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Wyświetlanie 1-15 z 15
Tytuł :
Publisher Correction: Whole-genome doubling confers unique genetic vulnerabilities on tumour cells.
Autorzy :
Quinton RJ; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
DiDomizio A; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
Vittoria MA; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
Kotýnková K; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
Ticas CJ; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
Patel S; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
Koga Y; Department of Medicine, Boston University School of Medicine, Boston, MA, USA.
Vakhshoorzadeh J; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
Hermance N; Department of Biology and Biotechnology, Worcester Polytechnic Institute, Worcester, MA, USA.
Kuroda TS; Department of Molecular Pathobiology and Cell Adhesion Biology, Mie University Graduate School of Medicine, Mie, Japan.
Parulekar N; Department of Medicine, Boston University School of Medicine, Boston, MA, USA.
Taylor AM; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.; Cancer Program, Broad Institute, Cambridge, MA, USA.; Department of Pathology and Cell Biology, Columbia University Medical Center, Member, Herbert Irving Comprehensive Cancer Center, New York, NY, USA.
Manning AL; Department of Biology and Biotechnology, Worcester Polytechnic Institute, Worcester, MA, USA.
Campbell JD; Department of Medicine, Boston University School of Medicine, Boston, MA, USA.; Cancer Program, Broad Institute, Cambridge, MA, USA.
Ganem NJ; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA. .; Department of Medicine, Boston University School of Medicine, Boston, MA, USA. .
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Źródło :
Nature [Nature] 2021 May 11. Date of Electronic Publication: 2021 May 11.
Typ publikacji :
Published Erratum
Tytuł :
Whole-genome doubling confers unique genetic vulnerabilities on tumour cells.
Autorzy :
Quinton RJ; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
DiDomizio A; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
Vittoria MA; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
Kotýnková K; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
Ticas CJ; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
Patel S; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
Koga Y; Department of Medicine, Boston University School of Medicine, Boston, MA, USA.
Vakhshoorzadeh J; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA.
Hermance N; Department of Biology and Biotechnology, Worcester Polytechnic Institute, Worcester, MA, USA.
Kuroda TS; Department of Molecular Pathobiology and Cell Adhesion Biology, Mie University Graduate School of Medicine, Mie, Japan.
Parulekar N; Department of Medicine, Boston University School of Medicine, Boston, MA, USA.
Taylor AM; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA, USA.; Cancer Program, Broad Institute, Cambridge, MA, USA.; Department of Pathology and Cell Biology, Columbia University Medical Center, Member, Herbert Irving Comprehensive Cancer Center, New York, NY, USA.
Manning AL; Department of Biology and Biotechnology, Worcester Polytechnic Institute, Worcester, MA, USA.
Campbell JD; Department of Medicine, Boston University School of Medicine, Boston, MA, USA.; Cancer Program, Broad Institute, Cambridge, MA, USA.
Ganem NJ; Department of Pharmacology & Experimental Therapeutics, Boston University School of Medicine, Boston, MA, USA. .; Department of Medicine, Boston University School of Medicine, Boston, MA, USA. .
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Źródło :
Nature [Nature] 2021 Feb; Vol. 590 (7846), pp. 492-497. Date of Electronic Publication: 2021 Jan 27.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Tetraploidy*
Genome, Human/*genetics
Mitosis/*drug effects
Neoplasms/*genetics
Neoplasms/*pathology
Abnormal Karyotype/drug effects ; Breast Neoplasms/genetics ; Breast Neoplasms/pathology ; Cell Line, Tumor ; Female ; Genes, Lethal/genetics ; Humans ; Kinesin/deficiency ; Kinesin/genetics ; Kinesin/metabolism ; M Phase Cell Cycle Checkpoints/drug effects ; Male ; Mitosis/genetics ; Proteasome Endopeptidase Complex/metabolism ; Reproducibility of Results ; Spindle Apparatus/drug effects
Czasopismo naukowe
Tytuł :
FBW7 Loss Promotes Chromosomal Instability and Tumorigenesis via Cyclin E1/CDK2-Mediated Phosphorylation of CENP-A.
Autorzy :
Takada M; Lineberger Comprehensive Cancer Center, University of North Carolina School of Medicine, Chapel Hill, North Carolina.
Zhang W; Biological Systems and Engineering Division, Lawrence Berkeley National Laboratory and Department of Molecular and Cell Biology, University of California, Berkeley, California.
Suzuki A; Department of Biology, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina.
Kuroda TS; Open Innovation Center Japan, Bayer Yakuhin, Ltd., Kita-ku, Osaka, Japan.
Yu Z; National Laboratory of Biomacromolecules, Institute of Biophysics, Chinese Academy of Sciences, Beijing, China.
Inuzuka H; Department of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts.
Gao D; Department of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts.
Wan L; Department of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts.
Zhuang M; Xin Hua Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Hu L; Lineberger Comprehensive Cancer Center, University of North Carolina School of Medicine, Chapel Hill, North Carolina.
Zhai B; St. Jude Children's Research Hospital, Memphis, Tennessee.
Fry CJ; Cell Signaling Technology, Danvers, Massachusetts.
Bloom K; Department of Biology, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina.
Li G; National Laboratory of Biomacromolecules, Institute of Biophysics, Chinese Academy of Sciences, Beijing, China.
Karpen GH; Biological Systems and Engineering Division, Lawrence Berkeley National Laboratory and Department of Molecular and Cell Biology, University of California, Berkeley, California. .
Wei W; Department of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts. .
Zhang Q; Lineberger Comprehensive Cancer Center, University of North Carolina School of Medicine, Chapel Hill, North Carolina. .; Department of Pathology and Laboratory Medicine, University of North Carolina, Chapel Hill, North Carolina.
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Źródło :
Cancer research [Cancer Res] 2017 Sep 15; Vol. 77 (18), pp. 4881-4893. Date of Electronic Publication: 2017 Jul 31.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Chromosomal Instability*
Autoantigens/*metabolism
Breast Neoplasms/*pathology
Cell Cycle Proteins/*metabolism
Cell Transformation, Neoplastic/*pathology
Chromosomal Proteins, Non-Histone/*metabolism
Colonic Neoplasms/*pathology
Cyclin E/*metabolism
Cyclin-Dependent Kinase 2/*metabolism
F-Box Proteins/*metabolism
Oncogene Proteins/*metabolism
Ubiquitin-Protein Ligases/*metabolism
Apoptosis ; Biomarkers, Tumor/metabolism ; Breast Neoplasms/genetics ; Breast Neoplasms/metabolism ; Cell Cycle ; Cell Proliferation ; Cell Transformation, Neoplastic/genetics ; Cell Transformation, Neoplastic/metabolism ; Centromere ; Centromere Protein A ; Colonic Neoplasms/genetics ; Colonic Neoplasms/metabolism ; F-Box-WD Repeat-Containing Protein 7 ; Female ; Histones/metabolism ; Humans ; Phosphorylation ; Tumor Cells, Cultured
Czasopismo naukowe
Tytuł :
Identification and biochemical analysis of Slac2-c/MyRIP as a Rab27A-, myosin Va/VIIa-, and actin-binding protein.
Autorzy :
Kuroda TS
Fukuda M
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Źródło :
Methods in enzymology [Methods Enzymol] 2005; Vol. 403, pp. 431-44.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Carrier Proteins/*metabolism
Dyneins/*metabolism
Microfilament Proteins/*metabolism
Myosin Heavy Chains/*metabolism
Myosin Type V/*metabolism
Myosins/*metabolism
rab GTP-Binding Proteins/*metabolism
Adaptor Proteins, Signal Transducing ; Animals ; COS Cells ; Chlorocebus aethiops ; Mice ; Myosin VIIa ; PC12 Cells ; Rats ; rab27 GTP-Binding Proteins
Czasopismo naukowe
Tytuł :
Functional analysis of slac2-a/melanophilin as a linker protein between Rab27A and myosin Va in melanosome transport.
Autorzy :
Kuroda TS
Itoh T
Fukuda M
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Źródło :
Methods in enzymology [Methods Enzymol] 2005; Vol. 403, pp. 419-31.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Carrier Proteins/*physiology
Melanosomes/*physiology
Myosin Heavy Chains/*physiology
Myosin Type V/*physiology
rab GTP-Binding Proteins/*physiology
Adaptor Proteins, Signal Transducing ; Animals ; Base Sequence ; Biological Transport ; Cells, Cultured ; DNA Primers ; Fluorescent Antibody Technique ; Mice ; Mice, Inbred C57BL ; RNA Interference ; RNA, Small Interfering ; rab27 GTP-Binding Proteins
Czasopismo naukowe
    Wyświetlanie 1-15 z 15

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