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    Wyświetlanie 1-12 z 12
    Tytuł:
    Overexpression of cathepsin K in mice decreases collagen deposition and lung resistance in response to bleomycin-induced pulmonary fibrosis.
    Autorzy:
    Srivastava M
    Steinwede K
    Kiviranta R
    Morko J
    Hoymann HG
    Länger F
    Buhling F
    Welte T
    Maus UA
    Srivastava, Mrigank
    Steinwede, Kathrin
    Kiviranta, Riku
    Morko, Jukka
    Hoymann, Heinz-Gerd
    Länger, Florian
    Buhling, Frank
    Welte, Tobias
    Maus, Ulrich A
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    Źródło:
    Respiratory Research; 2008, Vol. 9, p54-54, 1p
    Czasopismo naukowe
    Szczegóły
    Tytuł:
    Exploring alveolar recruitability using positive end-expiratory pressure in mice overexpressing TGF-β1: a structure-function analysis.
    Autorzy:
    Roeder F; Institute of Functional and Applied Anatomy, Hannover Medical School, Hannover, Germany.
    Röpke T; Department of Experimental Pneumology, Hannover Medical School, Hannover, Germany.
    Steinmetz LK; Department of Experimental Pneumology, Hannover Medical School, Hannover, Germany.
    Kolb M; Department of Medicine, Firestone Institute for Respiratory Health, McMaster University, Hamilton, ON, Canada.
    Maus UA; Department of Experimental Pneumology, Hannover Medical School, Hannover, Germany.; Biomedical Research in Endstage and Obstructive Lung Disease Hannover (BREATH), Member of the German Center for Lung Disease (DZL), Hannover, Germany.
    Smith BJ; Department of Bioengineering, College of Engineering Design and Computing, University of Colorado Denver|Anschutz Medical Campus, Aurora, CO, USA.; Department of Pediatric Pulmonary and Sleep Medicine, School of Medicine, University of Colorado Anschutz Medical Campus, Aurora, CO, USA.
    Knudsen L; Institute of Functional and Applied Anatomy, Hannover Medical School, Hannover, Germany. .; Biomedical Research in Endstage and Obstructive Lung Disease Hannover (BREATH), Member of the German Center for Lung Disease (DZL), Hannover, Germany. .
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    Źródło:
    Scientific reports [Sci Rep] 2024 Apr 06; Vol. 14 (1), pp. 8080. Date of Electronic Publication: 2024 Apr 06.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Transforming Growth Factor beta1*
    Pulmonary Atelectasis*
    Mice ; Animals ; Positive-Pressure Respiration/methods ; Lung ; Pulmonary Alveoli/physiology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Inflammation-inducible promoters to overexpress immune inhibitory factors by MSCs.
    Autorzy:
    Selich A; Hannover Medical School, Institute of Experimental Hematology, Building J11, HBZ, Level 01, Room, 6540, Hannover, Germany.
    Fleischauer J; Hannover Medical School, Institute of Experimental Hematology, Building J11, HBZ, Level 01, Room, 6540, Hannover, Germany.
    Roepke T; Division of Experimental Pneumology, Hannover Medical School, Hannover, Germany.
    Weisskoeppel L; Hannover Medical School, Institute of Experimental Hematology, Building J11, HBZ, Level 01, Room, 6540, Hannover, Germany.
    Galla M; Hannover Medical School, Institute of Experimental Hematology, Building J11, HBZ, Level 01, Room, 6540, Hannover, Germany.
    von Kaisenberg C; Department of Obstetrics and Gynecology, Hannover Medical School, Hannover, Germany.
    Maus UA; Division of Experimental Pneumology, Hannover Medical School, Hannover, Germany.; German Center for Lung Research, Partner Site BREATH, Hannover, Germany.
    Schambach A; Hannover Medical School, Institute of Experimental Hematology, Building J11, HBZ, Level 01, Room, 6540, Hannover, Germany.; Division of Hematology/Oncology, Boston Children's Hospital, Harvard Medical School, Boston, MA, USA.
    Rothe M; Hannover Medical School, Institute of Experimental Hematology, Building J11, HBZ, Level 01, Room, 6540, Hannover, Germany. .
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    Źródło:
    Stem cell research & therapy [Stem Cell Res Ther] 2023 Sep 23; Vol. 14 (1), pp. 270. Date of Electronic Publication: 2023 Sep 23.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Interleukin-10*/genetics
    Mesenchymal Stem Cells*
    Humans ; Animals ; Mice ; Transgenes ; Immunologic Factors ; Enzyme-Linked Immunosorbent Assay
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    S100A9 is indispensable for survival of pneumococcal pneumonia in mice.
    Autorzy:
    Ostermann L; Division of Experimental Pneumology, Hannover Medical School, Hannover, Germany.
    Seeliger B; Clinic for Pneumology, Hannover Medical School, Hannover, Germany.; German Center for Lung Research, Hannover, Germany.
    David S; Institute of Intensive Care Medicine, University Hospital Zurich, Zurich, Switzerland.
    Flasche C; Division of Experimental Pneumology, Hannover Medical School, Hannover, Germany.
    Maus R; Division of Experimental Pneumology, Hannover Medical School, Hannover, Germany.
    Reinboth MS; Division of Experimental Pneumology, Hannover Medical School, Hannover, Germany.
    Christmann M; Institute of Clinical Chemistry, Hannover Medical School, Hannover, Germany.
    Neumann K; Institute of Clinical Chemistry, Hannover Medical School, Hannover, Germany.
    Brand K; Institute of Clinical Chemistry, Hannover Medical School, Hannover, Germany.
    Seltmann S; Labopart Medical Laboratories, Dresden and Chemnitz, Germany.
    Bühling F; Labopart Medical Laboratories, Dresden and Chemnitz, Germany.
    Paton JC; Research Centre for Infectious Diseases, Department of Molecular and Biomedical Science, University of Adelaide, Adelaide, Australia.
    Roth J; Institute of Immunology, University of Münster, Münster, Germany.
    Vogl T; Institute of Immunology, University of Münster, Münster, Germany.
    Viemann D; Department of Pediatric Pneumology, Allergology and Neonatology, Hannover Medical School, Hannover, Germany.; Translational Pediatrics, Department of Pediatrics, University Hospital Würzburg, Germany.; Cluster of Excellence RESIST (EXC 2155), Hannover Medical School, Hannover, Germany.; Center for Infection Research, University Würzburg, Germany.
    Welte T; Clinic for Pneumology, Hannover Medical School, Hannover, Germany.; German Center for Lung Research, Hannover, Germany.
    Maus UA; Division of Experimental Pneumology, Hannover Medical School, Hannover, Germany.; German Center for Lung Research, Hannover, Germany.
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    Źródło:
    PLoS pathogens [PLoS Pathog] 2023 Jul 19; Vol. 19 (7), pp. e1011493. Date of Electronic Publication: 2023 Jul 19 (Print Publication: 2023).
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Pneumonia, Pneumococcal*
    Mice ; Animals ; Calgranulin B/genetics ; Calgranulin B/metabolism ; Lung ; Streptococcus pneumoniae/metabolism ; Calgranulin A/genetics ; Calgranulin A/metabolism ; Bacteria/metabolism ; Mice, Knockout
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Loss of Serpina1 in Mice Leads to Altered Gene Expression in Inflammatory and Metabolic Pathways.
    Autorzy:
    Meghadri SH; Department of Respiratory Medicine, Platform Bioinformatics, Member of the German Center for Lung Research (DZL), Biomedical Research in Endstage and Obstructive Lung Disease (BREATH), Hannover Medical School, 30625 Hannover, Germany.
    Martinez-Delgado B; Functional Genomics Unit, Rare Diseases Research Institute, Carlos III Health Institute, 28220 Majadahonda, Spain.
    Ostermann L; Division of Experimental Pneumology, Member of the German Center for Lung Research (DZL), Biomedical Research in Endstage and Obstructive Lung Disease (BREATH), Hannover Medical School, 30625 Hannover, Germany.
    Gomez-Mariano G; Functional Genomics Unit, Rare Diseases Research Institute, Carlos III Health Institute, 28220 Majadahonda, Spain.
    Perez-Luz S; Functional Genomics Unit, Rare Diseases Research Institute, Carlos III Health Institute, 28220 Majadahonda, Spain.
    Tumpara S; Department of Respiratory Medicine, Molecular Pneumology, Member of German Center for Lung Research (DZL), Biomedical Research School in Endstage and Obstructive Lung Disease (BREATH), Hannover Medical School, 30625 Hannover, Germany.
    Wrenger S; Department of Respiratory Medicine, Molecular Pneumology, Member of German Center for Lung Research (DZL), Biomedical Research School in Endstage and Obstructive Lung Disease (BREATH), Hannover Medical School, 30625 Hannover, Germany.
    DeLuca DS; Department of Respiratory Medicine, Platform Bioinformatics, Member of the German Center for Lung Research (DZL), Biomedical Research in Endstage and Obstructive Lung Disease (BREATH), Hannover Medical School, 30625 Hannover, Germany.
    Maus UA; Division of Experimental Pneumology, Member of the German Center for Lung Research (DZL), Biomedical Research in Endstage and Obstructive Lung Disease (BREATH), Hannover Medical School, 30625 Hannover, Germany.
    Welte T; Department of Respiratory Medicine, Molecular Pneumology, Member of German Center for Lung Research (DZL), Biomedical Research School in Endstage and Obstructive Lung Disease (BREATH), Hannover Medical School, 30625 Hannover, Germany.
    Janciauskiene S; Department of Respiratory Medicine, Molecular Pneumology, Member of German Center for Lung Research (DZL), Biomedical Research School in Endstage and Obstructive Lung Disease (BREATH), Hannover Medical School, 30625 Hannover, Germany.
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    Źródło:
    International journal of molecular sciences [Int J Mol Sci] 2022 Sep 09; Vol. 23 (18). Date of Electronic Publication: 2022 Sep 09.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    alpha 1-Antitrypsin Deficiency*/genetics
    alpha 1-Antitrypsin/*metabolism
    Animals ; Cholesterol ; Gene Expression ; Humans ; Metabolic Networks and Pathways ; Mice ; Mice, Inbred C57BL ; RNA, Small Interfering/metabolism ; Serine Proteinase Inhibitors ; alpha 1-Antitrypsin/genetics
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Role of matrix metalloprotease-2 and MMP-9 in experimental lung fibrosis in mice.
    Autorzy:
    Bormann T; Division of Experimental Pneumology, Hannover Medical School, Feodor-Lynen-Strasse 21, 30625, Hannover, Germany.
    Maus R; Division of Experimental Pneumology, Hannover Medical School, Feodor-Lynen-Strasse 21, 30625, Hannover, Germany.
    Stolper J; Division of Experimental Pneumology, Hannover Medical School, Feodor-Lynen-Strasse 21, 30625, Hannover, Germany.
    Tort Tarrés M; Division of Experimental Pneumology, Hannover Medical School, Feodor-Lynen-Strasse 21, 30625, Hannover, Germany.
    Brandenberger C; Institute of Functional and Applied Anatomy, Hannover Medical School, Carl-Neuberg-Strasse 1, 30625, Hannover, Germany.
    Wedekind D; Institute of Laboratory Animal Science, Hannover Medical School, Carl-Neuberg-Strasse 1, 30625, Hannover, Germany.
    Jonigk D; Department of Pathology, Hannover Medical School, Carl-Neuberg-Strasse 1, 30625, Hannover, Germany.
    Welte T; Clinic for Pneumology, Hannover Medical School, Carl-Neuberg-Strasse 1, 30625, Hannover, Germany.; German Center for Lung Research, Partner Site BREATH, Carl-Neuberg-Strasse 1, 30625, Hannover, Germany.
    Gauldie J; Department of Medicine, Pathology, and Molecular Medicine, McMaster University, 1280 Main St W, Hamilton, ON, L8S 4L8, Canada.
    Kolb M; Department of Medicine, Pathology, and Molecular Medicine, McMaster University, 1280 Main St W, Hamilton, ON, L8S 4L8, Canada.
    Maus UA; Division of Experimental Pneumology, Hannover Medical School, Feodor-Lynen-Strasse 21, 30625, Hannover, Germany. Maus..; German Center for Lung Research, Partner Site BREATH, Carl-Neuberg-Strasse 1, 30625, Hannover, Germany. Maus..
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    Źródło:
    Respiratory research [Respir Res] 2022 Jul 08; Vol. 23 (1), pp. 180. Date of Electronic Publication: 2022 Jul 08.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Idiopathic Pulmonary Fibrosis*/chemically induced
    Idiopathic Pulmonary Fibrosis*/genetics
    Idiopathic Pulmonary Fibrosis*/metabolism
    Matrix Metalloproteinase 2/*metabolism
    Matrix Metalloproteinase 9/*metabolism
    Animals ; Extracellular Matrix Proteins/metabolism ; Lung/metabolism ; Matrix Metalloproteinase 2/genetics ; Matrix Metalloproteinase 9/genetics ; Matrix Metalloproteinases/metabolism ; Mice
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    C-type Lectin Mincle Recognizes Glucosyl-diacylglycerol of Streptococcus pneumoniae and Plays a Protective Role in Pneumococcal Pneumonia.
    Autorzy:
    Behler-Janbeck F; Department of Experimental Pneumology, Hannover Medical School, Hannover, Germany.
    Takano T; Division of Molecular Immunology, Medical Institute of Bioregulation, Kyushu University, Fukuoka, Japan.
    Maus R; Department of Experimental Pneumology, Hannover Medical School, Hannover, Germany.
    Stolper J; Department of Experimental Pneumology, Hannover Medical School, Hannover, Germany.
    Jonigk D; Institute of Pathology, Hannover Medical School, Hannover, Germany.
    Tort Tarrés M; Department of Experimental Pneumology, Hannover Medical School, Hannover, Germany.
    Fuehner T; Clinic for Pneumology, Hannover Medical School, Hannover, Germany.
    Prasse A; Clinic for Pneumology, Hannover Medical School, Hannover, Germany.
    Welte T; Clinic for Pneumology, Hannover Medical School, Hannover, Germany.; German Center for Lung Research, partner site BREATH, Hannover, Germany.
    Timmer MS; School of Chemical and Physical Sciences, Victoria University of Wellington, Wellington, New Zealand.
    Stocker BL; School of Chemical and Physical Sciences, Victoria University of Wellington, Wellington, New Zealand.
    Nakanishi Y; Research Institute for Diseases of the Chest, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.
    Miyamoto T; Department of Natural Products Chemistry, Graduate School of Pharmaceutical Sciences, Kyushu University, Fukuoka, Japan.
    Yamasaki S; Division of Molecular Immunology, Medical Institute of Bioregulation, Kyushu University, Fukuoka, Japan.
    Maus UA; Department of Experimental Pneumology, Hannover Medical School, Hannover, Germany.; German Center for Lung Research, partner site BREATH, Hannover, Germany.
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    Źródło:
    PLoS pathogens [PLoS Pathog] 2016 Dec 06; Vol. 12 (12), pp. e1006038. Date of Electronic Publication: 2016 Dec 06 (Print Publication: 2016).
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Glycolipids/*metabolism
    Lectins, C-Type/*immunology
    Macrophages, Alveolar/*immunology
    Pneumonia, Pneumococcal/*immunology
    Receptors, Immunologic/*immunology
    Streptococcus pneumoniae/*immunology
    Animals ; Chromatography, High Pressure Liquid ; Flow Cytometry ; Glycolipids/immunology ; Humans ; Immunophenotyping ; Lectins, C-Type/metabolism ; Membrane Proteins/immunology ; Membrane Proteins/metabolism ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Neutrophils/immunology ; Real-Time Polymerase Chain Reaction ; Receptors, Immunologic/metabolism ; Streptococcus pneumoniae/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Overexpression of cathepsin K in mice decreases collagen deposition and lung resistance in response to bleomycin-induced pulmonary fibrosis.
    Autorzy:
    Srivastava M; Department of Pulmonary Medicine, Laboratory for Experimental Lung Research, Hannover School of Medicine, Hannover, Germany. />Steinwede K
    Kiviranta R
    Morko J
    Hoymann HG
    Länger F
    Buhling F
    Welte T
    Maus UA
    Pokaż więcej
    Źródło:
    Respiratory research [Respir Res] 2008 Jul 18; Vol. 9, pp. 54. Date of Electronic Publication: 2008 Jul 18.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Cathepsins/*metabolism
    Collagen/*metabolism
    Lung/*metabolism
    Pulmonary Fibrosis/*metabolism
    Animals ; Antibiotics, Antineoplastic ; Bleomycin ; Cathepsin K ; Cathepsins/genetics ; Disease Models, Animal ; Lung/pathology ; Mice ; Mice, Transgenic ; Pulmonary Fibrosis/chemically induced ; Pulmonary Fibrosis/prevention & control ; Respiratory Mucosa/metabolism ; Respiratory Mucosa/pathology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Basophils enhance immunological memory responses.
    Autorzy:
    Denzel A; Department of Internal Medicine II, University Hospital Regensburg, 93042 Regensburg, Germany.
    Maus UA
    Rodriguez Gomez M
    Moll C
    Niedermeier M
    Winter C
    Maus R
    Hollingshead S
    Briles DE
    Kunz-Schughart LA
    Talke Y
    Mack M
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    Źródło:
    Nature immunology [Nat Immunol] 2008 Jul; Vol. 9 (7), pp. 733-42. Date of Electronic Publication: 2008 May 30.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Immunologic Memory*
    Basophils/*immunology
    Adoptive Transfer ; Animals ; Antibody Formation ; Antigen Presentation/immunology ; Antigen-Presenting Cells/immunology ; B-Lymphocytes/immunology ; Bone Marrow/immunology ; Enzyme-Linked Immunosorbent Assay ; Female ; Flow Cytometry ; Interleukin-4/biosynthesis ; Interleukin-6/biosynthesis ; Lymphocyte Activation/immunology ; Mice ; Mice, Inbred C57BL ; Reverse Transcriptase Polymerase Chain Reaction ; Spleen/cytology ; Spleen/immunology ; T-Lymphocytes/immunology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Mediator responses of alveolar macrophages and kinetics of mononuclear phagocyte subset recruitment during acute primary and secondary mycobacterial infections in the lungs of mice.
    Autorzy:
    Srivastava M; Laboratory for Experimental Lung Research, Department of Pulmonary Medicine, Hannover School of Medicine, Hannover, Germany.
    Meinders A
    Steinwede K
    Maus R
    Lucke N
    Bühling F
    Ehlers S
    Welte T
    Maus UA
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    Źródło:
    Cellular microbiology [Cell Microbiol] 2007 Mar; Vol. 9 (3), pp. 738-52. Date of Electronic Publication: 2006 Oct 18.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Lung/*metabolism
    Macrophages, Alveolar/*metabolism
    Mycobacterium bovis/*growth & development
    Tuberculosis, Pulmonary/*genetics
    Animals ; CX3C Chemokine Receptor 1 ; Flow Cytometry ; Gene Expression Profiling ; Green Fluorescent Proteins/genetics ; Green Fluorescent Proteins/metabolism ; Lung/microbiology ; Lung/pathology ; Macrophages, Alveolar/microbiology ; Macrophages, Alveolar/pathology ; Mice ; Mice, Inbred BALB C ; Mice, Transgenic ; Microscopy, Fluorescence ; Monocytes/metabolism ; Monocytes/microbiology ; Monocytes/pathology ; Nod2 Signaling Adaptor Protein/genetics ; Nod2 Signaling Adaptor Protein/metabolism ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Receptors, Chemokine/genetics ; Receptors, Chemokine/metabolism ; Reverse Transcriptase Polymerase Chain Reaction ; Toll-Like Receptor 2/genetics ; Toll-Like Receptor 2/metabolism ; Toll-Like Receptor 4/genetics ; Toll-Like Receptor 4/metabolism ; Tuberculosis, Pulmonary/metabolism ; Tuberculosis, Pulmonary/microbiology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Endotoxin induced peritonitis elicits monocyte immigration into the lung: implications on alveolar space inflammatory responsiveness.
    Autorzy:
    Steinmüller M; Division of Pulmonary and Critical Care Medicine and Infectious Diseases, Department of Internal Medicine, Justus-Liebig-University, Giessen, Germany. />Srivastava M
    Kuziel WA
    Christman JW
    Seeger W
    Welte T
    Lohmeyer J
    Maus UA
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    Źródło:
    Respiratory research [Respir Res] 2006 Feb 18; Vol. 7, pp. 30. Date of Electronic Publication: 2006 Feb 18.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Monocytes/*immunology
    Peritonitis/*immunology
    Peritonitis/*pathology
    Pulmonary Alveoli/*immunology
    Pulmonary Alveoli/*pathology
    Receptors, Chemokine/*immunology
    Animals ; Cell Movement/drug effects ; Female ; Lipopolysaccharides ; Lymphocyte Activation/immunology ; Mice ; Mice, Inbred BALB C ; Monocytes/drug effects ; Peritonitis/chemically induced ; Pneumonia/immunology ; Pneumonia/pathology ; Pulmonary Alveoli/drug effects ; Receptors, CCR2
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
      Wyświetlanie 1-12 z 12

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