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Wyszukujesz frazę ""John, Alison E."" wg kryterium: Autor

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    Wyświetlanie 1-4 z 4
    Tytuł:
    The IL-33:ST2 axis is unlikely to play a central fibrogenic role in idiopathic pulmonary fibrosis.
    Autorzy:
    Stephenson KE; Division of Respiratory Medicine, School of Medicine, University of Nottingham, Nottingham, UK. .; Bioscience Asthma and Skin Immunity, Research and Early Development, Respiratory and Immunology, BioPharmaceuticals R&D, AstraZeneca, Cambridge, UK. .
    Porte J; Division of Respiratory Medicine, School of Medicine, University of Nottingham, Nottingham, UK.
    Kelly A; Bioscience Asthma and Skin Immunity, Research and Early Development, Respiratory and Immunology, BioPharmaceuticals R&D, AstraZeneca, Cambridge, UK.
    Wallace WA; Division of Pathology, University of Edinburgh, Edinburgh, UK.
    Huntington CE; Biologics Engineering, R&D, AstraZeneca, Cambridge, UK.
    Overed-Sayer CL; Bioscience COPD/IPF, Research and Early Development, Respiratory and Immunology, BioPharmaceuticals R&D, AstraZeneca, Cambridge, UK.
    Cohen ES; Bioscience Asthma and Skin Immunity, Research and Early Development, Respiratory and Immunology, BioPharmaceuticals R&D, AstraZeneca, Cambridge, UK.
    Jenkins RG; National Heart and Lung Institute, Imperial College London, London, UK.; Margaret Turner Warwick Centre for Fibrosing Lung Disease, Imperial College London, London, UK.; Interstitial lung disease unit, Royal Brompton Hospital, London, UK.
    John AE; Division of Respiratory Medicine, School of Medicine, University of Nottingham, Nottingham, UK.; National Heart and Lung Institute, Imperial College London, London, UK.; Margaret Turner Warwick Centre for Fibrosing Lung Disease, Imperial College London, London, UK.
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    Źródło:
    Respiratory research [Respir Res] 2023 Mar 23; Vol. 24 (1), pp. 89. Date of Electronic Publication: 2023 Mar 23.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Idiopathic Pulmonary Fibrosis*/chemically induced
    Idiopathic Pulmonary Fibrosis*/drug therapy
    Idiopathic Pulmonary Fibrosis*/metabolism
    Interleukin-33*/metabolism
    Animals ; Humans ; Mice ; Bleomycin/toxicity ; Fibroblasts/metabolism ; Interleukin-1 Receptor-Like 1 Protein/genetics ; Lung/metabolism ; Mice, Inbred C57BL ; Transforming Growth Factor beta/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Amplification of TGFβ Induced ITGB6 Gene Transcription May Promote Pulmonary Fibrosis.
    Autorzy:
    Tatler AL; Division of Respiratory Medicine-City, University of Nottingham, Nottingham, United Kingdom.
    Goodwin AT; Division of Respiratory Medicine-City, University of Nottingham, Nottingham, United Kingdom.
    Gbolahan O; Division of Respiratory Medicine-City, University of Nottingham, Nottingham, United Kingdom.
    Saini G; Division of Respiratory Medicine-City, University of Nottingham, Nottingham, United Kingdom.
    Porte J; Division of Respiratory Medicine-City, University of Nottingham, Nottingham, United Kingdom.
    John AE; Division of Respiratory Medicine-City, University of Nottingham, Nottingham, United Kingdom.
    Clifford RL; Division of Respiratory Medicine-City, University of Nottingham, Nottingham, United Kingdom.
    Violette SM; Biogen, Cambridge, Massachusetts, United States of America.
    Weinreb PH; Biogen, Cambridge, Massachusetts, United States of America.
    Parfrey H; Department of Pathology, University of Cambridge, Cambridge, United Kingdom.
    Wolters PJ; School of Medicine, University of California San Francisco, San Francisco, California, United States of America.
    Gauldie J; Department of Pathology and Molecular Medicine, McMaster University, Hamilton, Canada.
    Kolb M; Department of Pathology and Molecular Medicine, McMaster University, Hamilton, Canada.
    Jenkins G; Division of Respiratory Medicine-City, University of Nottingham, Nottingham, United Kingdom.
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    Źródło:
    PloS one [PLoS One] 2016 Aug 05; Vol. 11 (8), pp. e0158047. Date of Electronic Publication: 2016 Aug 05 (Print Publication: 2016).
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Idiopathic Pulmonary Fibrosis/*pathology
    Integrin beta Chains/*metabolism
    Transcription, Genetic/*drug effects
    Transforming Growth Factor beta/*pharmacology
    Animals ; Antigens, Neoplasm/genetics ; Antigens, Neoplasm/metabolism ; Binding Sites ; Cells, Cultured ; Epithelial Cells/cytology ; Epithelial Cells/drug effects ; Epithelial Cells/metabolism ; Humans ; Idiopathic Pulmonary Fibrosis/metabolism ; Integrin beta Chains/genetics ; Integrins/genetics ; Integrins/metabolism ; Lung/drug effects ; Lung/metabolism ; Lung/pathology ; Mice ; Mice, Knockout ; Mutagenesis, Site-Directed ; Promoter Regions, Genetic ; RNA Stability/drug effects ; Rats ; Rats, Sprague-Dawley ; Signal Transduction/drug effects ; Smad3 Protein/genetics ; Smad3 Protein/metabolism ; Smad4 Protein/genetics ; Smad4 Protein/metabolism ; Transforming Growth Factor beta/genetics ; Transforming Growth Factor beta/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Loss of ELK1 has differential effects on age-dependent organ fibrosis.
    Autorzy:
    Cairns, Jennifer T. (AUTHOR)
    Habgood, Anthony (AUTHOR)
    Edwards-Pritchard, Rochelle C. (AUTHOR)
    Joseph, Chitra (AUTHOR)
    John, Alison E. (AUTHOR)
    Wilkinson, Chloe (AUTHOR)
    Stewart, Iain D. (AUTHOR)
    Leslie, Jack (AUTHOR)
    Blaxall, Burns C. (AUTHOR)
    Susztak, Katalin (AUTHOR)
    Alberti, Siegfried (AUTHOR)
    Nordheim, Alfred (AUTHOR)
    Oakley, Fiona (AUTHOR)
    Jenkins, Gisli (AUTHOR)
    Tatler, Amanda L. (AUTHOR)
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    Źródło:
    International Journal of Biochemistry & Cell Biology. Mar2020, Vol. 120, pN.PAG-N.PAG. 1p.
    Czasopismo naukowe
    Szczegóły
      Wyświetlanie 1-4 z 4

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