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Wyświetlanie 1-5 z 5
Tytuł:
NK cells link obesity-induced adipose stress to inflammation and insulin resistance.
Autorzy:
Wensveen FM; Department of Histology and Embryology, Faculty of Medicine, University of Rijeka, Rijeka, Croatia.
Jelenčić V; Department of Histology and Embryology, Faculty of Medicine, University of Rijeka, Rijeka, Croatia.
Valentić S; Department of Histology and Embryology, Faculty of Medicine, University of Rijeka, Rijeka, Croatia.
Šestan M; Department of Histology and Embryology, Faculty of Medicine, University of Rijeka, Rijeka, Croatia.
Wensveen TT; Department of Internal Medicine, University Hospital Rijeka, Rijeka, Croatia.
Theurich S; Max Planck Institute for Metabolism Research Cologne, Cologne, Germany.
Glasner A; The Lautenberg Center for General and Tumor Immunology, The Hebrew University Hadassah Medical School, Jerusalem, Israel.
Mendrila D; Department of Surgery, University Hospital Rijeka, Rijeka, Croatia.
Štimac D; Department of Internal Medicine, University Hospital Rijeka, Rijeka, Croatia.
Wunderlich FT; Max Planck Institute for Metabolism Research Cologne, Cologne, Germany.
Brüning JC; Max Planck Institute for Metabolism Research Cologne, Cologne, Germany.
Mandelboim O; The Lautenberg Center for General and Tumor Immunology, The Hebrew University Hadassah Medical School, Jerusalem, Israel.
Polić B; Department of Histology and Embryology, Faculty of Medicine, University of Rijeka, Rijeka, Croatia.
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Źródło:
Nature immunology [Nat Immunol] 2015 Apr; Vol. 16 (4), pp. 376-85. Date of Electronic Publication: 2015 Mar 02.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adipocytes/*immunology
Insulin Resistance/*immunology
Intra-Abdominal Fat/*immunology
Killer Cells, Natural/*immunology
Macrophages/*immunology
Obesity/*immunology
Adipocytes/pathology ; Animals ; Antigens, Ly/genetics ; Antigens, Ly/immunology ; Cell Differentiation ; Female ; Gene Expression Regulation ; Humans ; Inflammation/genetics ; Inflammation/immunology ; Inflammation/pathology ; Insulin/immunology ; Interferon-gamma/biosynthesis ; Interferon-gamma/immunology ; Intra-Abdominal Fat/pathology ; Killer Cells, Natural/pathology ; Macrophages/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Natural Cytotoxicity Triggering Receptor 1/genetics ; Natural Cytotoxicity Triggering Receptor 1/immunology ; Obesity/genetics ; Obesity/pathology ; Signal Transduction
Czasopismo naukowe
Tytuł:
Role of NKG2D in obesity-induced adipose tissue inflammation and insulin resistance.
Autorzy:
Chung JJ; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, Missouri, United States of America.
Markiewicz MA; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, Missouri, United States of America.
Polić B; Department of Histology and Embryology, University of Rijeka School of Medicine, Rijeka, Croatia.
Shaw AS; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, Missouri, United States of America; Howard Hughes Medical Institute, Washington University School of Medicine, St. Louis, Missouri, United States of America.
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Źródło:
PloS one [PLoS One] 2014 Oct 15; Vol. 9 (10), pp. e110108. Date of Electronic Publication: 2014 Oct 15 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Insulin Resistance/*genetics
NK Cell Lectin-Like Receptor Subfamily K/*genetics
Obesity/*complications
Obesity/*genetics
Panniculitis/*etiology
3T3-L1 Cells ; Adipocytes/drug effects ; Adipocytes/metabolism ; Adipose Tissue/metabolism ; Adipose Tissue/pathology ; Animals ; Disease Models, Animal ; Gene Expression ; Insulin/metabolism ; Insulin/pharmacology ; Ligands ; Male ; Mice ; Mice, Knockout ; NK Cell Lectin-Like Receptor Subfamily K/metabolism ; Nuclear Matrix-Associated Proteins/genetics ; Nuclear Matrix-Associated Proteins/metabolism ; Nucleocytoplasmic Transport Proteins/genetics ; Nucleocytoplasmic Transport Proteins/metabolism ; Obesity/immunology ; Obesity/metabolism ; Protein Binding ; Tumor Necrosis Factor-alpha/metabolism ; Tumor Necrosis Factor-alpha/pharmacology
Czasopismo naukowe
    Wyświetlanie 1-5 z 5

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