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Wyświetlanie 61-80 z 1406
Tytuł:
Neuronal, but not glial, Contactin 2 negatively regulates axon regeneration in the injured adult optic nerve.
Autorzy:
Savvaki M; Department of Basic Science, Faculty of Medicine, University of Crete, Crete, Greece.; Institute of Molecular Biology & Biotechnology - FoRTH, Heraklion, Crete, Greece.
Kafetzis G; Department of Biology, University of Crete, Crete, Greece.; School of Life Sciences, University of Sussex, Brighton, UK.
Kaplanis SI; Department of Basic Science, Faculty of Medicine, University of Crete, Crete, Greece.; Institute of Molecular Biology & Biotechnology - FoRTH, Heraklion, Crete, Greece.
Ktena N; Department of Basic Science, Faculty of Medicine, University of Crete, Crete, Greece.; Institute of Molecular Biology & Biotechnology - FoRTH, Heraklion, Crete, Greece.
Theodorakis K; Institute of Molecular Biology & Biotechnology - FoRTH, Heraklion, Crete, Greece.
Karagogeos D; Department of Basic Science, Faculty of Medicine, University of Crete, Crete, Greece.; Institute of Molecular Biology & Biotechnology - FoRTH, Heraklion, Crete, Greece.
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Źródło:
The European journal of neuroscience [Eur J Neurosci] 2021 Mar; Vol. 53 (6), pp. 1705-1721. Date of Electronic Publication: 2021 Mar 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Axons*
Optic Nerve Injuries*
Animals ; Contactin 2 ; Mice ; Nerve Regeneration ; Neurons ; Optic Nerve
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
MEF2 transcription factors differentially contribute to retinal ganglion cell loss after optic nerve injury.
Autorzy:
Xia X; Mary M. and Sash A. Spencer Center for Vision Research, Department of Ophthalmology, Byers Eye Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.
Yu CY; Mary M. and Sash A. Spencer Center for Vision Research, Department of Ophthalmology, Byers Eye Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.
Bian M; Mary M. and Sash A. Spencer Center for Vision Research, Department of Ophthalmology, Byers Eye Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.
Sun CB; Mary M. and Sash A. Spencer Center for Vision Research, Department of Ophthalmology, Byers Eye Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.
Tanasa B; Mary M. and Sash A. Spencer Center for Vision Research, Department of Ophthalmology, Byers Eye Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.
Chang KC; Mary M. and Sash A. Spencer Center for Vision Research, Department of Ophthalmology, Byers Eye Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.
Bruffett DM; Mary M. and Sash A. Spencer Center for Vision Research, Department of Ophthalmology, Byers Eye Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.
Thakur H; Mary M. and Sash A. Spencer Center for Vision Research, Department of Ophthalmology, Byers Eye Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.
Shah SH; Mary M. and Sash A. Spencer Center for Vision Research, Department of Ophthalmology, Byers Eye Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.
Knasel C; Mary M. and Sash A. Spencer Center for Vision Research, Department of Ophthalmology, Byers Eye Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.
Cameron EG; Mary M. and Sash A. Spencer Center for Vision Research, Department of Ophthalmology, Byers Eye Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.
Kapiloff MS; Mary M. and Sash A. Spencer Center for Vision Research, Department of Ophthalmology, Byers Eye Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.; Department of Medicine and Stanford Cardiovascular Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.
Goldberg JL; Mary M. and Sash A. Spencer Center for Vision Research, Department of Ophthalmology, Byers Eye Institute, Stanford University School of Medicine, Palo Alto, CA, United States of America.
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Źródło:
PloS one [PLoS One] 2020 Dec 14; Vol. 15 (12), pp. e0242884. Date of Electronic Publication: 2020 Dec 14 (Print Publication: 2020).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
MEF2 Transcription Factors/*metabolism
Optic Nerve Injuries/*metabolism
Optic Nerve Injuries/*pathology
Retinal Ganglion Cells/*pathology
Alleles ; Animals ; Cell Count ; Humans ; MEF2 Transcription Factors/deficiency ; MEF2 Transcription Factors/genetics ; Mice ; Optic Nerve Injuries/genetics ; Point Mutation ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Mesoscopic cortical network reorganization during recovery of optic nerve injury in GCaMP6s mice.
Autorzy:
Groleau M; Laboratoire de Neurobiologie de la Cognition Visuelle, École d'Optométrie, Université de Montréal, Montréal, QC, H3T 1P1, Canada.
Nazari-Ahangarkolaee M; Department of Neuroscience, Canadian Centre for Behavioural Neuroscience (CCBN), University of Lethbridge, Lethbridge, AB, T1K 3M4, Canada.
Vanni MP; Laboratoire de Neurophotonique, École d'Optométrie, Université de Montréal, Montréal, QC, H3T 1P1, Canada.
Higgins JL; Laboratoire de Neurobiologie de la Cognition Visuelle, École d'Optométrie, Université de Montréal, Montréal, QC, H3T 1P1, Canada.
Vézina Bédard AS; Laboratoire de Neurobiologie de la Cognition Visuelle, École d'Optométrie, Université de Montréal, Montréal, QC, H3T 1P1, Canada.
Sabel BA; Institute of Medical Psychology, Medical Faculty, Otto-V.-Guericke University of Magdeburg, 39120, Magdeburg, Germany.
Mohajerani MH; Department of Neuroscience, Canadian Centre for Behavioural Neuroscience (CCBN), University of Lethbridge, Lethbridge, AB, T1K 3M4, Canada. .
Vaucher E; Laboratoire de Neurobiologie de la Cognition Visuelle, École d'Optométrie, Université de Montréal, Montréal, QC, H3T 1P1, Canada. .
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Źródło:
Scientific reports [Sci Rep] 2020 Dec 08; Vol. 10 (1), pp. 21472. Date of Electronic Publication: 2020 Dec 08.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Nerve Net/*physiopathology
Optic Nerve/*physiopathology
Optic Nerve Injuries/*physiopathology
Visual Cortex/*physiopathology
Animals ; Calcium/analysis ; Disease Models, Animal ; Female ; Male ; Mice, Inbred C57BL ; Nerve Crush ; Nerve Net/pathology ; Optic Nerve/pathology ; Optic Nerve Injuries/pathology ; Optic Nerve Injuries/therapy ; Visual Acuity ; Visual Cortex/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Neurofilament light chain as biomarker in idiopathic intracranial hypertension.
Autorzy:
Beier D; Department of Neurology, Odense University Hospital, Odense, Denmark.; Department of Clinical Research, University of Southern Denmark, Odense, Denmark.; OPEN, Odense Patient Data Explorative Network, Odense University Hospital, Odense, Denmark.
Korsbæk JJ; Danish Headache Center, Neurological Clinic, Rigshospitalet-Glostrup, Glostrup, Denmark.; University of Copenhagen, Copenhagen, Denmark.
Madsen JS; Department of Biochemistry and Immunology, Lillebaelt Hospital, University Hospital of Southern Denmark, Vejle, Denmark.; Department of Regional Health Research, Faculty of Health Sciences, University of Southern Denmark, Odense, Denmark.
Olsen DA; Department of Biochemistry and Immunology, Lillebaelt Hospital, University Hospital of Southern Denmark, Vejle, Denmark.
Molander LD; Department of Ophthalmology, Odense University Hospital, Odense, Denmark.
Hagen SM; Department of Ophthalmology, Rigshospitalet-Glostrup, Glostrup, Denmark.
Teunissen C; Department of Clinical Chemistry, Amsterdam University Medical Center, Amsterdam, the Netherlands.
Beier CP; Department of Neurology, Odense University Hospital, Odense, Denmark.; Department of Clinical Research, University of Southern Denmark, Odense, Denmark.
Jensen RH; Danish Headache Center, Neurological Clinic, Rigshospitalet-Glostrup, Glostrup, Denmark.; University of Copenhagen, Copenhagen, Denmark.
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Źródło:
Cephalalgia : an international journal of headache [Cephalalgia] 2020 Oct; Vol. 40 (12), pp. 1346-1354. Date of Electronic Publication: 2020 Jul 29.
Typ publikacji:
Journal Article; Observational Study
MeSH Terms:
Biomarkers/*cerebrospinal fluid
Neurofilament Proteins/*cerebrospinal fluid
Optic Nerve Injuries/*cerebrospinal fluid
Optic Nerve Injuries/*diagnosis
Pseudotumor Cerebri/*complications
Adult ; Female ; Humans ; Male ; Middle Aged ; Optic Nerve Injuries/etiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Topical ripasudil stimulates neuroprotection and axon regeneration in adult mice following optic nerve injury.
Autorzy:
Nishijima E; Visual Research Project, Tokyo Metropolitan Institute of Medical Science, 2-1-6 Kamikitazawa, Setagaya-ku, Tokyo, 156-8506, Japan.; Department of Ophthalmology, The Jikei University School of Medicine, Tokyo, Japan.
Namekata K; Visual Research Project, Tokyo Metropolitan Institute of Medical Science, 2-1-6 Kamikitazawa, Setagaya-ku, Tokyo, 156-8506, Japan. .
Kimura A; Visual Research Project, Tokyo Metropolitan Institute of Medical Science, 2-1-6 Kamikitazawa, Setagaya-ku, Tokyo, 156-8506, Japan.
Guo X; Visual Research Project, Tokyo Metropolitan Institute of Medical Science, 2-1-6 Kamikitazawa, Setagaya-ku, Tokyo, 156-8506, Japan.
Harada C; Visual Research Project, Tokyo Metropolitan Institute of Medical Science, 2-1-6 Kamikitazawa, Setagaya-ku, Tokyo, 156-8506, Japan.
Noro T; Visual Research Project, Tokyo Metropolitan Institute of Medical Science, 2-1-6 Kamikitazawa, Setagaya-ku, Tokyo, 156-8506, Japan.; Department of Ophthalmology, The Jikei University School of Medicine, Tokyo, Japan.
Nakano T; Department of Ophthalmology, The Jikei University School of Medicine, Tokyo, Japan.
Harada T; Visual Research Project, Tokyo Metropolitan Institute of Medical Science, 2-1-6 Kamikitazawa, Setagaya-ku, Tokyo, 156-8506, Japan. .
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Źródło:
Scientific reports [Sci Rep] 2020 Sep 24; Vol. 10 (1), pp. 15709. Date of Electronic Publication: 2020 Sep 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Axons/*drug effects
Isoquinolines/*therapeutic use
Nerve Regeneration/*drug effects
Neuroprotective Agents/*therapeutic use
Optic Nerve Injuries/*drug therapy
Sulfonamides/*therapeutic use
Animals ; Axons/physiology ; Isoquinolines/administration & dosage ; MAP Kinase Signaling System/drug effects ; Mice ; Nerve Crush ; Nerve Regeneration/physiology ; Neuroprotective Agents/administration & dosage ; Optic Nerve Injuries/metabolism ; Optic Nerve Injuries/physiopathology ; Phosphorylation/drug effects ; Retinal Ganglion Cells/drug effects ; Retinal Ganglion Cells/metabolism ; Sulfonamides/administration & dosage
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Dimethyl Fumarate Promotes the Survival of Retinal Ganglion Cells after Optic Nerve Injury, Possibly through the Nrf2/HO-1 Pathway.
Autorzy:
Mori S; Division of Ophthalmology, Department of Surgery, Kobe University Graduate School of Medicine, 7-5-2 Kusunoki-cho, Chuo-ku, Kobe 650-0017, Japan.
Kurimoto T; Division of Ophthalmology, Department of Surgery, Kobe University Graduate School of Medicine, 7-5-2 Kusunoki-cho, Chuo-ku, Kobe 650-0017, Japan.
Maeda H; Maeda Eye Clinic, 1-1-1 Uchihonmachi, Chuo-ku, Osaka 540-0012, Japan.
Nakamura M; Division of Ophthalmology, Department of Surgery, Kobe University Graduate School of Medicine, 7-5-2 Kusunoki-cho, Chuo-ku, Kobe 650-0017, Japan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Dec 30; Vol. 22 (1). Date of Electronic Publication: 2020 Dec 30.
Typ publikacji:
Journal Article
MeSH Terms:
Dimethyl Fumarate/*pharmacology
Heme Oxygenase-1/*metabolism
NF-E2-Related Factor 2/*metabolism
Optic Nerve Injuries/*metabolism
Retinal Ganglion Cells/*drug effects
Animals ; Blotting, Western ; Cell Survival/drug effects ; Disease Models, Animal ; Electroretinography ; Immunohistochemistry ; Male ; Mice, Inbred C57BL ; Optic Nerve Injuries/physiopathology ; Retinal Ganglion Cells/cytology ; Retinal Ganglion Cells/metabolism ; Signal Transduction/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Parabiosis reveals the correlation between the recruitment of circulating antigen presenting cells to the retina and the induction of spontaneous autoimmune uveoretinitis.
Autorzy:
McPherson, Scott W. (AUTHOR)
Heuss, Neal D. (AUTHOR)
Abedin, Md. (AUTHOR)
Roehrich, Heidi (AUTHOR)
Pierson, Mark J. (AUTHOR)
Gregerson, Dale S. (AUTHOR)
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Źródło:
Journal of Neuroinflammation. 12/9/2022, Vol. 19 Issue 1, p1-17. 17p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Partial optic nerve avulsion: A diagnostic challenge.
Autorzy:
Chia MA; Lions Eye Institute, Nedlands, Western Australia, Australia.
Shah VH; Lions Eye Institute, Nedlands, Western Australia, Australia.
Turner AW; Lions Eye Institute, Nedlands, Western Australia, Australia.
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Źródło:
Clinical & experimental ophthalmology [Clin Exp Ophthalmol] 2020 Jul; Vol. 48 (5), pp. 701-702. Date of Electronic Publication: 2020 Mar 16.
Typ publikacji:
Letter
MeSH Terms:
Eye Injuries*
Optic Nerve Injuries*/diagnosis
Optic Nerve Injuries*/etiology
Wounds, Nonpenetrating*
Humans ; Visual Acuity
Opinia redakcyjna
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Tau Gene Deletion does not Influence Axonal Regeneration and Retinal Neuron Survival in the Injured Mouse Visual System.
Autorzy:
Rodriguez L; CUO-Recherche, Département d'ophtalmologie, Faculté de médecine, Université Laval and Centre de recherche du CHU de Québec-Université Laval, Quebec, QC G1V 0A6, Canada.
Joly S; CUO-Recherche, Département d'ophtalmologie, Faculté de médecine, Université Laval and Centre de recherche du CHU de Québec-Université Laval, Quebec, QC G1V 0A6, Canada.
Mdzomba JB; CUO-Recherche, Département d'ophtalmologie, Faculté de médecine, Université Laval and Centre de recherche du CHU de Québec-Université Laval, Quebec, QC G1V 0A6, Canada.
Pernet V; CUO-Recherche, Département d'ophtalmologie, Faculté de médecine, Université Laval and Centre de recherche du CHU de Québec-Université Laval, Quebec, QC G1V 0A6, Canada.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Jun 08; Vol. 21 (11). Date of Electronic Publication: 2020 Jun 08.
Typ publikacji:
Journal Article
MeSH Terms:
Gene Deletion*
Axons/*metabolism
Nerve Regeneration/*genetics
Optic Nerve Injuries/*genetics
Optic Nerve Injuries/*metabolism
Retinal Ganglion Cells/*pathology
tau Proteins/*genetics
Animals ; Cell Death ; Cell Survival ; Disease Models, Animal ; Disease Susceptibility ; Mice ; Mice, Knockout ; Optic Nerve Injuries/pathology ; Retina/metabolism ; Retina/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Grease gun injury of the orbit: A rare case report.
Autorzy:
Chakraborti C; Department of Ophthalmology, North Bengal Medical College, Darjeeling, West Bengal, India.
Saha AK; Department of Ophthalmology, North Bengal Medical College, Darjeeling, West Bengal, India.
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Źródło:
Indian journal of ophthalmology [Indian J Ophthalmol] 2020 Aug; Vol. 68 (8), pp. 1701-1703.
Typ publikacji:
Case Reports
MeSH Terms:
Optic Nerve Injuries*
Orbit*/diagnostic imaging
Orbit*/surgery
Adult ; Humans ; India ; Lubricants ; Male ; Tomography, X-Ray Computed ; Young Adult
Raport
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Neuroinflammation and Optic Nerve Regeneration: Where Do We Stand in Elucidating Underlying Cellular and Molecular Players?
Autorzy:
Andries L; Department of Biology, Neural Circuit Development and Regeneration Research Group, KU Leuven, Leuven, Belgium.
De Groef L; Department of Biology, Neural Circuit Development and Regeneration Research Group, KU Leuven, Leuven, Belgium.
Moons L; Department of Biology, Neural Circuit Development and Regeneration Research Group, KU Leuven, Leuven, Belgium.
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Źródło:
Current eye research [Curr Eye Res] 2020 Mar; Vol. 45 (3), pp. 397-409. Date of Electronic Publication: 2019 Oct 07.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Axons/*physiology
Nerve Regeneration/*physiology
Optic Nerve/*physiopathology
Optic Nerve Injuries/*physiopathology
Animals ; Humans ; Optic Nerve/pathology ; Optic Nerve Injuries/metabolism ; Optic Nerve Injuries/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Philanthotoxin-343 attenuates retinal and optic nerve injury, and protects visual function in rats with N-methyl-D-aspartate-induced excitotoxicity.
Autorzy:
Fazel MF; Institute of Medical Science Technology, Universiti Kuala Lumpur, Kuala Lumpur, Malaysia.
Abu IF; Institute of Medical Science Technology, Universiti Kuala Lumpur, Kuala Lumpur, Malaysia.
Mohamad MHN; Institute of Medical Science Technology, Universiti Kuala Lumpur, Kuala Lumpur, Malaysia.
Agarwal R; School of Medicine, International Medical University, Bukit Jalil, Kuala Lumpur, Malaysia.
Iezhitsa I; School of Medicine, International Medical University, Bukit Jalil, Kuala Lumpur, Malaysia.
Bakar NS; Faculty of Medicine, Universiti Teknologi MARA, Sungai Buloh, Selangor, Malaysia.
Juliana N; Faculty of Medicine & Health Sciences, Universiti Sains Islam Malaysia, Indah, Kuala Lumpur, Malaysia.
Mellor IR; School of Life Sciences, Faculty of Medicine & Health Sciences, University of Nottingham, Nottingham, United Kingdom.
Franzyk H; Department of Drug Design and Pharmacology, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark.
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Źródło:
PloS one [PLoS One] 2020 Jul 24; Vol. 15 (7), pp. e0236450. Date of Electronic Publication: 2020 Jul 24 (Print Publication: 2020).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Neuroprotective Agents*/pharmacology
Neuroprotective Agents*/therapeutic use
Phenols*/pharmacology
Phenols*/therapeutic use
Polyamines*/pharmacology
Polyamines*/therapeutic use
Optic Nerve/*drug effects
Optic Nerve Injuries/*drug therapy
Retinal Ganglion Cells/*drug effects
Animals ; Male ; N-Methylaspartate/toxicity ; Optic Nerve/pathology ; Optic Nerve Injuries/chemically induced ; Rats ; Rats, Sprague-Dawley ; Retinal Ganglion Cells/pathology ; Vision, Ocular/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Neuroprotective effects of brain-derived neurotrophic factor against amyloid beta 1-40-induced retinal and optic nerve damage.
Autorzy:
Mohd Lazaldin MA; Centre for Neuroscience Research (NeuRon), Faculty of Medicine, Universiti Teknologi MARA, Sungai Buloh, Malaysia.
Iezhitsa I; Centre for Neuroscience Research (NeuRon), Faculty of Medicine, Universiti Teknologi MARA, Sungai Buloh, Malaysia.; Institute for Pathology, Laboratory and Forensic Medicine (I-PPerForM), Universiti Teknologi MARA, Sungai Buloh, Malaysia.; Research Centre for Innovative Medicines, Volgograd State Medical University, Volgograd, Russia.
Agarwal R; School of Medicine, International Medical University, Kuala Lumpur, Malaysia.
Bakar NS; Centre for Neuroscience Research (NeuRon), Faculty of Medicine, Universiti Teknologi MARA, Sungai Buloh, Malaysia.
Agarwal P; School of Medicine, International Medical University, Kuala Lumpur, Malaysia.
Mohd Ismail N; School of Medicine, International Medical University, Kuala Lumpur, Malaysia.
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Źródło:
The European journal of neuroscience [Eur J Neurosci] 2020 Jun; Vol. 51 (12), pp. 2394-2411. Date of Electronic Publication: 2020 Jan 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Neuroprotective Agents*/pharmacology
Optic Nerve Injuries*/drug therapy
Amyloid beta-Peptides/toxicity ; Animals ; Brain-Derived Neurotrophic Factor ; Optic Nerve ; Rats ; Rats, Sprague-Dawley ; Retinal Ganglion Cells
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Extracellular vesicles derived from human ES-MSCs protect retinal ganglion cells and preserve retinal function in a rodent model of optic nerve injury.
Autorzy:
Seyedrazizadeh SZ; Department of Brain and Cognitive Sciences, Cell Science Research Center, Royan Institute for Stem Cell Biology and Technology, ACECR, Tehran, Iran.
Poosti S; Department of Brain and Cognitive Sciences, Cell Science Research Center, Royan Institute for Stem Cell Biology and Technology, ACECR, Tehran, Iran.
Nazari A; Department of Stem Cells and Developmental Biology, Cell Science Research Center, Royan Institute for Stem Cell Biology and Technology, ACECR, Tehran, Iran.
Alikhani M; Department of Stem Cells and Developmental Biology, Cell Science Research Center, Royan Institute for Stem Cell Biology and Technology, ACECR, Tehran, Iran.
Shekari F; Department of Stem Cells and Developmental Biology, Cell Science Research Center, Royan Institute for Stem Cell Biology and Technology, ACECR, Tehran, Iran.
Pakdel F; Ophthalmology Department, Eye Research Center, Tehran University of Medical Sciences, Tehran, Iran.
Shahpasand K; Department of Brain and Cognitive Sciences, Cell Science Research Center, Royan Institute for Stem Cell Biology and Technology, ACECR, Tehran, Iran.
Satarian L; Department of Brain and Cognitive Sciences, Cell Science Research Center, Royan Institute for Stem Cell Biology and Technology, ACECR, Tehran, Iran. .; Department of Stem Cells and Developmental Biology, Cell Science Research Center, Royan Institute for Stem Cell Biology and Technology, ACECR, Tehran, Iran. .
Baharvand H; Department of Brain and Cognitive Sciences, Cell Science Research Center, Royan Institute for Stem Cell Biology and Technology, ACECR, Tehran, Iran. .; Department of Developmental Biology, University of Science and Culture, Tehran, Iran. .
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Źródło:
Stem cell research & therapy [Stem Cell Res Ther] 2020 May 27; Vol. 11 (1), pp. 203. Date of Electronic Publication: 2020 May 27.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Extracellular Vesicles*
Human Embryonic Stem Cells*
Optic Nerve Injuries*/therapy
Animals ; Disease Models, Animal ; Humans ; Retinal Ganglion Cells ; Rodentia
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Quantitative profiling of regional protein expression in rat retina after partial optic nerve transection using fluorescence difference two‑dimensional gel electrophoresis.
Autorzy:
Lam C; School of Optometry, Hong Kong Polytechnic University, Hong Kong 999077, SAR, P.R. China.
Li KK; School of Optometry, Hong Kong Polytechnic University, Hong Kong 999077, SAR, P.R. China.
Do CW; School of Optometry, Hong Kong Polytechnic University, Hong Kong 999077, SAR, P.R. China.
Chan H; School of Optometry, Hong Kong Polytechnic University, Hong Kong 999077, SAR, P.R. China.
To CH; School of Optometry, Hong Kong Polytechnic University, Hong Kong 999077, SAR, P.R. China.
Kwong JMK; Department of Ophthalmology, Stein Eye Institute, David Geffen School of Medicine, University of California Los Angeles, Los Angeles, CA 90095, USA.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2019 Sep; Vol. 20 (3), pp. 2734-2742. Date of Electronic Publication: 2019 Jul 25.
Typ publikacji:
Journal Article
MeSH Terms:
Proteome*
Proteomics*/methods
Optic Nerve Injuries/*metabolism
Retina/*metabolism
Retinal Ganglion Cells/*metabolism
Animals ; Disease Models, Animal ; Electrophoresis, Gel, Two-Dimensional ; Immunohistochemistry ; Optic Nerve Injuries/diagnosis ; Optic Nerve Injuries/etiology ; Rats ; Retinal Degeneration/diagnosis ; Retinal Degeneration/etiology ; Retinal Degeneration/metabolism ; Tandem Mass Spectrometry
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Visual recovery following optic nerve crush in male and female wild-type and TRIF-deficient mice.
Autorzy:
Du YL; Emory University College of Arts and Sciences, Neuroscience and Behavioral Biology Program, GA, USA.
Sergeeva EG; Department of Emergency Medicine, Emory University School of Medicine, Atlanta, GA, USA.
Stein DG; Emory University College of Arts and Sciences, Neuroscience and Behavioral Biology Program, GA, USA.; Department of Emergency Medicine, Emory University School of Medicine, Atlanta, GA, USA.
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Źródło:
Restorative neurology and neuroscience [Restor Neurol Neurosci] 2020; Vol. 38 (5), pp. 355-368.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adaptor Proteins, Vesicular Transport/*metabolism
Evoked Potentials, Visual/*physiology
Inflammation/*metabolism
Nerve Regeneration/*physiology
Optic Nerve Injuries/*metabolism
Recovery of Function/*physiology
Retinal Ganglion Cells/*physiology
Visual Perception/*physiology
Animals ; Behavior, Animal/physiology ; Disease Models, Animal ; Female ; Inflammation/immunology ; Male ; Mice ; Mice, Knockout ; Optic Nerve Injuries/immunology ; Signal Transduction/physiology ; Toll-Like Receptor 3/metabolism
Czasopismo naukowe
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