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Wyszukujesz frazę ""Mindy M. Tsai"" wg kryterium: Autor

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Wyświetlanie 1-20 z 22
Tytuł:
Mass Cytometry Phenotyping of Human Granulocytes Reveals Novel Basophil Functional Heterogeneity
Autorzy:
Nora Vivanco Gonzalez
John-Paul Oliveria
Dmitry Tebaykin
Geoffrey T. Ivison
Kaori Mukai
Mindy M. Tsai
Luciene Borges
Kari C. Nadeau
Stephen J. Galli
Albert G. Tsai
Sean C. Bendall
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Temat:
Immunology
Systems Biology
Science
Źródło:
iScience, Vol 23, Iss 11, Pp 101724- (2020)
Opis pliku:
electronic resource
Relacje:
http://www.sciencedirect.com/science/article/pii/S2589004220309214; https://doaj.org/toc/2589-0042
Dostęp URL:
https://doaj.org/article/ad8454f1b10241c68045d43551e2c1ae  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Tytuł:
Mast cells as sources of cytokines, chemokines, and growth factors.
Autorzy:
Mukai K; Department of Pathology, Stanford University School of Medicine, Stanford, CA, USA.; Sean N. Parker Center for Allergy and Asthma Research, Stanford University School of Medicine, Stanford, CA, USA.
Tsai M; Department of Pathology, Stanford University School of Medicine, Stanford, CA, USA.; Sean N. Parker Center for Allergy and Asthma Research, Stanford University School of Medicine, Stanford, CA, USA.
Saito H; Department of Allergy and Clinical Immunology, National Research Institute for Child Health & Development, Tokyo, Japan.
Galli SJ; Department of Pathology, Stanford University School of Medicine, Stanford, CA, USA.; Sean N. Parker Center for Allergy and Asthma Research, Stanford University School of Medicine, Stanford, CA, USA.; Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, CA, USA.
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Źródło:
Immunological reviews [Immunol Rev] 2018 Mar; Vol. 282 (1), pp. 121-150.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Review
MeSH Terms:
Chemokines/*metabolism
Cytokines/*metabolism
Hypersensitivity/*immunology
Mast Cells/*immunology
Animals ; Cell Communication ; Humans ; Immunity, Innate
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The tyrosine kinase inhibitor imatinib mesylate suppresses uric acid crystal-induced acute gouty arthritis in mice.
Autorzy:
Reber LL; Department of Pathology, Stanford University School of Medicine, Stanford, California, United States of America.; Sean N. Parker Center for Allergy Research, Stanford University School of Medicine, Stanford, California, United States of America.; Department of Immunology, Unit of Antibodies in Therapy and Pathology, Institut Pasteur, Paris, France.; Institut National de la Santé et de la Recherche Médicale, Paris, France.
Starkl P; Department of Pathology, Stanford University School of Medicine, Stanford, California, United States of America.; Sean N. Parker Center for Allergy Research, Stanford University School of Medicine, Stanford, California, United States of America.
Balbino B; Department of Immunology, Unit of Antibodies in Therapy and Pathology, Institut Pasteur, Paris, France.; Institut National de la Santé et de la Recherche Médicale, Paris, France.; Université Pierre et Marie Curie, Paris, France.
Sibilano R; Department of Pathology, Stanford University School of Medicine, Stanford, California, United States of America.; Sean N. Parker Center for Allergy Research, Stanford University School of Medicine, Stanford, California, United States of America.
Gaudenzio N; Department of Pathology, Stanford University School of Medicine, Stanford, California, United States of America.; Sean N. Parker Center for Allergy Research, Stanford University School of Medicine, Stanford, California, United States of America.
Rogalla S; Departments of Bioengineering, Radiology, and Pediatrics Division of Neonatology, Stanford University School of Medicine, Stanford, California, United States of America.; Molecular Imaging Program at Stanford, Stanford, California, United States of America.
Sensarn S; Departments of Bioengineering, Radiology, and Pediatrics Division of Neonatology, Stanford University School of Medicine, Stanford, California, United States of America.; Molecular Imaging Program at Stanford, Stanford, California, United States of America.
Kang D; Departments of Bioengineering, Radiology, and Pediatrics Division of Neonatology, Stanford University School of Medicine, Stanford, California, United States of America.; Molecular Imaging Program at Stanford, Stanford, California, United States of America.; Department of Life Science, Ewha Womans University, Seoul, Korea.
Raghu H; Department of Medicine, Stanford University School of Medicine, Stanford, California, United States of America.; Geriatric Research Education and Clinical Center, Veterans Affairs Palo Alto Health Care System, Palo Alto, California, United States of America.
Sokolove J; Department of Medicine, Stanford University School of Medicine, Stanford, California, United States of America.; Geriatric Research Education and Clinical Center, Veterans Affairs Palo Alto Health Care System, Palo Alto, California, United States of America.
Robinson WH; Department of Medicine, Stanford University School of Medicine, Stanford, California, United States of America.; Geriatric Research Education and Clinical Center, Veterans Affairs Palo Alto Health Care System, Palo Alto, California, United States of America.
Contag CH; Departments of Bioengineering, Radiology, and Pediatrics Division of Neonatology, Stanford University School of Medicine, Stanford, California, United States of America.; Molecular Imaging Program at Stanford, Stanford, California, United States of America.; Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, California, United States of America.
Tsai M; Department of Pathology, Stanford University School of Medicine, Stanford, California, United States of America.; Sean N. Parker Center for Allergy Research, Stanford University School of Medicine, Stanford, California, United States of America.
Galli SJ; Department of Pathology, Stanford University School of Medicine, Stanford, California, United States of America.; Sean N. Parker Center for Allergy Research, Stanford University School of Medicine, Stanford, California, United States of America.; Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, California, United States of America.
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Źródło:
PloS one [PLoS One] 2017 Oct 05; Vol. 12 (10), pp. e0185704. Date of Electronic Publication: 2017 Oct 05 (Print Publication: 2017).
Typ publikacji:
Journal Article
MeSH Terms:
Arthritis, Gouty/*prevention & control
Imatinib Mesylate/*pharmacology
Protein Kinase Inhibitors/*pharmacology
Protein-Tyrosine Kinases/*antagonists & inhibitors
Uric Acid/*adverse effects
Animals ; Crystallization ; Imatinib Mesylate/administration & dosage ; Injections, Intraperitoneal ; Mice ; Mice, Inbred C57BL ; Protein Kinase Inhibitors/administration & dosage ; Uric Acid/chemistry
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Targeting of Immune Cells by Dual TLR2/7 Ligands Suppresses Features of Allergic Th2 Immune Responses in Mice.
Autorzy:
Laiño J; Vice President's Research Group 1: Molecular Allergology, Paul-Ehrlich-Institut, Langen, Germany.
Wangorsch A; Vice President's Research Group 1: Molecular Allergology, Paul-Ehrlich-Institut, Langen, Germany.
Blanco F; Vice President's Research Group 1: Molecular Allergology, Paul-Ehrlich-Institut, Langen, Germany.
Wolfheimer S; Vice President's Research Group 1: Molecular Allergology, Paul-Ehrlich-Institut, Langen, Germany.
Krause M; Vice President's Research Group 1: Molecular Allergology, Paul-Ehrlich-Institut, Langen, Germany.
Flaczyk A; Vice President's Research Group 1: Molecular Allergology, Paul-Ehrlich-Institut, Langen, Germany.
Möller TM; Vice President's Research Group 1: Molecular Allergology, Paul-Ehrlich-Institut, Langen, Germany.
Tsai M; Department of Pathology and The Sean N. Parker Center for Allergy and Asthma Research, Stanford University School of Medicine, Stanford, CA, USA.
Galli S; Department of Pathology and The Sean N. Parker Center for Allergy and Asthma Research, Stanford University School of Medicine, Stanford, CA, USA.; Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, CA, USA.
Vieths S; Vice President's Research Group 1: Molecular Allergology, Paul-Ehrlich-Institut, Langen, Germany.
Toda M; Vice President's Research Group 1: Molecular Allergology, Paul-Ehrlich-Institut, Langen, Germany.
Scheurer S; Vice President's Research Group 1: Molecular Allergology, Paul-Ehrlich-Institut, Langen, Germany.
Schülke S; Vice President's Research Group 1: Molecular Allergology, Paul-Ehrlich-Institut, Langen, Germany.
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Źródło:
Journal of immunology research [J Immunol Res] 2017; Vol. 2017, pp. 7983217. Date of Electronic Publication: 2017 Oct 24.
Typ publikacji:
Journal Article
MeSH Terms:
Dendritic Cells/*immunology
Hypersensitivity/*drug therapy
Lipopeptides/*pharmacology
Membrane Glycoproteins/*agonists
Th2 Cells/*immunology
Toll-Like Receptor 2/*agonists
Toll-Like Receptor 7/*agonists
Animals ; Cells, Cultured ; Coculture Techniques ; Dendritic Cells/drug effects ; Humans ; Hypersensitivity/immunology ; Immunomodulation ; Lipopeptides/chemistry ; Lymphocyte Activation/drug effects ; Mice ; Mice, Inbred BALB C ; Mice, Inbred C57BL ; Mice, Transgenic ; Molecular Targeted Therapy ; Th1 Cells/immunology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
IgE and mast cells in host defense against parasites and venoms.
Autorzy:
Mukai K; Department of Pathology, Stanford University School of Medicine, Stanford, California 94305-5324, USA.; Sean N. Parker Center for Allergy and Asthma Research, Stanford University School of Medicine, Stanford, California 94305-5324, USA.
Tsai M; Department of Pathology, Stanford University School of Medicine, Stanford, California 94305-5324, USA.; Sean N. Parker Center for Allergy and Asthma Research, Stanford University School of Medicine, Stanford, California 94305-5324, USA.
Starkl P; CeMM Research Center for Molecular Medicine of the Austrian Academy of Sciences, and Department of Medicine I, Medical University of Vienna, 1090 Vienna, Austria.
Marichal T; Laboratory of Cellular and Molecular Immunology, GIGA-Research and Faculty of Veterinary Medicine, University of Liège, 4000 Liège, Belgium.
Galli SJ; Department of Pathology, Stanford University School of Medicine, Stanford, California 94305-5324, USA.; Sean N. Parker Center for Allergy and Asthma Research, Stanford University School of Medicine, Stanford, California 94305-5324, USA.; Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, California 94305-5324, USA.
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Źródło:
Seminars in immunopathology [Semin Immunopathol] 2016 Sep; Vol. 38 (5), pp. 581-603. Date of Electronic Publication: 2016 May 25.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms:
Immunity, Innate*
Host-Parasite Interactions/*immunology
Immunoglobulin E/*immunology
Mast Cells/*immunology
Mast Cells/*metabolism
Venoms/*immunology
Anaphylaxis/immunology ; Anaphylaxis/metabolism ; Animals ; Disease Resistance/immunology ; Helminthiasis/immunology ; Helminthiasis/metabolism ; Helminthiasis/parasitology ; Helminths/immunology ; Humans ; Hypersensitivity/immunology ; Hypersensitivity/metabolism ; Intestinal Diseases, Parasitic/immunology ; Intestinal Diseases, Parasitic/metabolism ; Parasites/immunology ; Parasitic Diseases/immunology ; Parasitic Diseases/transmission ; Th2 Cells/immunology ; Th2 Cells/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
RabGEF1/Rabex-5 Regulates TrkA-Mediated Neurite Outgrowth and NMDA-Induced Signaling Activation in NGF-Differentiated PC12 Cells.
Autorzy:
Tam SY; Department of Pathology, Stanford University School of Medicine, Stanford, California, United States of America.
Lilla JN; Department of Pathology, Stanford University School of Medicine, Stanford, California, United States of America.
Chen CC; Department of Pathology, Stanford University School of Medicine, Stanford, California, United States of America.
Kalesnikoff J; Department of Pathology, Stanford University School of Medicine, Stanford, California, United States of America.
Tsai M; Department of Pathology, Stanford University School of Medicine, Stanford, California, United States of America.
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Źródło:
PloS one [PLoS One] 2015 Nov 20; Vol. 10 (11), pp. e0142935. Date of Electronic Publication: 2015 Nov 20 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Differentiation/*genetics
Guanine Nucleotide Exchange Factors/*biosynthesis
Nerve Growth Factor/*metabolism
Receptor, trkA/*metabolism
Animals ; Gene Expression Regulation, Developmental ; Guanine Nucleotide Exchange Factors/genetics ; N-Methylaspartate/administration & dosage ; Nerve Growth Factor/genetics ; Neurites/metabolism ; Neurogenesis/genetics ; Neurons/metabolism ; PC12 Cells ; Protein Binding ; Rats ; Receptor, trkA/genetics ; Signal Transduction/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Evidence that mast cells are not required for healing of splinted cutaneous excisional wounds in mice.
Autorzy:
Nauta AC; Hagey Laboratory for Pediatric Regenerative Medicine, Division of Plastic and Reconstructive Surgery, Department of Surgery, Stanford University School of Medicine, Stanford, California, United States of America.
Grova M
Montoro DT
Zimmermann A
Tsai M
Gurtner GC
Galli SJ
Longaker MT
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Źródło:
PloS one [PLoS One] 2013; Vol. 8 (3), pp. e59167. Date of Electronic Publication: 2013 Mar 27.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Splints*
Wound Healing*
Mast Cells/*pathology
Skin/*pathology
Animals ; Collagen/metabolism ; Dermis/pathology ; Kinetics ; Mice ; Mice, Inbred C57BL
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
IgE and mast cells in allergic disease.
Autorzy:
Galli SJ; Department of Pathology, Stanford University School of Medicine, California, USA. />Tsai M
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Źródło:
Nature medicine [Nat Med] 2012 May 04; Vol. 18 (5), pp. 693-704. Date of Electronic Publication: 2012 May 04.
Typ publikacji:
Journal Article; Research Support, U.S. Gov't, P.H.S.; Review
MeSH Terms:
Hypersensitivity/*immunology
Immunoglobulin E/*physiology
Mast Cells/*physiology
Allergens/immunology ; Animals ; Humans ; Hypersensitivity/etiology ; Receptors, IgE/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
The development of allergic inflammation.
Autorzy:
Galli SJ; Department of Pathology, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, California 94305, USA. />Tsai M
Piliponsky AM
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Źródło:
Nature [Nature] 2008 Jul 24; Vol. 454 (7203), pp. 445-54.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Review
MeSH Terms:
Hypersensitivity/*immunology
Hypersensitivity/*pathology
Inflammation/*immunology
Inflammation/*pathology
Allergens/immunology ; Animals ; Chronic Disease ; Epithelium/immunology ; Humans ; Hypersensitivity/genetics ; Immunoglobulin E/immunology ; Inflammation/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Immunomodulatory mast cells: negative, as well as positive, regulators of immunity.
Autorzy:
Galli SJ; Department of Pathology, Stanford University School of Medicine, Stanford, California 94305-5324, USA. />Grimbaldeston M
Tsai M
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Źródło:
Nature reviews. Immunology [Nat Rev Immunol] 2008 Jun; Vol. 8 (6), pp. 478-86.
Typ publikacji:
Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Immunity, Cellular/*immunology
Mast Cells/*immunology
Mast Cells/*metabolism
Animals ; Disease Models, Animal ; Humans ; Immunity, Cellular/drug effects ; Immunity, Cellular/genetics ; Immunologic Factors/biosynthesis ; Immunologic Factors/metabolism ; Immunologic Factors/physiology ; Inflammation Mediators/physiology ; Mast Cells/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Neurotensin increases mortality and mast cells reduce neurotensin levels in a mouse model of sepsis.
Autorzy:
Piliponsky AM; Department of Pathology, Stanford University School of Medicine, Stanford, California 94305, USA.
Chen CC
Nishimura T
Metz M
Rios EJ
Dobner PR
Wada E
Wada K
Zacharias S
Mohanasundaram UM
Faix JD
Abrink M
Pejler G
Pearl RG
Tsai M
Galli SJ
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Źródło:
Nature medicine [Nat Med] 2008 Apr; Vol. 14 (4), pp. 392-8. Date of Electronic Publication: 2008 Mar 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
MeSH Terms:
Mast Cells/*metabolism
Neurotensin/*metabolism
Sepsis/*metabolism
Animals ; Cell Degranulation ; Disease Models, Animal ; Female ; Humans ; Hypotension/metabolism ; Hypotension/prevention & control ; Male ; Mast Cells/physiology ; Metalloendopeptidases/metabolism ; Mice ; Mice, Congenic ; Mice, Inbred C57BL ; Mice, Knockout ; Mice, Mutant Strains ; Neurotensin/antagonists & inhibitors ; Neurotensin/blood ; Neurotensin/deficiency ; Proto-Oncogene Proteins c-kit/genetics ; Proto-Oncogene Proteins c-kit/metabolism ; Receptors, Neurotensin/metabolism ; Sepsis/blood
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Mast cell-derived interleukin 10 limits skin pathology in contact dermatitis and chronic irradiation with ultraviolet B.
Autorzy:
Grimbaldeston MA; Department of Pathology, Stanford University School of Medicine, Stanford, California 94305-5176, USA.
Nakae S
Kalesnikoff J
Tsai M
Galli SJ
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Źródło:
Nature immunology [Nat Immunol] 2007 Oct; Vol. 8 (10), pp. 1095-104. Date of Electronic Publication: 2007 Sep 02.
Typ publikacji:
Journal Article
MeSH Terms:
Dermatitis, Allergic Contact/*pathology
Interleukin-10/*physiology
Mast Cells/*physiology
Skin/*pathology
Skin/*radiation effects
Animals ; Dermatitis, Allergic Contact/immunology ; Female ; Immunoglobulin G/physiology ; Mice ; Receptors, IgG/physiology ; Ultraviolet Rays
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Mast cells in the promotion and limitation of chronic inflammation.
Autorzy:
Metz M; Department of Pathology, Stanford University School of Medicine, Stanford, CA 94305-5324, USA.
Grimbaldeston MA
Nakae S
Piliponsky AM
Tsai M
Galli SJ
Pokaż więcej
Źródło:
Immunological reviews [Immunol Rev] 2007 Jun; Vol. 217, pp. 304-28.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Inflammation/*immunology
Mast Cells/*immunology
Acute Disease ; Animals ; Chronic Disease ; Disease Models, Animal ; Immunity, Innate ; Immunoglobulin E/immunology ; Inflammation/therapy ; Mast Cells/drug effects ; Mast Cells/ultrastructure ; Mice ; Proto-Oncogene Proteins c-kit/genetics ; Proto-Oncogene Proteins c-kit/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Mast cells in the development of adaptive immune responses.
Autorzy:
Galli SJ; Department of Pathology, Stanford University School of Medicine, Stanford, California 94305-5324, USA. />Nakae S
Tsai M
Pokaż więcej
Źródło:
Nature immunology [Nat Immunol] 2005 Feb; Vol. 6 (2), pp. 135-42.
Typ publikacji:
Journal Article; Research Support, U.S. Gov't, P.H.S.; Review
MeSH Terms:
Mast Cells/*cytology
Mast Cells/*immunology
Animals ; Antigen Presentation/immunology ; Cell Differentiation ; Humans ; Immune System Diseases/immunology ; Immune System Diseases/metabolism ; Lymphocytes/cytology ; Lymphocytes/immunology ; Mast Cells/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Mast cells as "tunable" effector and immunoregulatory cells: recent advances.
Autorzy:
Galli SJ; Department of Pathology, Stanford University School of Medicine, Stanford, California 94305, USA. />Kalesnikoff J
Grimbaldeston MA
Piliponsky AM
Williams CM
Tsai M
Pokaż więcej
Źródło:
Annual review of immunology [Annu Rev Immunol] 2005; Vol. 23, pp. 749-86.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.; Review
MeSH Terms:
Mast Cells/*immunology
Animals ; Asthma/immunology ; Basophils/immunology ; Dermatitis, Contact/immunology ; Humans ; Immunoglobulin E/metabolism ; Immunoglobulin G/metabolism ; Mice ; Mice, Mutant Strains ; Models, Immunological ; Receptors, IgE/metabolism ; Signal Transduction ; T-Lymphocytes/immunology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Mast cells promote homeostasis by limiting endothelin-1-induced toxicity.
Autorzy:
Maurer M; Department of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts 02215, USA.
Wedemeyer J
Metz M
Piliponsky AM
Weller K
Chatterjea D
Clouthier DE
Yanagisawa MM
Tsai M
Galli SJ
Pokaż więcej
Źródło:
Nature [Nature] 2004 Nov 25; Vol. 432 (7016), pp. 512-6. Date of Electronic Publication: 2004 Nov 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
MeSH Terms:
Drug-Related Side Effects and Adverse Reactions*
Egtazic Acid/*analogs & derivatives
Endothelin-1/*antagonists & inhibitors
Endothelin-1/*toxicity
Homeostasis/*physiology
Mast Cells/*physiology
Animals ; Body Temperature ; Body Weight ; Cell Degranulation/drug effects ; Cell Survival ; Chymases ; Diarrhea/chemically induced ; Egtazic Acid/pharmacology ; Endothelin-1/administration & dosage ; Endothelin-1/deficiency ; Female ; Injections, Intraperitoneal ; Mast Cells/cytology ; Mast Cells/enzymology ; Mast Cells/transplantation ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Mutation/genetics ; Oligopeptides/pharmacology ; Peptides, Cyclic/pharmacology ; Peritonitis/physiopathology ; Proto-Oncogene Proteins c-kit/genetics ; Proto-Oncogene Proteins c-kit/metabolism ; Serine Endopeptidases/metabolism ; Stem Cells/cytology ; Survival Rate
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
RabGEF1 is a negative regulator of mast cell activation and skin inflammation.
Autorzy:
Tam SY; Department of Pathology, Stanford University School of Medicine, Stanford, California 94305, USA. />Tsai M
Snouwaert JN
Kalesnikoff J
Scherrer D
Nakae S
Chatterjea D
Bouley DM
Galli SJ
Pokaż więcej
Źródło:
Nature immunology [Nat Immunol] 2004 Aug; Vol. 5 (8), pp. 844-52. Date of Electronic Publication: 2004 Jul 04.
Typ publikacji:
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
MeSH Terms:
Guanine Nucleotide Exchange Factors/*immunology
Inflammation/*immunology
Mast Cells/*immunology
Skin/*immunology
Amino Acid Sequence ; Animals ; Flow Cytometry ; Guanine Nucleotide Exchange Factors/genetics ; Humans ; Immunoblotting ; Mice ; Molecular Sequence Data ; Receptors, IgE/immunology ; Receptors, IgE/metabolism ; Sequence Homology, Amino Acid ; Signal Transduction/immunology ; Skin/pathology ; Two-Hybrid System Techniques ; ras Proteins/immunology ; ras Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Wyświetlanie 1-20 z 22

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