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Wyszukujesz frazę ""Jin, Huiling"" wg kryterium: Autor


Wyświetlanie 1-20 z 20
Tytuł:
Vidarabine, an anti-herpesvirus agent, prevents catecholamine-induced arrhythmias without adverse effect on heart function in mice
Autorzy:
Suita, KenjiAff1, Aff2
Fujita, Takayuki
Cai, Wenqian
Hidaka, Yuko
Jin, Huiling
Prajapati, Rajesh
Umemura, Masanari
Yokoyama, Utako
Sato, MotohikoAff1, Aff3
Knollmann, Björn C.
Okumura, SatoshiAff1, Aff2
Ishikawa, Yoshihiro
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Źródło:
Pflügers Archiv - European Journal of Physiology: European Journal of Physiology. 470(6):923-935
Czasopismo naukowe
Tytuł:
Norepinephrine-Induced Adrenergic Activation Strikingly Increased the Atrial Fibrillation Duration through β1- and α1-Adrenergic Receptor-Mediated Signaling in Mice.
Autorzy:
Suita, Kenji
Fujita, Takayuki
Hasegawa, Nozomi
Cai, Wenqian
Jin, Huiling
Hidaka, Yuko
Prajapati, Rajesh
Umemura, Masanari
Yokoyama, Utako
Sato, Motohiko
Okumura, Satoshi
Ishikawa, Yoshihiro
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Temat:
NORADRENALINE
SYMPATHOMIMETIC agents
ATRIAL fibrillation
ADRENERGIC receptors
LABORATORY mice
CELLULAR signal transduction
Źródło:
PLoS ONE; 7/23/2015, Vol. 10 Issue 7, p1-13, 13p
Czasopismo naukowe
Tytuł:
Role of cyclic AMP sensor Epac1 in masseter muscle hypertrophy and myosin heavy chain transition induced by β2-adrenoceptor stimulation.
Autorzy:
Ohnuki, Yoshiki
Umeki, Daisuke
Mototani, Yasumasa
Jin, Huiling
Cai, Wenqian
Shiozawa, Kouichi
Suita, Kenji
Saeki, Yasutake
Fujita, Takayuki
Ishikawa, Yoshihiro
Okumura, Satoshi
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Temat:
CYCLIC adenylic acid
ADENOSINE monophosphate
MASSETER muscle
MASTICATORY muscles
HYPERTROPHY
Źródło:
Journal of Physiology; Dec2014, Vol. 592 Issue 24, p5461-5475, 15p
Czasopismo naukowe
Tytuł:
Role of phosphodiesterase 4 expression in the Epac1 signaling-dependent skeletal muscle hypertrophic action of clenbuterol.
Autorzy:
Ohnuki Y; Department of Physiology, Tsurumi University School of Dental Medicine, Yokohama, Japan.
Umeki D; Department of Physiology, Tsurumi University School of Dental Medicine, Yokohama, Japan Department of Orthodontics, Tsurumi University School of Dental Medicine, Yokohama, Japan.
Mototani Y; Department of Physiology, Tsurumi University School of Dental Medicine, Yokohama, Japan.
Shiozawa K; Department of Physiology, Tsurumi University School of Dental Medicine, Yokohama, Japan.
Nariyama M; Department of Physiology, Tsurumi University School of Dental Medicine, Yokohama, Japan Department of Pediatric Dentistry, Tsurumi University School of Dental Medicine, Yokohama, Japan.
Ito A; Department of Physiology, Tsurumi University School of Dental Medicine, Yokohama, Japan Department of Orthodontics, Tsurumi University School of Dental Medicine, Yokohama, Japan.
Kawamura N; Department of Physiology, Tsurumi University School of Dental Medicine, Yokohama, Japan Department of Periodontology, Tsurumi University School of Dental Medicine, Yokohama, Japan.
Yagisawa Y; Department of Physiology, Tsurumi University School of Dental Medicine, Yokohama, Japan Department of Orthodontics, Tsurumi University School of Dental Medicine, Yokohama, Japan.
Jin H; Cardiovascular Research Institute, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Cai W; Cardiovascular Research Institute, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Suita K; Department of Physiology, Tsurumi University School of Dental Medicine, Yokohama, Japan Cardiovascular Research Institute, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Saeki Y; Department of Physiology, Tsurumi University School of Dental Medicine, Yokohama, Japan.
Fujita T; Cardiovascular Research Institute, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Ishikawa Y; Cardiovascular Research Institute, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Okumura S; Department of Physiology, Tsurumi University School of Dental Medicine, Yokohama, Japan Cardiovascular Research Institute, Yokohama City University Graduate School of Medicine, Yokohama, Japan .
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Źródło:
Physiological reports [Physiol Rep] 2016 May; Vol. 4 (10).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Clenbuterol/*toxicity
Cyclic Nucleotide Phosphodiesterases, Type 4/*biosynthesis
Guanine Nucleotide Exchange Factors/*metabolism
Muscle, Skeletal/*metabolism
Muscular Diseases/*chemically induced
Muscular Diseases/*metabolism
Adrenergic beta-Agonists/toxicity ; Animals ; Gene Expression Regulation, Enzymologic ; Hypertrophy/chemically induced ; Hypertrophy/metabolism ; Mice ; Mice, Inbred C57BL ; Mice, Inbred CBA ; Mice, Knockout ; Muscle, Skeletal/drug effects ; Signal Transduction/drug effects ; Signal Transduction/physiology
Czasopismo naukowe
    Wyświetlanie 1-20 z 20

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