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Wyszukujesz frazę ""MESH: Transcription Factors/metabolism"" wg kryterium: Temat


Tytuł :
Necroptosis microenvironment directs lineage commitment in liver cancer
Autorzy :
Seehawer, Marco
Heinzmann, Florian
D’Artista, Luana
Harbig, Jule
Roux, Pierre-François
Hoenicke, Lisa
Dang, Hien
Klotz, Sabrina
Robinson, Lucas
Doré, Grégory
Rozenblum, Nir
Kang, Tae-Won
Chawla, Rishabh
Buch, Thorsten
Vucur, Mihael
Roth, Mareike
Zuber, Johannes
Luedde, Tom
Sipos, Bence
Longerich, Thomas
Heikenwalder, Mathias
Wang, Xin Wei
Bischof, Oliver
Zender, Lars
Pokaż więcej
Temat :
Cancer epigenetics
Cancer microenvironment
MESH: Animals
MESH: Apoptosis*/genetics
MESH: Cytokines/metabolism
MESH: DNA Transposable Elements/genetics
MESH: DNA-Binding Proteins/genetics
MESH: DNA-Binding Proteins/metabolism
MESH: Disease Models, Animal
MESH: Epigenesis, Genetic/genetics
MESH: Female
MESH: Gene Expression Profiling
MESH: Genes, myc
MESH: Genes, ras
MESH: Carcinogenesis/genetics
MESH: Hepatocytes/metabolism
MESH: Hepatocytes/pathology
MESH: Humans
MESH: Liver Neoplasms/genetics
MESH: Liver Neoplasms/pathology
MESH: Male
MESH: Mice
MESH: Mosaicism
MESH: Necrosis*/genetics
MESH: Proto-Oncogene Proteins c-akt/genetics
MESH: Carcinoma, Hepatocellular/genetics
MESH: T-Box Domain Proteins/genetics
MESH: T-Box Domain Proteins/metabolism
MESH: Transcription Factors/genetics
MESH: Transcription Factors/metabolism
MESH: Tumor Microenvironment
MESH: Carcinoma, Hepatocellular/pathology
MESH: Cell Differentiation
MESH: Cell Lineage*/genetics
MESH: Cholangiocarcinoma/genetics
MESH: Cholangiocarcinoma/pathology
MESH: Cyclin-Dependent Kinase Inhibitor p16/deficiency
[SDV.CAN]Life Sciences [q-bio]/Cancer
Źródło :
Nature, Nature Publishing Group, 2018, 562 (7725), pp.69-75. ⟨10.1038/s41586-018-0519-y⟩
Tytuł :
Necroptosis microenvironment directs lineage commitment in liver cancer
Autorzy :
Seehawer, Marco
Heinzmann, Florian
D’Artista, Luana
Harbig, Jule
Roux, Pierre-François
Hoenicke, Lisa
Dang, Hien
Klotz, Sabrina
Robinson, Lucas
Doré, Grégory
Rozenblum, Nir
Kang, Tae-Won
Chawla, Rishabh
Buch, Thorsten
Vucur, Mihael
Roth, Mareike
Zuber, Johannes
Luedde, Tom
Sipos, Bence
Longerich, Thomas
Heikenwalder, Mathias
Wang, Xin Wei
Bischof, Oliver
Zender, Lars
Pokaż więcej
Temat :
Cancer epigenetics
Cancer microenvironment
MESH: Animals
MESH: Apoptosis*/genetics
MESH: Cytokines/metabolism
MESH: DNA Transposable Elements/genetics
MESH: DNA-Binding Proteins/genetics
MESH: DNA-Binding Proteins/metabolism
MESH: Disease Models, Animal
MESH: Epigenesis, Genetic/genetics
MESH: Female
MESH: Gene Expression Profiling
MESH: Genes, myc
MESH: Genes, ras
MESH: Carcinogenesis/genetics
MESH: Hepatocytes/metabolism
MESH: Hepatocytes/pathology
MESH: Humans
MESH: Liver Neoplasms/genetics
MESH: Liver Neoplasms/pathology
MESH: Male
MESH: Mice
MESH: Mosaicism
MESH: Necrosis*/genetics
MESH: Proto-Oncogene Proteins c-akt/genetics
MESH: Carcinoma, Hepatocellular/genetics
MESH: T-Box Domain Proteins/genetics
MESH: T-Box Domain Proteins/metabolism
MESH: Transcription Factors/genetics
MESH: Transcription Factors/metabolism
MESH: Tumor Microenvironment
MESH: Carcinoma, Hepatocellular/pathology
MESH: Cell Differentiation
MESH: Cell Lineage*/genetics
MESH: Cholangiocarcinoma/genetics
MESH: Cholangiocarcinoma/pathology
MESH: Cyclin-Dependent Kinase Inhibitor p16/deficiency
[SDV.CAN]Life Sciences [q-bio]/Cancer
Źródło :
Nature, Nature Publishing Group, 2018, 562 (7725), pp.69-75. ⟨10.1038/s41586-018-0519-y⟩
Tytuł :
Necroptosis microenvironment directs lineage commitment in liver cancer
Autorzy :
Seehawer, Marco
Heinzmann, Florian
D’Artista, Luana
Harbig, Jule
Roux, Pierre-François
Hoenicke, Lisa
Dang, Hien
Klotz, Sabrina
Robinson, Lucas
Doré, Grégory
Rozenblum, Nir
Kang, Tae-Won
Chawla, Rishabh
Buch, Thorsten
Vucur, Mihael
Roth, Mareike
Zuber, Johannes
Luedde, Tom
Sipos, Bence
Longerich, Thomas
Heikenwalder, Mathias
Wang, Xin Wei
Bischof, Oliver
Zender, Lars
Pokaż więcej
Temat :
Cancer epigenetics
Cancer microenvironment
MESH: Animals
MESH: Apoptosis*/genetics
MESH: Cytokines/metabolism
MESH: DNA Transposable Elements/genetics
MESH: DNA-Binding Proteins/genetics
MESH: DNA-Binding Proteins/metabolism
MESH: Disease Models, Animal
MESH: Epigenesis, Genetic/genetics
MESH: Female
MESH: Gene Expression Profiling
MESH: Genes, myc
MESH: Genes, ras
MESH: Carcinogenesis/genetics
MESH: Hepatocytes/metabolism
MESH: Hepatocytes/pathology
MESH: Humans
MESH: Liver Neoplasms/genetics
MESH: Liver Neoplasms/pathology
MESH: Male
MESH: Mice
MESH: Mosaicism
MESH: Necrosis*/genetics
MESH: Proto-Oncogene Proteins c-akt/genetics
MESH: Carcinoma, Hepatocellular/genetics
MESH: T-Box Domain Proteins/genetics
MESH: T-Box Domain Proteins/metabolism
MESH: Transcription Factors/genetics
MESH: Transcription Factors/metabolism
MESH: Tumor Microenvironment
MESH: Carcinoma, Hepatocellular/pathology
MESH: Cell Differentiation
MESH: Cell Lineage*/genetics
MESH: Cholangiocarcinoma/genetics
MESH: Cholangiocarcinoma/pathology
MESH: Cyclin-Dependent Kinase Inhibitor p16/deficiency
[SDV.CAN]Life Sciences [q-bio]/Cancer
Źródło :
Nature, Nature Publishing Group, 2018, 562 (7725), pp.69-75. ⟨10.1038/s41586-018-0519-y⟩
Tytuł :
Necroptosis microenvironment directs lineage commitment in liver cancer
Autorzy :
Seehawer, Marco
Heinzmann, Florian
D’Artista, Luana
Harbig, Jule
Roux, Pierre-François
Hoenicke, Lisa
Dang, Hien
Klotz, Sabrina
Robinson, Lucas
Doré, Grégory
Rozenblum, Nir
Kang, Tae-Won
Chawla, Rishabh
Buch, Thorsten
Vucur, Mihael
Roth, Mareike
Zuber, Johannes
Luedde, Tom
Sipos, Bence
Longerich, Thomas
Heikenwalder, Mathias
Wang, Xin Wei
Bischof, Oliver
Zender, Lars
Pokaż więcej
Temat :
MESH: Animals
MESH: Male
MESH: Disease Models, Animal
610 Medicine & health
MESH: Epigenesis, Genetic/genetics
MESH: Hepatocytes/pathology
MESH: Transcription Factors/metabolism
MESH: Cholangiocarcinoma/pathology
Cancer epigenetics
MESH: DNA-Binding Proteins/metabolism
MESH: Transcription Factors/genetics
MESH: Carcinoma, Hepatocellular/genetics
MESH: Cytokines/metabolism
[SDV.CAN]Life Sciences [q-bio]/Cancer
Institute of Laboratory Animal Science
MESH: Female
Cancer microenvironment
MESH: Liver Neoplasms/pathology
MESH: T-Box Domain Proteins/metabolism
MESH: DNA-Binding Proteins/genetics
MESH: Gene Expression Profiling
MESH: Genes, myc
MESH: Carcinogenesis/genetics
MESH: Apoptosis*/genetics
MESH: Necrosis*/genetics
MESH: Liver Neoplasms/genetics
MESH: Mosaicism
MESH: Hepatocytes/metabolism
MESH: Humans
MESH: Cholangiocarcinoma/genetics
digestive system diseases
MESH: Carcinoma, Hepatocellular/pathology
MESH: DNA Transposable Elements/genetics
MESH: Mice
MESH: Tumor Microenvironment
MESH: Proto-Oncogene Proteins c-akt/genetics
MESH: T-Box Domain Proteins/genetics
MESH: Cell Differentiation
570 Life sciences
biology
MESH: Cell Lineage*/genetics
MESH: Cyclin-Dependent Kinase Inhibitor p16/deficiency
MESH: Genes, ras
Źródło :
Nature, Nature Publishing Group, 2018, 562 (7725), pp.69-75. ⟨10.1038/s41586-018-0519-y⟩
Seehawer, Marco; Heinzmann, Florian; D’Artista, Luana; Harbig, Jule; Roux, Pierre-François; Hoenicke, Lisa; Dang, Hien; Klotz, Sabrina; Robinson, Lucas; Doré, Grégory; Rozenblum, Nir; Kang, Tae-Won; Chawla, Rishabh; Buch, Thorsten; Vucur, Mihael; Roth, Mareike; Zuber, Johannes; Luedde, Tom; Sipos, Bence; Longerich, Thomas; Heikenwälder, Mathias; Wang, Xin Wei; Bischof, Oliver; Zender, Lars (2018). Necroptosis microenvironment directs lineage commitment in liver cancer. Nature, 562(7725):69-75.
Opis pliku :
application/pdf
Tytuł :
Whi2 is a conserved negative regulator of TORC1 in response to low amino acids.
Autorzy :
Chen, Xianghui
Wang, Guiqin
Zhang, Yu
Dayhoff-Brannigan, Margaret
Diny, Nicola
Zhao, Mingjun
He, Ge
Sing, Cierra
Metz, Kyle
Stolp, Zachary
AOUACHERIA, Abdel
Cheng, Wen-Chih
Hardwick, J. Marie
Teng, Xinchen
Pokaż więcej
Temat :
[INFO.INFO-BI]Computer Science [cs]/Bioinformatics [q-bio.QM]
[SDV.BID.EVO]Life Sciences [q-bio]/Biodiversity/Populations and Evolution [q-bio.PE]
MESH: Transcription Factors/metabolism
MESH: Gene Expression Regulation
MESH: Membrane Proteins/genetics
MESH: Humans
[INFO.INFO-BT]Computer Science [cs]/Biotechnology
[SDV.GEN]Life Sciences [q-bio]/Genetics
[SDV.BC.BC]Life Sciences [q-bio]/Cellular Biology/Subcellular Processes [q-bio.SC]
MESH: Transcription Factors/genetics
[SDV.BBM.BC]Life Sciences [q-bio]/Biochemistry, Molecular Biology/Biomolecules [q-bio.BM]
[SDV.BC]Life Sciences [q-bio]/Cellular Biology
[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry, Molecular Biology/Molecular biology
QH426-470
MESH: COS Cells
Genetics
[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry, Molecular Biology/Genomics [q-bio.GN]
MESH: Membrane Proteins/metabolism
MESH: Intracellular Signaling Peptides and Proteins/genetics
MESH: Cercopithecus aethiops
MESH: Intracellular Signaling Peptides and Proteins/metabolism
Źródło :
PLoS Genetics, Vol 14, Iss 8, p e1007592 (2018)
PLoS Genetics, Public Library of Science, 2018, 14 (8), pp.e1007592. ⟨10.1371/journal.pgen.1007592⟩
Tytuł :
A recessive form of Hyper IgE Syndrome by disruption of ZNF341-dependent STAT3 transcription and activity
Autorzy :
Beziat, Vivien
Li, Juan
Lin, Jian-Xin
Ma, Cindy
Li, Peng
Bousfiha, Aziz
Pellier, Isabelle
Zoghi, Samaneh
Baris, Safa
Keles, Sevgi
Gray, Paul
Du, Ning
Wang, Yi
Zerbib, Yoann
Levy, Romain
Leclercq, Thibaut
About, Frédégonde
Lim, Ai Ing
Rao, Geetha
Payne, Kathryn
Pelham, Simon
Avery, Danielle
Deenick, Elissa
Pillay, Bethany
Chou, Janet
Guery, Romain
Belkadi, Aziz
Guerin, Antoine
Migaud, Melanie
Rattina, Vimel
Ailal, Fatima
Benhsaien, Ibtihal
Bouaziz, Matthieu
Habib, Tanwir
Chaussabel, Damien
Marr, Nico
El-Benna, Jamel
Grimbacher, Bodo
Wargon, Orli
Bustamante, Jacinta
Boisson, Bertrand
Müller-Fleckenstein, Ingrid
Fleckenstein, Bernhard
Chandesris, Marie-Olivia
Titeux, Matthias
Fraitag, Sylvie
Alyanakian, Marie-Alexandra
Leruez-Ville, Marianne
Picard, Capucine
Meyts, Isabelle
Di Santo, James
Hovnanian, Alain
Somer, Ayper
Ozen, Ahmet
Rezaei, Nima
Chatila, Talal
Abel, Laurent
Leonard, Warren
Tangye, Stuart
Puel, Anne
Casanova, Jean-Laurent
Pokaż więcej
Temat :
MESH: Cell Differentiation/immunology
MESH: Th17 Cells/metabolism
MESH: Male
MESH: Gene Expression Regulation/immunology
MESH: RNA, Messenger/metabolism
MESH: Transcription Factors/immunology
[SDV.IMM]Life Sciences [q-bio]/Immunology
MESH: Transcription Factors/metabolism
MESH: Homozygote
MESH: Th2 Cells/metabolism
MESH: Lymphocyte Count
MESH: STAT3 Transcription Factor/immunology
MESH: Transcription Factors/genetics
MESH: Transcription, Genetic/immunology
MESH: Cytokines/metabolism
MESH: Young Adult
MESH: Th17 Cells/immunology
MESH: Cell Nucleus/metabolism
MESH: Female
MESH: Adult
MESH: Cell Differentiation/genetics
MESH: Genes, Recessive/immunology
MESH: Whole Exome Sequencing
MESH: STAT3 Transcription Factor/metabolism
MESH: Adolescent
MESH: Pedigree
MESH: Job Syndrome/blood
MESH: Th2 Cells/immunology
MESH: Consanguinity
MESH: Exons/genetics
MESH: Humans
MESH: Immunoglobulin E/blood
MESH: STAT3 Transcription Factor/genetics
MESH: Cytokines/immunology
Article
MESH: Loss of Function Mutation
MESH: Middle Aged
MESH: Genes, Recessive/genetics
MESH: Promoter Regions, Genetic/genetics
MESH: Zinc Fingers/genetics
MESH: Job Syndrome/immunology
MESH: Immunoglobulin E/immunology
MESH: Job Syndrome/genetics
Źródło :
Science Immunology, American Association for the Advancement of Science, 2018, 3 (24), pp.eaat4956. ⟨10.1126/sciimmunol.aat4956⟩
Tytuł :
A recessive form of hyper-IgE syndrome by disruption of ZNF341-dependent STAT3 transcription and activity
Autorzy :
Beziat, Vivien
Li, Juan
Lin, Jian-Xin
Ma, Cindy
Li, Peng
Bousfiha, Aziz
Pellier, Isabelle
Zoghi, Samaneh
Baris, Safa
Keles, Sevgi
Gray, Paul
Du, Ning
Wang, Yi
Zerbib, Yoann
Levy, Romain
Leclercq, Thibaut
About, Frédégonde
Lim, Ai Ing
Rao, Geetha
Payne, Kathryn
Pelham, Simon
Avery, Danielle
Deenick, Elissa
Pillay, Bethany
Chou, Janet
Guery, Romain
Belkadi, Aziz
Guerin, Antoine
Migaud, Melanie
Rattina, Vimel
Ailal, Fatima
Benhsaien, Ibtihal
Bouaziz, Matthieu
Habib, Tanwir
Chaussabel, Damien
Marr, Nico
El-Benna, Jamel
Grimbacher, Bodo
Wargon, Orli
Bustamante, Jacinta
Boisson, Bertrand
Müller-Fleckenstein, Ingrid
Fleckenstein, Bernhard
Chandesris, Marie-Olivia
Titeux, Matthias
Fraitag, Sylvie
Alyanakian, Marie-Alexandra
Leruez-Ville, Marianne
Picard, Capucine
Meyts, Isabelle
Di Santo, James
Hovnanian, Alain
Somer, Ayper
Ozen, Ahmet
Rezaei, Nima
Chatila, Talal
Abel, Laurent
Leonard, Warren
Tangye, Stuart
Puel, Anne
Casanova, Jean-Laurent
Pokaż więcej
Temat :
MESH: Adolescent
MESH: Adult
MESH: Job Syndrome/genetics
MESH: Job Syndrome/immunology
MESH: Cell Differentiation/genetics
MESH: Loss of Function Mutation
MESH: Lymphocyte Count
MESH: Male
MESH: Middle Aged
MESH: Pedigree
MESH: Promoter Regions, Genetic/genetics
MESH: RNA, Messenger/metabolism
MESH: Gene Expression Regulation/immunology
MESH: STAT3 Transcription Factor/genetics
MESH: STAT3 Transcription Factor/immunology
MESH: STAT3 Transcription Factor/metabolism
MESH: Cell Differentiation/immunology
MESH: Th17 Cells/immunology
MESH: Th17 Cells/metabolism
MESH: Th2 Cells/immunology
MESH: Th2 Cells/metabolism
MESH: Transcription Factors/genetics
MESH: Transcription Factors/immunology
MESH: Genes, Recessive/genetics
MESH: Transcription Factors/metabolism
MESH: Transcription, Genetic/immunology
MESH: Whole Exome Sequencing
MESH: Young Adult
MESH: Cell Nucleus/metabolism
MESH: Zinc Fingers/genetics
MESH: Consanguinity
MESH: Cytokines/immunology
MESH: Cytokines/metabolism
MESH: Exons/genetics
MESH: Genes, Recessive/immunology
MESH: Female
MESH: Homozygote
MESH: Humans
MESH: Immunoglobulin E/blood
MESH: Immunoglobulin E/immunology
MESH: Job Syndrome/blood
[SDV.IMM]Life Sciences [q-bio]/Immunology
Źródło :
Science Immunology, American Association for the Advancement of Science, 2018, 3 (24), pp.eaat4956. ⟨10.1126/sciimmunol.aat4956⟩
Tytuł :
A recessive form of hyper-IgE syndrome by disruption of ZNF341-dependent STAT3 transcription and activity
Autorzy :
Beziat, Vivien
Li, Juan
Lin, Jian-Xin
Ma, Cindy
Li, Peng
Bousfiha, Aziz
Pellier, Isabelle
Zoghi, Samaneh
Baris, Safa
Keles, Sevgi
Gray, Paul
Du, Ning
Wang, Yi
Zerbib, Yoann
Levy, Romain
Leclercq, Thibaut
About, Frédégonde
Lim, Ai Ing
Rao, Geetha
Payne, Kathryn
Pelham, Simon
Avery, Danielle
Deenick, Elissa
Pillay, Bethany
Chou, Janet
Guery, Romain
Belkadi, Aziz
Guerin, Antoine
Migaud, Melanie
Rattina, Vimel
Ailal, Fatima
Benhsaien, Ibtihal
Bouaziz, Matthieu
Habib, Tanwir
Chaussabel, Damien
Marr, Nico
El-Benna, Jamel
Grimbacher, Bodo
Wargon, Orli
Bustamante, Jacinta
Boisson, Bertrand
Müller-Fleckenstein, Ingrid
Fleckenstein, Bernhard
Chandesris, Marie-Olivia
Titeux, Matthias
Fraitag, Sylvie
Alyanakian, Marie-Alexandra
Leruez-Ville, Marianne
Picard, Capucine
Meyts, Isabelle
Di Santo, James
Hovnanian, Alain
Somer, Ayper
Ozen, Ahmet
Rezaei, Nima
Chatila, Talal
Abel, Laurent
Leonard, Warren
Tangye, Stuart
Puel, Anne
Casanova, Jean-Laurent
Pokaż więcej
Temat :
MESH: Adolescent
MESH: Adult
MESH: Job Syndrome/genetics
MESH: Job Syndrome/immunology
MESH: Cell Differentiation/genetics
MESH: Loss of Function Mutation
MESH: Lymphocyte Count
MESH: Male
MESH: Middle Aged
MESH: Pedigree
MESH: Promoter Regions, Genetic/genetics
MESH: RNA, Messenger/metabolism
MESH: Gene Expression Regulation/immunology
MESH: STAT3 Transcription Factor/genetics
MESH: STAT3 Transcription Factor/immunology
MESH: STAT3 Transcription Factor/metabolism
MESH: Cell Differentiation/immunology
MESH: Th17 Cells/immunology
MESH: Th17 Cells/metabolism
MESH: Th2 Cells/immunology
MESH: Th2 Cells/metabolism
MESH: Transcription Factors/genetics
MESH: Transcription Factors/immunology
MESH: Genes, Recessive/genetics
MESH: Transcription Factors/metabolism
MESH: Transcription, Genetic/immunology
MESH: Whole Exome Sequencing
MESH: Young Adult
MESH: Cell Nucleus/metabolism
MESH: Zinc Fingers/genetics
MESH: Consanguinity
MESH: Cytokines/immunology
MESH: Cytokines/metabolism
MESH: Exons/genetics
MESH: Genes, Recessive/immunology
MESH: Female
MESH: Homozygote
MESH: Humans
MESH: Immunoglobulin E/blood
MESH: Immunoglobulin E/immunology
MESH: Job Syndrome/blood
[SDV.IMM]Life Sciences [q-bio]/Immunology
Źródło :
Science Immunology, American Association for the Advancement of Science, 2018, 3 (24), pp.eaat4956. ⟨10.1126/sciimmunol.aat4956⟩
Tytuł :
A recessive form of hyper-IgE syndrome by disruption of ZNF341-dependent STAT3 transcription and activity
Autorzy :
Beziat, Vivien
Li, Juan
Lin, Jian-Xin
Ma, Cindy
Li, Peng
Bousfiha, Aziz
Pellier, Isabelle
Zoghi, Samaneh
Baris, Safa
Keles, Sevgi
Gray, Paul
Du, Ning
Wang, Yi
Zerbib, Yoann
Levy, Romain
Leclercq, Thibaut
About, Frédégonde
Lim, Ai Ing
Rao, Geetha
Payne, Kathryn
Pelham, Simon
Avery, Danielle
Deenick, Elissa
Pillay, Bethany
Chou, Janet
Guery, Romain
Belkadi, Aziz
Guerin, Antoine
Migaud, Melanie
Rattina, Vimel
Ailal, Fatima
Benhsaien, Ibtihal
Bouaziz, Matthieu
Habib, Tanwir
Chaussabel, Damien
Marr, Nico
El-Benna, Jamel
Grimbacher, Bodo
Wargon, Orli
Bustamante, Jacinta
Boisson, Bertrand
Müller-Fleckenstein, Ingrid
Fleckenstein, Bernhard
Chandesris, Marie-Olivia
Titeux, Matthias
Fraitag, Sylvie
Alyanakian, Marie-Alexandra
Leruez-Ville, Marianne
Picard, Capucine
Meyts, Isabelle
Di Santo, James
Hovnanian, Alain
Somer, Ayper
Ozen, Ahmet
Rezaei, Nima
Chatila, Talal
Abel, Laurent
Leonard, Warren
Tangye, Stuart
Puel, Anne
Casanova, Jean-Laurent
Pokaż więcej
Temat :
MESH: Adolescent
MESH: Adult
MESH: Job Syndrome/genetics
MESH: Job Syndrome/immunology
MESH: Cell Differentiation/genetics
MESH: Loss of Function Mutation
MESH: Lymphocyte Count
MESH: Male
MESH: Middle Aged
MESH: Pedigree
MESH: Promoter Regions, Genetic/genetics
MESH: RNA, Messenger/metabolism
MESH: Gene Expression Regulation/immunology
MESH: STAT3 Transcription Factor/genetics
MESH: STAT3 Transcription Factor/immunology
MESH: STAT3 Transcription Factor/metabolism
MESH: Cell Differentiation/immunology
MESH: Th17 Cells/immunology
MESH: Th17 Cells/metabolism
MESH: Th2 Cells/immunology
MESH: Th2 Cells/metabolism
MESH: Transcription Factors/genetics
MESH: Transcription Factors/immunology
MESH: Genes, Recessive/genetics
MESH: Transcription Factors/metabolism
MESH: Transcription, Genetic/immunology
MESH: Whole Exome Sequencing
MESH: Young Adult
MESH: Cell Nucleus/metabolism
MESH: Zinc Fingers/genetics
MESH: Consanguinity
MESH: Cytokines/immunology
MESH: Cytokines/metabolism
MESH: Exons/genetics
MESH: Genes, Recessive/immunology
MESH: Female
MESH: Homozygote
MESH: Humans
MESH: Immunoglobulin E/blood
MESH: Immunoglobulin E/immunology
MESH: Job Syndrome/blood
[SDV.IMM]Life Sciences [q-bio]/Immunology
Źródło :
Science Immunology, American Association for the Advancement of Science, 2018, 3 (24), pp.eaat4956. ⟨10.1126/sciimmunol.aat4956⟩
Tytuł :
A recessive form of hyper-IgE syndrome by disruption of ZNF341-dependent STAT3 transcription and activity
Autorzy :
Beziat, Vivien
Li, Juan
Lin, Jian-Xin
Ma, Cindy
Li, Peng
Bousfiha, Aziz
Pellier, Isabelle
Zoghi, Samaneh
Baris, Safa
Keles, Sevgi
Gray, Paul
Du, Ning
Wang, Yi
Zerbib, Yoann
Levy, Romain
Leclercq, Thibaut
About, Frédégonde
Lim, Ai Ing
Rao, Geetha
Payne, Kathryn
Pelham, Simon
Avery, Danielle
Deenick, Elissa
Pillay, Bethany
Chou, Janet
Guery, Romain
Belkadi, Aziz
Guerin, Antoine
Migaud, Melanie
Rattina, Vimel
Ailal, Fatima
Benhsaien, Ibtihal
Bouaziz, Matthieu
Habib, Tanwir
Chaussabel, Damien
Marr, Nico
El-Benna, Jamel
Grimbacher, Bodo
Wargon, Orli
Bustamante, Jacinta
Boisson, Bertrand
Müller-Fleckenstein, Ingrid
Fleckenstein, Bernhard
Chandesris, Marie-Olivia
Titeux, Matthias
Fraitag, Sylvie
Alyanakian, Marie-Alexandra
Leruez-Ville, Marianne
Picard, Capucine
Meyts, Isabelle
Di Santo, James
Hovnanian, Alain
Somer, Ayper
Ozen, Ahmet
Rezaei, Nima
Chatila, Talal
Abel, Laurent
Leonard, Warren
Tangye, Stuart
Puel, Anne
Casanova, Jean-Laurent
Pokaż więcej
Temat :
MESH: Adolescent
MESH: Adult
MESH: Job Syndrome/genetics
MESH: Job Syndrome/immunology
MESH: Cell Differentiation/genetics
MESH: Loss of Function Mutation
MESH: Lymphocyte Count
MESH: Male
MESH: Middle Aged
MESH: Pedigree
MESH: Promoter Regions, Genetic/genetics
MESH: RNA, Messenger/metabolism
MESH: Gene Expression Regulation/immunology
MESH: STAT3 Transcription Factor/genetics
MESH: STAT3 Transcription Factor/immunology
MESH: STAT3 Transcription Factor/metabolism
MESH: Cell Differentiation/immunology
MESH: Th17 Cells/immunology
MESH: Th17 Cells/metabolism
MESH: Th2 Cells/immunology
MESH: Th2 Cells/metabolism
MESH: Transcription Factors/genetics
MESH: Transcription Factors/immunology
MESH: Genes, Recessive/genetics
MESH: Transcription Factors/metabolism
MESH: Transcription, Genetic/immunology
MESH: Whole Exome Sequencing
MESH: Young Adult
MESH: Cell Nucleus/metabolism
MESH: Zinc Fingers/genetics
MESH: Consanguinity
MESH: Cytokines/immunology
MESH: Cytokines/metabolism
MESH: Exons/genetics
MESH: Genes, Recessive/immunology
MESH: Female
MESH: Homozygote
MESH: Humans
MESH: Immunoglobulin E/blood
MESH: Immunoglobulin E/immunology
MESH: Job Syndrome/blood
[SDV.IMM]Life Sciences [q-bio]/Immunology
Źródło :
Science Immunology, American Association for the Advancement of Science, 2018, 3 (24), pp.eaat4956. ⟨10.1126/sciimmunol.aat4956⟩

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