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Tytuł :
Taraxasterol protects hippocampal neurons from oxygen-glucose deprivation-induced injury through activation of Nrf2 signalling pathway.
Autorzy :
He Y; Department of Neurology, AnKang Central Hospital, AnKang, China.
Jiang K; Department of Neurology, AnKang Central Hospital, AnKang, China.
Zhao X; Department of Neurology, AnKang Central Hospital, AnKang, China.
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Źródło :
Artificial cells, nanomedicine, and biotechnology [Artif Cells Nanomed Biotechnol] 2020 Dec; Vol. 48 (1), pp. 252-258.
Typ publikacji :
Journal Article
MeSH Terms :
Glucose/*deficiency
Hippocampus/*cytology
NF-E2-Related Factor 2/*metabolism
Neurons/*drug effects
Oxygen/*metabolism
Signal Transduction/*drug effects
Sterols/*pharmacology
Triterpenes/*pharmacology
Animals ; Apoptosis/drug effects ; Cytoprotection/drug effects ; Female ; Mice ; Neurons/cytology ; Neurons/metabolism ; Oxidative Stress/drug effects ; Pregnancy
Czasopismo naukowe
Tytuł :
Alleviation of brain injury by applying TGN-020 in the supraoptic nucleus via inhibiting vasopressin neurons in rats of focal ischemic stroke.
Autorzy :
Cui D; Department of Physiology, School of Basic Medical Sciences, Harbin Medical University, Harbin, China.
Jia S; Department of Physiology, School of Basic Medical Sciences, Harbin Medical University, Harbin, China.
Li T; Department of Physiology, School of Basic Medical Sciences, Harbin Medical University, Harbin, China.
Li D; Department of Physiology, School of Basic Medical Sciences, Harbin Medical University, Harbin, China.
Wang X; Department of Physiology, School of Basic Medical Sciences, Harbin Medical University, Harbin, China.
Liu X; Department of Physiology, School of Basic Medical Sciences, Harbin Medical University, Harbin, China.
Wang YF; Department of Physiology, School of Basic Medical Sciences, Harbin Medical University, Harbin, China. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2021 Jan 01; Vol. 264, pp. 118683. Date of Electronic Publication: 2020 Oct 28.
Typ publikacji :
Journal Article
MeSH Terms :
Brain Injuries/*metabolism
Ischemic Stroke/*metabolism
Neurons/*metabolism
Niacinamide/*analogs & derivatives
Supraoptic Nucleus/*metabolism
Thiadiazoles/*administration & dosage
Vasopressins/*metabolism
Animals ; Brain Injuries/drug therapy ; Ischemic Stroke/drug therapy ; Male ; Microinjections/methods ; Neurons/drug effects ; Niacinamide/administration & dosage ; Rats ; Rats, Wistar ; Supraoptic Nucleus/drug effects ; Vasopressins/antagonists & inhibitors
Czasopismo naukowe
Tytuł :
Cell cycle re-entry of neurons and reactive neuroblastosis in Huntington's disease: Possibilities for neural-glial transition in the brain.
Autorzy :
Manickam N; Laboratory of Stem Cells and Neuroregeneration, Department of Animal Science, School of Life Sciences, Bharathidasan University, Tiruchirappalli 620024, Tamil Nadu, India.
Radhakrishnan RK; Laboratory of Stem Cells and Neuroregeneration, Department of Animal Science, School of Life Sciences, Bharathidasan University, Tiruchirappalli 620024, Tamil Nadu, India.
Vergil Andrews JF; Laboratory of Stem Cells and Neuroregeneration, Department of Animal Science, School of Life Sciences, Bharathidasan University, Tiruchirappalli 620024, Tamil Nadu, India.
Selvaraj DB; Laboratory of Stem Cells and Neuroregeneration, Department of Animal Science, School of Life Sciences, Bharathidasan University, Tiruchirappalli 620024, Tamil Nadu, India.
Kandasamy M; Laboratory of Stem Cells and Neuroregeneration, Department of Animal Science, School of Life Sciences, Bharathidasan University, Tiruchirappalli 620024, Tamil Nadu, India; Faculty Recharge Programme, University Grants Commission (UGC-FRP), New Delhi 110002, India. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2020 Dec 15; Vol. 263, pp. 118569. Date of Electronic Publication: 2020 Oct 10.
Typ publikacji :
Journal Article; Review
MeSH Terms :
Cell Cycle*
Brain/*pathology
Huntington Disease/*pathology
Neuroglia/*pathology
Neurons/*pathology
Animals ; Brain/metabolism ; Humans ; Huntington Disease/metabolism ; Neuroglia/metabolism ; Neurons/metabolism
Czasopismo naukowe
Tytuł :
Perfluoroalkyl acids potentiate glutamate excitotoxicity in rat cerebellar granule neurons.
Autorzy :
Berntsen HF; Department of Production Animal Clinical Sciences NMBU-School of Veterinary Science, P.O. Box 369 sentrum, N-0102, Oslo, Norway; National Institute of Occupational Health, P.O. Box 8149 Dep N-0033, Oslo, Norway. Electronic address: .
Moldes-Anaya A; Research and Development (R&D) Section, PET Imaging Center, University Hospital of North Norway (UNN), Tromsø, Norway; Nuclear Medicine and Radiation Biology Research Group, Department of Clinical Medicine, UiT The Arctic University of Norway, Tromsø, Norway.
Bjørklund CG; Department of Production Animal Clinical Sciences NMBU-School of Veterinary Science, P.O. Box 369 sentrum, N-0102, Oslo, Norway.
Ragazzi L; Neurobiology Research Group, Department of Clinical Medicine, UiT The Arctic University of Norway, Tromsø, Norway.
Haug TM; Department of Oral Biology, University of Oslo, Oslo, Norway.
Strandabø RAU; Department of Biosciences, University of Oslo, Oslo, Norway.
Verhaegen S; Department of Production Animal Clinical Sciences NMBU-School of Veterinary Science, P.O. Box 369 sentrum, N-0102, Oslo, Norway.
Paulsen RE; Department of Pharmacy, Section for Pharmacology and Pharmaceutical Biosciences, University of Oslo, Oslo, Norway.
Ropstad E; Department of Production Animal Clinical Sciences NMBU-School of Veterinary Science, P.O. Box 369 sentrum, N-0102, Oslo, Norway.
Tasker RA; Department of Biomedical Sciences, University of Prince Edward Island, Charlottetown, PEI, Canada; Translational Neuropsychiatry Unit, Department of Clinical Medicine, Aarhus University, Aarhus, Denmark.
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Źródło :
Toxicology [Toxicology] 2020 Dec 01; Vol. 445, pp. 152610. Date of Electronic Publication: 2020 Oct 04.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Alkanesulfonic Acids/*toxicity
Caprylates/*toxicity
Cerebellum/*drug effects
Excitatory Amino Acid Agonists/*toxicity
Fluorocarbons/*toxicity
Glutamic Acid/*toxicity
Neurons/*drug effects
Alkanesulfonic Acids/administration & dosage ; Animals ; Caprylates/administration & dosage ; Cattle ; Cell Survival/drug effects ; Cell Survival/physiology ; Cells, Cultured ; Cerebellum/pathology ; Dose-Response Relationship, Drug ; Drug Synergism ; Excitatory Amino Acid Agonists/administration & dosage ; Female ; Fluorocarbons/administration & dosage ; Glutamic Acid/administration & dosage ; Male ; Neurons/pathology ; Rats ; Rats, Wistar
Czasopismo naukowe
Tytuł :
Core cell cycle machinery is crucially involved in both life and death of post-mitotic neurons.
Autorzy :
Marlier Q; Developmental Neurobiology Unit, GIGA Stem Cells/Neurosciences, University of Liège, Quartier Hopital (CHU), Avenue Hippocrate, 15, 4000, Liege, Belgium.
D'aes T; Developmental Neurobiology Unit, GIGA Stem Cells/Neurosciences, University of Liège, Quartier Hopital (CHU), Avenue Hippocrate, 15, 4000, Liege, Belgium.
Verteneuil S; Developmental Neurobiology Unit, GIGA Stem Cells/Neurosciences, University of Liège, Quartier Hopital (CHU), Avenue Hippocrate, 15, 4000, Liege, Belgium.
Vandenbosch R; Developmental Neurobiology Unit, GIGA Stem Cells/Neurosciences, University of Liège, Quartier Hopital (CHU), Avenue Hippocrate, 15, 4000, Liege, Belgium.
Malgrange B; Developmental Neurobiology Unit, GIGA Stem Cells/Neurosciences, University of Liège, Quartier Hopital (CHU), Avenue Hippocrate, 15, 4000, Liege, Belgium. .
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Źródło :
Cellular and molecular life sciences : CMLS [Cell Mol Life Sci] 2020 Nov; Vol. 77 (22), pp. 4553-4571. Date of Electronic Publication: 2020 May 31.
Typ publikacji :
Journal Article; Review
MeSH Terms :
Cell Cycle/*physiology
Mitosis/*physiology
Neurons/*physiology
Animals ; Apoptosis/physiology ; Cell Cycle Proteins/metabolism ; Humans ; Nervous System Diseases/metabolism ; Nervous System Diseases/physiopathology ; Neurons/metabolism
Czasopismo naukowe
Tytuł :
AMP-activated protein kinase (AMPK) signaling in GnRH neurons links energy status and reproduction.
Autorzy :
Franssen D; Instituto Maimónides de Investigación Biomédica de Córdoba (IMIBIC), 14004 Cordoba, Spain; Departament of Cell Biology, Physiology and Immunology, University of Cordoba, 14004 Cordoba, Spain; Hospital Universitario Reina Sofía, 14004 Cordoba, Spain.
Barroso A; Instituto Maimónides de Investigación Biomédica de Córdoba (IMIBIC), 14004 Cordoba, Spain; Departament of Cell Biology, Physiology and Immunology, University of Cordoba, 14004 Cordoba, Spain; Hospital Universitario Reina Sofía, 14004 Cordoba, Spain; CIBER Fisiopatología de la Obesidad y Nutrición (CIBEROBN), Instituto de Salud Carlos III, 14004 Córdoba, Spain.
Ruiz-Pino F; Instituto Maimónides de Investigación Biomédica de Córdoba (IMIBIC), 14004 Cordoba, Spain; Departament of Cell Biology, Physiology and Immunology, University of Cordoba, 14004 Cordoba, Spain; Hospital Universitario Reina Sofía, 14004 Cordoba, Spain; CIBER Fisiopatología de la Obesidad y Nutrición (CIBEROBN), Instituto de Salud Carlos III, 14004 Córdoba, Spain.
Vázquez MJ; Instituto Maimónides de Investigación Biomédica de Córdoba (IMIBIC), 14004 Cordoba, Spain; Departament of Cell Biology, Physiology and Immunology, University of Cordoba, 14004 Cordoba, Spain; Hospital Universitario Reina Sofía, 14004 Cordoba, Spain; CIBER Fisiopatología de la Obesidad y Nutrición (CIBEROBN), Instituto de Salud Carlos III, 14004 Córdoba, Spain.
García-Galiano D; Instituto Maimónides de Investigación Biomédica de Córdoba (IMIBIC), 14004 Cordoba, Spain; Departament of Cell Biology, Physiology and Immunology, University of Cordoba, 14004 Cordoba, Spain; Hospital Universitario Reina Sofía, 14004 Cordoba, Spain.
Castellano JM; Instituto Maimónides de Investigación Biomédica de Córdoba (IMIBIC), 14004 Cordoba, Spain; Departament of Cell Biology, Physiology and Immunology, University of Cordoba, 14004 Cordoba, Spain; Hospital Universitario Reina Sofía, 14004 Cordoba, Spain; CIBER Fisiopatología de la Obesidad y Nutrición (CIBEROBN), Instituto de Salud Carlos III, 14004 Córdoba, Spain.
Onieva R; Instituto Maimónides de Investigación Biomédica de Córdoba (IMIBIC), 14004 Cordoba, Spain; Departament of Cell Biology, Physiology and Immunology, University of Cordoba, 14004 Cordoba, Spain; Hospital Universitario Reina Sofía, 14004 Cordoba, Spain.
Ruiz-Cruz M; Instituto Maimónides de Investigación Biomédica de Córdoba (IMIBIC), 14004 Cordoba, Spain; Departament of Cell Biology, Physiology and Immunology, University of Cordoba, 14004 Cordoba, Spain; Hospital Universitario Reina Sofía, 14004 Cordoba, Spain.
Poutanen M; Research Centre for Integrative Physiology and Pharmacology, Institute of Biomedicine and Turku Center for Disease Modeling, University of Turku, Turku, Finland.
Gaytán F; Instituto Maimónides de Investigación Biomédica de Córdoba (IMIBIC), 14004 Cordoba, Spain; Departament of Cell Biology, Physiology and Immunology, University of Cordoba, 14004 Cordoba, Spain; Hospital Universitario Reina Sofía, 14004 Cordoba, Spain; CIBER Fisiopatología de la Obesidad y Nutrición (CIBEROBN), Instituto de Salud Carlos III, 14004 Córdoba, Spain.
Diéguez C; CIBER Fisiopatología de la Obesidad y Nutrición (CIBEROBN), Instituto de Salud Carlos III, 14004 Córdoba, Spain; NeurObesity Group, Department of Physiology, CIMUS, University of Santiago de Compostela-Instituto de Investigación Sanitaria, Santiago de Compostela, Spain.
Pinilla L; Instituto Maimónides de Investigación Biomédica de Córdoba (IMIBIC), 14004 Cordoba, Spain; Departament of Cell Biology, Physiology and Immunology, University of Cordoba, 14004 Cordoba, Spain; Hospital Universitario Reina Sofía, 14004 Cordoba, Spain; CIBER Fisiopatología de la Obesidad y Nutrición (CIBEROBN), Instituto de Salud Carlos III, 14004 Córdoba, Spain.
Lopez M; CIBER Fisiopatología de la Obesidad y Nutrición (CIBEROBN), Instituto de Salud Carlos III, 14004 Córdoba, Spain; NeurObesity Group, Department of Physiology, CIMUS, University of Santiago de Compostela-Instituto de Investigación Sanitaria, Santiago de Compostela, Spain.
Roa J; Instituto Maimónides de Investigación Biomédica de Córdoba (IMIBIC), 14004 Cordoba, Spain; Departament of Cell Biology, Physiology and Immunology, University of Cordoba, 14004 Cordoba, Spain; Hospital Universitario Reina Sofía, 14004 Cordoba, Spain; CIBER Fisiopatología de la Obesidad y Nutrición (CIBEROBN), Instituto de Salud Carlos III, 14004 Córdoba, Spain. Electronic address: .
Tena-Sempere M; Instituto Maimónides de Investigación Biomédica de Córdoba (IMIBIC), 14004 Cordoba, Spain; Departament of Cell Biology, Physiology and Immunology, University of Cordoba, 14004 Cordoba, Spain; Hospital Universitario Reina Sofía, 14004 Cordoba, Spain; CIBER Fisiopatología de la Obesidad y Nutrición (CIBEROBN), Instituto de Salud Carlos III, 14004 Córdoba, Spain; Research Centre for Integrative Physiology and Pharmacology, Institute of Biomedicine and Turku Center for Disease Modeling, University of Turku, Turku, Finland. Electronic address: .
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Źródło :
Metabolism: clinical and experimental [Metabolism] 2021 Feb; Vol. 115, pp. 154460. Date of Electronic Publication: 2020 Dec 04.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
AMP-Activated Protein Kinases/*metabolism
Energy Metabolism/*physiology
Gonadotropin-Releasing Hormone/*metabolism
Luteinizing Hormone/*blood
Neurons/*metabolism
Reproduction/*physiology
AMP-Activated Protein Kinases/genetics ; Animals ; Estrous Cycle/metabolism ; Female ; Kisspeptins/pharmacology ; Male ; Malnutrition/metabolism ; Mice ; Mice, Knockout ; Neurons/drug effects ; Phosphorylation ; Sex Characteristics ; Signal Transduction/drug effects ; Signal Transduction/physiology
Czasopismo naukowe
Tytuł :
Molecular mechanism of nanoparticulate TiO 2 induction of axonal development inhibition in rat primary cultured hippocampal neurons.
Autorzy :
Mu X; Department of Biochemistry and Molecular Biology, School of Basic Medical and Biological Sciences, Soochow University, Suzhou, China.
Li W; Department of Biochemistry and Molecular Biology, School of Basic Medical and Biological Sciences, Soochow University, Suzhou, China.
Ze X; Department of Orthopedics, The Second Affiliated Hospital of Soochow University, Suzhou, China.
Li L; Department of Biochemistry and Molecular Biology, School of Basic Medical and Biological Sciences, Soochow University, Suzhou, China.
Wang G; Department of Physiology and Neurobiology, School of Basic Medical and Biological Sciences, Soochow University, Suzhou, China.
Hong F; Jiangsu Collaborative Innovation Center of Regional Modern Agriculture and Environmental Protection, Huaiyin Normal University, Huaian, China.; Department of Biotechnology, School of Life Sciences, Huaiyin Normal University, Huaian, China.
Ze Y; Department of Biochemistry and Molecular Biology, School of Basic Medical and Biological Sciences, Soochow University, Suzhou, China.
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Źródło :
Environmental toxicology [Environ Toxicol] 2020 Aug; Vol. 35 (8), pp. 895-905. Date of Electronic Publication: 2020 Apr 24.
Typ publikacji :
Journal Article
MeSH Terms :
Nanoparticles/*toxicity
Neurons/*drug effects
Titanium/*toxicity
Animals ; Axons ; Hippocampus/drug effects ; Neurogenesis ; Neurons/metabolism ; Nogo Proteins/metabolism ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Tytuł :
NOGO-A immunolabeling is present in glial cells and some neurons of the recovering lumbar spinal cord in lizards.
Autorzy :
Alibardi L; Comparative Histolab Padova and Department of Biology, University of Bologna, Bologna, Italy.
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Źródło :
Journal of morphology [J Morphol] 2020 Oct; Vol. 281 (10), pp. 1260-1270. Date of Electronic Publication: 2020 Aug 08.
Typ publikacji :
Journal Article
MeSH Terms :
Lizards/*metabolism
Neuroglia/*metabolism
Neurons/*metabolism
Nogo Proteins/*metabolism
Spinal Cord/*cytology
Animals ; Axons/metabolism ; Axons/ultrastructure ; Behavior, Animal ; Brain/metabolism ; Nerve Regeneration/physiology ; Neuroglia/pathology ; Neuroglia/ultrastructure ; Neurons/ultrastructure ; Oligodendroglia/metabolism ; Oligodendroglia/ultrastructure ; Spinal Cord Injuries/metabolism ; Spinal Cord Injuries/pathology
Czasopismo naukowe
Tytuł :
Human Immunodeficiency Virus Type 1 and Methamphetamine-Mediated Mitochondrial Damage and Neuronal Degeneration in Human Neurons.
Autorzy :
Teodorof-Diedrich C; Department of Pediatrics, Division of Infectious Diseases, University of California San Diego, La Jolla, California, USA.
Spector SA; Department of Pediatrics, Division of Infectious Diseases, University of California San Diego, La Jolla, California, USA .; Rady Children's Hospital, San Diego, California, USA.
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Źródło :
Journal of virology [J Virol] 2020 Sep 29; Vol. 94 (20). Date of Electronic Publication: 2020 Sep 29 (Print Publication: 2020).
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
HIV Infections/*metabolism
HIV-1/*metabolism
Methamphetamine/*adverse effects
Mitochondria/*metabolism
Neurodegenerative Diseases/*metabolism
Neurons/*metabolism
Cells, Cultured ; Dynamins/genetics ; Dynamins/metabolism ; HIV Envelope Protein gp120/genetics ; HIV Envelope Protein gp120/metabolism ; HIV Infections/genetics ; HIV Infections/pathology ; HIV-1/genetics ; Humans ; Methamphetamine/pharmacology ; Microtubule-Associated Proteins/genetics ; Microtubule-Associated Proteins/metabolism ; Mitochondria/genetics ; Mitochondria/pathology ; Neurodegenerative Diseases/chemically induced ; Neurodegenerative Diseases/genetics ; Neurodegenerative Diseases/virology ; Neurons/pathology ; Neurons/virology ; Sequestosome-1 Protein/genetics ; Sequestosome-1 Protein/metabolism ; tat Gene Products, Human Immunodeficiency Virus/genetics ; tat Gene Products, Human Immunodeficiency Virus/metabolism
Czasopismo naukowe
Tytuł :
Extracellular tau induces microglial phagocytosis of living neurons in cell cultures.
Autorzy :
Pampuscenko K; Neuroscience Institute, Lithuanian University of Health Sciences, Kaunas, Lithuania.
Morkuniene R; Neuroscience Institute, Lithuanian University of Health Sciences, Kaunas, Lithuania.
Sneideris T; Institute of Biotechnology, Life Sciences Center, Vilnius University, Vilnius, Lithuania.
Smirnovas V; Institute of Biotechnology, Life Sciences Center, Vilnius University, Vilnius, Lithuania.
Budvytyte R; Institute of Biochemistry, Life Sciences Center, Vilnius University, Vilnius, Lithuania.
Valincius G; Institute of Biochemistry, Life Sciences Center, Vilnius University, Vilnius, Lithuania.
Brown GC; Department of Biochemistry, University of Cambridge, Cambridge, UK.
Borutaite V; Neuroscience Institute, Lithuanian University of Health Sciences, Kaunas, Lithuania.
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Źródło :
Journal of neurochemistry [J Neurochem] 2020 Aug; Vol. 154 (3), pp. 316-329. Date of Electronic Publication: 2019 Dec 29.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Neurons*/pathology
Microglia/*drug effects
Phagocytosis/*drug effects
tau Proteins/*pharmacology
Animals ; Cells, Cultured ; Coculture Techniques ; Microglia/metabolism ; Rats ; tau Proteins/metabolism
Czasopismo naukowe
Tytuł :
AgRP neurons control compulsive exercise and survival in an activity-based anorexia model.
Autorzy :
Miletta MC; Program in Integrative Cell Signaling and Neurobiology of Metabolism, Department of Comparative Medicine, Yale University School of Medicine, New Haven, CT, USA.
Iyilikci O; Program in Integrative Cell Signaling and Neurobiology of Metabolism, Department of Comparative Medicine, Yale University School of Medicine, New Haven, CT, USA.; Laboratory of Physiology of Behavior, Department of Comparative Medicine, Yale University School of Medicine, New Haven, CT, USA.
Shanabrough M; Program in Integrative Cell Signaling and Neurobiology of Metabolism, Department of Comparative Medicine, Yale University School of Medicine, New Haven, CT, USA.
Šestan-Peša M; Program in Integrative Cell Signaling and Neurobiology of Metabolism, Department of Comparative Medicine, Yale University School of Medicine, New Haven, CT, USA.
Cammisa A; Frank H. Netter MD School of Medicine, Quinnipiac University, North Haven, CT, USA.
Zeiss CJ; Program in Integrative Cell Signaling and Neurobiology of Metabolism, Department of Comparative Medicine, Yale University School of Medicine, New Haven, CT, USA.
Dietrich MO; Program in Integrative Cell Signaling and Neurobiology of Metabolism, Department of Comparative Medicine, Yale University School of Medicine, New Haven, CT, USA.; Laboratory of Physiology of Behavior, Department of Comparative Medicine, Yale University School of Medicine, New Haven, CT, USA.
Horvath TL; Program in Integrative Cell Signaling and Neurobiology of Metabolism, Department of Comparative Medicine, Yale University School of Medicine, New Haven, CT, USA. .
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Źródło :
Nature metabolism [Nat Metab] 2020 Nov; Vol. 2 (11), pp. 1204-1211. Date of Electronic Publication: 2020 Oct 26.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Agouti-Related Protein/*metabolism
Anorexia/*metabolism
Compulsive Exercise/*metabolism
Neurons/*metabolism
Animals ; Anorexia/psychology ; Behavior, Animal ; Body Weight ; Compulsive Exercise/psychology ; Diet ; Diet, High-Fat ; Female ; Food Deprivation ; Humans ; Male ; Maze Learning ; Mice ; Mice, Transgenic ; Nerve Fibers/metabolism ; Survival Analysis
Czasopismo naukowe
Tytuł :
Brainwide Genetic Sparse Cell Labeling to Illuminate the Morphology of Neurons and Glia with Cre-Dependent MORF Mice.
Autorzy :
Veldman MB; Center for Neurobehavioral Genetics, Jane and Terry Semel Institute for Neuroscience and Human Behavior, and Department of Psychiatry and Biobehavioral Sciences, Brain Research Institute, David Geffen School of Medicine at University of California, Los Angeles, Los Angeles, CA 90095, USA.
Park CS; Center for Neurobehavioral Genetics, Jane and Terry Semel Institute for Neuroscience and Human Behavior, and Department of Psychiatry and Biobehavioral Sciences, Brain Research Institute, David Geffen School of Medicine at University of California, Los Angeles, Los Angeles, CA 90095, USA.
Eyermann CM; Center for Neurobehavioral Genetics, Jane and Terry Semel Institute for Neuroscience and Human Behavior, and Department of Psychiatry and Biobehavioral Sciences, Brain Research Institute, David Geffen School of Medicine at University of California, Los Angeles, Los Angeles, CA 90095, USA.
Zhang JY; Center for Neurobehavioral Genetics, Jane and Terry Semel Institute for Neuroscience and Human Behavior, and Department of Psychiatry and Biobehavioral Sciences, Brain Research Institute, David Geffen School of Medicine at University of California, Los Angeles, Los Angeles, CA 90095, USA.
Zuniga-Sanchez E; Department of Biological Chemistry, Howard Hughes Medical Institute, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, USA.
Hirano AA; Department of Neurobiology, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, USA; Veterans Administration of Greater Los Angeles Health System, Los Angeles, CA 90073, USA.
Daigle TL; Allen Institute for Brain Science, Seattle, WA 98109, USA.
Foster NN; Center for Integrative Connectomics, University of Southern California Stevens Neuroimaging and Informatics Institute, Keck School of Medicine, USC, Los Angeles, CA, 90033, USA.
Zhu M; Center for Integrative Connectomics, University of Southern California Stevens Neuroimaging and Informatics Institute, Keck School of Medicine, USC, Los Angeles, CA, 90033, USA.
Langfelder P; Center for Neurobehavioral Genetics, Jane and Terry Semel Institute for Neuroscience and Human Behavior, and Department of Psychiatry and Biobehavioral Sciences, Brain Research Institute, David Geffen School of Medicine at University of California, Los Angeles, Los Angeles, CA 90095, USA.
Lopez IA; Cellular and Molecular Biology of the Inner Ear Laboratory, Department of Head and Neck Surgery, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, USA.
Brecha NC; Department of Neurobiology, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, USA; Veterans Administration of Greater Los Angeles Health System, Los Angeles, CA 90073, USA; Departments of Medicine and Ophthalmology, Stein Eye Institute, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, USA.
Zipursky SL; Department of Biological Chemistry, Howard Hughes Medical Institute, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, USA.
Zeng H; Allen Institute for Brain Science, Seattle, WA 98109, USA.
Dong HW; Center for Integrative Connectomics, University of Southern California Stevens Neuroimaging and Informatics Institute, Keck School of Medicine, USC, Los Angeles, CA, 90033, USA; Zilkha Neurogenetic Institute, and Department of Neurology, Keck School of Medicine, University of Southern California, Los Angeles, CA 90033, USA.
Yang XW; Center for Neurobehavioral Genetics, Jane and Terry Semel Institute for Neuroscience and Human Behavior, and Department of Psychiatry and Biobehavioral Sciences, Brain Research Institute, David Geffen School of Medicine at University of California, Los Angeles, Los Angeles, CA 90095, USA. Electronic address: .
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Źródło :
Neuron [Neuron] 2020 Oct 14; Vol. 108 (1), pp. 111-127.e6. Date of Electronic Publication: 2020 Aug 13.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Video-Audio Media
MeSH Terms :
Astrocytes/*ultrastructure
Brain/*ultrastructure
Microglia/*ultrastructure
Neurons/*ultrastructure
Retinal Horizontal Cells/*ultrastructure
Animals ; Astrocytes/metabolism ; Astrocytes/pathology ; Brain/metabolism ; Brain/pathology ; Frameshift Mutation/genetics ; Green Fluorescent Proteins/genetics ; Integrases ; Mice ; Mice, Transgenic ; Microglia/metabolism ; Microglia/pathology ; Microsatellite Repeats/genetics ; Neurons/metabolism ; Neurons/pathology ; Retinal Horizontal Cells/metabolism ; Retinal Horizontal Cells/pathology
Czasopismo naukowe
Tytuł :
Transfer of pathological α-synuclein from neurons to astrocytes via exosomes causes inflammatory responses after METH exposure.
Autorzy :
Meng Y; School of Forensic Medicine, Southern Medical University, Guangzhou, China.
Ding J; School of Forensic Medicine, Southern Medical University, Guangzhou, China.
Li C; School of Forensic Medicine, Southern Medical University, Guangzhou, China.
Fan H; School of Forensic Medicine, Southern Medical University, Guangzhou, China; School of Basic Medicine and Life Science, Hainan Medical University, Haikou, China.
He Y; School of Forensic Medicine, Southern Medical University, Guangzhou, China.
Qiu P; School of Forensic Medicine, Southern Medical University, Guangzhou, China. Electronic address: .
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Źródło :
Toxicology letters [Toxicol Lett] 2020 Oct 01; Vol. 331, pp. 188-199. Date of Electronic Publication: 2020 Jun 20.
Typ publikacji :
Journal Article
MeSH Terms :
Astrocytes/*drug effects
Central Nervous System Stimulants/*toxicity
Exosomes/*metabolism
Methamphetamine/*toxicity
Neurons/*drug effects
Neurotoxicity Syndromes/*metabolism
alpha-Synuclein/*metabolism
Animals ; Astrocytes/immunology ; Astrocytes/metabolism ; Cell Line, Tumor ; Coculture Techniques ; Cytokines/metabolism ; Hippocampus/cytology ; Humans ; Injections, Intraperitoneal ; Male ; Mice, Inbred C57BL ; Neurons/immunology ; Neurons/metabolism ; Neurotoxicity Syndromes/immunology ; Nuclear Receptor Subfamily 4, Group A, Member 2/genetics ; Primary Cell Culture ; Synucleinopathies/immunology ; Synucleinopathies/metabolism
Czasopismo naukowe

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