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Tytuł :
Iron is neurotoxic in retinal detachment and transferrin confers neuroprotection
Autorzy :
Daruich, Alejandra
Le Rouzic, Quentin
Jonet, Laurent
Naud, Marie-Christine
Kowalczuk, Laura
Pournaras, Jean-Antoine
Boatright, Jeffrey H.
Thomas, Aurélien
Turck, Natacha
Moulin, Alexandre
Behar-Cohen, Francine
Picard, Emilie
Pokaż więcej
Temat :
Aged
Animals
Apoptosis/drug effects
Disease Models, Animal
Eye Diseases, Hereditary/metabolism
Eye Diseases, Hereditary/surgery
Female
Humans
Iron/metabolism
Iron/pharmacology
Iron/toxicity
Mice
Mice, Inbred C57BL
Mice, Transgenic
Middle Aged
Necrosis
Neuroprotection
Photoreceptor Cells, Vertebrate/metabolism
Rats
Rats, Long-Evans
Rats, Wistar
Retina/metabolism
Retinal Detachment/metabolism
Retinal Detachment/surgery
Retinal Pigment Epithelium/metabolism
Subretinal Fluid/metabolism
Transferrin/genetics
Transferrin/metabolism
MESH: Aged
MESH: Animals
MESH: Female
MESH: Humans
MESH: Mice, Transgenic
MESH: Iron
MESH: Mice
MESH: Mice, Inbred C57BL
MESH: Middle Aged
MESH: Necrosis
MESH: Neuroprotection
MESH: Photoreceptor Cells, Vertebrate
MESH: Retina
MESH: Rats
MESH: Rats, Long-Evans
MESH: Rats, Wistar
MESH: Retinal Detachment
MESH: Apoptosis
MESH: Retinal Pigment Epithelium
MESH: Subretinal Fluid
MESH: Transferrin
MESH: Disease Models, Animal
MESH: Eye Diseases, Hereditary
[SDV.BBM]Life Sciences [q-bio]/Biochemistry, Molecular Biology
Multidisciplinary
Cancer research
Transferrin
chemistry.chemical_classification
chemistry
Retinal
chemistry.chemical_compound
Programmed cell death
Retinal pigment epithelium
medicine.anatomical_structure
medicine
business.industry
business
Necroptosis
Retina
Retinal detachment
medicine.disease
Źródło :
Science advances, vol. 5, no. 1, pp. eaau9940
Science Advances
Science Advances, American Association for the Advancement of Science (AAAS), 2019, 5 (1), pp.eaau9940. ⟨10.1126/sciadv.aau9940⟩
Crossref
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5fe9c3ad1e9397bf2e0403e52debf492
https://serval.unil.ch/resource/serval:BIB_66A0081D7AD0.P001/REF.pdf
Tytuł :
Necroptosis microenvironment directs lineage commitment in liver cancer
Autorzy :
Seehawer, Marco
Heinzmann, Florian
D’Artista, Luana
Harbig, Jule
Roux, Pierre-François
Hoenicke, Lisa
Dang, Hien
Klotz, Sabrina
Robinson, Lucas
Doré, Grégory
Rozenblum, Nir
Kang, Tae-Won
Chawla, Rishabh
Buch, Thorsten
Vucur, Mihael
Roth, Mareike
Zuber, Johannes
Luedde, Tom
Sipos, Bence
Longerich, Thomas
Heikenwalder, Mathias
Wang, Xin Wei
Bischof, Oliver
Zender, Lars
Pokaż więcej
Temat :
Cancer epigenetics
Cancer microenvironment
MESH: Animals
MESH: Apoptosis*/genetics
MESH: Cytokines/metabolism
MESH: DNA Transposable Elements/genetics
MESH: DNA-Binding Proteins/genetics
MESH: DNA-Binding Proteins/metabolism
MESH: Disease Models, Animal
MESH: Epigenesis, Genetic/genetics
MESH: Female
MESH: Gene Expression Profiling
MESH: Genes, myc
MESH: Genes, ras
MESH: Carcinogenesis/genetics
MESH: Hepatocytes/metabolism
MESH: Hepatocytes/pathology
MESH: Humans
MESH: Liver Neoplasms/genetics
MESH: Liver Neoplasms/pathology
MESH: Male
MESH: Mice
MESH: Mosaicism
MESH: Necrosis*/genetics
MESH: Proto-Oncogene Proteins c-akt/genetics
MESH: Carcinoma, Hepatocellular/genetics
MESH: T-Box Domain Proteins/genetics
MESH: T-Box Domain Proteins/metabolism
MESH: Transcription Factors/genetics
MESH: Transcription Factors/metabolism
MESH: Tumor Microenvironment
MESH: Carcinoma, Hepatocellular/pathology
MESH: Cell Differentiation
MESH: Cell Lineage*/genetics
MESH: Cholangiocarcinoma/genetics
MESH: Cholangiocarcinoma/pathology
MESH: Cyclin-Dependent Kinase Inhibitor p16/deficiency
[SDV.CAN]Life Sciences [q-bio]/Cancer
Article
Institute of Laboratory Animal Science
570 Life sciences
biology
610 Medicine & health
Multidisciplinary
Epigenome
Cancer research
Biology
Gene expression profiling
Cellular differentiation
Hepatic stellate cell
Liver cancer
medicine.disease
medicine
Tumor microenvironment
Necroptosis
Źródło :
Nature
Nature, Nature Publishing Group, 2018, 562 (7725), pp.69-75. ⟨10.1038/s41586-018-0519-y⟩
Seehawer, Marco; Heinzmann, Florian; D’Artista, Luana; Harbig, Jule; Roux, Pierre-François; Hoenicke, Lisa; Dang, Hien; Klotz, Sabrina; Robinson, Lucas; Doré, Grégory; Rozenblum, Nir; Kang, Tae-Won; Chawla, Rishabh; Buch, Thorsten; Vucur, Mihael; Roth, Mareike; Zuber, Johannes; Luedde, Tom; Sipos, Bence; Longerich, Thomas; Heikenwälder, Mathias; Wang, Xin Wei; Bischof, Oliver; Zender, Lars (2018). Necroptosis microenvironment directs lineage commitment in liver cancer. Nature, 562(7725):69-75.
Crossref
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::02caf225f5c633bfb7fa5ddb2d717963
https://hal.archives-ouvertes.fr/hal-02348982
Tytuł :
Comparative Study of Injury Models for Studying Muscle Regeneration in Mice
Autorzy :
Hardy, David
Besnard, Aurore
Latil, Mathilde
Jouvion, Grégory
Briand, David
Thépenier, Cédric
Pascal, Quentin
Guguin, Aurélie
Gayraud-Morel, Barbara
Cavaillon, Jean-Marc
Tajbakhsh, Shahragim
Rocheteau, Pierre
Chrétien, Fabrice
Pokaż więcej
Temat :
MESH: Animals
MESH: Barium Compounds
MESH: Macrophages
MESH: Mice
MESH: Mice, Inbred C57BL
MESH: Mice, Transgenic
MESH: Models, Animal
MESH: Muscle Development
MESH: Muscle, Skeletal
MESH: Myoblasts
MESH: Necrosis
MESH: Neovascularization, Physiologic
MESH: Chlorides
MESH: Regeneration
MESH: Satellite Cells, Skeletal Muscle
MESH: Stem Cells
MESH: Vascular Endothelial Growth Factor Receptor-2
MESH: Cobra Cardiotoxin Proteins
MESH: Cold Injury
MESH: Cytokines
MESH: Elapid Venoms
MESH: Fibrosis
MESH: Freezing
MESH: Green Fluorescent Proteins
[SDV]Life Sciences [q-bio]
[SDV.BC]Life Sciences [q-bio]/Cellular Biology
lcsh:Medicine
lcsh:R
lcsh:Science
lcsh:Q
MESH : Animals
MESH : Barium Compounds
MESH : Macrophages
MESH : Mice
MESH : Mice, Inbred C57BL
MESH : Mice, Transgenic
MESH : Models, Animal
MESH : Muscle Development
MESH : Muscle, Skeletal
MESH : Myoblasts
MESH : Necrosis
MESH : Neovascularization, Physiologic
MESH : Chlorides
MESH : Regeneration
MESH : Satellite Cells, Skeletal Muscle
MESH : Stem Cells
MESH : Vascular Endothelial Growth Factor Receptor-2
MESH : Cobra Cardiotoxin Proteins
MESH : Cold Injury
MESH : Cytokines
MESH : Elapid Venoms
MESH : Fibrosis
MESH : Freezing
MESH : Green Fluorescent Proteins
[ SDV ] Life Sciences [q-bio]
[ SDV.BC ] Life Sciences [q-bio]/Cellular Biology
Multidisciplinary
Myocyte
Stem cell
Biology
Skeletal muscle
medicine.anatomical_structure
medicine
Poison control
Muscle tissue
Regenerative medicine
Cardiotoxin
Regeneration (biology)
Pathology
medicine.medical_specialty
Research Article
Biology and Life Sciences
Developmental Biology
Morphogenesis
Regeneration
Muscle Regeneration
Organism Development
Immunology
Immune Response
Inflammation
Medicine and Health Sciences
Pathology and Laboratory Medicine
Signs and Symptoms
Anatomy
Histology
Physiology
Immune Physiology
Cytokines
Immune System
Innate Immune System
Molecular Development
Biological Tissue
Muscle Tissue
Cell Biology
Cellular Types
Animal Cells
Blood Cells
White Blood Cells
Macrophages
Immune Cells
Neurology
Neurobiology of Disease and Regeneration
Musculoskeletal System
Muscles
Skeletal Muscles
Źródło :
PLoS ONE
PLoS ONE, Public Library of Science, 2016, 11 (1), pp.e0147198. ⟨10.1371/journal.pone.0147198⟩
PLoS ONE, Vol 11, Iss 1, p e0147198 (2016)
PLoS ONE, Public Library of Science, 2016, 11 (1), pp.e0147198. 〈10.1371/journal.pone.0147198〉
Crossref
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a9a59bf3798e0703ed6ee10e60a0ec30
https://hal-pasteur.archives-ouvertes.fr/pasteur-01447903
Tytuł :
CT description of pancreatic and peripancreatic collections in the first month of acute pancreatitis: an interobserver variability study using the revised Atlanta classification 2012
Autorzy :
Badat, Neesmah
Pokaż więcej
Temat :
Severity
Acute pancreatitis
Computed tomography
Nécrose
Sévérité
Classification
Pancréatite aiguë
Scanner
Collection
MESH: Pancreatitis, Acute Necrotizing
MESH: Necrosis
MESH: Tomography, X-Ray Computed
[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
Źródło :
Médecine humaine et pathologie. 2017
Dostępność :
https://explore.openaire.eu/search/publication?articleId=od______2592::6b9e205692e8e2d948c0b9d8d9ab538e
https://dumas.ccsd.cnrs.fr/dumas-01731433
Tytuł :
Prognostic and Therapeutic Markers in Chordomas: A Study of 287 Tumors
Autorzy :
Tauziède-Espariat, Arnault
Bresson, Damien
Polivka, Marc
Bouazza, Schahrazed
Labrousse, François
Aronica, Eleonora
Pretet, Jean-Luc
Projetti, Fabrice
Herman, Philippe
Salle, Henri
Monnien, Franck
Valmary-Degano, Séverine
Laquerrière, Annie
Pocard, Marc
Chaigneau, Loïc
Isambert, Nicolas
Aubriot-Lorton, Marie-Hélène
Feuvret, Loïc
George, Bernard
Froelich, Sébastien
Adle-Biassette, Homa
Pokaż więcej
Temat :
Cellular and Molecular Neuroscience
Clinical Neurology
Neurology
General Medicine
Pathology and Forensic Medicine
Medicine
business.industry
business
KRAS
medicine.disease_cause
Pathology
medicine.medical_specialty
Survival analysis
Chordoma
medicine.disease
Vascular endothelial growth factor
chemistry.chemical_compound
chemistry
Oncology
Retrospective cohort study
Internal medicine
Univariate analysis
PI3K/AKT/mTOR pathway
Epidermal growth factor receptor
biology.protein
biology
MESH: Adolescent
MESH: Adult
MESH: Neoplasm Grading
MESH: Neoplasm, Residual
MESH: Aged
MESH: Prognosis
MESH: Protons
MESH: Retrospective Studies
MESH: Survival Analysis
MESH: Young Adult
MESH: Apoptosis
MESH: Biomarkers, Tumor
MESH: Female
MESH: Child
MESH: Child, Preschool
MESH: Chordoma
MESH: Cohort Studies
MESH: Disease-Free Survival
MESH: Humans
MESH: Immunohistochemistry
MESH: Infant
MESH: Infant, Newborn
MESH: Male
MESH: Middle Aged
MESH: Necrosis
[SDV.CAN]Life Sciences [q-bio]/Cancer
neoplasms
Źródło :
Journal of Neuropathology and Experimental Neurology, 75(2), 111-120. Lippincott Williams & Wilkins
Crossref
Journal of Neuropathology and Experimental Neurology
Journal of Neuropathology and Experimental Neurology, Lippincott, Williams & Wilkins, 2016, 75 (2), pp.111-120. ⟨10.1093/jnen/nlv010⟩
Journal of neuropathology and experimental neurology, 75(2), 111-120. Lippincott Williams and Wilkins
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8cc84c5470197e316ef52a52a1d34dc1
https://dare.uva.nl/personal/pure/en/publications/prognostic-and-therapeutic-markers-in-chordomas-a-study-of-287-tumors(eb36926b-f70b-44d6-b4b6-0963120328e5).html
Tytuł :
Dual Effects of Exercise in Dysferlinopathy
Autorzy :
Marie Villemeur
Fabrice Chrétien
Romain K. Gherardi
François-Jérôme Authier
Alice Marchand
Nathalie Bourg
Isabelle Richard
Olivier Biondi
Pokaż więcej
Temat :
Pathology and Forensic Medicine
Internal medicine
medicine.medical_specialty
medicine
Eccentric
Isometric exercise
Sarcolemma
Dysferlin
biology.protein
biology
Myopathy
medicine.symptom
Anatomy
Endocrinology
Dysferlinopathy
medicine.disease
Plasma membrane repair
Muscular dystrophy
MESH: Aging
MESH: Animals
MESH: Mice, Mutant Strains
MESH: Microscopy, Electron
MESH: Muscle Contraction
MESH: Muscle Fibers, Skeletal
MESH: Muscle Strength
MESH: Muscle, Skeletal
MESH: Muscular Dystrophies, Limb-Girdle
MESH: Necrosis
MESH: Physical Conditioning, Animal
MESH: Quadriceps Muscle
MESH: Running
MESH: Swimming
MESH: Cell Membrane
MESH: Dysferlin
MESH: Locomotion
MESH: Membrane Proteins
MESH: Mice
[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry, Molecular Biology/Molecular biology
[SDV.BIO]Life Sciences [q-bio]/Biotechnology
[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human genetics
Źródło :
Crossref
American Journal of Pathology
American Journal of Pathology, American Society for Investigative Pathology, 2013, 182 (6), pp.2298-2309. ⟨10.1016/j.ajpath.2013.02.045⟩
The American journal of pathology
The American journal of pathology, 2013, 182 (6), pp.2298-2309. ⟨10.1016/j.ajpath.2013.02.045⟩
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7f14c4908d054247dee93a64506c6a5f
https://api.elsevier.com/content/article/PII:S0002944013002526?httpAccept=text/xml
Tytuł :
Necrosis- and apoptosis-related Met cleavages have divergent functional consequences
Autorzy :
Doublet L
David Tulasne
Marie-Christine Copin
Dansin E
Rémi Montagne
Nadège Debreuck
Catherine Leroy
Delhem N
Baranzelli A
Henri Porte
Alessandro Furlan
Hervé Drobecq
Berbon M
Pokaż więcej
Temat :
MESH: ADAM Proteins/metabolism
MESH: Animals
MESH: Cell Survival
MESH: Enzyme Activation
MESH: Epithelial Cells/metabolism
MESH: HEK293 Cells
MESH: Hepatocyte Growth Factor/metabolism
MESH: Humans
MESH: Ionomycin/pharmacology
MESH: Lung Neoplasms/pathology
MESH: Mice
MESH: Necrosis/pathology
MESH: Apoptosis/drug effects
MESH: Neoplasm Metastasis/pathology
MESH: Proto-Oncogene Proteins c-met/metabolism
MESH: RNA Interference
MESH: RNA, Small Interfering
MESH: Signal Transduction
MESH: Apoptosis/physiology
MESH: Calcium/metabolism
MESH: Calpain/metabolism
MESH: Caspases/metabolism
MESH: Cell Line, Tumor
MESH: Cell Movement
MESH: Cell Proliferation
[SDV]Life Sciences [q-bio]
[SDV.BC]Life Sciences [q-bio]/Cellular Biology
Cancer Research
Cell Biology
Cellular and Molecular Neuroscience
Immunology
Programmed cell death
Hepatocyte growth factor
medicine.drug
medicine
Apoptosis
Cell biology
Receptor tyrosine kinase
biology.protein
biology
Proto-Oncogene Proteins c-met
Acetylation
Caspase
Signal transduction
Molecular biology
Original Article
Źródło :
Cell Death and Disease
Cell Death and Disease, Nature Publishing Group, 2015, 6 (5), pp.e1769-e1769. ⟨10.1038/cddis.2015.132⟩
Crossref
Cell Death & Disease
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0d293751c28a7934bbe5db4e461dd6cb
https://hal.archives-ouvertes.fr/hal-02413945
Tytuł :
PARP-1 is involved in autophagy induced by DNA damage
Autorzy :
Muñoz-Gámez, José Antonio
Rodríguez-Vargas, José Manuel
Quiles-Pérez, Rosa
Aguilar-Quesada, Rocío
Martín-Oliva, David
Murcia, Gilbert
Murcia, Josiane
Almendros, Antonio
Almodóvar, Mariano
Oliver, F Javier
Pokaż więcej
Temat :
Cell Biology
Molecular Biology
Autophagy
Cathepsin B
Programmed cell death
Transfection
PARP inhibitor
Cell biology
Biology
DNA damage
Poly ADP ribose polymerase
Cell
medicine.anatomical_structure
medicine
MESH: 1-Naphthylamine
MESH: 3T3 Cells
MESH: DNA Damage
MESH: Doxorubicin
MESH: Enzyme Activation
MESH: Gene Deletion
MESH: Mice
MESH: Microtubule-Associated Proteins
MESH: Mitochondria
MESH: Models, Biological
MESH: NAD
MESH: Naphthalimides
MESH: Adenosine Triphosphate
MESH: Necrosis
MESH: Poly(ADP-ribose) Polymerase Inhibitors
MESH: Poly(ADP-ribose) Polymerases
MESH: Protein Kinases
MESH: Proteins
MESH: Quinolones
MESH: Subcellular Fractions
MESH: TOR Serine-Threonine Kinases
MESH: Up-Regulation
MESH: Animals
MESH: Apoptosis
MESH: Apoptosis Regulatory Proteins
MESH: Autophagy
MESH: Autophagy-Related Protein 5
MESH: Beclin-1
MESH: Cell Survival
[SDV.BBM]Life Sciences [q-bio]/Biochemistry, Molecular Biology
[SDV.BBM.BS]Life Sciences [q-bio]/Biochemistry, Molecular Biology/Biomolecules [q-bio.BM]
Źródło :
Crossref
Autophagy
Autophagy, Taylor & Francis, 2009, 5 (1), pp.61-74. ⟨10.4161/auto.5.1.7272⟩
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b6af02a2a797b5a9e42b1b711d42af94
Tytuł :
RIPK1 both positively and negatively regulates RIPK3 oligomerization and necroptosis
Autorzy :
Orozco, S
Yatim, N
Werner, M R
Tran, H
Gunja, S Y
Tait, S WG
Albert, A
Green, D R
Oberst, A
Pokaż więcej
Temat :
MESH: Apoptosis/physiology
MESH: Caspase 8/metabolism
MESH: Protein Kinases/genetics
MESH: Protein Kinases/metabolism
MESH: Protein Multimerization
MESH: Protein Structure, Tertiary
MESH: RNA Interference
MESH: RNA, Small Interfering
MESH: Receptor-Interacting Protein Serine-Threonine Kinases/antagonists & inhibitors
MESH: Receptor-Interacting Protein Serine-Threonine Kinases/genetics
MESH: Receptor-Interacting Protein Serine-Threonine Kinases/metabolism
MESH: Receptors, Tumor Necrosis Factor, Type I
MESH: Cell Line
MESH: Tumor Necrosis Factor-alpha/antagonists & inhibitors
MESH: Tumor Necrosis Factor-alpha/metabolism
MESH: Cell Survival
MESH: Enzyme Activation
MESH: Humans
MESH: Imidazoles/pharmacology
MESH: Indoles/pharmacology
MESH: Necrosis/physiopathology
MESH: Phosphorylation
[SDV.IMM]Life Sciences [q-bio]/Immunology
Cell Biology
Molecular Biology
Programmed cell death
Enzyme activator
Biochemistry
Biology
Protein structure
Necroptosis
Receptor
Caspase 8
RIPK1
Cell biology
Cytosol
Original Paper
Źródło :
Cell Death and Differentiation
Cell Death and Differentiation, Nature Publishing Group, 2014, 21 (10), pp.1511-1521. ⟨10.1038/cdd.2014.76⟩
Crossref
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3113b69ac08c61599e8c895b8f8a2cc1
https://hal-pasteur.archives-ouvertes.fr/pasteur-01384184
Tytuł :
Listeria monocytogenes-Associated Biliary Tract Infections
Autorzy :
Charlier, Caroline
Fevre, Cindy
Travier, Laetitia
Cazenave, Benoît
Bracq-Dieye, Hélène
Podevin, Juliette
Assomany, Daher
Guilbert, Lydie
Bossard, Céline
Carpentier, Françoise
Cales, Valérie
Leclercq, Alexandre
Lecuit, Marc
Pokaż więcej
Temat :
MESH: Abdominal Pain
MESH: Adult
MESH: Humans
MESH: Immunosuppressive Agents
MESH: Aged
MESH: Listeria monocytogenes
MESH: Listeriosis
MESH: Male
MESH: Medication Errors
MESH: Middle Aged
MESH: Necrosis
MESH: Penicillins
MESH: Cholecystolithiasis
MESH: Population Surveillance
MESH: Retrospective Studies
MESH: Aged, 80 and over
MESH: Anti-Bacterial Agents
MESH: Bacteremia
MESH: Biliary Tract Diseases
MESH: Cholangitis
MESH: Cholecystectomy
MESH: Cholecystitis
MESH: Comorbidity
MESH: Cysts
MESH: Drug Resistance, Bacterial
MESH: Female
MESH: Fever
MESH: France
MESH: Gallbladder Diseases
[SDV]Life Sciences [q-bio]
[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseases
[SDV.MP]Life Sciences [q-bio]/Microbiology and Parasitology
[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology
Article
Observational Study
General Medicine
Cholecystitis
medicine.disease
medicine
Gallbladder
medicine.anatomical_structure
Chronic infection
business.industry
business
Gastroenterology
medicine.medical_specialty
Listeria
biology.organism_classification
biology
Surgery
Listeria monocytogenes
medicine.disease_cause
Internal medicine
Biliary Cyst
Biliary tract
Gallbladder disease
Źródło :
Medicine
Medicine, Lippincott, Williams & Wilkins, 2014, 93 (18), pp.e105. ⟨10.1097/MD.0000000000000105⟩
Crossref
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fd13d55764868778a023fac3ce6a3c73
https://hal-pasteur.archives-ouvertes.fr/pasteur-02391680
Tytuł :
Increased susceptibility to liver fibrosis with age is correlated with an altered inflammatory response
Autorzy :
Hélène Gilgenkrantz
Jacques-Emmanuel Guidotti
Bernard Fromenty
Abdelhamid Slama
Claudine Cosson
Meriem Mahrouf-Yorgov
Claudia Mitchell
Alexandra Collin de l'Hortet
Elsa Abdoun
Pokaż więcej
Temat :
MESH: Aging
MESH: Animals
MESH: Male
MESH: Mice
MESH: Mice, Inbred C57BL
MESH: Necrosis
MESH: Oxidative Stress
MESH: Carbon Tetrachloride
MESH: Cell Proliferation
MESH: Chronic Disease
MESH: Disease Susceptibility
MESH: Hepatocytes
MESH: Humans
MESH: Inflammation
MESH: Liver Cirrhosis
[SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterology
MESH : Aging
MESH : Animals
MESH : Male
MESH : Mice
MESH : Mice, Inbred C57BL
MESH : Necrosis
MESH : Oxidative Stress
MESH : Carbon Tetrachloride
MESH : Cell Proliferation
MESH : Chronic Disease
MESH : Disease Susceptibility
MESH : Hepatocytes
MESH : Humans
MESH : Inflammation
MESH : Liver Cirrhosis
[ SDV.MHEP.HEG ] Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterology
Geriatrics and Gerontology
Ageing
Liver fibrosis
Liver disorder
Hepatitis C virus
medicine.disease_cause
medicine
Inflammation
medicine.symptom
Biology
Oxidative stress
Immunology
Fibrosis
medicine.disease
Carbon tetrachloride
chemistry.chemical_compound
chemistry
Hepatocyte
medicine.anatomical_structure
Źródło :
Rejuvenation Research
Rejuvenation Research, Mary Ann Liebert, 2011, 14 (4), pp.353-63. ⟨10.1089/rej.2010.1146⟩
Rejuvenation Research, Mary Ann Liebert, 2011, 14 (4), pp.353-63. 〈10.1089/rej.2010.1146〉
Crossref
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f6f3a36d5a694bffd01377a9ff1c94f7
https://hal.archives-ouvertes.fr/hal-00740103
Tytuł :
IL-22 modulates IL-17A production and controls inflammation and tissue damage in experimental dengue infection
Autorzy :
Guabiraba, Rodrigo
Besnard, Anne-Gaëlle
Marques, Rafael E
Maillet, Isabelle
Fagundes, Caio T
Conceição, Thais M
Rust, Naiara M
Charreau, Sandrine
Paris, Isabelle
Lecron, Jean-Claude
Renauld, Jean-Christophe
Quesniaux, Valérie
Da Poian, Andrea T
Arruda, Luciana B
Souza, Danielle G
Ryffel, Bernhard
Teixeira, Mauro M
Pokaż więcej
Temat :
dengue virus
IL-17A
IL-22
infection
inflammation
MESH: Antigens, CD95
MESH: Liver
MESH: Lymphocyte Activation
MESH: Mice
MESH: Mice, Inbred C57BL
MESH: Mice, Transgenic
MESH: Necrosis
MESH: Protein Isoforms
MESH: T-Lymphocytes
MESH: Animals
MESH: Dengue
MESH: Apoptosis
MESH: Dengue Virus
MESH: Disease Models, Animal
MESH: Hep G2 Cells
MESH: Inflammation
MESH: Inflammation Mediators
MESH: Interleukin-17
MESH: Interleukin-6
MESH: Interleukins
MESH: Male
MESH: Mice, Knockout
MESH: B-Lymphocytes
MESH: Neutrophils
MESH: Receptors, Interleukin-17
MESH: Viral Load
MESH: CASP8 and FADD-Like Apoptosis Regulating Protein
MESH: Gene Expression Regulation
MESH: HEK293 Cells
MESH: Humans
MESH: Listeria monocytogenes
MESH: Listeriosis
[SDV.IMM]Life Sciences [q-bio]/Immunology
Źródło :
European Journal of Immunology
European Journal of Immunology, Wiley-VCH Verlag, 2013, 43 (6), pp.1529-44. ⟨10.1002/eji.201243229⟩
Dostępność :
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::4fbe16323169ce8ebc8e5bb17d66b20a
https://hal.archives-ouvertes.fr/hal-00944323
Tytuł :
Branched fungal β-glucan causes hyperinflammation and necrosis in phagocyte NADPH oxidase-deficient mice
Autorzy :
Mg Schäppi
Gaëtan Gavazzi
Fr Herrmann
Laurence Fiette
Karl-Heinz Krause
Christine Deffert
Dc Belli
Pokaż więcej
Temat :
Pathology and Forensic Medicine
Inflammation
medicine.symptom
medicine
Programmed cell death
Chronic granulomatous disease
medicine.disease
Immunology
NADPH oxidase
biology.protein
biology
NAD(P)H oxidase
Phagocyte
medicine.anatomical_structure
Lipoteichoic acid
Necrosis
MESH: Animals
MESH: Bacteria
MESH: Mice
MESH: Mice, Inbred C57BL
MESH: Mice, Mutant Strains
MESH: NADPH Oxidase
MESH: Necrosis
MESH: Phagocytes
MESH: Skin
MESH: beta-Glucans
MESH: Cell Death
MESH: Cell Wall
MESH: Dermatitis
MESH: Disease Models, Animal
MESH: Fungi
MESH: Granulomatous Disease, Chronic
MESH: Injections, Intradermal
MESH: Male
[SDV.IMM.II]Life Sciences [q-bio]/Immunology/Innate immunity
[SDV.BBM.BC]Life Sciences [q-bio]/Biochemistry, Molecular Biology/Biochemistry [q-bio.BM]
ddc:616.07
ddc:618.97
Animals
Bacteria/chemistry
Cell Death
Cell Wall/chemistry
Dermatitis/etiology/pathology
Disease Models Animal
Fungi/chemistry
Granulomatous Disease Chronic/etiology/pathology
Injections Intradermal
Male
Mice
Mice Inbred C57BL
Mice Mutant Strains
NADPH Oxidase/deficiency
Phagocytes/enzymology
Skin/pathology
Beta-Glucans/toxicity
Źródło :
Crossref
Journal of Pathology
Journal of Pathology, Wiley, 2008, 214 (4), pp.434-44. ⟨10.1002/path.2298⟩
The Journal of Pathology, Vol. 214, No 4 (2008) pp. 434-44
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b72fac346d0438a13b0d5e5bfb079e91
https://api.wiley.com/onlinelibrary/tdm/v1/articles/10.1002%2Fpath.2298

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