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Wyświetlanie 1-20 z 43
Tytuł:
ATG9A prevents TNF cytotoxicity by an unconventional lysosomal targeting pathway.
Autorzy:
Huyghe J; VIB Center for Inflammation Research, 9052 Ghent, Belgium.; Department of Biomedical Molecular Biology, Ghent University, 9052 Ghent, Belgium.
Priem D; VIB Center for Inflammation Research, 9052 Ghent, Belgium.; Department of Biomedical Molecular Biology, Ghent University, 9052 Ghent, Belgium.
Van Hove L; VIB Center for Inflammation Research, 9052 Ghent, Belgium.; Department of Biomedical Molecular Biology, Ghent University, 9052 Ghent, Belgium.
Gilbert B; VIB Center for Inflammation Research, 9052 Ghent, Belgium.; Department of Biomedical Molecular Biology, Ghent University, 9052 Ghent, Belgium.
Fritsch J; Department of Infection Prevention and Infectious Diseases, University Hospital Regensburg, 93053 Regensburg, Germany.
Uchiyama Y; Department of Cell Biology and Neuroscience, Juntendo University Graduate School of Medicine, 113-8654 Tokyo, Japan.
Hoste E; VIB Center for Inflammation Research, 9052 Ghent, Belgium.; Department of Biomedical Molecular Biology, Ghent University, 9052 Ghent, Belgium.
van Loo G; VIB Center for Inflammation Research, 9052 Ghent, Belgium.; Department of Biomedical Molecular Biology, Ghent University, 9052 Ghent, Belgium.
Bertrand MJM; VIB Center for Inflammation Research, 9052 Ghent, Belgium.; Department of Biomedical Molecular Biology, Ghent University, 9052 Ghent, Belgium.
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Źródło:
Science (New York, N.Y.) [Science] 2022 Dec 16; Vol. 378 (6625), pp. 1201-1207. Date of Electronic Publication: 2022 Dec 15.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Autophagy-Related Proteins*/genetics
Autophagy-Related Proteins*/metabolism
Membrane Proteins*/genetics
Membrane Proteins*/metabolism
Tumor Necrosis Factor-alpha*/metabolism
Vesicular Transport Proteins*/genetics
Vesicular Transport Proteins*/metabolism
Animals ; Mice ; Dermatitis/genetics ; Dermatitis/metabolism ; Dermatitis/pathology ; Disease Models, Animal ; Embryo Loss/genetics ; Embryo Loss/metabolism ; Embryo Loss/pathology ; Lysosomes/metabolism ; Receptors, Tumor Necrosis Factor, Type I/metabolism ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
RIPK1 ensures intestinal homeostasis by protecting the epithelium against apoptosis.
Autorzy:
Takahashi N; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium.
Vereecke L; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium.
Bertrand MJ; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium.
Duprez L; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium.
Berger SB; Pattern Recognition Receptor Discovery Performance Unit, Immuno-inflammation Therapeutic Area, GlaxoSmithKline, Collegeville, Pennsylvania 19426, USA.
Divert T; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium.
Gonçalves A; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium [3] VIB Bio Imaging Core Gent, Technologiepark 927, B-9052 Ghent, Belgium.
Sze M; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium.
Gilbert B; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium.
Kourula S; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium.
Goossens V; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium.
Lefebvre S; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium.
Günther C; Department of Medicine 1, Friedrich-Alexander-University, D-91054 Erlangen, Germany.
Becker C; Department of Medicine 1, Friedrich-Alexander-University, D-91054 Erlangen, Germany.
Bertin J; Pattern Recognition Receptor Discovery Performance Unit, Immuno-inflammation Therapeutic Area, GlaxoSmithKline, Collegeville, Pennsylvania 19426, USA.
Gough PJ; Pattern Recognition Receptor Discovery Performance Unit, Immuno-inflammation Therapeutic Area, GlaxoSmithKline, Collegeville, Pennsylvania 19426, USA.
Declercq W; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium.
van Loo G; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium.
Vandenabeele P; 1] VIB Inflammation Research Center, Technologiepark 927, B-9052 Ghent, Belgium [2] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium [3] Methusalem program, Ghent University, Technologiepark 927, B-9052 Ghent, Belgium.
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Źródło:
Nature [Nature] 2014 Sep 04; Vol. 513 (7516), pp. 95-9.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*/drug effects
Homeostasis*/drug effects
Epithelial Cells/*cytology
Epithelial Cells/*metabolism
Epithelium/*metabolism
Intestinal Mucosa/*metabolism
Intestines/*cytology
Receptor-Interacting Protein Serine-Threonine Kinases/*metabolism
Animals ; Anti-Bacterial Agents/pharmacology ; Caspase 8/genetics ; Caspase 8/metabolism ; Cell Survival/drug effects ; Epithelial Cells/drug effects ; Epithelial Cells/pathology ; Epithelium/drug effects ; Epithelium/pathology ; Female ; Gene Deletion ; Inflammation/metabolism ; Inflammation/pathology ; Intestines/drug effects ; Intestines/pathology ; Male ; Mice ; Mice, Knockout ; Myeloid Differentiation Factor 88/deficiency ; NF-kappa B/metabolism ; Necrosis ; Organoids/cytology ; Organoids/drug effects ; Organoids/enzymology ; Organoids/metabolism ; Protein Kinases/metabolism ; Receptor-Interacting Protein Serine-Threonine Kinases/deficiency ; Receptor-Interacting Protein Serine-Threonine Kinases/genetics ; Receptors, Tumor Necrosis Factor, Type I/deficiency ; Survival Analysis ; Tumor Necrosis Factors/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Hyperforin inhibits Akt1 kinase activity and promotes caspase-mediated apoptosis involving Bad and Noxa activation in human myeloid tumor cells.
Autorzy:
Merhi F; INSERM U872, Université Pierre et Marie Curie, Université Paris Descartes, Centre de Recherche des Cordeliers, Paris, France.
Tang R
Piedfer M
Mathieu J
Bombarda I
Zaher M
Kolb JP
Billard C
Bauvois B
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Źródło:
PloS one [PLoS One] 2011; Vol. 6 (10), pp. e25963. Date of Electronic Publication: 2011 Oct 06.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Caspases/*metabolism
Leukemia, Myeloid, Acute/*pathology
Phloroglucinol/*analogs & derivatives
Proto-Oncogene Proteins c-akt/*antagonists & inhibitors
Proto-Oncogene Proteins c-bcl-2/*metabolism
Terpenes/*pharmacology
bcl-Associated Death Protein/*metabolism
Adult ; Aged ; Aged, 80 and over ; Antineoplastic Agents/pharmacology ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Down-Regulation/drug effects ; Female ; Humans ; Intracellular Space/drug effects ; Intracellular Space/metabolism ; Leukemia, Myeloid, Acute/metabolism ; Male ; Middle Aged ; Mitochondrial Membranes/drug effects ; Mitochondrial Membranes/metabolism ; NF-kappa B/metabolism ; Phloroglucinol/pharmacology ; Phosphorylation/drug effects ; Proto-Oncogene Proteins c-akt/metabolism ; Signal Transduction/drug effects ; Up-Regulation/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
A PARP-1/JNK1 cascade participates in the synergistic apoptotic effect of TNFalpha and all-trans retinoic acid in APL cells.
Autorzy:
Mathieu J; INSERM Unité 685, Institut Universitaire d'Hématologie, Paris Cedex 10, France.
Flexor M
Lanotte M
Besançon F
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Źródło:
Oncogene [Oncogene] 2008 May 29; Vol. 27 (24), pp. 3361-70. Date of Electronic Publication: 2007 Dec 17.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Drug Synergism*
Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Leukemia, Promyelocytic, Acute/*metabolism
Mitogen-Activated Protein Kinase 8/*metabolism
Poly(ADP-ribose) Polymerases/*metabolism
Tretinoin/*pharmacology
Tumor Necrosis Factor-alpha/*pharmacology
Caspases/metabolism ; Cytochromes c/metabolism ; Enzyme Activation/drug effects ; Enzyme Inhibitors/pharmacology ; Flow Cytometry ; Humans ; Immunoblotting ; Leukemia, Promyelocytic, Acute/pathology ; Membrane Potential, Mitochondrial/drug effects ; Mitogen-Activated Protein Kinase 8/antagonists & inhibitors ; Mitogen-Activated Protein Kinase 8/genetics ; NF-kappa B/antagonists & inhibitors ; NF-kappa B/genetics ; NF-kappa B/metabolism ; Poly (ADP-Ribose) Polymerase-1 ; Poly(ADP-ribose) Polymerase Inhibitors ; Poly(ADP-ribose) Polymerases/genetics ; RNA, Small Interfering/pharmacology ; Recombinant Proteins ; Sarcoma, Ewing/metabolism ; Sarcoma, Ewing/pathology ; Tumor Cells, Cultured/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Arsenic trioxide represses NF-kappaB activation and increases apoptosis in ATRA-treated APL cells.
Autorzy:
Mathieu J; INSERM U685, Centre Hayem, Hôpital St. Louis, 1 Avenue Claude Vellefaux, 75475 Paris Cedex 10, France. julie.mathieu@stlouis.inserm.fr
Besançon F
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Źródło:
Annals of the New York Academy of Sciences [Ann N Y Acad Sci] 2006 Dec; Vol. 1090, pp. 203-8.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Arsenicals/*pharmacology
Leukemia, Promyelocytic, Acute/*pathology
NF-kappa B/*metabolism
Oxides/*pharmacology
Tretinoin/*pharmacology
Arsenic Trioxide ; Base Sequence ; DNA Probes ; Humans
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
[Induction of apoptosis of splenic lymphocytes in mice by accelerated carbon ions].
Autorzy:
Holl V; Laboratoire de Cancérologie Expérimentale et de Radiobiologie, Institut de Recherche contre les Cancers de l'Appareil Digestif, H pitaux Universitaires, Strasbourg, France.
Coelho D
Weltin D
Dufour P
Denis JM
Florentin I
Mathieu J
Gueulette J
Bischoff P
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Transliterated Title:
Induction de l'apoptose dans les lymphocytes spléniques de souris par un faisceau d'ions carbone.
Źródło:
Canadian journal of physiology and pharmacology [Can J Physiol Pharmacol] 2001 Feb; Vol. 79 (2), pp. 109-13.
Typ publikacji:
English Abstract; Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Carbon*
Heavy Ions*
Apoptosis/*radiation effects
Lymphocytes/*radiation effects
Spleen/*radiation effects
Animals ; Caspase Inhibitors ; Enzyme Inhibitors/pharmacology ; Female ; Ions ; Mice ; Mice, Inbred BALB C ; Mice, Inbred C3H ; Spleen/cytology ; X-Rays
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Wyświetlanie 1-20 z 43

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