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Tytuł :
Comparison of CD16-negative selection vs. MACSxpress system for isolation of blood eosinophils.
Autorzy :
Fukuchi M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Ueki S; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan. Electronic address: .
Saito H; Department of Otorhinolaryngology, Head & Neck Surgery, Akita University Hospital, Akita, Japan.
Miyabe Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan; Department of Otorhinolaryngology, Head & Neck Surgery, Akita University Hospital, Akita, Japan.
Konno Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Omokawa A; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Saga T; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Moritoki Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Takeda M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Yamada T; Department of Otorhinolaryngology, Head & Neck Surgery, Akita University Hospital, Akita, Japan.
Hirokawa M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
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Źródło :
Allergology international : official journal of the Japanese Society of Allergology [Allergol Int] 2019 Sep; Vol. 68S, pp. S11-S13. Date of Electronic Publication: 2019 May 06.
Typ publikacji :
Letter
MeSH Terms :
Immunomagnetic Separation*/methods
Immunomagnetic Separation*/standards
Eosinophils/*metabolism
Receptors, IgG/*metabolism
Eosinophils/cytology ; Eosinophils/immunology ; GPI-Linked Proteins/metabolism ; Humans
Opinia redakcyjna
Tytuł :
Critical role of CCL4 in eosinophil recruitment into the airway.
Autorzy :
Kobayashi Y; Airway Disease Section, Department of Otolaryngology, Kansai Medical University, Osaka, Japan.; Allergy Center, Kansai Medical University Hospital, Osaka, Japan.
Konno Y; Department of General Medical Practice and Laboratory Diagnostic Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Kanda A; Airway Disease Section, Department of Otolaryngology, Kansai Medical University, Osaka, Japan.; Allergy Center, Kansai Medical University Hospital, Osaka, Japan.
Yamada Y; Department of Allergy and Immunology, Gunma Children's Medical Center, Gunma, Japan.
Yasuba H; Department of Airway Medicine, Mitsubishi Kyoto Hospital, Kyoto, Japan.
Sakata Y; Central Research of Laboratory, Kansai Medical University, Osaka, Japan.
Fukuchi M; Department of General Medical Practice and Laboratory Diagnostic Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Tomoda K; Airway Disease Section, Department of Otolaryngology, Kansai Medical University, Osaka, Japan.
Iwai H; Airway Disease Section, Department of Otolaryngology, Kansai Medical University, Osaka, Japan.
Ueki S; Department of General Medical Practice and Laboratory Diagnostic Medicine, Akita University Graduate School of Medicine, Akita, Japan.
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Źródło :
Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology [Clin Exp Allergy] 2019 Jun; Vol. 49 (6), pp. 853-860. Date of Electronic Publication: 2019 Mar 29.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Chemokine CCL4/*immunology
Eosinophils/*immunology
Pulmonary Eosinophilia/*immunology
Animals ; Antibodies, Neutralizing/immunology ; Antibodies, Neutralizing/pharmacology ; Chemokine CCL4/antagonists & inhibitors ; Disease Models, Animal ; Eosinophils/pathology ; Humans ; Mice ; Mice, Inbred BALB C ; Pulmonary Eosinophilia/pathology
Czasopismo naukowe
Tytuł :
Charcot-Leyden crystal formation is closely associated with eosinophil extracellular trap cell death.
Autorzy :
Ueki S; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.; Division of Allergy and Inflammation, Department of Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA.
Tokunaga T; Division of Otorhinolaryngology, Head & Neck Surgery, University of Fukui, Fukui, Japan.
Melo RCN; Division of Allergy and Inflammation, Department of Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA.; Laboratory of Cellular Biology, Department of Biology, Federal University of Juiz de Fora, Juiz de Fora, Brazil.
Saito H; Department of Otorhinolaryngology, Head & Neck Surgery, Akita University Hospital, Akita, Japan.
Honda K; Department of Otorhinolaryngology, Head & Neck Surgery, Akita University Hospital, Akita, Japan.
Fukuchi M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Konno Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Takeda M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Yamamoto Y; Department of Molecular Pathology and Tumor Pathology, Akita University Graduate School of Medicine, Akita, Japan; and.
Hirokawa M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Fujieda S; Division of Otorhinolaryngology, Head & Neck Surgery, University of Fukui, Fukui, Japan.
Spencer LA; Division of Allergy and Inflammation, Department of Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA.
Weller PF; Division of Allergy and Inflammation, Department of Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA.; Division of Infectious Diseases, Department of Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA.
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Źródło :
Blood [Blood] 2018 Nov 15; Vol. 132 (20), pp. 2183-2187. Date of Electronic Publication: 2018 Aug 28.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Death*
Eosinophils/*pathology
Extracellular Traps/*immunology
Galectins/*analysis
Inflammation/*pathology
Cell Membrane/immunology ; Cell Membrane/pathology ; Crystallization ; Eosinophils/cytology ; Eosinophils/immunology ; Galectins/immunology ; Humans ; Inflammation/immunology
Czasopismo naukowe
Tytuł :
ETosis-derived DNA trap production in middle ear effusion is a common feature of eosinophilic otitis media.
Autorzy :
Ohta N; Division of Otolaryngology, Tohoku Medical and Pharmaceutical University, Sendai, Japan; Yamagata City Hospital Saiseikan, Yamagata, Japan. Electronic address: .
Ueki S; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Konno Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Hirokawa M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Kubota T; Yamagata City Hospital Saiseikan, Yamagata, Japan; Department of Otolaryngology Head and Neck Surgery, Yamagata University Faculty of Medicine, Yamagata, Japan.
Tomioka-Matsutani S; Yamagata City Hospital Saiseikan, Yamagata, Japan.
Suzuki T; Yamagata City Hospital Saiseikan, Yamagata, Japan.
Ishida Y; Division of Anatomy and Cell Biology, Tohoku Medical and Pharmaceutical University, Sendai, Japan.
Kawano T; Division of Pathophysiology, Department of Pharmaceutical Sciences, Faculty of Pharmaceutical Sciences, Tohoku Medical and Pharmaceutical University, Sendai, Japan.
Miyasaka T; Division of Pathophysiology, Department of Pharmaceutical Sciences, Faculty of Pharmaceutical Sciences, Tohoku Medical and Pharmaceutical University, Sendai, Japan.
Takahashi T; Division of Pathophysiology, Department of Pharmaceutical Sciences, Faculty of Pharmaceutical Sciences, Tohoku Medical and Pharmaceutical University, Sendai, Japan.
Suzuki T; Department of Otolaryngology Head and Neck Surgery, Kitasato University Faculty of Medicine, Sagamihara, Japan.
Ohno I; Center for Medical Education, Faculty of Medicine, Tohoku Medical and Pharmaceutical University, Sendai, Japan.
Kakehata S; Department of Otolaryngology Head and Neck Surgery, Yamagata University Faculty of Medicine, Yamagata, Japan.
Fujieda S; Division of Otorhinolaryngology - Head & Neck Surgery, Department of Sensory and Locomotor Medicine, Faculty of Medical Science, University of Fukui, Fukui, Japan.
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Źródło :
Allergology international : official journal of the Japanese Society of Allergology [Allergol Int] 2018 Jul; Vol. 67 (3), pp. 414-416. Date of Electronic Publication: 2017 Dec 11.
Typ publikacji :
Case Reports; Letter
MeSH Terms :
Eosinophils/*immunology
Eosinophils/*pathology
Extracellular Traps/*immunology
Otitis Media with Effusion/*diagnosis
Otitis Media with Effusion/*etiology
Adult ; Cell Death ; Female ; Humans ; Immunoglobulin E/immunology ; Male ; Middle Aged ; Symptom Assessment
Raport
Tytuł :
ICAM-1 upregulation is not required for retinoic acid-induced human eosinophil survival.
Autorzy :
Ueki S; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan. Electronic address: .
Nishikawa J; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Fukuchi M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan; Department of Dentistry and Oral Surgery, Akita University Hospital, 1-1-1 Hondo, Akita 010-8543, Japan.
Konno Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan; Department of Dentistry and Oral Surgery, Akita University Hospital, 1-1-1 Hondo, Akita 010-8543, Japan.
Takeda M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Moritoki Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Chihara J; Soseikai General Hospital, 101 Shimotoba-Hironagacho, Kyoto 612-8473, Japan.
Omokawa A; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Saga T; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Hirokawa M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
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Źródło :
Immunology letters [Immunol Lett] 2018 Apr; Vol. 196, pp. 68-73. Date of Electronic Publication: 2018 Feb 01.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Eosinophils/*drug effects
Intercellular Adhesion Molecule-1/*metabolism
Tretinoin/*pharmacology
Up-Regulation/*drug effects
Apoptosis/drug effects ; Apoptosis/genetics ; Benzoates/pharmacology ; Biphenyl Compounds/pharmacology ; Cell Survival/drug effects ; Cell Survival/genetics ; Cells, Cultured ; Eosinophils/cytology ; Eosinophils/metabolism ; Gene Expression/drug effects ; Humans ; Intercellular Adhesion Molecule-1/genetics ; Retinoid X Receptors/antagonists & inhibitors ; Retinoid X Receptors/genetics ; Retinoid X Receptors/metabolism ; Signal Transduction/drug effects ; Signal Transduction/genetics
Czasopismo naukowe
Tytuł :
Eosinophilic Otitis Media: the Aftermath of Eosinophil Extracellular Trap Cell Death.
Autorzy :
Ueki S; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1, Hondo, Akita, 010-8543, Japan. .
Ohta N; Department of Otolaryngology, Tohoku Medical and Pharmaceutical University, Sendai, Japan.
Takeda M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1, Hondo, Akita, 010-8543, Japan.
Konno Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1, Hondo, Akita, 010-8543, Japan.; Department of Dentistry and Oral Surgery, Akita University Hospital, Akita, Japan.
Hirokawa M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1, Hondo, Akita, 010-8543, Japan.
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Źródło :
Current allergy and asthma reports [Curr Allergy Asthma Rep] 2017 May; Vol. 17 (5), pp. 33.
Typ publikacji :
Journal Article; Review
MeSH Terms :
Eosinophils/*immunology
Extracellular Traps/*immunology
Otitis Media/*immunology
Animals ; Cell Death ; Humans
Czasopismo naukowe
Tytuł :
The effect of hepatocyte growth factor on secretory functions in human eosinophils.
Autorzy :
Yamauchi Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Ueki S; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan. Electronic address: .
Konno Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Ito W; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan; Nagareyama Tobu Clinic, 909-1 Nazukari, Nagareyama City, Chiba 270-0145, Japan.
Takeda M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Nakamura Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Nishikawa J; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Moritoki Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Omokawa A; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Saga T; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Hirokawa M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
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Źródło :
Cytokine [Cytokine] 2016 Dec; Vol. 88, pp. 45-50. Date of Electronic Publication: 2016 Aug 20.
Typ publikacji :
Journal Article
MeSH Terms :
Eosinophils/*metabolism
Hepatocyte Growth Factor/*pharmacology
Secretory Vesicles/*metabolism
Eosinophils/ultrastructure ; Female ; Humans ; Interleukin-5/metabolism ; Male ; Secretory Vesicles/ultrastructure ; Transforming Growth Factor beta/metabolism ; Vascular Endothelial Growth Factor A/metabolism
Czasopismo naukowe
Tytuł :
Functional analysis of free fatty acid receptor GPR120 in human eosinophils: implications in metabolic homeostasis.
Autorzy :
Konno Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Ueki S; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Takeda M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Kobayashi Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan; Department of Otolaryngology, Kansai Medical University, Shin-machi, Hirakata City, Osaka, Japan.
Tamaki M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Moritoki Y; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Oyamada H; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Itoga M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan; Department of Clinical Laboratory Medicine, Hirosaki University Graduate School of Medicine, Hirosaki, Japan.
Kayaba H; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan; Department of Clinical Laboratory Medicine, Hirosaki University Graduate School of Medicine, Hirosaki, Japan.
Omokawa A; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Hirokawa M; Department of General Internal Medicine and Clinical Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
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Źródło :
PloS one [PLoS One] 2015 Mar 19; Vol. 10 (3), pp. e0120386. Date of Electronic Publication: 2015 Mar 19 (Print Publication: 2015).
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Eosinophils/*drug effects
Methylamines/*pharmacology
Propionates/*pharmacology
Receptors, G-Protein-Coupled/*genetics
Receptors, G-Protein-Coupled/*metabolism
Apoptosis/drug effects ; Caspase 3/metabolism ; Eosinophils/metabolism ; Eosinophils/physiology ; Homeostasis/drug effects ; Humans ; Interleukin-4/metabolism ; Protein Kinase Inhibitors/pharmacology ; Receptors, G-Protein-Coupled/agonists ; fas Receptor/metabolism
Czasopismo naukowe
Tytuł :
Expression and functional roles of G-protein-coupled estrogen receptor (GPER) in human eosinophils.
Autorzy :
Tamaki M; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Konno Y; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Kobayashi Y; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan; Department of Otolaryngology, Kansai Medical University, 2-5-1 Shin-machi, Hirakata City, Osaka 573-1010, Japan.
Takeda M; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Itoga M; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan; Department of Clinical Laboratory Medicine, Hirosaki University Graduate School of Medicine, 5 Zaifu-cho, Hirosaki 036-8562, Japan.
Moritoki Y; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Oyamada H; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan.
Kayaba H; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan; Department of Clinical Laboratory Medicine, Hirosaki University Graduate School of Medicine, 5 Zaifu-cho, Hirosaki 036-8562, Japan.
Chihara J; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan; Soseikai General Hospital, 101 Shimotoba Hirosacho, Fushimi-ku, Kyoto City, Kyoto 612-8473, Japan.
Ueki S; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan. Electronic address: .
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Źródło :
Immunology letters [Immunol Lett] 2014 Jul; Vol. 160 (1), pp. 72-78. Date of Electronic Publication: 2014 Apr 06.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Gene Expression Regulation*/drug effects
Eosinophils/*metabolism
Receptors, Estrogen/*genetics
Receptors, Estrogen/*metabolism
Receptors, G-Protein-Coupled/*genetics
Receptors, G-Protein-Coupled/*metabolism
Apoptosis/drug effects ; Cell Degranulation/drug effects ; Cell Degranulation/immunology ; Chemokine CCL11/pharmacology ; Chemotaxis, Leukocyte/drug effects ; Chemotaxis, Leukocyte/immunology ; Eosinophils/drug effects ; Eosinophils/immunology ; Humans ; Hypersensitivity/genetics ; Hypersensitivity/immunology ; Hypersensitivity/metabolism ; Male
Czasopismo naukowe
Tytuł :
Eliciting eosinophil CCR3 expression by synthetic retinoids.
Autorzy :
Ueki S; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Nishikawa J; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Konno Y; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Tamaki M; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Takeda M; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Moritoki Y; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Chihara J; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
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Źródło :
Allergology international : official journal of the Japanese Society of Allergology [Allergol Int] 2014 May; Vol. 63 Suppl 1, pp. 67-8.
Typ publikacji :
Letter; Research Support, Non-U.S. Gov't
MeSH Terms :
Eosinophils/*drug effects
Eosinophils/*metabolism
Gene Expression Regulation/*drug effects
Receptors, CCR3/*genetics
Retinoids/*pharmacology
Humans ; Receptors, CCR3/metabolism
Raport
Tytuł :
The effect of pharmacological PI3Kγ inhibitor on eotaxin-induced human eosinophil functions.
Autorzy :
Saito Y; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Takeda M; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan. Electronic address: .
Nishikawa J; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Konno Y; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Tamaki M; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Itoga M; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Kobayashi Y; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Moritoki Y; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Ito W; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Chihara J; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
Ueki S; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan.
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Źródło :
Pulmonary pharmacology & therapeutics [Pulm Pharmacol Ther] 2014 Apr; Vol. 27 (2), pp. 164-9. Date of Electronic Publication: 2013 Dec 09.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Phosphoinositide-3 Kinase Inhibitors*
Chemokine CCL11/*metabolism
Eosinophils/*drug effects
Quinoxalines/*pharmacology
Thiazolidinediones/*pharmacology
Chemotaxis/drug effects ; Class Ib Phosphatidylinositol 3-Kinase/metabolism ; Down-Regulation/drug effects ; Enzyme-Linked Immunosorbent Assay ; Eosinophil-Derived Neurotoxin/metabolism ; Eosinophils/metabolism ; Flow Cytometry ; Humans ; Mitogen-Activated Protein Kinase 1/metabolism ; Mitogen-Activated Protein Kinase 3/metabolism ; Phosphorylation
Czasopismo naukowe
Tytuł :
Adiponectin attenuates human eosinophil adhesion and chemotaxis: implications in allergic inflammation.
Autorzy :
Yamamoto R; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine , Akita , Japan.
Ueki S
Moritoki Y
Kobayashi Y
Oyamada H
Konno Y
Tamaki M
Itoga M
Takeda M
Ito W
Chihara J
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Źródło :
The Journal of asthma : official journal of the Association for the Care of Asthma [J Asthma] 2013 Oct; Vol. 50 (8), pp. 828-35. Date of Electronic Publication: 2013 Jul 17.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Adiponectin/*immunology
Asthma/*immunology
Cell Adhesion/*immunology
Chemotaxis/*immunology
Eosinophils/*immunology
Calcium Signaling/immunology ; Cell Survival/immunology ; Eosinophils/cytology ; Flow Cytometry ; Humans ; Neutrophils/immunology ; RNA/chemistry ; RNA/genetics ; Receptors, Adiponectin/immunology ; Reverse Transcriptase Polymerase Chain Reaction
Czasopismo naukowe
Tytuł :
Retinoic acids up-regulate functional eosinophil-driving receptor CCR3.
Autorzy :
Ueki S; Department of Infection, Allergy, Clinical Immunology and Laboratory Medicine, Akita University Graduate School of Medicine, Akita, Japan. />Nishikawa J
Yamauchi Y
Konno Y
Tamaki M
Itoga M
Kobayashi Y
Takeda M
Moritoki Y
Ito W
Chihara J
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Źródło :
Allergy [Allergy] 2013 Jul; Vol. 68 (7), pp. 953-6. Date of Electronic Publication: 2013 Jun 06.
Typ publikacji :
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Chemotaxis, Leukocyte/*drug effects
Dermatitis, Atopic/*blood
Eosinophils/*drug effects
Receptors, CCR3/*genetics
Tretinoin/*pharmacology
Cells, Cultured ; Chemokine CCL24/genetics ; Chemokine CCL24/metabolism ; Chemotactic Factors, Eosinophil/genetics ; Chemotactic Factors, Eosinophil/metabolism ; Chemotaxis, Leukocyte/genetics ; Dermatitis, Atopic/genetics ; Eosinophils/immunology ; Gene Expression Regulation ; Humans ; Receptors, CCR3/metabolism ; Sensitivity and Specificity ; Signal Transduction/genetics ; Signal Transduction/immunology ; Up-Regulation
Czasopismo naukowe

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