Temat: "Mitochondrial Membrane Transport Proteins" - OpacWWW

Skocz do pozycji: 1.

Tytuł:: MTCH2 is a mitochondrial outer membrane protein insertase.
Autorzy:: Guna A; Whitehead Institute for Biomedical Research, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.
Stevens TA; Division of Biology and Biological Engineering, California Institute of Technology, 1200 East California Avenue, Pasadena, CA 91125, USA.
Inglis AJ; Division of Biology and Biological Engineering, California Institute of Technology, 1200 East California Avenue, Pasadena, CA 91125, USA.
Replogle JM; Whitehead Institute for Biomedical Research, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.; Medical Scientist Training Program, University of California, San Francisco, San Francisco, CA 94158, USA.; Tetrad Graduate Program, University of California, San Francisco, San Francisco, CA 94158, USA.; Howard Hughes Medical Institute, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.
Esantsi TK; Whitehead Institute for Biomedical Research, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.; Howard Hughes Medical Institute, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.
Muthukumar G; Whitehead Institute for Biomedical Research, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.
Shaffer KCL; Division of Biology and Biological Engineering, California Institute of Technology, 1200 East California Avenue, Pasadena, CA 91125, USA.
Wang ML; Whitehead Institute for Biomedical Research, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.; Division of Biology and Biological Engineering, California Institute of Technology, 1200 East California Avenue, Pasadena, CA 91125, USA.; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.
Pogson AN; Whitehead Institute for Biomedical Research, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.; Howard Hughes Medical Institute, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.
Jones JJ; Division of Biology and Biological Engineering, California Institute of Technology, 1200 East California Avenue, Pasadena, CA 91125, USA.
Lomenick B; Division of Biology and Biological Engineering, California Institute of Technology, 1200 East California Avenue, Pasadena, CA 91125, USA.
Chou TF; Division of Biology and Biological Engineering, California Institute of Technology, 1200 East California Avenue, Pasadena, CA 91125, USA.
Weissman JS; Whitehead Institute for Biomedical Research, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.; Howard Hughes Medical Institute, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.; David H. Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02142, USA.
Voorhees RM; Division of Biology and Biological Engineering, California Institute of Technology, 1200 East California Avenue, Pasadena, CA 91125, USA.; Pokaż więcej
Źródło:: Science (New York, N.Y.) [Science] 2022 Oct 21; Vol. 378 (6617), pp. 317-322. Date of Electronic Publication: 2022 Oct 20.
Typ publikacji:: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:: Mitochondrial Membrane Transport Proteins*/chemistry
Mitochondrial Membrane Transport Proteins*/genetics
Mitochondrial Membranes*/metabolism
Apoptosis*
Humans ; Endoplasmic Reticulum/metabolism ; K562 Cells

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Tytuł:: New insight into mitochondrial changes in vascular endothelial cells irradiated by gamma ray.
Autorzy:: Hu S; a Department of Cardiology , Chinese PLA General Hospital , Beijing , China.
Gao Y; b Beijing Institute of Radiation Medicine , Beijing , China.
Zhou H; a Department of Cardiology , Chinese PLA General Hospital , Beijing , China.
Kong F; b Beijing Institute of Radiation Medicine , Beijing , China.
Xiao F; b Beijing Institute of Radiation Medicine , Beijing , China.
Zhou P; b Beijing Institute of Radiation Medicine , Beijing , China.
Chen Y; a Department of Cardiology , Chinese PLA General Hospital , Beijing , China.; Pokaż więcej
Źródło:: International journal of radiation biology [Int J Radiat Biol] 2017 May; Vol. 93 (5), pp. 470-476. Date of Electronic Publication: 2017 Feb 08.
Typ publikacji:: Journal Article
MeSH Terms:: Gamma Rays*
Apoptosis/*physiology
Endothelial Cells/*physiology
Membrane Potential, Mitochondrial/*physiology
Mitochondria/*physiology
Mitochondrial Membrane Transport Proteins/*physiology
Apoptosis/radiation effects ; Cells, Cultured ; Dose-Response Relationship, Radiation ; Endothelial Cells/radiation effects ; Endothelial Cells/ultrastructure ; Humans ; Membrane Potential, Mitochondrial/radiation effects ; Mitochondria/radiation effects ; Mitochondria/ultrastructure ; Mitochondrial Membrane Transport Proteins/radiation effects ; Mitochondrial Membrane Transport Proteins/ultrastructure ; Mitochondrial Permeability Transition Pore ; Radiation Dosage ; Reactive Oxygen Species/metabolism

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Skocz do pozycji: 3.

Tytuł:: Low expression of mitofusin 1 is associated with mitochondrial dysfunction and apoptosis in porcine somatic cell nuclear transfer embryos.
Autorzy:: Park MR; Animal Biotechnology Division, National Institute of Animal Science, Rural Development Administration, Wanju-gun, Republic of Korea.
Hwang IS; Animal Biotechnology Division, National Institute of Animal Science, Rural Development Administration, Wanju-gun, Republic of Korea.
Kwak TU; Animal Biotechnology Division, National Institute of Animal Science, Rural Development Administration, Wanju-gun, Republic of Korea.
Lim JH; Animal Biotechnology Division, National Institute of Animal Science, Rural Development Administration, Wanju-gun, Republic of Korea.
Hwang S; Animal Biotechnology Division, National Institute of Animal Science, Rural Development Administration, Wanju-gun, Republic of Korea.
Cho SK; Department of Animal Science, Life and Industry Convergence Research Institute (RICRI), College of Natural Science, Pusan University, Miryang, Gyeongnam, Republic of Korea.; Pokaż więcej
Źródło:: Animal science journal = Nihon chikusan Gakkaiho [Anim Sci J] 2020 Jan-Dec; Vol. 91 (1), pp. e13430.
Typ publikacji:: Journal Article
MeSH Terms:: Blastocyst*
Apoptosis/*genetics
Embryo, Mammalian/*metabolism
Embryonic Development/*genetics
Gene Expression/*genetics
Gene Expression Regulation, Developmental/*genetics
Genetic Association Studies/*veterinary
Membrane Potential, Mitochondrial/*genetics
Mitochondrial Membrane Transport Proteins/*genetics
Mitochondrial Membrane Transport Proteins/*metabolism
Parthenogenesis/*genetics
Swine/*genetics
Swine/*physiology
Animals ; Cells, Cultured ; Embryo Culture Techniques/veterinary ; Fertilization in Vitro/veterinary ; Nuclear Transfer Techniques/veterinary ; Reactive Oxygen Species/metabolism

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Skocz do pozycji: 4.

Tytuł:: Rhynchophylline ameliorates myocardial ischemia/reperfusion injury through the modulation of mitochondrial mechanisms to mediate myocardial apoptosis.
Autorzy:: Qin QJ; Emergency Department, The Affiliated Hospital of Qingdao University, Qingdao, Shandong 266003, P.R. China.
Cui LQ; Department of Anesthesiology, Chengyang People's Hospital, Qingdao, Shandong 266109, P.R. China.
Li P; Emergency Department, The Affiliated Hospital of Qingdao University, Qingdao, Shandong 266003, P.R. China.
Wang YB; Emergency Department, The Affiliated Hospital of Qingdao University, Qingdao, Shandong 266003, P.R. China.
Zhang XZ; Emergency Department, The Affiliated Hospital of Qingdao University, Qingdao, Shandong 266003, P.R. China.
Guo ML; Emergency Department, The Affiliated Hospital of Qingdao University, Qingdao, Shandong 266003, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2019 Apr; Vol. 19 (4), pp. 2581-2590. Date of Electronic Publication: 2019 Jan 30.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Mitochondria/*drug effects
Mitochondria/*metabolism
Myocardial Reperfusion Injury/*metabolism
Myocytes, Cardiac/*drug effects
Myocytes, Cardiac/*metabolism
Oxindoles/*pharmacology
Animals ; Apoptosis/genetics ; Apoptosis Regulatory Proteins/genetics ; Apoptosis Regulatory Proteins/metabolism ; Biomarkers ; Calcium/metabolism ; Cell Survival/drug effects ; Gene Expression ; Matrix Metalloproteinases/metabolism ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; Myocardial Reperfusion Injury/drug therapy ; Myocardial Reperfusion Injury/etiology ; Oxidative Stress/drug effects ; Rats

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Skocz do pozycji: 5.

Tytuł:: Liraglutide protects renal mesangial cells against hyperglycemia‑mediated mitochondrial apoptosis by activating the ERK‑Yap signaling pathway and upregulating Sirt3 expression.
Autorzy:: Li J; Department of Geriatric Endocrinology, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.
Li N; Department of Geriatric Endocrinology, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.
Yan S; Department of Geriatric Endocrinology, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.
Lu Y; Department of Geriatric Endocrinology, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.
Miao X; Department of Geriatric Endocrinology, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.
Gu Z; Department of Geriatric Endocrinology, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.
Shao Y; Department of Outpatients, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2019 Apr; Vol. 19 (4), pp. 2849-2860. Date of Electronic Publication: 2019 Feb 08.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Hyperglycemia/*metabolism
Liraglutide/*pharmacology
Mesangial Cells/*drug effects
Mesangial Cells/*metabolism
Mitochondria/*drug effects
Mitochondria/*metabolism
Cell Cycle Proteins ; Cells, Cultured ; Dose-Response Relationship, Drug ; Extracellular Signal-Regulated MAP Kinases/metabolism ; Gene Expression Regulation/drug effects ; Glucose/metabolism ; Hyperglycemia/genetics ; Ion Channel Gating ; Membrane Potential, Mitochondrial/drug effects ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; Nuclear Proteins/metabolism ; Oxidative Stress/drug effects ; Protective Agents/pharmacology ; Reactive Oxygen Species/metabolism ; Signal Transduction/drug effects ; Sirtuin 3/genetics ; Transcription Factors/metabolism

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Skocz do pozycji: 6.

Tytuł:: Overexpression of appoptosin promotes mitochondrial damage in MIN6 cells.
Autorzy:: Wang T; Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, Fujian 361003, P.R. China.
Wei W; Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, Fujian 361003, P.R. China.
Mansai HAA; Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, Fujian 361003, P.R. China.
Huang C; Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, Fujian 361003, P.R. China.
Li L; Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, Fujian 361003, P.R. China.
Ye Q; Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, Fujian 361003, P.R. China.
Yin H; Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, Fujian 361003, P.R. China.
Yang C; Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, Fujian 361003, P.R. China.
Li X; Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, Fujian 361003, P.R. China.
Liu S; Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, Fujian 361003, P.R. China.
Yang S; Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, Fujian 361003, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2018 May; Vol. 17 (5), pp. 7149-7155. Date of Electronic Publication: 2018 Mar 16.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis*/drug effects
Up-Regulation*/drug effects
Insulin-Secreting Cells/*pathology
Mitochondria/*pathology
Mitochondrial Membrane Transport Proteins/*genetics
Solute Carrier Proteins/*genetics
Animals ; Caspase 3/metabolism ; Cell Line ; Cobalt/adverse effects ; Enzyme Activation/drug effects ; Insulin-Secreting Cells/cytology ; Insulin-Secreting Cells/drug effects ; Insulin-Secreting Cells/metabolism ; Mice ; Mitochondria/drug effects ; Mitochondria/genetics

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Skocz do pozycji: 7.

Tytuł:: Bax, Bak and beyond - mitochondrial performance in apoptosis.
Autorzy:: Peña-Blanco A; Interfaculty Institute of Biochemistry, Tübingen University, Germany.
García-Sáez AJ; Interfaculty Institute of Biochemistry, Tübingen University, Germany.; Max-Planck Institute for Intelligent Systems, Stuttgart, Germany.; Pokaż więcej
Źródło:: The FEBS journal [FEBS J] 2018 Feb; Vol. 285 (3), pp. 416-431. Date of Electronic Publication: 2017 Sep 04.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:: Apoptosis*
Mitochondrial Dynamics*
Models, Biological*
Apoptosis Regulatory Proteins/*metabolism
Mitochondrial Membranes/*metabolism
Animals ; Apoptosis Regulatory Proteins/agonists ; Apoptosis Regulatory Proteins/chemistry ; Dimerization ; Humans ; Lipid Bilayers/chemistry ; Lipid Bilayers/metabolism ; Lipid Mobilization ; Mitochondria/chemistry ; Mitochondria/enzymology ; Mitochondria/metabolism ; Mitochondrial Membrane Transport Proteins ; Mitochondrial Membranes/chemistry ; Mitochondrial Membranes/enzymology ; Porosity ; Protein Conformation ; Protein Interaction Domains and Motifs ; Protein Multimerization ; Proto-Oncogene Proteins c-bcl-2/agonists ; Proto-Oncogene Proteins c-bcl-2/chemistry ; Proto-Oncogene Proteins c-bcl-2/metabolism ; bcl-2 Homologous Antagonist-Killer Protein/agonists ; bcl-2 Homologous Antagonist-Killer Protein/chemistry ; bcl-2 Homologous Antagonist-Killer Protein/metabolism ; bcl-2-Associated X Protein/agonists ; bcl-2-Associated X Protein/chemistry ; bcl-2-Associated X Protein/metabolism

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Skocz do pozycji: 8.

Tytuł:: Isoflurane reduces hypoxia/reoxygenation-induced apoptosis and mitochondrial permeability transition in rat primary cultured cardiocytes.
Autorzy:: Wu W
Zhou X
Liu P
Fei W
Li L
Yun H; Department of Anesthesiology, Changzhou No,2 People's Hospital, the affiliated hospital of Nanjing Medical University, Changzhou 213003, China. .; Pokaż więcej
Źródło:: BMC anesthesiology [BMC Anesthesiol] 2014 Mar 10; Vol. 14, pp. 17. Date of Electronic Publication: 2014 Mar 10.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis/*drug effects
Isoflurane/*pharmacology
Mitochondria, Heart/*drug effects
Mitochondrial Membrane Transport Proteins/*antagonists & inhibitors
Myocytes, Cardiac/*drug effects
Animals ; Animals, Newborn ; Apoptosis/physiology ; Cell Hypoxia/drug effects ; Cell Hypoxia/physiology ; Cells, Cultured ; Male ; Mitochondria, Heart/metabolism ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; Myocytes, Cardiac/metabolism ; Rats ; Rats, Sprague-Dawley

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Skocz do pozycji: 9.

Tytuł:: Alpha-synuclein prevents the formation of spherical mitochondria and apoptosis under oxidative stress.
Autorzy:: Menges S; Department of Molecular Neurology, University Hospital Erlangen, Friedrich-Alexander-University (FAU) Erlangen-Nürnberg, 91054 Erlangen, Germany.
Minakaki G; Department of Molecular Neurology, University Hospital Erlangen, Friedrich-Alexander-University (FAU) Erlangen-Nürnberg, 91054 Erlangen, Germany.
Schaefer PM; Department of Neurology, Ulm University, 89081 Ulm, Germany.
Meixner H; Department of Molecular Neurology, University Hospital Erlangen, Friedrich-Alexander-University (FAU) Erlangen-Nürnberg, 91054 Erlangen, Germany.
Prots I; IZKF Junior Research Group III and BMBF Research Group Neuroscience, IZKF, FAU Erlangen-Nürnberg, 91054 Erlangen, Germany.; Department of Stem Cell Biology, Institute of Human Genetics, FAU Erlangen-Nürnberg, 91054 Erlangen, Germany.
Schlötzer-Schrehardt U; Department of Ophthalmology, University Hospital Erlangen, FAU Erlangen-Nürnberg, 91054 Erlangen, Germany.
Friedland K; Molecular and Clinical Pharmacy, Department of Chemistry and Pharmacy, FAU Erlangen-Nürnberg, 91054 Erlangen, Germany.
Winner B; IZKF Junior Research Group III and BMBF Research Group Neuroscience, IZKF, FAU Erlangen-Nürnberg, 91054 Erlangen, Germany.; Department of Stem Cell Biology, Institute of Human Genetics, FAU Erlangen-Nürnberg, 91054 Erlangen, Germany.
Outeiro TF; Department of Neurodegeneration and Restorative Research, University Medical Center Göttingen, 37073 Göttingen, Germany.; Max Planck Institute for Experimental Medicine, 37075 Göttingen, Germany.
Winklhofer KF; Molecular Cell Biology, Institute of Biochemistry and Pathobiochemistry, Ruhr-University Bochum, 44801 Bochum, Germany.
von Arnim CA; Department of Neurology, Ulm University, 89081 Ulm, Germany.
Xiang W; Institute of Biochemistry, FAU Erlangen-Nürnberg, 91054 Erlangen, Germany.
Winkler J; Department of Molecular Neurology, University Hospital Erlangen, Friedrich-Alexander-University (FAU) Erlangen-Nürnberg, 91054 Erlangen, Germany.
Klucken J; Department of Molecular Neurology, University Hospital Erlangen, Friedrich-Alexander-University (FAU) Erlangen-Nürnberg, 91054 Erlangen, Germany.; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2017 Feb 22; Vol. 7, pp. 42942. Date of Electronic Publication: 2017 Feb 22.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis/*drug effects
Hydrogen Peroxide/*toxicity
Mitochondria/*metabolism
Oxidative Stress/*drug effects
alpha-Synuclein/*metabolism
Animals ; Caspase 3/metabolism ; Cells, Cultured ; Dynamins ; GTP Phosphohydrolases/metabolism ; Humans ; Microtubule-Associated Proteins/metabolism ; Mitochondria/drug effects ; Mitochondrial Dynamics/drug effects ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Proteins/metabolism ; Mitophagy/drug effects ; Neurons/cytology ; Neurons/drug effects ; Neurons/metabolism ; Rats ; Rotenone/pharmacology ; Ubiquitin-Protein Ligases/metabolism

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Skocz do pozycji: 10.

Tytuł:: Caspase-Dependent and Caspase-Independent Pathways Are Involved in Cadmium-Induced Apoptosis in Primary Rat Proximal Tubular Cell Culture.
Autorzy:: Liu G; College of Veterinary Medicine, Yangzhou University, and Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, and Jiangsu Key Laboratory of Zoonosis, Yangzhou, Jiangsu, PR China.
Zou H; College of Veterinary Medicine, Yangzhou University, and Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, and Jiangsu Key Laboratory of Zoonosis, Yangzhou, Jiangsu, PR China.
Luo T; College of Veterinary Medicine, Yangzhou University, and Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, and Jiangsu Key Laboratory of Zoonosis, Yangzhou, Jiangsu, PR China.
Long M; College of Veterinary Medicine, Yangzhou University, and Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, and Jiangsu Key Laboratory of Zoonosis, Yangzhou, Jiangsu, PR China.
Bian J; College of Veterinary Medicine, Yangzhou University, and Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, and Jiangsu Key Laboratory of Zoonosis, Yangzhou, Jiangsu, PR China.
Liu X; College of Veterinary Medicine, Yangzhou University, and Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, and Jiangsu Key Laboratory of Zoonosis, Yangzhou, Jiangsu, PR China.
Gu J; College of Veterinary Medicine, Yangzhou University, and Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, and Jiangsu Key Laboratory of Zoonosis, Yangzhou, Jiangsu, PR China.
Yuan Y; College of Veterinary Medicine, Yangzhou University, and Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, and Jiangsu Key Laboratory of Zoonosis, Yangzhou, Jiangsu, PR China.
Song R; College of Veterinary Medicine, Yangzhou University, and Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, and Jiangsu Key Laboratory of Zoonosis, Yangzhou, Jiangsu, PR China.
Wang Y; College of Veterinary Medicine, Yangzhou University, and Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, and Jiangsu Key Laboratory of Zoonosis, Yangzhou, Jiangsu, PR China.
Zhu J; College of Veterinary Medicine, Yangzhou University, and Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, and Jiangsu Key Laboratory of Zoonosis, Yangzhou, Jiangsu, PR China.
Liu Z; College of Veterinary Medicine, Yangzhou University, and Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, and Jiangsu Key Laboratory of Zoonosis, Yangzhou, Jiangsu, PR China.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2016 Nov 18; Vol. 11 (11), pp. e0166823. Date of Electronic Publication: 2016 Nov 18 (Print Publication: 2016).
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Cadmium/*pharmacology
Caspases/*metabolism
Epithelial Cells/*drug effects
Epithelial Cells/*metabolism
Kidney Tubules, Proximal/*drug effects
Kidney Tubules, Proximal/*metabolism
Animals ; Caspase Inhibitors/pharmacology ; Cytochromes c/metabolism ; Endodeoxyribonucleases/metabolism ; Gene Silencing ; Membrane Proteins/genetics ; Mitochondria/drug effects ; Mitochondria/metabolism ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; Mitochondrial Proteins/genetics ; Oxidative Stress/drug effects ; Primary Cell Culture ; Rats ; Reactive Oxygen Species/metabolism ; Signal Transduction/drug effects

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Skocz do pozycji: 11.

Tytuł:: Cristae remodeling causes acidification detected by integrated graphene sensor during mitochondrial outer membrane permeabilization.
Autorzy:: Pham TD; Department of Biomedical Engineering, University of California, Irvine, CA, USA.
Pham PQ; Department of Chemical Engineering and Materials Science, University of California, Irvine, CA, USA.
Li J; Department of Chemical Engineering and Materials Science, University of California, Irvine, CA, USA.
Letai AG; Dana-Farber Cancer Institute, Harvard University, Boston, MA, USA.
Wallace DC; Center for Mitochondrial and Epigenomic Medicine, Children's Hospital of Philadelphia and Department of Pathology and Laboratory Medicine, University of Pennsylvania, Philadelphia, PA, USA.
Burke PJ; Department of Biomedical Engineering, University of California, Irvine, CA, USA.; Department of Chemical Engineering and Materials Science, University of California, Irvine, CA, USA.; Department of Electrical Engineering and Computer Science, University of California, Irvine, CA, USA.; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2016 Oct 27; Vol. 6, pp. 35907. Date of Electronic Publication: 2016 Oct 27.
Typ publikacji:: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms:: Apoptosis/*physiology
Mitochondrial Membranes/*metabolism
Bcl-2-Like Protein 11/metabolism ; Biosensing Techniques ; Carbonyl Cyanide m-Chlorophenyl Hydrazone/pharmacology ; Cell Line ; Cytochromes c/metabolism ; GTP Phosphohydrolases/metabolism ; Graphite ; HeLa Cells ; Humans ; Hydrogen-Ion Concentration ; Membrane Potential, Mitochondrial/drug effects ; Metalloendopeptidases/metabolism ; Mitochondrial Membrane Transport Proteins ; Mitochondrial Membranes/drug effects ; Nanostructures ; Permeability ; Proto-Oncogene Proteins c-bcl-2/metabolism ; bcl-2 Homologous Antagonist-Killer Protein/metabolism

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Skocz do pozycji: 12.

Tytuł:: Clematichinenoside (AR) Attenuates Hypoxia/Reoxygenation-Induced H9c2 Cardiomyocyte Apoptosis via a Mitochondria-Mediated Signaling Pathway.
Autorzy:: Ding H; State Key Laboratory of Natural Medicines, Department of Physiology, China Pharmaceutical University, Nanjing 210009, China. .; College of Pharmacy, Xinjiang Medical University, Urumqi 830011, China. .
Han R; College of Pharmacy, Xinjiang Medical University, Urumqi 830011, China. .
Chen X; College of Pharmacy, Xinjiang Medical University, Urumqi 830011, China. .
Fang W; State Key Laboratory of Natural Medicines, Department of Physiology, China Pharmaceutical University, Nanjing 210009, China. .
Liu M; Cancer Hospital Affiliated to Xinjiang Medical University, Urumqi 830011, China. .
Wang X; Xinjiang Key Laboratory of Medical Animal Model Research, Clinical Medical Research Institute of the First Affiliated Hospital of Xinjiang Medical University, Urumqi 830011, China. .
Wei Q; Xinjiang Key Laboratory of Medical Animal Model Research, Clinical Medical Research Institute of the First Affiliated Hospital of Xinjiang Medical University, Urumqi 830011, China. .
Kodithuwakku ND; Institute of Indigenous Medicine, University of Colombo, Rajagiriya 11600, Sri Lanka. darshi_.
Li Y; State Key Laboratory of Natural Medicines, Department of Physiology, China Pharmaceutical University, Nanjing 210009, China. .; Pokaż więcej
Źródło:: Molecules (Basel, Switzerland) [Molecules] 2016 May 30; Vol. 21 (6). Date of Electronic Publication: 2016 May 30.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Mitochondria/*drug effects
Mitochondria/*metabolism
Myocytes, Cardiac/*drug effects
Myocytes, Cardiac/*metabolism
Saponins/*pharmacology
Signal Transduction/*drug effects
Animals ; Biomarkers ; Caspase 3/metabolism ; Cell Line, Tumor ; Cell Survival/drug effects ; Cytochromes c/metabolism ; Hypoxia/metabolism ; Ion Channel Gating ; Membrane Potential, Mitochondrial/drug effects ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; Myocardial Reperfusion Injury/metabolism ; Protective Agents/chemistry ; Protective Agents/pharmacology ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Saponins/chemistry ; bcl-2-Associated X Protein/metabolism

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Skocz do pozycji: 13.

Tytuł:: Erastin Disrupts Mitochondrial Permeability Transition Pore (mPTP) and Induces Apoptotic Death of Colorectal Cancer Cells.
Autorzy:: Huo H; Department of General Surgery, The Ninth People's Hospital Affiliated to Shanghai Jiao-Tong University School of Medicine, Shanghai, China.
Zhou Z; Department of General Surgery, The Ninth People's Hospital Affiliated to Shanghai Jiao-Tong University School of Medicine, Shanghai, China.
Qin J; Department of General Surgery, The Ninth People's Hospital Affiliated to Shanghai Jiao-Tong University School of Medicine, Shanghai, China.
Liu W; Department of General Surgery, The Ninth People's Hospital Affiliated to Shanghai Jiao-Tong University School of Medicine, Shanghai, China.
Wang B; Department of General Surgery, The Ninth People's Hospital Affiliated to Shanghai Jiao-Tong University School of Medicine, Shanghai, China.
Gu Y; Department of General Surgery, The Ninth People's Hospital Affiliated to Shanghai Jiao-Tong University School of Medicine, Shanghai, China.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2016 May 12; Vol. 11 (5), pp. e0154605. Date of Electronic Publication: 2016 May 12 (Print Publication: 2016).
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Colorectal Neoplasms/*metabolism
Colorectal Neoplasms/*pathology
Mitochondrial Membrane Transport Proteins/*metabolism
Piperazines/*pharmacology
Animals ; Caspase 3/metabolism ; Caspase 9/metabolism ; Cell Line, Tumor ; Cell Proliferation/drug effects ; HT29 Cells ; Humans ; Mice, SCID ; Mitochondrial Permeability Transition Pore ; Piperazines/administration & dosage ; Reactive Oxygen Species/metabolism ; Voltage-Dependent Anion Channel 1/metabolism ; Xenograft Model Antitumor Assays

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Skocz do pozycji: 14.

Tytuł:: Involvement of intrinsic mitochondrial pathway in neosergeolide-induced apoptosis of human HL-60 leukemia cells: the role of mitochondrial permeability transition pore and DNA damage.
Autorzy:: Cavalcanti BC; National Laboratory of Experimental Oncology, Federal University of Ceará, CEP 60430-270, Fortaleza, CE, Brazil. nunim_ />da Costa PM
Carvalho AA
Rodrigues FA
Amorim RC
Silva EC
Pohlit AM
Costa-Lotufo LV
Moraes MO
Pessoa C; Pokaż więcej
Źródło:: Pharmaceutical biology [Pharm Biol] 2012 Aug; Vol. 50 (8), pp. 980-93.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Antineoplastic Agents, Phytogenic/*pharmacology
Apoptosis/*drug effects
DNA Fragmentation/*drug effects
Leukemia, Promyelocytic, Acute/*drug therapy
Mitochondria/*drug effects
Mitochondrial Membrane Transport Proteins/*metabolism
Quassins/*pharmacology
Antineoplastic Agents, Phytogenic/adverse effects ; Antineoplastic Agents, Phytogenic/antagonists & inhibitors ; Cell Proliferation/drug effects ; Cells, Cultured ; Comet Assay ; Cyclosporine/pharmacology ; Cytokinesis/drug effects ; HL-60 Cells ; Humans ; Inhibitory Concentration 50 ; Leukemia, Promyelocytic, Acute/metabolism ; Leukemia, Promyelocytic, Acute/pathology ; Leukocytes, Mononuclear/drug effects ; Membrane Potential, Mitochondrial/drug effects ; Micronucleus Tests ; Mitochondria/metabolism ; Mitochondrial Membrane Transport Proteins/antagonists & inhibitors ; Mitochondrial Permeability Transition Pore ; Neoplasm Proteins/antagonists & inhibitors ; Neoplasm Proteins/metabolism ; Quassins/adverse effects ; Quassins/antagonists & inhibitors ; Simaroubaceae/chemistry

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Skocz do pozycji: 15.

Tytuł:: The obesity-related peptide leptin sensitizes cardiac mitochondria to calcium-induced permeability transition pore opening and apoptosis.
Autorzy:: Martinez-Abundis E; Department of Physiology and Pharmacology, Schulich School of Medicine and Dentistry, University of Western Ontario, London, Ontario, Canada.
Rajapurohitam V
Haist JV
Gan XT
Karmazyn M; Pokaż więcej
Źródło:: PloS one [PLoS One] 2012; Vol. 7 (7), pp. e41612. Date of Electronic Publication: 2012 Jul 25.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis/*drug effects
Calcium/*pharmacology
Leptin/*pharmacology
Mitochondria/*metabolism
Mitochondrial Membrane Transport Proteins/*chemistry
Myocytes, Cardiac/*cytology
Obesity/*metabolism
Animals ; Digitonin/pharmacology ; Hypertrophy/chemically induced ; Hypertrophy/metabolism ; Hypertrophy/pathology ; Leptin/metabolism ; Mitochondria/drug effects ; Mitochondria/pathology ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; Myocytes, Cardiac/drug effects ; Myocytes, Cardiac/metabolism ; Myocytes, Cardiac/pathology ; Protein Conformation/drug effects ; Rats ; Rats, Sprague-Dawley ; STAT3 Transcription Factor/metabolism ; Time Factors ; rho-Associated Kinases/metabolism

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Skocz do pozycji: 16.

Tytuł:: Intra-mitochondrial degradation of Tim23 curtails the survival of cells rescued from apoptosis by caspase inhibitors.
Autorzy:: Goemans CG; Department of Biochemistry, University of Cambridge, Cambridge, UK. />Boya P
Skirrow CJ
Tolkovsky AM; Pokaż więcej
Źródło:: Cell death and differentiation [Cell Death Differ] 2008 Mar; Vol. 15 (3), pp. 545-54. Date of Electronic Publication: 2008 Jan 04.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis*
Caspase Inhibitors*
Mitochondrial Membrane Transport Proteins/*metabolism
Biological Transport ; Cell Cycle ; Cell Survival ; Cells, Cultured ; Cysteine Proteinase Inhibitors/pharmacology ; HeLa Cells ; Humans ; Mitochondria/enzymology ; Mitochondria/metabolism ; Mitochondrial Membrane Transport Proteins/antagonists & inhibitors ; Mitochondrial Membrane Transport Proteins/genetics ; Mitochondrial Precursor Protein Import Complex Proteins ; RNA Interference ; bcl-2 Homologous Antagonist-Killer Protein/metabolism ; bcl-2-Associated X Protein/metabolism

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Skocz do pozycji: 17.

Tytuł:: Involvement of mitochondrial permeability transition pore opening in alpha-bisabolol induced apoptosis.
Autorzy:: Cavalieri E; Dipartimento di Scienze Morfologico-Biomediche, Università di Verona, Italy.
Bergamini C
Mariotto S
Leoni S
Perbellini L
Darra E
Suzuki H
Fato R
Lenaz G; Pokaż więcej
Źródło:: The FEBS journal [FEBS J] 2009 Aug; Vol. 276 (15), pp. 3990-4000. Date of Electronic Publication: 2009 Jun 29.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis/*drug effects
Mitochondrial Membrane Transport Proteins/*physiology
Sesquiterpenes/*pharmacology
Breast Neoplasms ; Cell Line, Tumor ; Cell Survival/drug effects ; Female ; Fibroblasts/drug effects ; Fibroblasts/physiology ; Glioma ; Humans ; Male ; Mitochondrial Membrane Transport Proteins/drug effects ; Mitochondrial Permeability Transition Pore ; Monocyclic Sesquiterpenes ; Neutrophils/drug effects ; Neutrophils/physiology ; Prostatic Neoplasms ; T-Lymphocytes/drug effects ; T-Lymphocytes/physiology

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Skocz do pozycji: 18.

Tytuł:: Bufalin induces apoptosis in the U‑2OS human osteosarcoma cell line via triggering the mitochondrial pathway.
Autorzy:: Chen Y; Department of Pharmacy, People's Hospital of Zhangqiu, Shandong 250200, P.R. China.
Li M; Department of Orthopaedic Surgery, People's Hospital of Zhangqiu, Shandong 250200, P.R. China.
Li Z; Department of Orthopaedic Surgery, People's Hospital of Zhangqiu, Shandong 250200, P.R. China.
Gao P; Department of Orthopaedic Surgery, People's Hospital of Zhangqiu, Shandong 250200, P.R. China.
Zhou X; Department of Orthopaedic Surgery, People's Hospital of Zhangqiu, Shandong 250200, P.R. China.
Zhang J; Spinal Department of Orthopaedic Surgery, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan, Shandong 250014, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2016 Jan; Vol. 13 (1), pp. 817-22. Date of Electronic Publication: 2015 Nov 19.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Bufanolides/*pharmacology
Mitochondria/*metabolism
Osteosarcoma/*pathology
Signal Transduction/*drug effects
Blotting, Western ; Bufanolides/chemistry ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Cyclosporine/pharmacology ; Flow Cytometry ; Humans ; Membrane Potential, Mitochondrial/drug effects ; Microscopy, Fluorescence ; Mitochondria/drug effects ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore

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Skocz do pozycji: 19.

Tytuł:: Modulation of P2X4/P2X7/Pannexin-1 sensitivity to extracellular ATP via Ivermectin induces a non-apoptotic and inflammatory form of cancer cell death.
Autorzy:: Draganov D; Department of Immuno-Oncology, City of Hope, Duarte, CA, US.
Gopalakrishna-Pillai S; Department of Immuno-Oncology, City of Hope, Duarte, CA, US.
Chen YR; Diabetes &Metabolism Research Institute, City of Hope, Duarte, CA, US.
Zuckerman N; Department of Immuno-Oncology, City of Hope, Duarte, CA, US.
Moeller S; Department of Immuno-Oncology, City of Hope, Duarte, CA, US.
Wang C; Department of Immuno-Oncology, City of Hope, Duarte, CA, US.
Ann D; Diabetes &Metabolism Research Institute, City of Hope, Duarte, CA, US.
Lee PP; Department of Immuno-Oncology, City of Hope, Duarte, CA, US.; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2015 Nov 10; Vol. 5, pp. 16222. Date of Electronic Publication: 2015 Nov 10.
Typ publikacji:: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms:: Adenosine Triphosphate/*metabolism
Apoptosis/*drug effects
Connexins/*metabolism
Ivermectin/*toxicity
Nerve Tissue Proteins/*metabolism
Receptors, Purinergic P2X4/*metabolism
Receptors, Purinergic P2X7/*metabolism
Allosteric Regulation ; Animals ; Autophagy/drug effects ; Calcium-Calmodulin-Dependent Protein Kinase Type 2/metabolism ; Caspase 1/metabolism ; Cell Line, Tumor ; Humans ; Mice ; Mice, Inbred BALB C ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; NADPH Oxidases/antagonists & inhibitors ; NADPH Oxidases/metabolism ; Poly(ADP-ribose) Polymerases/metabolism ; RNA Interference ; Reactive Oxygen Species/metabolism ; Receptors, Purinergic P2X4/genetics ; Receptors, Purinergic P2X7/chemistry ; Receptors, Purinergic P2X7/genetics ; Signal Transduction/drug effects

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Skocz do pozycji: 20.

Tytuł:: Necroptosis Interfaces with MOMP and the MPTP in Mediating Cell Death.
Autorzy:: Karch J; Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati, Cincinnati, Ohio, United States of America.
Kanisicak O; Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati, Cincinnati, Ohio, United States of America.
Brody MJ; Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati, Cincinnati, Ohio, United States of America.
Sargent MA; Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati, Cincinnati, Ohio, United States of America.
Michael DM; Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati, Cincinnati, Ohio, United States of America.
Molkentin JD; Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati, Cincinnati, Ohio, United States of America; Howard Hughes Medical Institute, Cincinnati, Ohio, United States of America.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2015 Jun 10; Vol. 10 (6), pp. e0130520. Date of Electronic Publication: 2015 Jun 10 (Print Publication: 2015).
Typ publikacji:: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis*
Mitochondria/*metabolism
Mitochondrial Membrane Transport Proteins/*metabolism
Animals ; Peptidyl-Prolyl Isomerase F ; Cyclophilins/genetics ; Fibroblasts/metabolism ; Fibroblasts/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Mitochondrial Permeability Transition Pore ; Proto-Oncogene Proteins c-bcl-2/genetics ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Receptor-Interacting Protein Serine-Threonine Kinases/genetics ; bcl-2 Homologous Antagonist-Killer Protein/genetics ; bcl-2 Homologous Antagonist-Killer Protein/metabolism ; bcl-2-Associated X Protein/genetics ; bcl-2-Associated X Protein/metabolism

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