Temat: "Mice, Transgenic" - OpacWWW

Skocz do pozycji: 61.

Tytuł:: A Novel Cre Recombinase Mouse Strain for Cell-Specific Deletion of Floxed Genes in Ribbon Synapse-Forming Retinal Neurons.
Autorzy:: Suiwal S; Institute of Anatomy and Cell Biology, Department of Neuroanatomy, Medical School, Saarland University, 66421 Homburg, Germany.
Wartenberg P; Institute of Clinical and Experimental Pharmacology, Center for Molecular Signaling (PZMS) and Center for Gender-Specific Biology and Medicine (CGBM), Medical School, Saarland University, 66421 Homburg, Germany.
Boehm U; Institute of Clinical and Experimental Pharmacology, Center for Molecular Signaling (PZMS) and Center for Gender-Specific Biology and Medicine (CGBM), Medical School, Saarland University, 66421 Homburg, Germany.
Schmitz F; Institute of Anatomy and Cell Biology, Department of Neuroanatomy, Medical School, Saarland University, 66421 Homburg, Germany.
Schwarz K; Institute of Anatomy and Cell Biology, Department of Neuroanatomy, Medical School, Saarland University, 66421 Homburg, Germany.; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2024 Feb 05; Vol. 25 (3). Date of Electronic Publication: 2024 Feb 05.
Typ publikacji:: Journal Article
MeSH Terms:: tau Proteins*/metabolism
Retinal Neurons*/metabolism
Mice ; Animals ; Mice, Transgenic ; Synapses/metabolism ; Integrases/genetics ; Integrases/metabolism ; Green Fluorescent Proteins/metabolism

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Skocz do pozycji: 62.

Tytuł:: Perinatal choline supplementation prevents learning and memory deficits and reduces brain amyloid Aβ42 deposition in AppNL-G-F Alzheimer's disease model mice.
Autorzy:: Bellio TA; Department of Pathology & Laboratory Medicine, Boston University Chobanian & Avedisian School of Medicine, Boston, Massachusetts, United States of America.
Laguna-Torres JY; Department of Pathology & Laboratory Medicine, Boston University Chobanian & Avedisian School of Medicine, Boston, Massachusetts, United States of America.
Campion MS; Department of Pathology & Laboratory Medicine, Boston University Chobanian & Avedisian School of Medicine, Boston, Massachusetts, United States of America.
Chou J; Department of Pathology & Laboratory Medicine, Boston University Chobanian & Avedisian School of Medicine, Boston, Massachusetts, United States of America.
Yee S; Department of Pathology & Laboratory Medicine, Boston University Chobanian & Avedisian School of Medicine, Boston, Massachusetts, United States of America.
Blusztajn JK; Department of Pathology & Laboratory Medicine, Boston University Chobanian & Avedisian School of Medicine, Boston, Massachusetts, United States of America.
Mellott TJ; Department of Pathology & Laboratory Medicine, Boston University Chobanian & Avedisian School of Medicine, Boston, Massachusetts, United States of America.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2024 Feb 05; Vol. 19 (2), pp. e0297289. Date of Electronic Publication: 2024 Feb 05 (Print Publication: 2024).
Typ publikacji:: Journal Article
MeSH Terms:: Alzheimer Disease*/drug therapy
Alzheimer Disease*/prevention & control
Alzheimer Disease*/pathology
Amyloidosis*/pathology
Pregnancy ; Female ; Mice ; Animals ; Mice, Transgenic ; Lactation ; Disease Models, Animal ; Brain/metabolism ; Choline/pharmacology ; Memory Disorders/drug therapy ; Memory Disorders/prevention & control ; Memory Disorders/pathology ; Maze Learning ; Dietary Supplements ; Amyloid beta-Peptides/metabolism

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Skocz do pozycji: 63.

Tytuł:: Altered amyloid-β structure markedly reduces gliosis in the brain of mice harboring the Uppsala APP deletion.
Autorzy:: Pagnon de la Vega M; Department of Public Health and Caring Sciences, Geriatrics, Uppsala University, Uppsala, Sweden.
Syvänen S; Department of Public Health and Caring Sciences, Geriatrics, Uppsala University, Uppsala, Sweden.
Giedraitis V; Department of Public Health and Caring Sciences, Geriatrics, Uppsala University, Uppsala, Sweden.
Hooley M; UK Dementia Research Institute, Edinburgh Medical School, University of Edinburgh, Edinburgh, UK.; Centre for Discovery Brain Sciences, University of Edinburgh, Edinburgh, UK.
Konstantinidis E; Department of Public Health and Caring Sciences, Geriatrics, Uppsala University, Uppsala, Sweden.
Meier SR; Department of Public Health and Caring Sciences, Geriatrics, Uppsala University, Uppsala, Sweden.
Rokka J; Department of Public Health and Caring Sciences, Geriatrics, Uppsala University, Uppsala, Sweden.
Eriksson J; Department of Medicinal Chemistry, Division of Organic Pharmaceutical Chemistry, Uppsala University, Uppsala, Sweden.; PET Centre, Uppsala University Hospital, Uppsala, Sweden.
Aguilar X; Department of Public Health and Caring Sciences, Geriatrics, Uppsala University, Uppsala, Sweden.
Spires-Jones TL; UK Dementia Research Institute, Edinburgh Medical School, University of Edinburgh, Edinburgh, UK.; Centre for Discovery Brain Sciences, University of Edinburgh, Edinburgh, UK.
Lannfelt L; Department of Public Health and Caring Sciences, Geriatrics, Uppsala University, Uppsala, Sweden.; BioArctic AB, Stockholm, Sweden.
Nilsson LNG; Department of Pharmacology, University of Oslo and Oslo University Hospital, Oslo, Norway.
Erlandsson A; Department of Public Health and Caring Sciences, Geriatrics, Uppsala University, Uppsala, Sweden.
Hultqvist G; Department of Pharmacy, Uppsala University, Uppsala, Sweden.
Ingelsson M; Department of Public Health and Caring Sciences, Geriatrics, Uppsala University, Uppsala, Sweden.; Krembil Brain Institute, University Health Network, Toronto, ON, Canada.; Tanz Centre for Research in Neurodegenerative Diseases, Departments of Medicine and Laboratory Medicine and Pathobiology, University of Toronto, Toronto, ON, Canada.
Sehlin D; Department of Public Health and Caring Sciences, Geriatrics, Uppsala University, Uppsala, Sweden. .; Pokaż więcej
Źródło:: Acta neuropathologica communications [Acta Neuropathol Commun] 2024 Feb 05; Vol. 12 (1), pp. 22. Date of Electronic Publication: 2024 Feb 05.
Typ publikacji:: Journal Article
MeSH Terms:: Alzheimer Disease*/pathology
Amyloid beta-Peptides*/metabolism
Animals ; Humans ; Mice ; Amyloid beta-Protein Precursor/genetics ; Amyloid beta-Protein Precursor/metabolism ; Amyloid Precursor Protein Secretases/metabolism ; Brain/pathology ; Disease Models, Animal ; Gliosis/pathology ; Ligands ; Mice, Transgenic

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Skocz do pozycji: 64.

Tytuł:: Intersectional strategy to study cortical inhibitory parvalbumin-expressing interneurons.
Autorzy:: Palicz R; Center Anatomy, Institute for Neuroanatomy, University of Göttingen, Göttingen, Germany. .
Pater B; Center Anatomy, Institute for Neuroanatomy, University of Göttingen, Göttingen, Germany.
Truschow P; Center Anatomy, Institute for Neuroanatomy, University of Göttingen, Göttingen, Germany.
Witte M; Center Anatomy, Institute for Neuroanatomy, University of Göttingen, Göttingen, Germany.
Staiger JF; Center Anatomy, Institute for Neuroanatomy, University of Göttingen, Göttingen, Germany.; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2024 Feb 03; Vol. 14 (1), pp. 2829. Date of Electronic Publication: 2024 Feb 03.
Typ publikacji:: Journal Article
MeSH Terms:: Parvalbumins*/metabolism
Interneurons*/metabolism
Red Fluorescent Protein*
Mice ; Animals ; Neurons/metabolism ; Mice, Transgenic

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Skocz do pozycji: 65.

Tytuł:: Tmem119 expression is downregulated in a subset of brain metastasis-associated microglia.
Autorzy:: Ma W; Immunology, Metastasis and Microenvironment Program, The Wistar Institute, 3601 Spruce Street, 19104, Philadelphia, PA, USA. .
Oswald J; Immunology, Metastasis and Microenvironment Program, The Wistar Institute, 3601 Spruce Street, 19104, Philadelphia, PA, USA.
Rios Angulo A; Immunology, Metastasis and Microenvironment Program, The Wistar Institute, 3601 Spruce Street, 19104, Philadelphia, PA, USA.
Chen Q; Immunology, Metastasis and Microenvironment Program, The Wistar Institute, 3601 Spruce Street, 19104, Philadelphia, PA, USA. .; Pokaż więcej
Źródło:: BMC neuroscience [BMC Neurosci] 2024 Feb 02; Vol. 25 (1), pp. 6. Date of Electronic Publication: 2024 Feb 02.
Typ publikacji:: Journal Article
MeSH Terms:: Brain Neoplasms*/metabolism
Microglia*/metabolism
Animals ; Mice ; Brain/metabolism ; Macrophages/metabolism ; Mice, Transgenic ; Tumor Microenvironment

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Skocz do pozycji: 66.

Tytuł:: HDAC9-mediated calmodulin deacetylation induces memory impairment in Alzheimer's disease.
Autorzy:: Zhang HL; Jiangsu Key Laboratory of Neuropsychiatric Diseases, Institute of Neuroscience, Suzhou Medical College of Soochow University, Medical Center of Soochow University, Suzhou, China.
Hu S; Jiangsu Key Laboratory of Neuropsychiatric Diseases, Institute of Neuroscience, Suzhou Medical College of Soochow University, Medical Center of Soochow University, Suzhou, China.
Yang P; Key Laboratory of Brain Functional Genomics, Ministry of Education and Shanghai, School of Life Science, East China Normal University, Shanghai, China.
Long HC; Department of Neurology, The Affiliated Xingyi City Hospital of Guizhou Medical University, Xingyi, China.
Ma QH; Jiangsu Key Laboratory of Neuropsychiatric Diseases, Institute of Neuroscience, Suzhou Medical College of Soochow University, Medical Center of Soochow University, Suzhou, China.
Yin DM; Key Laboratory of Brain Functional Genomics, Ministry of Education and Shanghai, School of Life Science, East China Normal University, Shanghai, China.
Xu GY; Jiangsu Key Laboratory of Neuropsychiatric Diseases, Institute of Neuroscience, Suzhou Medical College of Soochow University, Medical Center of Soochow University, Suzhou, China.; Pokaż więcej
Źródło:: CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Feb; Vol. 30 (2), pp. e14573.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Alzheimer Disease*/metabolism
Neurodegenerative Diseases*
Mice ; Humans ; Animals ; Calmodulin ; Mice, Transgenic ; Memory Disorders/etiology ; Hippocampus/metabolism ; Disease Models, Animal ; Histone Deacetylases/metabolism ; Repressor Proteins/metabolism

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Skocz do pozycji: 67.

Tytuł:: Targeting blood brain barrier-Remote ischemic conditioning alleviates cognitive impairment in female APP/PS1 rats.
Autorzy:: Ma Y; International Science & Technology Cooperation Base of Geriatric, School of Public Health of North China University of Science and Technology, Tangshan, Hebei, China.
Sun W; School of Basic Medical Science, North China University of Science and Technology, Tangshan, Hebei, China.
Bai J; School of Basic Medical Science, North China University of Science and Technology, Tangshan, Hebei, China.
Gao F; International Science & Technology Cooperation Base of Geriatric, School of Public Health of North China University of Science and Technology, Tangshan, Hebei, China.
Ma H; International Science & Technology Cooperation Base of Geriatric, School of Public Health of North China University of Science and Technology, Tangshan, Hebei, China.
Liu H; International Science & Technology Cooperation Base of Geriatric, School of Public Health of North China University of Science and Technology, Tangshan, Hebei, China.
Hu J; School of Basic Medical Science, North China University of Science and Technology, Tangshan, Hebei, China.
Xu C; International Science & Technology Cooperation Base of Geriatric, School of Public Health of North China University of Science and Technology, Tangshan, Hebei, China.
Zhang X; International Science & Technology Cooperation Base of Geriatric, School of Public Health of North China University of Science and Technology, Tangshan, Hebei, China.
Liu Z; School of Basic Medical Science, North China University of Science and Technology, Tangshan, Hebei, China.
Yuan T; International Science & Technology Cooperation Base of Geriatric, School of Public Health of North China University of Science and Technology, Tangshan, Hebei, China.
Sun C; School of Basic Medical Science, North China University of Science and Technology, Tangshan, Hebei, China.
Huang Y; School of Basic Medical Science, North China University of Science and Technology, Tangshan, Hebei, China.
Wang R; International Science & Technology Cooperation Base of Geriatric, School of Public Health of North China University of Science and Technology, Tangshan, Hebei, China.; School of Basic Medical Science, North China University of Science and Technology, Tangshan, Hebei, China.; Pokaż więcej
Źródło:: CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Feb; Vol. 30 (2), pp. e14613.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Alzheimer Disease*/drug therapy
Cognitive Dysfunction*/diagnostic imaging
Cognitive Dysfunction*/therapy
Mice ; Rats ; Female ; Animals ; Blood-Brain Barrier/metabolism ; Amyloid beta-Peptides/metabolism ; Amyloid beta-Protein Precursor/genetics ; Amyloid beta-Protein Precursor/metabolism ; Amyloid Precursor Protein Secretases/genetics ; Mice, Transgenic ; Rats, Transgenic ; Aspartic Acid Endopeptidases/genetics ; Aspartic Acid Endopeptidases/metabolism ; Aspartic Acid Endopeptidases/therapeutic use ; Disease Models, Animal ; Presenilin-1/genetics ; Presenilin-1/metabolism

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Skocz do pozycji: 68.

Tytuł:: Pathological features and molecular signatures of early olfactory dysfunction in 3xTg-AD model mice.
Autorzy:: Yu H; Department of Fundamental Medicine, Wuxi School of Medicine, Jiangnan University, Wuxi, Jiangsu, P. R. China.; Affiliated Hospital of Jiangnan University, Wuxi, Jiangsu Province, P. R. China.
Wang F; Department of Fundamental Medicine, Wuxi School of Medicine, Jiangnan University, Wuxi, Jiangsu, P. R. China.
Jia D; The Affiliated Mental Health Center of Jiangnan University, Wuxi Central Rehabilitation Hospital, Wuxi, Jiangsu, P. R. China.
Bi S; Department of Fundamental Medicine, Wuxi School of Medicine, Jiangnan University, Wuxi, Jiangsu, P. R. China.
Gong J; Department of Fundamental Medicine, Wuxi School of Medicine, Jiangnan University, Wuxi, Jiangsu, P. R. China.
Wu JJ; Department of Fundamental Medicine, Wuxi School of Medicine, Jiangnan University, Wuxi, Jiangsu, P. R. China.
Mao Y; Department of Fundamental Medicine, Wuxi School of Medicine, Jiangnan University, Wuxi, Jiangsu, P. R. China.
Chen J; Department of Fundamental Medicine, Wuxi School of Medicine, Jiangnan University, Wuxi, Jiangsu, P. R. China.
Chai GS; Department of Fundamental Medicine, Wuxi School of Medicine, Jiangnan University, Wuxi, Jiangsu, P. R. China.; Pokaż więcej
Źródło:: CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Feb; Vol. 30 (2), pp. e14632.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Alzheimer Disease*/complications
Alzheimer Disease*/genetics
Alzheimer Disease*/metabolism
Olfaction Disorders*/genetics
Mice ; Animals ; Male ; Smell ; Neuroinflammatory Diseases ; Amyloid beta-Peptides/metabolism ; Mice, Transgenic ; Disease Models, Animal ; tau Proteins/genetics ; tau Proteins/metabolism

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Skocz do pozycji: 69.

Tytuł:: NCAPH drives breast cancer progression and identifies a gene signature that predicts luminal a tumour recurrence.
Autorzy:: Mendiburu-Eliçabe M; Instituto de Biología Molecular y Celular del Cáncer (IBMCC-CIC), Universidad de Salamanca/CSIC, Salamanca, Spain.; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.
García-Sancha N; Instituto de Biología Molecular y Celular del Cáncer (IBMCC-CIC), Universidad de Salamanca/CSIC, Salamanca, Spain.; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.
Corchado-Cobos R; Instituto de Biología Molecular y Celular del Cáncer (IBMCC-CIC), Universidad de Salamanca/CSIC, Salamanca, Spain.; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.
Martínez-López A; Departamento de Bioquímica y Biología Molecular, Facultad de Ciencias Químicas, Universidad Complutense, Madrid, Spain.; San Carlos Health Research Institute (IdISSC), Madrid, Spain.
Chang H; Biological Systems and Engineering Division, Lawrence Berkeley National Laboratory (LBNL), Berkeley, California, USA.; Berkeley Biomedical Data Science Center, Lawrence Berkeley National Laboratory (LBNL), Berkeley, California, USA.
Hua Mao J; Biological Systems and Engineering Division, Lawrence Berkeley National Laboratory (LBNL), Berkeley, California, USA.; Berkeley Biomedical Data Science Center, Lawrence Berkeley National Laboratory (LBNL), Berkeley, California, USA.
Blanco-Gómez A; Instituto de Biología Molecular y Celular del Cáncer (IBMCC-CIC), Universidad de Salamanca/CSIC, Salamanca, Spain.; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.
García-Casas A; Departamento de Bioquímica y Biología Molecular, Facultad de Ciencias Químicas, Universidad Complutense, Madrid, Spain.; San Carlos Health Research Institute (IdISSC), Madrid, Spain.
Castellanos-Martín A; Instituto de Biología Molecular y Celular del Cáncer (IBMCC-CIC), Universidad de Salamanca/CSIC, Salamanca, Spain.; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.
Salvador N; Departamento de Bioquímica y Biología Molecular, Facultad de Ciencias Químicas, Universidad Complutense, Madrid, Spain.; San Carlos Health Research Institute (IdISSC), Madrid, Spain.
Jiménez-Navas A; Instituto de Biología Molecular y Celular del Cáncer (IBMCC-CIC), Universidad de Salamanca/CSIC, Salamanca, Spain.; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.
Pérez-Baena MJ; Instituto de Biología Molecular y Celular del Cáncer (IBMCC-CIC), Universidad de Salamanca/CSIC, Salamanca, Spain.; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.
Sánchez-Martín MA; Departamento de Medicina, Universidad de Salamanca, Salamanca, Spain.; Servicio de Transgénesis, Plataforma Nucleus, Universidad de Salamanca, Salamanca, Spain.
Abad-Hernández MDM; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.; Departamento de Anatomía Patológica, Universidad de Salamanca, Salamanca, Spain.; Servicio de Anatomía Patológica, Hospital Universitario de Salamanca, Salamanca, Spain.
Carmen SD; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.; Departamento de Anatomía Patológica, Universidad de Salamanca, Salamanca, Spain.; Servicio de Anatomía Patológica, Hospital Universitario de Salamanca, Salamanca, Spain.
Claros-Ampuero J; Instituto de Biología Molecular y Celular del Cáncer (IBMCC-CIC), Universidad de Salamanca/CSIC, Salamanca, Spain.; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.; Servicio de Oncología, Hospital Universitario de Salamanca, Salamanca, Spain.
Cruz-Hernández JJ; Instituto de Biología Molecular y Celular del Cáncer (IBMCC-CIC), Universidad de Salamanca/CSIC, Salamanca, Spain.; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.; Departamento de Medicina, Universidad de Salamanca, Salamanca, Spain.; Servicio de Oncología, Hospital Universitario de Salamanca, Salamanca, Spain.
Rodríguez-Sánchez CA; Instituto de Biología Molecular y Celular del Cáncer (IBMCC-CIC), Universidad de Salamanca/CSIC, Salamanca, Spain.; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.; Departamento de Medicina, Universidad de Salamanca, Salamanca, Spain.; Servicio de Oncología, Hospital Universitario de Salamanca, Salamanca, Spain.
García-Cenador MB; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.; Departamento de Cirugía, Universidad de Salamanca, Salamanca, Spain.
García-Criado FJ; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.; Departamento de Cirugía, Universidad de Salamanca, Salamanca, Spain.
Vicente RS; Departamento de Informática y Automática, Universidad de Salamanca, Salamanca, España.
Castillo-Lluva S; Departamento de Bioquímica y Biología Molecular, Facultad de Ciencias Químicas, Universidad Complutense, Madrid, Spain.; San Carlos Health Research Institute (IdISSC), Madrid, Spain.
Pérez-Losada J; Instituto de Biología Molecular y Celular del Cáncer (IBMCC-CIC), Universidad de Salamanca/CSIC, Salamanca, Spain.; Biosanitary Research Institute of Salamanca (IBSAL), Salamanca, Spain.; Pokaż więcej
Źródło:: Clinical and translational medicine [Clin Transl Med] 2024 Feb; Vol. 14 (2), pp. e1554.
Typ publikacji:: Journal Article
MeSH Terms:: Breast Neoplasms*/drug therapy
Humans ; Mice ; Animals ; Female ; Neoplasm Recurrence, Local/genetics ; Gene Expression Profiling ; Prognosis ; Mice, Transgenic ; Nuclear Proteins/genetics ; Cell Cycle Proteins/genetics

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Skocz do pozycji: 70.

Tytuł:: Pericardial delta like non-canonical NOTCH ligand 1 (Dlk1) augments fibrosis in the heart through epithelial to mesenchymal transition.
Autorzy:: Jensen CH; Andersen Group, Department of Clinical Biochemistry, Odense University Hospital, Odense, Denmark.; Clinical Institute, University of Southern Denmark, Odense, Denmark.
Johnsen RH; Andersen Group, Department of Clinical Biochemistry, Odense University Hospital, Odense, Denmark.; Clinical Institute, University of Southern Denmark, Odense, Denmark.
Eskildsen T; Andersen Group, Department of Clinical Biochemistry, Odense University Hospital, Odense, Denmark.; Department of Cardiovascular and Renal Research, Institute of Molecular Medicine, University of Southern Denmark, Odense, Denmark.
Baun C; Department of Nuclear Medicine, Odense University Hospital, Odense, Denmark.
Ellman DG; Andersen Group, Department of Clinical Biochemistry, Odense University Hospital, Odense, Denmark.; Clinical Institute, University of Southern Denmark, Odense, Denmark.
Fang S; Andersen Group, Department of Clinical Biochemistry, Odense University Hospital, Odense, Denmark.; Clinical Institute, University of Southern Denmark, Odense, Denmark.
Bak ST; Andersen Group, Department of Clinical Biochemistry, Odense University Hospital, Odense, Denmark.; Clinical Institute, University of Southern Denmark, Odense, Denmark.
Hvidsten S; Department of Nuclear Medicine, Odense University Hospital, Odense, Denmark.
Larsen LA; Department of Cellular and Molecular Medicine, University of Copenhagen, Copenhagen, Denmark.
Rosager AM; Department of Clinical Pathology, Sydvestjysk Hospital, Esbjerg, Denmark.
Riber LP; Clinical Institute, University of Southern Denmark, Odense, Denmark.; Department of Cardiothoracic and Vascular Surgery, Odense University Hospital, Odense, Denmark.
Schneider M; Andersen Group, Department of Clinical Biochemistry, Odense University Hospital, Odense, Denmark.; Clinical Institute, University of Southern Denmark, Odense, Denmark.; Department of Cardiovascular and Renal Research, Institute of Molecular Medicine, University of Southern Denmark, Odense, Denmark.
De Mey J; Department of Cardiovascular and Renal Research, Institute of Molecular Medicine, University of Southern Denmark, Odense, Denmark.
Thomassen M; Clinical Institute, University of Southern Denmark, Odense, Denmark.; Department of Clinical Genetics, Odense University Hospital, Odense, Denmark.
Burton M; Clinical Institute, University of Southern Denmark, Odense, Denmark.; Department of Clinical Genetics, Odense University Hospital, Odense, Denmark.
Uchida S; Center for RNA Medicine, Department of Clinical Medicine, Aalborg University, Copenhagen, Denmark.
Laborda J; Department of Inorganic and Organic Chemistry and Biochemistry, University of Castilla-La Mancha Medical School, Albacete, Spain.
Andersen DC; Andersen Group, Department of Clinical Biochemistry, Odense University Hospital, Odense, Denmark.; Clinical Institute, University of Southern Denmark, Odense, Denmark.; Department of Cardiovascular and Renal Research, Institute of Molecular Medicine, University of Southern Denmark, Odense, Denmark.; Pokaż więcej
Źródło:: Clinical and translational medicine [Clin Transl Med] 2024 Feb; Vol. 14 (2), pp. e1565.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Epithelial-Mesenchymal Transition*/genetics
Myocardial Infarction*/genetics
Adult ; Animals ; Humans ; Mice ; Calcium-Binding Proteins/genetics ; Calcium-Binding Proteins/metabolism ; Cicatrix/metabolism ; Cicatrix/pathology ; Fibrosis ; Ligands ; Mice, Transgenic ; Pericardium/metabolism ; Thorax/pathology

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Skocz do pozycji: 71.

Tytuł:: Comparison of IL-2-antibody to IL-2-Fc with or without stereotactic radiation therapy in CEA immunocompetent mice with CEA positive tumors.
Autorzy:: Williams L; Department of Immunology and Theranostics, Riggs Diabetes, Metabolism, and Research Institute, Beckman Research Institute of City of Hope, Duarte, California, USA.
Li L; Department of Immunology and Theranostics, Riggs Diabetes, Metabolism, and Research Institute, Beckman Research Institute of City of Hope, Duarte, California, USA.
Yazaki PJ; Department of Immunology and Theranostics, Riggs Diabetes, Metabolism, and Research Institute, Beckman Research Institute of City of Hope, Duarte, California, USA.
Wong P; Department of Immunology and Theranostics, Riggs Diabetes, Metabolism, and Research Institute, Beckman Research Institute of City of Hope, Duarte, California, USA.
Hong T; Department of Immunology and Theranostics, Riggs Diabetes, Metabolism, and Research Institute, Beckman Research Institute of City of Hope, Duarte, California, USA.
Poku EK; Radiopharmacy, City of Hope, Duarte, California, USA.; Beckman Research Institute of City of Hope, Duarte, California, USA.
Hui S; Department of Radiation Oncology, City of Hope, Duarte, California, USA.; City of Hope Medical Center, Duarte, California, USA.
Ghimire H; Department of Radiation Oncology, City of Hope, Duarte, California, USA.; City of Hope Medical Center, Duarte, California, USA.
Shively JE; Department of Immunology and Theranostics, Riggs Diabetes, Metabolism, and Research Institute, Beckman Research Institute of City of Hope, Duarte, California, USA.
Kujawski M; Department of Immunology and Theranostics, Riggs Diabetes, Metabolism, and Research Institute, Beckman Research Institute of City of Hope, Duarte, California, USA.; Pokaż więcej
Źródło:: Cancer medicine [Cancer Med] 2024 Feb; Vol. 13 (3), pp. e6909. Date of Electronic Publication: 2024 Feb 05.
Typ publikacji:: Journal Article
MeSH Terms:: Radiosurgery*
Neoplasms*/therapy
Mice ; Animals ; Interleukin-2/pharmacology ; CD8-Positive T-Lymphocytes ; Antibodies ; Mice, Transgenic

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Skocz do pozycji: 72.

Tytuł:: Leptin haploinsufficiency exerts sex-dependent partial protection in SOD1 mice by reducing inflammatory pathways in the adipose tissue.
Autorzy:: Fernández-Beltrán LC; Neurological Disorders Group, Department of Neurology, Hospital Clínico San Carlos, Instituto de Investigación Sanitaria San Carlos (IdISSC), Madrid, Spain.; Department of Medicine, Universidad Complutense de Madrid, Madrid, Spain.
Ali Z; Neurological Disorders Group, Department of Neurology, Hospital Clínico San Carlos, Instituto de Investigación Sanitaria San Carlos (IdISSC), Madrid, Spain.; Mary Lyon Centre at MRC Harwell, Oxfordshire, UK.
Larrad-Sanz A; Department of Endocrinology and Nutrition, Hospital Clínico San Carlos, Instituto de Investigación Sanitaria San Carlos (IdISSC), Madrid, Spain.
Lopez-Carbonero JI; Neurological Disorders Group, Department of Neurology, Hospital Clínico San Carlos, Instituto de Investigación Sanitaria San Carlos (IdISSC), Madrid, Spain.
Godoy-Corchuelo JM; Neurological Disorders Group, Department of Neurology, Hospital Clínico San Carlos, Instituto de Investigación Sanitaria San Carlos (IdISSC), Madrid, Spain.
Jimenez-Coca I; Neurological Disorders Group, Department of Neurology, Hospital Clínico San Carlos, Instituto de Investigación Sanitaria San Carlos (IdISSC), Madrid, Spain.
Garcia-Toledo I; Neurological Disorders Group, Department of Neurology, Hospital Clínico San Carlos, Instituto de Investigación Sanitaria San Carlos (IdISSC), Madrid, Spain.
Bentley L; Mary Lyon Centre at MRC Harwell, Oxfordshire, UK.
Gomez-Pinedo U; Neurological Disorders Group, Department of Neurology, Hospital Clínico San Carlos, Instituto de Investigación Sanitaria San Carlos (IdISSC), Madrid, Spain.
Matias-Guiu JA; Neurological Disorders Group, Department of Neurology, Hospital Clínico San Carlos, Instituto de Investigación Sanitaria San Carlos (IdISSC), Madrid, Spain.
Gil-Moreno MJ; Neurological Disorders Group, Department of Neurology, Hospital Clínico San Carlos, Instituto de Investigación Sanitaria San Carlos (IdISSC), Madrid, Spain.
Matias-Guiu J; Neurological Disorders Group, Department of Neurology, Hospital Clínico San Carlos, Instituto de Investigación Sanitaria San Carlos (IdISSC), Madrid, Spain.
Corrochano S; Neurological Disorders Group, Department of Neurology, Hospital Clínico San Carlos, Instituto de Investigación Sanitaria San Carlos (IdISSC), Madrid, Spain. .; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2024 Feb 01; Vol. 14 (1), pp. 2671. Date of Electronic Publication: 2024 Feb 01.
Typ publikacji:: Journal Article
MeSH Terms:: Amyotrophic Lateral Sclerosis*/metabolism
Animals ; Female ; Humans ; Male ; Mice ; Adipose Tissue/metabolism ; Disease Models, Animal ; Haploinsufficiency ; Leptin/metabolism ; Mice, Transgenic ; Spinal Cord/metabolism ; Superoxide Dismutase/metabolism ; Superoxide Dismutase-1/genetics ; Superoxide Dismutase-1/metabolism

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Skocz do pozycji: 73.

Tytuł:: Presenilin-2 knock-In mice show severe depressive behavior via DVL3 downregulation.
Autorzy:: Yoo SS; College of Pharmacy and Medical Research Center, Chungbuk National University, Cheongju, South Korea.
Lee DW; Ministry of Food and Drug Safety (MFDS), Cheongju, South Korea.; Korea Health Industry Development Institute, Cheongju, South Korea.
Ham HJ; College of Pharmacy and Medical Research Center, Chungbuk National University, Cheongju, South Korea.
Yeo IJ; College of Pharmacy and Medical Research Center, Chungbuk National University, Cheongju, South Korea.
Chang JY; College of Pharmacy and Medical Research Center, Chungbuk National University, Cheongju, South Korea.
Yun J; College of Pharmacy and Medical Research Center, Chungbuk National University, Cheongju, South Korea.
Son DJ; College of Pharmacy and Medical Research Center, Chungbuk National University, Cheongju, South Korea.
Han SB; College of Pharmacy and Medical Research Center, Chungbuk National University, Cheongju, South Korea.
Hong JT; College of Pharmacy and Medical Research Center, Chungbuk National University, Cheongju, South Korea.; Pokaż więcej
Źródło:: CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Feb; Vol. 30 (2), pp. e14370. Date of Electronic Publication: 2023 Jul 27.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Alzheimer Disease*/metabolism
Amyloid beta-Peptides*/metabolism
Animals ; Mice ; Dishevelled Proteins/metabolism ; Down-Regulation ; Glycogen Synthase Kinase 3 beta ; Hydrocortisone ; Mice, Transgenic ; Presenilin-1/genetics ; Presenilin-2/metabolism

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Skocz do pozycji: 74.

Tytuł:: MKP-1 regulates the inflammatory activation of microglia against Alzheimer's disease.
Autorzy:: Li J; Institute of Neuroscience, Basic Medical College, Chongqing Medical University, Chongqing, China.; Department of Anatomy, Basic Medical College, Chongqing Medical University, Chongqing, China.
Wang L; Department of Basic Medicine, Chongqing College of Traditional Chinese Medicine, Chongqing, China.
Zeng Q; Institute of Neuroscience, Basic Medical College, Chongqing Medical University, Chongqing, China.; Department of Anatomy, Basic Medical College, Chongqing Medical University, Chongqing, China.
He J; Institute of Neuroscience, Basic Medical College, Chongqing Medical University, Chongqing, China.; Department of Anatomy, Basic Medical College, Chongqing Medical University, Chongqing, China.
Tang Q; Institute of Neuroscience, Basic Medical College, Chongqing Medical University, Chongqing, China.; Department of Anatomy, Basic Medical College, Chongqing Medical University, Chongqing, China.
Wang K; Institute of Neuroscience, Basic Medical College, Chongqing Medical University, Chongqing, China.; Department of Anatomy, Basic Medical College, Chongqing Medical University, Chongqing, China.
He G; Institute of Neuroscience, Basic Medical College, Chongqing Medical University, Chongqing, China.; Department of Anatomy, Basic Medical College, Chongqing Medical University, Chongqing, China.; Pokaż więcej
Źródło:: CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Feb; Vol. 30 (2), pp. e14409. Date of Electronic Publication: 2023 Aug 21.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Alzheimer Disease*/metabolism
Animals ; Mice ; Amyloid beta-Peptides/metabolism ; Amyloid beta-Protein Precursor/genetics ; Amyloid beta-Protein Precursor/metabolism ; Disease Models, Animal ; Mice, Transgenic ; Microglia/pathology ; Neuroinflammatory Diseases

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Skocz do pozycji: 75.

Tytuł:: SARM1 Promotes Neurodegeneration and Memory Impairment in Mouse Models of Alzheimer's Disease.
Autorzy:: Miao X; College of Pharmacy, Hangzhou Normal University, Hangzhou, Zhejiang 311121, China.; School of Mental Health, Wenzhou Medical University, Wenzhou, Zhejiang 325035, China.
Wu Q; School of Mental Health, Wenzhou Medical University, Wenzhou, Zhejiang 325035, China.
Du S; School of Mental Health, Wenzhou Medical University, Wenzhou, Zhejiang 325035, China.
Xiang L; School of Mental Health, Wenzhou Medical University, Wenzhou, Zhejiang 325035, China.
Zhou S; School of Mental Health, Wenzhou Medical University, Wenzhou, Zhejiang 325035, China.
Zhu J; School of the First Clinical Medical Sciences, School of Information and Engineering, Wenzhou Medical University, Wenzhou, Zhejiang 325205, China.
Chen Z; School of Mental Health, Wenzhou Medical University, Wenzhou, Zhejiang 325035, China.
Wang H; School of the First Clinical Medical Sciences, School of Information and Engineering, Wenzhou Medical University, Wenzhou, Zhejiang 325205, China.
Pan X; School of the First Clinical Medical Sciences, School of Information and Engineering, Wenzhou Medical University, Wenzhou, Zhejiang 325205, China.
Fan Y; School of the First Clinical Medical Sciences, School of Information and Engineering, Wenzhou Medical University, Wenzhou, Zhejiang 325205, China.
Zhang L; School of the First Clinical Medical Sciences, School of Information and Engineering, Wenzhou Medical University, Wenzhou, Zhejiang 325205, China.
Qian J; School of the First Clinical Medical Sciences, School of Information and Engineering, Wenzhou Medical University, Wenzhou, Zhejiang 325205, China.
Xing Y; School of the First Clinical Medical Sciences, School of Information and Engineering, Wenzhou Medical University, Wenzhou, Zhejiang 325205, China.
Xie Y; School of the First Clinical Medical Sciences, School of Information and Engineering, Wenzhou Medical University, Wenzhou, Zhejiang 325205, China.
Hu L; School of Mental Health, Wenzhou Medical University, Wenzhou, Zhejiang 325035, China.
Xu H; School of Mental Health, Wenzhou Medical University, Wenzhou, Zhejiang 325035, China.
Wang W; School of Mental Health, Wenzhou Medical University, Wenzhou, Zhejiang 325035, China.
Wang Y; Clinical Research Center, Affiliated Hangzhou First People's Hospital, Zhejiang University School of Medicine, Hangzhou, Zhejiang 310003, China.
Huang Z; College of Pharmacy, Hangzhou Normal University, Hangzhou, Zhejiang 311121, China.; School of Mental Health, Wenzhou Medical University, Wenzhou, Zhejiang 325035, China.; Pokaż więcej
Źródło:: Aging and disease [Aging Dis] 2024 Feb 01; Vol. 15 (1), pp. 390-407. Date of Electronic Publication: 2024 Feb 01.
Typ publikacji:: Journal Article
MeSH Terms:: Alzheimer Disease*/genetics
Mice ; Animals ; Nestin ; Mice, Transgenic ; Tumor Necrosis Factor-alpha ; Neuroinflammatory Diseases ; Memory Disorders/genetics ; Cytoskeletal Proteins/genetics ; Armadillo Domain Proteins/genetics

Czasopismo naukowe

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Skocz do pozycji: 76.

Tytuł:: Human APOE4 Protects High-Fat and High-Sucrose Diet Fed Targeted Replacement Mice against Fatty Liver Disease Compared to APOE3.
Autorzy:: Huebbe P; Institute of Human Nutrition and Food Science, Kiel University, D-24118 Kiel, Germany.
Bilke S; Institute of Experimental Medicine, Proteomics & Bioanalytics, Kiel University, D-24105 Kiel, Germany.
Rueter J; Institute of Human Nutrition and Food Science, Kiel University, D-24118 Kiel, Germany.
Schloesser A; Institute of Human Nutrition and Food Science, Kiel University, D-24118 Kiel, Germany.
Campbel G; Section Biomedical Imaging, Department of Radiology and Neuroradiology, Kiel University, D-24118 Kiel, Germany.
Glüer CC; Section Biomedical Imaging, Department of Radiology and Neuroradiology, Kiel University, D-24118 Kiel, Germany.
Lucius R; Anatomical Institute, Kiel University, D-24118 Kiel, Germany.
Röcken C; Department of Pathology, Kiel University and University Hospital Schleswig-Holstein, Campus Kiel, D-24105 Kiel, Germany.
Tholey A; Institute of Experimental Medicine, Proteomics & Bioanalytics, Kiel University, D-24105 Kiel, Germany.
Rimbach G; Institute of Human Nutrition and Food Science, Kiel University, D-24118 Kiel, Germany.; Pokaż więcej
Źródło:: Aging and disease [Aging Dis] 2024 Feb 01; Vol. 15 (1), pp. 259-281. Date of Electronic Publication: 2024 Feb 01.
Typ publikacji:: Journal Article
MeSH Terms:: Apolipoprotein E4*/genetics
Non-alcoholic Fatty Liver Disease*/etiology
Humans ; Mice ; Male ; Animals ; Apolipoprotein E3/genetics ; Mice, Transgenic ; Mice, Inbred C57BL ; Apolipoproteins E/genetics ; Diet

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Skocz do pozycji: 77.

Tytuł:: A Combination of Heavy Metals and Intracellular Pathway Modulators Induces Alzheimer Disease-like Pathologies in Organotypic Brain Slices.
Autorzy:: Korde DS; Laboratory of Psychiatry and Experimental Alzheimer's Research, Medical University of Innsbruck, 6020 Innsbruck, Austria.
Humpel C; Laboratory of Psychiatry and Experimental Alzheimer's Research, Medical University of Innsbruck, 6020 Innsbruck, Austria.; Pokaż więcej
Źródło:: Biomolecules [Biomolecules] 2024 Jan 30; Vol. 14 (2). Date of Electronic Publication: 2024 Jan 30.
Typ publikacji:: Journal Article
MeSH Terms:: Alzheimer Disease*/metabolism
Mice ; Animals ; Humans ; tau Proteins/metabolism ; Cadmium/metabolism ; Wortmannin/metabolism ; Mice, Transgenic ; Amyloid beta-Peptides/metabolism ; Neurofibrillary Tangles/metabolism ; Neurofibrillary Tangles/pathology ; Brain/metabolism ; Plaque, Amyloid/complications ; Plaque, Amyloid/metabolism ; Plaque, Amyloid/pathology ; Collagen/metabolism ; Hydrogels/metabolism ; Scopolamine Derivatives/metabolism

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Skocz do pozycji: 78.

Tytuł:: Pediococcus acidilactici reduces tau pathology and ameliorates behavioral deficits in models of neurodegenerative disorders.
Autorzy:: Zhang Y; Shunde Innovation School, Research Institute of Biology and Agriculture, University of Science and Technology Beijing, Beijing, 100083, China.; Zhongzhi International Institute of Agricultural Biosciences, Beijing, 100083, China.; Beijing Engineering Laboratory of Main Crop Bio-Tech Breeding, Beijing International Science and Technology Cooperation Base of Bio-Tech Breeding, Beijing Solidwill Sci-Tech Co. Ltd, Beijing, 100192, China.
Qian W; Shunde Innovation School, Research Institute of Biology and Agriculture, University of Science and Technology Beijing, Beijing, 100083, China.; Zhongzhi International Institute of Agricultural Biosciences, Beijing, 100083, China.
Zhang Y; Shunde Innovation School, Research Institute of Biology and Agriculture, University of Science and Technology Beijing, Beijing, 100083, China.; Zhongzhi International Institute of Agricultural Biosciences, Beijing, 100083, China.
Ma Y; Shunde Innovation School, Research Institute of Biology and Agriculture, University of Science and Technology Beijing, Beijing, 100083, China.; Zhongzhi International Institute of Agricultural Biosciences, Beijing, 100083, China.
Qian J; Shunde Innovation School, Research Institute of Biology and Agriculture, University of Science and Technology Beijing, Beijing, 100083, China.; Zhongzhi International Institute of Agricultural Biosciences, Beijing, 100083, China.
Li J; Zhongzhi International Institute of Agricultural Biosciences, Beijing, 100083, China.; Beijing Engineering Laboratory of Main Crop Bio-Tech Breeding, Beijing International Science and Technology Cooperation Base of Bio-Tech Breeding, Beijing Solidwill Sci-Tech Co. Ltd, Beijing, 100192, China.
Wei X; Shunde Innovation School, Research Institute of Biology and Agriculture, University of Science and Technology Beijing, Beijing, 100083, China.; Zhongzhi International Institute of Agricultural Biosciences, Beijing, 100083, China.; Beijing Engineering Laboratory of Main Crop Bio-Tech Breeding, Beijing International Science and Technology Cooperation Base of Bio-Tech Breeding, Beijing Solidwill Sci-Tech Co. Ltd, Beijing, 100192, China.
Long Y; Shunde Innovation School, Research Institute of Biology and Agriculture, University of Science and Technology Beijing, Beijing, 100083, China. .; Zhongzhi International Institute of Agricultural Biosciences, Beijing, 100083, China. .; Beijing Engineering Laboratory of Main Crop Bio-Tech Breeding, Beijing International Science and Technology Cooperation Base of Bio-Tech Breeding, Beijing Solidwill Sci-Tech Co. Ltd, Beijing, 100192, China. .
Wan X; Shunde Innovation School, Research Institute of Biology and Agriculture, University of Science and Technology Beijing, Beijing, 100083, China. .; Zhongzhi International Institute of Agricultural Biosciences, Beijing, 100083, China. .; Beijing Engineering Laboratory of Main Crop Bio-Tech Breeding, Beijing International Science and Technology Cooperation Base of Bio-Tech Breeding, Beijing Solidwill Sci-Tech Co. Ltd, Beijing, 100192, China. .; Pokaż więcej
Źródło:: Cell communication and signaling : CCS [Cell Commun Signal] 2024 Jan 30; Vol. 22 (1), pp. 84. Date of Electronic Publication: 2024 Jan 30.
Typ publikacji:: Video-Audio Media; Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Pediococcus acidilactici*/metabolism
Alzheimer Disease*/metabolism
Neurodegenerative Diseases*
Mice ; Animals ; Humans ; Aged ; Amyloid beta-Peptides/genetics ; Amyloid beta-Peptides/metabolism ; Caenorhabditis elegans/metabolism ; Uric Acid ; Mice, Transgenic ; Iron ; Phosphates

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Skocz do pozycji: 79.

Tytuł:: M13 phage grafted with peptide motifs as a tool to detect amyloid-β oligomers in brain tissue.
Autorzy:: Martins IM; CEB- Centre of Biological Engineering, University of Minho, 4710-057, Braga, Portugal. .; LABBELS - Associate Laboratory, Braga/Guimarães, Portugal. .; Netherlands Institute for Neuroscience, Amsterdam, the Netherlands. .; Swammerdam Institute for Life Sciences, University of Amsterdam, Amsterdam Neuroscience, Amsterdam, the Netherlands. .
Lima A; CEB- Centre of Biological Engineering, University of Minho, 4710-057, Braga, Portugal.; LABBELS - Associate Laboratory, Braga/Guimarães, Portugal.; Swammerdam Institute for Life Sciences, University of Amsterdam, Amsterdam Neuroscience, Amsterdam, the Netherlands.
de Graaff W; Swammerdam Institute for Life Sciences, University of Amsterdam, Amsterdam Neuroscience, Amsterdam, the Netherlands.
Cristóvão JS; Biosystems & Integrative Sciences Institute, Faculdade de Ciências, Universidade de Lisboa, Lisboa, Portugal.; Departamento de Química e Bioquímica, Faculdade de Ciências, Universidade de Lisboa, Lisboa, Portugal.
Brosens N; Swammerdam Institute for Life Sciences, University of Amsterdam, Amsterdam Neuroscience, Amsterdam, the Netherlands.
Aronica E; Amsterdam UMC location University of Amsterdam, Department of (Neuro)Pathology, Amsterdam Neuroscience, Amsterdam, the Netherlands.
Kluskens LD; CEB- Centre of Biological Engineering, University of Minho, 4710-057, Braga, Portugal.; LABBELS - Associate Laboratory, Braga/Guimarães, Portugal.
Gomes CM; Biosystems & Integrative Sciences Institute, Faculdade de Ciências, Universidade de Lisboa, Lisboa, Portugal.; Departamento de Química e Bioquímica, Faculdade de Ciências, Universidade de Lisboa, Lisboa, Portugal.
Azeredo J; CEB- Centre of Biological Engineering, University of Minho, 4710-057, Braga, Portugal.; LABBELS - Associate Laboratory, Braga/Guimarães, Portugal.
Kessels HW; Netherlands Institute for Neuroscience, Amsterdam, the Netherlands. .; Swammerdam Institute for Life Sciences, University of Amsterdam, Amsterdam Neuroscience, Amsterdam, the Netherlands. .; Pokaż więcej
Źródło:: Communications biology [Commun Biol] 2024 Jan 27; Vol. 7 (1), pp. 134. Date of Electronic Publication: 2024 Jan 27.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Amyloid beta-Peptides*/metabolism
Alzheimer Disease*
Humans ; Mice ; Animals ; Amyloid beta-Protein Precursor/metabolism ; Bacteriophage M13/metabolism ; Mice, Transgenic ; Brain/metabolism

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Skocz do pozycji: 80.

Tytuł:: TRPV4 Channels Promote Pathological, but Not Physiological, Cardiac Remodeling through the Activation of Calcineurin/NFAT and TRPC6.
Autorzy:: Yáñez-Bisbe L; Cardiovascular Diseases Research Group, Vall d'Hebron Institut de Recerca (VHIR), Hospital Universitari Vall d'Hebron, 08035 Barcelona, Spain.
Moya M; Cardiovascular Diseases Research Group, Vall d'Hebron Institut de Recerca (VHIR), Hospital Universitari Vall d'Hebron, 08035 Barcelona, Spain.
Rodríguez-Sinovas A; Cardiovascular Diseases Research Group, Vall d'Hebron Institut de Recerca (VHIR), Hospital Universitari Vall d'Hebron, 08035 Barcelona, Spain.; Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Instituto de Salud Carlos III, 28029 Madrid, Spain.
Ruiz-Meana M; Cardiovascular Diseases Research Group, Vall d'Hebron Institut de Recerca (VHIR), Hospital Universitari Vall d'Hebron, 08035 Barcelona, Spain.; Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Instituto de Salud Carlos III, 28029 Madrid, Spain.
Inserte J; Cardiovascular Diseases Research Group, Vall d'Hebron Institut de Recerca (VHIR), Hospital Universitari Vall d'Hebron, 08035 Barcelona, Spain.; Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Instituto de Salud Carlos III, 28029 Madrid, Spain.
Tajes M; Bio-Heart Cardiovascular Diseases Research Group, Bellvitge Biomedical Research Institute (IDIBELL), L'Hospitalet de Llobregat, 08908 Barcelona, Spain.
Batlle M; Institute for Biomedical Research August Pi i Sunyer (IDIBAPS), 08036 Barcelona, Spain.
Guasch E; Institute for Biomedical Research August Pi i Sunyer (IDIBAPS), 08036 Barcelona, Spain.; Cardiology Department, Hospital Clínic, 08036 Barcelona, Spain.
Mas-Stachurska A; Institute for Biomedical Research August Pi i Sunyer (IDIBAPS), 08036 Barcelona, Spain.; Cardiology Department, Hospital del Mar, 08003 Barcelona, Spain.
Miró E; Cardiovascular Diseases Research Group, Vall d'Hebron Institut de Recerca (VHIR), Hospital Universitari Vall d'Hebron, 08035 Barcelona, Spain.
Rivas N; Cardiovascular Diseases Research Group, Vall d'Hebron Institut de Recerca (VHIR), Hospital Universitari Vall d'Hebron, 08035 Barcelona, Spain.; Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Instituto de Salud Carlos III, 28029 Madrid, Spain.; Cardiology Department, Hospital Universitari Vall d'Hebron, 08035 Barcelona, Spain.
Ferreira González I; Cardiovascular Diseases Research Group, Vall d'Hebron Institut de Recerca (VHIR), Hospital Universitari Vall d'Hebron, 08035 Barcelona, Spain.; Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Instituto de Salud Carlos III, 28029 Madrid, Spain.; Cardiology Department, Hospital Universitari Vall d'Hebron, 08035 Barcelona, Spain.; Department of Medicine, Universitat Autònoma de Barcelona, 08193 Barcelona, Spain.; Centro de Investigación Biomédica en Red en Epidemiología y Salud Pública (CIBERESP), Instituto de Salud Carlos III, 28029 Madrid, Spain.
Garcia-Elias A; Department of Clinical Research, ASCIRES-CETIR Biomedic Group, 08029 Barcelona, Spain.
Benito B; Cardiovascular Diseases Research Group, Vall d'Hebron Institut de Recerca (VHIR), Hospital Universitari Vall d'Hebron, 08035 Barcelona, Spain.; Centro de Investigación Biomédica en Red de Enfermedades Cardiovasculares (CIBERCV), Instituto de Salud Carlos III, 28029 Madrid, Spain.; Cardiology Department, Hospital Universitari Vall d'Hebron, 08035 Barcelona, Spain.; Department of Medicine, Universitat Autònoma de Barcelona, 08193 Barcelona, Spain.; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2024 Jan 26; Vol. 25 (3). Date of Electronic Publication: 2024 Jan 26.
Typ publikacji:: Journal Article
MeSH Terms:: Calcineurin*/metabolism
Heart Failure*/metabolism
TRPV Cation Channels*/genetics
TRPV Cation Channels*/metabolism
Ventricular Remodeling*/genetics
Animals ; Mice ; Cells, Cultured ; Fibrosis ; Isoproterenol ; Mice, Transgenic ; Myocytes, Cardiac/metabolism ; NFATC Transcription Factors/genetics ; NFATC Transcription Factors/metabolism ; TRPC6 Cation Channel/genetics ; TRPC6 Cation Channel/metabolism

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