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Wyszukujesz frazę ""Murata, Yasuhiko"" wg kryterium: Autor


Wyświetlanie 1-6 z 6
Tytuł :
DNA-PKcs is activated under nutrient starvation and activates Akt, MST1, FoxO3a, and NDR1.
Autorzy :
Shiga S; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo- machi, Aoba-ku, Snedai, Miyagi-ken, 980-8575, Japan.
Murata Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo- machi, Aoba-ku, Snedai, Miyagi-ken, 980-8575, Japan.
Hashimoto T; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo- machi, Aoba-ku, Snedai, Miyagi-ken, 980-8575, Japan.
Urushihara Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo- machi, Aoba-ku, Snedai, Miyagi-ken, 980-8575, Japan.
Fujishima Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo- machi, Aoba-ku, Snedai, Miyagi-ken, 980-8575, Japan.
Kudo K; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo- machi, Aoba-ku, Snedai, Miyagi-ken, 980-8575, Japan.
Sonohara Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo- machi, Aoba-ku, Snedai, Miyagi-ken, 980-8575, Japan.
Kurusu M; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo- machi, Aoba-ku, Snedai, Miyagi-ken, 980-8575, Japan.
Takeda K; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo- machi, Aoba-ku, Snedai, Miyagi-ken, 980-8575, Japan; Department of Radiation Oncology, Tohoku University School of Medicine, 1-1 Seiryo- machi, Aoba-ku, Snedai, Miyagi-ken, 980-8575, Japan.
Jingu K; Department of Radiation Oncology, Tohoku University School of Medicine, 1-1 Seiryo- machi, Aoba-ku, Snedai, Miyagi-ken, 980-8575, Japan.
Hosoi Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo- machi, Aoba-ku, Snedai, Miyagi-ken, 980-8575, Japan. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 Jan 15; Vol. 521 (3), pp. 668-673. Date of Electronic Publication: 2019 Nov 01.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Enzyme Activation*
DNA-Activated Protein Kinase/*metabolism
Forkhead Box Protein O3/*metabolism
Glioblastoma/*metabolism
Protein-Serine-Threonine Kinases/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Cell Line, Tumor ; Hepatocyte Growth Factor/metabolism ; Humans ; Nutrients/metabolism ; Phosphorylation ; Proto-Oncogene Proteins/metabolism ; Signal Transduction ; Starvation/metabolism
Czasopismo naukowe
Tytuł :
Severe hypoxia increases expression of ATM and DNA-PKcs and it increases their activities through Src and AMPK signaling pathways.
Autorzy :
Hashimoto T; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8575, Japan.
Murata Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8575, Japan.
Urushihara Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8575, Japan.
Shiga S; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8575, Japan.
Takeda K; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8575, Japan.
Hosoi Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8575, Japan. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2018 Oct 20; Vol. 505 (1), pp. 13-19. Date of Electronic Publication: 2018 Sep 15.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Signal Transduction*
AMP-Activated Protein Kinases/*metabolism
Ataxia Telangiectasia Mutated Proteins/*metabolism
DNA-Activated Protein Kinase/*metabolism
Nuclear Proteins/*metabolism
src-Family Kinases/*metabolism
AMP-Activated Protein Kinases/genetics ; Blotting, Western ; Cell Hypoxia ; Cell Line, Transformed ; Cells, Cultured ; Fibroblasts/cytology ; Fibroblasts/metabolism ; Humans ; RNA Interference
Czasopismo naukowe
Tytuł :
Knockdown of AMPKα decreases ATM expression and increases radiosensitivity under hypoxia and nutrient starvation in an SV40-transformed human fibroblast cell line, LM217.
Autorzy :
Murata Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi-ken 980-8575, Japan.
Hashimoto T; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi-ken 980-8575, Japan.
Urushihara Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi-ken 980-8575, Japan.
Shiga S; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi-ken 980-8575, Japan.
Takeda K; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi-ken 980-8575, Japan; Department of Radiation Oncology, Tohoku University School of Medicine, 1-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi-ken 980-8575, Japan.
Jingu K; Department of Radiation Oncology, Tohoku University School of Medicine, 1-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi-ken 980-8575, Japan.
Hosoi Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi-ken 980-8575, Japan. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2018 Jan 22; Vol. 495 (4), pp. 2566-2572. Date of Electronic Publication: 2017 Dec 25.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Radiation Tolerance*
AMP-Activated Protein Kinases/*genetics
Ataxia Telangiectasia Mutated Proteins/*genetics
Cell Hypoxia/*radiation effects
Culture Media/*metabolism
Down-Regulation/*genetics
Neoplasms, Experimental/*radiotherapy
AMP-Activated Protein Kinases/metabolism ; Apoptosis/radiation effects ; Ataxia Telangiectasia Mutated Proteins/metabolism ; Cell Line ; Dose-Response Relationship, Radiation ; Down-Regulation/drug effects ; Gene Knockdown Techniques ; Genetic Vectors/genetics ; Hep G2 Cells ; Humans ; Neoplasms, Experimental/metabolism ; Neoplasms, Experimental/pathology ; Radiation Dosage ; Simian virus 40/genetics ; Transfection
Czasopismo naukowe
Tytuł :
NSAIDs diclofenac, indomethacin, and meloxicam highly upregulate expression of ICAM-1 and COX-2 induced by X-irradiation in human endothelial cells.
Autorzy :
Uehara Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi-ken 980-8575, Japan.
Murata Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi-ken 980-8575, Japan.
Shiga S; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi-ken 980-8575, Japan.
Hosoi Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi-ken 980-8575, Japan. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2016 Oct 28; Vol. 479 (4), pp. 847-852. Date of Electronic Publication: 2016 Sep 28.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents, Non-Steroidal/*pharmacology
Cyclooxygenase 2/*metabolism
Human Umbilical Vein Endothelial Cells/*drug effects
Human Umbilical Vein Endothelial Cells/*metabolism
Intercellular Adhesion Molecule-1/*metabolism
Anti-Inflammatory Agents, Non-Steroidal/adverse effects ; Contraindications ; Diclofenac/adverse effects ; Diclofenac/pharmacology ; E-Selectin/metabolism ; Heart/drug effects ; Heart/radiation effects ; Human Umbilical Vein Endothelial Cells/radiation effects ; Humans ; Indomethacin/adverse effects ; Indomethacin/pharmacology ; Meloxicam ; Myocardial Infarction/etiology ; Myocardium/metabolism ; NF-kappa B/antagonists & inhibitors ; NF-kappa B/metabolism ; Nitriles/pharmacology ; Risk Factors ; Sulfones/pharmacology ; Thiazines/adverse effects ; Thiazines/pharmacology ; Thiazoles/adverse effects ; Thiazoles/pharmacology ; Up-Regulation/drug effects ; Up-Regulation/radiation effects ; Vascular Cell Adhesion Molecule-1/metabolism
Czasopismo naukowe
Tytuł :
Activation of mTORC1 under nutrient starvation conditions increases cellular radiosensitivity in human liver cancer cell lines, HepG2 and HuH6.
Autorzy :
Murata Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Snedai, Miyagi-ken 980-8575, Japan.
Uehara Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Snedai, Miyagi-ken 980-8575, Japan.
Hosoi Y; Department of Radiation Biology, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Snedai, Miyagi-ken 980-8575, Japan. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2015 Dec 25; Vol. 468 (4), pp. 684-90. Date of Electronic Publication: 2015 Nov 14.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Radiation Tolerance*
Cell Survival/*radiation effects
Liver Neoplasms/*physiopathology
Liver Neoplasms/*radiotherapy
Multiprotein Complexes/*metabolism
TOR Serine-Threonine Kinases/*metabolism
Culture Media/metabolism ; Dose-Response Relationship, Radiation ; Hep G2 Cells ; Humans ; Mechanistic Target of Rapamycin Complex 1 ; Radiotherapy Dosage ; Up-Regulation
Czasopismo naukowe
Tytuł :
A novel Rieske-type protein derived from an apoptosis-inducing factor-like (AIFL) transcript with a retained intron 4 induces change in mitochondrial morphology and growth arrest.
Autorzy :
Murata Y; Department of Integrated Biosciences, Graduate School of Frontier Sciences, University of Tokyo, 5-1-5 Kashiwanoha, Kashiwa, Chiba 277-8561, Japan. />Furuyama I
Oda S
Mitani H
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2011 Apr 01; Vol. 407 (1), pp. 92-7. Date of Electronic Publication: 2011 Mar 02.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Alternative Splicing*
Apoptosis Inducing Factor/*genetics
Electron Transport Complex III/*genetics
Oryzias/*genetics
Animals ; Apoptosis/genetics ; Cell Cycle ; Cell Proliferation ; Introns/genetics ; Mitochondria/metabolism ; Mitochondria/ultrastructure ; Oryzias/embryology ; Protein Structure, Tertiary ; Transcription, Genetic
Czasopismo naukowe
    Wyświetlanie 1-6 z 6

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