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Wyszukujesz frazę ""MESH: Homozygote"" wg kryterium: Temat


Tytuł :
A recessive form of Hyper IgE Syndrome by disruption of ZNF341-dependent STAT3 transcription and activity
Autorzy :
Beziat, Vivien
Li, Juan
Lin, Jian-Xin
Ma, Cindy
Li, Peng
Bousfiha, Aziz
Pellier, Isabelle
Zoghi, Samaneh
Baris, Safa
Keles, Sevgi
Gray, Paul
Du, Ning
Wang, Yi
Zerbib, Yoann
Levy, Romain
Leclercq, Thibaut
About, Frédégonde
Lim, Ai Ing
Rao, Geetha
Payne, Kathryn
Pelham, Simon
Avery, Danielle
Deenick, Elissa
Pillay, Bethany
Chou, Janet
Guery, Romain
Belkadi, Aziz
Guerin, Antoine
Migaud, Melanie
Rattina, Vimel
Ailal, Fatima
Benhsaien, Ibtihal
Bouaziz, Matthieu
Habib, Tanwir
Chaussabel, Damien
Marr, Nico
El-Benna, Jamel
Grimbacher, Bodo
Wargon, Orli
Bustamante, Jacinta
Boisson, Bertrand
Müller-Fleckenstein, Ingrid
Fleckenstein, Bernhard
Chandesris, Marie-Olivia
Titeux, Matthias
Fraitag, Sylvie
Alyanakian, Marie-Alexandra
Leruez-Ville, Marianne
Picard, Capucine
Meyts, Isabelle
Di Santo, James
Hovnanian, Alain
Somer, Ayper
Ozen, Ahmet
Rezaei, Nima
Chatila, Talal
Abel, Laurent
Leonard, Warren
Tangye, Stuart
Puel, Anne
Casanova, Jean-Laurent
Pokaż więcej
Temat :
MESH: Cell Differentiation/immunology
MESH: Th17 Cells/metabolism
MESH: Male
MESH: Gene Expression Regulation/immunology
MESH: RNA, Messenger/metabolism
MESH: Transcription Factors/immunology
[SDV.IMM]Life Sciences [q-bio]/Immunology
MESH: Transcription Factors/metabolism
MESH: Homozygote
MESH: Th2 Cells/metabolism
MESH: Lymphocyte Count
MESH: STAT3 Transcription Factor/immunology
MESH: Transcription Factors/genetics
MESH: Transcription, Genetic/immunology
MESH: Cytokines/metabolism
MESH: Young Adult
MESH: Th17 Cells/immunology
MESH: Cell Nucleus/metabolism
MESH: Female
MESH: Adult
MESH: Cell Differentiation/genetics
MESH: Genes, Recessive/immunology
MESH: Whole Exome Sequencing
MESH: STAT3 Transcription Factor/metabolism
MESH: Adolescent
MESH: Pedigree
MESH: Job Syndrome/blood
MESH: Th2 Cells/immunology
MESH: Consanguinity
MESH: Exons/genetics
MESH: Humans
MESH: Immunoglobulin E/blood
MESH: STAT3 Transcription Factor/genetics
MESH: Cytokines/immunology
Article
MESH: Loss of Function Mutation
MESH: Middle Aged
MESH: Genes, Recessive/genetics
MESH: Promoter Regions, Genetic/genetics
MESH: Zinc Fingers/genetics
MESH: Job Syndrome/immunology
MESH: Immunoglobulin E/immunology
MESH: Job Syndrome/genetics
Źródło :
Science Immunology, American Association for the Advancement of Science, 2018, 3 (24), pp.eaat4956. ⟨10.1126/sciimmunol.aat4956⟩
Tytuł :
A recessive form of hyper-IgE syndrome by disruption of ZNF341-dependent STAT3 transcription and activity
Autorzy :
Beziat, Vivien
Li, Juan
Lin, Jian-Xin
Ma, Cindy
Li, Peng
Bousfiha, Aziz
Pellier, Isabelle
Zoghi, Samaneh
Baris, Safa
Keles, Sevgi
Gray, Paul
Du, Ning
Wang, Yi
Zerbib, Yoann
Levy, Romain
Leclercq, Thibaut
About, Frédégonde
Lim, Ai Ing
Rao, Geetha
Payne, Kathryn
Pelham, Simon
Avery, Danielle
Deenick, Elissa
Pillay, Bethany
Chou, Janet
Guery, Romain
Belkadi, Aziz
Guerin, Antoine
Migaud, Melanie
Rattina, Vimel
Ailal, Fatima
Benhsaien, Ibtihal
Bouaziz, Matthieu
Habib, Tanwir
Chaussabel, Damien
Marr, Nico
El-Benna, Jamel
Grimbacher, Bodo
Wargon, Orli
Bustamante, Jacinta
Boisson, Bertrand
Müller-Fleckenstein, Ingrid
Fleckenstein, Bernhard
Chandesris, Marie-Olivia
Titeux, Matthias
Fraitag, Sylvie
Alyanakian, Marie-Alexandra
Leruez-Ville, Marianne
Picard, Capucine
Meyts, Isabelle
Di Santo, James
Hovnanian, Alain
Somer, Ayper
Ozen, Ahmet
Rezaei, Nima
Chatila, Talal
Abel, Laurent
Leonard, Warren
Tangye, Stuart
Puel, Anne
Casanova, Jean-Laurent
Pokaż więcej
Temat :
MESH: Adolescent
MESH: Adult
MESH: Job Syndrome/genetics
MESH: Job Syndrome/immunology
MESH: Cell Differentiation/genetics
MESH: Loss of Function Mutation
MESH: Lymphocyte Count
MESH: Male
MESH: Middle Aged
MESH: Pedigree
MESH: Promoter Regions, Genetic/genetics
MESH: RNA, Messenger/metabolism
MESH: Gene Expression Regulation/immunology
MESH: STAT3 Transcription Factor/genetics
MESH: STAT3 Transcription Factor/immunology
MESH: STAT3 Transcription Factor/metabolism
MESH: Cell Differentiation/immunology
MESH: Th17 Cells/immunology
MESH: Th17 Cells/metabolism
MESH: Th2 Cells/immunology
MESH: Th2 Cells/metabolism
MESH: Transcription Factors/genetics
MESH: Transcription Factors/immunology
MESH: Genes, Recessive/genetics
MESH: Transcription Factors/metabolism
MESH: Transcription, Genetic/immunology
MESH: Whole Exome Sequencing
MESH: Young Adult
MESH: Cell Nucleus/metabolism
MESH: Zinc Fingers/genetics
MESH: Consanguinity
MESH: Cytokines/immunology
MESH: Cytokines/metabolism
MESH: Exons/genetics
MESH: Genes, Recessive/immunology
MESH: Female
MESH: Homozygote
MESH: Humans
MESH: Immunoglobulin E/blood
MESH: Immunoglobulin E/immunology
MESH: Job Syndrome/blood
[SDV.IMM]Life Sciences [q-bio]/Immunology
Źródło :
Science Immunology, American Association for the Advancement of Science, 2018, 3 (24), pp.eaat4956. ⟨10.1126/sciimmunol.aat4956⟩
Tytuł :
A recessive form of hyper-IgE syndrome by disruption of ZNF341-dependent STAT3 transcription and activity
Autorzy :
Beziat, Vivien
Li, Juan
Lin, Jian-Xin
Ma, Cindy
Li, Peng
Bousfiha, Aziz
Pellier, Isabelle
Zoghi, Samaneh
Baris, Safa
Keles, Sevgi
Gray, Paul
Du, Ning
Wang, Yi
Zerbib, Yoann
Levy, Romain
Leclercq, Thibaut
About, Frédégonde
Lim, Ai Ing
Rao, Geetha
Payne, Kathryn
Pelham, Simon
Avery, Danielle
Deenick, Elissa
Pillay, Bethany
Chou, Janet
Guery, Romain
Belkadi, Aziz
Guerin, Antoine
Migaud, Melanie
Rattina, Vimel
Ailal, Fatima
Benhsaien, Ibtihal
Bouaziz, Matthieu
Habib, Tanwir
Chaussabel, Damien
Marr, Nico
El-Benna, Jamel
Grimbacher, Bodo
Wargon, Orli
Bustamante, Jacinta
Boisson, Bertrand
Müller-Fleckenstein, Ingrid
Fleckenstein, Bernhard
Chandesris, Marie-Olivia
Titeux, Matthias
Fraitag, Sylvie
Alyanakian, Marie-Alexandra
Leruez-Ville, Marianne
Picard, Capucine
Meyts, Isabelle
Di Santo, James
Hovnanian, Alain
Somer, Ayper
Ozen, Ahmet
Rezaei, Nima
Chatila, Talal
Abel, Laurent
Leonard, Warren
Tangye, Stuart
Puel, Anne
Casanova, Jean-Laurent
Pokaż więcej
Temat :
MESH: Adolescent
MESH: Adult
MESH: Job Syndrome/genetics
MESH: Job Syndrome/immunology
MESH: Cell Differentiation/genetics
MESH: Loss of Function Mutation
MESH: Lymphocyte Count
MESH: Male
MESH: Middle Aged
MESH: Pedigree
MESH: Promoter Regions, Genetic/genetics
MESH: RNA, Messenger/metabolism
MESH: Gene Expression Regulation/immunology
MESH: STAT3 Transcription Factor/genetics
MESH: STAT3 Transcription Factor/immunology
MESH: STAT3 Transcription Factor/metabolism
MESH: Cell Differentiation/immunology
MESH: Th17 Cells/immunology
MESH: Th17 Cells/metabolism
MESH: Th2 Cells/immunology
MESH: Th2 Cells/metabolism
MESH: Transcription Factors/genetics
MESH: Transcription Factors/immunology
MESH: Genes, Recessive/genetics
MESH: Transcription Factors/metabolism
MESH: Transcription, Genetic/immunology
MESH: Whole Exome Sequencing
MESH: Young Adult
MESH: Cell Nucleus/metabolism
MESH: Zinc Fingers/genetics
MESH: Consanguinity
MESH: Cytokines/immunology
MESH: Cytokines/metabolism
MESH: Exons/genetics
MESH: Genes, Recessive/immunology
MESH: Female
MESH: Homozygote
MESH: Humans
MESH: Immunoglobulin E/blood
MESH: Immunoglobulin E/immunology
MESH: Job Syndrome/blood
[SDV.IMM]Life Sciences [q-bio]/Immunology
Źródło :
Science Immunology, American Association for the Advancement of Science, 2018, 3 (24), pp.eaat4956. ⟨10.1126/sciimmunol.aat4956⟩
Tytuł :
A recessive form of hyper-IgE syndrome by disruption of ZNF341-dependent STAT3 transcription and activity
Autorzy :
Beziat, Vivien
Li, Juan
Lin, Jian-Xin
Ma, Cindy
Li, Peng
Bousfiha, Aziz
Pellier, Isabelle
Zoghi, Samaneh
Baris, Safa
Keles, Sevgi
Gray, Paul
Du, Ning
Wang, Yi
Zerbib, Yoann
Levy, Romain
Leclercq, Thibaut
About, Frédégonde
Lim, Ai Ing
Rao, Geetha
Payne, Kathryn
Pelham, Simon
Avery, Danielle
Deenick, Elissa
Pillay, Bethany
Chou, Janet
Guery, Romain
Belkadi, Aziz
Guerin, Antoine
Migaud, Melanie
Rattina, Vimel
Ailal, Fatima
Benhsaien, Ibtihal
Bouaziz, Matthieu
Habib, Tanwir
Chaussabel, Damien
Marr, Nico
El-Benna, Jamel
Grimbacher, Bodo
Wargon, Orli
Bustamante, Jacinta
Boisson, Bertrand
Müller-Fleckenstein, Ingrid
Fleckenstein, Bernhard
Chandesris, Marie-Olivia
Titeux, Matthias
Fraitag, Sylvie
Alyanakian, Marie-Alexandra
Leruez-Ville, Marianne
Picard, Capucine
Meyts, Isabelle
Di Santo, James
Hovnanian, Alain
Somer, Ayper
Ozen, Ahmet
Rezaei, Nima
Chatila, Talal
Abel, Laurent
Leonard, Warren
Tangye, Stuart
Puel, Anne
Casanova, Jean-Laurent
Pokaż więcej
Temat :
MESH: Adolescent
MESH: Adult
MESH: Job Syndrome/genetics
MESH: Job Syndrome/immunology
MESH: Cell Differentiation/genetics
MESH: Loss of Function Mutation
MESH: Lymphocyte Count
MESH: Male
MESH: Middle Aged
MESH: Pedigree
MESH: Promoter Regions, Genetic/genetics
MESH: RNA, Messenger/metabolism
MESH: Gene Expression Regulation/immunology
MESH: STAT3 Transcription Factor/genetics
MESH: STAT3 Transcription Factor/immunology
MESH: STAT3 Transcription Factor/metabolism
MESH: Cell Differentiation/immunology
MESH: Th17 Cells/immunology
MESH: Th17 Cells/metabolism
MESH: Th2 Cells/immunology
MESH: Th2 Cells/metabolism
MESH: Transcription Factors/genetics
MESH: Transcription Factors/immunology
MESH: Genes, Recessive/genetics
MESH: Transcription Factors/metabolism
MESH: Transcription, Genetic/immunology
MESH: Whole Exome Sequencing
MESH: Young Adult
MESH: Cell Nucleus/metabolism
MESH: Zinc Fingers/genetics
MESH: Consanguinity
MESH: Cytokines/immunology
MESH: Cytokines/metabolism
MESH: Exons/genetics
MESH: Genes, Recessive/immunology
MESH: Female
MESH: Homozygote
MESH: Humans
MESH: Immunoglobulin E/blood
MESH: Immunoglobulin E/immunology
MESH: Job Syndrome/blood
[SDV.IMM]Life Sciences [q-bio]/Immunology
Źródło :
Science Immunology, American Association for the Advancement of Science, 2018, 3 (24), pp.eaat4956. ⟨10.1126/sciimmunol.aat4956⟩
Tytuł :
A recessive form of hyper-IgE syndrome by disruption of ZNF341-dependent STAT3 transcription and activity
Autorzy :
Beziat, Vivien
Li, Juan
Lin, Jian-Xin
Ma, Cindy
Li, Peng
Bousfiha, Aziz
Pellier, Isabelle
Zoghi, Samaneh
Baris, Safa
Keles, Sevgi
Gray, Paul
Du, Ning
Wang, Yi
Zerbib, Yoann
Levy, Romain
Leclercq, Thibaut
About, Frédégonde
Lim, Ai Ing
Rao, Geetha
Payne, Kathryn
Pelham, Simon
Avery, Danielle
Deenick, Elissa
Pillay, Bethany
Chou, Janet
Guery, Romain
Belkadi, Aziz
Guerin, Antoine
Migaud, Melanie
Rattina, Vimel
Ailal, Fatima
Benhsaien, Ibtihal
Bouaziz, Matthieu
Habib, Tanwir
Chaussabel, Damien
Marr, Nico
El-Benna, Jamel
Grimbacher, Bodo
Wargon, Orli
Bustamante, Jacinta
Boisson, Bertrand
Müller-Fleckenstein, Ingrid
Fleckenstein, Bernhard
Chandesris, Marie-Olivia
Titeux, Matthias
Fraitag, Sylvie
Alyanakian, Marie-Alexandra
Leruez-Ville, Marianne
Picard, Capucine
Meyts, Isabelle
Di Santo, James
Hovnanian, Alain
Somer, Ayper
Ozen, Ahmet
Rezaei, Nima
Chatila, Talal
Abel, Laurent
Leonard, Warren
Tangye, Stuart
Puel, Anne
Casanova, Jean-Laurent
Pokaż więcej
Temat :
MESH: Adolescent
MESH: Adult
MESH: Job Syndrome/genetics
MESH: Job Syndrome/immunology
MESH: Cell Differentiation/genetics
MESH: Loss of Function Mutation
MESH: Lymphocyte Count
MESH: Male
MESH: Middle Aged
MESH: Pedigree
MESH: Promoter Regions, Genetic/genetics
MESH: RNA, Messenger/metabolism
MESH: Gene Expression Regulation/immunology
MESH: STAT3 Transcription Factor/genetics
MESH: STAT3 Transcription Factor/immunology
MESH: STAT3 Transcription Factor/metabolism
MESH: Cell Differentiation/immunology
MESH: Th17 Cells/immunology
MESH: Th17 Cells/metabolism
MESH: Th2 Cells/immunology
MESH: Th2 Cells/metabolism
MESH: Transcription Factors/genetics
MESH: Transcription Factors/immunology
MESH: Genes, Recessive/genetics
MESH: Transcription Factors/metabolism
MESH: Transcription, Genetic/immunology
MESH: Whole Exome Sequencing
MESH: Young Adult
MESH: Cell Nucleus/metabolism
MESH: Zinc Fingers/genetics
MESH: Consanguinity
MESH: Cytokines/immunology
MESH: Cytokines/metabolism
MESH: Exons/genetics
MESH: Genes, Recessive/immunology
MESH: Female
MESH: Homozygote
MESH: Humans
MESH: Immunoglobulin E/blood
MESH: Immunoglobulin E/immunology
MESH: Job Syndrome/blood
[SDV.IMM]Life Sciences [q-bio]/Immunology
Źródło :
Science Immunology, American Association for the Advancement of Science, 2018, 3 (24), pp.eaat4956. ⟨10.1126/sciimmunol.aat4956⟩
Tytuł :
Mutations in DNAH1, which encodes an inner arm heavy chain dynein, lead to male infertility from multiple morphological abnormalities of the sperm flagella.
Autorzy :
Ben Khelifa, Mariem
Coutton, Charles
Zouari, Raoudha
Karaouzène, Thomas
Rendu, John
Bidart, Marie
Yassine, Sandra
Pierre, Virginie
Delaroche, Julie
Hennebicq, Sylviane
Grunwald, Didier
Escalier, Denise
Pernet-Gallay, Karine
Jouk, Pierre-Simon
Thierry-Mieg, Nicolas
Touré, Aminata
Arnoult, Christophe
Ray, Pierre F.
Pokaż więcej
Temat :
MESH: RNA Splice Sites
MESH: Male
MESH: RNA, Messenger
MESH: Homozygote
MESH: Genetic Variation
[SDV.GEN]Life Sciences [q-bio]/Genetics
MESH : Infertility, Male
[ SDV.GEN ] Life Sciences [q-bio]/Genetics
MESH : Male
MESH: Cilia
MESH : RNA, Messenger
Genetics
MESH : Axonemal Dyneins
MESH: Infertility, Male
MESH: Flagella
MESH : Genetic Variation
MESH: Mutation
MESH: Axoneme
MESH : Sperm Tail
Genetics(clinical)
MESH: Sperm Tail
MESH: Testis
MESH: Kartagener Syndrome
MESH : Sequence Analysis, DNA
MESH : Kartagener Syndrome
MESH: Humans
MESH : Axoneme
MESH : Cilia
MESH: Sperm Motility
MESH : Flagella
MESH : Mutation
MESH : Testis
MESH: Axonemal Dyneins
Report
MESH : Humans
MESH : RNA Splice Sites
[SDV]Life Sciences [q-bio]
MESH : Homozygote
MESH: Sequence Analysis, DNA
MESH : Sperm Motility
Źródło :
American Journal of Human Genetics
American Journal of Human Genetics, Elsevier (Cell Press), 2014, 94 (1), pp.95-104. ⟨10.1016/j.ajhg.2013.11.017⟩
American Journal of Human Genetics, Elsevier (Cell Press), 2014, 94 (1), pp.95-104. 〈10.1016/j.ajhg.2013.11.017〉
Tytuł :
Suppression of Hepcidin Expression and Iron Overload Mediate Salmonella Susceptibility in Ankyrin 1 ENU-Induced Mutant
Autorzy :
Yuki, Kyoko E
Eva, Megan M
Richer, Etienne
Chung, Dudley
Paquet, Marilène
Cellier, Mathieu
Canonne-Hergaux, François
Vaulont, Sophie
Vidal, Silvia M
Malo, Danielle
Pokaż więcej
Temat :
MESH: Salmonella typhimurium
Infectious Diseases
Genetic Mutation
MESH : Animals
Mutagenesis
MESH : Antimicrobial Cationic Peptides
MESH : Genetic Predisposition to Disease
MESH : Codon, Nonsense
[SDV.MHEP.HEM]Life Sciences [q-bio]/Human health and pathology/Hematology
MESH: Erythropoietin
MESH : Heme Oxygenase-1
MESH : Salmonella typhimurium
Immunology
MESH : Anemia, Hemolytic
MESH: Codon, Nonsense
MESH : Ankyrins
[ SDV ] Life Sciences [q-bio]
Biology
MESH: Genetic Predisposition to Disease
MESH : Hepcidins
MESH: Heterozygote
[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology
Bacterial Diseases
MESH: Mice
Science
[SDV]Life Sciences [q-bio]
MESH : Homozygote
Genetics of the Immune System
MESH: Animals
Research Article
Immunity to Infections
MESH : Iron
MESH : Survival Analysis
MESH: Homozygote
MESH: Antimicrobial Cationic Peptides
Immunity
MESH : Gene Expression
[ SDV.MP.BAC ] Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology
MESH: Membrane Proteins
MESH: Ethylnitrosourea
MESH: Iron Overload
Genetics
Salmonella
MESH : Membrane Proteins
Immune Response
MESH: Ankyrins
MESH : Ethylnitrosourea
MESH : Growth Differentiation Factor 15
MESH: Growth Differentiation Factor 15
MESH : Liver
MESH: Survival Analysis
MESH : Erythrocytes
MESH : Salmonella Infections
MESH : Erythropoietin
MESH: Hepcidins
Microbiology
MESH: Liver
Medicine
Animal Models of Infection
MESH: Erythrocytes
[ SDV.MHEP.HEM ] Life Sciences [q-bio]/Human health and pathology/Hematology
MESH: Gene Expression
MESH: Anemia, Hemolytic
MESH: Salmonella Infections
MESH : Iron Overload
Virology
Clinical Immunology
MESH: Iron
MESH: Heme Oxygenase-1
MESH : Heterozygote
MESH : Mice
Źródło :
PLoS ONE, Vol 8, Iss 2, p e55331 (2013)
PLoS ONE, Public Library of Science, 2013, 8 (2), pp.e55331. ⟨10.1371/journal.pone.0055331⟩
PLoS ONE, Public Library of Science, 2013, 8 (2), pp.e55331. 〈10.1371/journal.pone.0055331〉
Tytuł :
Hypomorphic homozygous mutations in phosphoglucomutase 3 (PGM3) impair immunity and increase serum IgE levels.
Autorzy :
Sassi, Atfa
Lazaroski, Sandra
Wu, Gang
Haslam, Stuart M
Fliegauf, Manfred
Mellouli, Fethi
Patiroglu, Turkan
Unal, Ekrem
Ozdemir, Mehmet Akif
Jouhadi, Zineb
Khadir, Khadija
Ben-Khemis, Leila
Ben-Ali, Meriem
Ben-Mustapha, Imen
Borchani, Lamia
Pfeifer, Dietmar
Jakob, Thilo
Khemiri, Monia
Asplund, A Charlotta
Gustafsson, Manuela O
Lundin, Karin E
Falk-Sörqvist, Elin
Moens, Lotte N
Gungor, Hatice Eke
Engelhardt, Karin R
Dziadzio, Magdalena
Stauss, Hans
Fleckenstein, Bernhard
Meier, Rebecca
Prayitno, Khairunnadiya
Maul-Pavicic, Andrea
Schaffer, Sandra
Rakhmanov, Mirzokhid
Henneke, Philipp
Kraus, Helene
Eibel, Hermann
Kölsch, Uwe
Nadifi, Sellama
Nilsson, Mats
Bejaoui, Mohamed
Schäffer, Alejandro A
Smith, C I Edvard
Dell, Anne
Barbouche, Mohamed-Ridha
Grimbacher, Bodo
Pokaż więcej
Temat :
MESH: T-Lymphocytes
MESH: Male
MESH: Infant
[SDV.IMM]Life Sciences [q-bio]/Immunology
MESH: Homozygote
MESH: Humans
MESH: Genetic Diseases, Inborn
MESH: Tunisia
MESH: Chromosomes, Human, Pair 6
MESH: Mutation, Missense
MESH: Immunity
MESH: Cell Proliferation
MESH: Amino Acid Substitution
MESH: Child
MESH: Job Syndrome
MESH: Phosphoglucomutase
MESH: Female
[SDV]Life Sciences [q-bio]
MESH: Adult
MESH: Glycosylation
MESH: Genetic Linkage
MESH: Immunoglobulin E
Źródło :
Journal of Allergy and Clinical Immunology, Elsevier, 2014, 133 (5), pp.1410-9, 1419.e1-13. ⟨10.1016/j.jaci.2014.02.025⟩
Journal of Allergy and Clinical Immunology

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