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Wyszukujesz frazę ""Kidney Tubules, Proximal"" wg kryterium: Temat


Tytuł :
Modulation of Tubular pH by Acetazolamide in a Ca Transport Deficient Mice Facilitates Calcium Nephrolithiasis.
Autorzy :
Awuah Boadi E; Calcium Signaling Laboratory, Research Service, Veterans Affairs Medical Center, 50 Irving Street, NW, Washington, DC 20422, USA.
Shin S; Calcium Signaling Laboratory, Research Service, Veterans Affairs Medical Center, 50 Irving Street, NW, Washington, DC 20422, USA.
Yeroushalmi S; Calcium Signaling Laboratory, Research Service, Veterans Affairs Medical Center, 50 Irving Street, NW, Washington, DC 20422, USA.; Division of Renal Diseases & Hypertension, Department of Medicine, The George Washington University, Washington, DC 20037, USA.
Choi BE; Calcium Signaling Laboratory, Research Service, Veterans Affairs Medical Center, 50 Irving Street, NW, Washington, DC 20422, USA.
Li P; Calcium Signaling Laboratory, Research Service, Veterans Affairs Medical Center, 50 Irving Street, NW, Washington, DC 20422, USA.
Bandyopadhyay BC; Calcium Signaling Laboratory, Research Service, Veterans Affairs Medical Center, 50 Irving Street, NW, Washington, DC 20422, USA.; Division of Renal Diseases & Hypertension, Department of Medicine, The George Washington University, Washington, DC 20037, USA.; Department of Biomedical Engineering, The Catholic University of America, 620 Michigan Avenue NE, Washington, DC 20064, USA.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Mar 17; Vol. 22 (6). Date of Electronic Publication: 2021 Mar 17.
Typ publikacji :
Journal Article
MeSH Terms :
Acetazolamide/*pharmacology
Calcium/*metabolism
Kidney Tubules, Proximal/*metabolism
Kidney Tubules, Proximal/*pathology
Nephrolithiasis/*metabolism
Nephrolithiasis/*pathology
Animals ; Biological Transport/drug effects ; Calcinosis/complications ; Endoplasmic Reticulum Stress/drug effects ; Fibrosis ; Hydrogen-Ion Concentration ; Inflammation/pathology ; Kidney Tubules, Proximal/drug effects ; Mice ; Nephrolithiasis/urine ; Oxidative Stress/drug effects ; TRPC Cation Channels/metabolism ; Up-Regulation/drug effects
Czasopismo naukowe
Tytuł :
Danegaptide Prevents TGFβ1-Induced Damage in Human Proximal Tubule Epithelial Cells of the Kidney.
Autorzy :
Squires PE; School of Life Sciences, Joseph Banks Laboratories, University of Lincoln, Lincoln LN6 7DL, UK.
Price GW; School of Life Sciences, Joseph Banks Laboratories, University of Lincoln, Lincoln LN6 7DL, UK.
Mouritzen U; Ciana Therapeutics, Ved Hegnet 2, 2960 Rungsted Kyst, Copenhagen, Denmark.
Potter JA; School of Life Sciences, Joseph Banks Laboratories, University of Lincoln, Lincoln LN6 7DL, UK.
Williams BM; School of Life Sciences, Joseph Banks Laboratories, University of Lincoln, Lincoln LN6 7DL, UK.
Hills CE; School of Life Sciences, Joseph Banks Laboratories, University of Lincoln, Lincoln LN6 7DL, UK.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Mar 10; Vol. 22 (6). Date of Electronic Publication: 2021 Mar 10.
Typ publikacji :
Journal Article
MeSH Terms :
Epithelial Cells*/metabolism
Epithelial Cells*/pathology
Kidney Tubules, Proximal*/injuries
Kidney Tubules, Proximal*/metabolism
Kidney Tubules, Proximal*/pathology
Renal Insufficiency, Chronic*/drug therapy
Renal Insufficiency, Chronic*/metabolism
Renal Insufficiency, Chronic*/pathology
Dipeptides/*pharmacology
Transforming Growth Factor beta1/*metabolism
Cell Line ; Humans
Czasopismo naukowe
Tytuł :
Activation of G protein-coupled estrogen receptor 1 ameliorates proximal tubular injury and proteinuria in Dahl salt-sensitive female rats.
Autorzy :
Gohar EY; Cardio-Renal Physiology and Medicine Section, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama.
Almutlaq RN; Cardio-Renal Physiology and Medicine Section, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama.
Daugherty EM; Cardio-Renal Physiology and Medicine Section, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama.
Butt MK; Cardio-Renal Physiology and Medicine Section, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama.
Jin C; Cardio-Renal Physiology and Medicine Section, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama.
Pollock JS; Cardio-Renal Physiology and Medicine Section, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama.
Pollock DM; Cardio-Renal Physiology and Medicine Section, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama.
De Miguel C; Cardio-Renal Physiology and Medicine Section, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama.
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Źródło :
American journal of physiology. Regulatory, integrative and comparative physiology [Am J Physiol Regul Integr Comp Physiol] 2021 Mar 01; Vol. 320 (3), pp. R297-R306. Date of Electronic Publication: 2021 Jan 06.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Albuminuria/*prevention & control
Cyclopentanes/*pharmacology
Kidney Diseases/*prevention & control
Kidney Glomerulus/*drug effects
Kidney Tubules, Proximal/*drug effects
Quinolines/*pharmacology
Receptors, G-Protein-Coupled/*agonists
Albuminuria/etiology ; Albuminuria/metabolism ; Albuminuria/pathology ; Animals ; Arterial Pressure ; Cell Adhesion Molecules/metabolism ; Disease Models, Animal ; Female ; Hypertension/etiology ; Hypertension/physiopathology ; Kidney Diseases/etiology ; Kidney Diseases/metabolism ; Kidney Diseases/pathology ; Kidney Glomerulus/metabolism ; Kidney Glomerulus/pathology ; Kidney Tubules, Proximal/metabolism ; Kidney Tubules, Proximal/pathology ; Rats, Inbred Dahl ; Receptors, G-Protein-Coupled/metabolism ; Signal Transduction ; Sodium Chloride, Dietary
Czasopismo naukowe
Tytuł :
Putting the pressure on endocytosis in the kidney.
Autorzy :
Van Giel D; Laboratory of Ion Channel Research, TRP Research Platform Leuven, VIB Center for Brain and Disease Research, Department of Cellular and Molecular Medicine, Faculty of Medicine, KU Leuven, Belgium.
Vennekens R; Laboratory of Ion Channel Research, TRP Research Platform Leuven, VIB Center for Brain and Disease Research, Department of Cellular and Molecular Medicine, Faculty of Medicine, KU Leuven, Belgium. Electronic address: .
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Źródło :
Cell calcium [Cell Calcium] 2021 Mar; Vol. 94, pp. 102338. Date of Electronic Publication: 2020 Dec 26.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't; Comment
MeSH Terms :
Kidney Tubules, Proximal*
TRPV Cation Channels*
Albumins ; Endocytosis
Czasopismo naukowe
Tytuł :
Proximal tubule LPA1 and LPA2 receptors use divergent signaling pathways to additively increase profibrotic cytokine secretion.
Autorzy :
Geng H; Department of Pathology, University of Texas Health Science Center, San Antonio, Texas.
Lan R; Department of Pathology, University of Texas Health Science Center, San Antonio, Texas.
Liu Y; Department of Pathology, University of Texas Health Science Center, San Antonio, Texas.
Chen W; Department of Pathology, University of Texas Health Science Center, San Antonio, Texas.
Wu M; Department of Pathology, University of Texas Health Science Center, San Antonio, Texas.
Saikumar P; Department of Pathology, University of Texas Health Science Center, San Antonio, Texas.
Weinberg JM; Department of Medicine, University of Michigan Medical Center, Ann Arbor, Michigan.
Venkatachalam MA; Department of Pathology, University of Texas Health Science Center, San Antonio, Texas.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2021 Mar 01; Vol. 320 (3), pp. F359-F374. Date of Electronic Publication: 2021 Jan 11.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Acute Kidney Injury/*metabolism
Cytokines/*metabolism
Kidney Tubules, Proximal/*metabolism
Receptors, Lysophosphatidic Acid/*metabolism
Reperfusion Injury/*metabolism
Acute Kidney Injury/genetics ; Acute Kidney Injury/pathology ; Animals ; Cell Line ; Connective Tissue Growth Factor/metabolism ; Disease Models, Animal ; ErbB Receptors/metabolism ; Extracellular Signal-Regulated MAP Kinases/metabolism ; Fibrosis ; Humans ; JNK Mitogen-Activated Protein Kinases/metabolism ; Kidney Tubules, Proximal/pathology ; Lymphokines/metabolism ; Male ; Mice ; Phosphorylation ; Platelet-Derived Growth Factor/metabolism ; Rats, Sprague-Dawley ; Receptors, Lysophosphatidic Acid/genetics ; Reperfusion Injury/genetics ; Reperfusion Injury/pathology ; Signal Transduction ; Transcription Factor AP-1/metabolism ; Transforming Growth Factor beta1/metabolism
Czasopismo naukowe
Tytuł :
Pharmacological inhibition of Vanin-1 is not protective in models of acute and chronic kidney disease.
Autorzy :
Unterschemmann K; Research and Early Development, Bayer Pharmaceuticals, Wuppertal, Germany.
Ehrmann A; Drug Discovery Sciences, Bayer Pharmaceuticals, Wuppertal, Germany.
Herzig I; Drug Discovery Sciences, Bayer Pharmaceuticals, Wuppertal, Germany.
Andreevski AL; Research and Early Development, Bayer Pharmaceuticals, Wuppertal, Germany.
Lustig K; Research and Early Development, Bayer Pharmaceuticals, Wuppertal, Germany.
Schmeck C; Drug Discovery Sciences, Bayer Pharmaceuticals, Wuppertal, Germany.
Eitner F; Research and Early Development, Bayer Pharmaceuticals, Wuppertal, Germany.
Grundmann M; Research and Early Development, Bayer Pharmaceuticals, Wuppertal, Germany.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2021 Jan 01; Vol. 320 (1), pp. F61-F73. Date of Electronic Publication: 2020 Nov 16.
Typ publikacji :
Journal Article
MeSH Terms :
Acute Kidney Injury/*prevention & control
Amidohydrolases/*antagonists & inhibitors
Enzyme Inhibitors/*pharmacology
Kidney Tubules, Proximal/*drug effects
Nephritis, Hereditary/*prevention & control
Oxidative Stress/*drug effects
Renal Insufficiency, Chronic/*prevention & control
Reperfusion Injury/*prevention & control
Acute Kidney Injury/enzymology ; Acute Kidney Injury/genetics ; Acute Kidney Injury/pathology ; Amidohydrolases/genetics ; Amidohydrolases/metabolism ; Animals ; Apoptosis/drug effects ; Autoantigens/genetics ; Autoantigens/metabolism ; Cell Line ; Collagen Type IV/genetics ; Collagen Type IV/metabolism ; Disease Models, Animal ; Enzyme Inhibitors/pharmacokinetics ; Fibrosis ; GPI-Linked Proteins/antagonists & inhibitors ; GPI-Linked Proteins/genetics ; GPI-Linked Proteins/metabolism ; Humans ; Kidney Tubules, Proximal/enzymology ; Kidney Tubules, Proximal/pathology ; Male ; Mice, Inbred C57BL ; Mice, Knockout ; Nephritis, Hereditary/enzymology ; Nephritis, Hereditary/genetics ; Nephritis, Hereditary/pathology ; Renal Insufficiency, Chronic/enzymology ; Renal Insufficiency, Chronic/genetics ; Renal Insufficiency, Chronic/pathology ; Reperfusion Injury/enzymology ; Reperfusion Injury/genetics ; Reperfusion Injury/pathology
Czasopismo naukowe
Tytuł :
Autophagy-mediated cytoplasmic accumulation of p53 leads to apoptosis through DRAM-BAX in cadmium-exposed human proximal tubular cells.
Autorzy :
Lee HY; Department of Anesthesiology and Pain Medicine, South Korea.
Oh SH; School of Medicine, Chosun University, 309 Pilmundaero, Dong-gu, Gwangju, 61452, South Korea. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2021 Jan 01; Vol. 534, pp. 128-133. Date of Electronic Publication: 2020 Dec 13.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Autophagy/*drug effects
Cadmium/*toxicity
Kidney Tubules, Proximal/*cytology
Tumor Suppressor Protein p53/*metabolism
Apoptosis/physiology ; Autophagy/physiology ; Autophagy-Related Protein 5/genetics ; Autophagy-Related Protein 5/metabolism ; Cell Line ; Epithelial Cells ; Humans ; Kidney Tubules, Proximal/drug effects ; Kidney Tubules, Proximal/metabolism ; Membrane Proteins/metabolism ; Poly (ADP-Ribose) Polymerase-1/metabolism ; RNA Interference ; Tumor Suppressor Protein p53/genetics ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł :
Protective effect of carnosine on hydrogen peroxide-induced oxidative stress in human kidney tubular epithelial cells.
Autorzy :
Cao Y; Department of Endocrinology, The First Affiliated Hospital of Anhui Medical University, 218 Jixi Road, Hefei, Anhui, 230022, China.
Xu J; Department of Endocrinology, The First Affiliated Hospital of Anhui Medical University, 218 Jixi Road, Hefei, Anhui, 230022, China.
Cui D; Department of Endocrinology, The First Affiliated Hospital of Anhui Medical University, 218 Jixi Road, Hefei, Anhui, 230022, China.
Liu L; Department of Endocrinology, The First Affiliated Hospital of Anhui Medical University, 218 Jixi Road, Hefei, Anhui, 230022, China.
Zhang S; Department of Endocrinology, The First Affiliated Hospital of Anhui Medical University, 218 Jixi Road, Hefei, Anhui, 230022, China.
Shen B; School of Basic Medical Sciences, Anhui Medical University, 81 Meishan Road, Hefei, Anhui, 230032, China.
Wu Y; Department of Nephrology, The First Affiliated Hospital of Anhui Medical University, 218 Jixi Road, Hefei, Anhui, 230022, China. Electronic address: .
Zhang Q; Department of Endocrinology, The First Affiliated Hospital of Anhui Medical University, 218 Jixi Road, Hefei, Anhui, 230022, China. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2021 Jan 01; Vol. 534, pp. 576-582. Date of Electronic Publication: 2020 Dec 01.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Carnosine/*pharmacology
Kidney Tubules, Proximal/*drug effects
Kidney Tubules, Proximal/*metabolism
Oxidative Stress/*drug effects
Apoptosis/drug effects ; Cell Line ; Cell Survival/drug effects ; Diabetic Nephropathies/drug therapy ; Diabetic Nephropathies/metabolism ; Diabetic Nephropathies/pathology ; Epithelial Cells/drug effects ; Epithelial Cells/metabolism ; Epithelial Cells/pathology ; Humans ; Hydrogen Peroxide/toxicity ; Kidney Tubules, Proximal/pathology ; Membrane Potential, Mitochondrial/drug effects ; NADPH Oxidase 4/metabolism ; Reactive Oxygen Species/metabolism ; Signal Transduction/drug effects ; Superoxide Dismutase/metabolism
Czasopismo naukowe
Tytuł :
Potential Protection Effect of ER Homeostasis of N -(2-Hydroxyethyl)adenosine Isolated from Cordyceps cicadae in Nonsteroidal Anti-Inflammatory Drug-Stimulated Human Proximal Tubular Cells.
Autorzy :
Chyau CC; Research Institute of Biotechnology, Hungkuang University, Taichung 43302, Taiwan.
Wu HL; Research Institute of Biotechnology, Hungkuang University, Taichung 43302, Taiwan.
Peng CC; Graduate Institute of Clinical Medicine, College of Medicine, Taipei Medical University, Taipei 11031, Taiwan.
Huang SH; Research Institute of Biotechnology, Hungkuang University, Taichung 43302, Taiwan.
Chen CC; Grape King Biotechnology Center, Chung-Li City 320054, Taiwan.
Chen CH; Department of Nephrology, Taichung Veterans General Hospital, Taichung 40705, Taiwan.
Peng RY; Research Institute of Biotechnology, Hungkuang University, Taichung 43302, Taiwan.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Feb 04; Vol. 22 (4). Date of Electronic Publication: 2021 Feb 04.
Typ publikacji :
Journal Article
MeSH Terms :
Homeostasis*
Adenosine/*analogs & derivatives
Anti-Inflammatory Agents, Non-Steroidal/*pharmacology
Cordyceps/*chemistry
Endoplasmic Reticulum Stress/*drug effects
Kidney Tubules, Proximal/*drug effects
Protective Agents/*pharmacology
Adenosine/pharmacology ; Gene Expression Regulation ; Humans ; Kidney Tubules, Proximal/metabolism ; Oxidative Stress
SCR Organism :
Cordyceps cicadae
Czasopismo naukowe
Tytuł :
Cellular extrusion bioprinting improves kidney organoid reproducibility and conformation.
Autorzy :
Lawlor KT; Murdoch Children's Research Institute, Melbourne, Victoria, Australia.
Vanslambrouck JM; Murdoch Children's Research Institute, Melbourne, Victoria, Australia.
Higgins JW; Organovo, San Diego, CA, USA.
Chambon A; Organovo, San Diego, CA, USA.
Bishard K; Organovo, San Diego, CA, USA.
Arndt D; Organovo, San Diego, CA, USA.
Er PX; Murdoch Children's Research Institute, Melbourne, Victoria, Australia.
Wilson SB; Murdoch Children's Research Institute, Melbourne, Victoria, Australia.
Howden SE; Murdoch Children's Research Institute, Melbourne, Victoria, Australia.
Tan KS; Murdoch Children's Research Institute, Melbourne, Victoria, Australia.
Li F; Murdoch Children's Research Institute, Melbourne, Victoria, Australia.
Hale LJ; Murdoch Children's Research Institute, Melbourne, Victoria, Australia.
Shepherd B; Organovo, San Diego, CA, USA.
Pentoney S; Organovo, San Diego, CA, USA.
Presnell SC; Organovo, San Diego, CA, USA.
Chen AE; Organovo, San Diego, CA, USA.
Little MH; Murdoch Children's Research Institute, Melbourne, Victoria, Australia. .; Department of Anatomy and Neuroscience, The University of Melbourne, Melbourne, Victoria, Australia. .; Department of Paediatrics, The University of Melbourne, Melbourne, Victoria, Australia. .
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Źródło :
Nature materials [Nat Mater] 2021 Feb; Vol. 20 (2), pp. 260-271. Date of Electronic Publication: 2020 Nov 23.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Bioprinting*
Kidney Tubules, Proximal/*metabolism
Organoids/*metabolism
Pluripotent Stem Cells/*metabolism
Humans ; Kidney Tubules, Proximal/cytology ; Organoids/cytology ; Pluripotent Stem Cells/cytology
Czasopismo naukowe
Tytuł :
Cisplatin-Mediated Upregulation of APE2 Binding to MYH9 Provokes Mitochondrial Fragmentation and Acute Kidney Injury.
Autorzy :
Hu Y; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio.
Yang C; Department of Medical Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts.; Department of Clinical Laboratory, the 4th Hospital of Harbin Medical University, Harbin, China.
Amorim T; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio.
Maqbool M; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio.
Lin J; Department of Medicine, University of California San Diego, La Jolla, California.
Li C; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio.; College of Food Science and Technology, Agricultural University of Hebei, Baoding, Hebei, China.
Fang C; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio.; Department of Clinical Laboratory, the 4th Hospital of Harbin Medical University, Harbin, China.
Xue L; Department of Medical Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts.; Department of Clinical Laboratory, the 4th Hospital of Harbin Medical University, Harbin, China.
Kwart A; Department of Medical Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts.; Division of Hand Surgery, Department of Orthopedic Surgery, NYU Langone Orthopedic Hospital, New York, New York.
Fang H; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio.
Yin M; Image Core, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio.
Janocha AJ; The Department of Inflammation and Immunity, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio.
Tsuchimoto D; Department of Immunobiology and Neuroscience, Medical Institute of Bioregulation, Kyushu University, Higashi-ku, Japan.
Nakabeppu Y; Department of Immunobiology and Neuroscience, Medical Institute of Bioregulation, Kyushu University, Higashi-ku, Japan.
Jiang X; Department of Clinical Laboratory, the 4th Hospital of Harbin Medical University, Harbin, China.
Mejia-Garcia A; Department of Hematology and Medical Oncology, Taussig Cancer Institute, Cleveland Clinic, Cleveland, Ohio.
Anwer F; Department of Hematology and Medical Oncology, Taussig Cancer Institute, Cleveland Clinic, Cleveland, Ohio.
Khouri J; Department of Hematology and Medical Oncology, Taussig Cancer Institute, Cleveland Clinic, Cleveland, Ohio.
Qi X; Department of Physiology and Biophysics, Case Western Reserve University School of Medicine, Cleveland, Ohio.
Zheng QY; Department of Otolaryngology-Head and Neck Surgery, Case Western Reserve University, Cleveland, Ohio.
Yu JS; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio.; Department of Radiation Oncology, Taussig Cancer Institute, Cleveland Clinic, Cleveland, Ohio.
Yan S; Department of Biological Sciences, University of North Carolina at Charlotte, Charlotte, North Carolina.
LaFramboise T; Department of Genetics and Genome Sciences, Case Western Reserve University School of Medicine, Cleveland, Ohio.
Anderson KC; Department of Medical Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts.
Herlitz LC; Department of Laboratory Medicine, Robert J. Tomsich Pathology and Laboratory Medicine Institute, Cleveland Clinic, Cleveland, Ohio.
Munshi NC; Department of Medical Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts.; VA Boston Healthcare System, Boston, Massachusetts.
Lin J; Department of Genetics and Genome Sciences, Case Western Reserve University School of Medicine, Cleveland, Ohio. .
Zhao J; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio. .
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Źródło :
Cancer research [Cancer Res] 2021 Feb 01; Vol. 81 (3), pp. 713-723. Date of Electronic Publication: 2020 Dec 07.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Acute Kidney Injury/*chemically induced
Antineoplastic Agents/*adverse effects
Cisplatin/*adverse effects
DNA-(Apurinic or Apyrimidinic Site) Lyase/*metabolism
Endonucleases/*metabolism
Kidney Tubules, Proximal/*drug effects
Multifunctional Enzymes/*metabolism
Myosin Heavy Chains/*metabolism
Acute Kidney Injury/prevention & control ; Animals ; Carboplatin/adverse effects ; DNA Damage ; DNA, Mitochondrial/drug effects ; DNA-(Apurinic or Apyrimidinic Site) Lyase/drug effects ; DNA-(Apurinic or Apyrimidinic Site) Lyase/genetics ; Endonucleases/drug effects ; Endonucleases/genetics ; Hearing Loss, Sensorineural/chemically induced ; Humans ; Kidney Tubules, Proximal/metabolism ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Mice, Transgenic ; Mitochondria/drug effects ; Mitochondria/metabolism ; Mitochondrial Diseases/genetics ; Multifunctional Enzymes/drug effects ; Multifunctional Enzymes/genetics ; Mutation ; Myosin Heavy Chains/genetics ; Nephritis/chemically induced ; Oxaliplatin/adverse effects ; Phenotype ; Thrombocytopenia/chemically induced ; Up-Regulation/drug effects
SCR Disease Name :
Macrothrombocytopenia progressive deafness
Czasopismo naukowe
Tytuł :
Renal ischemia-reperfusion leads to hypertension and changes in proximal tubule Na transport and renin-angiotensin-aldosterone system: Role of NADPH oxidase.
Autorzy :
Lima NKS; Department of Physiology and Pharmacology, Federal University of Pernambuco, Recife, Brazil.
Farias WRA; Department of Physiology and Pharmacology, Federal University of Pernambuco, Recife, Brazil.
Cirilo MAS; Department of Physiology and Pharmacology, Federal University of Pernambuco, Recife, Brazil.
Oliveira AG; Department of Physiology and Pharmacology, Federal University of Pernambuco, Recife, Brazil.
Farias JS; Department of Physiology and Pharmacology, Federal University of Pernambuco, Recife, Brazil.
Aires RS; Department of Physiology and Pharmacology, Federal University of Pernambuco, Recife, Brazil.
Muzi-Filho H; Carlos Chagas Filho Institute of Biophysics, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil; National Center for Structural Biology and Bioimaging/CENABIO, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil.
Paixão ADO; Department of Physiology and Pharmacology, Federal University of Pernambuco, Recife, Brazil; National Center for Structural Biology and Bioimaging/CENABIO, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil.
Vieira LD; Department of Physiology and Pharmacology, Federal University of Pernambuco, Recife, Brazil; National Center for Structural Biology and Bioimaging/CENABIO, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2021 Feb 01; Vol. 266, pp. 118879. Date of Electronic Publication: 2020 Dec 10.
Typ publikacji :
Journal Article
MeSH Terms :
Renin-Angiotensin System*
Acute Kidney Injury/*complications
Hypertension/*pathology
Kidney Tubules, Proximal/*pathology
NADPH Oxidases/*metabolism
Reperfusion Injury/*complications
Sodium/*metabolism
Acute Kidney Injury/metabolism ; Acute Kidney Injury/pathology ; Aldosterone/metabolism ; Animals ; Hypertension/enzymology ; Hypertension/etiology ; Kidney Tubules, Proximal/metabolism ; Male ; Oxidative Stress ; Rats ; Rats, Wistar ; Reperfusion Injury/metabolism ; Reperfusion Injury/pathology
Czasopismo naukowe
Tytuł :
Renoprotective effect of Stat1 deletion in murine aristolochic acid nephropathy.
Autorzy :
Feng W; Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama.
Ying WZ; Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama.
Li X; Department of Cell, Developmental and Integrative Biology, University of Alabama at Birmingham, Birmingham, Alabama.
Curtis LM; Department of Cell, Developmental and Integrative Biology, University of Alabama at Birmingham, Birmingham, Alabama.
Sanders PW; Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama.; Department of Cell, Developmental and Integrative Biology, University of Alabama at Birmingham, Birmingham, Alabama.; Department of Veterans Affairs Medical Center, Birmingham, Alabama.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2021 Jan 01; Vol. 320 (1), pp. F87-F96. Date of Electronic Publication: 2020 Dec 07.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms :
Aristolochic Acids*
Gene Deletion*
Extracellular Matrix/*metabolism
Kidney Tubules, Proximal/*metabolism
Renal Insufficiency, Chronic/*prevention & control
STAT1 Transcription Factor/*deficiency
Animals ; Disease Models, Animal ; Extracellular Matrix/pathology ; Fibrosis ; Hepatitis A Virus Cellular Receptor 1/metabolism ; Integrin beta Chains/metabolism ; Kidney Tubules, Proximal/pathology ; Male ; Mice, Inbred C57BL ; Mice, Knockout ; Phosphorylation ; Renal Insufficiency, Chronic/chemically induced ; Renal Insufficiency, Chronic/metabolism ; Renal Insufficiency, Chronic/pathology ; STAT1 Transcription Factor/genetics ; Signal Transduction ; Smad2 Protein/metabolism ; Smad3 Protein/metabolism
Czasopismo naukowe
Tytuł :
Mechanism of how carbamylation reduces albumin binding to FcRn contributing to increased vascular clearance.
Autorzy :
Yadav SPS; Division of Nephrology, Department of Medicine, Indiana University School of Medicine, Indianapolis, Indiana.
Sandoval RM; Division of Nephrology, Department of Medicine, Indiana University School of Medicine, Indianapolis, Indiana.
Zhao J; Department of Chemistry and Biochemistry, Texas Tech University, Lubbock, Texas.
Huang Y; Department of Chemistry and Biochemistry, Texas Tech University, Lubbock, Texas.
Wang E; Department of Cell Systems and Anatomy, UT Health San Antonio, San Antonio, Texas.
Kumar S; Division of Nephrology, Department of Medicine, Indiana University School of Medicine, Indianapolis, Indiana.
Campos-Bilderback SB; Division of Nephrology, Department of Medicine, Indiana University School of Medicine, Indianapolis, Indiana.
Rhodes G; Division of Nephrology, Department of Medicine, Indiana University School of Medicine, Indianapolis, Indiana.
Mechref Y; Department of Chemistry and Biochemistry, Texas Tech University, Lubbock, Texas.
Molitoris BA; Division of Nephrology, Department of Medicine, Indiana University School of Medicine, Indianapolis, Indiana.; Department of Cellular and Integrative Physiology, Indiana University School of Medicine, Indianapolis, Indiana.
Wagner MC; Division of Nephrology, Department of Medicine, Indiana University School of Medicine, Indianapolis, Indiana.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2021 Jan 01; Vol. 320 (1), pp. F114-F129. Date of Electronic Publication: 2020 Dec 07.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Histocompatibility Antigens Class I/*metabolism
Kidney Tubules, Proximal/*metabolism
Liver/*metabolism
Receptors, Fc/*metabolism
Renal Insufficiency, Chronic/*metabolism
Serum Albumin/*metabolism
Animals ; Chromatography, Liquid ; Disease Models, Animal ; Glomerular Filtration Rate ; Kidney Tubules, Proximal/physiopathology ; Lysine ; Male ; Microscopy, Fluorescence, Multiphoton ; Protein Binding ; Protein Carbamylation ; Rats, Inbred Strains ; Rats, Sprague-Dawley ; Receptors, Cell Surface/metabolism ; Renal Insufficiency, Chronic/pathology ; Renal Insufficiency, Chronic/physiopathology ; Scattering, Small Angle ; Tandem Mass Spectrometry ; Time Factors ; X-Ray Diffraction
Czasopismo naukowe
Tytuł :
Two New Aristolochic Acid Analogues from the Roots of Aristolochia contorta with Significant Cytotoxic Activity.
Autorzy :
Ji HJ; School of Chinese Materia Medica, Nanjing University of Chinese Medicine, Xianlin Road #138, Nanjing 210023, China.; Shanghai Institue of Materia Medica, Chinese Academy of Sciences, Shanghai 201203, China.; Department of Pharmacy, Yancheng School of Clinical Medicine of Nanjing Medical University, Yancheng 224001, China.
Li JY; Shanghai Institue of Materia Medica, Chinese Academy of Sciences, Shanghai 201203, China.
Wu SF; Shanghai Institue of Materia Medica, Chinese Academy of Sciences, Shanghai 201203, China.
Wu WY; School of Chinese Materia Medica, Nanjing University of Chinese Medicine, Xianlin Road #138, Nanjing 210023, China.
Yao CL; Shanghai Institue of Materia Medica, Chinese Academy of Sciences, Shanghai 201203, China.
Yao S; Shanghai Institue of Materia Medica, Chinese Academy of Sciences, Shanghai 201203, China.
Zhang JQ; Shanghai Institue of Materia Medica, Chinese Academy of Sciences, Shanghai 201203, China.
Guo DA; School of Chinese Materia Medica, Nanjing University of Chinese Medicine, Xianlin Road #138, Nanjing 210023, China.; Shanghai Institue of Materia Medica, Chinese Academy of Sciences, Shanghai 201203, China.
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Źródło :
Molecules (Basel, Switzerland) [Molecules] 2020 Dec 23; Vol. 26 (1). Date of Electronic Publication: 2020 Dec 23.
Typ publikacji :
Journal Article
MeSH Terms :
Aristolochia/*chemistry
Aristolochic Acids/*pharmacology
Carcinogens/*pharmacology
Cytotoxins/*pharmacology
Kidney Tubules, Proximal/*pathology
Plant Roots/*chemistry
Cell Proliferation ; Cells, Cultured ; Humans ; Kidney Tubules, Proximal/drug effects
Czasopismo naukowe
Tytuł :
Cre/loxP approach-mediated downregulation of Pik3c3 inhibits the hypertrophic growth of renal proximal tubule cells.
Autorzy :
Liu T; Departments of Cellular Biology & Anatomy and Medicine, Medical College of Georgia, Augusta University, Augusta, Georgia.
Yuan J; Departments of Cellular Biology & Anatomy and Medicine, Medical College of Georgia, Augusta University, Augusta, Georgia.
Dai C; Departments of Cellular Biology & Anatomy and Medicine, Medical College of Georgia, Augusta University, Augusta, Georgia.
Xu J; Departments of Cellular Biology & Anatomy and Medicine, Medical College of Georgia, Augusta University, Augusta, Georgia.
Li S; Department of Biochemistry & Molecular Biology, School of Basic Medical Sciences, Kunming Medical University, Kunming, Yunnan, China.
Humphreys BD; Division of Nephrology, Department of Medicine, Washington University School of Medicine in St. Louis, St. Louis, Missouri.
Kleven DT; Department of Pathology, Medical College of Georgia, Augusta University, Augusta, Georgia.
Chen JK; Departments of Cellular Biology & Anatomy and Medicine, Medical College of Georgia, Augusta University, Augusta, Georgia.
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Źródło :
Journal of cellular physiology [J Cell Physiol] 2020 Dec; Vol. 235 (12), pp. 9958-9973. Date of Electronic Publication: 2020 May 31.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Class III Phosphatidylinositol 3-Kinases/*genetics
Kidney/*drug effects
Kidney Tubules, Proximal/*growth & development
Nephrons/*growth & development
Sodium-Phosphate Cotransporter Proteins, Type IIa/*genetics
Animals ; Class III Phosphatidylinositol 3-Kinases/antagonists & inhibitors ; Extracellular Matrix Proteins/genetics ; Gene Expression Regulation, Developmental/genetics ; Humans ; Integrases/genetics ; Kidney/growth & development ; Kidney/pathology ; Kidney/surgery ; Kidney Tubules, Proximal/metabolism ; Mechanistic Target of Rapamycin Complex 1/genetics ; Mice ; Nephrectomy ; Nephrons/metabolism ; Phosphorylation/genetics ; Protein-Lysine 6-Oxidase/genetics ; Ribosomal Protein S6 Kinases, 90-kDa/genetics ; Signal Transduction/drug effects ; Sirolimus/pharmacology
Czasopismo naukowe
Tytuł :
MicroRNA-133a-Dependent Inhibition of Proximal Tubule Angiotensinogen by Renal TNF (Tumor Necrosis Factor).
Autorzy :
Hao S; From the Department of Pharmacology, New York Medical College, Valhalla.
Salzo J; From the Department of Pharmacology, New York Medical College, Valhalla.
Zhao H; From the Department of Pharmacology, New York Medical College, Valhalla.
Hao M; From the Department of Pharmacology, New York Medical College, Valhalla.
Darzynkiewicz Z; From the Department of Pharmacology, New York Medical College, Valhalla.
Ferreri NR; From the Department of Pharmacology, New York Medical College, Valhalla.
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Źródło :
Hypertension (Dallas, Tex. : 1979) [Hypertension] 2020 Dec; Vol. 76 (6), pp. 1744-1752. Date of Electronic Publication: 2020 Nov 02.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Angiotensinogen/*genetics
Gene Expression Regulation/*genetics
Kidney Tubules, Proximal/*metabolism
MicroRNAs/*genetics
Tumor Necrosis Factor-alpha/*genetics
Angiotensinogen/metabolism ; Animals ; Blood Pressure/drug effects ; Blood Pressure/physiology ; Gene Expression Regulation/drug effects ; Kidney/drug effects ; Kidney/metabolism ; Kidney Tubules, Proximal/drug effects ; Male ; Mice, Inbred C57BL ; RNA Interference ; Sodium Chloride, Dietary/administration & dosage ; Tumor Necrosis Factor-alpha/administration & dosage ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Tytuł :
SARS-CoV-2 receptor networks in diabetic and COVID-19-associated kidney disease.
Autorzy :
Menon R; Department of Computational Medicine and Bioinformatics, University of Michigan, Ann Arbor, Michigan, USA.
Otto EA; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Sealfon R; Center for Computational Biology, Flatiron Institute, New York, New York, USA.
Nair V; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Wong AK; Center for Computational Biology, Flatiron Institute, New York, New York, USA.
Theesfeld CL; Lewis-Sigler Institute for Integrative Genomics, Princeton University, Princeton, New Jersey, USA.
Chen X; Center for Computational Biology, Flatiron Institute, New York, New York, USA.
Wang Y; Lewis-Sigler Institute for Integrative Genomics, Princeton University, Princeton, New Jersey, USA; Department of Computer Science, Princeton University, Princeton, New Jersey, USA.
Boppana AS; Department of Computer Science, Princeton University, Princeton, New Jersey, USA.
Luo J; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Yang Y; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Kasson PM; Departments of Molecular Physiology and Biomedical Engineering, University of Virginia, Charlottesville, Virginia, USA.
Schaub JA; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Berthier CC; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Eddy S; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Lienczewski CC; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Godfrey B; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Dagenais SL; Advanced Genomics Core, Biomedical Research Core Facility, University of Michigan, Ann Arbor, Michigan, USA.
Sohaney R; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Hartman J; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Fermin D; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Subramanian L; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Looker HC; Chronic Kidney Disease Section, National Institute of Diabetes and Digestive and Kidney Diseases, Phoenix, Arizona, USA.
Harder JL; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Mariani LH; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Hodgin JB; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Sexton JZ; Department of Internal Medicine, Division of Gastroenterology and Hepatology, University of Michigan, Ann Arbor, Michigan, USA.
Wobus CE; Department of Microbiology and Immunology, University of Michigan, Ann Arbor, Michigan, USA.
Naik AS; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA.
Nelson RG; Chronic Kidney Disease Section, National Institute of Diabetes and Digestive and Kidney Diseases, Phoenix, Arizona, USA.
Troyanskaya OG; Center for Computational Biology, Flatiron Institute, New York, New York, USA; Lewis-Sigler Institute for Integrative Genomics, Princeton University, Princeton, New Jersey, USA; Department of Computer Science, Princeton University, Princeton, New Jersey, USA. Electronic address: .
Kretzler M; Department of Internal Medicine, Division of Nephrology, University of Michigan, Ann Arbor, Michigan, USA. Electronic address: .
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Źródło :
Kidney international [Kidney Int] 2020 Dec; Vol. 98 (6), pp. 1502-1518. Date of Electronic Publication: 2020 Oct 08.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Angiotensin-Converting Enzyme 2/*metabolism
COVID-19/*metabolism
Diabetic Nephropathies/*metabolism
Kidney Tubules, Proximal/*metabolism
SARS-CoV-2/*metabolism
Adult ; Aged ; Angiotensin Receptor Antagonists/pharmacology ; Angiotensin Receptor Antagonists/therapeutic use ; Angiotensin-Converting Enzyme Inhibitors/pharmacology ; Angiotensin-Converting Enzyme Inhibitors/therapeutic use ; COVID-19/complications ; COVID-19/virology ; Case-Control Studies ; Diabetic Nephropathies/drug therapy ; Female ; Gene Expression Profiling ; Gene Regulatory Networks ; Host-Pathogen Interactions ; Humans ; Kidney Tubules, Proximal/drug effects ; Male ; Middle Aged
Czasopismo naukowe
Tytuł :
Hnf4a Is Required for the Development of Cdh6-Expressing Progenitors into Proximal Tubules in the Mouse Kidney.
Autorzy :
Marable SS; Division of Pediatric Urology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio.; Division of Developmental Biology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio.
Chung E; Division of Pediatric Urology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio.; Division of Developmental Biology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio.
Park JS; Division of Pediatric Urology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio .; Division of Developmental Biology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio.; Department of Surgery, University of Cincinnati College of Medicine, Cincinnati, Ohio.
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Źródło :
Journal of the American Society of Nephrology : JASN [J Am Soc Nephrol] 2020 Nov; Vol. 31 (11), pp. 2543-2558. Date of Electronic Publication: 2020 Aug 06.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Cadherins/*metabolism
Hepatocyte Nuclear Factor 4/*genetics
Hepatocyte Nuclear Factor 4/*metabolism
Kidney Tubules, Proximal/*metabolism
Lectins/*metabolism
Stem Cells/*metabolism
Animals ; Cadherins/genetics ; Cell Differentiation/genetics ; Cell Proliferation ; Disease Models, Animal ; Fanconi Syndrome/genetics ; Female ; Gene Expression Regulation/genetics ; Gene Ontology ; Kidney Tubules, Proximal/pathology ; Kidney Tubules, Proximal/physiopathology ; Mice ; Mice, Knockout ; Phenotype ; Renal Reabsorption/genetics ; Stem Cells/physiology
Czasopismo naukowe
Tytuł :
Empagliflozin Inhibits Basal and IL-1β-Mediated MCP-1/CCL2 and Endothelin-1 Expression in Human Proximal Tubular Cells.
Autorzy :
Pirklbauer M; Department of Internal Medicine IV-Nephrology and Hypertension, Medical University Innsbruck, Anichstrasse 35, 6020 Innsbruck, Austria.
Bernd M; Department of Internal Medicine IV-Nephrology and Hypertension, Medical University Innsbruck, Anichstrasse 35, 6020 Innsbruck, Austria.
Fuchs L; Department of Internal Medicine IV-Nephrology and Hypertension, Medical University Innsbruck, Anichstrasse 35, 6020 Innsbruck, Austria.
Staudinger P; Department of Internal Medicine IV-Nephrology and Hypertension, Medical University Innsbruck, Anichstrasse 35, 6020 Innsbruck, Austria.
Corazza U; Department of Internal Medicine IV-Nephrology and Hypertension, Medical University Innsbruck, Anichstrasse 35, 6020 Innsbruck, Austria.
Leierer J; Department of Internal Medicine IV-Nephrology and Hypertension, Medical University Innsbruck, Anichstrasse 35, 6020 Innsbruck, Austria.
Mayer G; Department of Internal Medicine IV-Nephrology and Hypertension, Medical University Innsbruck, Anichstrasse 35, 6020 Innsbruck, Austria.
Schramek H; Department of Internal Medicine IV-Nephrology and Hypertension, Medical University Innsbruck, Anichstrasse 35, 6020 Innsbruck, Austria.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2020 Nov 01; Vol. 21 (21). Date of Electronic Publication: 2020 Nov 01.
Typ publikacji :
Journal Article
MeSH Terms :
Benzhydryl Compounds/*pharmacology
Chemokine CCL2/*genetics
Endothelin-1/*genetics
Gene Expression/*drug effects
Glucosides/*pharmacology
Interleukin-1beta/*pharmacology
Kidney Tubules, Proximal/*drug effects
Cell Line ; Chemokine CCL2/metabolism ; Endothelin-1/metabolism ; Gene Expression Profiling/methods ; Humans ; Kidney Tubules, Proximal/cytology ; Kidney Tubules, Proximal/metabolism ; Oligonucleotide Array Sequence Analysis/methods
Czasopismo naukowe

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