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Tytuł:
Noninvasive Imaging OX40 Activated T Cells Provides Early Warning of Rheumatoid Arthritis.
Autorzy:
Wen G; Department of Orthopedics, The Second Affiliated Hospital of Harbin Medical University, Harbin, 150086, China.
Lei H; Department of Rheumatology and Immunology, The Second Affiliated Hospital of Harbin Medical University, Harbin, 150086, China.
Qi B; Department of Orthopedics, The Second Affiliated Hospital of Harbin Medical University, Harbin, 150086, China.
Duan S; Department of Orthopedics, The Second Affiliated Hospital of Harbin Medical University, Harbin, 150086, China.
Xiao Z; Molecular Imaging Research Center of Harbin Medical University, Harbin, 150001, China.
Han C; Department of Orthopedics, The Second Affiliated Hospital of Harbin Medical University, Harbin, 150086, China.
Xia Y; Department of Orthopedics, The Second Affiliated Hospital of Harbin Medical University, Harbin, 150086, China.
Jing C; Department of Orthopedics, The Second Affiliated Hospital of Harbin Medical University, Harbin, 150086, China.
Liu J; Department of Orthopedics, The Second Affiliated Hospital of Harbin Medical University, Harbin, 150086, China. .
Li C; Department of Orthopedics, The Second Affiliated Hospital of Harbin Medical University, Harbin, 150086, China. .
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Źródło:
Molecular imaging and biology [Mol Imaging Biol] 2023 Aug; Vol. 25 (4), pp. 621-629. Date of Electronic Publication: 2023 Apr 03.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Receptors, Tumor Necrosis Factor*/metabolism
Arthritis, Rheumatoid*/diagnostic imaging
Arthritis, Rheumatoid*/metabolism
Mice ; Animals ; Membrane Glycoproteins/metabolism ; Tumor Necrosis Factors/metabolism ; Receptors, OX40/metabolism ; Tumor Necrosis Factor Receptor Superfamily, Member 7/metabolism ; Tissue Distribution ; T-Lymphocytes/metabolism ; Antibodies, Monoclonal/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
Hypomethylation of Tumor necrosis factor-like cytokine 1A(TL1A) and its decoy receptor 3 expressive level increase has diagnostic value in HBV-associated cirrhosis.
Autorzy:
Wei XF; Department of Hepatology, Qilu Hospital of Shandong University, Jinan, 250012, PR China. Electronic address: .
Zhu JY; Department of Hepatology, Qilu Hospital of Shandong University, Jinan, 250012, PR China.
Liu HH; Department of Hepatology, Qilu Hospital of Shandong University, Jinan, 250012, PR China.
Su X; Department of Hepatology, Qilu Hospital of Shandong University, Jinan, 250012, PR China.
Li JH; Department of Hepatology, Qilu Hospital of Shandong University, Jinan, 250012, PR China.
Fan YC; Department of Hepatology, Qilu Hospital of Shandong University, Jinan, 250012, PR China; Institute of Hepatology, Shandong University, Jinan, 250012, PR China.
Wang K; Department of Hepatology, Qilu Hospital of Shandong University, Jinan, 250012, PR China; Institute of Hepatology, Shandong University, Jinan, 250012, PR China; Shenzhen Research Institute of Shandong University, Shenzhen, 518000, PR China. Electronic address: .
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Źródło:
Virology [Virology] 2023 Aug; Vol. 585, pp. 91-99. Date of Electronic Publication: 2023 Jun 08.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Hepatitis B virus*/genetics
Hepatitis B virus*/metabolism
Tumor Necrosis Factor Ligand Superfamily Member 15*/genetics
Tumor Necrosis Factor Ligand Superfamily Member 15*/metabolism
Humans ; Fibrosis ; Liver Cirrhosis/diagnosis ; Liver Cirrhosis/genetics ; Tumor Necrosis Factor-alpha
Czasopismo naukowe
Szczegóły
Tytuł:
Identification of a novel role for TL1A/DR3 deficiency in acute respiratory distress syndrome that exacerbates alveolar epithelial disruption.
Autorzy:
Zhang D; Department of Respiratory, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, 250021, Shandong, China.
Zhang J; Department of Respiratory and Intensive Care Unit, The First Affiliated Hospital of Shandong First Medical University and Shandong Provincial Qianfoshan Hospital, Shandong Institute of Respiratory Diseases, Jinan, China.
Zhang J; Department of Respiratory, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, 250021, Shandong, China.
Ji X; Department of Respiratory and Intensive Care Unit, The First Affiliated Hospital of Shandong First Medical University and Shandong Provincial Qianfoshan Hospital, Shandong Institute of Respiratory Diseases, Jinan, China.
Qi Q; Department of Respiratory and Intensive Care Unit, The First Affiliated Hospital of Shandong First Medical University and Shandong Provincial Qianfoshan Hospital, Shandong Institute of Respiratory Diseases, Jinan, China.
Xu J; Department of Respiratory and Intensive Care Unit, The First Affiliated Hospital of Shandong First Medical University and Shandong Provincial Qianfoshan Hospital, Shandong Institute of Respiratory Diseases, Jinan, China.
Pan Y; Department of Respiratory, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, 250021, Shandong, China.
Liu X; Department of Respiratory and Intensive Care Unit, The First Affiliated Hospital of Shandong First Medical University and Shandong Provincial Qianfoshan Hospital, Shandong Institute of Respiratory Diseases, Jinan, China.
Sun F; Department of Respiratory and Intensive Care Unit, The First Affiliated Hospital of Shandong First Medical University and Shandong Provincial Qianfoshan Hospital, Shandong Institute of Respiratory Diseases, Jinan, China.
Zeng R; Department of Respiratory, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, 250021, Shandong, China.
Dong L; Department of Respiratory, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, 250021, Shandong, China. .; Department of Respiratory and Intensive Care Unit, The First Affiliated Hospital of Shandong First Medical University and Shandong Provincial Qianfoshan Hospital, Shandong Institute of Respiratory Diseases, Jinan, China. .
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Źródło:
Respiratory research [Respir Res] 2023 Jul 11; Vol. 24 (1), pp. 182. Date of Electronic Publication: 2023 Jul 11.
Typ publikacji:
Journal Article
MeSH Terms:
Respiratory Distress Syndrome*/chemically induced
Respiratory Distress Syndrome*/genetics
Receptors, Tumor Necrosis Factor, Member 25*/genetics
Tumor Necrosis Factor Ligand Superfamily Member 15*/genetics
Animals ; Mice ; Epithelium ; Ligands ; Tumor Necrosis Factor-alpha
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Early depletion of M1 macrophages retards the progression of glucocorticoid-associated osteonecrosis of the femoral head.
Autorzy:
Cheng Y; Department of Orthopedics Trauma and Microsurgery, Zhongnan Hospital of Wuhan University, Wuhan 430071, Hubei, China.
Chen H; Department of Orthopedics Trauma and Microsurgery, Zhongnan Hospital of Wuhan University, Wuhan 430071, Hubei, China.
Duan P; Department of Orthopedics Trauma and Microsurgery, Zhongnan Hospital of Wuhan University, Wuhan 430071, Hubei, China.
Zhang H; Department of Orthopedics Trauma and Microsurgery, Zhongnan Hospital of Wuhan University, Wuhan 430071, Hubei, China.
Yu Y; Department of Orthopedics Trauma and Microsurgery, Zhongnan Hospital of Wuhan University, Wuhan 430071, Hubei, China.
Yu J; Department of Orthopedics Trauma and Microsurgery, Zhongnan Hospital of Wuhan University, Wuhan 430071, Hubei, China.
Yu Z; Department of Orthopedics Trauma and Microsurgery, Zhongnan Hospital of Wuhan University, Wuhan 430071, Hubei, China.
Zheng L; Department of Orthopedics Trauma and Microsurgery, Zhongnan Hospital of Wuhan University, Wuhan 430071, Hubei, China.
Ye X; Department of Orthopedics Trauma and Microsurgery, Zhongnan Hospital of Wuhan University, Wuhan 430071, Hubei, China.
Pan Z; Department of Orthopedics Trauma and Microsurgery, Zhongnan Hospital of Wuhan University, Wuhan 430071, Hubei, China. Electronic address: .
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Źródło:
International immunopharmacology [Int Immunopharmacol] 2023 Sep; Vol. 122, pp. 110639. Date of Electronic Publication: 2023 Jul 21.
Typ publikacji:
Journal Article
MeSH Terms:
Glucocorticoids*
Femur Head Necrosis*/chemically induced
Femur Head Necrosis*/metabolism
Humans ; Rats ; Mice ; Animals ; Femur Head ; Tumor Necrosis Factor-alpha ; NF-kappa B ; Culture Media, Conditioned ; Macrophages/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
Deletion of the non-adjacent genes UL148 and UL148D impairs human cytomegalovirus-mediated TNF receptor 2 surface upregulation.
Autorzy:
Le-Trilling VTK; Institute for Virology, University Hospital Essen, University of Duisburg-Essen, Essen, Germany.
Maaßen F; Institute for Virology, University Hospital Essen, University of Duisburg-Essen, Essen, Germany.
Katschinski B; Institute for Virology, University Hospital Essen, University of Duisburg-Essen, Essen, Germany.
Hengel H; Institute of Virology, Medical Center and Faculty of Medicine, University of Freiburg, Freiburg, Germany.
Trilling M; Institute for Virology, University Hospital Essen, University of Duisburg-Essen, Essen, Germany.
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Źródło:
Frontiers in immunology [Front Immunol] 2023 Aug 03; Vol. 14, pp. 1170300. Date of Electronic Publication: 2023 Aug 03 (Print Publication: 2023).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cytomegalovirus*/genetics
Receptors, Tumor Necrosis Factor, Type II*/genetics
Viral Fusion Proteins*
Humans ; Transcriptional Activation ; Tumor Necrosis Factor-alpha/genetics ; Up-Regulation
Czasopismo naukowe
Szczegóły
Tytuł:
Double-negative T cells regulate the progression of liver fibrosis through Th9 cells differentiation.
Autorzy:
Han C; The Second Affiliated Hospital of Jiaxing University, Jiaxing, China.
Pei H; Jilin Agricultural University, Changchun, China.
Sheng Y; The Second Affiliated Hospital of Jiaxing University, Jiaxing, China.
Wang J; The Second Affiliated Hospital of Jiaxing University, Jiaxing, China.
Zhou X; The Second Affiliated Hospital of Jiaxing University, Jiaxing, China.
Li W; The Second Affiliated Hospital of Jiaxing University, Jiaxing, China.
Wu S; The Second Affiliated Hospital of Jiaxing University, Jiaxing, China.
Yang Y; The Second Affiliated Hospital of Jiaxing University, Jiaxing, China.
Sheng J; The Second Affiliated Hospital of Jiaxing University, Jiaxing, China.
Cao C; The Second Affiliated Hospital of Jiaxing University, Jiaxing, China.
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Źródło:
Liver international : official journal of the International Association for the Study of the Liver [Liver Int] 2023 Jul; Vol. 43 (7), pp. 1604-1613. Date of Electronic Publication: 2023 May 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
T-Lymphocytes, Helper-Inducer*
Receptors, Tumor Necrosis Factor, Type II*/metabolism
Mice ; Animals ; STAT5 Transcription Factor/metabolism ; Tumor Necrosis Factor-alpha ; NF-kappa B/metabolism ; Interleukin-9/metabolism ; Cell Differentiation ; Liver Cirrhosis/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
IL-10 in combination with IL-12 and TNF-α attenuates CXCL8/CXCR1 axis in peritoneal macrophages of mice infected with Staphylococcus aureus through the TNFR1-IL-1R-NF-κB pathway.
Autorzy:
Dutta P; Department of Physiology, Immunology Laboratory, University of Calcutta, University Colleges of Science and Technology, 92 APC Road, Calcutta 700009, West Bengal, India.
Bishayi B; Department of Physiology, Immunology Laboratory, University of Calcutta, University Colleges of Science and Technology, 92 APC Road, Calcutta 700009, West Bengal, India. Electronic address: biswa_.
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Źródło:
International immunopharmacology [Int Immunopharmacol] 2023 Jul; Vol. 120, pp. 110297. Date of Electronic Publication: 2023 May 17.
Typ publikacji:
Journal Article
MeSH Terms:
Receptors, Tumor Necrosis Factor, Type I*/therapeutic use
Receptors, Tumor Necrosis Factor, Type I*/metabolism
Staphylococcal Infections*/metabolism
Animals ; Male ; Mice ; Cytokines/metabolism ; Interleukin-10 ; Interleukin-12/therapeutic use ; Macrophages, Peritoneal/metabolism ; NF-kappa B ; Reactive Oxygen Species ; Receptors, Interleukin-8A/metabolism ; Staphylococcus aureus/metabolism ; Tumor Necrosis Factor-alpha
Czasopismo naukowe
Szczegóły
Tytuł:
Daily dose of metformin caused acute kidney injury with lactic acidosis: a case report.
Autorzy:
Ariga M; Laboratory of Pharmaceutics, Department of Biomedical Pharmaceutics, Gifu Pharmaceutical University, 1-25-4 Daigakunishi, Gifu, 501-1196, Japan.
Hagita J; Kariya Toyota General Hospital, Aichi, Japan.
Soda M; Laboratory of Pharmaceutics, Department of Biomedical Pharmaceutics, Gifu Pharmaceutical University, 1-25-4 Daigakunishi, Gifu, 501-1196, Japan.
Oida Y; Laboratory of Pharmaceutics, Department of Biomedical Pharmaceutics, Gifu Pharmaceutical University, 1-25-4 Daigakunishi, Gifu, 501-1196, Japan.
Teramachi H; Laboratory of Clinical Pharmacy, Department of Pharmacy Practice and Science, Gifu Pharmaceutical University, Gifu, Japan.
Kitaichi K; Laboratory of Pharmaceutics, Department of Biomedical Pharmaceutics, Gifu Pharmaceutical University, 1-25-4 Daigakunishi, Gifu, 501-1196, Japan. .
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Źródło:
Journal of medical case reports [J Med Case Rep] 2023 Sep 16; Vol. 17 (1), pp. 393. Date of Electronic Publication: 2023 Sep 16.
Typ publikacji:
Case Reports; Journal Article
MeSH Terms:
Metformin*/adverse effects
Diabetes Mellitus, Type 2*/complications
Diabetes Mellitus, Type 2*/drug therapy
Acidosis, Lactic*/chemically induced
Acute Kidney Injury*/chemically induced
Kidney Tubular Necrosis, Acute*
Male ; Humans ; Middle Aged ; Creatinine ; Necrosis
Czasopismo naukowe
Szczegóły
Tytuł:
A TNF-α blocking peptide that reduces NF-κB and MAPK activity for attenuating inflammation.
Autorzy:
Wang Y; School of Life Science and Technology, Changchun University of Science and Technology, Changchun 130013, China.
Ye R; School of Medicine, Shanghai University, Shanghai 200444, China.
Fan L; Department of Pharmacy, Shanghai Pudong New Area People's Hospital, Shanghai 201299, China.
Zhao X; Department of Critical Care Medicine, Shanghai Tenth People's Hospital, School of Medicine, Tongji University, Shanghai 200072,China.
Li L; School of Medicine, Shanghai University, Shanghai 200444, China.
Zheng H; School of Medicine, Shanghai University, Shanghai 200444, China.
Qiu Y; Department of Pharmacy, Shanghai Pudong New Area People's Hospital, Shanghai 201299, China. Electronic address: .
He X; School of Life Science and Technology, Changchun University of Science and Technology, Changchun 130013, China. Electronic address: .
Lu Y; School of Medicine, Shanghai University, Shanghai 200444, China; Department of Critical Care Medicine, Shanghai Tenth People's Hospital, School of Medicine, Tongji University, Shanghai 200072,China. Electronic address: .
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Źródło:
Bioorganic & medicinal chemistry [Bioorg Med Chem] 2023 Sep 07; Vol. 92, pp. 117420. Date of Electronic Publication: 2023 Jul 20.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
NF-kappa B*/metabolism
Tumor Necrosis Factor-alpha*/metabolism
Humans ; Molecular Docking Simulation ; Tumor Necrosis Factor Inhibitors/therapeutic use ; Inflammation/drug therapy ; Peptides/pharmacology ; Peptides/therapeutic use
Czasopismo naukowe
Szczegóły
Tytuł:
Dual-Targeted Therapy in Pediatric Inflammatory Bowel Disease: A Comprehensive Review.
Autorzy:
Yerushalmy-Feler A; Pediatric Gastroenterology Institute, 'Dana-Dwek' Children's Hospital, Tel Aviv Sourasky Medical Center, Affiliated to the Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Brauner C; Pediatric Gastroenterology Institute, 'Dana-Dwek' Children's Hospital, Tel Aviv Sourasky Medical Center, Affiliated to the Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Cohen S; Pediatric Gastroenterology Institute, 'Dana-Dwek' Children's Hospital, Tel Aviv Sourasky Medical Center, Affiliated to the Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel. .
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Źródło:
Paediatric drugs [Paediatr Drugs] 2023 Sep; Vol. 25 (5), pp. 489-498. Date of Electronic Publication: 2023 Jun 15.
Typ publikacji:
Review; Journal Article
MeSH Terms:
Tumor Necrosis Factor Inhibitors*/therapeutic use
Inflammatory Bowel Diseases*/drug therapy
Adult ; Humans ; Child ; Tumor Necrosis Factor-alpha ; Endoscopy ; Combined Modality Therapy ; Ustekinumab
Czasopismo naukowe
Szczegóły
Tytuł:
Anti-tumor necrosis factor therapy in Bulgarian pediatric patients with inflammatory bowel disease - an 8-year experience of a referral center.
Autorzy:
Shentova-Eneva RR; Medical University of Sofia, Sofia, Bulgaria.
Kofinova DR; Medical University of Sofia, Sofia, Bulgaria.
Baycheva MZ; Medical University of Sofia, Sofia, Bulgaria.
Hadzhiyski PG; Medical University of Sofia, Sofia, Bulgaria.
Naydenov HB; Medical University of Sofia, Sofia, Bulgaria.
Yaneva PG; Medical University of Sofia, Sofia, Bulgaria.
Lazarova EA; Medical University of Sofia, Sofia, Bulgaria.
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Źródło:
Folia medica [Folia Med (Plovdiv)] 2023 Aug 31; Vol. 65 (4), pp. 605-611.
Typ publikacji:
Journal Article
MeSH Terms:
Tumor Necrosis Factor Inhibitors*
Inflammatory Bowel Diseases*/drug therapy
Humans ; Child ; Bulgaria ; Referral and Consultation ; Necrosis
Czasopismo naukowe
Szczegóły
Tytuł:
A Model of iPSC-Derived Macrophages with TNFAIP3 Overexpression Reveals the Peculiarities of TNFAIP3 Protein Expression and Function in Human Macrophages.
Autorzy:
Sheveleva O; Laboratory of Cellular and Molecular Basis of Histogenesis, Koltzov Institute of Developmental Biology of Russian Academy of Sciences, Vavilova Str., 26, 119334 Moscow, Russia.
Protasova E; Laboratory of Cellular and Molecular Basis of Histogenesis, Koltzov Institute of Developmental Biology of Russian Academy of Sciences, Vavilova Str., 26, 119334 Moscow, Russia.
Nenasheva T; Laboratory of Cellular and Molecular Basis of Histogenesis, Koltzov Institute of Developmental Biology of Russian Academy of Sciences, Vavilova Str., 26, 119334 Moscow, Russia.
Butorina N; Laboratory of Cellular and Molecular Basis of Histogenesis, Koltzov Institute of Developmental Biology of Russian Academy of Sciences, Vavilova Str., 26, 119334 Moscow, Russia.
Melnikova V; Laboratory of Comparative Developmental Physiology, Koltzov Institute of Developmental Biology of Russian Academy of Sciences, Vavilova Str., 26, 119334 Moscow, Russia.
Gerasimova T; Laboratory of Cellular and Molecular Basis of Histogenesis, Koltzov Institute of Developmental Biology of Russian Academy of Sciences, Vavilova Str., 26, 119334 Moscow, Russia.
Sakovnich O; Laboratory of Cellular and Molecular Basis of Histogenesis, Koltzov Institute of Developmental Biology of Russian Academy of Sciences, Vavilova Str., 26, 119334 Moscow, Russia.
Kurinov A; Laboratory of Regeneration Problems, Koltzov Institute of Developmental Biology of Russian Academy of Sciences, Vavilova Str., 26, 119334 Moscow, Russia.
Grigor'eva E; Laboratory of Developmental Epigenetics, Institute of Cytology and Genetics, Siberian Branch of Russian Academy of Sciences, Lavrentyev Ave., 10, 630090 Novosibirsk, Russia.
Medvedev S; Laboratory of Developmental Epigenetics, Institute of Cytology and Genetics, Siberian Branch of Russian Academy of Sciences, Lavrentyev Ave., 10, 630090 Novosibirsk, Russia.
Lyadova I; Laboratory of Cellular and Molecular Basis of Histogenesis, Koltzov Institute of Developmental Biology of Russian Academy of Sciences, Vavilova Str., 26, 119334 Moscow, Russia.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Aug 16; Vol. 24 (16). Date of Electronic Publication: 2023 Aug 16.
Typ publikacji:
Journal Article
MeSH Terms:
Tumor Necrosis Factor-alpha*
Induced Pluripotent Stem Cells*
Humans ; Tumor Necrosis Factor alpha-Induced Protein 3/genetics ; Macrophages ; Inflammation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Morphological and molecular-biological features of glioblastoma progression in tolerant and susceptible to hypoxia Wistar rats.
Autorzy:
Dzhalilova DS; Avtsyn Research Institute of Human Morphology of Federal State Budgetary Scientific Institution 'Petrovsky National Research Centre of Surgery', 3 Tsyurupy Street, Moscow, Russia, 117418. .
Zolotova NA; Avtsyn Research Institute of Human Morphology of Federal State Budgetary Scientific Institution 'Petrovsky National Research Centre of Surgery', 3 Tsyurupy Street, Moscow, Russia, 117418.
Mkhitarov VA; Avtsyn Research Institute of Human Morphology of Federal State Budgetary Scientific Institution 'Petrovsky National Research Centre of Surgery', 3 Tsyurupy Street, Moscow, Russia, 117418.
Kosyreva AM; Avtsyn Research Institute of Human Morphology of Federal State Budgetary Scientific Institution 'Petrovsky National Research Centre of Surgery', 3 Tsyurupy Street, Moscow, Russia, 117418.; Research Institute of Molecular and Cellular Medicine, RUDN University, 6 Miklukho-Maklaya St, Moscow, Russia, 117198.
Tsvetkov IS; Avtsyn Research Institute of Human Morphology of Federal State Budgetary Scientific Institution 'Petrovsky National Research Centre of Surgery', 3 Tsyurupy Street, Moscow, Russia, 117418.
Khalansky AS; Avtsyn Research Institute of Human Morphology of Federal State Budgetary Scientific Institution 'Petrovsky National Research Centre of Surgery', 3 Tsyurupy Street, Moscow, Russia, 117418.
Alekseeva AI; Avtsyn Research Institute of Human Morphology of Federal State Budgetary Scientific Institution 'Petrovsky National Research Centre of Surgery', 3 Tsyurupy Street, Moscow, Russia, 117418.
Fatkhudinov TH; Avtsyn Research Institute of Human Morphology of Federal State Budgetary Scientific Institution 'Petrovsky National Research Centre of Surgery', 3 Tsyurupy Street, Moscow, Russia, 117418.; Research Institute of Molecular and Cellular Medicine, RUDN University, 6 Miklukho-Maklaya St, Moscow, Russia, 117198.
Makarova OV; Avtsyn Research Institute of Human Morphology of Federal State Budgetary Scientific Institution 'Petrovsky National Research Centre of Surgery', 3 Tsyurupy Street, Moscow, Russia, 117418.
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Źródło:
Scientific reports [Sci Rep] 2023 Aug 04; Vol. 13 (1), pp. 12694. Date of Electronic Publication: 2023 Aug 04.
Typ publikacji:
Journal Article
MeSH Terms:
Tumor Necrosis Factor-alpha*
Glioblastoma*/complications
Rats ; Male ; Animals ; Rats, Wistar ; Hypoxia/pathology ; Disease Susceptibility ; Necrosis/complications ; Hypoxia-Inducible Factor 1, alpha Subunit
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Polymorphisms indicating risk of inflammatory bowel disease or antigenicity to anti-TNF drugs as biomarkers of response in children.
Autorzy:
Zapata-Cobo P; Instituto de Investigación Sanitaria Gregorio Marañón, Hospital General Universitario Gregorio Marañón, Madrid, Spain.
Salvador-Martín S; Instituto de Investigación Sanitaria Gregorio Marañón, Hospital General Universitario Gregorio Marañón, Madrid, Spain.
Velasco M; Hospital Universitario Infantil Niño Jesús, Madrid, Spain.
Palomino LM; Hospital Universitario Infantil Niño Jesús, Madrid, Spain.
Clemente S; Hospital Vall d'Hebrón, Barcelona, Spain.
Segarra O; Hospital Vall d'Hebrón, Barcelona, Spain.
Moreno-Álvarez A; Hospital Universitario A Coruña, A Coruña, Spain.
Fernández-Lorenzo A; Hospital Universitario A Coruña, A Coruña, Spain.
Pérez-Moneo B; Hospital Universitario Infanta Leonor, Madrid, Spain.
Montraveta M; Hospital Universitario Germans Trias i Pujol, Badalona, Spain.
Sánchez C; Instituto de Investigación Sanitaria Gregorio Marañón, Hospital General Universitario Gregorio Marañón, Madrid, Spain.
Tolín M; Instituto de Investigación Sanitaria Gregorio Marañón, Hospital General Universitario Gregorio Marañón, Madrid, Spain.
Loverdos I; Hospital Universitario Parc Taulí, Sabadell, Spain.
Fobelo MJ; Hospital Universitario Virgen de Valme, Universidad de Sevilla, Sevilla, Spain.
Navas-López VM; Hospital Regional Universitario de Málaga, Spain.
Magallares L; Hospital Universitario la Paz, Madrid, Spain.
García-Romero R; Hospital Universitario Miguel Servet, Zaragoza, Spain.
Sánchez-Hernández JG; Complejo Asistencial Universitario de Salamanca, Salamanca, Spain.
Rodríguez A; Hospital Universitario Virgen del Rocio, Sevilla, Spain.
Bossacoma F; Servicio de Gastroenterología, Hepatología y Nutrición Pediátrica, Hospital Sant Joan de Dèu, Barcelona, Spain.
Balboa MJ; Hospital Universitario Virgen de la Macarena, Sevilla, Spain.
Salcedo E; Hospital Universitario Doce de Octubre, Madrid, Spain.
Sanjurjo-Sáez M; Instituto de Investigación Sanitaria Gregorio Marañón, Hospital General Universitario Gregorio Marañón, Madrid, Spain.
López-Fernández LA; Instituto de Investigación Sanitaria Gregorio Marañón, Hospital General Universitario Gregorio Marañón, Madrid, Spain. Electronic address: .
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Źródło:
Pharmacological research [Pharmacol Res] 2023 Aug; Vol. 194, pp. 106859. Date of Electronic Publication: 2023 Jul 19.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Tumor Necrosis Factor Inhibitors*/therapeutic use
Inflammatory Bowel Diseases*/drug therapy
Inflammatory Bowel Diseases*/genetics
Humans ; Child ; Tumor Necrosis Factor-alpha/genetics ; Prospective Studies ; Polymorphism, Single Nucleotide ; Biomarkers
Czasopismo naukowe
Szczegóły
Tytuł:
Comparative Efficacy of Secukinumab Versus Tumor Necrosis Factor Inhibitors for the Treatment of Takayasu Arteritis.
Autorzy:
Tian X; Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, National Clinical Research Center for Dermatologic and Immunologic Diseases, Beijing, China.
Li M; Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, National Clinical Research Center for Dermatologic and Immunologic Diseases, Beijing, China.
Jiang N; Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, National Clinical Research Center for Dermatologic and Immunologic Diseases, Beijing, China.
Zhao Y; Department of Rheumatology, Weihai Municipal Hospital, Cheeloo College of Medicine, Shandong University, Weihai, China.
Li J; Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, National Clinical Research Center for Dermatologic and Immunologic Diseases, Beijing, China.
Zhou Y; Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, National Clinical Research Center for Dermatologic and Immunologic Diseases, Beijing, China.
Wang Y; Department of Ultrasonography, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China.
Wang Y; Department of Ultrasonography, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China.
Li T; Department of Rheumatology, Beijing Anzhen Hospital, Capital Medical University, Beijing, China.
Yang Y; Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, National Clinical Research Center for Dermatologic and Immunologic Diseases, Beijing, China.
Wang Y; Department of Epidemiology and Bio-statistics, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China.
Merkel PA; Division of Rheumatology, Department of Medicine; and Division of Epidemiology, Department of Biostatistics, Epidemiology, and Informatics, University of Pennsylvania, Philadelphia.
Zeng X; Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, National Clinical Research Center for Dermatologic and Immunologic Diseases, Beijing, China.
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Źródło:
Arthritis & rheumatology (Hoboken, N.J.) [Arthritis Rheumatol] 2023 Aug; Vol. 75 (8), pp. 1415-1423. Date of Electronic Publication: 2023 Jun 08.
Typ publikacji:
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Takayasu Arteritis*/drug therapy
Tumor Necrosis Factor Inhibitors*/therapeutic use
Humans ; Cohort Studies ; Glucocorticoids ; Immunosuppressive Agents/therapeutic use ; Prospective Studies ; Treatment Outcome ; Tumor Necrosis Factor-alpha
Czasopismo naukowe
Szczegóły
Tytuł:
Short Peptides of Innate Immunity Protein Tag7 (PGLYRP1) Selectively Induce Inhibition or Activation of Tumor Cell Death via TNF Receptor.
Autorzy:
Yurkina DM; Institute of Gene Biology (RAS), Moscow 119334, Russia.
Sharapova TN; Institute of Gene Biology (RAS), Moscow 119334, Russia.
Romanova EA; Institute of Gene Biology (RAS), Moscow 119334, Russia.
Yashin DV; Institute of Gene Biology (RAS), Moscow 119334, Russia.
Sashchenko LP; Institute of Gene Biology (RAS), Moscow 119334, Russia.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Jul 12; Vol. 24 (14). Date of Electronic Publication: 2023 Jul 12.
Typ publikacji:
Journal Article
MeSH Terms:
Cytokines*/metabolism
Neoplasms*
Receptors, Tumor Necrosis Factor, Type I*/genetics
Humans ; Apoptosis ; Immunity, Innate ; Peptides/pharmacology ; Tumor Necrosis Factor-alpha
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Role of tumor necrosis factor-alpha in the central nervous system: a focus on autoimmune disorders.
Autorzy:
Gonzalez Caldito N; Department of Neurology, Northwestern Memorial Hospital, Feinberg School of Medicine, Northwestern University, Chicago, IL, United States.
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Źródło:
Frontiers in immunology [Front Immunol] 2023 Jul 07; Vol. 14, pp. 1213448. Date of Electronic Publication: 2023 Jul 07 (Print Publication: 2023).
Typ publikacji:
Journal Article; Review
MeSH Terms:
Tumor Necrosis Factor-alpha*
Multiple Sclerosis*
Humans ; Tumor Necrosis Factor Inhibitors ; Central Nervous System ; Cytokines
Czasopismo naukowe
Szczegóły
Tytuł:
Tumor necrosis factor-alpha inhibitors and acne fulminans: Friend or foe?
Autorzy:
Barry KK; Department of Dermatology, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts, USA.; Tufts University School of Medicine, Boston, Massachusetts, USA.
Neale HD; University of Massachusetts Medical School, Worcester, Massachusetts, USA.
Hawryluk EB; Department of Dermatology, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts, USA.
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Źródło:
Pediatric dermatology [Pediatr Dermatol] 2023 Jul-Aug; Vol. 40 (4), pp. 678-680. Date of Electronic Publication: 2022 Dec 27.
Typ publikacji:
Case Reports
MeSH Terms:
Tumor Necrosis Factor-alpha*
Acne Vulgaris*/drug therapy
Humans ; Tumor Necrosis Factor Inhibitors/therapeutic use ; Skin/pathology ; Adalimumab/adverse effects ; Immunologic Factors/therapeutic use
Raport
Szczegóły
Tytuł:
Discovery of a Non-competitive TNFR1 Antagonist Affibody with Picomolar Monovalent Potency That Does Not Affect TNFR2 Function.
Autorzy:
Vunnam N; Department of Biomedical Engineering, University of Minnesota, Minneapolis, Minnesota 55455, United States.
Been M; Department of Biomedical Engineering, University of Minnesota, Minneapolis, Minnesota 55455, United States.
Huber E; Department of Biomedical Engineering, University of Minnesota, Minneapolis, Minnesota 55455, United States.
Paulson C; Department of Biomedical Engineering, University of Minnesota, Minneapolis, Minnesota 55455, United States.
Szymonski S; Department of Biomedical Engineering, University of Minnesota, Minneapolis, Minnesota 55455, United States.
Hackel BJ; Department of Chemical Engineering and Materials Science, University of Minnesota, Minneapolis, Minnesota 55455, United States.
Sachs JN; Department of Biomedical Engineering, University of Minnesota, Minneapolis, Minnesota 55455, United States.
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Źródło:
Molecular pharmaceutics [Mol Pharm] 2023 Apr 03; Vol. 20 (4), pp. 1884-1897. Date of Electronic Publication: 2023 Mar 10.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Receptors, Tumor Necrosis Factor, Type I*/chemistry
Receptors, Tumor Necrosis Factor, Type I*/metabolism
Receptors, Tumor Necrosis Factor, Type II*/chemistry
Receptors, Tumor Necrosis Factor, Type II*/metabolism
Ligands ; Tumor Necrosis Factor Inhibitors ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
Tranexamic acid for management of gross hematuria due to renal papillary necrosis in sickle cell disease.
Autorzy:
Cochrane AM; Department of Pediatrics, Washington University in St. Louis School of Medicine, St. Louis, Missouri, USA.
Barton KT; Division of Pediatric Nephrology, Washington University in St. Louis School of Medicine, St. Louis, Missouri, USA.
Turner MN; St. Louis Children's Hospital, St. Louis, Missouri, USA.
Hulbert ML; Division of Pediatric Hematology/Oncology, Washington University in St. Louis School of Medicine, St. Louis, Missouri, USA.
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Źródło:
Pediatric blood & cancer [Pediatr Blood Cancer] 2023 Oct; Vol. 70 (10), pp. e30549. Date of Electronic Publication: 2023 Jul 12.
Typ publikacji:
Letter
MeSH Terms:
Tranexamic Acid*/therapeutic use
Anemia, Sickle Cell*/complications
Anemia, Sickle Cell*/drug therapy
Kidney Papillary Necrosis*/complications
Kidney Papillary Necrosis*/drug therapy
Sickle Cell Trait*
Humans ; Hematuria/drug therapy ; Hematuria/etiology ; Necrosis
Opinia redakcyjna
Szczegóły
Wyświetlanie 1-20 z 376139

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