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Wyszukujesz frazę ""Niepmann ST"" wg kryterium: Autor


Wyświetlanie 1-2 z 2
Tytuł:
CX3CR1 is a prerequisite for the development of cardiac hypertrophy and left ventricular dysfunction in mice upon transverse aortic constriction.
Autorzy:
Weisheit CK; Department of Anesthesiology and Intensive Care Medicine, University Hospital Bonn, Bonn, Germany.
Kleiner JL; Department of Anesthesiology and Intensive Care Medicine, University Hospital Bonn, Bonn, Germany.
Rodrigo MB; Department of Anesthesiology and Intensive Care Medicine, University Hospital Bonn, Bonn, Germany.
Niepmann ST; Heart Center Bonn, Clinic for Internal Medicine II, University Hospital Bonn, Bonn, Germany.
Zimmer S; Heart Center Bonn, Clinic for Internal Medicine II, University Hospital Bonn, Bonn, Germany.
Duerr GD; Department of Cardiac Surgery, University Clinical Centre Bonn, Bonn, Germany.
Coburn M; Department of Anesthesiology and Intensive Care Medicine, University Hospital Bonn, Bonn, Germany.
Kurts C; Institute of Experimental Immunology, University Hospital Bonn, Bonn, Germany.
Frede S; Department of Anesthesiology and Intensive Care Medicine, University Hospital Bonn, Bonn, Germany.
Eichhorn L; Department of Anesthesiology and Intensive Care Medicine, University Hospital Bonn, Bonn, Germany.
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Źródło:
PloS one [PLoS One] 2021 Jan 07; Vol. 16 (1), pp. e0243788. Date of Electronic Publication: 2021 Jan 07 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Hypertrophy, Left Ventricular*/immunology
Hypertrophy, Left Ventricular*/metabolism
Ventricular Dysfunction, Left*/immunology
Ventricular Dysfunction, Left*/metabolism
Ventricular Remodeling*
CX3C Chemokine Receptor 1/*metabolism
Animals ; Cytokines/metabolism ; Disease Models, Animal ; Female ; Macrophages/metabolism ; Mice ; Mice, Inbred C57BL
Czasopismo naukowe
Tytuł:
Sodium thiocyanate treatment attenuates atherosclerotic plaque formation and improves endothelial regeneration in mice.
Autorzy:
Zietzer A; Medical Department II, University Hospital Bonn, Bonn, Germany.
Niepmann ST; Medical Department II, University Hospital Bonn, Bonn, Germany.
Camara B; Medical Department II, University Hospital Bonn, Bonn, Germany.
Lenart MA; Medical Department II, University Hospital Bonn, Bonn, Germany.
Jansen F; Medical Department II, University Hospital Bonn, Bonn, Germany.
Becher MU; Medical Department II, University Hospital Bonn, Bonn, Germany.
Andrié R; Medical Department II, University Hospital Bonn, Bonn, Germany.
Nickenig G; Medical Department II, University Hospital Bonn, Bonn, Germany.
Tiyerili V; Medical Department II, University Hospital Bonn, Bonn, Germany.
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Źródło:
PloS one [PLoS One] 2019 Apr 02; Vol. 14 (4), pp. e0214476. Date of Electronic Publication: 2019 Apr 02 (Print Publication: 2019).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Regeneration*
Endothelium, Vascular/*metabolism
Plaque, Atherosclerotic/*metabolism
Thiocyanates/*pharmacology
Animals ; Aorta/metabolism ; Atherosclerosis ; Blood Pressure ; Body Weight ; Carotid Arteries/pathology ; Granulocytes/metabolism ; Heart ; Heart Rate/drug effects ; Hydrogen Peroxide ; Mice ; Mice, Inbred C57BL ; Mice, Knockout, ApoE ; Neointima/pathology ; Neutrophils/metabolism ; Oxidative Stress ; Peroxidase/metabolism ; Plaque, Atherosclerotic/drug therapy ; Reactive Oxygen Species/metabolism ; Thiocyanates/metabolism ; Vasodilation/drug effects
Czasopismo naukowe
    Wyświetlanie 1-2 z 2

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