Cardiovascular Research Center, University of Missouri, Columbia, Missouri, United States of America. Leo MD; Department of Physiology, University of Tennessee Health Science Center, Memphis, Tennessee, United States of America. JaggarJH; Department of Physiology, University of Tennessee Health Science Center, Memphis, Tennessee, United States of America. Braun AP; Department of Physiology and Pharmacology, University of Calgary, Calgary, Alberta, Canada. Hill MA; Dalton Cardiovascular Research Center, University of Missouri, Columbia, Missouri, United States of America; Department of Medical Pharmacology and Physiology, University of Missouri, Columbia, Missouri, United States of America.
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Źródło:
PloS one [PLoS One] 2014 Jun 12; Vol. 9 (6), pp. e98863. Date of Electronic Publication: 2014 Jun 12 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Contributions of KATP and KCa channels to cerebral arteriolar dilation to hypercapnia in neonatal brain.
Autorzy:
Nnorom CC; Departments of Physiology and Pediatrics, University of Tennessee Health Science Center, Memphis, Tennessee. Davis C; Departments of Physiology and Pediatrics, University of Tennessee Health Science Center, Memphis, Tennessee. Fedinec AL; Departments of Physiology and Pediatrics, University of Tennessee Health Science Center, Memphis, Tennessee. Howell K; Departments of Physiology and Pediatrics, University of Tennessee Health Science Center, Memphis, Tennessee. JaggarJH; Departments of Physiology and Pediatrics, University of Tennessee Health Science Center, Memphis, Tennessee. Parfenova H; Departments of Physiology and Pediatrics, University of Tennessee Health Science Center, Memphis, Tennessee. Pourcyrous M; Departments of Physiology and Pediatrics, University of Tennessee Health Science Center, Memphis, Tennessee. Leffler CW; Departments of Physiology and Pediatrics, University of Tennessee Health Science Center, Memphis, Tennessee.
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Źródło:
Physiological reports [Physiol Rep] 2014 Aug 28; Vol. 2 (8). Date of Electronic Publication: 2014 Aug 28 (Print Publication: 2014).
Eugenol dilates mesenteric arteries and reduces systemic BP by activating endothelial cell TRPV4 channels.
Autorzy:
Peixoto-Neves D; Department of Physiology, University of Tennessee Health Science Center, Memphis, TN, USA.; Laboratório de Eletrofisiologia, Instituto Superior de Ciências Biomédicas, Universidade Estadual do Ceará, Fortaleza, Ceará, Brazil. Wang Q; Department of Physiology, University of Tennessee Health Science Center, Memphis, TN, USA. Leal-Cardoso JH; Laboratório de Eletrofisiologia, Instituto Superior de Ciências Biomédicas, Universidade Estadual do Ceará, Fortaleza, Ceará, Brazil. Rossoni LV; Laboratório de Fisiologia Vascular, Instituto de Ciências Biomédicas, Universidade de São Paulo, São Paulo, Brazil. JaggarJH; Department of Physiology, University of Tennessee Health Science Center, Memphis, TN, USA.
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Źródło:
British journal of pharmacology [Br J Pharmacol] 2015 Jul; Vol. 172 (14), pp. 3484-94. Date of Electronic Publication: 2015 May 19.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
Calcium Channels and Oxidative Stress Mediate a Synergistic Disruption of Tight Junctions by Ethanol and Acetaldehyde in Caco-2 Cell Monolayers.
Autorzy:
Samak G; Department of Physiology, University of Tennessee Health Science Center, 894 Union Avenue, Memphis TN 38163, USA. Gangwar R; Department of Physiology, University of Tennessee Health Science Center, 894 Union Avenue, Memphis TN 38163, USA. Meena AS; Department of Physiology, University of Tennessee Health Science Center, 894 Union Avenue, Memphis TN 38163, USA. Rao RG; Department of Physiology, University of Tennessee Health Science Center, 894 Union Avenue, Memphis TN 38163, USA. Shukla PK; Department of Physiology, University of Tennessee Health Science Center, 894 Union Avenue, Memphis TN 38163, USA. Manda B; Department of Physiology, University of Tennessee Health Science Center, 894 Union Avenue, Memphis TN 38163, USA. Narayanan D; Department of Physiology, University of Tennessee Health Science Center, 894 Union Avenue, Memphis TN 38163, USA. JaggarJH; Department of Physiology, University of Tennessee Health Science Center, 894 Union Avenue, Memphis TN 38163, USA. Rao R; Department of Physiology, University of Tennessee Health Science Center, 894 Union Avenue, Memphis TN 38163, USA.
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Źródło:
Scientific reports [Sci Rep] 2016 Dec 13; Vol. 6, pp. 38899. Date of Electronic Publication: 2016 Dec 13.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
Calmodulin is responsible for Ca -dependent regulation of TRPA1 Channels.
Autorzy:
Hasan R; School of Medicine, Medical Sciences &Nutrition, University of Aberdeen, Foresterhill, Aberdeen AB25 2ZD, United Kingdom.; Department of Physiology, University of Tennessee Health Science Center, Memphis, TN 38163, USA.; Department of Pharmacology, University of Cambridge, Tennis Court Road, Cambridge, CB2 1PD, United Kingdom. Leeson-Payne AT; School of Medicine, Medical Sciences &Nutrition, University of Aberdeen, Foresterhill, Aberdeen AB25 2ZD, United Kingdom. JaggarJH; Department of Physiology, University of Tennessee Health Science Center, Memphis, TN 38163, USA. Zhang X; School of Medicine, Medical Sciences &Nutrition, University of Aberdeen, Foresterhill, Aberdeen AB25 2ZD, United Kingdom.; Department of Pharmacology, University of Cambridge, Tennis Court Road, Cambridge, CB2 1PD, United Kingdom.; Schcool of Life &Health Sciences, Aston University, Aston triangle, Birmingham B4 7ET, United Kingdom.
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Źródło:
Scientific reports [Sci Rep] 2017 Mar 23; Vol. 7, pp. 45098. Date of Electronic Publication: 2017 Mar 23.
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