Autor: "Meinel, S." - OpacWWW

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Tytuł:: The mineralocorticoid receptor leads to increased expression of EGFR and T-type calcium channels that support HL-1 cell hypertrophy.
Autorzy:: Stroedecke K; Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Magdeburger Str. 6, 06097, Halle, Saale, Germany.
Meinel S; Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Magdeburger Str. 6, 06097, Halle, Saale, Germany.
Markwardt F; Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Magdeburger Str. 6, 06097, Halle, Saale, Germany.
Kloeckner U; Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Magdeburger Str. 6, 06097, Halle, Saale, Germany.
Straetz N; Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Magdeburger Str. 6, 06097, Halle, Saale, Germany.
Quarch K; Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Magdeburger Str. 6, 06097, Halle, Saale, Germany.
Schreier B; Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Magdeburger Str. 6, 06097, Halle, Saale, Germany.
Kopf M; Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Magdeburger Str. 6, 06097, Halle, Saale, Germany.
Gekle M; Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Magdeburger Str. 6, 06097, Halle, Saale, Germany.
Grossmann C; Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Magdeburger Str. 6, 06097, Halle, Saale, Germany. .; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2021 Jun 24; Vol. 11 (1), pp. 13229. Date of Electronic Publication: 2021 Jun 24.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Calcium Channels, T-Type/*genetics
ErbB Receptors/*genetics
Hypertrophy/*genetics
Receptors, Mineralocorticoid/*genetics
Aldosterone/genetics ; Animals ; Cardiovascular System/drug effects ; Cardiovascular System/pathology ; Cell Line ; Female ; Genotype ; HEK293 Cells ; Humans ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Mineralocorticoid Receptor Antagonists/pharmacology ; Myocytes, Cardiac/drug effects ; Polymorphism, Single Nucleotide/genetics ; Rats

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