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Wyszukujesz frazę ""Myocytes, Cardiac"" wg kryterium: Temat


Tytuł:
Anserine, a Histidine-Containing Dipeptide, Suppresses Pressure Overload-Induced Systolic Dysfunction by Inhibiting Histone Acetyltransferase Activity of p300 in Mice.
Autorzy:
Sunagawa Y; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.; Division of Translational Research, National Hospital Organization Kyoto Medical Center, Kyoto 612-8555, Japan.; Shizuoka General Hospital, Shizuoka 420-8527, Japan.
Tsukabe R; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.
Irokawa Y; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.
Funamoto M; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.; Division of Translational Research, National Hospital Organization Kyoto Medical Center, Kyoto 612-8555, Japan.; Department of Pharmacology, Institute of Biomedical Sciences, Tokushima University Graduate School, Tokushima 770-8503, Japan.
Suzuki Y; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.
Yamada M; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.
Shimizu S; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.; Division of Translational Research, National Hospital Organization Kyoto Medical Center, Kyoto 612-8555, Japan.
Katanasaka Y; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.; Division of Translational Research, National Hospital Organization Kyoto Medical Center, Kyoto 612-8555, Japan.; Shizuoka General Hospital, Shizuoka 420-8527, Japan.
Hamabe-Horiike T; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.; Division of Translational Research, National Hospital Organization Kyoto Medical Center, Kyoto 612-8555, Japan.; Shizuoka General Hospital, Shizuoka 420-8527, Japan.
Kawase Y; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.
Naruta R; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.
Shimizu K; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.; Division of Translational Research, National Hospital Organization Kyoto Medical Center, Kyoto 612-8555, Japan.
Mori K; Shizuoka General Hospital, Shizuoka 420-8527, Japan.; Graduate School of Public Health, Shizuoka Graduate University of Public Health, Shizuoka 420-0881, Japan.; Department of Molecular and Clinical Pharmacology, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.
Hosomi R; Laboratory of Food and Nutritional Sciences, Faculty of Chemistry, Materials and Bioengineering, Kansai University, Osaka 564-8680, Japan.
Komiyama M; Division of Translational Research, National Hospital Organization Kyoto Medical Center, Kyoto 612-8555, Japan.
Hasegawa K; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.; Division of Translational Research, National Hospital Organization Kyoto Medical Center, Kyoto 612-8555, Japan.
Morimoto T; Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka, Shizuoka 422-8526, Japan.; Division of Translational Research, National Hospital Organization Kyoto Medical Center, Kyoto 612-8555, Japan.; Shizuoka General Hospital, Shizuoka 420-8527, Japan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2024 Feb 16; Vol. 25 (4). Date of Electronic Publication: 2024 Feb 16.
Typ publikacji:
Journal Article
MeSH Terms:
Anserine*/pharmacology
Cardiomyopathies*/metabolism
Heart Failure*/metabolism
Myocytes, Cardiac*/drug effects
Myocytes, Cardiac*/metabolism
p300-CBP Transcription Factors*/antagonists & inhibitors
Animals ; Humans ; Male ; Mice ; Acetylation ; Cardiomegaly/genetics ; Enzyme Inhibitors/pharmacology ; Histones/metabolism ; Mice, Inbred C57BL ; Phenylephrine/pharmacology
Czasopismo naukowe
Tytuł:
Cinnamaldehyde protects cardiomyocytes from oxygen-glucose deprivation/reoxygenation-induced lipid peroxidation and DNA damage via activating the Nrf2 pathway.
Autorzy:
Li YG; Department of Cardiology, Central Hospital of Jiaozuo Coal Industry (Group) Co., LTD, Jiaozuo, Henan, China.
Li JH; Department of Cardiology, Central Hospital of Jiaozuo Coal Industry (Group) Co., LTD, Jiaozuo, Henan, China.
Wang HQ; Department of Cardiology, Central Hospital of Jiaozuo Coal Industry (Group) Co., LTD, Jiaozuo, Henan, China.
Liao J; Université Paris Dauphine, Paris, France.
Du XY; Department of Cardiology, Central Hospital of Jiaozuo Coal Industry (Group) Co., LTD, Jiaozuo, Henan, China.
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Źródło:
Chemical biology & drug design [Chem Biol Drug Des] 2024 Feb; Vol. 103 (2), pp. e14489.
Typ publikacji:
Journal Article
MeSH Terms:
Acrolein*/analogs & derivatives
Acrolein*/pharmacology
Myocytes, Cardiac*/drug effects
Myocytes, Cardiac*/metabolism
NF-E2-Related Factor 2*/drug effects
NF-E2-Related Factor 2*/metabolism
Oxygen*/metabolism
Animals ; Rats ; Apoptosis ; DNA Damage ; Glucose/pharmacology ; Glutathione Disulfide/genetics ; Glutathione Disulfide/metabolism ; Glutathione Disulfide/pharmacology ; Lipid Peroxidation ; NF-kappa B/metabolism ; Oxidative Stress ; Reactive Oxygen Species/metabolism ; Superoxide Dismutase/metabolism
Czasopismo naukowe
Tytuł:
Give heart cells a beat: An interactive museum exhibit that synchronizes stem cell-derived cardiomyocytes to visitors' heartbeat.
Autorzy:
Perez-Bermejo JA; Gladstone Institutes, San Francisco, CA, USA.
Reisman SJ; Exploratorium, San Francisco, CA, USA.
Ma J; Exploratorium, San Francisco, CA, USA.
Carrison-Stone D; Exploratorium, San Francisco, CA, USA.
Cerrito C; Exploratorium, San Francisco, CA, USA.
Ribeiro AJS; Gladstone Institutes, San Francisco, CA, USA.
Conklin BR; Gladstone Institutes, San Francisco, CA, USA; Innovative Genomics Institute, Berkeley, CA 94704, USA; University of California, San Francisco Departments of Medicine and Ophthalmology, San Francisco, CA 94143, USA. Electronic address: .
Yu K; Exploratorium, San Francisco, CA, USA. Electronic address: .
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Źródło:
Stem cell reports [Stem Cell Reports] 2024 Mar 12; Vol. 19 (3), pp. 426-433. Date of Electronic Publication: 2024 Feb 08.
Typ publikacji:
Journal Article
MeSH Terms:
Myocytes, Cardiac*
Museums*
Humans ; Heart Rate ; Stem Cells
Czasopismo naukowe
Tytuł:
Geometry and length control of 3D engineered heart tissues using direct laser writing.
Autorzy:
Karakan MÇ; Department of Mechanical Engineering, Boston University, Boston, MA 02215, USA. .; Photonics Center, Boston University, Boston, MA 02215, USA.; Department of Biomedical Engineering, Boston University, Boston, MA 02215, USA.; Wyss Institute for Biologically Inspired Engineering, Harvard University, Boston, MA 02115, USA.
Ewoldt JK; Department of Biomedical Engineering, Boston University, Boston, MA 02215, USA.; Wyss Institute for Biologically Inspired Engineering, Harvard University, Boston, MA 02115, USA.
Segarra AJ; Photonics Center, Boston University, Boston, MA 02215, USA.; Department of Biomedical Engineering, Polytechnic University of Puerto Rico, San Juan 00918, Puerto Rico.
Sundaram S; Department of Biomedical Engineering, Boston University, Boston, MA 02215, USA.; Wyss Institute for Biologically Inspired Engineering, Harvard University, Boston, MA 02115, USA.
Wang MC; Department of Biomedical Engineering, Boston University, Boston, MA 02215, USA.; Wyss Institute for Biologically Inspired Engineering, Harvard University, Boston, MA 02115, USA.; Harvard-MIT Program in Health Sciences and Technology, Institute for Medical Engineering and Science, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.
White AE; Department of Mechanical Engineering, Boston University, Boston, MA 02215, USA. .; Photonics Center, Boston University, Boston, MA 02215, USA.; Department of Biomedical Engineering, Boston University, Boston, MA 02215, USA.; Division of Materials Science and Engineering, Boston University, Boston, Massachusetts 02215, USA.; Department of Physics, Boston University, Boston, MA 02215, USA.
Chen CS; Department of Biomedical Engineering, Boston University, Boston, MA 02215, USA.; Wyss Institute for Biologically Inspired Engineering, Harvard University, Boston, MA 02115, USA.
Ekinci KL; Department of Mechanical Engineering, Boston University, Boston, MA 02215, USA. .; Photonics Center, Boston University, Boston, MA 02215, USA.; Division of Materials Science and Engineering, Boston University, Boston, Massachusetts 02215, USA.
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Źródło:
Lab on a chip [Lab Chip] 2024 Mar 12; Vol. 24 (6), pp. 1685-1701. Date of Electronic Publication: 2024 Mar 12.
Typ publikacji:
Journal Article
MeSH Terms:
Myocytes, Cardiac*
Tissue Engineering*/methods
Lasers ; Myocardial Contraction
Czasopismo naukowe
Tytuł:
ATP5me alleviates high glucose-induced myocardial cell injury.
Autorzy:
Hou Q; Obstetrical Department, the First People's Hospital of Yunnan Province, the Affiliated Hospital of Kunming University of Science and Technology, No.157 Jinbi Road, Kunming, Yunnan, 650032, China.
Yan F; Obstetrical Department, the First People's Hospital of Yunnan Province, the Affiliated Hospital of Kunming University of Science and Technology, No.157 Jinbi Road, Kunming, Yunnan, 650032, China.
Li X; Obstetrical Department, the First People's Hospital of Yunnan Province, the Affiliated Hospital of Kunming University of Science and Technology, No.157 Jinbi Road, Kunming, Yunnan, 650032, China.
Liu H; Obstetrical Department, the First People's Hospital of Yunnan Province, the Affiliated Hospital of Kunming University of Science and Technology, No.157 Jinbi Road, Kunming, Yunnan, 650032, China.
Yang X; Obstetrical Department, the First People's Hospital of Yunnan Province, the Affiliated Hospital of Kunming University of Science and Technology, No.157 Jinbi Road, Kunming, Yunnan, 650032, China.
Dong X; Obstetrical Department, the First People's Hospital of Yunnan Province, the Affiliated Hospital of Kunming University of Science and Technology, No.157 Jinbi Road, Kunming, Yunnan, 650032, China. Electronic address: .
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Źródło:
International immunopharmacology [Int Immunopharmacol] 2024 Mar 10; Vol. 129, pp. 111626. Date of Electronic Publication: 2024 Feb 05.
Typ publikacji:
Journal Article
MeSH Terms:
Myocytes, Cardiac*/metabolism
Heart Injuries*
Rats ; Animals ; Reactive Oxygen Species/metabolism ; Myocardium/metabolism ; Oxidative Stress ; Glucose/metabolism ; Apoptosis ; Superoxide Dismutase/metabolism
Czasopismo naukowe
Tytuł:
N-Acetyltransferase 10 represses Uqcr11 and Uqcrb independently of ac4C modification to promote heart regeneration.
Autorzy:
Ma W; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China.; Institute of Clinical Pharmacy, NHC Key Laboratory of Cell Transplantation, the Heilongjiang Key Laboratory of Drug Research, Harbin Medical University, Harbin, China.
Tian Y; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China.
Shi L; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China.
Liang J; Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Tianjin, China.
Ouyang Q; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China.
Li J; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China.
Chen H; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China.
Sun H; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China.
Ji H; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China.
Liu X; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.; Department of Laboratory Medicine at The Fourth Affiliated Hospital, Harbin Medical University, Harbin, China.
Huang W; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China.
Gao X; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China.
Jin X; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China.
Wang X; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.
Liu Y; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.
Yu Y; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.
Guo X; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China.
Tian Y; Department of Pathophysiology and the Key Laboratory of Cardiovascular Pathophysiology, Harbin Medical University, Harbin, China.
Yang F; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China.
Li F; Department of Laboratory Medicine and Pathology, University of Minnesota, Minneapolis, MN, USA.
Wang N; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China.
Cai B; Department of Pharmacy at the Second Affiliated Hospital, Harbin Medical University, Harbin, China. .; Department of Pharmacology at College of Pharmacy (National Key Laboratory of Frigid Zone Cardiovascular Diseases, Key Laboratory of Cardiovascular Research, Ministry of Education), Harbin Medical University, Harbin, China. .; Institute of Clinical Pharmacy, NHC Key Laboratory of Cell Transplantation, the Heilongjiang Key Laboratory of Drug Research, Harbin Medical University, Harbin, China. .
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Źródło:
Nature communications [Nat Commun] 2024 Mar 08; Vol. 15 (1), pp. 2137. Date of Electronic Publication: 2024 Mar 08.
Typ publikacji:
Journal Article
MeSH Terms:
Myocytes, Cardiac*/metabolism
Protein Processing, Post-Translational*
Animals ; Female ; Humans ; Male ; Mice ; Acetyltransferases/metabolism ; N-Terminal Acetyltransferases/metabolism ; RNA, Messenger/metabolism ; Swine
Czasopismo naukowe
Tytuł:
Strengthening cardiac therapy pipelines using human pluripotent stem cell-derived cardiomyocytes.
Autorzy:
Raniga K; The Biodiscovery Institute, University of Nottingham, Nottingham NG7 2RD, UK; Pathology, Non-Clinical Safety, GlaxoSmithKline R&D, Stevenage SG1 2NY, UK. Electronic address: .
Nasir A; The Biodiscovery Institute, University of Nottingham, Nottingham NG7 2RD, UK.
Vo NTN; The Biodiscovery Institute, University of Nottingham, Nottingham NG7 2RD, UK.
Vaidyanathan R; FUJIFILM Cellular Dynamics, Inc., Madison, WI, USA.
Dickerson S; FUJIFILM Cellular Dynamics, Inc., Madison, WI, USA.
Hilcove S; FUJIFILM Cellular Dynamics, Inc., Madison, WI, USA.
Mosqueira D; The Biodiscovery Institute, University of Nottingham, Nottingham NG7 2RD, UK.
Mirams GR; Centre for Mathematical Medicine & Biology, School of Mathematical Sciences, University of Nottingham, Nottingham NG7 2RD, UK.
Clements P; Pathology, Non-Clinical Safety, GlaxoSmithKline R&D, Stevenage SG1 2NY, UK.
Hicks R; BioPharmaceuticals R&D Cell Therapy Department, Research and Early Development, Cardiovascular, Renal, and Metabolism (CVRM), BioPharmaceuticals R&D, AstraZeneca, Gothenburg, Sweden; School of Cardiovascular and Metabolic Medicine & Sciences, King's College London, London WC2R 2LS, UK.
Pointon A; Safety Sciences, Clinical Pharmacology and Safety Sciences, BioPharmaceuticals R&D, AstraZeneca, Cambridge CB2 0AA, UK.
Stebbeds W; LifeArc, Stevenage SG1 2FX, UK.
Francis J; Mechanstic Biology and Profiling, Discovery Sciences, BioPharmaceuticals R&D, AstraZeneca, Cambridge CB2 0AA, UK.
Denning C; The Biodiscovery Institute, University of Nottingham, Nottingham NG7 2RD, UK. Electronic address: .
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Źródło:
Cell stem cell [Cell Stem Cell] 2024 Mar 07; Vol. 31 (3), pp. 292-311. Date of Electronic Publication: 2024 Feb 15.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Myocytes, Cardiac*
Induced Pluripotent Stem Cells*
Humans ; Cell Differentiation
Czasopismo naukowe
Tytuł:
Canonical Wnt signaling directs the generation of functional human PSC-derived atrioventricular canal cardiomyocytes in bioprinted cardiac tissues.
Autorzy:
Ye C; Institute of Cardiovascular Diseases, Xiamen Cardiovascular Hospital, School of Medicine, Xiamen University, Xiamen 361006, Fujian, China.
Yang C; State Key Laboratory of Molecular Vaccinology and Molecular Diagnostics, National Institute of Diagnostics and Vaccine Development in Infectious Diseases, School of Life Sciences, School of Public Health, Xiamen University, Xiamen 361102, Fujian, China.
Zhang H; Institute of Cardiovascular Diseases, Xiamen Cardiovascular Hospital, School of Medicine, Xiamen University, Xiamen 361006, Fujian, China.
Gao R; Institute of Cardiovascular Diseases, Xiamen Cardiovascular Hospital, School of Medicine, Xiamen University, Xiamen 361006, Fujian, China.
Liao Y; Institute of Cardiovascular Diseases, Xiamen Cardiovascular Hospital, School of Medicine, Xiamen University, Xiamen 361006, Fujian, China.
Zhang Y; State Key Laboratory of Molecular Vaccinology and Molecular Diagnostics, National Institute of Diagnostics and Vaccine Development in Infectious Diseases, School of Life Sciences, School of Public Health, Xiamen University, Xiamen 361102, Fujian, China.
Jie L; Institute of Cardiovascular Diseases, Xiamen Cardiovascular Hospital, School of Medicine, Xiamen University, Xiamen 361006, Fujian, China.
Zhang Y; Institute of Cardiovascular Diseases, Xiamen Cardiovascular Hospital, School of Medicine, Xiamen University, Xiamen 361006, Fujian, China.
Cheng T; State Key Laboratory of Molecular Vaccinology and Molecular Diagnostics, National Institute of Diagnostics and Vaccine Development in Infectious Diseases, School of Life Sciences, School of Public Health, Xiamen University, Xiamen 361102, Fujian, China.
Wang Y; Institute of Cardiovascular Diseases, Xiamen Cardiovascular Hospital, School of Medicine, Xiamen University, Xiamen 361006, Fujian, China. Electronic address: .
Ren J; Institute of Cardiovascular Diseases, Xiamen Cardiovascular Hospital, School of Medicine, Xiamen University, Xiamen 361006, Fujian, China. Electronic address: .
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Źródło:
Cell stem cell [Cell Stem Cell] 2024 Mar 07; Vol. 31 (3), pp. 398-409.e5. Date of Electronic Publication: 2024 Feb 15.
Typ publikacji:
Journal Article
MeSH Terms:
Myocytes, Cardiac*/metabolism
Wnt Signaling Pathway*
Heart Septal Defects*
Humans ; Endocardial Cushions ; Heart Ventricles ; Cell Differentiation
SCR Disease Name:
Complete atrioventricular septal defect
Czasopismo naukowe
Tytuł:
Nanoplastics causes heart aging/myocardial cell senescence through the Ca /mtDNA/cGAS-STING signaling cascade.
Autorzy:
Wang K; Department of Cardiology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou, China.
Du Y; Department of Cardiology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou, China.
Li P; Department of Cardiology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou, China.
Guan C; Department of Cardiology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou, China.
Zhou M; Department of Cardiology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou, China.
Wu L; Department of Cardiology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou, China.
Liu Z; Department of Cardiology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou, China.
Huang Z; Department of Cardiology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou, China. .
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Źródło:
Journal of nanobiotechnology [J Nanobiotechnology] 2024 Mar 06; Vol. 22 (1), pp. 96. Date of Electronic Publication: 2024 Mar 06.
Typ publikacji:
Journal Article
MeSH Terms:
Myocytes, Cardiac*
DNA, Mitochondrial*
Humans ; Microplastics ; Cellular Senescence ; Mitochondria ; Signal Transduction
Czasopismo naukowe
Tytuł:
GRAF1 Acts as a Downstream Mediator of Parkin to Regulate Mitophagy in Cardiomyocytes.
Autorzy:
Zhu Q; Department of Pathology and Laboratory Medicine, University of North Carolina, Chapel Hill, NC 27599, USA.
Combs ME; Department of Pathology and Laboratory Medicine, University of North Carolina, Chapel Hill, NC 27599, USA.
Bowles DE; Division of Surgical Sciences, Duke University Medical Center, Durham, NC 27710, USA.
Gross RT; Division of Surgical Sciences, Duke University Medical Center, Durham, NC 27710, USA.
Mendiola Pla M; Division of Surgical Sciences, Duke University Medical Center, Durham, NC 27710, USA.
Mack CP; Department of Pathology and Laboratory Medicine, University of North Carolina, Chapel Hill, NC 27599, USA.; McAllister Heart Institute, University of North Carolina, Chapel Hill, NC 27599, USA.
Taylor JM; Department of Pathology and Laboratory Medicine, University of North Carolina, Chapel Hill, NC 27599, USA.; McAllister Heart Institute, University of North Carolina, Chapel Hill, NC 27599, USA.
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Źródło:
Cells [Cells] 2024 Mar 04; Vol. 13 (5). Date of Electronic Publication: 2024 Mar 04.
Typ publikacji:
Journal Article
MeSH Terms:
GTPase-Activating Proteins*/metabolism
Mitophagy*/physiology
Myocytes, Cardiac*/metabolism
Phosphatidylinositol Phosphates*
Ubiquitin-Protein Ligases*/metabolism
Animals ; Humans ; Mice ; Actins ; Protein Kinases/metabolism
Czasopismo naukowe
Tytuł:
Defective Biomechanics and Pharmacological Rescue of Human Cardiomyocytes with Filamin C Truncations.
Autorzy:
Lazzarino M; CNR-IOM, Area Science Park, 34149 Trieste, Italy.; Cardiovascular Institute, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
Zanetti M; CNR-IOM, Area Science Park, 34149 Trieste, Italy.; Cardiovascular Institute, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
Chen SN; Cardiovascular Institute, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
Gao S; Cardiovascular Institute, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
Peña B; Cardiovascular Institute, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.; Bioengineering Department, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
Lam CK; Stanford Cardiovascular Institute, Stanford University, Stanford, CA 94305, USA.
Wu JC; Stanford Cardiovascular Institute, Stanford University, Stanford, CA 94305, USA.
Taylor MRG; Cardiovascular Institute, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
Mestroni L; Cardiovascular Institute, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
Sbaizero O; Cardiovascular Institute, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.; Engineering and Architecture Department, University of Trieste, 34127 Trieste, Italy.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2024 Mar 03; Vol. 25 (5). Date of Electronic Publication: 2024 Mar 03.
Typ publikacji:
Journal Article
MeSH Terms:
Myocytes, Cardiac*/metabolism
Induced Pluripotent Stem Cells*
Humans ; Biomechanical Phenomena ; Filamins/metabolism ; Actins/metabolism ; Myocardium
Czasopismo naukowe
Tytuł:
mTORC1 hyperactivation and resultant suppression of macroautophagy contribute to the induction of cardiomyocyte necroptosis by catecholamine surges.
Autorzy:
Cai M; Heart Center, Shanghai Sixth People's Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Division of Basic Biomedical Sciences, Sanford School of Medicine of the University of South Dakota, Vermillion, South Dakota, USA.
Wu P; Division of Basic Biomedical Sciences, Sanford School of Medicine of the University of South Dakota, Vermillion, South Dakota, USA.; Department of Cardiology, Xiamen Cardiovascular Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen, China.
Ni W; Department of Cardiology, Xiamen Cardiovascular Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen, China.
Huang D; Heart Center, Shanghai Sixth People's Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Wang X; Division of Basic Biomedical Sciences, Sanford School of Medicine of the University of South Dakota, Vermillion, South Dakota, USA.
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Źródło:
Physiological reports [Physiol Rep] 2024 Mar; Vol. 12 (5), pp. e15966.
Typ publikacji:
Journal Article
MeSH Terms:
Catecholamines*
Myocytes, Cardiac*
Animals ; Mice ; Rats ; Mice, Inbred C57BL ; Macroautophagy ; Necroptosis ; Isoproterenol/toxicity ; Mechanistic Target of Rapamycin Complex 1 ; Necrosis
Czasopismo naukowe
Tytuł:
Do calcium channel blockers applied to cardiomyocytes cause increased channel expression resulting in reduced efficacy?
Autorzy:
Jæger KH; Simula Research Laboratory, Oslo, Norway. .
Charwat V; Organos Inc., Berkeley, CA, USA.
Wall S; Simula Research Laboratory, Oslo, Norway.; Organos Inc., Berkeley, CA, USA.
Healy KE; Department of Bioengineering, University of California, Berkeley, CA, USA.; Department of Material Science and Engineering, University of California, Berkeley, CA, USA.
Tveito A; Simula Research Laboratory, Oslo, Norway.
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Źródło:
NPJ systems biology and applications [NPJ Syst Biol Appl] 2024 Mar 01; Vol. 10 (1), pp. 22. Date of Electronic Publication: 2024 Mar 01.
Typ publikacji:
Journal Article
MeSH Terms:
Myocytes, Cardiac*
Induced Pluripotent Stem Cells*
Humans ; Calcium Channel Blockers/pharmacology ; Calcium ; Calcium Channels
Czasopismo naukowe
Tytuł:
Evaluation of the cardiotoxicity potential of bisphenol analogues in human induced pluripotent stem cells derived cardiomyocytes.
Autorzy:
Lee SG; Department of Stem Cell Biology, School of Medicine, Konkuk University, 120 Neungdong-Ro, Gwangjin-Gu, Seoul 05029, Republic of Korea; College of Veterinary Medicine, Konkuk University, Seoul 05029, Republic of Korea.
Song GE; College of Veterinary Medicine, Konkuk University, Seoul 05029, Republic of Korea.
Seok J; College of Veterinary Medicine, Konkuk University, Seoul 05029, Republic of Korea.
Kim J; College of Veterinary Medicine, Konkuk University, Seoul 05029, Republic of Korea.
Kim MW; College of Veterinary Medicine, Konkuk University, Seoul 05029, Republic of Korea.
Rhee J; College of Veterinary Medicine, Konkuk University, Seoul 05029, Republic of Korea.
Park S; College of Veterinary Medicine, Konkuk University, Seoul 05029, Republic of Korea.
Jeong KS; College of Veterinary Medicine, Konkuk University, Seoul 05029, Republic of Korea.
Lee S; College of Veterinary Medicine, Konkuk University, Seoul 05029, Republic of Korea.
Lee YH; College of Veterinary Medicine, Konkuk University, Seoul 05029, Republic of Korea.
Jeong Y; College of Veterinary Medicine, Konkuk University, Seoul 05029, Republic of Korea.
Chung HM; Department of Stem Cell Biology, School of Medicine, Konkuk University, 120 Neungdong-Ro, Gwangjin-Gu, Seoul 05029, Republic of Korea; Miraecell Bio Co. Ltd., Seoul 04795, Republic of Korea.
Kim CY; College of Veterinary Medicine, Konkuk University, Seoul 05029, Republic of Korea. Electronic address: .
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Źródło:
Ecotoxicology and environmental safety [Ecotoxicol Environ Saf] 2024 Mar 01; Vol. 272, pp. 116108. Date of Electronic Publication: 2024 Feb 15.
Typ publikacji:
Journal Article
MeSH Terms:
Myocytes, Cardiac*
Induced Pluripotent Stem Cells*/physiology
Benzhydryl Compounds*
Animals ; Humans ; Cardiotoxicity/etiology ; Phenols/toxicity
Czasopismo naukowe
Tytuł:
Optogenetic confirmation of transverse-tubular membrane excitability in intact cardiac myocytes.
Autorzy:
Scardigli M; Department of Experimental and Clinical Medicine, University of Florence, Florence, Italy.
Pásek M; Institute of Thermomechanics, Czech Academy of Science, Prague, Czech Republic.; Department of Physiology, Faculty of Medicine, Masaryk University, Brno, Czech Republic.
Santini L; Department of Neurology, Psychology, Drug Sciences and Child Health, University of Florence, Florence, Italy.
Palandri C; Department of Neurology, Psychology, Drug Sciences and Child Health, University of Florence, Florence, Italy.
Conti E; European Laboratory for Non-Linear Spectroscopy - LENS, Sesto Fiorentino, Italy.; Neuroscience Institute, National Research Council, Pisa, Italy.
Crocini C; DZHK (German Centre for Cardiovascular Research), Partner Site Berlin, Berlin, Germany.; Max Rubner Center for Cardiovascular Metabolic Renal Research (MRC), Deutsches Herzzentrum der Charité (DHZC), Charité-Universitätsmedizin Berlin, Berlin, Germany.
Campione M; Institute of Neuroscience (IN-CNR) and Department of Biomedical Science, University of Padua, Padua, Italy.
Loew LM; Center for Cell Analysis and Modeling, University of Connecticut, Farmington, CT, USA.
de Vries AAF; Laboratory of Experimental Cardiology, Department of Cardiology, Leiden University Medical Center, Leiden, The Netherlands.
Pijnappels DA; Laboratory of Experimental Cardiology, Department of Cardiology, Leiden University Medical Center, Leiden, The Netherlands.
Pavone FS; European Laboratory for Non-Linear Spectroscopy - LENS, Sesto Fiorentino, Italy.
Poggesi C; Department of Experimental and Clinical Medicine, University of Florence, Florence, Italy.
Cerbai E; Department of Neurology, Psychology, Drug Sciences and Child Health, University of Florence, Florence, Italy.; European Laboratory for Non-Linear Spectroscopy - LENS, Sesto Fiorentino, Italy.
Coppini R; Department of Neurology, Psychology, Drug Sciences and Child Health, University of Florence, Florence, Italy.
Kohl P; Institute for Experimental Cardiovascular Medicine, University Heart Center and Medical Faculty, University of Freiburg, Freiburg, Germany.
Ferrantini C; Department of Experimental and Clinical Medicine, University of Florence, Florence, Italy.
Sacconi L; European Laboratory for Non-Linear Spectroscopy - LENS, Sesto Fiorentino, Italy.; Institute for Experimental Cardiovascular Medicine, University Heart Center and Medical Faculty, University of Freiburg, Freiburg, Germany.; Institute of Clinical Physiology, National Research Council (IFC-CNR), Florence, Italy.
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Źródło:
The Journal of physiology [J Physiol] 2024 Mar; Vol. 602 (5), pp. 791-808. Date of Electronic Publication: 2024 Feb 13.
Typ publikacji:
Journal Article
MeSH Terms:
Myocytes, Cardiac*/metabolism
Optogenetics*
Sarcolemma/metabolism ; Cell Membrane ; Membrane Potentials ; Action Potentials/physiology
Czasopismo naukowe
Tytuł:
Lin28a cardiomyocyte-specific modified mRNA translation system induces cardiomyocyte cell division and cardiac repair.
Autorzy:
Magadum A; Cardiovascular Research Institute, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Department of Genetics and Genomic Sciences, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Black Family Stem Cell Institute, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Center for Translational Medicine, Temple University School of Medicine, Philadelphia, PA 19140, USA. Electronic address: .
Sun J; Department of Biomedical Engineering, School of Medicine, School of Engineering, University of Alabama at Birmingham, Birmingham, AL 35294-4440, USA.
Singh N; Cardiovascular Research Institute, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Department of Genetics and Genomic Sciences, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Black Family Stem Cell Institute, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Department of Sports Biosciences, Central University of Rajasthan, Ajmer, India.
Kurian AA; Cardiovascular Research Institute, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Department of Genetics and Genomic Sciences, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Black Family Stem Cell Institute, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA.
Chepurko E; Cardiovascular Research Institute, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Department of Genetics and Genomic Sciences, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Black Family Stem Cell Institute, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA.
Fargnoli A; Cardiovascular Research Institute, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Department of Genetics and Genomic Sciences, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Black Family Stem Cell Institute, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA.
Hajjar R; Gene & Cell Therapy Institute, Massachusetts General Brigham, Boston, MA 02199, USA.
Zhang J; Department of Biomedical Engineering, School of Medicine, School of Engineering, University of Alabama at Birmingham, Birmingham, AL 35294-4440, USA; Department of Medicine, Division of Cardiovascular Disease, School of Medicine, University of Alabama at Birmingham, Birmingham, AL 35294-4440, USA. Electronic address: .
Zangi L; Cardiovascular Research Institute, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Department of Genetics and Genomic Sciences, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA; Black Family Stem Cell Institute, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA. Electronic address: .
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Źródło:
Journal of molecular and cellular cardiology [J Mol Cell Cardiol] 2024 Mar; Vol. 188, pp. 61-64. Date of Electronic Publication: 2024 Jan 30.
Typ publikacji:
Journal Article
MeSH Terms:
Myocytes, Cardiac*
Cardiac Surgical Procedures*
Animals ; Humans ; Adult ; COVID-19 Vaccines ; Cell Division ; Protein Biosynthesis ; Mammals
Czasopismo naukowe
Tytuł:
Cardiomyocyte maturation alters molecular stress response capacities and determines cell survival upon mitochondrial dysfunction.
Autorzy:
Schraps N; Department of Pediatric Cardiology, Justus Liebig University, Gießen, Germany.
Tirre M; Department of Pediatric Cardiology, University Hospital Münster, Münster, Germany.
Pyschny S; Department of Pediatric Cardiology, University Hospital Münster, Münster, Germany.
Reis A; Institute of Physiology, Justus Liebig University, Gießen, Germany.
Schlierbach H; Institute of Neuropathology, Justus Liebig University, Gießen, Germany.
Seidl M; Institute of Pharmacology and Toxicology, Westfälische Wilhelms University, Münster, Germany.
Kehl HG; Department of Pediatric Cardiology, University Hospital Münster, Münster, Germany.
Schänzer A; Institute of Neuropathology, Justus Liebig University, Gießen, Germany.
Heger J; Institute of Physiology, Justus Liebig University, Gießen, Germany.
Jux C; Department of Pediatric Cardiology, Justus Liebig University, Gießen, Germany; Department of Pediatric Cardiology, University Hospital Münster, Münster, Germany.
Drenckhahn JD; Department of Pediatric Cardiology, Justus Liebig University, Gießen, Germany; Department of Pediatric Cardiology, University Hospital Münster, Münster, Germany. Electronic address: .
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Źródło:
Free radical biology & medicine [Free Radic Biol Med] 2024 Mar; Vol. 213, pp. 248-265. Date of Electronic Publication: 2024 Jan 22.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Myocytes, Cardiac*/metabolism
Mitochondrial Diseases*/metabolism
Adult ; Mice ; Humans ; Animals ; Reactive Oxygen Species/metabolism ; Cell Survival ; Electron Transport Complex III/metabolism ; Antioxidants/metabolism ; Adenosine Triphosphate/metabolism ; Mammals/metabolism
Czasopismo naukowe
Tytuł:
GDF11 mitigates high glucose-induced cardiomyocytes apoptosis by inhibiting the ALKBH5-FOXO3-CDR1as/Hippo signaling pathway.
Autorzy:
Shao Y; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China; Cancer Institute, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao 266071, China.
Li M; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China; Key Laboratory of Basic and Application Research of Beiyao (Heilongjiang University of Chinese Medicine), Ministry of Education, Heilongjiang University of Chinese Medicine, 24 Heping Road, Xiangfang District, Harbin 150040, China.
Wang Y; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Qu Y; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Gong M; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China; College of Bioinformatics Science and Technology, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Yu Q; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China; Heart, Lung, and Blood Vascular Medicine Institute, Division of Cardiology, Department of Medicine, University of Pittsburgh School of Medicine, Pittsburgh, PA 15261, USA.
Yang X; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Liu D; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Li H; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Wang Y; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Sun H; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Zhang Y; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Zhang X; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Liu T; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Liu J; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Gong T; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Sun Y; The third ward of obstetrics and Gynecology, the Second Hospital of Harbin Medical University, Xuefu Road, Nangang District, Harbin, Heilongjiang 150081, China.
Du Z; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China.
Jiao L; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China. Electronic address: .
Zhang Y; Department of Pharmacology, College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, China. Electronic address: .
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Źródło:
Biochimica et biophysica acta. Molecular cell research [Biochim Biophys Acta Mol Cell Res] 2024 Mar; Vol. 1871 (3), pp. 119656. Date of Electronic Publication: 2024 Jan 03.
Typ publikacji:
Journal Article
MeSH Terms:
Myocytes, Cardiac*/metabolism
Diabetic Cardiomyopathies*/chemically induced
Diabetic Cardiomyopathies*/metabolism
Mice ; Animals ; Hippo Signaling Pathway ; Mice, Inbred C57BL ; Growth Differentiation Factors/genetics ; Growth Differentiation Factors/metabolism ; Growth Differentiation Factors/pharmacology ; Glucose/pharmacology ; Glucose/metabolism ; Apoptosis/genetics
Czasopismo naukowe
Tytuł:
STING activation in cardiomyocytes drives hypertrophy-associated heart failure via NF-κB-mediated inflammatory response.
Autorzy:
Wang L; Department of Pharmacology, School of Pharmacy, China Pharmaceutical University, Nanjing, Jiangsu 210009, China.
Zhang S; Department of Pharmacology, School of Pharmacy, China Pharmaceutical University, Nanjing, Jiangsu 210009, China.
Liu H; Department of Pharmacology, School of Pharmacy, China Pharmaceutical University, Nanjing, Jiangsu 210009, China.
Gao L; Department of Pharmacology, School of Pharmacy, China Pharmaceutical University, Nanjing, Jiangsu 210009, China.
He L; Department of Pharmacology, School of Pharmacy, China Pharmaceutical University, Nanjing, Jiangsu 210009, China.
Chen Y; Department of General Surgery, The Second People's Hospital of Hefei, Hefei Hospital Affiliated to Anhui Medical University, Hefei 230011, China.
Zhang J; Department of Pathophysiology, The Fourth Affiliated Hospital of Anhui Medical University, Hefei, China.
Yang M; Department of Pathophysiology, The Fourth Affiliated Hospital of Anhui Medical University, Hefei, China.
He C; Department of Pharmacology, School of Pharmacy, China Pharmaceutical University, Nanjing, Jiangsu 210009, China; State Key Laboratory of Natural Medicines, China Pharmaceutical University, Nanjing 211198, China. Electronic address: .
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Źródło:
Biochimica et biophysica acta. Molecular basis of disease [Biochim Biophys Acta Mol Basis Dis] 2024 Mar; Vol. 1870 (3), pp. 166997. Date of Electronic Publication: 2023 Dec 22.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Heart Failure*/genetics
Heart Failure*/metabolism
Myocytes, Cardiac*/metabolism
Animals ; Humans ; Mice ; Angiotensin II/metabolism ; Cardiomegaly/genetics ; Cardiomegaly/metabolism ; Myocardium/metabolism ; NF-kappa B/metabolism
Czasopismo naukowe
Tytuł:
Maternal androgen excess inhibits fetal cardiomyocytes proliferation through RB-mediated cell cycle arrest and induces cardiac hypertrophy in adulthood.
Autorzy:
Huo Y; National Children's Medical Center, Children's Hospital of Fudan University, Fudan University, Shanghai, 201102, China.; National Health Commission (NHC) Key Laboratory of Neonatal Diseases, Fudan University, 399 Wanyuan Road, Minhang, Shanghai, 201102, China.
Wang W; Guangzhou Center for Disease Control and Prevention, Guangzhou, 510080, China.
Zhang J; National Children's Medical Center, Children's Hospital of Fudan University, Fudan University, Shanghai, 201102, China.; National Health Commission (NHC) Key Laboratory of Neonatal Diseases, Fudan University, 399 Wanyuan Road, Minhang, Shanghai, 201102, China.; Institute of Pediatrics, Children's Hospital of Fudan University, Shanghai, 201102, China.
Xu D; National Children's Medical Center, Children's Hospital of Fudan University, Fudan University, Shanghai, 201102, China.; National Health Commission (NHC) Key Laboratory of Neonatal Diseases, Fudan University, 399 Wanyuan Road, Minhang, Shanghai, 201102, China.
Bai F; National Children's Medical Center, Children's Hospital of Fudan University, Fudan University, Shanghai, 201102, China.; National Health Commission (NHC) Key Laboratory of Neonatal Diseases, Fudan University, 399 Wanyuan Road, Minhang, Shanghai, 201102, China.
Gui Y; National Children's Medical Center, Children's Hospital of Fudan University, Fudan University, Shanghai, 201102, China. yhgui_.; National Health Commission (NHC) Key Laboratory of Neonatal Diseases, Fudan University, 399 Wanyuan Road, Minhang, Shanghai, 201102, China. yhgui_.; Cardiovascular Center, Children's Hospital of Fudan University, Shanghai, 201102, China. yhgui_.
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Źródło:
Journal of endocrinological investigation [J Endocrinol Invest] 2024 Mar; Vol. 47 (3), pp. 603-617. Date of Electronic Publication: 2023 Aug 29.
Typ publikacji:
Journal Article
MeSH Terms:
Androgens*/adverse effects
Myocytes, Cardiac*
Humans ; Adult ; Pregnancy ; Female ; Animals ; Mice ; Androsterone ; Cell Cycle Checkpoints ; Cell Proliferation ; Cell Cycle Proteins ; Dihydrotestosterone ; Cardiomegaly/chemically induced
Czasopismo naukowe

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