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Tytuł :
Respiratory complex and tissue lineage drive recurrent mutations in tumour mtDNA.
Autorzy :
Gorelick AN; Computational Oncology Service, Memorial Sloan Kettering Cancer Center, New York, NY, USA.; Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
Kim M; Computational Oncology Service, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
Chatila WK; Computational Oncology Service, Memorial Sloan Kettering Cancer Center, New York, NY, USA.; Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center, New York, NY, USA.; Marie-Josée and Henry R. Kravis Center for Molecular Oncology, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
La K; Laboratory of Metabolic Regulation and Genetics, Rockefeller University, New York, NY, USA.
Hakimi AA; Urology Service, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
Berger MF; Marie-Josée and Henry R. Kravis Center for Molecular Oncology, Memorial Sloan Kettering Cancer Center, New York, NY, USA.; Department of Pathology, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
Taylor BS; Computational Oncology Service, Memorial Sloan Kettering Cancer Center, New York, NY, USA.; Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center, New York, NY, USA.; Marie-Josée and Henry R. Kravis Center for Molecular Oncology, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
Gammage PA; CRUK Beatson Institute, Glasgow, UK. .; Institute of Cancer Sciences, University of Glasgow, Glasgow, UK. .
Reznik E; Computational Oncology Service, Memorial Sloan Kettering Cancer Center, New York, NY, USA. .; Marie-Josée and Henry R. Kravis Center for Molecular Oncology, Memorial Sloan Kettering Cancer Center, New York, NY, USA. .; Urology Service, Memorial Sloan Kettering Cancer Center, New York, NY, USA. .
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Źródło :
Nature metabolism [Nat Metab] 2021 Apr; Vol. 3 (4), pp. 558-570. Date of Electronic Publication: 2021 Apr 08.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms :
Oxidative Phosphorylation*
Cell Lineage/*genetics
DNA, Mitochondrial/*genetics
DNA, Neoplasm/*genetics
Adenosine Triphosphate/biosynthesis ; Colorectal Neoplasms/genetics ; Exome/genetics ; Genome, Human/genetics ; Genome, Mitochondrial ; Genotype ; Humans ; Membrane Potential, Mitochondrial/physiology ; Mitochondria/genetics ; Mutation/genetics ; Phenotype ; RNA/genetics
Czasopismo naukowe
Tytuł :
Mitochondrial oxidative phosphorylation and mitophagy in myocardial ischaemia/reperfusion: effects of chloroquine.
Autorzy :
Dhanabalan K; Division of Medical Physiology, Department of Biomedical Sciences, Faculty of Medicine and Health Sciences, University of Stellenbosch, South Africa.
Huisamen B; Division of Medical Physiology, Department of Biomedical Sciences, Faculty of Medicine and Health Sciences, University of Stellenbosch, South Africa.
Lochner A; Division of Medical Physiology, Department of Biomedical Sciences, Faculty of Medicine and Health Sciences, University of Stellenbosch, South Africa. Email: .
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Źródło :
Cardiovascular journal of Africa [Cardiovasc J Afr] 2020 Jul/Aug; Vol. 31 (4), pp. 169-179. Date of Electronic Publication: 2019 Dec 12.
Typ publikacji :
Journal Article
MeSH Terms :
Chloroquine/*pharmacology
Mitochondria, Heart/*drug effects
Mitophagy/*drug effects
Myocardial Reperfusion Injury/*drug therapy
Myocytes, Cardiac/*drug effects
Oxidative Phosphorylation/*drug effects
Animals ; Disease Models, Animal ; Dynamins/metabolism ; Isolated Heart Preparation ; Male ; Mitochondria, Heart/metabolism ; Mitochondria, Heart/pathology ; Myocardial Reperfusion Injury/metabolism ; Myocardial Reperfusion Injury/pathology ; Myocytes, Cardiac/metabolism ; Myocytes, Cardiac/pathology ; Phosphorylation ; Protein Kinases/metabolism ; Rats, Wistar ; Sequestosome-1 Protein/metabolism ; Signal Transduction ; Time Factors ; Ubiquitin-Protein Ligases/metabolism ; rab GTP-Binding Proteins/metabolism
Czasopismo naukowe
Tytuł :
TXNIP contributes to bone loss via promoting the mitochondrial oxidative phosphorylation during glucocorticoid-induced osteoporosis.
Autorzy :
Mo Y; Department of Pharmacology, Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical University, Zhanjiang, Guangdong, China; Department of Orthopedics and Traumatology, Nanning Hospital of Traditional Chinese Medicine, Guangxi University of Chinese Medicine, Nanning, Guangxi, China.
Lai W; Department of Pharmacology, Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical University, Zhanjiang, Guangdong, China; Department of Phamacy, Yuebei people's Hospital, Shaoguan, Guangdong, China.
Zhong Y; Department of Pharmacology, Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical University, Zhanjiang, Guangdong, China.
Hu Z; Department of Endocrinology, The Second Affiliated Hospital of Guangzhou Medical University, Guangzhou, Guangdong, China.
You M; Xiamen Medical College, Xiamen, Fujian, China.
Du M; Department of Pharmacology, Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical University, Zhanjiang, Guangdong, China.
Wang P; Department of Pharmacology, Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical University, Zhanjiang, Guangdong, China.
Wu X; Department of Pharmacology, Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical University, Zhanjiang, Guangdong, China.
Chen C; Department of Pharmacology, Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical University, Zhanjiang, Guangdong, China.
He H; Department of Orthopedics and Traumatology, Nanning Hospital of Traditional Chinese Medicine, Guangxi University of Chinese Medicine, Nanning, Guangxi, China.
Gao Z; Department of Orthopedics and Traumatology, Nanning Hospital of Traditional Chinese Medicine, Guangxi University of Chinese Medicine, Nanning, Guangxi, China.
Xu Y; Xiamen Medical College, Xiamen, Fujian, China.
Wang D; Department of Traditional Chinese Medicine, Shenzhen Hospital, Southern Medical University, Shenzhen, Guangdong, China; Department of the Ministry of Science and Technology, Guangxi International Zhuang Medicine Hospital, Nanning, Guangxi 530201, China. Electronic address: .
Cui L; Department of Pharmacology, Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical University, Zhanjiang, Guangdong, China. Electronic address: .
Yang Y; Department of Pharmacology, Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical University, Zhanjiang, Guangdong, China. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2021 Feb 01; Vol. 266, pp. 118938. Date of Electronic Publication: 2020 Dec 19.
Typ publikacji :
Journal Article
MeSH Terms :
Oxidative Phosphorylation*
Bone Resorption/*etiology
Carrier Proteins/*physiology
Cell Cycle Proteins/*metabolism
Glucocorticoids/*toxicity
Mitochondria/*pathology
Osteoporosis/*pathology
Thioredoxins/*physiology
Animals ; Bone Resorption/metabolism ; Bone Resorption/pathology ; Cell Cycle Proteins/genetics ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Mitochondria/metabolism ; Osteoporosis/chemically induced ; Osteoporosis/metabolism ; Oxidative Stress ; Rats ; Rats, Sprague-Dawley ; Reactive Oxygen Species/metabolism ; Signal Transduction
Czasopismo naukowe
Tytuł :
Her4 promotes cancer metabolic reprogramming via the c-Myc-dependent signaling axis.
Autorzy :
Han J; Department of Orthopedics, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai Bone Tumor Institution, Shanghai 200080, PR China.
Zhang Y; Department of Orthopedics, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai Bone Tumor Institution, Shanghai 200080, PR China.
Xu J; Department of Orthopedics, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai Bone Tumor Institution, Shanghai 200080, PR China.
Zhang T; Department of Orthopedics, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai Bone Tumor Institution, Shanghai 200080, PR China.
Wang H; Department of Orthopedics, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai Bone Tumor Institution, Shanghai 200080, PR China.
Wang Z; Department of Orthopedics, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai Bone Tumor Institution, Shanghai 200080, PR China.
Jiang Y; Department of Orthopedics, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai Bone Tumor Institution, Shanghai 200080, PR China.
Zhou L; Department of Orthopedics, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai Bone Tumor Institution, Shanghai 200080, PR China.
Yang M; Department of Orthopedics, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai Bone Tumor Institution, Shanghai 200080, PR China.
Hua Y; Department of Orthopedics, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai Bone Tumor Institution, Shanghai 200080, PR China. Electronic address: .
Cai Z; Department of Orthopedics, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai Bone Tumor Institution, Shanghai 200080, PR China. Electronic address: .
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Źródło :
Cancer letters [Cancer Lett] 2021 Jan 01; Vol. 496, pp. 57-71. Date of Electronic Publication: 2020 Oct 07.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cellular Reprogramming*
Gene Expression Regulation, Neoplastic*
Metabolome*
Oxidative Phosphorylation*
Osteosarcoma/*pathology
Proto-Oncogene Proteins c-myc/*metabolism
Receptor, ErbB-4/*metabolism
Animals ; Apoptosis ; Biomarkers, Tumor/genetics ; Biomarkers, Tumor/metabolism ; Bone Neoplasms/genetics ; Bone Neoplasms/metabolism ; Bone Neoplasms/pathology ; Cell Proliferation ; Female ; Glycolysis ; Humans ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Osteosarcoma/genetics ; Osteosarcoma/metabolism ; Proto-Oncogene Proteins c-myc/genetics ; Receptor, ErbB-4/genetics ; Signal Transduction ; Tumor Cells, Cultured ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
Blue-Native Electrophoresis to Study the OXPHOS Complexes.
Autorzy :
Fernandez-Vizarra E; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK. .
Zeviani M; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK.
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Źródło :
Methods in molecular biology (Clifton, N.J.) [Methods Mol Biol] 2021; Vol. 2192, pp. 287-311.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Oxidative Phosphorylation*
Electrophoresis, Gel, Two-Dimensional/*methods
Mitochondria/*chemistry
Multiprotein Complexes/*chemistry
Native Polyacrylamide Gel Electrophoresis/*methods
Animals ; Blood Donors ; Humans ; Leukocytes/cytology ; Liver/cytology ; Mice ; Mice, Inbred C57BL ; Mitochondria/metabolism ; Mitochondrial Membranes/chemistry ; Mitochondrial Membranes/metabolism ; Multiprotein Complexes/metabolism ; Myocardium/cytology
Czasopismo naukowe
Tytuł :
Novel regulatory roles of UCP1 in osteoblasts.
Autorzy :
Mukherjee S; Department of Biotechnology, Daegu University, Gyeongsan, Gyeongbuk 38453, Republic of Korea.
Yun JW; Department of Biotechnology, Daegu University, Gyeongsan, Gyeongbuk 38453, Republic of Korea. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2021 Jul 01; Vol. 276, pp. 119427. Date of Electronic Publication: 2021 Mar 27.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Differentiation*
Osteogenesis*
Oxidative Phosphorylation*
Mesenchymal Stem Cells/*cytology
Osteoblasts/*physiology
Uncoupling Protein 1/*metabolism
Animals ; Autophagy ; Cells, Cultured ; Mice ; Osteoblasts/cytology ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Tytuł :
Simulated Microgravity Suppresses Osteogenic Differentiation of Mesenchymal Stem Cells by Inhibiting Oxidative Phosphorylation.
Autorzy :
Liu L; Key Laboratory of Biorheological Science and Technology, Ministry of Education, College of Bioengineering, Chongqing University, Chongqing 400030, China.
Cheng Y; Key Laboratory of Biorheological Science and Technology, Ministry of Education, College of Bioengineering, Chongqing University, Chongqing 400030, China.
Wang J; Key Laboratory of Biorheological Science and Technology, Ministry of Education, College of Bioengineering, Chongqing University, Chongqing 400030, China.
Ding Z; Key Laboratory of Biorheological Science and Technology, Ministry of Education, College of Bioengineering, Chongqing University, Chongqing 400030, China.
Halim A; Key Laboratory of Biorheological Science and Technology, Ministry of Education, College of Bioengineering, Chongqing University, Chongqing 400030, China.
Luo Q; Key Laboratory of Biorheological Science and Technology, Ministry of Education, College of Bioengineering, Chongqing University, Chongqing 400030, China.
Song G; Key Laboratory of Biorheological Science and Technology, Ministry of Education, College of Bioengineering, Chongqing University, Chongqing 400030, China.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2020 Dec 21; Vol. 21 (24). Date of Electronic Publication: 2020 Dec 21.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Differentiation*
Osteogenesis*
Oxidative Phosphorylation*
Mesenchymal Stem Cells/*cytology
Sirtuin 1/*metabolism
Weightlessness Simulation/*adverse effects
Animals ; Cells, Cultured ; Male ; Mesenchymal Stem Cells/physiology ; Rats ; Rats, Sprague-Dawley ; Signal Transduction ; Sirtuin 1/genetics
Czasopismo naukowe
Tytuł :
Dysfunctional Mitochondrial Dynamic and Oxidative Phosphorylation Precedes Cardiac Dysfunction in R120G-αB-Crystallin-Induced Desmin-Related Cardiomyopathy.
Autorzy :
Alam S; Department of Pathology and Translational Pathobiology Louisiana State University Health Sciences Center Shreveport LA.
Abdullah CS; Department of Pathology and Translational Pathobiology Louisiana State University Health Sciences Center Shreveport LA.
Aishwarya R; Department of Molecular and Cellular Physiology Louisiana State University Health Sciences Center Shreveport LA.
Morshed M; Department of Pathology and Translational Pathobiology Louisiana State University Health Sciences Center Shreveport LA.
Nitu SS; Department of Pathology and Translational Pathobiology Louisiana State University Health Sciences Center Shreveport LA.
Miriyala S; Department of Cellular Biology and Anatomy Louisiana State University Health Sciences Center Shreveport LA.
Panchatcharam M; Department of Cellular Biology and Anatomy Louisiana State University Health Sciences Center Shreveport LA.
Kevil CG; Department of Pathology and Translational Pathobiology Louisiana State University Health Sciences Center Shreveport LA.; Department of Molecular and Cellular Physiology Louisiana State University Health Sciences Center Shreveport LA.; Department of Cellular Biology and Anatomy Louisiana State University Health Sciences Center Shreveport LA.
Orr AW; Department of Pathology and Translational Pathobiology Louisiana State University Health Sciences Center Shreveport LA.; Department of Molecular and Cellular Physiology Louisiana State University Health Sciences Center Shreveport LA.; Department of Cellular Biology and Anatomy Louisiana State University Health Sciences Center Shreveport LA.
Bhuiyan MS; Department of Pathology and Translational Pathobiology Louisiana State University Health Sciences Center Shreveport LA.; Department of Molecular and Cellular Physiology Louisiana State University Health Sciences Center Shreveport LA.
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Źródło :
Journal of the American Heart Association [J Am Heart Assoc] 2020 Dec; Vol. 9 (23), pp. e017195. Date of Electronic Publication: 2020 Nov 19.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Oxidative Phosphorylation*
Cardiomyopathies/*etiology
Cardiomyopathies/*pathology
Mitochondrial Dynamics/*physiology
Animals ; Cardiomyopathies/metabolism ; Desmin ; Disease Models, Animal ; Mice ; Mice, Transgenic ; alpha-Crystallin B Chain
Czasopismo naukowe
Tytuł :
Targeting Mitochondrial Metabolism in Prostate Cancer with Triterpenoids.
Autorzy :
Mamouni K; Georgia Cancer Center, Augusta University, Augusta, GA 30912, USA.; Research Service, Charlie Norwood VA Medical Center, Augusta, GA 30904, USA.
Kallifatidis G; Georgia Cancer Center, Augusta University, Augusta, GA 30912, USA.; Research Service, Charlie Norwood VA Medical Center, Augusta, GA 30904, USA.; Department of Biological Sciences, Augusta University, Augusta, GA 30912, USA.
Lokeshwar BL; Georgia Cancer Center, Augusta University, Augusta, GA 30912, USA.; Research Service, Charlie Norwood VA Medical Center, Augusta, GA 30904, USA.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Feb 28; Vol. 22 (5). Date of Electronic Publication: 2021 Feb 28.
Typ publikacji :
Journal Article; Review
MeSH Terms :
Glycolysis*
Oxidative Phosphorylation*
Oxidative Stress*
Mitochondria/*drug effects
Prostatic Neoplasms/*drug therapy
Triterpenes/*pharmacology
Animals ; Humans ; Male ; Mitochondria/pathology ; Prostatic Neoplasms/metabolism ; Prostatic Neoplasms/pathology
Czasopismo naukowe
Tytuł :
Validation of a method evaluating T cell metabolic potential in compliance with ICH Q2 (R1).
Autorzy :
Mercier-Letondal P; Univ. Bourgogne Franche-Comté, INSERM, EFS BFC, UMR1098 RIGHT Interactions Greffon-Hôte-Tumeur/Ingénierie Cellulaire et Génique, 25000, Besançon, France. .
Marton C; Univ. Bourgogne Franche-Comté, INSERM, EFS BFC, UMR1098 RIGHT Interactions Greffon-Hôte-Tumeur/Ingénierie Cellulaire et Génique, 25000, Besançon, France.
Godet Y; Univ. Bourgogne Franche-Comté, INSERM, EFS BFC, UMR1098 RIGHT Interactions Greffon-Hôte-Tumeur/Ingénierie Cellulaire et Génique, 25000, Besançon, France.
Galaine J; Univ. Bourgogne Franche-Comté, INSERM, EFS BFC, UMR1098 RIGHT Interactions Greffon-Hôte-Tumeur/Ingénierie Cellulaire et Génique, 25000, Besançon, France.; Etablissement Français du Sang Bourgogne Franche-Comté, Activité Médicaments de Thérapie Innovante, 25000, Besançon, France.
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Źródło :
Journal of translational medicine [J Transl Med] 2021 Jan 06; Vol. 19 (1), pp. 21. Date of Electronic Publication: 2021 Jan 06.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Respiration*
Oxidative Phosphorylation*
Glucose ; Glycolysis ; Oxygen Consumption
Czasopismo naukowe
Tytuł :
Oxygen Consumption Evaluation: An Important Indicator of Metabolic State, Cellular Function, and Cell Fate Along Neural Deregulation.
Autorzy :
Brito MD; Department of Physiological Science, Santa Casa de São Paulo School of Medical Science. R. Doutor Cesário Motta Júnior, São Paulo, SP, Brazil.
Silva LFSE; Department of Physiological Science, Santa Casa de São Paulo School of Medical Science. R. Doutor Cesário Motta Júnior, São Paulo, SP, Brazil.
Siena A; Department of Physiological Science, Santa Casa de São Paulo School of Medical Science. R. Doutor Cesário Motta Júnior, São Paulo, SP, Brazil.
Chipara M; Institute of Cancer and Genomic Sciences, Institute of Biomedical Research, College of Medical and Dental Sciences, University of Birmingham, Birmingham, UK.
Sarkar S; Institute of Cancer and Genomic Sciences, Institute of Biomedical Research, College of Medical and Dental Sciences, University of Birmingham, Birmingham, UK.
Rosenstock TR; Department of Pharmacology, Institute of Biomedical Science, University of São Paulo, São Paulo, Brazil. .; Institute of Cancer and Genomic Sciences, Institute of Biomedical Research, College of Medical and dental Sciences, University of Birmingham, Birmingham, UK. .
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Źródło :
Methods in molecular biology (Clifton, N.J.) [Methods Mol Biol] 2021; Vol. 2240, pp. 207-230.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Biological Assay*
Oxidative Phosphorylation*/drug effects
Oxygen Consumption*/drug effects
Adenosine Triphosphate/*metabolism
Brain/*metabolism
Mitochondria/*metabolism
Neurons/*metabolism
Animals ; Brain/drug effects ; Cell Line ; Humans ; In Vitro Techniques ; Mitochondria/drug effects ; Neurons/drug effects ; Primary Cell Culture ; Rats ; Time Factors
Czasopismo naukowe
Tytuł :
Effect of dietary restriction and subsequent realimentation on hepatic oxidative phosphorylation in cattle.
Autorzy :
Keogh K; Animal and Bioscience Research Department, Animal and Grassland Research and Innovation Centre, Teagasc Grange, Dunsany, Co. Meath C15 PW93, Ireland.
McKenna C; Animal and Bioscience Research Department, Animal and Grassland Research and Innovation Centre, Teagasc Grange, Dunsany, Co. Meath C15 PW93, Ireland; School of Biochemistry & Immunology, Trinity College Dublin, Dublin 2 D02 R590, Ireland.
Porter RK; School of Biochemistry & Immunology, Trinity College Dublin, Dublin 2 D02 R590, Ireland.
Waters SM; Animal and Bioscience Research Department, Animal and Grassland Research and Innovation Centre, Teagasc Grange, Dunsany, Co. Meath C15 PW93, Ireland.
Kenny DA; Animal and Bioscience Research Department, Animal and Grassland Research and Innovation Centre, Teagasc Grange, Dunsany, Co. Meath C15 PW93, Ireland. Electronic address: .
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Źródło :
Animal : an international journal of animal bioscience [Animal] 2021 Jan; Vol. 15 (1), pp. 100009. Date of Electronic Publication: 2020 Dec 10.
Typ publikacji :
Journal Article
MeSH Terms :
Food Deprivation*
Oxidative Phosphorylation*
Animal Feed/analysis ; Animals ; Cattle ; Diet/veterinary ; Liver/metabolism ; Male
Czasopismo naukowe
Tytuł :
Mitochondrial oxidative phosphorylation in cutaneous melanoma.
Autorzy :
Kumar PR; Bob Champion Research and Education Building, Norwich Medical School, University of East Anglia, Norwich, UK.
Moore JA; Bob Champion Research and Education Building, Norwich Medical School, University of East Anglia, Norwich, UK.
Bowles KM; Bob Champion Research and Education Building, Norwich Medical School, University of East Anglia, Norwich, UK.; Department of Haematology, Norfolk and Norwich University Hospital, Norwich, UK.
Rushworth SA; Bob Champion Research and Education Building, Norwich Medical School, University of East Anglia, Norwich, UK. .
Moncrieff MD; Bob Champion Research and Education Building, Norwich Medical School, University of East Anglia, Norwich, UK. .; Department of Plastic and Reconstructive Surgery, Norfolk and Norwich University Hospital, Norwich, NR4 7UY, UK. .
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Źródło :
British journal of cancer [Br J Cancer] 2021 Jan; Vol. 124 (1), pp. 115-123. Date of Electronic Publication: 2020 Nov 18.
Typ publikacji :
Journal Article; Review
MeSH Terms :
Oxidative Phosphorylation*
Melanoma/*metabolism
Melanoma/*pathology
Mitochondria/*metabolism
Skin Neoplasms/*metabolism
Skin Neoplasms/*pathology
Tumor Microenvironment/*physiology
Animals ; Humans
SCR Disease Name :
Melanoma, Cutaneous Malignant
Czasopismo naukowe
Tytuł :
Mitochondrial protein gene expression and the oxidative phosphorylation pathway associated with feed efficiency and energy balance in dairy cattle.
Autorzy :
Dorji J; School of Applied Systems Biology, La Trobe University, Bundoora, Victoria, Australia, 3083; Agriculture Victoria, AgriBio, Centre for AgriBioscience, Bundoora, Victoria, Australia, 3083. Electronic address: .
MacLeod IM; Agriculture Victoria, AgriBio, Centre for AgriBioscience, Bundoora, Victoria, Australia, 3083.
Chamberlain AJ; Agriculture Victoria, AgriBio, Centre for AgriBioscience, Bundoora, Victoria, Australia, 3083.
Vander Jagt CJ; Agriculture Victoria, AgriBio, Centre for AgriBioscience, Bundoora, Victoria, Australia, 3083.
Ho PN; Agriculture Victoria, AgriBio, Centre for AgriBioscience, Bundoora, Victoria, Australia, 3083.
Khansefid M; Agriculture Victoria, AgriBio, Centre for AgriBioscience, Bundoora, Victoria, Australia, 3083.
Mason BA; Agriculture Victoria, AgriBio, Centre for AgriBioscience, Bundoora, Victoria, Australia, 3083.
Prowse-Wilkins CP; Agriculture Victoria, AgriBio, Centre for AgriBioscience, Bundoora, Victoria, Australia, 3083; Faculty of Veterinary and Agricultural Sciences, The University of Melbourne, Victoria, Australia, 3010.
Marett LC; Faculty of Veterinary and Agricultural Sciences, The University of Melbourne, Victoria, Australia, 3010; Agriculture Victoria, Ellinbank Dairy Centre, Ellinbank, Victoria, Australia, 3821.
Wales WJ; Faculty of Veterinary and Agricultural Sciences, The University of Melbourne, Victoria, Australia, 3010; Agriculture Victoria, Ellinbank Dairy Centre, Ellinbank, Victoria, Australia, 3821.
Cocks BG; School of Applied Systems Biology, La Trobe University, Bundoora, Victoria, Australia, 3083; Agriculture Victoria, AgriBio, Centre for AgriBioscience, Bundoora, Victoria, Australia, 3083.
Pryce JE; School of Applied Systems Biology, La Trobe University, Bundoora, Victoria, Australia, 3083; Agriculture Victoria, AgriBio, Centre for AgriBioscience, Bundoora, Victoria, Australia, 3083.
Daetwyler HD; School of Applied Systems Biology, La Trobe University, Bundoora, Victoria, Australia, 3083; Agriculture Victoria, AgriBio, Centre for AgriBioscience, Bundoora, Victoria, Australia, 3083.
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Źródło :
Journal of dairy science [J Dairy Sci] 2021 Jan; Vol. 104 (1), pp. 575-587. Date of Electronic Publication: 2020 Nov 06.
Typ publikacji :
Journal Article
MeSH Terms :
Animal Feed*
Oxidative Phosphorylation*
Cattle/*genetics
Mitochondrial Proteins/*genetics
Animals ; Cattle/metabolism ; Eating/genetics ; Energy Metabolism ; Female ; Gene Expression ; Genome ; Lactation ; Milk ; Phenotype ; Sequence Analysis, RNA/veterinary
Czasopismo naukowe
Tytuł :
Shear stress activates mitochondrial oxidative phosphorylation by reducing plasma membrane cholesterol in vascular endothelial cells.
Autorzy :
Yamamoto K; Laboratory of System Physiology, Department of Biomedical Engineering, Graduate School of Medicine, The University of Tokyo, 113-0033 Tokyo, Japan; .
Nogimori Y; Laboratory of System Physiology, Department of Biomedical Engineering, Graduate School of Medicine, The University of Tokyo, 113-0033 Tokyo, Japan.; Pediatrics, Reproductive, Developmental and Aging Sciences, Graduate School of Medicine, The University of Tokyo, 113-8655 Tokyo, Japan.
Imamura H; Laboratory of Functional Biology, Graduate School of Biostudies, Kyoto University, 606-8501 Kyoto, Japan.
Ando J; Laboratory of Biomedical Engineering, School of Medicine, Dokkyo Medical University, 321-0293 Tochigi, Japan .
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Źródło :
Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2020 Dec 29; Vol. 117 (52), pp. 33660-33667. Date of Electronic Publication: 2020 Dec 14.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Oxidative Phosphorylation*
Stress, Mechanical*
Cell Membrane/*metabolism
Cholesterol/*metabolism
Endothelial Cells/*metabolism
Mitochondria/*metabolism
Adenosine Triphosphate/metabolism ; Aorta/cytology ; Biosensing Techniques ; Endocytosis ; Humans ; Lung/blood supply ; Mutation/genetics ; beta-Cyclodextrins/pharmacology
Czasopismo naukowe
Tytuł :
Why All the Fuss about Oxidative Phosphorylation (OXPHOS)?
Autorzy :
Xu Y; Department of Medicinal Chemistry, College of Pharmacy, Rogel Cancer Center, University of Michigan, Ann Arbor, Michigan 48109, United States.
Xue D; Department of Medicinal Chemistry, College of Pharmacy, Rogel Cancer Center, University of Michigan, Ann Arbor, Michigan 48109, United States.
Bankhead A 3rd; Department of Computational Medicine and Bioinformatics, University of Michigan Medical School, Ann Arbor, Michigan 48109, United States.; Department of Biostatistics, University of Michigan, School of Public Health, Ann Arbor, Michigan 48109, United States.
Neamati N; Department of Medicinal Chemistry, College of Pharmacy, Rogel Cancer Center, University of Michigan, Ann Arbor, Michigan 48109, United States.
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Źródło :
Journal of medicinal chemistry [J Med Chem] 2020 Dec 10; Vol. 63 (23), pp. 14276-14307. Date of Electronic Publication: 2020 Oct 26.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Review
MeSH Terms :
Oxidative Phosphorylation*
Animals ; Binding Sites ; Humans ; Molecular Structure ; Mutation ; Neoplastic Stem Cells/metabolism ; Tumor Microenvironment ; Up-Regulation
Czasopismo naukowe
Tytuł :
Benefits of BCG-induced metabolic switch from oxidative phosphorylation to aerobic glycolysis in autoimmune and nervous system diseases.
Autorzy :
Faustman DL; From the, Department of Medicine, Immunobiology Laboratories, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.
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Źródło :
Journal of internal medicine [J Intern Med] 2020 Dec; Vol. 288 (6), pp. 641-650. Date of Electronic Publication: 2020 Mar 18.
Typ publikacji :
Journal Article; Review
MeSH Terms :
Glycolysis*
Oxidative Phosphorylation*
BCG Vaccine/*therapeutic use
Central Nervous System Diseases/*drug therapy
Central Nervous System Diseases/*metabolism
Diabetes Mellitus, Type 1/*drug therapy
Diabetes Mellitus, Type 1/*metabolism
BCG Vaccine/administration & dosage ; BCG Vaccine/immunology ; Central Nervous System Diseases/immunology ; Diabetes Mellitus, Type 1/complications ; Diabetes Mellitus, Type 1/immunology ; Drug Administration Schedule ; Humans ; Hypoglycemia/prevention & control ; Islets of Langerhans/physiology ; Regeneration ; Tuberculosis/immunology ; Tuberculosis/prevention & control
Czasopismo naukowe
Tytuł :
Metabolic rewiring in drug resistant cells exhibit higher OXPHOS and fatty acids as preferred major source to cellular energetics.
Autorzy :
Salunkhe S; Shilpee Dutt Laboratory, Tata Memorial Centre, Advanced Centre for Treatment, Research and Education in Cancer (ACTREC), Kharghar, Navi Mumbai 410210, India; Homi Bhabha National Institute, Training School Complex, Anushakti Nagar, Mumbai 400085, India.
Mishra SV; Shilpee Dutt Laboratory, Tata Memorial Centre, Advanced Centre for Treatment, Research and Education in Cancer (ACTREC), Kharghar, Navi Mumbai 410210, India; Homi Bhabha National Institute, Training School Complex, Anushakti Nagar, Mumbai 400085, India.
Ghorai A; Shilpee Dutt Laboratory, Tata Memorial Centre, Advanced Centre for Treatment, Research and Education in Cancer (ACTREC), Kharghar, Navi Mumbai 410210, India.
Hole A; Chilakapati Laboratory, Tata Memorial Centre, Advanced Centre for Treatment, Research and Education in Cancer (ACTREC), Kharghar, Navi Mumbai 410210, India.
Chandrani P; Integrated Genomics Laboratory, Advanced Centre for Treatment Research Education in Cancer (ACTREC), India.
Dutt A; Integrated Genomics Laboratory, Advanced Centre for Treatment Research Education in Cancer (ACTREC), India; Homi Bhabha National Institute, Training School Complex, Anushakti Nagar, Mumbai 400085, India.
Chilakapati M; Chilakapati Laboratory, Tata Memorial Centre, Advanced Centre for Treatment, Research and Education in Cancer (ACTREC), Kharghar, Navi Mumbai 410210, India; Homi Bhabha National Institute, Training School Complex, Anushakti Nagar, Mumbai 400085, India.
Dutt S; Shilpee Dutt Laboratory, Tata Memorial Centre, Advanced Centre for Treatment, Research and Education in Cancer (ACTREC), Kharghar, Navi Mumbai 410210, India; Homi Bhabha National Institute, Training School Complex, Anushakti Nagar, Mumbai 400085, India. Electronic address: .
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Źródło :
Biochimica et biophysica acta. Bioenergetics [Biochim Biophys Acta Bioenerg] 2020 Dec 01; Vol. 1861 (12), pp. 148300. Date of Electronic Publication: 2020 Aug 25.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Drug Resistance, Neoplasm*/drug effects
Energy Metabolism*/drug effects
Oxidative Phosphorylation*/drug effects
Fatty Acids/*metabolism
Cell Proliferation/drug effects ; Cell Respiration/drug effects ; Cell Survival/drug effects ; Gene Expression Regulation, Leukemic/drug effects ; Glycolysis/drug effects ; HL-60 Cells ; Humans ; Lipids/analysis ; Metabolic Networks and Pathways/drug effects ; Mitoxantrone/pharmacology ; Oxygen Consumption/drug effects ; THP-1 Cells
Czasopismo naukowe
Tytuł :
Mitochondrial Damage and the Road to Exhaustion.
Autorzy :
Li W; Suzhou Institute of Systems Medicine, Suzhou, Jiangsu 215123, China; Center for Systems Medicine, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences and Peking Union Medical College, 100005 Beijing, China.
Cheng H; Suzhou Institute of Systems Medicine, Suzhou, Jiangsu 215123, China; Center for Systems Medicine, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences and Peking Union Medical College, 100005 Beijing, China; Key Laboratory of Synthetic Biology Regulatory Element, Chinese Academy of Medical Sciences, Beijing, China.
Li G; Suzhou Institute of Systems Medicine, Suzhou, Jiangsu 215123, China; Center for Systems Medicine, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences and Peking Union Medical College, 100005 Beijing, China; Key Laboratory of Synthetic Biology Regulatory Element, Chinese Academy of Medical Sciences, Beijing, China. Electronic address: .
Zhang L; Suzhou Institute of Systems Medicine, Suzhou, Jiangsu 215123, China; Center for Systems Medicine, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences and Peking Union Medical College, 100005 Beijing, China. Electronic address: .
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Źródło :
Cell metabolism [Cell Metab] 2020 Dec 01; Vol. 32 (6), pp. 905-907.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't; Comment
MeSH Terms :
Oxidative Phosphorylation*
T-Lymphocytes*
Mitochondria/metabolism ; Mitophagy ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Tytuł :
Histone H2AX promotes neuronal health by controlling mitochondrial homeostasis.
Autorzy :
Weyemi U; The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205.
Paul BD; The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205.
Bhattacharya D; The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205.
Malla AP; The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205.
Boufraqech M; Endocrine Oncology Branch, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892.
Harraz MM; The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205.
Bonner WM; Developmental Therapeutics Branch, Laboratory of Molecular Pharmacology, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892.
Snyder SH; The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205; .; Department of Psychiatry and Behavioral Sciences, Johns Hopkins University School of Medicine, Baltimore, MD 21205.; Department of Pharmacology and Molecular Sciences, Johns Hopkins University School of Medicine, Baltimore, MD 21205.
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Źródło :
Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2019 Apr 09; Vol. 116 (15), pp. 7471-7476. Date of Electronic Publication: 2019 Mar 25.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Oxidative Phosphorylation*
Histones/*metabolism
Mitochondria/*metabolism
Neural Stem Cells/*metabolism
Neurons/*metabolism
Animals ; Cell Death ; Electron Transport/physiology ; Histones/genetics ; Mice ; Mice, Knockout ; Mitochondria/genetics ; Neural Stem Cells/cytology ; Neurons/cytology ; Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha/genetics ; Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha/metabolism ; Phosphorylation
Czasopismo naukowe

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