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Wyświetlanie 1-7 z 7
Tytuł :
Autophagy activated by silibinin contributes to glioma cell death via induction of oxidative stress-mediated BNIP3-dependent nuclear translocation of AIF.
Autorzy :
Wang C; Department of Neurosurgery, First Hospital of Jilin University, 130021, Changchun, China.; Research Center of Neuroscience, First Hospital of Jilin University, 130021, Changchun, China.
He C; Department of Neurosurgery, First Hospital of Jilin University, 130021, Changchun, China.; Research Center of Neuroscience, First Hospital of Jilin University, 130021, Changchun, China.
Lu S; Department of Neurosurgery, First Hospital of Jilin University, 130021, Changchun, China.; Research Center of Neuroscience, First Hospital of Jilin University, 130021, Changchun, China.
Wang X; Department of Neurosurgery, First Hospital of Jilin University, 130021, Changchun, China.; Research Center of Neuroscience, First Hospital of Jilin University, 130021, Changchun, China.
Wang L; Department of Neurosurgery, First Hospital of Jilin University, 130021, Changchun, China.; Research Center of Neuroscience, First Hospital of Jilin University, 130021, Changchun, China.
Liang S; Department of Neurosurgery, First Hospital of Jilin University, 130021, Changchun, China.; Research Center of Neuroscience, First Hospital of Jilin University, 130021, Changchun, China.
Wang X; Department of Radiotherapy, Second Hospital of Jilin University, 130021, Changchun, China.
Piao M; Department of Anesthesiology, First Hospital of Jilin University, 130021, Changchun, China.
Cui J; Department of Histology and Embryology, College of Basic Medical Sciences, Jilin University, 130021, Changchun, China.
Chi G; Key Laboratory of Pathobiology, Ministry of Education, Jilin University, 130021, Changchun, China.
Ge P; Department of Neurosurgery, First Hospital of Jilin University, 130021, Changchun, China. .; Research Center of Neuroscience, First Hospital of Jilin University, 130021, Changchun, China. .
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Źródło :
Cell death & disease [Cell Death Dis] 2020 Aug 14; Vol. 11 (8), pp. 630. Date of Electronic Publication: 2020 Aug 14.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis Inducing Factor/*metabolism
Autophagy/*drug effects
Cell Nucleus/*metabolism
Glioma/*metabolism
Glioma/*pathology
Membrane Proteins/*metabolism
Oxidative Stress/*drug effects
Proto-Oncogene Proteins/*metabolism
Silybin/*pharmacology
Animals ; Cell Death/drug effects ; Cell Line, Tumor ; Cell Nucleus/drug effects ; Cell Proliferation/drug effects ; Female ; Glutathione/metabolism ; Glycolysis/drug effects ; Humans ; Hydrogen Peroxide/metabolism ; Mice, Inbred BALB C ; Mice, Nude ; Mitochondria/drug effects ; Mitochondria/metabolism ; Mitochondria/pathology ; Models, Biological ; Phosphorylation/drug effects ; Protein Transport/drug effects ; Silybin/chemistry ; Tumor Suppressor Protein p53/metabolism
Czasopismo naukowe
Tytuł :
MLKL contributes to shikonin-induced glioma cell necroptosis via promotion of chromatinolysis.
Autorzy :
Ding Y; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
He C; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Lu S; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Wang X; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Wang C; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Wang L; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Zhang J; Department of Neurosurgery, Sun Yat-sen University Cancer Center, State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine, Guangzhou, 510060, China.
Piao M; Department of Anesthesiology, First Hospital of Jilin University, Changchun, 130021, China.
Chi G; Key Laboratory of Pathobiology, Ministry of Education, Jilin University, Changchun, 130021, China.
Luo Y; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Sai K; Department of Neurosurgery, Sun Yat-sen University Cancer Center, State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine, Guangzhou, 510060, China. Electronic address: .
Ge P; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China. Electronic address: .
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Źródło :
Cancer letters [Cancer Lett] 2019 Dec 28; Vol. 467, pp. 58-71. Date of Electronic Publication: 2019 Sep 24.
Typ publikacji :
Journal Article
MeSH Terms :
DNA, Neoplasm/*chemistry
Glioma/*drug therapy
Naphthoquinones/*administration & dosage
Protein Kinases/*metabolism
Animals ; Apoptosis Inducing Factor/metabolism ; Cell Line, Tumor ; DNA Fragmentation ; DNA, Neoplasm/drug effects ; Gene Expression Regulation, Neoplastic/drug effects ; Glioma/genetics ; Glioma/metabolism ; Humans ; Mice ; Mitochondria/metabolism ; Naphthoquinones/pharmacology ; Necroptosis ; Rats ; Reactive Oxygen Species/metabolism ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
RSL3 induced autophagic death in glioma cells via causing glycolysis dysfunction.
Autorzy :
Wang X; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Lu S; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
He C; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Wang C; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Wang L; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Piao M; Department of Anesthesiology, First Hospital of Jilin University, Changchun, 130021, China.
Chi G; Key Laboratory of Pathobiology, Ministry of Education, Jilin University, Changchun, 130021, China.
Luo Y; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Ge P; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2019 Oct 20; Vol. 518 (3), pp. 590-597. Date of Electronic Publication: 2019 Aug 21.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Antineoplastic Agents/*pharmacology
Autophagic Cell Death/*drug effects
Carbolines/*pharmacology
Glioma/*drug therapy
Glycolysis/*drug effects
Animals ; Antineoplastic Agents/therapeutic use ; Carbolines/therapeutic use ; Cell Line, Tumor ; Enzyme Inhibitors/pharmacology ; Glioma/metabolism ; Glioma/pathology ; Glutathione Peroxidase/antagonists & inhibitors ; Glutathione Peroxidase/metabolism ; Humans ; Mice, Inbred BALB C ; Mice, Nude ; Rats
Czasopismo naukowe
Tytuł :
Pseudolaric acid B triggers ferroptosis in glioma cells via activation of Nox4 and inhibition of xCT.
Autorzy :
Wang Z; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Ding Y; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Wang X; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Lu S; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Wang C; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
He C; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Wang L; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Piao M; Department of Anesthesiology, First Hospital of Jilin University, Changchun, 130021, China.
Chi G; Key Laboratory of Pathobiology, Ministry of Education, Jilin University, Changchun, 130021, China.
Luo Y; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Ge P; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China. Electronic address: .
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Źródło :
Cancer letters [Cancer Lett] 2018 Aug 01; Vol. 428, pp. 21-33. Date of Electronic Publication: 2018 Apr 24.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Brain Neoplasms/*drug therapy
Diterpenes/*pharmacology
Glioma/*drug therapy
Lipid Peroxidation/*drug effects
Amino Acid Transport System y+/metabolism ; Animals ; Brain Neoplasms/pathology ; Cell Line, Tumor/transplantation ; Cyclohexylamines/pharmacology ; Disease Models, Animal ; Diterpenes/therapeutic use ; Down-Regulation/drug effects ; Gene Expression Regulation, Neoplastic/drug effects ; Glioma/pathology ; Glutathione/metabolism ; Humans ; Hydrogen Peroxide/metabolism ; Iron/metabolism ; Mice ; Mice, Nude ; Mitochondria/drug effects ; Mitochondria/metabolism ; NADPH Oxidase 4/metabolism ; Phenylenediamines/pharmacology ; Rats ; Reactive Oxygen Species/metabolism ; Up-Regulation/drug effects
Czasopismo naukowe
Tytuł :
RIP1 and RIP3 contribute to shikonin-induced glycolysis suppression in glioma cells via increase of intracellular hydrogen peroxide.
Autorzy :
Lu B; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Wang Z; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Ding Y; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Wang X; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Lu S; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Wang C; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
He C; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Piao M; Department of Anesthesiology, First Hospital of Jilin University, Changchun, 130021, China.
Chi G; Key Laboratory of Pathobiology, Ministry of Education, Jilin University, Changchun, 130021, China.
Luo Y; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China.
Ge P; Department of Neurosurgery, First Hospital of Jilin University, Changchun, 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun, 130021, China. Electronic address: .
Pokaż więcej
Źródło :
Cancer letters [Cancer Lett] 2018 Jul 01; Vol. 425, pp. 31-42. Date of Electronic Publication: 2018 Mar 30.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Glioma/*drug therapy
Glycolysis/*drug effects
Hydrogen Peroxide/*metabolism
Naphthoquinones/*administration & dosage
Nuclear Pore Complex Proteins/*metabolism
RNA-Binding Proteins/*metabolism
Receptor-Interacting Protein Serine-Threonine Kinases/*metabolism
Animals ; Cell Line, Tumor ; Cysteine/metabolism ; Gene Expression Regulation, Neoplastic/drug effects ; Glioma/metabolism ; Glutathione/metabolism ; Humans ; Mice ; Naphthoquinones/pharmacology ; Rats ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
RIP1 and RIP3 contribute to shikonin-induced DNA double-strand breaks in glioma cells via increase of intracellular reactive oxygen species.
Autorzy :
Zhou Z; Department of Neurosurgery, First Hospital of Jilin University, Changchun 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun 130021, China.
Lu B; Department of Neurosurgery, First Hospital of Jilin University, Changchun 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun 130021, China.
Wang C; Department of Neurosurgery, First Hospital of Jilin University, Changchun 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun 130021, China.
Wang Z; Department of Neurosurgery, First Hospital of Jilin University, Changchun 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun 130021, China.
Luo T; Research Center of Neuroscience, First Hospital of Jilin University, Changchun 130021, China; Department of Neurology, First Hospital of Jilin University, Changchun 130021, China.
Piao M; Department of Anesthesiology, First Hospital of Jilin University, Changchun 130021, China.
Meng F; Department of Neurosurgery, People's Hospital of Jilin Province, Changchun 130021, China.
Chi G; Key Laboratory of Pathobiology, Ministry of Education, Jilin University, Changchun 130021, China.
Luo Y; Department of Neurosurgery, First Hospital of Jilin University, Changchun 130021, China.
Ge P; Department of Neurosurgery, First Hospital of Jilin University, Changchun 130021, China; Research Center of Neuroscience, First Hospital of Jilin University, Changchun 130021, China. Electronic address: .
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Źródło :
Cancer letters [Cancer Lett] 2017 Apr 01; Vol. 390, pp. 77-90. Date of Electronic Publication: 2017 Jan 17.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
DNA Breaks, Double-Stranded*/drug effects
Reactive Oxygen Species*
Glioma/*drug therapy
Nuclear Pore Complex Proteins/*metabolism
RNA-Binding Proteins/*metabolism
Receptor-Interacting Protein Serine-Threonine Kinases/*metabolism
Animals ; Cell Line, Tumor ; Disease Models, Animal ; Heterografts ; Mice ; Naphthoquinones/pharmacology ; Nuclear Pore Complex Proteins/genetics ; RNA-Binding Proteins/genetics ; Receptor-Interacting Protein Serine-Threonine Kinases/genetics ; Up-Regulation
Czasopismo naukowe
    Wyświetlanie 1-7 z 7

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