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Wyświetlanie 1-20 z 111
Tytuł:
Phospho-Tyr705 of STAT3 is a therapeutic target for sepsis through regulating inflammation and coagulation.
Autorzy:
Xu S; Department of Intensive Care Unit, The First Affiliated Hospital of Wenzhou Medical University, Nanbaixiang road, Wenzhou, Zhejiang, 325000, P.R. China.
Pan X; Department of Intensive Care Unit, The First Affiliated Hospital of Wenzhou Medical University, Nanbaixiang road, Wenzhou, Zhejiang, 325000, P.R. China.
Mao L; Department of Intensive Care Unit, The First Affiliated Hospital of Wenzhou Medical University, Nanbaixiang road, Wenzhou, Zhejiang, 325000, P.R. China.
Pan H; Wenzhou Medical University, Wenzhou, Zhejiang, P.R. China.
Xu W; Department of Intensive Care Unit, The First Affiliated Hospital of Wenzhou Medical University, Nanbaixiang road, Wenzhou, Zhejiang, 325000, P.R. China.
Hu Y; Department of Intensive Care Unit, The First Affiliated Hospital of Wenzhou Medical University, Nanbaixiang road, Wenzhou, Zhejiang, 325000, P.R. China.
Yu X; Department of Intensive Care Unit, The First Affiliated Hospital of Wenzhou Medical University, Nanbaixiang road, Wenzhou, Zhejiang, 325000, P.R. China.
Chen Z; Department of Intensive Care Unit, The First Affiliated Hospital of Wenzhou Medical University, Nanbaixiang road, Wenzhou, Zhejiang, 325000, P.R. China.
Qian S; Department of Intensive Care Unit, The First Affiliated Hospital of Wenzhou Medical University, Nanbaixiang road, Wenzhou, Zhejiang, 325000, P.R. China.
Ye Y; Department of Blood Transfusion, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, P.R. China.
Huang Y; Department of Intensive Care Unit, The First Affiliated Hospital of Wenzhou Medical University, Nanbaixiang road, Wenzhou, Zhejiang, 325000, P.R. China.
Pan J; Department of Intensive Care Unit, The First Affiliated Hospital of Wenzhou Medical University, Nanbaixiang road, Wenzhou, Zhejiang, 325000, P.R. China. .
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Źródło:
Cell communication and signaling : CCS [Cell Commun Signal] 2020 Jul 08; Vol. 18 (1), pp. 104. Date of Electronic Publication: 2020 Jul 08.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Blood Coagulation*/drug effects
Molecular Targeted Therapy*
Inflammation/*blood
Inflammation/*complications
Phosphotyrosine/*metabolism
STAT3 Transcription Factor/*metabolism
Sepsis/*blood
Sepsis/*complications
Aminosalicylic Acids ; Animals ; Benzofurans/pharmacology ; Cecum/pathology ; Cell Membrane Permeability/drug effects ; Endothelial Cells/drug effects ; Endothelial Cells/metabolism ; Humans ; Inflammation Mediators/metabolism ; Ligation ; Lipopolysaccharides ; Macrophages/drug effects ; Macrophages/metabolism ; Male ; Mice ; Mice, Inbred C57BL ; Models, Biological ; Naphthoquinones/pharmacology ; Punctures ; RAW 264.7 Cells ; Sulfonamides ; Suppression, Genetic/drug effects ; Survival Analysis ; Thromboplastin/metabolism ; Toll-Like Receptor 4/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Tim-3 regulates inflammatory cytokine expression and Th17 cell response induced by monocytes from patients with chronic hepatitis B.
Autorzy:
Wang J; Department of Infectious Disease, The Affiliated Hospital of Xuzhou Medical University, Xuzhou, China.
Li C; Department of Infectious Disease, The Affiliated Hospital of Xuzhou Medical University, Xuzhou, China.
Fu J; Department of Infectious Disease, The Affiliated Hospital of Xuzhou Medical University, Xuzhou, China.
Wang X; Department of Infectious Disease, The Affiliated Hospital of Xuzhou Medical University, Xuzhou, China.
Feng X; Central Laboratory of the Affiliated Hospital of Xuzhou Medical University, Xuzhou, China.
Pan X; Department of Infectious Disease, The Affiliated Hospital of Xuzhou Medical University, Xuzhou, China.
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Źródło:
Scandinavian journal of immunology [Scand J Immunol] 2019 May; Vol. 89 (5), pp. e12755. Date of Electronic Publication: 2019 Mar 18.
Typ publikacji:
Journal Article
MeSH Terms:
Hepatitis A Virus Cellular Receptor 2/*metabolism
Hepatitis B virus/*physiology
Hepatitis B, Chronic/*immunology
Inflammation/*immunology
Monocytes/*immunology
Th17 Cells/*immunology
Adult ; Cells, Cultured ; Cytokines/metabolism ; Female ; Galectins/metabolism ; Hepatitis A Virus Cellular Receptor 2/genetics ; Humans ; Immunologic Memory ; Immunomodulation ; Inflammation Mediators/metabolism ; Male ; Middle Aged ; Signal Transduction ; Up-Regulation ; Young Adult
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Sustained elevation of NF-κB activity sensitizes offspring of maternal inflammation to hypertension via impairing PGC-1α recovery.
Autorzy:
Deng Y; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Zhang Q; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Luo H; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Chen X; Diagosis and Treatment Center for Servicemen, Southwest Hospital, Third Military Medical University, Chongqing, China.
Han Q; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Wang F; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Huang P; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Lai W; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Guan X; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Pan X; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Ji Y; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Guo W; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Che L; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Tang Y; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Gu L; The Center for Disease Control and Prevention of Chengdu Military Command, Chengdu, China.
Yu J; Division of Hematology, Department of Internal Medicine, The Ohio State University, Columbus, Ohio, USA.
Namaka M; Colleges of Pharmacy and Medicine, University of Manitoba, Winnipeg, MB, Canada.; Joint Laboratory of Biological Psychiatry Between Shantou University Medical College and the College of Medicine University of Manitoba, Shantou, China.
Deng Y; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
Li X; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing, China.
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Źródło:
Scientific reports [Sci Rep] 2016 Sep 12; Vol. 6, pp. 32642. Date of Electronic Publication: 2016 Sep 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Hypertension/*physiopathology
Inflammation/*physiopathology
NF-kappa B/*genetics
Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha/*genetics
Animals ; Antioxidants/metabolism ; Aorta, Thoracic/metabolism ; Aorta, Thoracic/pathology ; Blood Pressure/genetics ; Desoxycorticosterone/metabolism ; Disease Models, Animal ; Female ; Gene Expression Regulation ; Humans ; Hypertension/chemically induced ; Hypertension/complications ; Hypertension/genetics ; Inflammation/chemically induced ; Inflammation/complications ; Inflammation/genetics ; Lipopolysaccharides/toxicity ; Maternal Exposure ; Rats ; Reactive Oxygen Species/metabolism ; Signal Transduction/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Maternal inflammation activated ROS-p38 MAPK predisposes offspring to heart damages caused by isoproterenol via augmenting ROS generation.
Autorzy:
Zhang Q; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Deng Y; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Lai W; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Guan X; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Sun X; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Han Q; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Wang F; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Pan X; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Ji Y; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Luo H; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Huang P; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Tang Y; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Gu L; The Center for Disease Control and Prevention of Chengdu Military Command, Chengdu 610021, China.
Dan G; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Yu J; Division of Hematology, Department of Internal Medicine, The Ohio State University, Columbus, Ohio 43210, USA.
Namaka M; Colleges of Pharmacy and Medicine, University of Manitoba, Apotex Center 750, McDermot Avenue, Winnipeg, R3E 0T5, MB, Canada.; Joint Laboratory of Biological Psychiatry between Shantou University Medical College and the College of Medicine University of Manitoba, Shantou 515063, China.
Zhang J; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Deng Y; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
Li X; Institute of Materia Medica, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.; Center of Translational Medicine, College of Pharmacy, Third Military Medical University, Chongqing 400038, China.
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Źródło:
Scientific reports [Sci Rep] 2016 Jul 22; Vol. 6, pp. 30146. Date of Electronic Publication: 2016 Jul 22.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Heart/*physiopathology
Heart Diseases/*metabolism
Inflammation/*metabolism
Reactive Oxygen Species/*metabolism
p38 Mitogen-Activated Protein Kinases/*metabolism
Animals ; Antioxidants/metabolism ; Female ; Heart/drug effects ; Heart Diseases/chemically induced ; Imidazoles/pharmacology ; Inflammation/chemically induced ; Isoproterenol/pharmacology ; Male ; NADPH Oxidases/metabolism ; Oxidative Stress/drug effects ; Oxidative Stress/physiology ; Pregnancy ; Pyridines/pharmacology ; Rats ; Rats, Sprague-Dawley ; Signal Transduction/drug effects ; Signal Transduction/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Natural Hirudin Increases Rat Flap Viability by Anti-Inflammation via PARs/p38/NF-κB Pathway.
Autorzy:
Peng L; Guangxi Medical University, Nanning, Guangxi 530021, China.
Pan X; Guangxi Medical University, Nanning, Guangxi 530021, China.
Yin G; Department of Plastic and Aesthetic Surgery, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi 530021, China.
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Źródło:
BioMed research international [Biomed Res Int] 2015; Vol. 2015, pp. 597264. Date of Electronic Publication: 2015 Dec 07.
Typ publikacji:
Journal Article
MeSH Terms:
Hirudins/*administration & dosage
Inflammation/*drug therapy
Signal Transduction/*drug effects
Skin/*drug effects
Animals ; Gene Expression Regulation/drug effects ; Humans ; Inflammation/genetics ; Inflammation/pathology ; Intercellular Adhesion Molecule-1/genetics ; Intercellular Adhesion Molecule-1/metabolism ; Interleukin-6/genetics ; Interleukin-6/metabolism ; Phosphorylation ; Rats ; Skin/metabolism ; Skin/pathology ; Transcription Factor RelA/genetics ; Transcription Factor RelA/metabolism ; Tumor Necrosis Factor-alpha/genetics ; Tumor Necrosis Factor-alpha/metabolism ; p38 Mitogen-Activated Protein Kinases/genetics ; p38 Mitogen-Activated Protein Kinases/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Regulation of microRNA-155 in atherosclerotic inflammatory responses by targeting MAP3K10.
Autorzy:
Zhu J; Department of Cardiology, First Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, People's Republic of China.
Chen T
Yang L
Li Z
Wong MM
Zheng X
Pan X
Zhang L
Yan H
Pokaż więcej
Źródło:
PloS one [PLoS One] 2012; Vol. 7 (11), pp. e46551. Date of Electronic Publication: 2012 Nov 26.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Atherosclerosis/*genetics
Inflammation/*genetics
MAP Kinase Kinase Kinases/*genetics
MicroRNAs/*genetics
Animals ; Apolipoproteins E/deficiency ; Apolipoproteins E/genetics ; Atherosclerosis/metabolism ; Atherosclerosis/pathology ; Biomarkers/metabolism ; Coronary Artery Disease/genetics ; Coronary Artery Disease/metabolism ; Cytokines/metabolism ; Gene Expression ; Gene Expression Regulation ; Humans ; Inflammation/metabolism ; Inflammation Mediators/metabolism ; Leukocytes, Mononuclear/metabolism ; Lipoproteins, LDL/metabolism ; MAP Kinase Kinase Kinases/metabolism ; MAP Kinase Signaling System ; Macrophages/metabolism ; Male ; Mice ; Mice, Knockout ; MicroRNAs/blood ; MicroRNAs/metabolism ; Plaque, Atherosclerotic/genetics ; Plaque, Atherosclerotic/metabolism ; Plaque, Atherosclerotic/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Exploring the Correlation Between the Systemic Immune Inflammation Index (SII), Systemic Inflammatory Response Index (SIRI), and Type 2 Diabetic Retinopathy
Autorzy:
Wang S
Pan X
Jia B
Chen S
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Temat:
systemic immune inflammation index
system inflammatory response index
type 2 diabetic retinopathy
inflammation
Specialties of internal medicine
RC581-951
Źródło:
Diabetes, Metabolic Syndrome and Obesity, Vol Volume 16, Pp 3827-3836 (2023)
Opis pliku:
electronic resource
Relacje:
https://www.dovepress.com/exploring-the-correlation-between-the-systemic-immune-inflammation-ind-peer-reviewed-fulltext-article-DMSO; https://doaj.org/toc/1178-7007
Dostęp URL:
https://doaj.org/article/9db20627ca634f84a0e628043580ba73  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Tytuł:
Effects of Semaglutide on Cardiac Protein Expression and Cardiac Function of Obese Mice
Autorzy:
Pan X
Yue L
Ban J
Ren L
Chen S
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Temat:
semaglutide
lipid metabolism
deps
obesity
inflammation
oxidative stress
Pathology
RB1-214
Therapeutics. Pharmacology
RM1-950
Źródło:
Journal of Inflammation Research, Vol Volume 15, Pp 6409-6425 (2022)
Opis pliku:
electronic resource
Relacje:
https://www.dovepress.com/effects-of-semaglutide-on-cardiac-protein-expression-and-cardiac-funct-peer-reviewed-fulltext-article-JIR; https://doaj.org/toc/1178-7031
Dostęp URL:
https://doaj.org/article/cb0b4e5467e04f319dad6c9c9a77b6fd  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Tytuł:
Bibliometric analysis of recent research on the association between TRPV1 and inflammation
Autorzy:
Pan Xu
Ru-Ru Shao
Yuan He
Pokaż więcej
Temat:
TRPV1 channel
inflammation
bibliometrics
CiteSpace
Vosviewer
Therapeutics. Pharmacology
RM1-950
Physiology
QP1-981
Źródło:
Channels, Vol 17, Iss 1 (2023)
Opis pliku:
electronic resource
Relacje:
https://doaj.org/toc/1933-6950; https://doaj.org/toc/1933-6969
Dostęp URL:
https://doaj.org/article/fb65581a065f469cbe866f6f3c0bbd56  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Wyświetlanie 1-20 z 111

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