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Wyszukujesz frazę ""Mitochondrial Transmembrane Permeability-Driven Necrosis"" wg kryterium: Temat


Wyświetlanie 1-13 z 13
Tytuł:
Mitochondrial Permeability Transition, Cell Death and Neurodegeneration.
Autorzy:
Baev AY; Laboratory of Experimental Biophysics, Centre for Advanced Technologies, Tashkent 100174, Uzbekistan.; Department of Biophysics, Faculty of Biology, National University of Uzbekistan, Tashkent 100174, Uzbekistan.
Vinokurov AY; Cell Physiology and Pathology Laboratory, Orel State University, Orel 302026, Russia.
Potapova EV; Cell Physiology and Pathology Laboratory, Orel State University, Orel 302026, Russia.
Dunaev AV; Cell Physiology and Pathology Laboratory, Orel State University, Orel 302026, Russia.
Angelova PR; Department of Clinical and Movement Neurosciences, UCL Queen Square Institute of Neurology, Queen Square, London WC1N 3BG, UK.
Abramov AY; Department of Clinical and Movement Neurosciences, UCL Queen Square Institute of Neurology, Queen Square, London WC1N 3BG, UK.
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Źródło:
Cells [Cells] 2024 Apr 08; Vol. 13 (7). Date of Electronic Publication: 2024 Apr 08.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Mitochondrial Transmembrane Permeability-Driven Necrosis*
Neurodegenerative Diseases*/metabolism
Humans ; Mitochondria/metabolism ; Cell Death/physiology ; Necrosis/metabolism
Czasopismo naukowe
Tytuł:
Mitochondrial Permeability Transition.
Autorzy:
Bernardi P; Department of Biomedical Sciences, University of Padova, Via Ugo Bassi 58/B, I-35131 Padova, Italy.
Pavlov E; College of Dentistry, Department of Molecular Pathobiology, New York University, New York, NY 10010, USA.
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Źródło:
Cells [Cells] 2022 Nov 30; Vol. 11 (23). Date of Electronic Publication: 2022 Nov 30.
Typ publikacji:
Editorial
MeSH Terms:
Mitochondrial Transmembrane Permeability-Driven Necrosis*
Mitochondrial Membranes*
Mitochondrial Permeability Transition Pore ; Permeability
Opinia redakcyjna
Tytuł:
The mitochondrial permeability transition: Recent progress and open questions.
Autorzy:
Bernardi P; Department of Biomedical Sciences and CNR Neuroscience Institute, University of Padova, Italy.
Carraro M; Department of Biomedical Sciences and CNR Neuroscience Institute, University of Padova, Italy.
Lippe G; Department of Medicine, University of Udine, Italy.
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Źródło:
The FEBS journal [FEBS J] 2022 Nov; Vol. 289 (22), pp. 7051-7074. Date of Electronic Publication: 2021 Nov 12.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
Mitochondrial Transmembrane Permeability-Driven Necrosis*
Mitochondrial Permeability Transition Pore*
Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Proton-Translocating ATPases/genetics ; Mitochondrial Proton-Translocating ATPases/metabolism ; Permeability ; Adenosine Triphosphate/metabolism ; Calcium/metabolism
Czasopismo naukowe
Tytuł:
Refractive Index Imaging Reveals That Elimination of the ATP Synthase C Subunit Does Not Prevent the Adenine Nucleotide Translocase-Dependent Mitochondrial Permeability Transition.
Autorzy:
Neginskaya MA; Department of Molecular Pathobiology, New York University, 345 East 24th Street, New York, NY 10010, USA.; Department of Medicine, Albert Einstein College of Medicine, 1300 Morris Park Ave, New York, NY 10461, USA.
Morris SE; Department of Molecular Pathobiology, New York University, 345 East 24th Street, New York, NY 10010, USA.
Pavlov EV; Department of Molecular Pathobiology, New York University, 345 East 24th Street, New York, NY 10010, USA.
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Źródło:
Cells [Cells] 2023 Jul 27; Vol. 12 (15). Date of Electronic Publication: 2023 Jul 27.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Mitochondrial Membrane Transport Proteins*/metabolism
Mitochondria*/metabolism
Mitochondrial Permeability Transition Pore/metabolism ; Mitochondrial Transmembrane Permeability-Driven Necrosis ; Refractometry ; Mitochondrial ADP, ATP Translocases/metabolism ; Adenosine Triphosphate/metabolism
Czasopismo naukowe
Tytuł:
Diplacone Isolated from Paulownia tomentosa Mature Fruit Induces Ferroptosis-Mediated Cell Death through Mitochondrial Ca Influx and Mitochondrial Permeability Transition.
Autorzy:
Kang MJ; Natural Product Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Cheongju 28116, Republic of Korea.
Ryu HW; Natural Product Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Cheongju 28116, Republic of Korea.
Oh ES; Natural Product Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Cheongju 28116, Republic of Korea.; Department of Biological Sciences, College of Bioscience and Biotechnology, Chungnam National University, Daejeon 34134, Republic of Korea.
Song YN; Natural Product Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Cheongju 28116, Republic of Korea.; Department of Biological Sciences, College of Bioscience and Biotechnology, Chungnam National University, Daejeon 34134, Republic of Korea.
Huh YH; Korea Basic Science Institute, Cheongju 28119, Republic of Korea.
Park JY; Natural Product Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Cheongju 28116, Republic of Korea.; Departments of Anatomy & Cell Biology, Department of Medical Science, College of Medicine, Chungnam National University, Daejeon 35015, Republic of Korea.
Oh SM; Natural Product Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Cheongju 28116, Republic of Korea.
Lee SY; Natural Product Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Cheongju 28116, Republic of Korea.
Park YJ; Natural Product Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Cheongju 28116, Republic of Korea.
Kim DY; Natural Product Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Cheongju 28116, Republic of Korea.
Ro H; Department of Biological Sciences, College of Bioscience and Biotechnology, Chungnam National University, Daejeon 34134, Republic of Korea.
Hong ST; Departments of Anatomy & Cell Biology, Department of Medical Science, College of Medicine, Chungnam National University, Daejeon 35015, Republic of Korea.
Lee SU; Natural Product Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Cheongju 28116, Republic of Korea.
Moon DO; Department of Biology Education, Daegu University, 201 Daegudae-ro, Gyeongsan-si 38453, Republic of Korea.
Kim MO; Natural Product Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Cheongju 28116, Republic of Korea.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Apr 11; Vol. 24 (8). Date of Electronic Publication: 2023 Apr 11.
Typ publikacji:
Journal Article
MeSH Terms:
Ferroptosis*
Humans ; Mitochondrial Transmembrane Permeability-Driven Necrosis ; Fruit/metabolism ; Cell Death/physiology ; Reactive Oxygen Species/metabolism ; Glutathione/metabolism ; Lipid Peroxidation ; Mitochondrial Permeability Transition Pore/metabolism
Czasopismo naukowe
Tytuł:
Transport of Ca -Dependent Permeability Transition in Rat Liver Mitochondria under the Streptozotocin-Induced Type I Diabetes.
Autorzy:
Belosludtsev KN; Laboratory of mitochondrial transport, Institute of Theoretical and Experimental Biophysics, Russian Academy of Sciences, Institutskaya 3, 142290 Pushchino, Moscow region, Russia. .; Department of biochemistry, cell biology and microbiology, Mari State University, pl. Lenina 1, Yoshkar-Ola, 424001 Mari El, Russia. .
Talanov EY; Laboratory of mitochondrial transport, Institute of Theoretical and Experimental Biophysics, Russian Academy of Sciences, Institutskaya 3, 142290 Pushchino, Moscow region, Russia.
Starinets VS; Department of biochemistry, cell biology and microbiology, Mari State University, pl. Lenina 1, Yoshkar-Ola, 424001 Mari El, Russia.
Agafonov AV; Laboratory of mitochondrial transport, Institute of Theoretical and Experimental Biophysics, Russian Academy of Sciences, Institutskaya 3, 142290 Pushchino, Moscow region, Russia.
Dubinin MV; Department of biochemistry, cell biology and microbiology, Mari State University, pl. Lenina 1, Yoshkar-Ola, 424001 Mari El, Russia.
Belosludtseva NV; Laboratory of mitochondrial transport, Institute of Theoretical and Experimental Biophysics, Russian Academy of Sciences, Institutskaya 3, 142290 Pushchino, Moscow region, Russia.
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Źródło:
Cells [Cells] 2019 Aug 30; Vol. 8 (9). Date of Electronic Publication: 2019 Aug 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Mitochondrial Transmembrane Permeability-Driven Necrosis*
Calcium/*metabolism
Diabetes Mellitus, Experimental/*metabolism
Mitochondria, Liver/*metabolism
Animals ; Male ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Tytuł:
Mitochondrial Permeability Transition Causes Mitochondrial Reactive Oxygen Species- and Caspase 3-Dependent Atrophy of Single Adult Mouse Skeletal Muscle Fibers.
Autorzy:
Skinner SK; Department of Physical Therapy, University of Florida, Gainesville, FL 32610, USA.
Solania A; Departments of Molecular Medicine and Integrative Structural and Computational Biology, Scripps Research, La Jolla, San Diego, CA 92037, USA.
Wolan DW; Departments of Molecular Medicine and Integrative Structural and Computational Biology, Scripps Research, La Jolla, San Diego, CA 92037, USA.
Cohen MS; Department of Chemical Physiology and Biochemistry, Oregon Health and Science University, Portland, OR 97239, USA.
Ryan TE; Department of Applied Physiology and Kinesiology, University of Florida, Gainesville, FL 32610, USA.
Hepple RT; Department of Physical Therapy, University of Florida, Gainesville, FL 32610, USA.
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Źródło:
Cells [Cells] 2021 Sep 29; Vol. 10 (10). Date of Electronic Publication: 2021 Sep 29.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Caspase 3/*metabolism
Mitochondrial Transmembrane Permeability-Driven Necrosis/*physiology
Muscle Fibers, Skeletal/*metabolism
Reactive Oxygen Species/*metabolism
Animals ; Disease Models, Animal ; Male ; Mice ; Muscle Fibers, Skeletal/enzymology
Czasopismo naukowe
Tytuł:
Conjugation of Aminoadamantane and γ-Carboline Pharmacophores Gives Rise to Unexpected Properties of Multifunctional Ligands.
Autorzy:
Bachurin SO; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Makhaeva GF; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Shevtsova EF; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Aksinenko AY; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Grigoriev VV; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Shevtsov PN; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Goreva TV; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Epishina TA; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Kovaleva NV; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Pushkareva EA; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Boltneva NP; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Lushchekina SV; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.; Emanuel Institute of Biochemical Physics, Russian Academy of Sciences, 119334 Moscow, Russia.
Gabrelyan AV; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Zamoyski VL; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Dubova LG; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Rudakova EV; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Fisenko VP; I.M.Sechenov First Moscow State Medical University, 8 Build. 2 Trubetskaya Str., 119991 Moscow, Russia.
Bovina EV; Institute of Physiologically Active Compounds, Russian Academy of Sciences, 142432 Chernogolovka, Russia.
Richardson RJ; Department of Environmental Health Sciences, University of Michigan, Ann Arbor, MI 48109, USA.
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2021 Sep 11; Vol. 26 (18). Date of Electronic Publication: 2021 Sep 11.
Typ publikacji:
Journal Article
MeSH Terms:
Amantadine/*chemistry
Amantadine/*pharmacology
Carbolines/*chemistry
Carbolines/*pharmacology
Cholinesterase Inhibitors/*chemistry
Cholinesterase Inhibitors/*pharmacology
Acetylcholinesterase/chemistry ; Amantadine/analogs & derivatives ; Animals ; Butyrylcholinesterase/chemistry ; Carboxylesterase/chemistry ; Catalytic Domain ; Cell Line ; Cholinesterase Inhibitors/chemical synthesis ; Horses ; Humans ; Kinetics ; Ligands ; Memantine/chemistry ; Memantine/pharmacology ; Mitochondrial Transmembrane Permeability-Driven Necrosis/drug effects ; Molecular Docking Simulation ; Propidium/chemistry ; Rats ; Receptors, N-Methyl-D-Aspartate/chemistry ; Receptors, N-Methyl-D-Aspartate/metabolism ; Structure-Activity Relationship ; Swine ; Tubulin/drug effects ; Tubulin Modulators/chemical synthesis ; Tubulin Modulators/chemistry ; Tubulin Modulators/pharmacology
Czasopismo naukowe
Tytuł:
Bisindolylpyrrole triggers transient mitochondrial permeability transitions to cause apoptosis in a VDAC1/2 and cyclophilin D-dependent manner via the ANT-associated pore.
Autorzy:
Koushi M; Department of Morphophysiology, Faculty of Pharmaceutical Sciences, Josai International University, 1 Gumyo, Togane, Chiba, 283-8555, Japan.
Aoyama Y; Department of Morphophysiology, Faculty of Pharmaceutical Sciences, Josai International University, 1 Gumyo, Togane, Chiba, 283-8555, Japan.
Kamei Y; Department of Morphophysiology, Faculty of Pharmaceutical Sciences, Josai International University, 1 Gumyo, Togane, Chiba, 283-8555, Japan.
Asakai R; Department of Morphophysiology, Faculty of Pharmaceutical Sciences, Josai International University, 1 Gumyo, Togane, Chiba, 283-8555, Japan. .
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Źródło:
Scientific reports [Sci Rep] 2020 Oct 12; Vol. 10 (1), pp. 16751. Date of Electronic Publication: 2020 Oct 12.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Apoptosis/*genetics
Peptidyl-Prolyl Isomerase F/*genetics
Peptidyl-Prolyl Isomerase F/*metabolism
Cytoprotection/*drug effects
Cytoprotection/*genetics
Mitochondria, Liver/*metabolism
Mitochondrial ADP, ATP Translocases/*genetics
Mitochondrial ADP, ATP Translocases/*metabolism
Mitochondrial Transmembrane Permeability-Driven Necrosis/*drug effects
Pyrroles/*pharmacology
Voltage-Dependent Anion Channel 1/*genetics
Voltage-Dependent Anion Channel 1/*metabolism
Voltage-Dependent Anion Channel 2/*genetics
Voltage-Dependent Anion Channel 2/*metabolism
Adenosine Diphosphate ; Animals ; Calcium/metabolism ; Gene Knockdown Techniques ; HeLa Cells ; Humans ; Protein Kinase C/metabolism ; Protein Kinase C/physiology ; RNA, Small Interfering/genetics ; Rats
Czasopismo naukowe
Tytuł:
Comparison of Effects of Metformin, Phenformin, and Inhibitors of Mitochondrial Complex I on Mitochondrial Permeability Transition and Ischemic Brain Injury.
Autorzy:
Skemiene K; Neuroscience Institute, Lithuanian University of Health Sciences, LT-50161 Kaunas, Lithuania.
Rekuviene E; Neuroscience Institute, Lithuanian University of Health Sciences, LT-50161 Kaunas, Lithuania.
Jekabsone A; Neuroscience Institute, Lithuanian University of Health Sciences, LT-50161 Kaunas, Lithuania.
Cizas P; Neuroscience Institute, Lithuanian University of Health Sciences, LT-50161 Kaunas, Lithuania.
Morkuniene R; Neuroscience Institute, Lithuanian University of Health Sciences, LT-50161 Kaunas, Lithuania.
Borutaite V; Neuroscience Institute, Lithuanian University of Health Sciences, LT-50161 Kaunas, Lithuania.
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Źródło:
Biomolecules [Biomolecules] 2020 Oct 01; Vol. 10 (10). Date of Electronic Publication: 2020 Oct 01.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Brain Injuries/*drug therapy
Metformin/*administration & dosage
Mitochondria/*drug effects
Phenformin/*administration & dosage
Animals ; Brain/drug effects ; Brain/metabolism ; Brain/pathology ; Brain Injuries/metabolism ; Brain Injuries/pathology ; Electron Transport Complex I/genetics ; Humans ; Male ; Mitochondria/genetics ; Mitochondrial Permeability Transition Pore/antagonists & inhibitors ; Mitochondrial Permeability Transition Pore/metabolism ; Mitochondrial Transmembrane Permeability-Driven Necrosis/drug effects ; Necrosis/drug therapy ; Necrosis/metabolism ; Necrosis/pathology ; Neurons/drug effects ; Neurons/metabolism ; Neurons/pathology ; Neuroprotective Agents/administration & dosage ; Oxidative Phosphorylation/drug effects ; Rats
Czasopismo naukowe
Tytuł:
Cell Death in the Kidney.
Autorzy:
Priante G; Kidney Histomorphology and Molecular Biology Laboratory, Clinical Nephrology, Department of Medicine - DIMED, University of Padua, via Giustiniani 2, 35128 Padova, Italy. .
Gianesello L; Kidney Histomorphology and Molecular Biology Laboratory, Clinical Nephrology, Department of Medicine - DIMED, University of Padua, via Giustiniani 2, 35128 Padova, Italy.
Ceol M; Kidney Histomorphology and Molecular Biology Laboratory, Clinical Nephrology, Department of Medicine - DIMED, University of Padua, via Giustiniani 2, 35128 Padova, Italy.
Del Prete D; Kidney Histomorphology and Molecular Biology Laboratory, Clinical Nephrology, Department of Medicine - DIMED, University of Padua, via Giustiniani 2, 35128 Padova, Italy.
Anglani F; Kidney Histomorphology and Molecular Biology Laboratory, Clinical Nephrology, Department of Medicine - DIMED, University of Padua, via Giustiniani 2, 35128 Padova, Italy.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2019 Jul 23; Vol. 20 (14). Date of Electronic Publication: 2019 Jul 23.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Acute Kidney Injury/*metabolism
Apoptosis Regulatory Proteins/*genetics
Calcinosis/*metabolism
Epithelial Cells/*metabolism
Kidney/*metabolism
Necrosis/*metabolism
Reperfusion Injury/*metabolism
Acute Kidney Injury/drug therapy ; Acute Kidney Injury/genetics ; Acute Kidney Injury/pathology ; Animals ; Apoptosis/drug effects ; Apoptosis/genetics ; Apoptosis Regulatory Proteins/classification ; Apoptosis Regulatory Proteins/metabolism ; Calcinosis/genetics ; Calcinosis/pathology ; Calcinosis/prevention & control ; Epithelial Cells/drug effects ; Epithelial Cells/pathology ; Ferroptosis/drug effects ; Ferroptosis/genetics ; Gene Expression Regulation ; Humans ; Immunogenic Cell Death/drug effects ; Immunogenic Cell Death/genetics ; Kidney/drug effects ; Kidney/pathology ; Mitochondrial Transmembrane Permeability-Driven Necrosis/drug effects ; Mitochondrial Transmembrane Permeability-Driven Necrosis/genetics ; Necroptosis/drug effects ; Necroptosis/genetics ; Necrosis/genetics ; Necrosis/pathology ; Necrosis/prevention & control ; Protective Agents/pharmacology ; Pyroptosis/drug effects ; Pyroptosis/genetics ; Reperfusion Injury/drug therapy ; Reperfusion Injury/genetics ; Reperfusion Injury/pathology
Czasopismo naukowe
Tytuł:
Cyclophilin D binds to the acidic C-terminus region of α-Synuclein and affects its aggregation characteristics.
Autorzy:
Torpey J; NMR Centre for Structural Biology and Institute of Systems, Molecular and Integrative Biology, University of Liverpool, Liverpool, L69 7ZB, UK.
Madine J; NMR Centre for Structural Biology and Institute of Systems, Molecular and Integrative Biology, University of Liverpool, Liverpool, L69 7ZB, UK.
Wood A; NMR Centre for Structural Biology and Institute of Systems, Molecular and Integrative Biology, University of Liverpool, Liverpool, L69 7ZB, UK.
Lian LY; NMR Centre for Structural Biology and Institute of Systems, Molecular and Integrative Biology, University of Liverpool, Liverpool, L69 7ZB, UK. .
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Źródło:
Scientific reports [Sci Rep] 2020 Jun 23; Vol. 10 (1), pp. 10159. Date of Electronic Publication: 2020 Jun 23.
Typ publikacji:
Journal Article
MeSH Terms:
Protein Aggregates*
Protein Aggregation, Pathological*
Peptidyl-Prolyl Isomerase F/*metabolism
Peptidyl-Prolyl Isomerase F/*physiology
alpha-Synuclein/*metabolism
Catalysis ; Cyclohexane Monoterpenes ; In Vitro Techniques ; Mitochondria/metabolism ; Mitochondrial Transmembrane Permeability-Driven Necrosis ; Molecular Chaperones ; Protein Binding ; Solubility
Czasopismo naukowe
Tytuł:
Calreticulin is a Critical Cell Survival Factor in Malignant Neoplasms.
Autorzy:
Han A; Sue and Bill Gross Stem Cell Research Center, Department of Molecular Biology and Biochemistry, University of California-Irvine, Irvine, California, United States of America.
Li C; Sue and Bill Gross Stem Cell Research Center, Department of Molecular Biology and Biochemistry, University of California-Irvine, Irvine, California, United States of America.
Zahed T; Sue and Bill Gross Stem Cell Research Center, Department of Molecular Biology and Biochemistry, University of California-Irvine, Irvine, California, United States of America.
Wong M; Sue and Bill Gross Stem Cell Research Center, Department of Molecular Biology and Biochemistry, University of California-Irvine, Irvine, California, United States of America.
Smith I; Center for Autism Research and Translation, University of California-Irvine, Irvine, California, United States of America.
Hoedel K; Sue and Bill Gross Stem Cell Research Center, Department of Molecular Biology and Biochemistry, University of California-Irvine, Irvine, California, United States of America.
Green D; Department of Immunology, St. Jude Children's Research Hospital, Memphis, Tennessee, United States of America.
Boiko AD; Sue and Bill Gross Stem Cell Research Center, Department of Molecular Biology and Biochemistry, University of California-Irvine, Irvine, California, United States of America.
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Źródło:
PLoS biology [PLoS Biol] 2019 Sep 30; Vol. 17 (9), pp. e3000402. Date of Electronic Publication: 2019 Sep 30 (Print Publication: 2019).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Calreticulin/*metabolism
Neoplasms/*metabolism
Calcium/metabolism ; Cell Line, Tumor ; Down-Regulation ; Humans ; Mitochondrial Transmembrane Permeability-Driven Necrosis ; Neoplasms/mortality ; Tumor Suppressor Protein p53/metabolism
Czasopismo naukowe
    Wyświetlanie 1-13 z 13

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