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Wyszukujesz frazę ""Lang, Sabine M."" wg kryterium: Autor

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    Wyświetlanie 1-13 z 13
    Tytuł:
    Tick tock, tick tock: Mouse culture and tissue aging captured by an epigenetic clock.
    Autorzy:
    Minteer C; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, USA.
    Morselli M; Department of Molecular, Cell, and Developmental Biology, University of California Los Angeles, Los Angeles, California, USA.
    Meer M; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, USA.
    Cao J; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, USA.; Rutgers Cancer Institute of New Jersey, New Brunswick, New Jersey, USA.
    Higgins-Chen A; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, USA.
    Lang SM; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, USA.
    Pellegrini M; Department of Molecular, Cell, and Developmental Biology, University of California Los Angeles, Los Angeles, California, USA.
    Yan Q; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, USA.
    Levine ME; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, USA.
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    Źródło:
    Aging cell [Aging Cell] 2022 Feb; Vol. 21 (2), pp. e13553. Date of Electronic Publication: 2022 Feb 01.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Epigenesis, Genetic*
    Fibroblasts*
    Aging/genetics ; Animals ; DNA Methylation/genetics ; Epigenomics ; Mice
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    KDM5 histone demethylases repress immune response via suppression of STING.
    Autorzy:
    Wu L; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, United States of America.
    Cao J; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, United States of America.
    Cai WL; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, United States of America.
    Lang SM; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, United States of America.
    Horton JR; Department of Molecular and Cellular Oncology, The University of Texas MD Anderson Cancer Center, Houston, Texas, United States of America.
    Jansen DJ; National Center for Advancing Translational Sciences, National Institutes of Health, Rockville, Maryland, United States of America.
    Liu ZZ; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, United States of America.
    Chen JF; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, United States of America.
    Zhang M; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, United States of America.
    Mott BT; National Center for Advancing Translational Sciences, National Institutes of Health, Rockville, Maryland, United States of America.
    Pohida K; National Center for Advancing Translational Sciences, National Institutes of Health, Rockville, Maryland, United States of America.
    Rai G; National Center for Advancing Translational Sciences, National Institutes of Health, Rockville, Maryland, United States of America.
    Kales SC; National Center for Advancing Translational Sciences, National Institutes of Health, Rockville, Maryland, United States of America.
    Henderson MJ; National Center for Advancing Translational Sciences, National Institutes of Health, Rockville, Maryland, United States of America.
    Hu X; National Center for Advancing Translational Sciences, National Institutes of Health, Rockville, Maryland, United States of America.
    Jadhav A; National Center for Advancing Translational Sciences, National Institutes of Health, Rockville, Maryland, United States of America.
    Maloney DJ; National Center for Advancing Translational Sciences, National Institutes of Health, Rockville, Maryland, United States of America.
    Simeonov A; National Center for Advancing Translational Sciences, National Institutes of Health, Rockville, Maryland, United States of America.
    Zhu S; Institute of Immunology and the CAS Key Laboratory of Innate Immunity and Chronic Disease, School of Life Sciences and Medical Center, University of Science and Technology of China, Hefei, China.
    Iwasaki A; Department of Immunobiology, Yale School of Medicine, New Haven, Connecticut, United States of America.; Howard Hughes Medical Institute, Chevy Chase, Maryland, United States of America.
    Hall MD; National Center for Advancing Translational Sciences, National Institutes of Health, Rockville, Maryland, United States of America.
    Cheng X; Department of Molecular and Cellular Oncology, The University of Texas MD Anderson Cancer Center, Houston, Texas, United States of America.
    Shadel GS; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, United States of America.; Department of Genetics, Yale School of Medicine, New Haven, Connecticut, United States of America.; Salk Institute for Biological Studies, La Jolla, California, United States of America.
    Yan Q; Department of Pathology, Yale School of Medicine, New Haven, Connecticut, United States of America.
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    Źródło:
    PLoS biology [PLoS Biol] 2018 Aug 06; Vol. 16 (8), pp. e2006134. Date of Electronic Publication: 2018 Aug 06 (Print Publication: 2018).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Histone Demethylases/*physiology
    Jumonji Domain-Containing Histone Demethylases/*physiology
    Membrane Proteins/*physiology
    Nuclear Proteins/*physiology
    Repressor Proteins/*physiology
    Cell Line ; Cytosol/metabolism ; DNA/metabolism ; Histone Methyltransferases/physiology ; Histones/physiology ; Humans ; Immunity, Innate/physiology ; Immunotherapy ; Interferons/metabolism ; Interferons/physiology ; MCF-7 Cells ; Membrane Proteins/metabolism ; Signal Transduction
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Mitochondrial DNA stress primes the antiviral innate immune response.
    Autorzy:
    West AP; Department of Pathology, Yale School of Medicine, New Haven, Connecticut 06520, USA.
    Khoury-Hanold W; Department of Immunobiology, Yale School of Medicine, New Haven, Connecticut 06520, USA.
    Staron M; Department of Immunobiology, Yale School of Medicine, New Haven, Connecticut 06520, USA.
    Tal MC; Department of Immunobiology, Yale School of Medicine, New Haven, Connecticut 06520, USA.
    Pineda CM; Department of Pathology, Yale School of Medicine, New Haven, Connecticut 06520, USA.
    Lang SM; Department of Pathology, Yale School of Medicine, New Haven, Connecticut 06520, USA.
    Bestwick M; Department of Pathology, Yale School of Medicine, New Haven, Connecticut 06520, USA.
    Duguay BA; Li Ka Shing Institute of Virology, Department of Medical Microbiology and Immunology, University of Alberta, Edmonton, Alberta T6G 2S2, Canada.
    Raimundo N; Department of Pathology, Yale School of Medicine, New Haven, Connecticut 06520, USA.
    MacDuff DA; Department of Pathology and Immunology, Washington University School of Medicine, St Louis, Missouri 63110, USA.
    Kaech SM; 1] Department of Immunobiology, Yale School of Medicine, New Haven, Connecticut 06520, USA [2] Howard Hughes Medical Institute, Chevy Chase, Maryland 20815-6789, USA.
    Smiley JR; Li Ka Shing Institute of Virology, Department of Medical Microbiology and Immunology, University of Alberta, Edmonton, Alberta T6G 2S2, Canada.
    Means RE; Department of Pathology, Yale School of Medicine, New Haven, Connecticut 06520, USA.
    Iwasaki A; 1] Department of Immunobiology, Yale School of Medicine, New Haven, Connecticut 06520, USA [2] Howard Hughes Medical Institute, Chevy Chase, Maryland 20815-6789, USA.
    Shadel GS; 1] Department of Pathology, Yale School of Medicine, New Haven, Connecticut 06520, USA [2] Department of Genetics, Yale School of Medicine, New Haven, Connecticut 06520, USA.
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    Źródło:
    Nature [Nature] 2015 Apr 23; Vol. 520 (7548), pp. 553-7. Date of Electronic Publication: 2015 Feb 02.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Stress, Physiological*
    DNA, Mitochondrial/*metabolism
    Herpesvirus 1, Human/*immunology
    Immunity, Innate/*immunology
    Animals ; Cell Line ; DNA-Binding Proteins/deficiency ; DNA-Binding Proteins/genetics ; DNA-Binding Proteins/metabolism ; Female ; Gene Expression Regulation/genetics ; Gene Expression Regulation/immunology ; High Mobility Group Proteins/deficiency ; High Mobility Group Proteins/genetics ; High Mobility Group Proteins/metabolism ; Humans ; Interferon Regulatory Factor-3/metabolism ; Interferon Type I/immunology ; Membrane Proteins/metabolism ; Mice ; Nucleotidyltransferases/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    The MARCH family E3 ubiquitin ligase K5 alters monocyte metabolism and proliferation through receptor tyrosine kinase modulation.
    Autorzy:
    Karki R; Department of Pathology, Yale University School of Medicine, New Haven, Connecticut, United States of America.
    Lang SM
    Means RE
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    Źródło:
    PLoS pathogens [PLoS Pathog] 2011 Apr; Vol. 7 (4), pp. e1001331. Date of Electronic Publication: 2011 Apr 07.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Herpesvirus 8, Human/*enzymology
    Immediate-Early Proteins/*metabolism
    Monocytes/*metabolism
    Receptor Protein-Tyrosine Kinases/*metabolism
    Ubiquitin-Protein Ligases/*metabolism
    Cell Line, Tumor ; Cell Proliferation ; Down-Regulation ; Endocytosis ; Glucose/metabolism ; Humans ; Lactic Acid/metabolism ; Monocytes/virology ; Phosphorylation ; Proto-Oncogene Proteins c-akt/metabolism ; Sarcoma, Kaposi/immunology ; Sarcoma, Kaposi/pathology ; Ubiquitin/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Placental viral infection sensitizes to endotoxin-induced pre-term labor: a double hit hypothesis.
    Autorzy:
    Cardenas I; Department of Obstetrics Gynecology and Reproductive Sciences, Reproductive Immunology Unit, School of Medicine, Yale University, New Haven, CT 06520, USA.
    Mor G
    Aldo P
    Lang SM
    Stabach P
    Sharp A
    Romero R
    Mazaki-Tovi S
    Gervasi M
    Means RE
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    Źródło:
    American journal of reproductive immunology (New York, N.Y. : 1989) [Am J Reprod Immunol] 2011 Feb; Vol. 65 (2), pp. 110-7. Date of Electronic Publication: 2010 Aug 13.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural
    MeSH Terms:
    Endotoxins/*pharmacology
    Lipopolysaccharides/*pharmacology
    Obstetric Labor, Premature/*etiology
    Placenta Diseases/*virology
    Rhadinovirus/*pathogenicity
    Animals ; Bacterial Infections/immunology ; Bacterial Infections/microbiology ; Female ; Fetal Death ; Herpesviridae Infections/virology ; Humans ; Labor, Induced ; Mice ; Mice, Inbred C57BL ; NIH 3T3 Cells ; Placenta/drug effects ; Placenta/virology ; Placenta Diseases/immunology ; Placenta Diseases/microbiology ; Pregnancy ; Tumor Virus Infections/virology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    The Kaposi’s sarcoma-associated herpesvirus E3 ubiquitin ligase K5 acts as a novel oncogene, altering cellular metabolism and signaling: implications for tumorigenesis
    Autorzy:
    Means Robert E
    Lang Sabine M
    Karki Roshan
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    Temat:
    Neoplasms. Tumors. Oncology. Including cancer and carcinogens
    RC254-282
    Infectious and parasitic diseases
    RC109-216
    Źródło:
    Infectious Agents and Cancer, Vol 5, Iss Suppl 1, p A79 (2010)
    Opis pliku:
    electronic resource
    Relacje:
    https://doaj.org/toc/1750-9378
    Dostęp URL:
    https://doaj.org/article/db18f544a26a46999fc5cd1366c27b54  Link otwiera się w nowym oknie
    Czasopismo naukowe
    Szczegóły
    Tytuł:
    Identification of a nef allele that causes lymphocyte activation and acute disease in...
    Autorzy:
    Du, Zhenjian
    Lang, Sabine M.
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    Źródło:
    Cell. 8/25/95, Vol. 82 Issue 4, p665. 9p. 12 Color Photographs, 9 Black and White Photographs, 6 Diagrams, 4 Graphs.
    Czasopismo naukowe
    Szczegóły
      Wyświetlanie 1-13 z 13

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