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Tytuł:
The potential role of glucose metabolism, lipid metabolism, and amino acid metabolism in the treatment of Parkinson's disease.
Autorzy:
Li H; Department of Biochemistry and Molecular Biology, School of Basic Medical Science, Southwest Medical University, Luzhou, China.
Zeng F; Department of Biochemistry and Molecular Biology, School of Basic Medical Science, Southwest Medical University, Luzhou, China.
Huang C; Department of Dermatology, The Affiliated Hospital of Southwest Medical University, Luzhou, China.
Pu Q; Department of Biochemistry and Molecular Biology, School of Basic Medical Science, Southwest Medical University, Luzhou, China.
Thomas ER; Department of Microbiology, NEIGRIHMS, Shillong, India.
Chen Y; Department of Dermatology, The Affiliated Hospital of Southwest Medical University, Luzhou, China.
Li X; Department of Biochemistry and Molecular Biology, School of Basic Medical Science, Southwest Medical University, Luzhou, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Feb; Vol. 30 (2), pp. e14411. Date of Electronic Publication: 2023 Aug 14.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Parkinson Disease*/metabolism
Neurodegenerative Diseases*
Humans ; Lipid Metabolism ; alpha-Synuclein/metabolism ; Levodopa ; Dopaminergic Neurons/metabolism ; Glucose
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
PGAM5 knockout causes depressive-like behaviors in mice via ATP deficiency in the prefrontal cortex.
Autorzy:
Cui W; Department of Imaging Diagnostics, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Chen C; The Second Affiliated Hospital of Guangzhou University of Chinese Medicine, Guangzhou, China.
Gong L; Department of Imaging Diagnostics, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Wen J; Department of Imaging Diagnostics, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Yang S; Department of Imaging Diagnostics, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Zheng M; Department of Pharmacy, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Gao B; School of Traditional Chinese Medicine, Southern Medical University, Guangzhou, China.
You J; School of Traditional Chinese Medicine, Southern Medical University, Guangzhou, China.
Lin X; Department of Imaging Diagnostics, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Hao Y; Department of Imaging Diagnostics, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Chen Z; Department of Imaging Diagnostics, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Wu Z; Department of Imaging Diagnostics, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Gao L; Department of Imaging Diagnostics, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Tang J; Department of Imaging Diagnostics, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Yuan Z; Centre for Cognitive and Brain Sciences, University of Macau, Taipa, China.
Sun X; School of Traditional Chinese Medicine, Southern Medical University, Guangzhou, China.
Jing L; Traditional Chinese Medicine Integrated Hospital, Southern Medical University, Guangzhou, China.
Wen G; Department of Imaging Diagnostics, Nanfang Hospital, Southern Medical University, Guangzhou, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Feb; Vol. 30 (2), pp. e14377. Date of Electronic Publication: 2023 Aug 25.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Depression*/diagnostic imaging
Depression*/genetics
Depression*/metabolism
Depressive Disorder, Major*/diagnostic imaging
Depressive Disorder, Major*/genetics
Depressive Disorder, Major*/metabolism
Mice ; Animals ; Adenosine Triphosphate/metabolism ; Prefrontal Cortex/metabolism ; Energy Metabolism ; Stress, Psychological/metabolism ; Mice, Knockout ; Phosphoprotein Phosphatases/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Tracing the path of disruption: C isotope applications in traumatic brain injury-induced metabolic dysfunction.
Autorzy:
Peper CJ; Clinical Neuroscience Research Center, Departments of Neurosurgery and Neurology, Tulane University School of Medicine, New Orleans, Louisiana, USA.
Kilgore MD; Clinical Neuroscience Research Center, Departments of Neurosurgery and Neurology, Tulane University School of Medicine, New Orleans, Louisiana, USA.
Jiang Y; Clinical Neuroscience Research Center, Departments of Neurosurgery and Neurology, Tulane University School of Medicine, New Orleans, Louisiana, USA.
Xiu Y; Clinical Neuroscience Research Center, Departments of Neurosurgery and Neurology, Tulane University School of Medicine, New Orleans, Louisiana, USA.
Xia W; Clinical Neuroscience Research Center, Departments of Neurosurgery and Neurology, Tulane University School of Medicine, New Orleans, Louisiana, USA.
Wang Y; Clinical Neuroscience Research Center, Departments of Neurosurgery and Neurology, Tulane University School of Medicine, New Orleans, Louisiana, USA.
Shi M; Clinical Neuroscience Research Center, Departments of Neurosurgery and Neurology, Tulane University School of Medicine, New Orleans, Louisiana, USA.
Zhou D; Clinical Neuroscience Research Center, Departments of Neurosurgery and Neurology, Tulane University School of Medicine, New Orleans, Louisiana, USA.
Dumont AS; Clinical Neuroscience Research Center, Departments of Neurosurgery and Neurology, Tulane University School of Medicine, New Orleans, Louisiana, USA.
Wang X; Clinical Neuroscience Research Center, Departments of Neurosurgery and Neurology, Tulane University School of Medicine, New Orleans, Louisiana, USA.; Neuroscience Program, Tulane Brain Institute, Tulane University, New Orleans, Louisiana, USA.
Liu N; Clinical Neuroscience Research Center, Departments of Neurosurgery and Neurology, Tulane University School of Medicine, New Orleans, Louisiana, USA.; Neuroscience Program, Tulane Brain Institute, Tulane University, New Orleans, Louisiana, USA.; Tulane University Translational Sciences Institute, New Orleans, Louisiana, USA.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14693.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Brain Injuries, Traumatic*/metabolism
Animals ; Humans ; Energy Metabolism ; Isotopes
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The role of glutamate and glutamine metabolism and related transporters in nerve cells.
Autorzy:
Zhang D; Department of Pediatrics, Shengjing Hospital of China Medical University, Shenyang, Liaoning, China.; Medical Research Center, Liaoning Key Laboratory of Research and Application of Animal Models for Environment and Metabolic Diseases, Shengjing Hospital of China Medical University, Shenyang, Liaoning, China.
Hua Z; Department of Pediatrics, Shengjing Hospital of China Medical University, Shenyang, Liaoning, China.; Medical Research Center, Liaoning Key Laboratory of Research and Application of Animal Models for Environment and Metabolic Diseases, Shengjing Hospital of China Medical University, Shenyang, Liaoning, China.
Li Z; Department of Pediatrics, Shengjing Hospital of China Medical University, Shenyang, Liaoning, China.; Medical Research Center, Liaoning Key Laboratory of Research and Application of Animal Models for Environment and Metabolic Diseases, Shengjing Hospital of China Medical University, Shenyang, Liaoning, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Feb; Vol. 30 (2), pp. e14617.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Glutamic Acid*/metabolism
Glutamine*
Amino Acids/metabolism ; Brain/metabolism ; Neurons/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Sevoflurane causes cognitive impairment by inducing iron deficiency and inhibiting the proliferation of neural precursor cells in infant mice.
Autorzy:
Zuo Y; Laboratory of Molecular Iron Metabolism, College of Life Science, Hebei Normal University, Shijiazhuang, Hebei Province, China.
Xie J; Laboratory of Molecular Iron Metabolism, College of Life Science, Hebei Normal University, Shijiazhuang, Hebei Province, China.
Zhang X; Laboratory of Molecular Iron Metabolism, College of Life Science, Hebei Normal University, Shijiazhuang, Hebei Province, China.
Thirupathi A; Faculty of Sports Science, Ningbo University, Ningbo, China.
Liu X; The Second Affiliated Hospital of Hebei Medical University, Shijiazhuang, China.
Zhang D; Laboratory of Molecular Iron Metabolism, College of Life Science, Hebei Normal University, Shijiazhuang, Hebei Province, China.
Zhang J; Laboratory of Molecular Iron Metabolism, College of Life Science, Hebei Normal University, Shijiazhuang, Hebei Province, China.
Shi Z; Laboratory of Molecular Iron Metabolism, College of Life Science, Hebei Normal University, Shijiazhuang, Hebei Province, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Feb; Vol. 30 (2), pp. e14612.
Typ publikacji:
Journal Article
MeSH Terms:
Neural Stem Cells*/metabolism
Cognitive Dysfunction*/chemically induced
Cognitive Dysfunction*/metabolism
Iron Deficiencies*
Humans ; Mice ; Animals ; Sevoflurane/toxicity ; Bromodeoxyuridine/metabolism ; Mice, Inbred C57BL ; Neurons/metabolism ; Cell Proliferation ; Iron/metabolism ; Hippocampus/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
BRCC3 mediates inflammation and pyroptosis in cerebral ischemia/reperfusion injury by activating the NLRP6 inflammasome.
Autorzy:
Huang X; Department of Pathology, Chongqing Medical University, Chongqing, China.; Department of Pathology, Chongqing Three Gorges Medical College, Wanzhou, China.
Tan J; Department of Pathophysiology, Chongqing Medical University, Chongqing, China.
Ji Y; Department of Pathology, Chongqing Medical University, Chongqing, China.
Luo J; Department of Pathology, Chongqing Medical University, Chongqing, China.; Department of Neurology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China.
Zhao Y; Department of Pathology, Chongqing Medical University, Chongqing, China.
Zhao J; Department of Pathophysiology, Chongqing Medical University, Chongqing, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14697.
Typ publikacji:
Journal Article
MeSH Terms:
Brain Ischemia*/metabolism
Reperfusion Injury*/metabolism
Animals ; Humans ; Mice ; Deubiquitinating Enzymes ; Infarction, Middle Cerebral Artery/pathology ; Inflammasomes/metabolism ; Intracellular Signaling Peptides and Proteins ; Neuroinflammatory Diseases ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Pyroptosis/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
ERBB1 alleviates secondary brain injury induced by experimental intracerebral hemorrhage in rats by modulating neuronal death via PLC-γ/PKC pathway.
Autorzy:
Li B; Department of Neurosurgery & Brain and Nerve Research Laboratory, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu Province, China.; Department of Neurosurgery, Yancheng City No. 1 People's Hospital, Yancheng First Hospital, Affiliated Hospital of Nanjing University Medical School, Yancheng, Jiangsu Province, China.
Wu J; Department of Neurosurgery & Brain and Nerve Research Laboratory, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu Province, China.
Cao D; Department of Neurosurgery & Brain and Nerve Research Laboratory, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu Province, China.; Department of Neurosurgery, The Affiliated Hospital of Yangzhou University, Yangzhou, Jiangsu Province, China.
Cao C; Department of Neurosurgery & Brain and Nerve Research Laboratory, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu Province, China.; Department of Neurocritical Intensive Care Unit, Jiangyin Clinical College of Xuzhou Medical College, Jiangyin, Jiangsu Province, China.
Zhang J; Department of Neurosurgery & Brain and Nerve Research Laboratory, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu Province, China.
Li X; Department of Neurosurgery & Brain and Nerve Research Laboratory, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu Province, China.
Li H; Department of Neurosurgery & Brain and Nerve Research Laboratory, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu Province, China.
Shen H; Department of Neurosurgery & Brain and Nerve Research Laboratory, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu Province, China.
Yu Z; Department of Neurosurgery & Brain and Nerve Research Laboratory, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu Province, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14679.
Typ publikacji:
Journal Article
MeSH Terms:
Brain Injuries*/metabolism
Cerebral Hemorrhage*/complications
Cerebral Hemorrhage*/metabolism
Quinazolines*
ErbB Receptors*/metabolism
Animals ; Rats ; Apoptosis ; Cytokines/metabolism ; Phospholipase C gamma/metabolism ; Rats, Sprague-Dawley ; Tyrphostins ; Protein Kinase C/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Hyperforin ameliorates neuroinflammation and white matter lesions by regulating microglial VEGFR 2 /SRC pathway in vascular cognitive impairment mice.
Autorzy:
Gao X; Department of Neurology, Shanghai Changhai Hospital, Second Military Medical University/Naval Medical University, Shanghai, China.
Chen J; Department of Neurology, Shanghai Changhai Hospital, Second Military Medical University/Naval Medical University, Shanghai, China.
Yin G; Department of Neurology, Shanghai Changhai Hospital, Second Military Medical University/Naval Medical University, Shanghai, China.
Liu Y; Department of Neurology, Shanghai Changhai Hospital, Second Military Medical University/Naval Medical University, Shanghai, China.
Gu Z; Department of Neurology, Shanghai Changhai Hospital, Second Military Medical University/Naval Medical University, Shanghai, China.
Sun R; Department of Neurology, Shanghai Changhai Hospital, Second Military Medical University/Naval Medical University, Shanghai, China.
Sun X; Department of Neurology, Shanghai Changhai Hospital, Second Military Medical University/Naval Medical University, Shanghai, China.
Jiao X; Department of Neurology, Shanghai Changhai Hospital, Second Military Medical University/Naval Medical University, Shanghai, China.
Wang L; Department of Neurology, Shanghai Changhai Hospital, Second Military Medical University/Naval Medical University, Shanghai, China.
Wang N; Department of Neurology, Shanghai Changhai Hospital, Second Military Medical University/Naval Medical University, Shanghai, China.
Zhang Y; Department of Psychiatry, Faculty of Medicine and Dentistry, University of Alberta, Edmonton, Alberta, Canada.
Kan Y; Department of Neurology, Shanghai Changhai Hospital, Second Military Medical University/Naval Medical University, Shanghai, China.
Bi X; Department of Neurology, Shanghai Changhai Hospital, Second Military Medical University/Naval Medical University, Shanghai, China.
Du B; Department of Neurology, Shanghai Changhai Hospital, Second Military Medical University/Naval Medical University, Shanghai, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14666.
Typ publikacji:
Journal Article
MeSH Terms:
White Matter*/metabolism
Cognitive Dysfunction*/drug therapy
Cognitive Dysfunction*/metabolism
Terpenes*
Phloroglucinol/*analogs & derivatives
Mice ; Animals ; Microglia ; Neuroinflammatory Diseases ; Tumor Necrosis Factor-alpha/metabolism ; Interleukin-6/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
SKN-1/NRF2 upregulation by vitamin A is conserved from nematodes to mammals and is critical for lifespan extension in Caenorhabditis elegans.
Autorzy:
Sirakawin C; Laboratory of Molecular Neurobiology, Sheng Yushou Center of Cell Biology and Immunology, Department of Genetics and Developmental Biology, School of Life Sciences and Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
Lin D; Laboratory of Molecular Neurobiology, Sheng Yushou Center of Cell Biology and Immunology, Department of Genetics and Developmental Biology, School of Life Sciences and Biotechnology, Shanghai Jiao Tong University, Shanghai, China.; Key Laboratory for Molecular Enzymology and Engineering, School of Life Sciences, Jilin University, Changchun, China.
Zhou Z; Laboratory of Molecular Neurobiology, Sheng Yushou Center of Cell Biology and Immunology, Department of Genetics and Developmental Biology, School of Life Sciences and Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
Wang X; Shanghai Key Laboratory of Pancreatic Diseases, Institute of Translational Medicine, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Kelleher R; University of Warwick, School of Life Sciences, Coventry, UK.
Huang S; Laboratory of Molecular Neurobiology, Sheng Yushou Center of Cell Biology and Immunology, Department of Genetics and Developmental Biology, School of Life Sciences and Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
Long W; Laboratory of Molecular Neurobiology, Sheng Yushou Center of Cell Biology and Immunology, Department of Genetics and Developmental Biology, School of Life Sciences and Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
Pires-daSilva A; University of Warwick, School of Life Sciences, Coventry, UK.
Liu Y; Laboratory of Molecular Neurobiology, Sheng Yushou Center of Cell Biology and Immunology, Department of Genetics and Developmental Biology, School of Life Sciences and Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
Wang J; Shanghai Key Laboratory of Pancreatic Diseases, Institute of Translational Medicine, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Vinnikov IA; Laboratory of Molecular Neurobiology, Sheng Yushou Center of Cell Biology and Immunology, Department of Genetics and Developmental Biology, School of Life Sciences and Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
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Źródło:
Aging cell [Aging Cell] 2024 Mar; Vol. 23 (3), pp. e14064. Date of Electronic Publication: 2023 Dec 15.
Typ publikacji:
Journal Article
MeSH Terms:
Caenorhabditis elegans*/metabolism
Caenorhabditis elegans Proteins*/metabolism
Animals ; Humans ; Mice ; Longevity/genetics ; DNA-Binding Proteins/genetics ; DNA-Binding Proteins/metabolism ; Vitamin A/pharmacology ; Vitamin A/metabolism ; NF-E2-Related Factor 2/metabolism ; Up-Regulation ; Oxidative Stress ; Mammals/metabolism ; Micronutrients/metabolism ; Micronutrients/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Acetyl-11-keto-beta-boswellic acid modulates macrophage polarization and Schwann cell migration to accelerate spinal cord injury repair in rats.
Autorzy:
Wang Y; Department of Veterinary Medicine, Northeast Agricultural University, Harbin, Heilongjiang, China.
Xiong Z; Department of Veterinary Medicine, Northeast Agricultural University, Harbin, Heilongjiang, China.
Qiao Y; School of Life Sciences, Northeast Agricultural University, Harbin, Heilongjiang, China.
Zhang Q; Department of Veterinary Medicine, Northeast Agricultural University, Harbin, Heilongjiang, China.
Zhou G; Department of Veterinary Medicine, Northeast Agricultural University, Harbin, Heilongjiang, China.
Zhou C; Department of Veterinary Medicine, Northeast Agricultural University, Harbin, Heilongjiang, China.
Ma X; Department of Veterinary Medicine, Northeast Agricultural University, Harbin, Heilongjiang, China.
Jiang X; Department of Veterinary Medicine, Northeast Agricultural University, Harbin, Heilongjiang, China.
Yu W; Department of Veterinary Medicine, Northeast Agricultural University, Harbin, Heilongjiang, China.; Key Laboratory of the Provincial Education, Department of Heilongjiang for Common Animal Disease Prevention and Treatment, Northeast Agricultural University, Harbin, Heilongjiang, China.; Institute of Chinese Veterinary Medicine, Northeast Agricultural University, Harbin, Heilongjiang, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14642.
Typ publikacji:
Journal Article
MeSH Terms:
Interleukin-10*/metabolism
Spinal Cord Injuries*/metabolism
Triterpenes*
Rats ; Animals ; Antioxidants/pharmacology ; NF-E2-Related Factor 2/metabolism ; Macrophages/metabolism ; Spinal Cord/metabolism ; Cell Movement
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Fat mass and obesity associated protein inhibits neuronal ferroptosis via the FYN/Drp1 axis and alleviate cerebral ischemia/reperfusion injury.
Autorzy:
Zhang Y; Department of Emergency, Hunan Provincial People's Hospital, The First Affiliated Hospital of Hunan Normal University, Changsha, Hunan, China.
Gong X; Department of Neurosurgery, Hunan Provincial People's Hospital, The First Affiliated Hospital of Hunan Normal University, Changsha, Hunan, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14636.
Typ publikacji:
Journal Article
MeSH Terms:
Brain Ischemia*/genetics
Brain Ischemia*/metabolism
Ferroptosis*
Reperfusion Injury*/genetics
Reperfusion Injury*/metabolism
Animals ; Mice ; Alpha-Ketoglutarate-Dependent Dioxygenase FTO/metabolism ; Neurons/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CYP1B1 affects the integrity of the blood-brain barrier and oxidative stress in the striatum: An investigation of manganese-induced neurotoxicity.
Autorzy:
Wu J; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.; Department of Pharmacy, Taikang Tongji (Wuhan) Hospital, Wuhu, China.
Li Y; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.; Department of Pharmacy, The First Affiliated Hospital of Wannan Medical College, Wuhu, China.
Tian S; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.
Na S; Zhongnan Hospital of Wuhan University, Institute of Hepatobiliary Diseases of Wuhan University, Transplant Center of Wuhan University, Hubei Key Laboratory of Medical Technology on Transplantation, Wuhan, Hubei, China.
Wei H; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.
Wu Y; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.
Yang Y; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.
Shen Z; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.
Ding J; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.
Bao S; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.
Liu S; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.
Li L; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.
Feng R; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.
Zhu Y; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.
He C; Demonstration Center for Experimental Basic Medicine Education, School of Basic Medical Sciences, Wuhan University, Wuhan, China.
Yue J; Department of Pharmacology, School of Basic Medical Sciences, Wuhan University, Wuhan, China.; Hubei Province Key Laboratory of Allergy and Immunology, Wuhan, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14633.
Typ publikacji:
Journal Article
MeSH Terms:
Blood-Brain Barrier*/metabolism
Cytochrome P-450 CYP1B1*/metabolism
Neuroblastoma*
Animals ; Humans ; Mice ; alpha-Synuclein/metabolism ; Biotin/metabolism ; Dopamine/metabolism ; Endothelial Cells/metabolism ; Manganese/toxicity ; Oxidative Stress
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
HuR-positive stress granules: Potential targets for age-related osteoporosis.
Autorzy:
Huai Y; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.; Department of Orthopedics, Tangdu Hospital, Air Force Military Medical University, Xi'an, China.
Wang X; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Mao W; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Wang X; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Zhao Y; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Chu X; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Huang Q; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Ru K; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Zhang L; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Li Y; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Chen Z; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Qian A; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
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Źródło:
Aging cell [Aging Cell] 2024 Mar; Vol. 23 (3), pp. e14053. Date of Electronic Publication: 2024 Feb 20.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/metabolism
Osteoporosis*/metabolism
Stress Granules*
Humans ; Osteoblasts/metabolism ; Osteogenesis ; RNA-Binding Proteins/metabolism ; ELAV-Like Protein 1/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Selective HDAC6 inhibition protects against blood-brain barrier dysfunction after intracerebral hemorrhage.
Autorzy:
Peng C; Department of Neurology, Second Xiangya Hospital, Central South University, Changsha, Hunan, China.; Department of Neurology, Hunan Provincial Rehabilitation Hospital, Hunan University of Medicine, Changsha, Hunan, China.
Wang Y; Department of Neurology, Second Xiangya Hospital, Central South University, Changsha, Hunan, China.
Hu Z; Department of Neurology, Second Xiangya Hospital, Central South University, Changsha, Hunan, China.
Chen C; Department of Neurology, Second Xiangya Hospital, Central South University, Changsha, Hunan, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14429. Date of Electronic Publication: 2023 Sep 04.
Typ publikacji:
Journal Article
MeSH Terms:
Blood-Brain Barrier*/metabolism
Tubulin*/metabolism
Animals ; Humans ; Rats ; Brain/metabolism ; Cerebral Hemorrhage/complications ; Cerebral Hemorrhage/drug therapy ; Cerebral Hemorrhage/genetics ; Disease Models, Animal ; Endothelial Cells/metabolism ; Histone Deacetylase 6/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Potentiated GABAergic neuronal activities in the basolateral amygdala alleviate stress-induced depressive behaviors.
Autorzy:
Asim M; Department of Neuroscience, City University of Hong Kong, Kowloon Tong, People's Republic of China.; Department of Biomedical Science, City University of Hong Kong, Kowloon Tong, People's Republic of China.
Wang H; Department of Neuroscience, City University of Hong Kong, Kowloon Tong, People's Republic of China.; Department of Biomedical Science, City University of Hong Kong, Kowloon Tong, People's Republic of China.
Chen X; Department of Neuroscience, City University of Hong Kong, Kowloon Tong, People's Republic of China.; City University of Hong Kong Shenzhen Research Institute, Shenzhen, People's Republic of China.
He J; Department of Neuroscience, City University of Hong Kong, Kowloon Tong, People's Republic of China.; City University of Hong Kong Shenzhen Research Institute, Shenzhen, People's Republic of China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14422. Date of Electronic Publication: 2023 Sep 16.
Typ publikacji:
Journal Article
MeSH Terms:
Basolateral Nuclear Complex*/metabolism
Depressive Disorder, Major*/metabolism
Humans ; Mice ; Animals ; Calcium-Calmodulin-Dependent Protein Kinase Type 2/metabolism ; GABAergic Neurons/metabolism ; gamma-Aminobutyric Acid/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Gastrodin improves nerve cell injury and behaviors of depressed mice through Caspase-3-mediated apoptosis.
Autorzy:
Pei H; College of Chinese Medicinal Materials, Jilin Agricultural University, Changchun, China.
Shen H; Department of Neurology, The Second Affiliated Hospital of Jiaxing University, Jiaxing, China.
Bi J; College of Chinese Medicinal Materials, Jilin Agricultural University, Changchun, China.
He Z; College of Chinese Medicinal Materials, Jilin Agricultural University, Changchun, China.
Zhai L; Department of Neurology, The Second Affiliated Hospital of Jiaxing University, Jiaxing, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14444. Date of Electronic Publication: 2023 Aug 31.
Typ publikacji:
Journal Article
MeSH Terms:
Depression*/drug therapy
Depression*/metabolism
Neurons*/metabolism
Benzyl Alcohols*
Glucosides*
Mice ; Animals ; Caspase 3/metabolism ; Apoptosis ; Stress, Psychological/metabolism ; Disease Models, Animal ; Hippocampus/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Phosphodiesterase inhibitor ameliorates senescent changes of renal interstitial pericytes in aging kidney.
Autorzy:
Kim HD; Department of Internal Medicine, Eunpyeong St. Mary's Hospital, College of Medicine, The Catholic University of Korea, Seoul, Republic of Korea.
Kim EN; Department of Internal Medicine, College of Medicine, The Catholic University of Korea, Seoul, Republic of Korea.
Lim JH; Department of Internal Medicine, College of Medicine, The Catholic University of Korea, Seoul, Republic of Korea.
Kim Y; Department of Internal Medicine, Seoul St. Mary's Hospital, College of Medicine, The Catholic University of Korea, Seoul, Republic of Korea.
Ban TH; Department of Internal Medicine, Eunpyeong St. Mary's Hospital, College of Medicine, The Catholic University of Korea, Seoul, Republic of Korea.
Lee H; Department of Internal Medicine, Seoul National University Hospital, Seoul, Republic of Korea.
Kim YS; Department of Internal Medicine, Seoul National University Hospital, Seoul, Republic of Korea.
Park CW; Department of Internal Medicine, Seoul St. Mary's Hospital, College of Medicine, The Catholic University of Korea, Seoul, Republic of Korea.
Choi BS; Department of Internal Medicine, Eunpyeong St. Mary's Hospital, College of Medicine, The Catholic University of Korea, Seoul, Republic of Korea.
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Źródło:
Aging cell [Aging Cell] 2024 Mar; Vol. 23 (3), pp. e14075. Date of Electronic Publication: 2023 Dec 28.
Typ publikacji:
Journal Article
MeSH Terms:
Pericytes*/metabolism
Kidney Diseases*/metabolism
Humans ; Mice ; Animals ; Aged ; Phosphodiesterase Inhibitors/metabolism ; Endothelial Cells/metabolism ; Albuminuria/metabolism ; Albuminuria/pathology ; Mice, Inbred C57BL ; Kidney/metabolism ; Aging ; Fibrosis ; Inflammation/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Inhibition of HDAC6 promotes microvascular endothelial cells to phagocytize myelin debris and reduces inflammatory response to accelerate the repair of spinal cord injury.
Autorzy:
Wu C; Department of Traumatology and Orthopedics, Affiliated Hospital of Nanjing University of Chinese Medicine, Nanjing, China.; Laboratory of New Techniques of Restoration & Reconstruction, Institute of Traumatology & Orthopedics, Nanjing University of Chinese Medicine, Nanjing, China.
Pan Y; Laboratory of Chinese Medicine Nursing Intervention for Chronic Diseases, Nanjing University of Chinese Medicine, Nanjing, China.
Wang L; School of Chinese Medicine, School of Integrated Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, China.
Liu M; School of Chinese Medicine, School of Integrated Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, China.
Tu P; Department of Traumatology and Orthopedics, Affiliated Hospital of Nanjing University of Chinese Medicine, Nanjing, China.; Laboratory of New Techniques of Restoration & Reconstruction, Institute of Traumatology & Orthopedics, Nanjing University of Chinese Medicine, Nanjing, China.
Chen S; School of Chinese Medicine, School of Integrated Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, China.
Shi L; School of Chinese Medicine, School of Integrated Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, China.
Yan D; Department of Traumatology and Orthopedics, Affiliated Hospital of Nanjing University of Chinese Medicine, Nanjing, China.; Laboratory of New Techniques of Restoration & Reconstruction, Institute of Traumatology & Orthopedics, Nanjing University of Chinese Medicine, Nanjing, China.
Ma Y; Department of Traumatology and Orthopedics, Affiliated Hospital of Nanjing University of Chinese Medicine, Nanjing, China.; Laboratory of New Techniques of Restoration & Reconstruction, Institute of Traumatology & Orthopedics, Nanjing University of Chinese Medicine, Nanjing, China.; School of Chinese Medicine, School of Integrated Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, China.
Guo Y; Department of Traumatology and Orthopedics, Affiliated Hospital of Nanjing University of Chinese Medicine, Nanjing, China.; Laboratory of New Techniques of Restoration & Reconstruction, Institute of Traumatology & Orthopedics, Nanjing University of Chinese Medicine, Nanjing, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14439. Date of Electronic Publication: 2023 Aug 29.
Typ publikacji:
Journal Article
MeSH Terms:
Myelin Sheath*/metabolism
Spinal Cord Injuries*/drug therapy
Spinal Cord Injuries*/metabolism
Humans ; Endothelial Cells/metabolism ; Histone Deacetylase 6/antagonists & inhibitors ; Histone Deacetylase 6/metabolism ; Immunoglobulin G/pharmacology ; Immunoglobulin M/metabolism ; Macrophages ; Spinal Cord/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Luteolin alleviates depression-like behavior by modulating glycerophospholipid metabolism in the hippocampus and prefrontal cortex of LOD rats.
Autorzy:
Wu X; Integrative Medicine Research Center, School of Basic Medical Sciences, Guangzhou University of Chinese Medicine, Guangzhou University of Chinese Medicine, Guangzhou, China.
Xu H; Integrative Medicine Research Center, School of Basic Medical Sciences, Guangzhou University of Chinese Medicine, Guangzhou University of Chinese Medicine, Guangzhou, China.
Zeng N; Department of Rehabilitation Medicine, The People's Hospital of Longhua District, Shenzhen, China.
Li H; Key Laboratory of Depression Animal Model Based on TCM Syndrome, Key Laboratory of TCM for Prevention and Treatment of Brain Diseases with Cognitive Dysfunction, Jiangxi University of Chinese Medicine, Nanchang, China.
Yao G; Integrative Medicine Research Center, School of Basic Medical Sciences, Guangzhou University of Chinese Medicine, Guangzhou University of Chinese Medicine, Guangzhou, China.
Liu K; Integrative Medicine Research Center, School of Basic Medical Sciences, Guangzhou University of Chinese Medicine, Guangzhou University of Chinese Medicine, Guangzhou, China.
Yan C; Integrative Medicine Research Center, School of Basic Medical Sciences, Guangzhou University of Chinese Medicine, Guangzhou University of Chinese Medicine, Guangzhou, China.
Wu L; Integrative Medicine Research Center, School of Basic Medical Sciences, Guangzhou University of Chinese Medicine, Guangzhou University of Chinese Medicine, Guangzhou, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14455. Date of Electronic Publication: 2023 Sep 16.
Typ publikacji:
Journal Article
MeSH Terms:
Luteolin*/pharmacology
Luteolin*/therapeutic use
Luteolin*/metabolism
Depression*/drug therapy
Rats ; Animals ; Chromatography, Liquid ; Molecular Docking Simulation ; Tandem Mass Spectrometry ; Prefrontal Cortex/metabolism ; Glycerophospholipids/metabolism ; Hippocampus/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Human umbilical cord mesenchymal stem cell-derived exosomes attenuate neuroinflammation and oxidative stress through the NRF2/NF-κB/NLRP3 pathway.
Autorzy:
Che J; Department of Anesthesiology, Fudan University Shanghai Cancer Center, Shanghai, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China.
Wang H; Department of Anesthesiology, Fudan University Shanghai Cancer Center, Shanghai, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China.
Dong J; Department of Anesthesiology, Fudan University Shanghai Cancer Center, Shanghai, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China.
Wu Y; Department of Anesthesiology, Fudan University Shanghai Cancer Center, Shanghai, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China.
Zhang H; Shanghai Key Laboratory of Clinical Geriatric Medicine, Huadong Hospital, Fudan University, Shanghai, China.
Fu L; Shanghai Key Laboratory of Clinical Geriatric Medicine, Huadong Hospital, Fudan University, Shanghai, China.
Zhang J; Department of Anesthesiology, Fudan University Shanghai Cancer Center, Shanghai, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14454. Date of Electronic Publication: 2023 Sep 12.
Typ publikacji:
Journal Article
MeSH Terms:
Exosomes*/metabolism
Mesenchymal Stem Cells*/metabolism
Animals ; Humans ; Mice ; Anti-Inflammatory Agents/pharmacology ; Cytokines/metabolism ; Inflammasomes/metabolism ; Lipopolysaccharides/toxicity ; Mice, Inbred C57BL ; Neuroinflammatory Diseases ; NF-E2-Related Factor 2/metabolism ; NF-kappa B/metabolism ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Oxidative Stress ; Umbilical Cord/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Wyświetlanie 1-20 z 106560

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