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Wyszukujesz frazę ""Membrane Potential, Mitochondrial"" wg kryterium: Temat


Tytuł :
Superoxide produced by mitochondrial complex III plays a pivotal role in the execution of ferroptosis induced by cysteine starvation.
Autorzy :
Homma T; Department of Biochemistry and Molecular Biology, Graduate School of Medical Science, Yamagata University, 2-2-2 Iidanishi, Yamagata, 990-9585, Japan. Electronic address: .
Kobayashi S; Department of Biochemistry and Molecular Biology, Graduate School of Medical Science, Yamagata University, 2-2-2 Iidanishi, Yamagata, 990-9585, Japan.
Sato H; Department of Medical Technology, Faculty of Medicine, Niigata University, Niigata, 951-8518, Japan.
Fujii J; Department of Biochemistry and Molecular Biology, Graduate School of Medical Science, Yamagata University, 2-2-2 Iidanishi, Yamagata, 990-9585, Japan.
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Źródło :
Archives of biochemistry and biophysics [Arch Biochem Biophys] 2021 Mar 30; Vol. 700, pp. 108775. Date of Electronic Publication: 2021 Jan 23.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Ferroptosis*
Membrane Potential, Mitochondrial*
Cysteine/*deficiency
Electron Transport Complex III/*metabolism
Mitochondrial Membranes/*metabolism
Superoxides/*metabolism
Animals ; HeLa Cells ; Humans ; Mice
Czasopismo naukowe
Tytuł :
Ndufs1 Deficiency Aggravates the Mitochondrial Membrane Potential Dysfunction in Pressure Overload-Induced Myocardial Hypertrophy.
Autorzy :
Zou R; Heart Center, Guangdong Provincial Key Laboratory of Research in Structural Birth Defect Disease, Guangzhou Women and Children's Medical Center, Guangzhou Medical University, Guangzhou 510623, China.
Tao J; Department of Cardiovascular Surgery, Sun Yat-sen Memorial Hospital, Sun Yat-sen University, Guangzhou, Guangdong 510120, China.
Qiu J; Department of Cardiovascular Surgery, Sun Yat-sen Memorial Hospital, Sun Yat-sen University, Guangzhou, Guangdong 510120, China.
Shi W; Department of Paediatrics, Guangdong Provincial Key Laboratory of Research in Structural Birth Defect Disease, Guangzhou Women and Children's Medical Center, Guangzhou Medical University, Guangzhou 510623, China.
Zou M; Heart Center, Guangdong Provincial Key Laboratory of Research in Structural Birth Defect Disease, Guangzhou Women and Children's Medical Center, Guangzhou Medical University, Guangzhou 510623, China.
Chen W; Heart Center, Guangdong Provincial Key Laboratory of Research in Structural Birth Defect Disease, Guangzhou Women and Children's Medical Center, Guangzhou Medical University, Guangzhou 510623, China.
Li W; Heart Center, Guangdong Provincial Key Laboratory of Research in Structural Birth Defect Disease, Guangzhou Women and Children's Medical Center, Guangzhou Medical University, Guangzhou 510623, China.
Zhou N; Heart Center, Guangdong Provincial Key Laboratory of Research in Structural Birth Defect Disease, Guangzhou Women and Children's Medical Center, Guangzhou Medical University, Guangzhou 510623, China.
Wang S; Department of Surgical Nursing, Guangzhou Women and Children's Medical Center, Guangzhou Medical University, Guangzhou 510623, China.
Ma L; Heart Center, Guangdong Provincial Key Laboratory of Research in Structural Birth Defect Disease, Guangzhou Women and Children's Medical Center, Guangzhou Medical University, Guangzhou 510623, China.
Chen X; Heart Center, Guangdong Provincial Key Laboratory of Research in Structural Birth Defect Disease, Guangzhou Women and Children's Medical Center, Guangzhou Medical University, Guangzhou 510623, China.
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Źródło :
Oxidative medicine and cellular longevity [Oxid Med Cell Longev] 2021 Mar 03; Vol. 2021, pp. 5545261. Date of Electronic Publication: 2021 Mar 03 (Print Publication: 2021).
Typ publikacji :
Journal Article
MeSH Terms :
Membrane Potential, Mitochondrial*
Pressure*
Cardiomegaly/*metabolism
Myocardium/*metabolism
Myocardium/*pathology
NADH Dehydrogenase/*deficiency
Angiotensin II ; Animals ; Atrial Natriuretic Factor/metabolism ; Biomarkers/metabolism ; Cardiomegaly/pathology ; Constriction, Pathologic ; Down-Regulation ; Male ; Mice, Inbred C57BL ; Mitochondria, Heart/metabolism ; Myocytes, Cardiac/metabolism ; Myosin Heavy Chains/metabolism ; NADH Dehydrogenase/metabolism ; Natriuretic Peptide, Brain/metabolism ; RNA-Seq ; Rats ; Single-Cell Analysis
Czasopismo naukowe
Tytuł :
Forces, fluxes, and fuels: tracking mitochondrial metabolism by integrating measurements of membrane potential, respiration, and metabolites.
Autorzy :
Jones AE; Department of Molecular and Medical Pharmacology, University of California, Los Angeles, California.
Sheng L; Department of Molecular and Medical Pharmacology, University of California, Los Angeles, California.
Acevedo A; Department of Molecular and Medical Pharmacology, University of California, Los Angeles, California.
Veliova M; Department of Molecular and Medical Pharmacology, University of California, Los Angeles, California.; Department of Medicine, University of California, Los Angeles, California.
Shirihai OS; Department of Molecular and Medical Pharmacology, University of California, Los Angeles, California.; Department of Medicine, University of California, Los Angeles, California.
Stiles L; Department of Molecular and Medical Pharmacology, University of California, Los Angeles, California.; Department of Medicine, University of California, Los Angeles, California.
Divakaruni AS; Department of Molecular and Medical Pharmacology, University of California, Los Angeles, California.
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Źródło :
American journal of physiology. Cell physiology [Am J Physiol Cell Physiol] 2021 Jan 01; Vol. 320 (1), pp. C80-C91. Date of Electronic Publication: 2020 Nov 04.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Review
MeSH Terms :
Biological Assay*
Energy Metabolism*
Membrane Potential, Mitochondrial*
Metabolomics*
Organelle Biogenesis*
Mitochondria/*metabolism
Animals ; Biomarkers/metabolism ; Cell Line ; Cell Respiration ; Humans ; Isotope Labeling ; Oxygen Consumption
Czasopismo naukowe
Tytuł :
Stop codon read-through of mammalian MTCH2 leading to an unstable isoform regulates mitochondrial membrane potential.
Autorzy :
Manjunath LE; Department of Biochemistry, Indian Institute of Science, Bengaluru, Karnataka, India.
Singh A; Department of Biochemistry, Indian Institute of Science, Bengaluru, Karnataka, India.
Sahoo S; Department of Biochemistry, Indian Institute of Science, Bengaluru, Karnataka, India.
Mishra A; Department of Biochemistry, Indian Institute of Science, Bengaluru, Karnataka, India.
Padmarajan J; Department of Biochemistry, Indian Institute of Science, Bengaluru, Karnataka, India.
Basavaraju CG; Department of Biochemistry, Indian Institute of Science, Bengaluru, Karnataka, India.
Eswarappa SM; Department of Biochemistry, Indian Institute of Science, Bengaluru, Karnataka, India. Electronic address: .
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Źródło :
The Journal of biological chemistry [J Biol Chem] 2020 Dec 11; Vol. 295 (50), pp. 17009-17026. Date of Electronic Publication: 2020 Oct 07.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Membrane Potential, Mitochondrial*
Protein Biosynthesis*
3' Untranslated Regions/*genetics
Codon, Terminator/*genetics
Mitochondrial Membrane Transport Proteins/*metabolism
RNA, Messenger/*genetics
Animals ; Aorta/cytology ; Aorta/metabolism ; Base Sequence ; CRISPR-Cas Systems/genetics ; Cattle ; Endothelium, Vascular/cytology ; Endothelium, Vascular/metabolism ; HEK293 Cells ; Humans ; Mitochondrial Membrane Transport Proteins/antagonists & inhibitors ; Mitochondrial Membrane Transport Proteins/genetics ; Protein Isoforms ; Ribosomes/metabolism
Czasopismo naukowe
Tytuł :
δ-opioid receptor activation protects against Parkinson's disease-related mitochondrial dysfunction by enhancing PINK1/Parkin-dependent mitophagy.
Autorzy :
Xu Y; Department of Neurosurgery, The First People's Hospital of Changzhou, Changzhou, Jiangsu, China.; Modern Medical Research Center, The Third Affiliated Hospital of Soochow University, Changzhou, Jiangsu, China.; Shanghai Key Laboratory of Acupuncture Mechanism and Acupoint Function, Department of Aeronautics and Astronautics, Fudan University, Shanghai, China.
Zhi F; Department of Neurosurgery, The First People's Hospital of Changzhou, Changzhou, Jiangsu, China.; Modern Medical Research Center, The Third Affiliated Hospital of Soochow University, Changzhou, Jiangsu, China.
Mao J; Department of Neurosurgery, The First People's Hospital of Changzhou, Changzhou, Jiangsu, China.; Modern Medical Research Center, The Third Affiliated Hospital of Soochow University, Changzhou, Jiangsu, China.
Peng Y; Department of Neurosurgery, The First People's Hospital of Changzhou, Changzhou, Jiangsu, China.
Shao N; Department of Neurosurgery, The First People's Hospital of Changzhou, Changzhou, Jiangsu, China.
Balboni G; Department of Life and Environment Sciences, University of Cagliari, Cagliari, Italy.
Yang Y; Department of Neurosurgery, The First People's Hospital of Changzhou, Changzhou, Jiangsu, China.; Modern Medical Research Center, The Third Affiliated Hospital of Soochow University, Changzhou, Jiangsu, China.
Xia Y; Shanghai Key Laboratory of Acupuncture Mechanism and Acupoint Function, Department of Aeronautics and Astronautics, Fudan University, Shanghai, China.
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Źródło :
Aging [Aging (Albany NY)] 2020 Nov 10; Vol. 12 (24), pp. 25035-25059. Date of Electronic Publication: 2020 Nov 10.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Hypoxia/*genetics
Membrane Potential, Mitochondrial/*genetics
Mitochondria/*metabolism
Mitophagy/*genetics
Protein Kinases/*genetics
Receptors, Opioid, delta/*genetics
Ubiquitin-Protein Ligases/*genetics
1-Methyl-4-phenylpyridinium/toxicity ; Animals ; Benzimidazoles/pharmacology ; Gene Knockdown Techniques ; Herbicides/toxicity ; Membrane Potential, Mitochondrial/drug effects ; Mitochondria/drug effects ; Mitophagy/drug effects ; Oligopeptides/pharmacology ; PC12 Cells ; Parkinsonian Disorders/chemically induced ; Parkinsonian Disorders/genetics ; Parkinsonian Disorders/metabolism ; Protein Kinases/drug effects ; Protein Kinases/metabolism ; Rats ; Receptors, Opioid, delta/agonists ; Receptors, Opioid, delta/metabolism ; Superoxides/metabolism ; Ubiquitin-Protein Ligases/drug effects ; Ubiquitin-Protein Ligases/metabolism ; Up-Regulation
Czasopismo naukowe
Tytuł :
Differences in Mitochondrial Membrane Potential Identify Distinct Populations of Human Cardiac Mesenchymal Progenitor Cells.
Autorzy :
Gambini E; Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino-IRCCS, Via Carlo Parea 4, 20138 Milan, Italy.
Martinelli I; Institute of Metabolic and Cardiovascular Diseases (I2MC), Institut National de la Santé et de la Recherche Médicale (INSERM), Université de Toulouse, 31432 Toulouse, France.
Stadiotti I; Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino-IRCCS, Via Carlo Parea 4, 20138 Milan, Italy.
Vinci MC; Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino-IRCCS, Via Carlo Parea 4, 20138 Milan, Italy.
Scopece A; Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino-IRCCS, Via Carlo Parea 4, 20138 Milan, Italy.
Eramo L; Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino-IRCCS, Via Carlo Parea 4, 20138 Milan, Italy.
Sommariva E; Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino-IRCCS, Via Carlo Parea 4, 20138 Milan, Italy.
Resta J; Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino-IRCCS, Via Carlo Parea 4, 20138 Milan, Italy.; Institute of Metabolic and Cardiovascular Diseases (I2MC), Institut National de la Santé et de la Recherche Médicale (INSERM), Université de Toulouse, 31432 Toulouse, France.
Benaouadi S; Institute of Metabolic and Cardiovascular Diseases (I2MC), Institut National de la Santé et de la Recherche Médicale (INSERM), Université de Toulouse, 31432 Toulouse, France.
Cogliati E; Treviso Tissue Bank Foundation, Via Antonio Scarpa 9, 31100 Treviso, Italy.
Paolin A; Treviso Tissue Bank Foundation, Via Antonio Scarpa 9, 31100 Treviso, Italy.
Parini A; Institute of Metabolic and Cardiovascular Diseases (I2MC), Institut National de la Santé et de la Recherche Médicale (INSERM), Université de Toulouse, 31432 Toulouse, France.
Pompilio G; Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino-IRCCS, Via Carlo Parea 4, 20138 Milan, Italy.; Dipartimento di Scienze Cliniche e di Comunità, Università degli Studi di Milano, Via Festa del Perdono 7, 20122 Milan, Italy.
Savagner F; Institute of Metabolic and Cardiovascular Diseases (I2MC), Institut National de la Santé et de la Recherche Médicale (INSERM), Université de Toulouse, 31432 Toulouse, France.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2020 Oct 10; Vol. 21 (20). Date of Electronic Publication: 2020 Oct 10.
Typ publikacji :
Journal Article
MeSH Terms :
Membrane Potential, Mitochondrial*
Mesenchymal Stem Cells/*cytology
Mesenchymal Stem Cells/*metabolism
Myocytes, Cardiac/*cytology
Myocytes, Cardiac/*metabolism
Biomarkers ; Cell Differentiation ; Energy Metabolism ; Gene Expression Regulation ; Humans ; Immunophenotyping ; Mitochondria/genetics ; Mitochondria/metabolism ; Muscle Development/genetics ; Osteogenesis/genetics
Czasopismo naukowe
Tytuł :
Alum triggers infiltration of human neutrophils ex vivo and causes lysosomal destabilization and mitochondrial membrane potential-dependent NET-formation.
Autorzy :
Reithofer M; Institute of Pathophysiology and Allergy Research, Center for Pathophysiology, Infectiology and Immunology, Medical University of Vienna, Vienna, Austria.
Karacs J; Institute of Pathophysiology and Allergy Research, Center for Pathophysiology, Infectiology and Immunology, Medical University of Vienna, Vienna, Austria.
Strobl J; Department of Dermatology, Medical University of Vienna, Vienna, Austria.
Kitzmüller C; Institute of Pathophysiology and Allergy Research, Center for Pathophysiology, Infectiology and Immunology, Medical University of Vienna, Vienna, Austria.
Polak D; Institute of Pathophysiology and Allergy Research, Center for Pathophysiology, Infectiology and Immunology, Medical University of Vienna, Vienna, Austria.
Seif K; Department of Surgery, Medical University of Vienna, Vienna, Austria.
Kamalov M; Institute of Biological Chemistry, Department of Chemistry, University of Vienna, Vienna, Austria.
Becker CFW; Institute of Biological Chemistry, Department of Chemistry, University of Vienna, Vienna, Austria.
Greiner G; Department of Laboratory Medicine, Medical University of Vienna, Vienna, Austria.
Schmetterer K; Department of Laboratory Medicine, Medical University of Vienna, Vienna, Austria.
Stary G; Department of Dermatology, Medical University of Vienna, Vienna, Austria.; Ludwig Boltzmann Institute for Rare and Undiagnosed Diseases, Vienna, Austria.; CeMM Research Center for Molecular Medicine, Vienna, Austria.
Bohle B; Institute of Pathophysiology and Allergy Research, Center for Pathophysiology, Infectiology and Immunology, Medical University of Vienna, Vienna, Austria.
Jahn-Schmid B; Institute of Pathophysiology and Allergy Research, Center for Pathophysiology, Infectiology and Immunology, Medical University of Vienna, Vienna, Austria.
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Źródło :
FASEB journal : official publication of the Federation of American Societies for Experimental Biology [FASEB J] 2020 Oct; Vol. 34 (10), pp. 14024-14041. Date of Electronic Publication: 2020 Aug 29.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Extracellular Traps*
Membrane Potential, Mitochondrial*
Neutrophil Infiltration*
Adjuvants, Immunologic/*pharmacology
Aluminum Hydroxide/*pharmacology
Lysosomes/*metabolism
Neutrophils/*drug effects
Calcium/metabolism ; Cells, Cultured ; Glycolysis ; Humans ; Mitochondria/metabolism ; NADPH Oxidases/metabolism ; Neutrophils/cytology ; Neutrophils/immunology ; Oxidative Phosphorylation
Czasopismo naukowe
Tytuł :
BAFF attenuates oxidative stress-induced cell death by the regulation of mitochondria membrane potential via Syk activation in WiL2-NS B lymphoblasts.
Autorzy :
Park S; Department of Integrative Bioscience and Biotechnology, Sejong University, 209 Neungdong-ro Kwangjin-gu, Seoul, 05006, Republic of Korea.
Jang JW; Department of Integrative Bioscience and Biotechnology, Sejong University, 209 Neungdong-ro Kwangjin-gu, Seoul, 05006, Republic of Korea.
Moon EY; Department of Integrative Bioscience and Biotechnology, Sejong University, 209 Neungdong-ro Kwangjin-gu, Seoul, 05006, Republic of Korea. .
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Źródło :
Scientific reports [Sci Rep] 2020 Jul 16; Vol. 10 (1), pp. 11784. Date of Electronic Publication: 2020 Jul 16.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Death*/genetics
Membrane Potential, Mitochondrial*/genetics
Oxidative Stress*/genetics
B-Cell Activating Factor/*genetics
Syk Kinase/*metabolism
B-Cell Activating Factor/metabolism ; B-Lymphocytes/immunology ; B-Lymphocytes/metabolism ; B-Lymphocytes/pathology ; Caspase 3/metabolism ; Cell Line, Tumor ; Humans ; Hydrogen Peroxide/metabolism ; Matrix Metalloproteinases/metabolism ; Phosphorylation ; Protein Binding ; Protein Kinase Inhibitors/pharmacology ; Reactive Oxygen Species/metabolism ; Syk Kinase/antagonists & inhibitors
Czasopismo naukowe
Tytuł :
Knockdown of sorcin increases HEI-OC1 cell damage induced by cisplatin in vitro.
Autorzy :
Wang D; ENT Institute and Otorhinolaryngology Department, Affiliated Eye and ENT Hospital, Fudan University and Key Laboratory of Hearing Medicine of National Health and Family Planning Commission (NHFPC), Shanghai, 200031, China.
Shi S; ENT Institute and Otorhinolaryngology Department, Affiliated Eye and ENT Hospital, Fudan University and Key Laboratory of Hearing Medicine of National Health and Family Planning Commission (NHFPC), Shanghai, 200031, China.
Hsieh YL; ENT Institute and Otorhinolaryngology Department, Affiliated Eye and ENT Hospital, Fudan University and Key Laboratory of Hearing Medicine of National Health and Family Planning Commission (NHFPC), Shanghai, 200031, China.
Wang J; ENT Institute and Otorhinolaryngology Department, Affiliated Eye and ENT Hospital, Fudan University and Key Laboratory of Hearing Medicine of National Health and Family Planning Commission (NHFPC), Shanghai, 200031, China.
Wang H; ENT Institute and Otorhinolaryngology Department, Affiliated Eye and ENT Hospital, Fudan University and Key Laboratory of Hearing Medicine of National Health and Family Planning Commission (NHFPC), Shanghai, 200031, China.
Wang W; ENT Institute and Otorhinolaryngology Department, Affiliated Eye and ENT Hospital, Fudan University and Key Laboratory of Hearing Medicine of National Health and Family Planning Commission (NHFPC), Shanghai, 200031, China. Electronic address: .
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Źródło :
Archives of biochemistry and biophysics [Arch Biochem Biophys] 2021 Apr 15; Vol. 701, pp. 108752. Date of Electronic Publication: 2021 Mar 03.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Calcium-Binding Proteins/*biosynthesis
Cisplatin/*adverse effects
Hair Cells, Auditory/*metabolism
MAP Kinase Signaling System/*drug effects
Ototoxicity/*metabolism
Animals ; Apoptosis/genetics ; Calcium-Binding Proteins/genetics ; Caspase 3/genetics ; Caspase 3/metabolism ; Cell Line ; Cisplatin/pharmacology ; Gene Knockdown Techniques ; Hair Cells, Auditory/pathology ; MAP Kinase Signaling System/genetics ; Membrane Potential, Mitochondrial/drug effects ; Membrane Potential, Mitochondrial/genetics ; Mice ; Ototoxicity/genetics ; Ototoxicity/pathology ; bcl-2-Associated X Protein/genetics ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł :
Exploiting pyocyanin to treat mitochondrial disease due to respiratory complex III dysfunction.
Autorzy :
Peruzzo R; Department of Biology, University of Padova, Padova, Italy.; Department of Molecular and Cell Biology, University of California, Berkeley, CA, USA.
Corrà S; Department of Biology, University of Padova, Padova, Italy.
Costa R; Department of Biology, University of Padova, Padova, Italy.
Brischigliaro M; Department of Biology, University of Padova, Padova, Italy.
Varanita T; Department of Biology, University of Padova, Padova, Italy.
Biasutto L; CNR Institute of Neuroscience, Padova, Italy.; Department of Biomedical Sciences, University of Padova, Padova, Italy.
Rampazzo C; Department of Biology, University of Padova, Padova, Italy.
Ghezzi D; Unit of Medical Genetics and Neurogenetics, Fondazione IRCCS Istituto Neurologico Carlo Besta, Milano, Italy.; Department of Pathophysiology and Transplantation, University of Milan, Milan, Italy.
Leanza L; Department of Biology, University of Padova, Padova, Italy.
Zoratti M; CNR Institute of Neuroscience, Padova, Italy.; Department of Biomedical Sciences, University of Padova, Padova, Italy.
Zeviani M; Department of Neurosciences, University of Padova, Padova, Italy.; Venetian Institute of Molecular Medicine, Padova, Italy.
De Pittà C; Department of Biology, University of Padova, Padova, Italy.
Viscomi C; Department of Biomedical Sciences, University of Padova, Padova, Italy.
Costa R; Department of Biology, University of Padova, Padova, Italy.
Szabò I; Department of Biology, University of Padova, Padova, Italy. .; CNR Institute of Neuroscience, Padova, Italy. .
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Źródło :
Nature communications [Nat Commun] 2021 Apr 08; Vol. 12 (1), pp. 2103. Date of Electronic Publication: 2021 Apr 08.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Adenosine Triphosphate/*biosynthesis
Electron Transport Complex III/*metabolism
Membrane Proteins/*genetics
Mitochondrial Diseases/*drug therapy
Mitochondrial Proteins/*genetics
Pyocyanine/*pharmacology
ATPases Associated with Diverse Cellular Activities/genetics ; Animals ; Animals, Genetically Modified ; Cell Line ; Drosophila melanogaster ; Electron Transport Complex III/genetics ; Humans ; Membrane Potential, Mitochondrial/drug effects ; Membrane Potential, Mitochondrial/physiology ; Mice ; Mitochondrial Diseases/pathology ; Molecular Chaperones/genetics ; Oxidation-Reduction/drug effects ; Pyocyanine/metabolism ; Reactive Oxygen Species/metabolism ; Zebrafish
Czasopismo naukowe
Tytuł :
Effect of mtDNA depletion from C6 glioma cells and characteristics of the generated C6ρ0 cells.
Autorzy :
Jin Y; Key Laboratory of Tibetan Medicine Research, Northwest Institute of Plateau Biology, Chinese Academy of Sciences, Xining, Qinghai 810008, P.R. China.
Luan G; Key Laboratory of Tibetan Medicine Research, Northwest Institute of Plateau Biology, Chinese Academy of Sciences, Xining, Qinghai 810008, P.R. China.
Li J; Center for Mitochondria and Healthy Aging, College of Life Sciences, Yantai University, Yantai, Shandong 264005, P.R. China.
Wang H; Key Laboratory of Tibetan Medicine Research, Northwest Institute of Plateau Biology, Chinese Academy of Sciences, Xining, Qinghai 810008, P.R. China.
Wang Z; Center for Mitochondria and Healthy Aging, College of Life Sciences, Yantai University, Yantai, Shandong 264005, P.R. China.
Bai B; Key Laboratory of Tibetan Medicine Research, Northwest Institute of Plateau Biology, Chinese Academy of Sciences, Xining, Qinghai 810008, P.R. China.
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Źródło :
Molecular medicine reports [Mol Med Rep] 2021 Apr; Vol. 23 (4). Date of Electronic Publication: 2021 Feb 12.
Typ publikacji :
Journal Article
MeSH Terms :
Brain Neoplasms/*genetics
DNA, Mitochondrial/*genetics
Glioma/*genetics
Mitochondria/*genetics
Adenosine Triphosphate/metabolism ; Animals ; Brain Neoplasms/metabolism ; Brain Neoplasms/pathology ; Cell Line, Tumor ; Cell Proliferation/genetics ; DNA, Mitochondrial/metabolism ; Glioma/metabolism ; Glioma/pathology ; Intracellular Space/metabolism ; Membrane Potential, Mitochondrial/genetics ; Membrane Potential, Mitochondrial/physiology ; Mitochondria/metabolism ; Rats ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Tytuł :
Effect of photobiomodulation on CCC-ESF reactive oxygen species steady-state in high glucose mediums.
Autorzy :
Chen H; School of Life Sciences, Tiangong University, Xiqing District, Binshui West Road, Tianjin, 300387, China.; Academy of Medical Engineering and Translational Medicine, Tianjin University, Tianjin, 300072, China.; Tianjin Photoelectric Detection Technology and Systems Key Laboratory, Tianjin, 300387, China.
Tu M; School of Life Sciences, Tiangong University, Xiqing District, Binshui West Road, Tianjin, 300387, China.
Shi J; School of Life Sciences, Tiangong University, Xiqing District, Binshui West Road, Tianjin, 300387, China.; Tianjin Photoelectric Detection Technology and Systems Key Laboratory, Tianjin, 300387, China.
Wang Y; School of Life Sciences, Tiangong University, Xiqing District, Binshui West Road, Tianjin, 300387, China.
Hou Z; School of Life Sciences, Tiangong University, Xiqing District, Binshui West Road, Tianjin, 300387, China.
Wang J; School of Life Sciences, Tiangong University, Xiqing District, Binshui West Road, Tianjin, 300387, China. .; Tianjin Photoelectric Detection Technology and Systems Key Laboratory, Tianjin, 300387, China. .
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Źródło :
Lasers in medical science [Lasers Med Sci] 2021 Apr; Vol. 36 (3), pp. 555-562. Date of Electronic Publication: 2020 Jul 09.
Typ publikacji :
Journal Article
MeSH Terms :
Low-Level Light Therapy*
Culture Media/*chemistry
Embryo, Mammalian/*cytology
Fibroblasts/*radiation effects
Glucose/*pharmacology
Reactive Oxygen Species/*metabolism
Skin/*cytology
Animals ; Antioxidants/metabolism ; Cell Proliferation/drug effects ; Cell Proliferation/radiation effects ; Cell Survival/drug effects ; Cell Survival/radiation effects ; Dose-Response Relationship, Radiation ; Fibroblasts/cytology ; Fibroblasts/drug effects ; Humans ; Interleukin-1beta/metabolism ; Membrane Potential, Mitochondrial/drug effects ; Membrane Potential, Mitochondrial/radiation effects ; NF-kappa B/metabolism ; Oxidative Stress/drug effects ; Oxidative Stress/radiation effects ; Superoxide Dismutase/metabolism ; Tumor Necrosis Factor-alpha/metabolism ; Wound Healing/drug effects ; Wound Healing/radiation effects
Czasopismo naukowe
Tytuł :
The 3-deoxysappanchalcone induces ROS-mediated apoptosis and cell cycle arrest via JNK/p38 MAPKs signaling pathway in human esophageal cancer cells.
Autorzy :
Kwak AW; Department of Pharmacy, College of Pharmacy, Mokpo National University, Jeonnam 58554, Republic of Korea.
Lee MJ; Department of Pharmacy, College of Pharmacy, Mokpo National University, Jeonnam 58554, Republic of Korea.
Lee MH; College of Korean Medicine, Dongshin University, Naju, Jeonnam 58245, Republic of Korea.
Yoon G; Department of Pharmacy, College of Pharmacy, Mokpo National University, Jeonnam 58554, Republic of Korea.
Cho SS; Department of Pharmacy, College of Pharmacy, Mokpo National University, Jeonnam 58554, Republic of Korea.
Chae JI; Department of Dental Pharmacology, School of Dentistry, Jeonbuk National University, Jeonju 54896, Republic of Korea. Electronic address: .
Shim JH; Department of Pharmacy, College of Pharmacy, Mokpo National University, Jeonnam 58554, Republic of Korea; The China-US (Henan) Hormel Cancer Institute, Zhengzhou, Henan, 450008, PR China; Department of Biomedicine, Health & Life Convergence Sciences, BK21 Four, Biomedical and Healthcare Research Institute, Mokpo National University, Jeonnam 58554, Republic of Korea. Electronic address: .
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Źródło :
Phytomedicine : international journal of phytotherapy and phytopharmacology [Phytomedicine] 2021 Jun; Vol. 86, pp. 153564. Date of Electronic Publication: 2021 Apr 04.
Typ publikacji :
Journal Article
MeSH Terms :
Antineoplastic Combined Chemotherapy Protocols/*pharmacology
Chalcones/*pharmacology
Esophageal Neoplasms/*drug therapy
Esophageal Squamous Cell Carcinoma/*drug therapy
Apoptosis/drug effects ; Cell Cycle Checkpoints ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Endoplasmic Reticulum Stress/drug effects ; Esophageal Neoplasms/metabolism ; Esophageal Neoplasms/pathology ; Esophageal Squamous Cell Carcinoma/metabolism ; Esophageal Squamous Cell Carcinoma/pathology ; Humans ; MAP Kinase Signaling System/drug effects ; Membrane Potential, Mitochondrial/drug effects ; Reactive Oxygen Species/metabolism ; Signal Transduction/drug effects ; p38 Mitogen-Activated Protein Kinases/metabolism
Czasopismo naukowe
Tytuł :
Costunolide attenuates oxygen‑glucose deprivation/reperfusion‑induced mitochondrial‑mediated apoptosis in PC12 cells.
Autorzy :
Meng L; Key Laboratory of Hui Ethnic Medicine Modernization, Ministry of Education, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region 750004, P.R. China.
Ma H; Key Laboratory of Hui Ethnic Medicine Modernization, Ministry of Education, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region 750004, P.R. China.
Meng J; Key Laboratory of Hui Ethnic Medicine Modernization, Ministry of Education, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region 750004, P.R. China.
Li T; Key Laboratory of Hui Ethnic Medicine Modernization, Ministry of Education, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region 750004, P.R. China.
Zhu Y; Key Laboratory of Hui Ethnic Medicine Modernization, Ministry of Education, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region 750004, P.R. China.
Zhao Q; Key Laboratory of Hui Ethnic Medicine Modernization, Ministry of Education, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region 750004, P.R. China.
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Źródło :
Molecular medicine reports [Mol Med Rep] 2021 Jun; Vol. 23 (6). Date of Electronic Publication: 2021 Mar 31.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Hypoxia*
Apoptosis/*drug effects
Glucose/*deficiency
Mitochondria/*metabolism
Neuroprotective Agents/*pharmacology
Sesquiterpenes/*pharmacology
Animals ; Calcium/metabolism ; Caspases/metabolism ; L-Lactate Dehydrogenase/metabolism ; Membrane Potential, Mitochondrial ; PC12 Cells ; Rats
Czasopismo naukowe
Tytuł :
Avermectin induced DNA damage to the apoptosis and autophagy in human lung epithelial A549 cells.
Autorzy :
Diao L; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai 200237, China.
Tang N; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai 200237, China.
Zhang C; Department of Pathology, UT southwestern Medical Center, Dallas, TX 75390, United States.
Cheng J; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai 200237, China.
Zhang Z; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai 200237, China.
Wang S; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai 200237, China.
Wu C; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai 200237, China.
Zhang L; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai 200237, China.
Tao L; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai 200237, China.
Li Z; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai 200237, China.
Zhang Y; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai 200237, China. Electronic address: .
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Źródło :
Ecotoxicology and environmental safety [Ecotoxicol Environ Saf] 2021 Jun 01; Vol. 215, pp. 112129. Date of Electronic Publication: 2021 Mar 16.
Typ publikacji :
Journal Article
MeSH Terms :
Insecticides/*toxicity
Ivermectin/*analogs & derivatives
A549 Cells ; Apoptosis/drug effects ; Autophagy/drug effects ; Cell Proliferation/drug effects ; Cytochromes c/metabolism ; DNA Damage/drug effects ; Humans ; Ivermectin/toxicity ; Lung/metabolism ; Membrane Potential, Mitochondrial/drug effects ; Mitochondria/drug effects ; Proto-Oncogene Proteins c-bcl-2
Czasopismo naukowe
Tytuł :
A Combined Antitumor Strategy Mediated by a New Targeted Nanosystem to Hepatocellular Carcinoma.
Autorzy :
Farinha D; CNC - Center for Neuroscience and Cell Biology (CNC), University of Coimbra, Coimbra, Portugal.; Institute for Interdisciplinary Research (IIIUC), University of Coimbra, Coimbra, Portugal.
Migawa M; Ionis Pharmaceuticals Inc, Carlsbad, USA.
Sarmento-Ribeiro A; CNC - Center for Neuroscience and Cell Biology (CNC), University of Coimbra, Coimbra, Portugal.; Laboratory of Oncobiology and Hematology (LOH) and University Clinic of Hematology, Faculty of Medicine, University of Coimbra, Coimbra, Portugal.; Clinical Hematology Department, Centro Hospitalar Universitário de Coimbra (CHUC), Coimbra, Portugal.
Faneca H; CNC - Center for Neuroscience and Cell Biology (CNC), University of Coimbra, Coimbra, Portugal.; Institute for Interdisciplinary Research (IIIUC), University of Coimbra, Coimbra, Portugal.
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Źródło :
International journal of nanomedicine [Int J Nanomedicine] 2021 May 18; Vol. 16, pp. 3385-3405. Date of Electronic Publication: 2021 May 18 (Print Publication: 2021).
Typ publikacji :
Journal Article
MeSH Terms :
Carcinoma, Hepatocellular/*drug therapy
Liver Neoplasms/*drug therapy
Molecular Targeted Therapy/*methods
Nanomedicine/*methods
Animals ; Apoptosis/drug effects ; Carcinoma, Hepatocellular/pathology ; Caspases/metabolism ; Cell Line, Tumor ; Cell Survival/drug effects ; Humans ; Liver Neoplasms/pathology ; Membrane Potential, Mitochondrial/drug effects ; Sorafenib/pharmacology ; Sorafenib/therapeutic use
Czasopismo naukowe
Tytuł :
Improved bioavailability and anticancer efficacy of Hesperetin on breast cancer via a self-assembled rebaudioside A nanomicelles system.
Autorzy :
Wang J; Department of Pharmacy, College of Chemical Engineering, Qingdao University of Science and Technology, Qingdao 266042, China.
Li Q; Department of Pharmacy, College of Chemical Engineering, Qingdao University of Science and Technology, Qingdao 266042, China.
Chen Z; Department of Pharmacy, College of Chemical Engineering, Qingdao University of Science and Technology, Qingdao 266042, China.
Qi X; Department of Pharmacy, College of Chemical Engineering, Qingdao University of Science and Technology, Qingdao 266042, China.
Wu X; Department of Pharmacy, College of Chemical Engineering, Qingdao University of Science and Technology, Qingdao 266042, China.
Di G; School of Basic Medicine, Qingdao University, Qingdao 266071, China.
Fan J; Department of Pharmaceutical Analysis, School of Pharmacy, Nanjing Medical University, Nanjing 211166, China.
Guo C; Department of Pharmacy, College of Chemical Engineering, Qingdao University of Science and Technology, Qingdao 266042, China. Electronic address: .
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Źródło :
Toxicology and applied pharmacology [Toxicol Appl Pharmacol] 2021 May 15; Vol. 419, pp. 115511. Date of Electronic Publication: 2021 Apr 02.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Drug Carriers*
Nanoparticles*
Antineoplastic Agents, Phytogenic/*pharmacology
Breast Neoplasms/*drug therapy
Diterpenes, Kaurane/*chemistry
Hesperidin/*pharmacology
Administration, Oral ; Animals ; Antineoplastic Agents, Phytogenic/administration & dosage ; Antineoplastic Agents, Phytogenic/chemistry ; Antineoplastic Agents, Phytogenic/pharmacokinetics ; Apoptosis/drug effects ; Biological Availability ; Breast Neoplasms/metabolism ; Breast Neoplasms/pathology ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Drug Compounding ; Drug Liberation ; Female ; Hesperidin/administration & dosage ; Hesperidin/chemistry ; Hesperidin/pharmacokinetics ; Humans ; Membrane Potential, Mitochondrial/drug effects ; Mice ; Micelles ; Phosphatidylinositol 3-Kinases/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Rats, Sprague-Dawley ; Reactive Oxygen Species/metabolism ; Signal Transduction ; Solubility ; Tissue Distribution ; Tumor Burden/drug effects
Czasopismo naukowe
Tytuł :
Roles of ERK/Akt signals in mitochondria-dependent and endoplasmic reticulum stress-triggered neuronal cell apoptosis induced by 4-methyl-2,4-bis(4-hydroxyphenyl)pent-1-ene, a major active metabolite of bisphenol A.
Autorzy :
Huang CF; School of Chinese Medicine, College of Chinese Medicine, China Medical University, Taichung, 404, Taiwan; Department of Nursing, College of Medical and Health Science, Asia University, Taichung, 413, Taiwan.
Liu SH; Institute of Toxicology, College of Medicine, National Taiwan University, Taipei, 100, Taiwan.
Su CC; Department of Otorhinolaryngology, Head and Neck Surgery, Changhua Christian Hospital, Changhua County, 500, Taiwan; School of Medicine, Kaohsiung Medical University, Kaohsiung, 807, Taiwan.
Fang KM; Department of Otolaryngology, Far Eastern Memorial Hospital, New Taipei City, 220, Taiwan.
Yen CC; Department of Occupational Safety and Health, College of Health Care and Management, Chung Shan Medical University, Taichung, 402, Taiwan.
Yang CY; Department of Surgery, National Taiwan University Hospital, and Department of Surgery, College of Medicine, National Taiwan University, Taipei, 100, Taiwan.
Tang FC; Department of Occupational Medicine, Changhua Christian Hospital, Changhua County, 500, Taiwan.
Liu JM; Division of Urology, Department of Surgery, Taoyuan General Hospital, Ministry of Health and Welfare, Taoyuan, 330, Taiwan.
Wu CC; Department of Public Health, China Medical University, Taichung, 404, Taiwan.
Lee KI; Department of Emergency, Taichung Tzu Chi Hospital, Buddhist Tzu Chi Medical Foundation, Taichung, 427, Taiwan. Electronic address: .
Chen YW; Department of Physiology and Graduate Institute of Basic Medical Science, School of Medicine, College of Medicine, China Medical University, Taichung, 404, Taiwan. Electronic address: .
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Źródło :
Toxicology [Toxicology] 2021 May 15; Vol. 455, pp. 152764. Date of Electronic Publication: 2021 Mar 23.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Benzhydryl Compounds/*toxicity
Endoplasmic Reticulum Stress/*drug effects
Neurons/*drug effects
Phenols/*toxicity
Animals ; Benzhydryl Compounds/administration & dosage ; Cell Line, Tumor ; Cytochromes c/drug effects ; Dose-Response Relationship, Drug ; MAP Kinase Signaling System/drug effects ; Membrane Potential, Mitochondrial/drug effects ; Mice ; Mitochondria/metabolism ; Neurons/pathology ; Phenols/administration & dosage ; Proto-Oncogene Proteins c-akt/metabolism
Czasopismo naukowe
Tytuł :
A novel 1-((3-(2-toluyl)-4,5-dihydroisoxazol-5-yl)methyl)-4-(trifluoromethyl)pyrimidin-2(1H)-one activates intrinsic mitochondria-dependent pathway and decreases angiogenesis in PC-3 cells.
Autorzy :
Silva Dos Santos M; Department of Basic Health Sciences, Laboratory of Genetic Toxicology, Federal University of Health Sciences of Porto Alegre (UFCSPA), Porto Alegre, RS, Brazil.
Lobo MM; Chemical Core Heterocycles (NUQUIMHE), Chemistry Department, Federal University of Santa Maria, Santa Maria (UFSM), RS, Brazil.
Amaral SS; Department of Pharmacosciences, Federal University of Health Sciences of Porto Alegre (UFCSPA), Porto Alegre, RS, Brazil.
Zanatta N; Chemical Core Heterocycles (NUQUIMHE), Chemistry Department, Federal University of Santa Maria, Santa Maria (UFSM), RS, Brazil.
Viau CM; Department of Basic Health Sciences, Laboratory of Genetic Toxicology, Federal University of Health Sciences of Porto Alegre (UFCSPA), Porto Alegre, RS, Brazil. Electronic address: .
Saffi J; Department of Basic Health Sciences, Laboratory of Genetic Toxicology, Federal University of Health Sciences of Porto Alegre (UFCSPA), Porto Alegre, RS, Brazil. Electronic address: .
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Źródło :
European journal of pharmacology [Eur J Pharmacol] 2021 May 15; Vol. 899, pp. 174028. Date of Electronic Publication: 2021 Mar 13.
Typ publikacji :
Journal Article
MeSH Terms :
Neovascularization, Pathologic*
Angiogenesis Inhibitors/*pharmacology
Apoptosis/*drug effects
Mitochondria/*drug effects
Prostatic Neoplasms/*drug therapy
Pyrimidinones/*pharmacology
Ataxia Telangiectasia Mutated Proteins/metabolism ; DNA Damage ; Down-Regulation ; G2 Phase Cell Cycle Checkpoints/drug effects ; Histones/metabolism ; Humans ; Lipid Peroxidation/drug effects ; Male ; Membrane Potential, Mitochondrial/drug effects ; Mitochondria/metabolism ; Mitochondria/pathology ; PC-3 Cells ; Phosphorylation ; Prostatic Neoplasms/metabolism ; Prostatic Neoplasms/pathology ; Reactive Oxygen Species/metabolism ; Signal Transduction ; Topoisomerase I Inhibitors/pharmacology ; Vascular Endothelial Growth Factor A/metabolism
Czasopismo naukowe
Tytuł :
Novel arylidene malonate derivative, KM-34, showed neuroprotective effects on in vitro and in vivo models of ischemia/reperfusion.
Autorzy :
Ramírez-Sánchez J; Laboratorio de Neurofarmacología Experimental, Centro de Investigación y Desarrollo de Medicamentos, La Habana, 10600, Cuba. Electronic address: .
Wong-Guerra M; Laboratorio de Neurofarmacología Experimental, Centro de Investigación y Desarrollo de Medicamentos, La Habana, 10600, Cuba.
Fonseca-Fonseca LA; Laboratorio de Neurofarmacología Experimental, Centro de Investigación y Desarrollo de Medicamentos, La Habana, 10600, Cuba.
Simões-Pires EN; Programa de Pós-graduação em Bioquímica, Departamento de Bioquímica, ICBS, Universidade Federal do Rio Grande do Sul, Porto Alegre, RS, 90035-003, Brazil.
García-Pupo L; Laboratorio de Neurofarmacología Experimental, Centro de Investigación y Desarrollo de Medicamentos, La Habana, 10600, Cuba.
Ochoa-Rodríguez E; Laboratorio de Síntesis Orgánica, Facultad de Química, Universidad de La Habana, La Habana, 10400, Cuba.
Verdecia-Reyes Y; Laboratorio de Síntesis Orgánica, Facultad de Química, Universidad de La Habana, La Habana, 10400, Cuba.
Delgado-Hernández R; Laboratorio de Neurofarmacología Experimental, Centro de Investigación y Desarrollo de Medicamentos, La Habana, 10600, Cuba; Guest professor at Universidad de Santander (UDES), Bucaramanga, 680003, Colombia.
Salbego C; Programa de Pós-graduação em Bioquímica, Departamento de Bioquímica, ICBS, Universidade Federal do Rio Grande do Sul, Porto Alegre, RS, 90035-003, Brazil; Departamento de Bioquímica, PPG em Bioquímica, PPG em Educação em Ciência, Instituto de Ciências Básicas da Saúde, Universidade Federal do Rio Grande do Sul, Porto Alegre, RS, 90035-003, Brazil.
Souza DO; Programa de Pós-graduação em Bioquímica, Departamento de Bioquímica, ICBS, Universidade Federal do Rio Grande do Sul, Porto Alegre, RS, 90035-003, Brazil; Departamento de Bioquímica, PPG em Bioquímica, PPG em Educação em Ciência, Instituto de Ciências Básicas da Saúde, Universidade Federal do Rio Grande do Sul, Porto Alegre, RS, 90035-003, Brazil.
Pardo-Andreu GL; Centro de Estudio para las Investigaciones y Evaluaciones Biológicas, Instituto de Farmacia y Alimentos, La Habana, 13600, Cuba.
Nuñez-Figueredo Y; Laboratorio de Neurofarmacología Experimental, Centro de Investigación y Desarrollo de Medicamentos, La Habana, 10600, Cuba.
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Źródło :
European journal of pharmacology [Eur J Pharmacol] 2021 May 15; Vol. 899, pp. 174025. Date of Electronic Publication: 2021 Mar 17.
Typ publikacji :
Journal Article
MeSH Terms :
Brain/*drug effects
Catechols/*pharmacology
Infarction, Middle Cerebral Artery/*drug therapy
Neuroprotective Agents/*pharmacology
Reperfusion Injury/*prevention & control
Animals ; Behavior, Animal/drug effects ; Brain/metabolism ; Brain/pathology ; Brain/physiopathology ; Disease Models, Animal ; Infarction, Middle Cerebral Artery/metabolism ; Infarction, Middle Cerebral Artery/pathology ; Infarction, Middle Cerebral Artery/physiopathology ; Locomotion/drug effects ; Male ; Membrane Potential, Mitochondrial/drug effects ; Mitochondria/drug effects ; Mitochondria/metabolism ; Mitochondria/pathology ; Mitochondrial Swelling/drug effects ; Motor Activity/drug effects ; Oxidative Stress/drug effects ; Rats, Wistar ; Reperfusion Injury/metabolism ; Reperfusion Injury/pathology ; Reperfusion Injury/physiopathology ; Tissue Culture Techniques
Czasopismo naukowe

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