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Tytuł:
A novel trypsin of Trichinella spiralis mediates larval invasion of gut epithelium via binding to PAR2 and activating ERK1/2 pathway.
Autorzy:
Han LL; Department of Parasitology, Medical College, Zhengzhou University, Zhengzhou, China.
Lu QQ; Department of Parasitology, Medical College, Zhengzhou University, Zhengzhou, China.
Zheng WW; Department of Parasitology, Medical College, Zhengzhou University, Zhengzhou, China.
Li YL; Department of Parasitology, Medical College, Zhengzhou University, Zhengzhou, China.
Song YY; Department of Parasitology, Medical College, Zhengzhou University, Zhengzhou, China.
Zhang XZ; Department of Parasitology, Medical College, Zhengzhou University, Zhengzhou, China.
Long SR; Department of Parasitology, Medical College, Zhengzhou University, Zhengzhou, China.
Liu RD; Department of Parasitology, Medical College, Zhengzhou University, Zhengzhou, China.
Wang ZQ; Department of Parasitology, Medical College, Zhengzhou University, Zhengzhou, China.
Cui J; Department of Parasitology, Medical College, Zhengzhou University, Zhengzhou, China.
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Źródło:
PLoS neglected tropical diseases [PLoS Negl Trop Dis] 2024 Jan 02; Vol. 18 (1), pp. e0011874. Date of Electronic Publication: 2024 Jan 02 (Print Publication: 2024).
Typ publikacji:
Journal Article
MeSH Terms:
Trichinella spiralis*/metabolism
Trichinella spiralis*/pathogenicity
Trichinellosis*/genetics
Trichinellosis*/metabolism
Receptor, PAR-2*/metabolism
Animals ; Humans ; Mice ; Caco-2 Cells ; Epithelium/metabolism ; Helminth Proteins/metabolism ; Larva/physiology ; MAP Kinase Signaling System ; Mice, Inbred BALB C ; Protein Kinases ; Trypsin/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Single-Nucleotide Polymorphisms of the PAR2 and IL-17A Genes Are Significantly Associated with Chronic Pain.
Autorzy:
Soeda M; Addictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan.; Department of Oral Health and Clinical Science, Tokyo Dental College, Tokyo 101-0061, Japan.
Ohka S; Addictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan.
Nishizawa D; Addictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan.
Iseki M; Department of Anesthesiology & Pain Medicine, Juntendo University School of Medicine, Tokyo 113-8431, Japan.
Yamaguchi K; Department of Anesthesiology & Pain Medicine, Juntendo University School of Medicine, Tokyo 113-8431, Japan.
Arita H; Department of Anesthesiology, Pain Relief Center, JR Tokyo General Hospital, Tokyo 151-8528, Japan.
Hanaoka K; Department of Anesthesiology, Pain Relief Center, JR Tokyo General Hospital, Tokyo 151-8528, Japan.
Kato J; Department of Anesthesiology, Nihon University School of Medicine, Tokyo 173-8610, Japan.
Ogawa S; University Research Center, Nihon University, Tokyo 173-8610, Japan.
Hiranuma A; Addictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan.; Department of Surgery, Toho University Sakura Medical Center, Chiba 285-8741, Japan.
Hasegawa J; Addictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan.
Nakayama K; Addictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan.
Ebata Y; Addictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan.
Hayashida M; Addictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan.; Department of Anesthesiology & Pain Medicine, Juntendo University School of Medicine, Tokyo 113-8431, Japan.; Department of Anesthesiology, Saitama Medical University International Medical Center, Saitama 350-1298, Japan.
Ichinohe T; Department of Dental Anesthesiology, Tokyo Dental College, Tokyo 101-0061, Japan.
Fukuda KI; Department of Oral Health and Clinical Science, Tokyo Dental College, Tokyo 101-0061, Japan.
Ikeda K; Addictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Dec 18; Vol. 24 (24). Date of Electronic Publication: 2023 Dec 18.
Typ publikacji:
Journal Article
MeSH Terms:
Autoimmune Diseases*
Chronic Pain*/genetics
Interleukin-17*/genetics
Receptor, PAR-2*/genetics
Humans ; Case-Control Studies ; Genetic Predisposition to Disease ; Polymorphism, Single Nucleotide
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Protease secretions by the invading blastocyst induce calcium oscillations in endometrial epithelial cells via the protease-activated receptor 2.
Autorzy:
Hennes A; Laboratory of Endometrium, Endometriosis and Reproductive Medicine, Department of Development and Regeneration, KU Leuven, Herestraat 49 Box 611, 3000, Leuven, Belgium.; Laboratory of Ion Channel Research, Department of Cellular and Molecular Medicine, VIB Center for Brain & Disease Research, KU Leuven, Herestraat 49 Box 802, 3000, Leuven, Belgium.
Devroe J; Laboratory of Endometrium, Endometriosis and Reproductive Medicine, Department of Development and Regeneration, KU Leuven, Herestraat 49 Box 611, 3000, Leuven, Belgium.; Laboratory of Ion Channel Research, Department of Cellular and Molecular Medicine, VIB Center for Brain & Disease Research, KU Leuven, Herestraat 49 Box 802, 3000, Leuven, Belgium.; Leuven University Fertility Center, University Hospitals Leuven, Herestraat 49, 3000, Leuven, Belgium.
De Clercq K; Laboratory of Endometrium, Endometriosis and Reproductive Medicine, Department of Development and Regeneration, KU Leuven, Herestraat 49 Box 611, 3000, Leuven, Belgium.; Laboratory of Ion Channel Research, Department of Cellular and Molecular Medicine, VIB Center for Brain & Disease Research, KU Leuven, Herestraat 49 Box 802, 3000, Leuven, Belgium.
Ciprietti M; Laboratory of Endometrium, Endometriosis and Reproductive Medicine, Department of Development and Regeneration, KU Leuven, Herestraat 49 Box 611, 3000, Leuven, Belgium.; Laboratory of Ion Channel Research, Department of Cellular and Molecular Medicine, VIB Center for Brain & Disease Research, KU Leuven, Herestraat 49 Box 802, 3000, Leuven, Belgium.
Held K; Laboratory of Endometrium, Endometriosis and Reproductive Medicine, Department of Development and Regeneration, KU Leuven, Herestraat 49 Box 611, 3000, Leuven, Belgium.; Laboratory of Ion Channel Research, Department of Cellular and Molecular Medicine, VIB Center for Brain & Disease Research, KU Leuven, Herestraat 49 Box 802, 3000, Leuven, Belgium.
Luyten K; Laboratory of Endometrium, Endometriosis and Reproductive Medicine, Department of Development and Regeneration, KU Leuven, Herestraat 49 Box 611, 3000, Leuven, Belgium.
Van Ranst N; Laboratory of Ion Channel Research, Department of Cellular and Molecular Medicine, VIB Center for Brain & Disease Research, KU Leuven, Herestraat 49 Box 802, 3000, Leuven, Belgium.
Maenhoudt N; Laboratory of Tissue Plasticity in Health and Disease, Cluster of Stem Cell and Developmental Biology, Department of Development and Regeneration, KU Leuven, Herestraat 49 Box 804, 3000, Leuven, Belgium.
Peeraer K; Laboratory of Endometrium, Endometriosis and Reproductive Medicine, Department of Development and Regeneration, KU Leuven, Herestraat 49 Box 611, 3000, Leuven, Belgium.; Leuven University Fertility Center, University Hospitals Leuven, Herestraat 49, 3000, Leuven, Belgium.
Vankelecom H; Laboratory of Tissue Plasticity in Health and Disease, Cluster of Stem Cell and Developmental Biology, Department of Development and Regeneration, KU Leuven, Herestraat 49 Box 804, 3000, Leuven, Belgium.
Voets T; Laboratory of Ion Channel Research, Department of Cellular and Molecular Medicine, VIB Center for Brain & Disease Research, KU Leuven, Herestraat 49 Box 802, 3000, Leuven, Belgium.
Vriens J; Laboratory of Endometrium, Endometriosis and Reproductive Medicine, Department of Development and Regeneration, KU Leuven, Herestraat 49 Box 611, 3000, Leuven, Belgium. .; Laboratory of Ion Channel Research, Department of Cellular and Molecular Medicine, VIB Center for Brain & Disease Research, KU Leuven, Herestraat 49 Box 802, 3000, Leuven, Belgium. .
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Źródło:
Reproductive biology and endocrinology : RB&E [Reprod Biol Endocrinol] 2023 Apr 15; Vol. 21 (1), pp. 37. Date of Electronic Publication: 2023 Apr 15.
Typ publikacji:
Journal Article
MeSH Terms:
Calcium Signaling*
Receptor, PAR-2*/genetics
Receptor, PAR-2*/metabolism
Female ; Humans ; Peptide Hydrolases/metabolism ; Calcium/metabolism ; Endometrium/metabolism ; Blastocyst/physiology ; Embryo Implantation/physiology ; Epithelial Cells/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Involvement of Mast-Cell-Tryptase- and Protease-Activated Receptor 2-Mediated Signaling and Urothelial Barrier Dysfunction with Reduced Uroplakin II Expression in Bladder Hyperactivity Induced by Chronic Bladder Ischemia in the Rat.
Autorzy:
Akaihata H; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Matsuoka K; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Hata J; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Harigane Y; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Yaginuma K; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Endo Y; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Imai H; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Matsuoka Y; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Onagi A; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Tanji R; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Honda-Takinami R; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Hoshi S; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Koguchi T; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Sato Y; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Kataoka M; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Uemura M; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
Igawa Y; Department of Urology, Nagano Prefectural Shinshu Medical Center, 1332 Suzaka, Nagano 382-8577, Japan.
Kojima Y; Department of Urology, Fukushima Medical University, 1 Hikarigaoka, Fukushima 960-1295, Japan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Feb 16; Vol. 24 (4). Date of Electronic Publication: 2023 Feb 16.
Typ publikacji:
Journal Article
MeSH Terms:
Ischemia*/metabolism
Receptor, PAR-2*/metabolism
Tryptases*/metabolism
Urinary Bladder*/blood supply
Urinary Bladder*/metabolism
Urinary Bladder, Overactive*/metabolism
Animals ; Rats ; Mast Cells/metabolism ; Uroplakin II/metabolism ; Urothelium/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Platelet-activating factor and protease-activated receptor 2 cooperate to promote neutrophil recruitment and lung inflammation through nuclear factor-kappa B transactivation.
Autorzy:
Silva IS; Laboratory of Inflammation and Proteases, Department of Pharmacology, Institute of Biological Sciences (ICB), Federal University of Minas Gerais (UFMG), Belo Horizonte, Minas Gerais, Brazil.
Almeida AD; Laboratory of Inflammation and Proteases, Department of Pharmacology, Institute of Biological Sciences (ICB), Federal University of Minas Gerais (UFMG), Belo Horizonte, Minas Gerais, Brazil.
Lima Filho ACM; Department of Physiology and Biophysics, ICB/UFMG, Belo Horizonte, Minas Gerais, Brazil.
Fernandes-Braga W; Laboratory of Atherosclerosis and Nutritional Biochemistry (LABIN-UFMG), Department of Biochemistry and Immunology, ICB/UFMG, Belo Horizonte, Minas Gerais, Brazil.
Barra A; Laboratory of Inflammation and Proteases, Department of Pharmacology, Institute of Biological Sciences (ICB), Federal University of Minas Gerais (UFMG), Belo Horizonte, Minas Gerais, Brazil.
Oliveira HMC; Department of Morphology, ICB/UFMG, Belo Horizonte, Minas Gerais, Brazil.
Cassali GD; Department of Pathology, ICB/UFMG, Belo Horizonte, Minas Gerais, Brazil.
Capettini LSA; Laboratory of Vascular Biology, Department of Pharmacology, ICB/UFMG, Belo Horizonte, Minas Gerais, Brazil.
Menezes GB; Department of Morphology, ICB/UFMG, Belo Horizonte, Minas Gerais, Brazil.
Alvarez-Leite JI; Laboratory of Atherosclerosis and Nutritional Biochemistry (LABIN-UFMG), Department of Biochemistry and Immunology, ICB/UFMG, Belo Horizonte, Minas Gerais, Brazil.
Leite MF; Department of Physiology and Biophysics, ICB/UFMG, Belo Horizonte, Minas Gerais, Brazil.
Klein A; Laboratory of Inflammation and Proteases, Department of Pharmacology, Institute of Biological Sciences (ICB), Federal University of Minas Gerais (UFMG), Belo Horizonte, Minas Gerais, Brazil. .
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Źródło:
Scientific reports [Sci Rep] 2023 Dec 07; Vol. 13 (1), pp. 21637. Date of Electronic Publication: 2023 Dec 07.
Typ publikacji:
Journal Article
MeSH Terms:
NF-kappa B*/metabolism
Pneumonia*/chemically induced
Mice ; Animals ; Platelet Activating Factor/metabolism ; Receptor, PAR-2/genetics ; Receptor, PAR-2/metabolism ; Receptors, G-Protein-Coupled/metabolism ; Aprotinin/metabolism ; Neutrophil Infiltration ; Transcriptional Activation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Punicalagin suppresses inflammation in ventilator-induced lung injury through protease-activated receptor-2 inhibition-induced inhibition of NLR family pyrin domain containing-3 inflammasome activation.
Autorzy:
Zhang W; Center for Rehabilitation Medicine, Rehabilitation & Sports Medicine Research Institute of Zhejiang Province, Department of Rehabilitation Medicine, Zhejiang Provincial People's Hospital, Affiliated People's Hospital, Hangzhou Medical College, Hangzhou City, China.
Zhu Q; Emergency and Critical Care Center, Department of Pulmonary and Critical Care Medicine, Zhejiang Provincial People's Hospital, Affiliated People's Hospital, Hangzhou Medical College, Hangzhou City, China.
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Źródło:
Chemical biology & drug design [Chem Biol Drug Des] 2022 Aug; Vol. 100 (2), pp. 218-229. Date of Electronic Publication: 2022 May 04.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Hydrolyzable Tannins*/pharmacology
Inflammasomes*/metabolism
NLR Family, Pyrin Domain-Containing 3 Protein*/metabolism
Receptor, PAR-2*/antagonists & inhibitors
Receptor, PAR-2*/metabolism
Ventilator-Induced Lung Injury*/drug therapy
Ventilator-Induced Lung Injury*/metabolism
Ventilator-Induced Lung Injury*/pathology
Animals ; Cytokines/metabolism ; Inflammation/drug therapy ; Lung/metabolism ; Mice ; Pyrin Domain ; Rats
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Involvement of PAR-2 in the Induction of Cell-Specific Matrix Metalloproteinase-2 by Activated Protein C in Cutaneous Wound Healing.
Autorzy:
Julovi SM; Sutton Arthritis Research Laboratory, Kolling Institute of Medical Research, University of Sydney, Pacific Highway, St. Leonards, NSW 2065, Australia.; Kidney Injury Group, Centre for Transplant and Renal Research, Westmead Institute for Medical Research, Westmead, NSW 2045, Australia.
McKelvey K; Sutton Arthritis Research Laboratory, Kolling Institute of Medical Research, University of Sydney, Pacific Highway, St. Leonards, NSW 2065, Australia.
Minhas N; Sutton Arthritis Research Laboratory, Kolling Institute of Medical Research, University of Sydney, Pacific Highway, St. Leonards, NSW 2065, Australia.
Chan YA; Sutton Arthritis Research Laboratory, Kolling Institute of Medical Research, University of Sydney, Pacific Highway, St. Leonards, NSW 2065, Australia.
Xue M; Sutton Arthritis Research Laboratory, Kolling Institute of Medical Research, University of Sydney, Pacific Highway, St. Leonards, NSW 2065, Australia.
Jackson CJ; Sutton Arthritis Research Laboratory, Kolling Institute of Medical Research, University of Sydney, Pacific Highway, St. Leonards, NSW 2065, Australia.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Dec 27; Vol. 25 (1). Date of Electronic Publication: 2023 Dec 27.
Typ publikacji:
Journal Article
MeSH Terms:
Matrix Metalloproteinase 2*/genetics
Protein C*
Humans ; Animals ; Male ; Mice ; Mice, Inbred C57BL ; Matrix Metalloproteinase 9/genetics ; Endopeptidases ; Mice, Knockout ; Receptor, PAR-2/genetics ; Wound Healing
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Coagulation Factor Xa Has No Effects on the Expression of PAR1, PAR2, and PAR4 and No Proinflammatory Effects on HL-1 Cells.
Autorzy:
Ruf L; Institute of Clinical Chemistry and Pathobiochemistry, Department of Pathobiochemistry, Otto-von-Guericke-University Magdeburg, Leipziger Str. 44, 39120 Magdeburg, Germany.
Bukowska A; Institute of Clinical Chemistry and Pathobiochemistry, Department of Pathobiochemistry, Otto-von-Guericke-University Magdeburg, Leipziger Str. 44, 39120 Magdeburg, Germany.
Gardemann A; Institute of Clinical Chemistry and Pathobiochemistry, Department of Pathobiochemistry, Otto-von-Guericke-University Magdeburg, Leipziger Str. 44, 39120 Magdeburg, Germany.
Goette A; Institute of Clinical Chemistry and Pathobiochemistry, Department of Pathobiochemistry, Otto-von-Guericke-University Magdeburg, Leipziger Str. 44, 39120 Magdeburg, Germany.; Department of Cardiology and Intensive Care Medicine, St. Vincenz-Hospital Paderborn, Am Busdorf 2, 33098 Paderborn, Germany.
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Źródło:
Cells [Cells] 2023 Dec 15; Vol. 12 (24). Date of Electronic Publication: 2023 Dec 15.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Atrial Fibrillation*
Factor Xa*/metabolism
Animals ; Mice ; NF-kappa B/metabolism ; Receptor, PAR-1/metabolism ; Receptor, PAR-2/metabolism ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
GB83, an Agonist of PAR2 with a Unique Mechanism of Action Distinct from Trypsin and PAR2-AP.
Autorzy:
Heo Y; College of Pharmacy, Yonsei Institute of Pharmaceutical Sciences, Yonsei University, Incheon 21983, Korea.
Yang E; College of Pharmacy, Yonsei Institute of Pharmaceutical Sciences, Yonsei University, Incheon 21983, Korea.
Lee Y; College of Pharmacy, Yonsei Institute of Pharmaceutical Sciences, Yonsei University, Incheon 21983, Korea.
Seo Y; College of Pharmacy, Yonsei Institute of Pharmaceutical Sciences, Yonsei University, Incheon 21983, Korea.; New Drug Development Center, Daegu Gyeongbuk Medical Innovation Foundation, Daegu 41061, Korea.
Ryu K; College of Pharmacy, Yonsei Institute of Pharmaceutical Sciences, Yonsei University, Incheon 21983, Korea.
Jeon H; College of Pharmacy, Yonsei Institute of Pharmaceutical Sciences, Yonsei University, Incheon 21983, Korea.
Namkung W; College of Pharmacy, Yonsei Institute of Pharmaceutical Sciences, Yonsei University, Incheon 21983, Korea.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Sep 13; Vol. 23 (18). Date of Electronic Publication: 2022 Sep 13.
Typ publikacji:
Journal Article
MeSH Terms:
Calcium*/metabolism
Receptor, PAR-2*/metabolism
Peptides ; Trypsin ; beta-Arrestins
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Granzyme K initiates IL-6 and IL-8 release from epithelial cells by activating protease-activated receptor 2.
Autorzy:
Kaiserman D; Department of Biochemistry & Molecular Biology, Biomedicine Discovery Institute, Monash University, Clayton, VIC, Australia.
Zhao P; Monash Institute of Pharmaceutical Sciences, Monash University, Parkville, VIC, Australia.; Australian Research Council Centre of Excellence in Convergent Bio-Nano Science and Technology, Monash University, Parkville, VIC, Australia.
Rowe CL; Department of Biochemistry & Molecular Biology, Biomedicine Discovery Institute, Monash University, Clayton, VIC, Australia.
Leong A; Monash Institute of Pharmaceutical Sciences, Monash University, Parkville, VIC, Australia.; Australian Research Council Centre of Excellence in Convergent Bio-Nano Science and Technology, Monash University, Parkville, VIC, Australia.
Barlow N; Monash Institute of Pharmaceutical Sciences, Monash University, Parkville, VIC, Australia.
Joeckel LT; Department of Biochemistry & Molecular Biology, Biomedicine Discovery Institute, Monash University, Clayton, VIC, Australia.
Hitchen C; Department of Biochemistry & Molecular Biology, Biomedicine Discovery Institute, Monash University, Clayton, VIC, Australia.
Stewart SE; Department of Biochemistry & Molecular Biology, Biomedicine Discovery Institute, Monash University, Clayton, VIC, Australia.
Hollenberg MD; Department of Physiology & Pharmacology, Department of Medicine, University of Calgary, Calgary AB, Canada.
Bunnett N; Department of Molecular Pathobiology, Department of Neuroscience and Physiology, Neuroscience Institute, New York University, New York, NY, United States of America.
Suhrbier A; QIMR Berghofer Medical Research Institute, Brisbane, QLD, Australia.; Australian Infectious Disease Research Centre, University of Queensland, Brisbane, QLD, Australia.
Bird PI; Department of Biochemistry & Molecular Biology, Biomedicine Discovery Institute, Monash University, Clayton, VIC, Australia.
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Źródło:
PloS one [PLoS One] 2022 Jul 26; Vol. 17 (7), pp. e0270584. Date of Electronic Publication: 2022 Jul 26 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, N.I.H., Extramural
MeSH Terms:
Receptor, PAR-1*/metabolism
Receptor, PAR-2*/metabolism
Endopeptidases/metabolism ; Endothelial Cells/metabolism ; Epithelial Cells/metabolism ; Granzymes/metabolism ; Interleukin-6/metabolism ; Interleukin-8/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Melanocore uptake by keratinocytes occurs through phagocytosis and involves protease-activated receptor-2 internalization.
Autorzy:
Moreiras H; CEDOC-Chronic Diseases Research Center, NOVA Medical School (NMS), Faculdade de Ciências Médicas, Universidade NOVA de Lisboa, Lisbon, Portugal.
Bento-Lopes L; CEDOC-Chronic Diseases Research Center, NOVA Medical School (NMS), Faculdade de Ciências Médicas, Universidade NOVA de Lisboa, Lisbon, Portugal.
Neto MV; CEDOC-Chronic Diseases Research Center, NOVA Medical School (NMS), Faculdade de Ciências Médicas, Universidade NOVA de Lisboa, Lisbon, Portugal.
Escrevente C; CEDOC-Chronic Diseases Research Center, NOVA Medical School (NMS), Faculdade de Ciências Médicas, Universidade NOVA de Lisboa, Lisbon, Portugal.
Cabaço LC; CEDOC-Chronic Diseases Research Center, NOVA Medical School (NMS), Faculdade de Ciências Médicas, Universidade NOVA de Lisboa, Lisbon, Portugal.
Hall MJ; CEDOC-Chronic Diseases Research Center, NOVA Medical School (NMS), Faculdade de Ciências Médicas, Universidade NOVA de Lisboa, Lisbon, Portugal.
Ramalho JS; CEDOC-Chronic Diseases Research Center, NOVA Medical School (NMS), Faculdade de Ciências Médicas, Universidade NOVA de Lisboa, Lisbon, Portugal.
Seabra MC; CEDOC-Chronic Diseases Research Center, NOVA Medical School (NMS), Faculdade de Ciências Médicas, Universidade NOVA de Lisboa, Lisbon, Portugal.
Barral DC; CEDOC-Chronic Diseases Research Center, NOVA Medical School (NMS), Faculdade de Ciências Médicas, Universidade NOVA de Lisboa, Lisbon, Portugal.
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Źródło:
Traffic (Copenhagen, Denmark) [Traffic] 2022 Jun; Vol. 23 (6), pp. 331-345. Date of Electronic Publication: 2022 May 15.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Melanins*/metabolism
Receptor, PAR-2*/metabolism
Actins/metabolism ; Keratinocytes/metabolism ; Melanocytes/metabolism ; Melanosomes/metabolism ; Phagocytosis/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Proteinase-activated receptor-2 antagonist C391 inhibits Alternaria-induced airway epithelial signalling and asthma indicators in acute exposure mouse models.
Autorzy:
Rivas CM; Physiological Sciences Graduate Interdisciplinary Program, University of Arizona, Tucson, Arizona, USA.; Asthma and Airway Disease Research Center, University of Arizona Health Sciences, Tucson, Arizona, USA.
Yee MC; Biomedical Sciences, University of California Riverside, Riverside, California, USA.
Addison KJ; Asthma and Airway Disease Research Center, University of Arizona Health Sciences, Tucson, Arizona, USA.; Department of Cellular and Molecular Medicine, University of Arizona Health Sciences, Tucson, Arizona, USA.
Lovett M; Physiological Sciences Graduate Interdisciplinary Program, University of Arizona, Tucson, Arizona, USA.
Pal K; Biomedical Sciences, University of California Riverside, Riverside, California, USA.
Ledford JG; Asthma and Airway Disease Research Center, University of Arizona Health Sciences, Tucson, Arizona, USA.; Department of Cellular and Molecular Medicine, University of Arizona Health Sciences, Tucson, Arizona, USA.
Dussor G; School of Behavioral and Brain Sciences and Center for Advanced Pain Studies, University of Texas at Dallas, Richardson, Texas, USA.
Price TJ; School of Behavioral and Brain Sciences and Center for Advanced Pain Studies, University of Texas at Dallas, Richardson, Texas, USA.
Vagner J; Bio5 Collaborative Research Institute, University of Arizona, Tucson, Arizona, USA.
DeFea KA; Biomedical Sciences, University of California Riverside, Riverside, California, USA.; Corporate Headquarters, PARMedics, Inc., Temecula, California, USA.
Boitano S; Physiological Sciences Graduate Interdisciplinary Program, University of Arizona, Tucson, Arizona, USA.; Asthma and Airway Disease Research Center, University of Arizona Health Sciences, Tucson, Arizona, USA.; Department of Cellular and Molecular Medicine, University of Arizona Health Sciences, Tucson, Arizona, USA.; Bio5 Collaborative Research Institute, University of Arizona, Tucson, Arizona, USA.; Department of Physiology, University of Arizona Health Sciences, Tucson, Arizona, USA.
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Źródło:
British journal of pharmacology [Br J Pharmacol] 2022 May; Vol. 179 (10), pp. 2208-2222. Date of Electronic Publication: 2022 Feb 01.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Asthma*/drug therapy
Asthma*/metabolism
Receptor, PAR-2*
Allergens ; Alternaria/metabolism ; Animals ; HEK293 Cells ; Humans ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Involvement of PAR2 in platelet-derived growth factor receptor-α-positive cell proliferation in the colon of diabetic mice.
Autorzy:
Li YJ; Department of Anatomy and Physiology, Shanghai Jiao Tong University College of Basic Medical Science, Shanghai, China.; Department of Pediatric Surgery, Xinhua Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Ao JP; State Key Laboratory of Oncogenes and Related Genes, School of Medicine, Shanghai Cancer Institute, Renji Hospital, Shanghai Jiao Tong University, Shanghai, China.
Huang X; Department of Anatomy and Physiology, Shanghai Jiao Tong University College of Basic Medical Science, Shanghai, China.
Lu HL; Department of Anatomy and Physiology, Shanghai Jiao Tong University College of Basic Medical Science, Shanghai, China.
Fu HY; Department of Anatomy and Physiology, Shanghai Jiao Tong University College of Basic Medical Science, Shanghai, China.
Song NN; Department of Anatomy and Physiology, Shanghai Jiao Tong University College of Basic Medical Science, Shanghai, China.
Xu WX; Department of Anatomy and Physiology, Shanghai Jiao Tong University College of Basic Medical Science, Shanghai, China.; Department of Pediatric Surgery, Xinhua Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Chen J; Department of Pediatric Surgery, Xinhua Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
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Źródło:
Physiological reports [Physiol Rep] 2021 Nov; Vol. 9 (21), pp. e15099.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Proliferation*
Colon/*metabolism
Diabetes Mellitus, Experimental/*metabolism
Receptor, PAR-2/*metabolism
Receptor, Platelet-Derived Growth Factor alpha/*metabolism
Animals ; Cells, Cultured ; Colon/cytology ; Colon/drug effects ; Male ; Mice ; Mice, Inbred ICR ; Myocytes, Smooth Muscle/metabolism ; Myocytes, Smooth Muscle/physiology ; NIH 3T3 Cells ; Oligopeptides/pharmacology ; Phosphatidylinositol 3-Kinases/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Receptor, PAR-2/agonists ; Receptor, PAR-2/genetics ; Receptor, Platelet-Derived Growth Factor alpha/genetics ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Histone 2B Facilitates Plasminogen-Enhanced Endothelial Migration through Protease-Activated Receptor 1 (PAR1) and Protease-Activated Receptor 2 (PAR2).
Autorzy:
Das M; Department of Cardiovascular and Metabolic Sciences, Lerner Research Institute, Cleveland Clinic, Cleveland, OH 44195, USA.
Ithychanda SS; Department of Cardiovascular and Metabolic Sciences, Lerner Research Institute, Cleveland Clinic, Cleveland, OH 44195, USA.
Plow EF; Department of Cardiovascular and Metabolic Sciences, Lerner Research Institute, Cleveland Clinic, Cleveland, OH 44195, USA.
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Źródło:
Biomolecules [Biomolecules] 2022 Jan 26; Vol. 12 (2). Date of Electronic Publication: 2022 Jan 26.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Receptor, PAR-1*
Receptor, PAR-2*
Endothelial Cells/metabolism ; Histones/metabolism ; Plasminogen/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Activated Protein C Protects against Murine Contact Dermatitis by Suppressing Protease-Activated Receptor 2.
Autorzy:
Xue M; Sutton Arthritis Research Laboratory, Institute of Bone and Joint Research, Kolling Institute, Faculty of Medicine and Health, University of Sydney at Royal North Shore Hospital, St Leonards, NSW 2065, Australia.; The Australian Arthritis and Autoimmune Biobank Collaborative (A3BC), Institute of Bone and Joint Research, Kolling Institute, Faculty of Medicine and Health, University of Sydney at Royal North Shore Hospital, St Leonards, NSW 2065, Australia.
Lin H; Sutton Arthritis Research Laboratory, Institute of Bone and Joint Research, Kolling Institute, Faculty of Medicine and Health, University of Sydney at Royal North Shore Hospital, St Leonards, NSW 2065, Australia.; The Australian Arthritis and Autoimmune Biobank Collaborative (A3BC), Institute of Bone and Joint Research, Kolling Institute, Faculty of Medicine and Health, University of Sydney at Royal North Shore Hospital, St Leonards, NSW 2065, Australia.
Zhao R; Sutton Arthritis Research Laboratory, Institute of Bone and Joint Research, Kolling Institute, Faculty of Medicine and Health, University of Sydney at Royal North Shore Hospital, St Leonards, NSW 2065, Australia.
Fryer C; Sutton Arthritis Research Laboratory, Institute of Bone and Joint Research, Kolling Institute, Faculty of Medicine and Health, University of Sydney at Royal North Shore Hospital, St Leonards, NSW 2065, Australia.
March L; The Australian Arthritis and Autoimmune Biobank Collaborative (A3BC), Institute of Bone and Joint Research, Kolling Institute, Faculty of Medicine and Health, University of Sydney at Royal North Shore Hospital, St Leonards, NSW 2065, Australia.
Jackson CJ; Sutton Arthritis Research Laboratory, Institute of Bone and Joint Research, Kolling Institute, Faculty of Medicine and Health, University of Sydney at Royal North Shore Hospital, St Leonards, NSW 2065, Australia.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Jan 03; Vol. 23 (1). Date of Electronic Publication: 2022 Jan 03.
Typ publikacji:
Journal Article
MeSH Terms:
Dermatitis, Contact/*metabolism
Protein C/*metabolism
Receptor, PAR-2/*genetics
Skin/*metabolism
Animals ; Dermatitis, Contact/pathology ; Dinitrofluorobenzene/toxicity ; Female ; Gene Expression Regulation ; Humans ; Inflammation ; Mice ; Mice, Knockout ; Receptor, PAR-1/genetics ; Receptor, PAR-2/metabolism ; Skin/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Bone marrow derived mast cells injected into the osteoarthritic knee joints of mice induced by sodium monoiodoacetate enhanced spontaneous pain through activation of PAR2 and action of extracellular ATP.
Autorzy:
Habuchi H; Multidisciplinary Pain Center, Aichi Medical University, Nagakute, Aichi, Japan.
Izumi M; Department of Orthopedic Surgery, Kochi Medical School, Kochi University, Okochokohasu, Nankoku, Kochi, Japan.
Dan J; Department of Orthopedic Surgery, Kochi Medical School, Kochi University, Okochokohasu, Nankoku, Kochi, Japan.
Ushida T; Multidisciplinary Pain Center, Aichi Medical University, Nagakute, Aichi, Japan.
Ikeuchi M; Department of Orthopedic Surgery, Kochi Medical School, Kochi University, Okochokohasu, Nankoku, Kochi, Japan.
Takeuchi K; Department of Medical Cell Biology, School of Medicine, Aichi Medical University, Nagakute, Aichi, Japan.
Habuchi O; Multidisciplinary Pain Center, Aichi Medical University, Nagakute, Aichi, Japan.
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Źródło:
PloS one [PLoS One] 2021 Jun 04; Vol. 16 (6), pp. e0252590. Date of Electronic Publication: 2021 Jun 04 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adenosine Triphosphate/*metabolism
Arthritis, Experimental/*therapy
Chronic Pain/*pathology
Knee Joint/*pathology
Mast Cells/*transplantation
Receptor, PAR-2/*metabolism
Adenosine Triphosphate/analysis ; Animals ; Arthritis, Experimental/chemically induced ; Arthritis, Experimental/pathology ; Bone Marrow Cells/cytology ; Chemokine CXCL2/genetics ; Chemokine CXCL2/metabolism ; Chenodeoxycholic Acid/analogs & derivatives ; Chenodeoxycholic Acid/toxicity ; Chronic Pain/etiology ; Disease Models, Animal ; Knee Joint/metabolism ; Male ; Mast Cells/cytology ; Mast Cells/metabolism ; Matrix Metalloproteinase 9/genetics ; Matrix Metalloproteinase 9/metabolism ; Mice ; Mice, Inbred C57BL ; Mice, Transgenic ; Oligopeptides/administration & dosage ; Receptor, PAR-2/agonists ; Receptor, PAR-2/antagonists & inhibitors ; Receptors, Purinergic/metabolism ; Synovial Fluid/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Trypsin induces an aversive response in zebrafish by PAR2 activation in keratinocytes.
Autorzy:
Alsrhani A; Department of Biological Sciences, University of North Texas, Denton, TX, United States of America.
Raman R; Department of Biological Sciences, University of North Texas, Denton, TX, United States of America.
Jagadeeswaran P; Department of Biological Sciences, University of North Texas, Denton, TX, United States of America.
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Źródło:
PloS one [PLoS One] 2021 Oct 08; Vol. 16 (10), pp. e0257774. Date of Electronic Publication: 2021 Oct 08 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Receptor, PAR-2/*genetics
Skin/*metabolism
Trypsin/*metabolism
Zebrafish/*genetics
Animals ; Cell Line ; Gills/metabolism ; Keratinocytes/drug effects ; Keratinocytes/metabolism ; Larva/drug effects ; Larva/genetics ; Neomycin/pharmacology ; Receptor, PAR-2/antagonists & inhibitors ; Signal Transduction/drug effects ; Signal Transduction/genetics ; Skin/drug effects ; Trypsin/adverse effects ; Zebrafish/metabolism ; Zinc Sulfate/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Protease-Activated Receptor-2 and Phospholipid Metabolism Analysis in Hyperuricemia-Induced Renal Injury.
Autorzy:
Sui X; Department of Nephrology, The Second Affiliated Hospital of Shenzhen University, Shenzhen 518000, Guangdong, China.
Xie T; Department of Nephrology, The People's Hospital of Baoan Shenzhen, The Second School of Clinical Medicine, Southern Medical University, Shenzhen 518000, Guangdong, China.
Xu Y; Department of Nephrology, The Second Affiliated Hospital of Shenzhen University, Shenzhen 518000, Guangdong, China.
Zhang A; Department of Nephrology, The Second Affiliated Hospital of Shenzhen University, Shenzhen 518000, Guangdong, China.
Zhang Y; Department of Nephrology, The Second Affiliated Hospital of Shenzhen University, Shenzhen 518000, Guangdong, China.
Gu F; Department of Nephrology, Shenzhen Baoan People's Hospital (Group) The Second People's Hospital, Shenzhen 518000, Guangdong, China.
Li L; Department of Nephrology, The Second Affiliated Hospital of Shenzhen University, Shenzhen 518000, Guangdong, China.
Xu Z; Department of Nephrology, The People's Hospital of Baoan Shenzhen, The Second School of Clinical Medicine, Southern Medical University, Shenzhen 518000, Guangdong, China.
Chen J; Department of Nephrology, The Second Affiliated Hospital of Shenzhen University, Shenzhen 518000, Guangdong, China.; Department of Nephrology, The People's Hospital of Baoan Shenzhen, The Second School of Clinical Medicine, Southern Medical University, Shenzhen 518000, Guangdong, China.
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Źródło:
Mediators of inflammation [Mediators Inflamm] 2023 Jul 13; Vol. 2023, pp. 5007488. Date of Electronic Publication: 2023 Jul 13 (Print Publication: 2023).
Typ publikacji:
Journal Article
MeSH Terms:
Hyperuricemia*/drug therapy
Rats ; Animals ; NF-kappa B/metabolism ; Signal Transduction/physiology ; Proto-Oncogene Proteins c-akt/metabolism ; Receptor, PAR-2/metabolism ; Phosphatidylinositol 3-Kinases/metabolism ; Kidney/metabolism ; Phospholipids/metabolism ; Phospholipids/therapeutic use
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
PAR2-Induced Tissue Factor Synthesis by Primary Cultures of Human Kidney Tubular Epithelial Cells Is Modified by Glucose Availability.
Autorzy:
Humphries TLR; Centre for Kidney Disease Research, Translational Research Institute, Faulty of Medicine, The University of Queensland at the Princess Alexandra, Brisbane, QLD 4072, Australia.
Shen K; Centre for Kidney Disease Research, Translational Research Institute, Faulty of Medicine, The University of Queensland at the Princess Alexandra, Brisbane, QLD 4072, Australia.
Iyer A; Australian Research Council Centre of Excellence in Advanced Molecular Imaging, Institute for Molecular Bioscience, The University of Queensland, Brisbane, QLD 4072, Australia.; Centre for Inflammation and Disease Research, Institute for Molecular Bioscience, The University of Queensland, Brisbane, QLD 4072, Australia.
Johnson DW; Centre for Kidney Disease Research, Translational Research Institute, Faulty of Medicine, The University of Queensland at the Princess Alexandra, Brisbane, QLD 4072, Australia.; Department of Nephrology, The University of Queensland at Princess Alexandra Hospital, Brisbane, QLD 4102, Australia.
Gobe GC; Centre for Kidney Disease Research, Translational Research Institute, Faulty of Medicine, The University of Queensland at the Princess Alexandra, Brisbane, QLD 4072, Australia.; School of Biomedical Sciences, Faculty of Medicine, The University of Queensland at the Translational Research Institute, Brisbane, QLD 4072, Australia.
Nikolic-Paterson D; Department of Nephrology, Monash Medical Centre and Monash University Centre for Inflammatory Diseases, Melbourne, VIC 3168, Australia.
Fairlie DP; Australian Research Council Centre of Excellence in Advanced Molecular Imaging, Institute for Molecular Bioscience, The University of Queensland, Brisbane, QLD 4072, Australia.; Centre for Inflammation and Disease Research, Institute for Molecular Bioscience, The University of Queensland, Brisbane, QLD 4072, Australia.
Vesey DA; Centre for Kidney Disease Research, Translational Research Institute, Faulty of Medicine, The University of Queensland at the Princess Alexandra, Brisbane, QLD 4072, Australia.; Department of Nephrology, The University of Queensland at Princess Alexandra Hospital, Brisbane, QLD 4102, Australia.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Jul 14; Vol. 22 (14). Date of Electronic Publication: 2021 Jul 14.
Typ publikacji:
Journal Article
MeSH Terms:
Epithelial Cells/*metabolism
Gene Expression Regulation/*drug effects
Glucose/*pharmacology
Kidney Tubules/*metabolism
Receptor, PAR-2/*metabolism
Thromboplastin/*metabolism
Epithelial Cells/drug effects ; Humans ; Kidney Tubules/drug effects ; Receptor, PAR-2/genetics ; Sweetening Agents/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Expression of protease activated receptor-2 is reduced in renal cell carcinoma biopsies and cell lines.
Autorzy:
Morais C; Centre for Kidney Disease Research, The University of Queensland, Translational Research Institute, Brisbane, Australia.; Department of Urology, Princess Alexandra Hospital, Brisbane, Australia.
Rajandram R; Department of Surgery, Faculty of Medicine, University of Malaya, Kuala Lumpur, Wilayah Persekutuan, Malaysia.
Blakeney JS; Centre for Inflammation and Disease Research, Institute for Molecular Bioscience, The University of Queensland, Brisbane, QLD, Australia.
Iyer A; Centre for Inflammation and Disease Research, Institute for Molecular Bioscience, The University of Queensland, Brisbane, QLD, Australia.
Suen JY; Centre for Inflammation and Disease Research, Institute for Molecular Bioscience, The University of Queensland, Brisbane, QLD, Australia.
Johnson DW; Centre for Kidney Disease Research, The University of Queensland, Translational Research Institute, Brisbane, Australia.; Department of Nephrology, Princess Alexandra Hospital, Brisbane, Australia.
Gobe GC; Centre for Kidney Disease Research, The University of Queensland, Translational Research Institute, Brisbane, Australia.
Fairlie DP; Centre for Inflammation and Disease Research, Institute for Molecular Bioscience, The University of Queensland, Brisbane, QLD, Australia.
Vesey DA; Centre for Kidney Disease Research, The University of Queensland, Translational Research Institute, Brisbane, Australia.; Department of Nephrology, Princess Alexandra Hospital, Brisbane, Australia.
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Źródło:
PloS one [PLoS One] 2021 Mar 25; Vol. 16 (3), pp. e0248983. Date of Electronic Publication: 2021 Mar 25 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Carcinoma, Renal Cell/*metabolism
Carcinoma, Renal Cell/*pathology
Kidney Neoplasms/*metabolism
Kidney Neoplasms/*pathology
Receptor, PAR-2/*metabolism
Biopsy ; Calcium/metabolism ; Carcinoma, Renal Cell/genetics ; Cell Line, Tumor ; Epithelial Cells/metabolism ; Gene Expression Regulation, Neoplastic ; Humans ; Kidney Neoplasms/genetics ; Kidney Tubules/pathology ; Receptor, PAR-2/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
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