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Wyszukujesz frazę ""Nitric Oxide Synthase Type II metabolism"" wg kryterium: Temat


Tytuł :
Inflammation leads to distinct populations of extracellular vesicles from microglia
Autorzy :
Yang, Yiyi
Boza-Serrano, Antonio
Dunning, Christopher J. R.
Clausen, Bettina Hjelm
Lambertsen, Kate Lykke
Deierborg, Tomas
Pokaż więcej
Temat :
Microglia
Extracellular vesicles (EVs)
Neuroinflammation
TNF
lcsh:Neurology. Diseases of the nervous system
lcsh:RC346-429
Research
Cellular and Molecular Neuroscience
Neurology
Immunology
General Neuroscience
Tumor necrosis factor alpha
Western blot
medicine.diagnostic_test
medicine
Interleukin
Lipopolysaccharide
chemistry.chemical_compound
chemistry
medicine.anatomical_structure
Cytokine
medicine.medical_treatment
Inflammation
medicine.symptom
Cell biology
Microglia/drug effects
Protein Transport/drug effects
Nitric Oxide Synthase Type II/metabolism
Tumor Necrosis Factor-alpha/deficiency
Etanercept/pharmacology
Mice, Inbred C57BL
Infarction, Middle Cerebral Artery/complications
Male
Lipopolysaccharides/pharmacology
Signal Transduction/drug effects
Mice, Knockout
Animals
Inflammation/etiology
Immunosuppressive Agents/pharmacology
Mice
Cytokines/metabolism
Extracellular Vesicles/pathology
Cell Line, Transformed
Gene Expression Regulation/drug effects
Disease Models, Animal
Źródło :
Journal of Neuroinflammation, Vol 15, Iss 1, Pp 1-19 (2018)
Journal of Neuroinflammation
Crossref
Yang, Y, Boza-Serrano, A, Dunning, C J R, Clausen, B H, Lambertsen, K L & Deierborg, T 2018, ' Inflammation leads to distinct populations of extracellular vesicles from microglia ', Journal of Neuroinflammation, vol. 15, 168 . https://doi.org/10.1186/s12974-018-1204-7
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fa5b9f02a66c45696b513383261ae0bf
http://link.springer.com/article/10.1186/s12974-018-1204-7
Tytuł :
Aberrant up-regulation of iNOS/NO system is correlated with an increased abundance of Foxp3+ cells and reduced effector/memory cell markers expression during colorectal cancer: immunomodulatory effects of cetuximab combined with chemotherapy
Autorzy :
Chafia Touil-Boukoffa
Sonia Ait-Younes
Olivier Morales
Oussama Medjeber
Hassen Mahfouf
Hayet Rafa
Sabah Hetit
Nadira Delhem
Said Belhadef
Yvan de Launoit
Mourad Belkhelfa
Hayat Ait-Kaci
Sarra Benkhelifa
Pokaż więcej
Temat :
Host immune responses
Immunomodulation
iNOS/NO system
Cetuximab and chemotherapy
Colorectal Cancer
MESH: Adult
MESH: Aged
MESH: Forkhead Transcription Factors/metabolism
MESH: Humans
MESH: Immunologic Factors/metabolism
MESH: Male
MESH: Middle Aged
MESH: Nitric Oxide/metabolism
MESH: Nitric Oxide Synthase Type II/metabolism
MESH: T-Lymphocytes, Regulatory/drug effects
MESH: Up-Regulation/drug effects
MESH: Up-Regulation/physiology
MESH: Aged, 80 and over
MESH: Biomarkers, Tumor/metabolism
MESH: Cetuximab/therapeutic use
MESH: Colorectal Neoplasms/drug therapy
MESH: Colorectal Neoplasms/metabolism
MESH: Down-Regulation/drug effects
MESH: Down-Regulation/physiology
MESH: Female
[SDV]Life Sciences [q-bio]
[SDV.CAN]Life Sciences [q-bio]/Cancer
Pharmacology (medical)
Pharmacology
Immunology
Chemotherapy
medicine.medical_treatment
medicine
FOXP3
Immune system
Memory T cell
medicine.anatomical_structure
Colorectal cancer
medicine.disease
Cancer research
Biomarker (medicine)
Inflammation
medicine.symptom
Cetuximab
medicine.drug
business.industry
business
Źródło :
Inflammopharmacology
Inflammopharmacology, Springer Verlag, 2019, 27 (4), pp.685-700. ⟨10.1007/s10787-019-00566-9⟩
Crossref
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::90c14e2c54400f0d81e84b20996df275
https://hal.archives-ouvertes.fr/hal-02391591
Tytuł :
Pregabalin Ameliorates Lipopolysaccharide-Induced Pancreatic Inflammation in Aged Rats
Autorzy :
Ozmen, O.
Topsakal, Şenay
Pokaż więcej
Temat :
Animals
Anti-Inflammatory Agents/*pharmacology
Caspase 3/metabolism
Cytoprotection
Disease Models, Animal
Female
Granulocyte Colony-Stimulating Factor/metabolism
Inflammation Mediators/metabolism
Interleukin-6/metabolism
Lipopolysaccharides
Nitric Oxide Synthase Type II/metabolism
Pancreas/*drug effects/metabolism/pathology
Pancreatitis/blood/chemically induced/pathology/*prevention & control
Pregabalin/*pharmacology
Rats, Wistar
Serum Amyloid A Protein/metabolism
Signal Transdu
Immunology and Allergy
Endocrinology, Diabetes and Metabolism
Lipopolysaccharide
chemistry.chemical_compound
chemistry
Internal medicine
medicine.medical_specialty
medicine
Sepsis
medicine.disease
business.industry
business
Granulocyte
medicine.anatomical_structure
Pathogenesis
Pancreas
Pregabalin
medicine.drug
Endocrinology
Nitric oxide synthase
biology.protein
biology
Endocrine system
Immunohistochemistry
Pathology
Rat
amylase
caspase 3
glucose
granulocyte colony stimulating factor
inducible nitric oxide synthase
interleukin 6
triacylglycerol lipase
antiinflammatory agent
autacoid
Casp3 protein, rat
Il6 protein, rat
Nos2 protein, rat
serum amyloid A
aging
animal experiment
animal model
animal tissue
Article
biochemical analysis
cell infiltration
controlled study
female
histopathology
immune response
leucocyte infiltration
neutrophil chemotaxis
nonhuman
pancreatitis
protein expression
sepsis
single drug dose
animal
blood
cell protection
disease model
drug effect
metabolism
signal transduction
Wistar rat
Anti-Inflammatory Agents
Granulocyte Colony-Stimulating Factor
Inflammation Mediators
Interleukin-6
Nitric Oxide Synthase Type II
Serum Amyloid A Protein
Źródło :
Crossref
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::23f9114be52657faf470b2e0452280f8
Tytuł :
All-Trans Retinoic Acid Modulates TLR4/NF-κB Signaling Pathway Targeting TNF-αand Nitric Oxide Synthase 2 Expression in Colonic Mucosa during Ulcerative Colitis and Colitis Associated Cancer
Autorzy :
Rafa, Hayet
Benkhelifa, Sarra
AitYounes, Sonia
Saoula, Houria
Belhadef, Said
Belkhelfa, Mourad
Boukercha, Aziza
Toumi, Ryma
Soufli, Imene
Moralès, Olivier
de Launoit, Yvan
Mahfouf, Hassen
Nakmouche, M’hamed
Delhem, Nadira
Touil-Boukoffa, Chafia
Pokaż więcej
Temat :
[SDV.CAN]Life Sciences [q-bio]/Cancer
MESH: Aged
MESH: Blotting, Western
MESH: Colitis/blood
MESH: Colitis, Ulcerative/blood
MESH: Colitis, Ulcerative/metabolism
MESH: Colorectal Neoplasms/blood
MESH: Colorectal Neoplasms/metabolism
MESH: Enzyme-Linked Immunosorbent Assay
MESH: Female
MESH: Humans
MESH: Immunohistochemistry
MESH: Intestinal Mucosa/metabolism
MESH: Male
MESH: Middle Aged
MESH: Nitric Oxide/blood
MESH: Nitric Oxide Synthase Type II/metabolism
MESH: Real-Time Polymerase Chain Reaction
MESH: Tumor Necrosis Factor-alpha/blood
[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry, Molecular Biology/Genomics [q-bio.GN]
[SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterology
Research Article
lcsh:Pathology
lcsh:RB1-214
Article Subject
Cell Biology
Immunology
Cancer research
Signal transduction
Colorectal cancer
medicine.disease
medicine
Tumor necrosis factor alpha
Ulcerative colitis
Biology
Retinoic acid
chemistry.chemical_compound
chemistry
Ex vivo
TLR4
Nitric oxide synthase 2
biology.protein
Źródło :
Mediators of Inflammation
Mediators of Inflammation, Hindawi Publishing Corporation, 2017, 2017, pp.1-16. ⟨10.1155/2017/7353252⟩
Mediators of Inflammation, Hindawi Publishing Corporation, 2017, 2017, pp.7353252. ⟨10.1155/2017/7353252⟩
Mediators of Inflammation, Vol 2017 (2017)
Crossref
Opis pliku :
text/xhtml
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::61aabb23e503ebb00e3a943766bdaf55
https://hal.archives-ouvertes.fr/hal-03059784
Tytuł :
Evaluation of anti-proliferative and anti-inflammatory activities of Pelagia noctiluca venom in Lipopolysaccharide/Interferon-γ stimulated RAW264.7 macrophages
Autorzy :
Ayed, Yosra
Sghaier, Rabiaa Manel
Laouini, Dhafer
Bacha, Hassen
Pokaż więcej
Temat :
[ SDV ] Life Sciences [q-bio]
Venom
Pelagia noctiluca
Nitric oxide
MTT assay
Cell proliferation
Anti-inflammatory activity
MESH: Animals
MESH: Anti-Inflammatory Agents/isolation & purification
MESH: Macrophages/drug effects
MESH: Macrophages/metabolism
MESH: Anti-Inflammatory Agents/pharmacology
MESH: Mice
MESH: Nitric Oxide Synthase Type II/antagonists & inhibitors
MESH: Nitric Oxide Synthase Type II/metabolism
MESH: Cell Line
MESH: Cell Proliferation/drug effects
MESH: Cell Proliferation/physiology
MESH: Cell Survival/drug effects
MESH: Dose-Response Relationship, Drug
MESH: Cell Survival/physiology
MESH: Cnidarian Venoms/isolation & purification
MESH: Cnidarian Venoms/pharmacology
MESH: Drug Evaluation, Preclinical/methods
MESH: Humans
MESH: Interferon-gamma/toxicity
MESH: K562 Cells
MESH: Leukocytes, Mononuclear/drug effects
MESH: Leukocytes, Mononuclear/metabolism
MESH: Lipopolysaccharides/toxicity
[SDV]Life Sciences [q-bio]
[SDV.SP]Life Sciences [q-bio]/Pharmaceutical sciences
Źródło :
Biomedicine and Pharmacotherapy
Biomedicine and Pharmacotherapy, Elsevier Masson, 2016, 84, pp.1986-1991. 〈10.1016/j.biopha.2016.11.010 〉
Biomedicine and Pharmacotherapy, Elsevier Masson, 2016, 84, pp.1986-1991. ⟨10.1016/j.biopha.2016.11.010⟩
Dostępność :
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::c7649e6147030f8328541f8af88684e2
https://hal-riip.archives-ouvertes.fr/pasteur-01455071
Tytuł :
IL-23/IL-17A Axis Correlates with the Nitric Oxide Pathway in Inflammatory Bowel Disease: Immunomodulatory Effect of Retinoic Acid
Autorzy :
Rafa, Hayet
Saoula, Houria
Belkhelfa, Mourad
Medjeber, Oussama
Soufli, Imene
Toumi, Ryma
De Launoit, Yvan
Moralès, Olivier
Nakmouche, M'hamed
Delhem, Nadira
Touil-Boukoffa, Chafia
Pokaż więcej
Temat :
MESH: Adult
MESH: Algeria
MESH: Interleukin-17/metabolism
MESH: Interleukin-23/blood
MESH: Interleukin-23/metabolism
MESH: Interleukin-6/blood
MESH: Interleukin-6/metabolism
MESH: Male
MESH: Middle Aged
MESH: Nitric Oxide/metabolism
MESH: Nitric Oxide Synthase Type II/genetics
MESH: Nitric Oxide Synthase Type II/metabolism
MESH: Cells, Cultured
MESH: Receptors, Cytokine/genetics
MESH: Receptors, Cytokine/metabolism
MESH: STAT3 Transcription Factor/genetics
MESH: STAT3 Transcription Factor/metabolism
MESH: Signal Transduction/drug effects
MESH: T-Lymphocyte Subsets/drug effects
MESH: T-Lymphocyte Subsets/immunology
MESH: Th17 Cells/drug effects
MESH: Th17 Cells/immunology
MESH: Tretinoin/therapeutic use
MESH: Female
MESH: Young Adult
MESH: Gene Expression Regulation/drug effects
MESH: Humans
MESH: Immunomodulation
MESH: Inflammatory Bowel Diseases/drug therapy
MESH: Inflammatory Bowel Diseases/immunology
MESH: Interleukin-17/blood
[SDV]Life Sciences [q-bio]
[SDV.CAN]Life Sciences [q-bio]/Cancer
Virology
Cell Biology
Immunology
Interleukin
Biology
Proinflammatory cytokine
Peripheral blood mononuclear cell
Cytokine
medicine.medical_treatment
medicine
STAT3
biology.protein
Inflammatory bowel disease
medicine.disease
Pathogenesis
Immune dysregulation
medicine.disease_cause
Źródło :
Journal of Interferon and Cytokine Research
Journal of Interferon and Cytokine Research, Mary Ann Liebert, 2013, 33 (7), pp.355-368. ⟨10.1089/jir.2012.0063⟩
Crossref
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a681e8219310a30fbf46f18c46e3e251
https://hal.archives-ouvertes.fr/hal-02395835
Tytuł :
Membrane-bound TNF induces protective immune responses to M. bovis BCG infection: regulation of memTNF and TNF receptors comparing two memTNF molecules
Autorzy :
Ruth Bisig
Sonia Schuepbach-Mallepell
Irene Garcia
Olivier Gaide
Maria L. Olleros
Dominique Vesin
George Kollias
Marie-Laure Santiago-Raber
Pokaż więcej
Temat :
ddc:616.07
Animals
Bone Marrow Cells/cytology
Cell Membrane/metabolism
Chemokines/metabolism
Disease Resistance/immunology
Gene Knock-In Techniques
Interferon-gamma/immunology/metabolism
Intracellular Space/immunology/metabolism/microbiology
Macrophages/immunology/metabolism/microbiology
Mice
Mice, Inbred C57BL
Mycobacterium bovis/immunology/pathogenicity
NF-kappa B/metabolism
Nitric Oxide/metabolism
Nitric Oxide Synthase Type II/metabolism
Receptors, Tumor Necrosis Factor, Type II/chemistry/metabolism
Solubility
Tuberculosis/immunology/veterinary
Tumor Necrosis Factor-alpha/genetics/metabolism
lcsh:Medicine
lcsh:R
lcsh:Science
lcsh:Q
Multidisciplinary
Interferon gamma
medicine.drug
medicine
Cell biology
Inflammation
medicine.symptom
NFKB1
Tumor necrosis factor alpha
Biology
Immune system
Receptor
Immunology
Intracellular
Chemokine
biology.protein
Research Article
Immune System
Cytokines
Immunity
Adaptive Immunity
Innate Immunity
Immune Response
Medicine
Infectious Diseases
Bacterial Diseases
Tuberculosis
Bovine Tuberculosis in Humans
Infectious Disease Control
Źródło :
PLOS ONE, Vol. 7, No 5 (2012) P. e31469
PLoS ONE, Vol 7, Iss 5, p e31469 (2012)
Crossref
PLoS ONE
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1cd404e1fd097bea881eea6444c804f4
https://archive-ouverte.unige.ch/unige:32287
Tytuł :
CX(3)CR1(+) CD115(+) CD135(+) common macrophage/DC precursors and the role of CX(3)CR1 in their response to inflammation
Autorzy :
Auffray, Cedric
Fogg, Darin K.
Narni-Mancinelli, Emilie
Senechal, Brigitte
Trouillet, Celine
Saederup, Noah
Leemput, Julia
Bigot, Karine
Campisi, Laura
Abitbol, Marc
Molina, Thierry
Charo, Israel
Hume, David A.
Cumano, Ana
Lauvau, Gregoire
Geissmann, Frederic
Pokaż więcej
Temat :
Animals
Bone Marrow Cells/cytology
Bone Marrow Cells/immunology
Cell Differentiation
Cell Movement
Cell Proliferation
Cell Survival
Dendritic Cells/cytology
Dendritic Cells/enzymology
Dendritic Cells/immunology
Inflammation/enzymology
Inflammation/immunology
Listeria monocytogenes
Listeriosis/immunology
Macrophages/cytology
Macrophages/enzymology
Macrophages/immunology
Mice
Monocytes/cytology
Monocytes/immunology
Nitric Oxide Synthase Type II/metabolism
Phenotype
Reactive Oxygen Species/metabolism
Receptor, Macrophage Colony-Stimulating Factor/metabolism
Receptors, Chemokine/deficiency
Receptors, Chemokine/metabolism
Spleen/cytology
Spleen
Stem Cells
Tumor Necrosis Factor-alpha/biosynthesis
fms-Like Tyrosine Kinase 3/metabolism
Immunology
Immunology and Allergy
Article
MESH: Animals
MESH: Bone Marrow Cells
MESH: Macrophages
MESH: Mice
MESH: Monocytes
MESH: Nitric Oxide Synthase Type II
MESH: Phenotype
MESH: Reactive Oxygen Species
MESH: Receptor, Macrophage Colony-Stimulating Factor
MESH: Receptors, Chemokine
MESH: Spleen
MESH: Stem Cells
MESH: Cell Differentiation
MESH: Tumor Necrosis Factor-alpha
MESH: fms-Like Tyrosine Kinase 3
MESH: Cell Movement
MESH: Cell Proliferation
MESH: Cell Survival
MESH: Dendritic Cells
MESH: Inflammation
MESH: Listeria Infections
MESH: Listeria monocytogenes
[SDV.IMM]Life Sciences [q-bio]/Immunology
MESH : Animals
MESH : Bone Marrow Cells
MESH : Macrophages
MESH : Mice
MESH : Monocytes
MESH : Nitric Oxide Synthase Type II
MESH : Phenotype
MESH : Reactive Oxygen Species
MESH : Receptor, Macrophage Colony-Stimulating Factor
MESH : Receptors, Chemokine
MESH : Spleen
MESH : Stem Cells
MESH : Cell Differentiation
MESH : Tumor Necrosis Factor-alpha
MESH : fms-Like Tyrosine Kinase 3
MESH : Cell Movement
MESH : Cell Proliferation
MESH : Cell Survival
MESH : Dendritic Cells
MESH : Inflammation
MESH : Listeria Infections
MESH : Listeria monocytogenes
[ SDV.IMM ] Life Sciences [q-bio]/Immunology
Stem cell
Biology
Fms-Like Tyrosine Kinase 3
Tumor necrosis factor alpha
Inflammation
medicine.symptom
medicine
Myeloid
medicine.anatomical_structure
CX3CR1
Dendritic cell
Cell biology
hemic and immune systems
Źródło :
Auffray, C, Fogg, D K, Narni-Mancinelli, E, Senechal, B, Trouillet, C, Saederup, N, Leemput, J, Bigot, K, Campisi, L, Abitbol, M, Molina, T, Charo, I, Hume, D A, Cumano, A, Lauvau, G & Geissmann, F 2009, ' CX(3)CR1(+) CD115(+) CD135(+) common macrophage/DC precursors and the role of CX(3)CR1 in their response to inflammation ', Journal of Experimental Medicine, vol. 206, no. 3, pp. 595-606 . https://doi.org/10.1084/jem.20081385
The Journal of Experimental Medicine
Journal of Experimental Medicine
Journal of Experimental Medicine, Rockefeller University Press, 2009, 206 (3), pp.595-606. ⟨10.1084/jem.20081385⟩
The Journal of Experimental Medecine
The Journal of Experimental Medecine, The Rockefeller University Press, 2009, 206 (3), pp.595-606. 〈10.1084/jem.20081385〉
Crossref
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2f67d62a1cd1a75779217f556259f301
https://www.pure.ed.ac.uk/ws/files/12572592/J_Exp_Med_2009_Auffray_pdf.pdf
Tytuł :
Effects of the PPAR-beta agonist GW501516 in an in vitro model of brain inflammation and antibody-induced demyelination
Autorzy :
Defaux, A.
Zurich, M.G.
Braissant, O.
Honegger, P.
Monnet-Tschudi, F.
Pokaż więcej
Temat :
Animals
Anti-Inflammatory Agents/pharmacology
Anti-Inflammatory Agents/therapeutic use
Antibodies/immunology
Astrocytes/cytology
Astrocytes/metabolism
Brain/cytology
Cells, Cultured
Demyelinating Diseases/drug therapy
Demyelinating Diseases/immunology
Encephalitis/drug therapy
Encephalitis/immunology
Encephalomyelitis, Autoimmune, Experimental/drug therapy
Encephalomyelitis, Autoimmune, Experimental/immunology
Glial Fibrillary Acidic Protein/genetics
Glial Fibrillary Acidic Protein/metabolism
Interferon-gamma/immunology
Interferon-gamma/pharmacology
Interleukin-6/genetics
Interleukin-6/metabolism
Lipopolysaccharides/immunology
Lipopolysaccharides/pharmacology
Microglia/cytology
Microglia/metabolism
Myelin Basic Proteins/genetics
Myelin Basic Proteins/metabolism
Neurofilament Proteins/genetics
Neurofilament Proteins/metabolism
Nitric Oxide Synthase Type II/genetics
Nitric Oxide Synthase Type II/metabolism
PPAR gamma/genetics
PPAR gamma/metabolism
PPAR-beta/agonists
PPAR-beta/genetics
Rats
Thiazoles/pharmacology
Thiazoles/therapeutic use
Tumor Necrosis Factor-alpha/genetics
Tumor Necrosis Factor-alpha/metabolism
Źródło :
Journal of Neuroinflammation, vol. 6, pp. 15
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=od______1900::83e5e22f33d355d34cdbd2f28badca79
https://serval.unil.ch/notice/serval:BIB_A74CAED83BA4
Tytuł :
Low- and high-density lipoproteins modulate function, apoptosis, and proliferation of primary human and murine pancreatic beta-cells
Autorzy :
Rütti, Sabine
Ehses, Jan A.
Sibler, Rahel A.
Prazak, Richard
Rohrer, Lucia
Georgopoulos, Spiros
Meier, Daniel T.
Niclauss, Nadja
Berney, Thierry
Donath, Marc Y.
von Eckardstein, Arnold
Pokaż więcej
Temat :
ddc:617
Animals
Antigens, CD95/metabolism
Apolipoprotein A-I/metabolism
Apoptosis
CASP8 and FADD-Like Apoptosis Regulating Protein/metabolism
Cell Proliferation
Cell Survival
Cells, Cultured
Female
Glucose/metabolism
Humans
Insulin/secretion
Insulin-Secreting Cells/physiology
Interleukin-1beta/metabolism
Lipoproteins, HDL/physiology
Lipoproteins, LDL/physiology
Lysophospholipids/metabolism
Male
Mice
Mice, Inbred C57BL
Mice, Knockout
Middle Aged
Nitric Oxide Synthase Type II/metabolism
Receptors, LDL/metabolism
Scavenger Receptors, Class B/metabolism
Sphingosine/analogs & derivatives/metabolism
Clinic for Endocrinology and Diabetology
Center for Integrative Human Physiology
Institute of Clinical Chemistry
570 Life sciences
biology
610 Medicine & health
540 Chemistry
Endocrinology
Programmed cell death
Apolipoprotein A1
biology.protein
medicine.medical_specialty
medicine
Islet
geography.geographical_feature_category
geography
Sphingolipid
Internal medicine
Lipoprotein
Scavenger receptor
LDL receptor
lipids (amino acids, peptides, and proteins)
Źródło :
Endocrinology, Vol. 150, No 10 (2009) pp. 4521-30
Rütti, S; Ehses, J A; Sibler, Rahel; Prazak, R; Rohrer, L; Georgopoulos, S; Meier, D T; Niclauss, N; Berney, T; Donath, M Y; von Eckardstein, Arnold (2009). Low-and high-density lipoproteins modulate function, apoptosis, and proliferation of primary human and murine pancreatic beta-cells. Endocrinology, 150(10):4521-4530.
Crossref
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::25513697d42dc2ebe84188aa87b57fd7
Tytuł :
PARP1 is required for adhesion molecule expression in atherogenesis
Autorzy :
Giovanni G. Camici
Jan Borén
Tobias von Lukowicz
Christian M. Matter
Paul O. Hassa
Monika Gersbach
Christine Lohmann
Felix C. Tanner
Michael O. Hottiger
Thomas F. Lüscher
Barbara E. Stähli
Vincent Braunersreuther
François Mach
Bernhard Odermatt
Pokaż więcej
Temat :
ddc:616
Animals
Apolipoproteins E/genetics/metabolism
Atherosclerosis/enzymology/immunology/pathology/prevention & control
Cell Adhesion Molecules/metabolism
Cholesterol/blood
Disease Models, Animal
E-Selectin/metabolism
Enzyme Inhibitors/pharmacology
Inflammation/enzymology/immunology/pathology/prevention & control
Inflammation Mediators/blood
Macrophages/pathology
Male
Mice
Mice, Inbred C57BL
Mice, Knockout
Necrosis
Nitric Oxide Synthase Type II/metabolism
P-Selectin/metabolism
Phenanthrenes/pharmacology
Poly(ADP-ribose) Polymerases/antagonists & inhibitors/genetics/metabolism
T-Lymphocytes/pathology
Vascular Cell Adhesion Molecule-1/metabolism
Physiology (medical)
Cardiology and Cardiovascular Medicine
Physiology
Immunology
Apolipoprotein E
E-selectin
biology.protein
biology
Endothelial activation
Cancer research
Cell adhesion
Inflammation
medicine.symptom
medicine
Cell adhesion molecule
PARP1
Nitric oxide synthase
Department of Molecular Mechanisms of Disease
Center for Integrative Human Physiology
Institute of Pathology and Molecular Pathology
Clinic for Cardiology
Institute of Physiology
570 Life sciences
610 Medicine & health
Źródło :
Cardiovascular Research, Vol. 78, No 1 (2008) pp. 158-66
Crossref
von Lukowicz, T; Hassa, P O; Lohmann, C; Borén, J; Braunersreuther, V; Mach, F; Odermatt, B; Gersbach, M; Camici, G G; Stähli, B E; Tanner, F C; Hottiger, Michael O; Lüscher, T F; Matter, C M (2008). PARP1 is required for adhesion molecule expression in atherogenesis. Cardiovascular Research, 78(1):158-166.
Opis pliku :
application/pdf
Dostępność :
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f2287753e12011f9dbe9d333930ba633

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