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Tytuł:
The International Mouse Phenotyping Consortium: comprehensive knockout phenotyping underpinning the study of human disease.
Autorzy:
Groza T; European Bioinformatics Institute, European Molecular Biology Laboratory, Welcome Genome Campus, Hinxton CB10 1SD, UK.
Gomez FL; European Bioinformatics Institute, European Molecular Biology Laboratory, Welcome Genome Campus, Hinxton CB10 1SD, UK.
Mashhadi HH; European Bioinformatics Institute, European Molecular Biology Laboratory, Welcome Genome Campus, Hinxton CB10 1SD, UK.
Muñoz-Fuentes V; European Bioinformatics Institute, European Molecular Biology Laboratory, Welcome Genome Campus, Hinxton CB10 1SD, UK.
Gunes O; European Bioinformatics Institute, European Molecular Biology Laboratory, Welcome Genome Campus, Hinxton CB10 1SD, UK.
Wilson R; European Bioinformatics Institute, European Molecular Biology Laboratory, Welcome Genome Campus, Hinxton CB10 1SD, UK.
Cacheiro P; William Harvey Research Institute, Queen Mary University of London, London EC1M 6BQ, UK.
Frost A; Mary Lyon Centre at MRC Harwell, Harwell Campus OX11 7UE, UK.
Keskivali-Bond P; Mary Lyon Centre at MRC Harwell, Harwell Campus OX11 7UE, UK.
Vardal B; Mary Lyon Centre at MRC Harwell, Harwell Campus OX11 7UE, UK.
McCoy A; Mary Lyon Centre at MRC Harwell, Harwell Campus OX11 7UE, UK.
Cheng TK; Mary Lyon Centre at MRC Harwell, Harwell Campus OX11 7UE, UK.
Santos L; Research Data Team, The Turing Institute, 96 Euston Rd, London NW1 2DB, UK.
Wells S; Mary Lyon Centre at MRC Harwell, Harwell Campus OX11 7UE, UK.
Smedley D; William Harvey Research Institute, Queen Mary University of London, London EC1M 6BQ, UK.
Mallon AM; Research Data Team, The Turing Institute, 96 Euston Rd, London NW1 2DB, UK.
Parkinson H; European Bioinformatics Institute, European Molecular Biology Laboratory, Welcome Genome Campus, Hinxton CB10 1SD, UK.
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Źródło:
Nucleic acids research [Nucleic Acids Res] 2023 Jan 06; Vol. 51 (D1), pp. D1038-D1045.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Databases, Factual*
Mice, Knockout*
Disease Models, Animal*
Animals ; Humans ; Mice ; Phenotype
Czasopismo naukowe
Szczegóły
Tytuł:
Caspase-11 deficiency attenuates neutrophil recruitment into the atherosclerotic lesion in apolipoprotein E-deficient mice.
Autorzy:
Karasawa T; Division of Inflammation Research, Center for Molecular Medicine, Jichi Medical University, Shimotsuke, Tochigi, Japan. Electronic address: .
Komada T; Division of Inflammation Research, Center for Molecular Medicine, Jichi Medical University, Shimotsuke, Tochigi, Japan.
Baatarjav C; Division of Inflammation Research, Center for Molecular Medicine, Jichi Medical University, Shimotsuke, Tochigi, Japan.
Aizawa E; Division of Inflammation Research, Center for Molecular Medicine, Jichi Medical University, Shimotsuke, Tochigi, Japan.
Mizushina Y; Division of Inflammation Research, Center for Molecular Medicine, Jichi Medical University, Shimotsuke, Tochigi, Japan.
Fujimura K; Division of Inflammation Research, Center for Molecular Medicine, Jichi Medical University, Shimotsuke, Tochigi, Japan.
Gunji Y; Division of Inflammation Research, Center for Molecular Medicine, Jichi Medical University, Shimotsuke, Tochigi, Japan.
Komori S; Division of Inflammation Research, Center for Molecular Medicine, Jichi Medical University, Shimotsuke, Tochigi, Japan.
Aizawa H; Division of Inflammation Research, Center for Molecular Medicine, Jichi Medical University, Shimotsuke, Tochigi, Japan.
Jing Tao CB; Division of Inflammation Research, Center for Molecular Medicine, Jichi Medical University, Shimotsuke, Tochigi, Japan.
Matsumura T; Division of Inflammation Research, Center for Molecular Medicine, Jichi Medical University, Shimotsuke, Tochigi, Japan.
Takahashi M; Division of Inflammation Research, Center for Molecular Medicine, Jichi Medical University, Shimotsuke, Tochigi, Japan. Electronic address: .
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Źródło:
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2023 Dec 17; Vol. 686, pp. 149158. Date of Electronic Publication: 2023 Oct 26.
Typ publikacji:
Journal Article
MeSH Terms:
Atherosclerosis*/metabolism
Plaque, Atherosclerotic*/pathology
Animals ; Humans ; Mice ; Inflammasomes/metabolism ; Caspases ; Neutrophil Infiltration ; Mice, Knockout ; Apolipoproteins E/genetics ; Apolipoproteins/pharmacology ; Mice, Inbred C57BL
Czasopismo naukowe
Szczegóły
Tytuł:
Galectin-3 contributes to acute cardiac dysfunction and toxicity by increasing oxidative stress and fibrosis in doxorubicin-treated mice.
Autorzy:
Seropian IM; Pontificia Universidad Católica Argentina, Facultad de Medicina, Instituto de Investigaciones Biomédicas UCA-CONICET, Laboratorio de Patología Cardiovascular Experimental e Hipertensión Arterial, Buenos Aires, Argentina; Servicio de Hemodinamia y Cardiología Intervencionista, Hospital Italiano de Buenos Aires, Argentina.
Fontana Estevez FS; Pontificia Universidad Católica Argentina, Facultad de Medicina, Instituto de Investigaciones Biomédicas UCA-CONICET, Laboratorio de Patología Cardiovascular Experimental e Hipertensión Arterial, Buenos Aires, Argentina.
Villaverde A; Pontificia Universidad Católica Argentina, Facultad de Medicina, Instituto de Investigaciones Biomédicas UCA-CONICET, Laboratorio de Patología Cardiovascular Experimental e Hipertensión Arterial, Buenos Aires, Argentina.
Cacciagiú L; Hospital General de Agudos Teodoro Álvarez, Laboratorio Central, Sección Bioquímica, Argentina.
Bustos R; Pontificia Universidad Católica Argentina, Facultad de Medicina, Instituto de Investigaciones Biomédicas UCA-CONICET, Laboratorio de Patología Cardiovascular Experimental e Hipertensión Arterial, Buenos Aires, Argentina.
Touceda V; Pontificia Universidad Católica Argentina, Facultad de Medicina, Instituto de Investigaciones Biomédicas UCA-CONICET, Laboratorio de Patología Cardiovascular Experimental e Hipertensión Arterial, Buenos Aires, Argentina.
Penas F; Instituto de Investigaciones Biomédicas en Retrovirus y SIDA, CONICET-Universidad de Buenos Aires, Buenos Aires, Argentina.
Selser C; Pontificia Universidad Católica Argentina, Facultad de Medicina, Instituto de Investigaciones Biomédicas UCA-CONICET, Laboratorio de Patología Cardiovascular Experimental e Hipertensión Arterial, Buenos Aires, Argentina.
Morales C; Universidad de Buenos Aires, Facultad de Medicina, Departamento de Patología, Buenos Aires, Argentina.
Miksztowicz V; Pontificia Universidad Católica Argentina, Facultad de Medicina, Instituto de Investigaciones Biomédicas UCA-CONICET, Laboratorio de Patología Cardiovascular Experimental e Hipertensión Arterial, Buenos Aires, Argentina.
González GE; Pontificia Universidad Católica Argentina, Facultad de Medicina, Instituto de Investigaciones Biomédicas UCA-CONICET, Laboratorio de Patología Cardiovascular Experimental e Hipertensión Arterial, Buenos Aires, Argentina. Electronic address: .
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Źródło:
International journal of cardiology [Int J Cardiol] 2023 Dec 15; Vol. 393, pp. 131386. Date of Electronic Publication: 2023 Sep 22.
Typ publikacji:
Journal Article
MeSH Terms:
Galectin 3*/genetics
Galectin 3*/metabolism
Cardiomyopathies*/metabolism
Humans ; Mice ; Male ; Animals ; Thiobarbituric Acid Reactive Substances/adverse effects ; Thiobarbituric Acid Reactive Substances/metabolism ; Doxorubicin/toxicity ; Oxidative Stress ; Myocytes, Cardiac/metabolism ; Mice, Knockout ; Cardiotoxicity/metabolism ; Fibrosis ; Inflammation/metabolism ; Apoptosis
Czasopismo naukowe
Szczegóły
Tytuł:
Importance of nuclear localization signal-fused Cas9 in the production of genome-edited mice via embryo electroporation.
Autorzy:
Niinuma S; Division of Science and Engineering, Graduate School of Arts and Science, Iwate University, Iwate, 020-8551, Japan.
Wake Y; Division of Science and Engineering, Graduate School of Arts and Science, Iwate University, Iwate, 020-8551, Japan.
Nakagawa Y; Department of Chemistry and Biological Sciences, Faculty of Science and Engineering, Iwate University, Iwate, 020-8551, Japan.
Kaneko T; Division of Science and Engineering, Graduate School of Arts and Science, Iwate University, Iwate, 020-8551, Japan; Department of Chemistry and Biological Sciences, Faculty of Science and Engineering, Iwate University, Iwate, 020-8551, Japan. Electronic address: .
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Źródło:
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2023 Dec 10; Vol. 685, pp. 149140. Date of Electronic Publication: 2023 Oct 27.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
CRISPR-Cas Systems*/genetics
Nuclear Localization Signals*/genetics
Mice ; Animals ; Mice, Knockout ; Gene Editing/methods ; Electroporation/methods
Czasopismo naukowe
Szczegóły
Tytuł:
TMEM16A deficiency in alveolar type 2 epithelial cells protected against endoplasmic reticulum stress-induced ferroptosis during acute lung injury.
Autorzy:
Jiang W; Department of Thoracic Surgery, Renmin Hospital of Wuhan University, Wuhan, China.
Ren J; Department of Otorhinolaryngology Head and Neck Surgery, Renmin Hospital of Wuhan University, Wuhan, China.
Zhou H; Department of Respiratory and Critical Care Medicine, Renmin Hospital of Wuhan University, Wuhan, China.
He R; Department of Thoracic Surgery, Renmin Hospital of Wuhan University, Wuhan, China.
Li D; Department of Thoracic Surgery, Renmin Hospital of Wuhan University, Wuhan, China.
Xiong R; Department of Thoracic Surgery, Renmin Hospital of Wuhan University, Wuhan, China.
He Z; Department of Thoracic Surgery, Renmin Hospital of Wuhan University, Wuhan, China.
Cheng D; Department of Respiratory and Critical Care Medicine, Renmin Hospital of Wuhan University, Wuhan, China. Electronic address: .
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Źródło:
International immunopharmacology [Int Immunopharmacol] 2023 Dec; Vol. 125 (Pt B), pp. 111208. Date of Electronic Publication: 2023 Nov 15.
Typ publikacji:
Journal Article
MeSH Terms:
Acute Lung Injury*/chemically induced
Ferroptosis*
Sepsis*/pathology
Animals ; Mice ; Alveolar Epithelial Cells/pathology ; Endoplasmic Reticulum Stress ; Lipopolysaccharides/pharmacology ; Lung/pathology ; Mice, Knockout
Czasopismo naukowe
Szczegóły
Tytuł:
The chromatin remodeling protein BRG1 contributes to liver ischemia-reperfusion injury by regulating NOXA expression.
Autorzy:
Li N; Key Laboratory of Targeted Intervention of Cardiovascular Disease and Collaborative Innovation Center for Cardiovascular Translational Medicine, Departments of Human Anatomy and Pathophysiology, Nanjing Medial University, Nanjing, China.
Liu H; Key Laboratory of Targeted Intervention of Cardiovascular Disease and Collaborative Innovation Center for Cardiovascular Translational Medicine, Departments of Human Anatomy and Pathophysiology, Nanjing Medial University, Nanjing, China.
Xue Y; Key Laboratory of Targeted Intervention of Cardiovascular Disease and Collaborative Innovation Center for Cardiovascular Translational Medicine, Departments of Human Anatomy and Pathophysiology, Nanjing Medial University, Nanjing, China.
Zhu Q; Department of General Surgery, Children's Hospital of Nanjing Medical University, Nanjing, China. Electronic address: .
Fan Z; Department of Pathology, Nanjing Drum Tower Hospital, Nanjing, China. Electronic address: .
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Źródło:
Life sciences [Life Sci] 2023 Dec 01; Vol. 334, pp. 122235. Date of Electronic Publication: 2023 Nov 03.
Typ publikacji:
Journal Article
MeSH Terms:
Chromatin Assembly and Disassembly*
Reperfusion Injury*/metabolism
Mice ; Animals ; Liver/metabolism ; Hepatocytes/metabolism ; Apoptosis Regulatory Proteins/metabolism ; Apoptosis ; Mice, Knockout ; Mice, Inbred C57BL
Czasopismo naukowe
Szczegóły
Tytuł:
α-Synuclein is the major platelet isoform but is dispensable for activation, secretion, and thrombosis.
Autorzy:
Smith AN; Department of Molecular and Cellular Biochemistry, College of Medicine, University of Kentucky, Lexington, KY, USA.
Joshi S; Department of Molecular and Cellular Biochemistry, College of Medicine, University of Kentucky, Lexington, KY, USA.
Chanzu H; Department of Molecular and Cellular Biochemistry, College of Medicine, University of Kentucky, Lexington, KY, USA.
Alfar HR; Department of Molecular and Cellular Biochemistry, College of Medicine, University of Kentucky, Lexington, KY, USA.
Shravani Prakhya K; Department of Molecular and Cellular Biochemistry, College of Medicine, University of Kentucky, Lexington, KY, USA.
Whiteheart SW; Department of Molecular and Cellular Biochemistry, College of Medicine, University of Kentucky, Lexington, KY, USA.
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Źródło:
Platelets [Platelets] 2023 Dec; Vol. 34 (1), pp. 2267147. Date of Electronic Publication: 2023 Nov 05.
Typ publikacji:
Journal Article
MeSH Terms:
alpha-Synuclein*/genetics
alpha-Synuclein*/metabolism
Thrombosis*/metabolism
Animals ; Mice ; Blood Platelets/metabolism ; Cytoplasmic Granules/metabolism ; Exocytosis/physiology ; Mice, Knockout ; Platelet Activation ; Protein Isoforms/metabolism ; SNARE Proteins/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
Taste receptor type 1 member 3 mediates diet-induced cognitive impairment in mice.
Autorzy:
Lee KH; Department of Food and Nutrition, College of Human Ecology, Seoul National University, Seoul 08826, Republic of Korea; Department of Environmental Health Sciences, Graduate School of Public Health, Seoul National University, Seoul 08826, Republic of Korea.
Song JW; Department of Food and Nutrition, College of Human Ecology, Seoul National University, Seoul 08826, Republic of Korea.
Kim CS; Department of Food and Nutrition, College of Human Ecology, Seoul National University, Seoul 08826, Republic of Korea.
Seong H; Department of Food and Nutrition, College of Human Ecology, Seoul National University, Seoul 08826, Republic of Korea.
Shin DM; Department of Food and Nutrition, College of Human Ecology, Seoul National University, Seoul 08826, Republic of Korea; Research Institute of Human Ecology, Seoul National University, Seoul 08826, Republic of Korea. Electronic address: .
Shon WJ; Department of Food and Nutrition, College of Human Ecology, Seoul National University, Seoul 08826, Republic of Korea; Research Institute of Human Ecology, Seoul National University, Seoul 08826, Republic of Korea. Electronic address: .
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Źródło:
Life sciences [Life Sci] 2023 Dec 01; Vol. 334, pp. 122194. Date of Electronic Publication: 2023 Oct 20.
Typ publikacji:
Journal Article
MeSH Terms:
Cognitive Dysfunction*/etiology
Diet, Western*
Receptors, G-Protein-Coupled*/metabolism
Animals ; Male ; Mice ; AMP-Activated Protein Kinases/metabolism ; Mice, Inbred C57BL ; Mice, Knockout ; Sirtuin 3/metabolism ; Taste
Czasopismo naukowe
Szczegóły
Tytuł:
Fibroblast growth factor 21 protects the liver from apoptosis in a type 1 diabetes mouse model via regulating L-lactate homeostasis.
Autorzy:
Yan J; School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, Zhejiang, China.
Xie J; School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, Zhejiang, China.
Xu S; School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, Zhejiang, China.
Guo Y; School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, Zhejiang, China.
Ji K; School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, Zhejiang, China.
Li C; School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, Zhejiang, China.
Gao H; School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, Zhejiang, China; Oujiang Laboratory (Zhejiang Lab for Regenerative Medicine, Vision and Brain Health), Wenzhou 325035, Zhejiang, China. Electronic address: .
Zhao L; School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, Zhejiang, China. Electronic address: .
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Źródło:
Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie [Biomed Pharmacother] 2023 Dec; Vol. 168, pp. 115737. Date of Electronic Publication: 2023 Oct 18.
Typ publikacji:
Journal Article
MeSH Terms:
Diabetes Mellitus, Type 1*/drug therapy
Diabetes Mellitus, Type 1*/metabolism
Mice ; Male ; Animals ; AMP-Activated Protein Kinases/metabolism ; Mice, Inbred C57BL ; Liver ; Fibroblast Growth Factors/pharmacology ; Fibroblast Growth Factors/metabolism ; Disease Models, Animal ; Glucose/metabolism ; Homeostasis ; Apoptosis ; Mice, Knockout
Czasopismo naukowe
Szczegóły
Tytuł:
Angiotensin II type 1 receptor-associated protein in immune cells: a possible key factor in the pathogenesis of visceral obesity.
Autorzy:
Tsukamoto S; Department of Medical Science and Cardiorenal Medicine, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Suzuki T; Department of Medical Science and Cardiorenal Medicine, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Wakui H; Department of Medical Science and Cardiorenal Medicine, Yokohama City University Graduate School of Medicine, Yokohama, Japan. Electronic address: .
Uehara T; Department of Medical Science and Cardiorenal Medicine, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Ichikawa J; Department of Immunology, Yokohama City University Graduate School of Medicine, Yokohama, Kanagawa, Japan.
Okuda H; Department of Immunology, Yokohama City University Graduate School of Medicine, Yokohama, Kanagawa, Japan.
Haruhara K; Department of Medical Science and Cardiorenal Medicine, Yokohama City University Graduate School of Medicine, Yokohama, Japan; Division of Nephrology and Hypertension, Department of Internal Medicine, The Jikei University School of Medicine, Tokyo, Japan.
Azushima K; Department of Medical Science and Cardiorenal Medicine, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Abe E; Department of Medical Science and Cardiorenal Medicine, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Tanaka S; Department of Medical Science and Cardiorenal Medicine, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Taguchi S; Department of Medical Science and Cardiorenal Medicine, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Hirota K; Department of Medical Science and Cardiorenal Medicine, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Kinguchi S; Department of Medical Science and Cardiorenal Medicine, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
Yamashita A; Department of Investigative Medicine Graduate School of Medicine, University of the Ryukyus, Okinawa, Japan.
Tamura T; Department of Immunology, Yokohama City University Graduate School of Medicine, Yokohama, Kanagawa, Japan; Advanced Medical Research Center, Yokohama City University, Japan.
Tamura K; Department of Medical Science and Cardiorenal Medicine, Yokohama City University Graduate School of Medicine, Yokohama, Japan.
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Źródło:
Metabolism: clinical and experimental [Metabolism] 2023 Dec; Vol. 149, pp. 155706. Date of Electronic Publication: 2023 Oct 13.
Typ publikacji:
Journal Article
MeSH Terms:
Obesity, Abdominal*/complications
Insulin Resistance*
Mice ; Animals ; Receptor, Angiotensin, Type 1/metabolism ; Adipose Tissue/metabolism ; Obesity/metabolism ; Weight Gain ; Diet, High-Fat ; Mice, Knockout ; Mice, Inbred C57BL
Czasopismo naukowe
Szczegóły
Tytuł:
Brain-derived neurotrophic factor knock-out mice develop non-alcoholic steatohepatitis.
Autorzy:
Ichimura-Shimizu M; Department of Pathology and Laboratory Medicine, Tokushima University Graduate School, Tokushima, Japan.
Kojima M; Department of Applied Bioscience, College of Bioscience and Chemistry, Kanazawa Institute of Technology, Ishikawa, Japan.; Biomedical Research Institute, National Institute of Advanced Industrial Science and Technology (AIST), Osaka, Japan.; Core Research for Evolutional Science and Technology (CREST), Japan Science and Technology Agency (JST), Kawaguchi, Japan.
Suzuki S; Department of Anatomy and Neurobiology, Faculty of Medicine, Kagawa University, Kagawa, Japan.; Health and Medical Research Institute, National Institute of Advanced Industrial Science and Technology (AIST), Takamatsu, Japan.
Miyata M; Department of Applied Bioscience, College of Bioscience and Chemistry, Kanazawa Institute of Technology, Ishikawa, Japan.
Osaki Y; Department of Pathology and Laboratory Medicine, Tokushima University Graduate School, Tokushima, Japan.
Matsui K; Biomedical Research Institute, National Institute of Advanced Industrial Science and Technology (AIST), Osaka, Japan.
Mizui T; Biomedical Research Institute, National Institute of Advanced Industrial Science and Technology (AIST), Osaka, Japan.; Core Research for Evolutional Science and Technology (CREST), Japan Science and Technology Agency (JST), Kawaguchi, Japan.
Tsuneyama K; Department of Pathology and Laboratory Medicine, Tokushima University Graduate School, Tokushima, Japan.
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Źródło:
The Journal of pathology [J Pathol] 2023 Dec; Vol. 261 (4), pp. 465-476. Date of Electronic Publication: 2023 Oct 02.
Typ publikacji:
Journal Article
MeSH Terms:
Non-alcoholic Fatty Liver Disease*/genetics
Non-alcoholic Fatty Liver Disease*/metabolism
Humans ; Mice ; Animals ; Brain-Derived Neurotrophic Factor/genetics ; Brain-Derived Neurotrophic Factor/metabolism ; Mice, Knockout ; Liver/pathology ; Inflammation/pathology ; Obesity/complications ; Mice, Inbred C57BL ; Disease Models, Animal ; Diet, High-Fat
Czasopismo naukowe
Szczegóły
Tytuł:
Sympathetic overdrive and unrestrained adipose lipolysis drive alcohol-induced hepatic steatosis in rodents.
Autorzy:
Zhou C; Department of Medicine and Diabetes, Obesity, and Metabolism Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Ruiz HH; Department of Medicine and Diabetes, Obesity, and Metabolism Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Ling L; Department of Medicine and Diabetes, Obesity, and Metabolism Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Maurizi G; Department of Medicine and Diabetes, Obesity, and Metabolism Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Sakamoto K; Department of Medicine and Diabetes, Obesity, and Metabolism Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA; Division of Endocrinology, Department of Medicine, Rutgers Robert Wood Johnson Medical School, New Brunswick, NJ, USA.
Liberini CG; Department of Medicine and Diabetes, Obesity, and Metabolism Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Wang L; Department of Medicine and Diabetes, Obesity, and Metabolism Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Stanley A; Department of Medicine and Diabetes, Obesity, and Metabolism Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Egritag HE; Department of Medicine and Diabetes, Obesity, and Metabolism Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Sanz SM; Department of Medicine and Diabetes, Obesity, and Metabolism Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Lindtner C; Department of Medicine and Diabetes, Obesity, and Metabolism Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Butera MA; Department of Medicine and Diabetes, Obesity, and Metabolism Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA; Division of Endocrinology, Department of Medicine, Rutgers Robert Wood Johnson Medical School, New Brunswick, NJ, USA.
Buettner C; Department of Medicine and Diabetes, Obesity, and Metabolism Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA; Division of Endocrinology, Department of Medicine, Rutgers Robert Wood Johnson Medical School, New Brunswick, NJ, USA. Electronic address: .
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Źródło:
Molecular metabolism [Mol Metab] 2023 Dec; Vol. 78, pp. 101813. Date of Electronic Publication: 2023 Sep 29.
Typ publikacji:
Journal Article
MeSH Terms:
Fatty Liver, Alcoholic*
Fatty Liver*/pathology
Liver Diseases, Alcoholic*
Rats ; Mice ; Animals ; Lipolysis ; Rodentia/metabolism ; Insulin/metabolism ; Ethanol/adverse effects ; Mice, Knockout ; Obesity
Czasopismo naukowe
Szczegóły
Tytuł:
Conditional loss of Ube3d in the retinal pigment epithelium accelerates age-associated alterations in the retina of mice.
Autorzy:
Tao T; Department of Ophthalmology, Peking University People's Hospital, Eye Diseases and Optometry Institute, Beijing, PR China.; Beijing Key Laboratory of Diagnosis and Therapy of Retinal and Choroid Diseases, Beijing, PR China.; College of Optometry, Peking University Health Science Center, Beijing, PR China.; Department of Ophthalmology, The First Affiliated Hospital of Anhui Medical University, Hefei, PR China.
Xu N; Department of Ophthalmology, Peking University People's Hospital, Eye Diseases and Optometry Institute, Beijing, PR China.; Beijing Key Laboratory of Diagnosis and Therapy of Retinal and Choroid Diseases, Beijing, PR China.; College of Optometry, Peking University Health Science Center, Beijing, PR China.
Li J; Department of Ophthalmology, Peking University People's Hospital, Eye Diseases and Optometry Institute, Beijing, PR China.; Beijing Key Laboratory of Diagnosis and Therapy of Retinal and Choroid Diseases, Beijing, PR China.; College of Optometry, Peking University Health Science Center, Beijing, PR China.
Zhao M; Department of Ophthalmology, Peking University People's Hospital, Eye Diseases and Optometry Institute, Beijing, PR China.; Beijing Key Laboratory of Diagnosis and Therapy of Retinal and Choroid Diseases, Beijing, PR China.; College of Optometry, Peking University Health Science Center, Beijing, PR China.
Li X; Department of Ophthalmology, Peking University People's Hospital, Eye Diseases and Optometry Institute, Beijing, PR China.; Beijing Key Laboratory of Diagnosis and Therapy of Retinal and Choroid Diseases, Beijing, PR China.; College of Optometry, Peking University Health Science Center, Beijing, PR China.; Department of Ophthalmology, Xiamen Eye Center of Xiamen University, Xiamen, PR China.
Huang L; Department of Ophthalmology, Peking University People's Hospital, Eye Diseases and Optometry Institute, Beijing, PR China.; Beijing Key Laboratory of Diagnosis and Therapy of Retinal and Choroid Diseases, Beijing, PR China.; College of Optometry, Peking University Health Science Center, Beijing, PR China.
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Źródło:
The Journal of pathology [J Pathol] 2023 Dec; Vol. 261 (4), pp. 442-454. Date of Electronic Publication: 2023 Sep 29.
Typ publikacji:
Journal Article
MeSH Terms:
Retinal Pigment Epithelium*/metabolism
Macular Degeneration*/genetics
Macular Degeneration*/pathology
Mice ; Humans ; Animals ; Retina/metabolism ; Phagocytosis ; Mice, Knockout ; Proteasome Endopeptidase Complex/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
MiR-484 promotes nonalcoholic fatty liver disease progression in mice via downregulation of Sorbs2.
Autorzy:
Jia Y; Department of Hepatobiliary Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Pan H; Department of Hepatobiliary Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Liu J; Key Laboratory of Coal Science and Technology of Ministry of Education, College of Chemistry and Chemical Engineering, Taiyuan University of Technology, Taiyuan, China.
Huang T; Department of Hepatobiliary Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Han K; Heping Hospital Affiliated to Changzhi Medical College, Changzhi, China.
Mei Q; Institute of Reproductive Health and Center for Reproductive Medicine, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zeng L; Institute of Reproductive Health and Center for Reproductive Medicine, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zhou J; Institute of Reproductive Health and Center for Reproductive Medicine, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zhang Y; Department of Hepatobiliary Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Wuhan National High Magnetic Field Center, Huazhong University of Science and Technology, Wuhan, China.
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Źródło:
Obesity (Silver Spring, Md.) [Obesity (Silver Spring)] 2023 Dec; Vol. 31 (12), pp. 2972-2985. Date of Electronic Publication: 2023 Sep 26.
Typ publikacji:
Journal Article
MeSH Terms:
Non-alcoholic Fatty Liver Disease*/genetics
Non-alcoholic Fatty Liver Disease*/metabolism
MicroRNAs*/genetics
MicroRNAs*/metabolism
Male ; Humans ; Animals ; Mice ; Down-Regulation ; Liver/metabolism ; Hepatocytes/metabolism ; Lipid Metabolism/genetics ; Diet, High-Fat ; Mice, Knockout ; Mice, Inbred C57BL ; RNA-Binding Proteins/genetics ; Adaptor Proteins, Signal Transducing/genetics
Czasopismo naukowe
Szczegóły
Tytuł:
Revisiting the truncated lamin A produced by a commonly used strain of Lmna knockout mice.
Autorzy:
Kim JR; Departments of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
Kim PH; Departments of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
Presnell A; Departments of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
Tu Y; Departments of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
Young SG; Departments of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.; Departments of Human Genetics, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.
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Źródło:
Nucleus (Austin, Tex.) [Nucleus] 2023 Dec; Vol. 14 (1), pp. 2262308. Date of Electronic Publication: 2023 Sep 27.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
MeSH Terms:
Fibroblasts*
Lamin Type A*/genetics
Animals ; Mice ; Cell Nucleus ; Mice, Knockout
Czasopismo naukowe
Szczegóły
Tytuł:
Hyperlipidemia impairs bone regeneration of closed bone defects and tooth extraction wounds in mice.
Autorzy:
Liu W; Department of Periodontology, School and Hospital of Stomatology, Cheeloo College of Medicine, Shandong University & Shandong Key Laboratory of Oral Tissue Regeneration & Shandong Engineering Laboratory for Dental Materials and Oral Tissue Regeneration & Shandong Provincial Clinical Research Center for Oral Diseases, Jinan, China.
Yang J; Department of Dentistry, People's Hospital of Ningxia Hui Autonomous Region, Yinchuan, China.
Li S; Dental Department, Huaihe Hospital of Henan University, Kaifeng, China.
Ye X; Department of Endodontics, School and Hospital of Stomatology, Cheeloo College of Medicine, Shandong University & Shandong Key Laboratory of Oral Tissue Regeneration & Shandong Engineering Laboratory for Dental Materials and Oral Tissue Regeneration & Shandong Provincial Clinical Research Center for Oral Diseases, Jinan, China.
Yang P; Department of Periodontology, School and Hospital of Stomatology, Cheeloo College of Medicine, Shandong University & Shandong Key Laboratory of Oral Tissue Regeneration & Shandong Engineering Laboratory for Dental Materials and Oral Tissue Regeneration & Shandong Provincial Clinical Research Center for Oral Diseases, Jinan, China.
Zhang J; Department of Endodontics, School and Hospital of Stomatology, Cheeloo College of Medicine, Shandong University & Shandong Key Laboratory of Oral Tissue Regeneration & Shandong Engineering Laboratory for Dental Materials and Oral Tissue Regeneration & Shandong Provincial Clinical Research Center for Oral Diseases, Jinan, China.
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Źródło:
Journal of periodontal research [J Periodontal Res] 2023 Dec; Vol. 58 (6), pp. 1201-1211. Date of Electronic Publication: 2023 Aug 16.
Typ publikacji:
Journal Article
MeSH Terms:
Hyperlipidemias*/complications
Alveolar Ridge Augmentation*/methods
Alveolar Bone Loss*/surgery
Animals ; Mice ; Tooth Socket/surgery ; Mice, Inbred C57BL ; Mice, Knockout, ApoE ; Bone Regeneration ; Tooth Extraction/methods ; Apolipoproteins E
Czasopismo naukowe
Szczegóły
Tytuł:
Celastrol as an intestinal FXR inhibitor triggers tripolide-induced intestinal bleeding: Underlying mechanism of gastrointestinal injury induced by Tripterygium wilfordii.
Autorzy:
Dai M; Department of Integrated Traditional Chinese and Western Medicine, Laboratory of Metabolomics and Drug-induced Liver Injury, Frontiers Science Center for Disease-Related Molecular Network, West China Hospital, Sichuan University, Chengdu 610041, China; School of Public Health, Ningbo University Health Science Center, Ningbo 315211, China; State Key Laboratory of Phytochemistry and Plant Resources in West China, Kunming Institute of Botany, Chinese Academy of Sciences, Kunming 650201, China.
Peng W; Department of Integrated Traditional Chinese and Western Medicine, Laboratory of Metabolomics and Drug-induced Liver Injury, Frontiers Science Center for Disease-Related Molecular Network, West China Hospital, Sichuan University, Chengdu 610041, China; Institute of Rare Diseases, West China Hospital of Sichuan University, Chengdu 610041, China.
Lin L; State Key Laboratory of Phytochemistry and Plant Resources in West China, Kunming Institute of Botany, Chinese Academy of Sciences, Kunming 650201, China.
Wu ZE; Department of Integrated Traditional Chinese and Western Medicine, Laboratory of Metabolomics and Drug-induced Liver Injury, Frontiers Science Center for Disease-Related Molecular Network, West China Hospital, Sichuan University, Chengdu 610041, China.
Zhang T; Department of Integrated Traditional Chinese and Western Medicine, Laboratory of Metabolomics and Drug-induced Liver Injury, Frontiers Science Center for Disease-Related Molecular Network, West China Hospital, Sichuan University, Chengdu 610041, China; State Key Laboratory of Phytochemistry and Plant Resources in West China, Kunming Institute of Botany, Chinese Academy of Sciences, Kunming 650201, China.
Zhao Q; Department of Integrated Traditional Chinese and Western Medicine, Laboratory of Metabolomics and Drug-induced Liver Injury, Frontiers Science Center for Disease-Related Molecular Network, West China Hospital, Sichuan University, Chengdu 610041, China.
Cheng Y; Department of Integrated Traditional Chinese and Western Medicine, Laboratory of Metabolomics and Drug-induced Liver Injury, Frontiers Science Center for Disease-Related Molecular Network, West China Hospital, Sichuan University, Chengdu 610041, China.
Lin Q; Department of Integrated Traditional Chinese and Western Medicine, Laboratory of Metabolomics and Drug-induced Liver Injury, Frontiers Science Center for Disease-Related Molecular Network, West China Hospital, Sichuan University, Chengdu 610041, China.
Zhang B; Department of Integrated Traditional Chinese and Western Medicine, Laboratory of Metabolomics and Drug-induced Liver Injury, Frontiers Science Center for Disease-Related Molecular Network, West China Hospital, Sichuan University, Chengdu 610041, China.
Liu A; School of Public Health, Ningbo University Health Science Center, Ningbo 315211, China.
Rao Q; Department of Integrated Traditional Chinese and Western Medicine, Laboratory of Metabolomics and Drug-induced Liver Injury, Frontiers Science Center for Disease-Related Molecular Network, West China Hospital, Sichuan University, Chengdu 610041, China.
Huang J; State Key Laboratory of Phytochemistry and Plant Resources in West China, Kunming Institute of Botany, Chinese Academy of Sciences, Kunming 650201, China.
Zhao J; School of Pharmaceutical Sciences, South-Central Minzu University, Wuhan 430074, China. Electronic address: .
Gonzalez FJ; Center for Cancer Research, National Cancer Institute, NIH, Bethesda, MD 20892, USA.
Li F; Department of Integrated Traditional Chinese and Western Medicine, Laboratory of Metabolomics and Drug-induced Liver Injury, Frontiers Science Center for Disease-Related Molecular Network, West China Hospital, Sichuan University, Chengdu 610041, China; State Key Laboratory of Respiratory Health and Multimorbidity, West China Hospital, Sichuan University, Chengdu, Sichuan 610041, China. Electronic address: .
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Źródło:
Phytomedicine : international journal of phytotherapy and phytopharmacology [Phytomedicine] 2023 Dec; Vol. 121, pp. 155054. Date of Electronic Publication: 2023 Sep 02.
Typ publikacji:
Journal Article
MeSH Terms:
Triterpenes*/chemistry
Animals ; Mice ; Tripterygium/chemistry ; Molecular Docking Simulation ; Gastrointestinal Hemorrhage ; Mice, Knockout
Czasopismo naukowe
Szczegóły
Tytuł:
A+T rich interaction domain protein 3a (Arid3a) impairs Mertk-mediated efferocytosis in cholestasis.
Autorzy:
Chen R; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China.
Huang B; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China.
Lian M; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China.
Wei Y; Department of Gastroenterology and Hepatology, Zhongshan Hospital, Fudan University; 180 Fenglin Road, Shanghai 200032, China.
Miao Q; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China.
Liang J; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China.
Ou Y; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China.
Liang X; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China.
Zhang H; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China.
Li Y; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China.
Xiao X; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China.
Wang Q; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China.
You Z; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China.
Chai J; Department of Gastroenterology, Institute of Digestive Diseases of PLA, Cholestatic Liver Diseases Center and Center for Metabolic Associated Fatty Liver Disease, Southwest Hospital, Third Military Medical University (Army Medical University), Chongqing 400038, China.
Gershwin ME; Division of Rheumatology, Department of Medicine, Allergy and Clinical Immunology, University of California at Davis, Davis, CA, USA. Electronic address: .
Tang R; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China. Electronic address: ruqi_.
Ma X; Division of Gastroenterology and Hepatology, Key Laboratory of Gastroenterology and Hepatology, Ministry of Health, State Key Laboratory for Oncogenes and Related Genes, Renji Hospital, School of Medicine, Shanghai Jiao Tong University; Shanghai Institute of Digestive Disease; 145 Middle Shandong Road, Shanghai 200001, China; Institute of Aging & Tissue Regeneration, Renji Hospital, Shanghai Jiaotong University School of Medicine, Shanghai, China. Electronic address: .
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Źródło:
Journal of hepatology [J Hepatol] 2023 Dec; Vol. 79 (6), pp. 1478-1490. Date of Electronic Publication: 2023 Sep 01.
Typ publikacji:
Journal Article
MeSH Terms:
Cholestasis*/metabolism
Liver Diseases*/metabolism
Mice ; Animals ; c-Mer Tyrosine Kinase/genetics ; c-Mer Tyrosine Kinase/metabolism ; Phagocytosis/physiology ; Macrophages/metabolism ; Mice, Knockout
Czasopismo naukowe
Szczegóły
Tytuł:
Therapeutic role of mesenchymal stem cell-derived extracellular vesicles in neuroinflammation and cognitive dysfunctions induced by binge-like ethanol treatment in adolescent mice.
Autorzy:
Mellado S; Department of Physiology, School of Medicine and Dentistry, University of Valencia, Valencia, Spain.
Cuesta CM; Department of Physiology, School of Medicine and Dentistry, University of Valencia, Valencia, Spain.
Montagud S; Department of Psychobiology, Facultad de Psicología, Universitat de Valencia, Valencia, Spain.
Rodríguez-Arias M; Department of Psychobiology, Facultad de Psicología, Universitat de Valencia, Valencia, Spain.
Moreno-Manzano V; Neuronal and Tissue Regeneration Laboratory, Príncipe Felipe Research Center, Valencia, Spain.
Guerri C; Príncipe Felipe Research Center, Valencia, Spain.
Pascual M; Department of Physiology, School of Medicine and Dentistry, University of Valencia, Valencia, Spain.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2023 Dec; Vol. 29 (12), pp. 4018-4031. Date of Electronic Publication: 2023 Jun 28.
Typ publikacji:
Journal Article
MeSH Terms:
Extracellular Vesicles*
Cognitive Dysfunction*/therapy
Mesenchymal Stem Cells*
Mice ; Animals ; Female ; Ethanol/toxicity ; Neuroinflammatory Diseases ; Mice, Knockout
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
TRAF5 regulates intestinal mucosal Th1/Th17 cell immune responses via Runx1 in colitis mice.
Autorzy:
Li M; Department of Gastroenterology, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Clinical Center and Key Lab of Intestinal and Colorectal Diseases, Wuhan, China.
Gan C; Department of Gastroenterology, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Clinical Center and Key Lab of Intestinal and Colorectal Diseases, Wuhan, China.
Zhang R; Department of Gastroenterology, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Clinical Center and Key Lab of Intestinal and Colorectal Diseases, Wuhan, China.
Wang J; Department of Gastroenterology, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Clinical Center and Key Lab of Intestinal and Colorectal Diseases, Wuhan, China.
Wang Y; Department of Gastroenterology, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Clinical Center and Key Lab of Intestinal and Colorectal Diseases, Wuhan, China.
Zhu W; Department of Gastroenterology, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Clinical Center and Key Lab of Intestinal and Colorectal Diseases, Wuhan, China.
Liu L; Department of Gastroenterology, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Clinical Center and Key Lab of Intestinal and Colorectal Diseases, Wuhan, China.
Shang J; Department of Gastroenterology, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Clinical Center and Key Lab of Intestinal and Colorectal Diseases, Wuhan, China.
Zhao Q; Department of Gastroenterology, Zhongnan Hospital of Wuhan University, Wuhan, China.; Hubei Clinical Center and Key Lab of Intestinal and Colorectal Diseases, Wuhan, China.
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Źródło:
Immunology [Immunology] 2023 Dec; Vol. 170 (4), pp. 495-509. Date of Electronic Publication: 2023 Aug 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Colitis*
Inflammatory Bowel Diseases*
Animals ; Mice ; Th17 Cells ; TNF Receptor-Associated Factor 5/metabolism ; Intestinal Mucosa ; Immunity ; Th1 Cells ; Mice, Inbred C57BL ; CD4-Positive T-Lymphocytes ; Mice, Knockout ; Disease Models, Animal ; Core Binding Factor Alpha 2 Subunit/genetics ; Core Binding Factor Alpha 2 Subunit/metabolism
Czasopismo naukowe
Szczegóły
Wyświetlanie 1-20 z 75282

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