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Tytuł:
Heat shock protein 40 of Streptococcus pneumoniae induces immune response of human dendritic cells via TLR4-dependent p38 MAPK and JNK signaling pathways.
Autorzy:
Liu JJ; Clinical Laboratory, Xiamen Children's Hospital, Xiamen, China.
Lin JC; Clinical Laboratory, Xiamen Children's Hospital, Xiamen, China.
Chen LN; Clinical Laboratory, Xiamen Children's Hospital, Xiamen, China.
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Źródło:
Immunity, inflammation and disease [Immun Inflamm Dis] 2022 Dec; Vol. 10 (12), pp. e735.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
p38 Mitogen-Activated Protein Kinases*
MAP Kinase Signaling System*
Humans ; HSP40 Heat-Shock Proteins ; Streptococcus pneumoniae ; Interleukin-6 ; Tumor Necrosis Factor-alpha ; Toll-Like Receptor 4 ; Immunity ; Dendritic Cells
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Regulation of the p38-MAPK pathway by hyperosmolarity and by WNK kinases.
Autorzy:
Liu Z; Cell Biology Program, The Hospital for Sick Children, PGCRL 19-9715, 686 Bay St., Toronto, ON, M5G 0A4, Canada.; Biochemistry Department, University of Toronto, Toronto, ON, Canada.
Demian W; Cell Biology Program, The Hospital for Sick Children, PGCRL 19-9715, 686 Bay St., Toronto, ON, M5G 0A4, Canada.; Biochemistry Department, University of Toronto, Toronto, ON, Canada.
Persaud A; Cell Biology Program, The Hospital for Sick Children, PGCRL 19-9715, 686 Bay St., Toronto, ON, M5G 0A4, Canada.
Jiang C; Cell Biology Program, The Hospital for Sick Children, PGCRL 19-9715, 686 Bay St., Toronto, ON, M5G 0A4, Canada.
Subramanaya AR; Department of Medicine and Cell Biology, University of Pittsburgh, Pittsburgh, USA.
Rotin D; Cell Biology Program, The Hospital for Sick Children, PGCRL 19-9715, 686 Bay St., Toronto, ON, M5G 0A4, Canada. .; Biochemistry Department, University of Toronto, Toronto, ON, Canada. .
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Źródło:
Scientific reports [Sci Rep] 2022 Aug 25; Vol. 12 (1), pp. 14480. Date of Electronic Publication: 2022 Aug 25.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
MAP Kinase Signaling System*
p38 Mitogen-Activated Protein Kinases*/metabolism
Enzyme Activation ; MAP Kinase Kinase 3/metabolism ; TNF Receptor-Associated Factor 2/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Dimethyl Itaconate Reduces α-MSH-Induced Pigmentation via Modulation of AKT and p38 MAPK Signaling Pathways in B16F10 Mouse Melanoma Cells.
Autorzy:
Jang S; Department of Pharmaceutical Engineering & Biotechnology, Sun Moon University, Asan 31460, Korea.
Chi WJ; Microorganism Resources Division, National Institute of Biological Resources, Incheon 17058, Korea.
Kim SY; Department of Pharmaceutical Engineering & Biotechnology, Sun Moon University, Asan 31460, Korea.
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2022 Jun 29; Vol. 27 (13). Date of Electronic Publication: 2022 Jun 29.
Typ publikacji:
Journal Article
MeSH Terms:
Hyperpigmentation*/metabolism
MAP Kinase Signaling System*/drug effects
Melanoma, Experimental*/drug therapy
Melanoma, Experimental*/metabolism
Proto-Oncogene Proteins c-akt*/metabolism
p38 Mitogen-Activated Protein Kinases*/metabolism
Animals ; Cell Line, Tumor ; Glycogen Synthase Kinase 3 beta/metabolism ; Melanins/metabolism ; Mice ; Microphthalmia-Associated Transcription Factor/genetics ; Microphthalmia-Associated Transcription Factor/metabolism ; Monophenol Monooxygenase/metabolism ; NF-E2-Related Factor 2/metabolism ; Pigmentation ; Signal Transduction ; Succinates ; alpha-MSH/metabolism ; alpha-MSH/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
MAPK Pathways in Ocular Pathophysiology: Potential Therapeutic Drugs and Challenges.
Autorzy:
Moustardas P; Division of Ophthalmology, Department of Biomedical and Clinical Sciences, Linköping University, 58185 Linköping, Sweden.
Aberdam D; INSERM U1138, Centre des Cordeliers, 75270 Paris, France.; Université de Paris Cité, 75014 Paris, France.
Lagali N; Division of Ophthalmology, Department of Biomedical and Clinical Sciences, Linköping University, 58185 Linköping, Sweden.; Department of Ophthalmology, Sørlandet Hospital Arendal, 4838 Arendal, Norway.
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Źródło:
Cells [Cells] 2023 Feb 14; Vol. 12 (4). Date of Electronic Publication: 2023 Feb 14.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
Mitogen-Activated Protein Kinases*/metabolism
p38 Mitogen-Activated Protein Kinases*/metabolism
Signal Transduction ; Phosphorylation ; MAP Kinase Signaling System/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Activation of ERK and p38 Reduces AZD8055-Mediated Inhibition of Protein Synthesis in Hepatocellular Carcinoma HepG2 Cell Line.
Autorzy:
Jee HY; Department of Biological Sciences, Konkuk University, Seoul 05029, Korea.
Lee YG; Department of Biological Sciences, Konkuk University, Seoul 05029, Korea.
Lee S; Department of Biological Sciences, Konkuk University, Seoul 05029, Korea.
Elvira R; Soonchunhyang Institute of Medi-Bio Science (SIMS), Soonchunhyang University, Cheonan 31151, Korea.
Seo HE; Department of Biological Sciences, Konkuk University, Seoul 05029, Korea.
Lee JY; Department of Biological Sciences, Konkuk University, Seoul 05029, Korea.
Han J; Soonchunhyang Institute of Medi-Bio Science (SIMS), Soonchunhyang University, Cheonan 31151, Korea.
Lee K; Department of Biological Sciences, Konkuk University, Seoul 05029, Korea.; Korea Hemp Institute, Konkuk University, Seoul 05029, Korea.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Oct 30; Vol. 22 (21). Date of Electronic Publication: 2021 Oct 30.
Typ publikacji:
Journal Article
MeSH Terms:
MAP Kinase Signaling System*
Carcinoma, Hepatocellular/*metabolism
Liver Neoplasms/*metabolism
Morpholines/*pharmacology
Neoplasm Proteins/*metabolism
Protein Biosynthesis/*drug effects
p38 Mitogen-Activated Protein Kinases/*metabolism
Carcinoma, Hepatocellular/pathology ; Enzyme Activation/drug effects ; Hep G2 Cells ; Humans ; Liver Neoplasms/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Atypical p38 Signaling, Activation, and Implications for Disease.
Autorzy:
Burton JC; Department of Pharmaceutical and Biomedical Sciences, College of Pharmacy, University of Georgia, Athens, GA 30602, USA.
Antoniades W; Department of Pharmaceutical and Biomedical Sciences, College of Pharmacy, University of Georgia, Athens, GA 30602, USA.
Okalova J; Department of Pharmaceutical and Biomedical Sciences, College of Pharmacy, University of Georgia, Athens, GA 30602, USA.
Roos MM; Department of Pharmaceutical and Biomedical Sciences, College of Pharmacy, University of Georgia, Athens, GA 30602, USA.
Grimsey NJ; Department of Pharmaceutical and Biomedical Sciences, College of Pharmacy, University of Georgia, Athens, GA 30602, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Apr 17; Vol. 22 (8). Date of Electronic Publication: 2021 Apr 17.
Typ publikacji:
Journal Article; Review
MeSH Terms:
MAP Kinase Signaling System*
Cardiovascular Diseases/*metabolism
Communicable Diseases/*metabolism
Neurodegenerative Diseases/*metabolism
p38 Mitogen-Activated Protein Kinases/*metabolism
Animals ; Humans
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
S100A16 suppresses the proliferation, migration and invasion of colorectal cancer cells in part via the JNK/p38 MAPK pathway.
Autorzy:
Ou S; Guangdong Provincial Key Laboratory of Gastroenterology, Department of Gastroenterology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong 510515, P.R. China.
Liao Y; Guangdong Provincial Key Laboratory of Gastroenterology, Department of Gastroenterology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong 510515, P.R. China.
Shi J; Guangdong Provincial Key Laboratory of Gastroenterology, Department of Gastroenterology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong 510515, P.R. China.
Tang J; Department of Gastroenterology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi 530021, P.R. China.
Ye Y; Guangdong Provincial Key Laboratory of Gastroenterology, Department of Gastroenterology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong 510515, P.R. China.
Wu F; Guangdong Provincial Key Laboratory of Gastroenterology, Department of Gastroenterology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong 510515, P.R. China.
Wang W; Guangdong Provincial Key Laboratory of Gastroenterology, Department of Gastroenterology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong 510515, P.R. China.
Fei J; Guangdong Provincial Key Laboratory of Gastroenterology, Department of Gastroenterology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong 510515, P.R. China.
Xie F; Guangdong Provincial Key Laboratory of Gastroenterology, Department of Gastroenterology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong 510515, P.R. China.
Bai L; Guangdong Provincial Key Laboratory of Gastroenterology, Department of Gastroenterology, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong 510515, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2021 Feb; Vol. 23 (2). Date of Electronic Publication: 2020 Dec 23.
Typ publikacji:
Journal Article
MeSH Terms:
Cell Movement*
Cell Proliferation*
MAP Kinase Signaling System*
Colorectal Neoplasms/*metabolism
MAP Kinase Kinase 4/*metabolism
Neoplasm Proteins/*metabolism
S100 Proteins/*metabolism
p38 Mitogen-Activated Protein Kinases/*metabolism
Colorectal Neoplasms/genetics ; Colorectal Neoplasms/pathology ; Female ; HCT116 Cells ; Humans ; MAP Kinase Kinase 4/genetics ; Male ; Neoplasm Invasiveness ; Neoplasm Proteins/genetics ; S100 Proteins/genetics ; p38 Mitogen-Activated Protein Kinases/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
β-elemene regulates M1-M2 macrophage balance through the ERK/JNK/P38 MAPK signaling pathway.
Autorzy:
Zhou Y; Research Center for Food Safety, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan.; Department of Applied Biological Chemistry, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan.
Takano T; Research Center for Food Safety, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan.; Department of Applied Biological Chemistry, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan.
Li X; Department of Applied Biological Chemistry, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan.
Wang Y; Research Center for Food Safety, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan.; Department of Applied Biological Chemistry, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan.
Wang R; Research Center for Food Safety, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan.; Department of Applied Biological Chemistry, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan.
Zhu Z; Department of Applied Biological Chemistry, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan.; Key Laboratory of Industrial Fermentation Microbiology of the Ministry of Education, Tianjin Key Laboratory of Industrial Microbiology, College of Biotechnology, Tianjin University of Science and Technology, National Engineering Laboratory for Industrial Enzymes, Tianjin, 300457, P. R. China.
Tanokura M; Research Center for Food Safety, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan. .; Department of Applied Biological Chemistry, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan. .
Miyakawa T; Department of Applied Biological Chemistry, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan. .
Hachimura S; Research Center for Food Safety, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan. .; Department of Applied Biological Chemistry, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, 113-8657, Japan. .
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Źródło:
Communications biology [Commun Biol] 2022 May 31; Vol. 5 (1), pp. 519. Date of Electronic Publication: 2022 May 31.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Macrophages*/metabolism
p38 Mitogen-Activated Protein Kinases*/metabolism
Animals ; Cytokines/metabolism ; Inflammation/drug therapy ; Inflammation/metabolism ; MAP Kinase Signaling System ; Mice ; Obesity/drug therapy ; Obesity/etiology ; Obesity/metabolism ; Sesquiterpenes
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Lablab Purpureus Protects HaCaT Cells from Oxidative Stress-Induced Cell Death through Nrf2-Mediated Heme Oxygenase-1 Expression via the Activation of p38 and ERK1/2.
Autorzy:
Diniyah N; Department of Food Science and Biotechnology, Graduate School, Kyungpook National University, Daegu 41566, Korea.; Faculty of Agricultural Technology, University of Jember, Jember 68121, East Java, Indonesia.
Alam MB; Department of Food Science and Biotechnology, Graduate School, Kyungpook National University, Daegu 41566, Korea.; Food and Bio-Industry Research Institute, Kyungpook National University, Daegu 41566, Korea.
Choi HJ; Department of Food Science and Biotechnology, Graduate School, Kyungpook National University, Daegu 41566, Korea.
Lee SH; Department of Food Science and Biotechnology, Graduate School, Kyungpook National University, Daegu 41566, Korea.; Food and Bio-Industry Research Institute, Kyungpook National University, Daegu 41566, Korea.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Nov 14; Vol. 21 (22). Date of Electronic Publication: 2020 Nov 14.
Typ publikacji:
Journal Article
MeSH Terms:
MAP Kinase Signaling System*/drug effects
MAP Kinase Signaling System*/radiation effects
Oxidative Stress*/drug effects
Oxidative Stress*/radiation effects
Plant Extracts*/chemistry
Plant Extracts*/pharmacology
Fabaceae/*chemistry
Free Radical Scavengers/*pharmacology
Heme Oxygenase-1/*biosynthesis
Keratinocytes/*metabolism
NF-E2-Related Factor 2/*metabolism
Ultraviolet Rays/*adverse effects
p38 Mitogen-Activated Protein Kinases/*metabolism
Cell Line ; Free Radical Scavengers/chemistry ; Humans ; Mitogen-Activated Protein Kinase 1/metabolism ; Mitogen-Activated Protein Kinase 3/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
p38 Signalling Pathway.
Autorzy:
Sanz-Ezquerro JJ; Department of Molecular and Cellular Biology, Centro Nacional de Biotecnología/CSIC (CNB-CSIC), Campus-UAM, 28049 Madrid, Spain.
Cuenda A; Department of Immunology and Oncology, Centro Nacional de Biotecnología/CSIC (CNB-CSIC), Campus-UAM, 28049 Madrid, Spain.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Jan 20; Vol. 22 (3). Date of Electronic Publication: 2021 Jan 20.
Typ publikacji:
Editorial; Introductory Journal Article
MeSH Terms:
MAP Kinase Signaling System*
p38 Mitogen-Activated Protein Kinases/*metabolism
Animals ; Humans
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Icariin attenuates thioacetamide‑induced bone loss via the RANKL‑p38/ERK‑NFAT signaling pathway.
Autorzy:
Cheng L; School of Medical Technology and Information Engineering, Zhejiang Chinese Medical University, Hangzhou, Zhejiang 310053, P.R. China.
Jin X; School of Medical Technology and Information Engineering, Zhejiang Chinese Medical University, Hangzhou, Zhejiang 310053, P.R. China.
Shen H; School of Medical Technology and Information Engineering, Zhejiang Chinese Medical University, Hangzhou, Zhejiang 310053, P.R. China.
Chen X; School of Medical Technology and Information Engineering, Zhejiang Chinese Medical University, Hangzhou, Zhejiang 310053, P.R. China.
Chen J; School of Medical Technology and Information Engineering, Zhejiang Chinese Medical University, Hangzhou, Zhejiang 310053, P.R. China.
Xu B; Department of General Surgery, Sir Run Run Shaw Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang 310016, P.R. China.
Xu J; School of Medical Technology and Information Engineering, Zhejiang Chinese Medical University, Hangzhou, Zhejiang 310053, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2022 Apr; Vol. 25 (4). Date of Electronic Publication: 2022 Feb 16.
Typ publikacji:
Journal Article
MeSH Terms:
Bone Resorption/*drug therapy
Bone Resorption/*metabolism
Flavonoids/*pharmacology
Protective Agents/*pharmacology
RANK Ligand/*metabolism
Transcription Factors/*metabolism
p38 Mitogen-Activated Protein Kinases/*metabolism
Alkaline Phosphatase/blood ; Animals ; Body Weight/drug effects ; Bone Resorption/chemically induced ; Calcium/blood ; Cell Differentiation/drug effects ; Collagen Type I/blood ; Disease Models, Animal ; Femur/diagnostic imaging ; Femur/drug effects ; Femur/metabolism ; Flavonoids/therapeutic use ; MAP Kinase Signaling System/drug effects ; Magnesium/blood ; Male ; Osteoclasts/drug effects ; Peptides/blood ; Phosphorus/blood ; Protective Agents/therapeutic use ; Rats, Sprague-Dawley ; Thioacetamide/toxicity ; X-Ray Microtomography ; Rats
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
p38 MAPK Pathway in the Heart: New Insights in Health and Disease.
Autorzy:
Romero-Becerra R; Centro Nacional de Investigaciones Cardiovasculares (CNIC), 28029 Madrid, Spain.
Santamans AM; Centro Nacional de Investigaciones Cardiovasculares (CNIC), 28029 Madrid, Spain.
Folgueira C; Centro Nacional de Investigaciones Cardiovasculares (CNIC), 28029 Madrid, Spain.
Sabio G; Centro Nacional de Investigaciones Cardiovasculares (CNIC), 28029 Madrid, Spain.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Oct 08; Vol. 21 (19). Date of Electronic Publication: 2020 Oct 08.
Typ publikacji:
Journal Article; Review
MeSH Terms:
MAP Kinase Signaling System*/drug effects
Arrhythmias, Cardiac/*metabolism
Cardiomegaly/*metabolism
Heart Failure/*metabolism
Myocardium/*metabolism
Reperfusion Injury/*metabolism
p38 Mitogen-Activated Protein Kinases/*metabolism
Animals ; Humans ; Isoenzymes/antagonists & inhibitors ; Isoenzymes/metabolism ; Protein Kinase Inhibitors/pharmacology ; Protein Kinase Inhibitors/therapeutic use ; Regeneration/physiology ; Reperfusion Injury/drug therapy ; Treatment Outcome ; p38 Mitogen-Activated Protein Kinases/antagonists & inhibitors
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Caenorhabditis elegans mounts a p38 MAPK pathway-mediated defence to Cutibacterium acnes infection.
Autorzy:
Huang X; Department of Dermatology, Nanfang Hospital, Southern Medical University, Guangzhou, China.; Division of Infectious Diseases, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, Rhode Island, USA.
Pan W; Division of Infectious Diseases, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, Rhode Island, USA.
Kim W; College of Pharmacy, Graduate School of Pharmaceutical Sciences, Ewha Womans University, Seoul, South Korea.
White A; Division of Infectious Diseases, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, Rhode Island, USA.; Department of Molecular Pharmacology, Physiology and Biotechnology at Brown University, Providence, Rhode Island, USA.
Li S; Division of Infectious Diseases, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, Rhode Island, USA.
Li H; Department of Molecular and Medical Pharmacology, Crump Institute for Molecular Imaging, David Geffen School of Medicine, UCLA, Los Angeles, California, USA.; UCLA-DOE Institute for Genomics and Proteomics, Los Angeles, California, USA.
Lee K; Division of Infectious Diseases, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, Rhode Island, USA.
Fuchs BB; Division of Infectious Diseases, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, Rhode Island, USA.
Zeng K; Department of Dermatology, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Mylonakis E; Division of Infectious Diseases, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, Rhode Island, USA.
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Źródło:
Cellular microbiology [Cell Microbiol] 2020 Oct; Vol. 22 (10), pp. e13234. Date of Electronic Publication: 2020 Jul 15.
Typ publikacji:
Journal Article
MeSH Terms:
MAP Kinase Signaling System*
Caenorhabditis elegans/*metabolism
Caenorhabditis elegans/*microbiology
Caenorhabditis elegans Proteins/*metabolism
Lectins, C-Type/*metabolism
Propionibacteriaceae/*pathogenicity
p38 Mitogen-Activated Protein Kinases/*metabolism
Animals ; Caenorhabditis elegans/immunology ; Caenorhabditis elegans Proteins/genetics ; Down-Regulation ; Immunity, Innate ; Lectins, C-Type/genetics ; MAP Kinase Kinase 4/metabolism ; Mitogen-Activated Protein Kinases/metabolism ; Phosphorylation ; Receptors, G-Protein-Coupled/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Notch4 mediates vascular remodeling via ERK/JNK/P38 MAPK signaling pathways in hypoxic pulmonary hypertension.
Autorzy:
Guo M; Department of Pulmonary and Critical Care Medicine, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1095 Jiefang Avenue, Wuhan, 430030, China.; Key Laboratory of Respiratory Diseases, National Ministry of Health of the People's Republic of China and National Clinical Research Center for Respiratory Disease, Wuhan, China.
Zhang M; Department of Pulmonary and Critical Care Medicine, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1095 Jiefang Avenue, Wuhan, 430030, China.; Key Laboratory of Respiratory Diseases, National Ministry of Health of the People's Republic of China and National Clinical Research Center for Respiratory Disease, Wuhan, China.
Cao X; Department of Pediatrics, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Fang X; Department of Pulmonary and Critical Care Medicine, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1095 Jiefang Avenue, Wuhan, 430030, China.; Key Laboratory of Respiratory Diseases, National Ministry of Health of the People's Republic of China and National Clinical Research Center for Respiratory Disease, Wuhan, China.
Li K; Department of Pulmonary and Critical Care Medicine, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1095 Jiefang Avenue, Wuhan, 430030, China.; Key Laboratory of Respiratory Diseases, National Ministry of Health of the People's Republic of China and National Clinical Research Center for Respiratory Disease, Wuhan, China.
Qin L; Department of Pulmonary and Critical Care Medicine, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1095 Jiefang Avenue, Wuhan, 430030, China.; Key Laboratory of Respiratory Diseases, National Ministry of Health of the People's Republic of China and National Clinical Research Center for Respiratory Disease, Wuhan, China.
He Y; Department of Pulmonary and Critical Care Medicine, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1095 Jiefang Avenue, Wuhan, 430030, China.; Key Laboratory of Respiratory Diseases, National Ministry of Health of the People's Republic of China and National Clinical Research Center for Respiratory Disease, Wuhan, China.
Zhao J; Department of Pulmonary and Critical Care Medicine, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1095 Jiefang Avenue, Wuhan, 430030, China.; Key Laboratory of Respiratory Diseases, National Ministry of Health of the People's Republic of China and National Clinical Research Center for Respiratory Disease, Wuhan, China.
Xu Y; Department of Pulmonary and Critical Care Medicine, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1095 Jiefang Avenue, Wuhan, 430030, China.; Key Laboratory of Respiratory Diseases, National Ministry of Health of the People's Republic of China and National Clinical Research Center for Respiratory Disease, Wuhan, China.
Liu X; Department of Pulmonary and Critical Care Medicine, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1095 Jiefang Avenue, Wuhan, 430030, China.; Key Laboratory of Respiratory Diseases, National Ministry of Health of the People's Republic of China and National Clinical Research Center for Respiratory Disease, Wuhan, China.
Li X; Department of Pulmonary and Critical Care Medicine, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1095 Jiefang Avenue, Wuhan, 430030, China. .; Key Laboratory of Respiratory Diseases, National Ministry of Health of the People's Republic of China and National Clinical Research Center for Respiratory Disease, Wuhan, China. .
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Źródło:
Respiratory research [Respir Res] 2022 Jan 11; Vol. 23 (1), pp. 6. Date of Electronic Publication: 2022 Jan 11.
Typ publikacji:
Journal Article
MeSH Terms:
Gene Expression Regulation*
Hypertension, Pulmonary/*genetics
Hypoxia/*complications
Myocytes, Smooth Muscle/*metabolism
Receptor, Notch4/*genetics
Vascular Remodeling/*genetics
p38 Mitogen-Activated Protein Kinases/*genetics
Animals ; Apoptosis ; Cell Proliferation ; Cells, Cultured ; Disease Models, Animal ; Hypertension, Pulmonary/etiology ; Hypertension, Pulmonary/pathology ; Hypoxia/genetics ; Hypoxia/metabolism ; MAP Kinase Signaling System ; Male ; Myocytes, Smooth Muscle/pathology ; Pulmonary Artery/metabolism ; Pulmonary Artery/pathology ; Pulmonary Artery/physiopathology ; Rats ; Rats, Sprague-Dawley ; Receptor, Notch4/biosynthesis ; Signal Transduction ; Up-Regulation ; p38 Mitogen-Activated Protein Kinases/biosynthesis
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Effect of Acupuncture on the p38 Signaling Pathway in Several Nervous System Diseases: A Systematic Review.
Autorzy:
Wei TH; Department of Chinese Medicine, China Medical University Hospital, Taichung 40447, Taiwan.
Hsieh CL; Department of Chinese Medicine, China Medical University Hospital, Taichung 40447, Taiwan.; Chinese Medicine Research Center, China Medical University, Taichung 40402, Taiwan.; Graduate Institute of Acupuncture Science, College of Chinese Medicine, China Medical University, Taichung 40402, Taiwan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Jun 30; Vol. 21 (13). Date of Electronic Publication: 2020 Jun 30.
Typ publikacji:
Journal Article; Review; Systematic Review
MeSH Terms:
Acupuncture Therapy*
MAP Kinase Signaling System*
Nervous System Diseases/*therapy
p38 Mitogen-Activated Protein Kinases/*metabolism
Animals ; Brain-Derived Neurotrophic Factor/metabolism ; Humans ; Motion Sickness/metabolism ; Motion Sickness/therapy ; Nerve Regeneration ; Nervous System Diseases/metabolism ; TRPV Cation Channels/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Hydroxytyrosol Plays Antiatherosclerotic Effects through Regulating Lipid Metabolism via Inhibiting the p38 Signal Pathway.
Autorzy:
Zhang X; Department of Cardiology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China.
Qin Y; Department of Cardiology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China.
Wan X; Department of Cardiology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China.
Liu H; Department of Cardiology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China.
Iv C; Department of Cardiology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China.
Ruan W; Department of Cardiology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China.
Lu L; Department of Cardiology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China.
He L; Department of Cardiology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China.; Bio-X Institutes, Key Laboratory for the Genetics of Developmental and Neuropsychiatric Disorders (Ministry of Education), Shanghai Jiaotong University, Shanghai 200240, China.
Guo X; Department of Cardiology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China.
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Źródło:
BioMed research international [Biomed Res Int] 2020 Jun 22; Vol. 2020, pp. 5036572. Date of Electronic Publication: 2020 Jun 22 (Print Publication: 2020).
Typ publikacji:
Journal Article
MeSH Terms:
Atherosclerosis*/drug therapy
Atherosclerosis*/genetics
Atherosclerosis*/metabolism
Lipid Metabolism/*drug effects
MAP Kinase Signaling System/*drug effects
Phenylethyl Alcohol/*analogs & derivatives
p38 Mitogen-Activated Protein Kinases/*metabolism
Animals ; Lipid Metabolism/genetics ; MAP Kinase Signaling System/genetics ; Mice ; Mice, Knockout, ApoE ; Phenylethyl Alcohol/pharmacology ; p38 Mitogen-Activated Protein Kinases/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The MKK-Dependent Phosphorylation of p38α Is Augmented by Arginine Methylation on Arg49/Arg149 during Erythroid Differentiation.
Autorzy:
Liu MY; Institute of Biopharmaceutical Sciences, National Yang-Ming University, Taipei 112, Taiwan.
Hua WK; Institute of Biopharmaceutical Sciences, National Yang-Ming University, Taipei 112, Taiwan.
Chen CJ; Institute of Microbiology and Immunology, National Yang-Ming University, Taipei 112, Taiwan.
Lin WJ; Institute of Biopharmaceutical Sciences, National Yang-Ming University, Taipei 112, Taiwan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 May 17; Vol. 21 (10). Date of Electronic Publication: 2020 May 17.
Typ publikacji:
Journal Article
MeSH Terms:
Arginine/*metabolism
Erythropoiesis/*genetics
MAP Kinase Kinase 3/*metabolism
MAP Kinase Signaling System/*genetics
Protein-Arginine N-Methyltransferases/*metabolism
Repressor Proteins/*metabolism
p38 Mitogen-Activated Protein Kinases/*metabolism
Cell Line, Tumor ; Enzyme Activation/drug effects ; Erythropoiesis/drug effects ; Gene Knockdown Techniques ; Humans ; MAP Kinase Kinase 3/genetics ; MAP Kinase Signaling System/drug effects ; Mass Spectrometry ; Methylation ; Mutation ; Phosphorylation ; Protein Binding ; Protein Processing, Post-Translational ; Protein-Arginine N-Methyltransferases/genetics ; Recombinant Proteins/genetics ; Recombinant Proteins/isolation & purification ; Recombinant Proteins/metabolism ; Repressor Proteins/genetics ; p38 Mitogen-Activated Protein Kinases/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Toxoplasma gondii dense granule protein GRA24 drives MyD88-independent p38 MAPK activation, IL-12 production and induction of protective immunity.
Autorzy:
Mercer HL; Center for Evolutionary and Theoretical Immunology and Department of Biology, University of New Mexico, Albuquerque, New Mexico, United States of America.
Snyder LM; Center for Evolutionary and Theoretical Immunology and Department of Biology, University of New Mexico, Albuquerque, New Mexico, United States of America.
Doherty CM; Center for Evolutionary and Theoretical Immunology and Department of Biology, University of New Mexico, Albuquerque, New Mexico, United States of America.
Fox BA; Department of Microbiology and Immunology, Geisel School of Medicine at Dartmouth, Lebanon, New Hampshire, United States of America.
Bzik DJ; Department of Microbiology and Immunology, Geisel School of Medicine at Dartmouth, Lebanon, New Hampshire, United States of America.
Denkers EY; Center for Evolutionary and Theoretical Immunology and Department of Biology, University of New Mexico, Albuquerque, New Mexico, United States of America.
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Źródło:
PLoS pathogens [PLoS Pathog] 2020 May 15; Vol. 16 (5), pp. e1008572. Date of Electronic Publication: 2020 May 15 (Print Publication: 2020).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Interleukin-12/*immunology
MAP Kinase Signaling System/*immunology
Macrophages/*immunology
Myeloid Differentiation Factor 88/*immunology
Protozoan Proteins/*immunology
Toxoplasma/*immunology
Toxoplasmosis/*immunology
p38 Mitogen-Activated Protein Kinases/*immunology
Animals ; Enzyme Activation/genetics ; Enzyme Activation/immunology ; Interleukin-12/genetics ; MAP Kinase Signaling System/genetics ; Macrophages/parasitology ; Macrophages/pathology ; Mice ; Mice, Knockout ; Myeloid Differentiation Factor 88/genetics ; Protozoan Proteins/genetics ; Toxoplasma/genetics ; Toxoplasmosis/genetics ; Toxoplasmosis/pathology ; p38 Mitogen-Activated Protein Kinases/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CDDO-Me Inhibits Microglial Activation and Monocyte Infiltration by Abrogating NFκB- and p38 MAPK-Mediated Signaling Pathways Following Status Epilepticus.
Autorzy:
Kim JE; Department of Anatomy and Neurobiology, Institute of Epilepsy Research, College of Medicine, Hallym University, Chuncheon, Kangwon-Do 24252, Korea.
Park H; Department of Anatomy and Neurobiology, Institute of Epilepsy Research, College of Medicine, Hallym University, Chuncheon, Kangwon-Do 24252, Korea.
Lee JE; Department of Anatomy and Neurobiology, Institute of Epilepsy Research, College of Medicine, Hallym University, Chuncheon, Kangwon-Do 24252, Korea.
Kang TC; Department of Anatomy and Neurobiology, Institute of Epilepsy Research, College of Medicine, Hallym University, Chuncheon, Kangwon-Do 24252, Korea.
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Źródło:
Cells [Cells] 2020 May 01; Vol. 9 (5). Date of Electronic Publication: 2020 May 01.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
MAP Kinase Signaling System*
Microglia/*metabolism
Monocytes/*metabolism
NF-kappa B/*metabolism
Oleanolic Acid/*analogs & derivatives
Status Epilepticus/*pathology
p38 Mitogen-Activated Protein Kinases/*metabolism
Animals ; Chemokine CCL2/metabolism ; Frontal Lobe/pathology ; Male ; Microglia/drug effects ; Models, Biological ; Monocytes/drug effects ; NF-E2-Related Factor 2/metabolism ; Oleanolic Acid/pharmacology ; Parietal Lobe/pathology ; Peptides/pharmacology ; Phosphorylation/drug effects ; Phosphoserine/metabolism ; Rats, Sprague-Dawley ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The Fusion Protein rFlaA:Betv1 Modulates DC Responses by a p38-MAPK and COX2-Dependent Secretion of PGE 2 from Epithelial Cells.
Autorzy:
Lin YJ; Molecular Allergology, Paul-Ehrlich-Institut, 63225 Langen, Germany.
Flaczyk A; Molecular Allergology, Paul-Ehrlich-Institut, 63225 Langen, Germany.; Department of Anesthesia, Critical Care and Pain Medicine, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02114, USA.
Wolfheimer S; Molecular Allergology, Paul-Ehrlich-Institut, 63225 Langen, Germany.
Goretzki A; Molecular Allergology, Paul-Ehrlich-Institut, 63225 Langen, Germany.
Jamin A; Molecular Allergology, Paul-Ehrlich-Institut, 63225 Langen, Germany.
Wangorsch A; Molecular Allergology, Paul-Ehrlich-Institut, 63225 Langen, Germany.
Vieths S; Molecular Allergology, Paul-Ehrlich-Institut, 63225 Langen, Germany.
Scheurer S; Molecular Allergology, Paul-Ehrlich-Institut, 63225 Langen, Germany.
Schülke S; Molecular Allergology, Paul-Ehrlich-Institut, 63225 Langen, Germany.
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Źródło:
Cells [Cells] 2021 Dec 04; Vol. 10 (12). Date of Electronic Publication: 2021 Dec 04.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cyclooxygenase 2/*metabolism
Dendritic Cells/*metabolism
Dinoprostone/*metabolism
Epithelial Cells/*metabolism
Recombinant Fusion Proteins/*pharmacology
p38 Mitogen-Activated Protein Kinases/*metabolism
Animals ; Cell Line ; Chemokine CCL2/metabolism ; Chemokine CCL20/metabolism ; Chemokines/metabolism ; Dendritic Cells/drug effects ; Epithelial Cells/drug effects ; Interleukin-6/metabolism ; MAP Kinase Signaling System/drug effects ; Mice, Inbred BALB C ; Models, Biological ; NF-kappa B/metabolism ; Phosphorylation/drug effects ; Solubility ; Toll-Like Receptor 5/metabolism ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
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