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Tytuł :
ROR2 has a protective role in melanoma by inhibiting Akt activity, cell-cycle progression, and proliferation.
Autorzy :
Castro MV; Centro de Estudios Biomédicos, Básicos, Aplicados y Desarrollo (CEBBAD), Universidad Maimónides, 1405, Buenos Aires, Argentina.; Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), 1425, Buenos Aires, Argentina.
Barbero GA; Centro de Estudios Biomédicos, Básicos, Aplicados y Desarrollo (CEBBAD), Universidad Maimónides, 1405, Buenos Aires, Argentina.; Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), 1425, Buenos Aires, Argentina.
Villanueva MB; Centro de Estudios Biomédicos, Básicos, Aplicados y Desarrollo (CEBBAD), Universidad Maimónides, 1405, Buenos Aires, Argentina.; Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), 1425, Buenos Aires, Argentina.
Grumolato L; INSERM U982, Institute for Research and Innovation in Biomedicine, University of Rouen, 76183, Rouen, France.
Nsengimana J; Biostatistics Research Group, Population Health Sciences Institute, Faculty of Medical Sciences, Newcastle University, Newcastle upon Tyne, NE2 4HH, UK.
Newton-Bishop J; Leeds Institute of Medical Research, Leeds, LS2 9JT, UK.
Illescas E; Centro de Estudios Biomédicos, Básicos, Aplicados y Desarrollo (CEBBAD), Universidad Maimónides, 1405, Buenos Aires, Argentina.
Quezada MJ; Centro de Estudios Biomédicos, Básicos, Aplicados y Desarrollo (CEBBAD), Universidad Maimónides, 1405, Buenos Aires, Argentina.; Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), 1425, Buenos Aires, Argentina.
Lopez-Bergami P; Centro de Estudios Biomédicos, Básicos, Aplicados y Desarrollo (CEBBAD), Universidad Maimónides, 1405, Buenos Aires, Argentina. .; Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), 1425, Buenos Aires, Argentina. .; Centro de Estudios Biomédicos, Biotecnológicos, Ambientales y Diagnóstico, Universidad Maimonides, Hidalgo 775, 6th Floor, Lab 602., 1405, Buenos Aires, Argentina. .
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Źródło :
Journal of biomedical science [J Biomed Sci] 2021 Nov 13; Vol. 28 (1), pp. 76. Date of Electronic Publication: 2021 Nov 13.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Cycle*
Cell Proliferation*
Melanoma/*genetics
Proto-Oncogene Proteins c-akt/*genetics
Receptor Tyrosine Kinase-like Orphan Receptors/*genetics
Proto-Oncogene Proteins c-akt/metabolism
Czasopismo naukowe
Tytuł :
MUSTN1 is an indispensable factor in the proliferation, differentiation and apoptosis of skeletal muscle satellite cells in chicken.
Autorzy :
Hu Z; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, 61130, China.
Xu H; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, 61130, China.
Lu Y; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, 61130, China.
He Q; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, 61130, China.
Yan C; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, 61130, China.
Zhao X; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, 61130, China.
Tian Y; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, 61130, China.
Yang C; Animal Breeding and Genetics Key Laboratory of Sichuan Province, Sichuan Animal Science Academy, Chengdu, 610066, China.
Zhang Z; Animal Breeding and Genetics Key Laboratory of Sichuan Province, Sichuan Animal Science Academy, Chengdu, 610066, China.
Qiu M; Animal Breeding and Genetics Key Laboratory of Sichuan Province, Sichuan Animal Science Academy, Chengdu, 610066, China. Electronic address: .
Wang Y; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, 61130, China. Electronic address: .
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Źródło :
Experimental cell research [Exp Cell Res] 2021 Oct 15; Vol. 407 (2), pp. 112833. Date of Electronic Publication: 2021 Sep 16.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis*
Cell Differentiation*
Cell Proliferation*
Gene Expression Regulation, Developmental*
Muscle Development*
Nuclear Proteins/*metabolism
Satellite Cells, Skeletal Muscle/*cytology
Animals ; Chickens ; MicroRNAs ; Nuclear Proteins/genetics ; Satellite Cells, Skeletal Muscle/metabolism
Czasopismo naukowe
Tytuł :
Salt Stress Promotes Abscisic Acid Accumulation to Affect Cell Proliferation and Expansion of Primary Roots in Rice.
Autorzy :
Huang Y; Biotechnology Research Institute, Chinese Academy of Agricultural Sciences, Beijing 100081, China.
Zhou J; Biotechnology Research Institute, Chinese Academy of Agricultural Sciences, Beijing 100081, China.
Li Y; Biotechnology Research Institute, Chinese Academy of Agricultural Sciences, Beijing 100081, China.
Quan R; Biotechnology Research Institute, Chinese Academy of Agricultural Sciences, Beijing 100081, China.; National Key Facility of Crop Gene Resources and Genetic Improvement, Beijing 100081, China.
Wang J; Biotechnology Research Institute, Chinese Academy of Agricultural Sciences, Beijing 100081, China.; National Key Facility of Crop Gene Resources and Genetic Improvement, Beijing 100081, China.
Huang R; Biotechnology Research Institute, Chinese Academy of Agricultural Sciences, Beijing 100081, China.; National Key Facility of Crop Gene Resources and Genetic Improvement, Beijing 100081, China.
Qin H; Biotechnology Research Institute, Chinese Academy of Agricultural Sciences, Beijing 100081, China.; National Key Facility of Crop Gene Resources and Genetic Improvement, Beijing 100081, China.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Oct 08; Vol. 22 (19). Date of Electronic Publication: 2021 Oct 08.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Proliferation*
Gene Expression Regulation, Plant*
Salt Stress*
Abscisic Acid/*metabolism
Oryza/*growth & development
Plant Proteins/*metabolism
Plant Roots/*growth & development
Oryza/genetics ; Oryza/metabolism ; Plant Proteins/genetics ; Plant Roots/genetics ; Plant Roots/metabolism
Czasopismo naukowe
Tytuł :
Lysophosphatidic Acid Mediates Imiquimod-Induced Psoriasis-like Symptoms by Promoting Keratinocyte Proliferation through LPAR1/ROCK2/PI3K/AKT Signaling Pathway.
Autorzy :
Kim D; Lee Gil Ya Cancer and Diabetes Institute, Gachon University, Incheon 21999, Korea.
Kim HJ; College of Pharmacy and Gachon Institute of Pharmaceutical Sciences, Gachon University, Incheon 21936, Korea.
Baek JO; Department of Dermatology, Gachon University College of Medicine, Gil Medical Center, Incheon 21565, Korea.
Roh JY; Department of Dermatology, Gachon University College of Medicine, Gil Medical Center, Incheon 21565, Korea.
Jun HS; Lee Gil Ya Cancer and Diabetes Institute, Gachon University, Incheon 21999, Korea.; College of Pharmacy and Gachon Institute of Pharmaceutical Sciences, Gachon University, Incheon 21936, Korea.; Gachon Medical Research Institute, Gil Hospital, Incheon 21565, Korea.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Oct 05; Vol. 22 (19). Date of Electronic Publication: 2021 Oct 05.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Proliferation*
Gene Expression Regulation/*drug effects
Imiquimod/*toxicity
Keratinocytes/*cytology
Lysophospholipids/*pharmacology
Psoriasis/*drug therapy
Animals ; Apoptosis ; Biomarkers/metabolism ; Cell Cycle ; Cells, Cultured ; Humans ; Interferon Inducers/toxicity ; Keratinocytes/metabolism ; Male ; Mice ; Mice, Inbred BALB C ; Phosphatidylinositol 3-Kinases/genetics ; Phosphatidylinositol 3-Kinases/metabolism ; Proto-Oncogene Proteins c-akt/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; Psoriasis/chemically induced ; Psoriasis/metabolism ; Psoriasis/pathology ; Receptors, Lysophosphatidic Acid/genetics ; Receptors, Lysophosphatidic Acid/metabolism ; rho-Associated Kinases/genetics ; rho-Associated Kinases/metabolism
Czasopismo naukowe
Tytuł :
circHIPK3 regulates proliferation and differentiation of myoblast through the miR-7/TCF12 pathway.
Autorzy :
Gao M; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
Li X; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
Yang Z; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
Zhao S; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
Ling X; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
Li J; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
Xing K; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
Qi X; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
Wang X; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
Xiao L; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
Ni H; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
Guo Y; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
Sheng X; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
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Źródło :
Journal of cellular physiology [J Cell Physiol] 2021 Oct; Vol. 236 (10), pp. 6793-6805. Date of Electronic Publication: 2021 Mar 21.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Differentiation*
Cell Proliferation*
Muscle Development*
Basic Helix-Loop-Helix Transcription Factors/*metabolism
MicroRNAs/*metabolism
Myoblasts, Skeletal/*metabolism
RNA, Circular/*metabolism
Animals ; Basic Helix-Loop-Helix Transcription Factors/genetics ; Cell Line ; Gene Expression Regulation ; Mice ; MicroRNAs/genetics ; RNA, Circular/genetics ; Signal Transduction
Czasopismo naukowe
Tytuł :
Knockout of Calcyclin Binding Protein Impedes the Growth of Breast Cancer Cells by Regulating Cell Apoptosis and β-Catenin Signaling.
Autorzy :
Wang N; The Second Department of Medicine Oncology, General Hospital of Ningxia Medical University, Yinchuan, China.
Wang Y; The First Department of Medicine Oncology, General Hospital of Ningxia Medical University, Yinchuan, China.
Wang H; The First Department of Medicine Oncology, General Hospital of Ningxia Medical University, Yinchuan, China.
Luo N; The Second Department of Medicine Oncology, General Hospital of Ningxia Medical University, Yinchuan, China.
Yang W; The Second Department of Medicine Oncology, General Hospital of Ningxia Medical University, Yinchuan, China.
Zhao Z; Clinical Laboratory Center, General Hospital of Ningxia Medical University, Yinchuan, China.; Ningxia Key Laboratory of Clinical and Pathogenic Microbiology, General Hospital of Ningxia Medical University, Yinchuan, China.
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Źródło :
DNA and cell biology [DNA Cell Biol] 2021 Oct; Vol. 40 (10), pp. 1317-1324. Date of Electronic Publication: 2021 Sep 29.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Cell Proliferation*
Breast Neoplasms/*metabolism
Calcium-Binding Proteins/*metabolism
Carcinoma/*metabolism
beta Catenin/*metabolism
Calcium-Binding Proteins/genetics ; Female ; Humans ; MCF-7 Cells ; Middle Aged ; Signal Transduction
Czasopismo naukowe
Tytuł :
Stimulation of vascular smooth muscle cell proliferation by stiff matrix via the IK Ca channel-dependent Ca signaling.
Autorzy :
Jia X; Beijing Advanced Innovation Centre for Biomedical Engineering, Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, China.; School of Engineering Medicine, Beihang University, No.37, Xueyuan Road, haidian district, Beijing, China.; School of Biomedical Sciences, Faculty of Biological Sciences, University of Leeds, Leeds, UK.
Yang Q; Beijing Advanced Innovation Centre for Biomedical Engineering, Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, China.; School of Engineering Medicine, Beihang University, No.37, Xueyuan Road, haidian district, Beijing, China.
Gao C; Beijing Advanced Innovation Centre for Biomedical Engineering, Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, China.; School of Engineering Medicine, Beihang University, No.37, Xueyuan Road, haidian district, Beijing, China.
Chen X; Beijing Advanced Innovation Centre for Biomedical Engineering, Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, China.; School of Engineering Medicine, Beihang University, No.37, Xueyuan Road, haidian district, Beijing, China.
Li Y; Beijing Advanced Innovation Centre for Biomedical Engineering, Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, China.; School of Engineering Medicine, Beihang University, No.37, Xueyuan Road, haidian district, Beijing, China.
Su H; Beijing Advanced Innovation Centre for Biomedical Engineering, Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, China.; School of Engineering Medicine, Beihang University, No.37, Xueyuan Road, haidian district, Beijing, China.
Zheng Y; Beijing Advanced Innovation Centre for Biomedical Engineering, Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, China.; School of Engineering Medicine, Beihang University, No.37, Xueyuan Road, haidian district, Beijing, China.
Zhang S; Beijing Advanced Innovation Centre for Biomedical Engineering, Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, China.; School of Engineering Medicine, Beihang University, No.37, Xueyuan Road, haidian district, Beijing, China.
Wang Z; Beijing Advanced Innovation Centre for Biomedical Engineering, Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, China.; School of Engineering Medicine, Beihang University, No.37, Xueyuan Road, haidian district, Beijing, China.
Wang H; Beijing Advanced Innovation Centre for Biomedical Engineering, Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, China.; School of Engineering Medicine, Beihang University, No.37, Xueyuan Road, haidian district, Beijing, China.
Jiang LH; School of Biomedical Sciences, Faculty of Biological Sciences, University of Leeds, Leeds, UK.; Sino-UK Joint Laboratory of Brain Function and Injury of Henan Province and Department of Physiology and Pathophysiology, Xinxiang Medical University, Xinxiang, China.
Sun Y; Beijing Advanced Innovation Centre for Biomedical Engineering, Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, China.; School of Engineering Medicine, Beihang University, No.37, Xueyuan Road, haidian district, Beijing, China.
Fan Y; Beijing Advanced Innovation Centre for Biomedical Engineering, Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, China.; School of Engineering Medicine, Beihang University, No.37, Xueyuan Road, haidian district, Beijing, China.
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Źródło :
Journal of cellular physiology [J Cell Physiol] 2021 Oct; Vol. 236 (10), pp. 6897-6906. Date of Electronic Publication: 2021 Mar 01.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Calcium Signaling*
Cell Proliferation*
Mechanotransduction, Cellular*
Dimethylpolysiloxanes/*chemistry
Intermediate-Conductance Calcium-Activated Potassium Channels/*metabolism
Muscle, Smooth, Vascular/*metabolism
Myocytes, Smooth Muscle/*metabolism
Animals ; Cell Culture Techniques ; Cell Line ; Extracellular Signal-Regulated MAP Kinases/metabolism ; Intermediate-Conductance Calcium-Activated Potassium Channels/genetics ; Rats
Czasopismo naukowe
Tytuł :
Metabolic flexibility maintains proliferation and migration of FGFR signaling-deficient lymphatic endothelial cells.
Autorzy :
Song H; Cardiovascular Biology Research Program, Oklahoma Medical Research Foundation, Oklahoma City, Oklahoma, USA.
Zhu J; Cardiovascular Biology Research Program, Oklahoma Medical Research Foundation, Oklahoma City, Oklahoma, USA.
Li P; Cardiovascular Biology Research Program, Oklahoma Medical Research Foundation, Oklahoma City, Oklahoma, USA.
Han F; Cardiovascular Biology Research Program, Oklahoma Medical Research Foundation, Oklahoma City, Oklahoma, USA.
Fang L; Department of Cardiovascular Sciences, Center for Cardiovascular Regeneration, Houston Methodist Research Institute, Houston, Texas, USA; Department of Ophthalmology, Blanton Eye Institute, Houston Methodist Institute for Academic Medicine, Houston Methodist Research Institute, Houston, Texas, USA; Department of Cardiothoracic Surgeries, Weill Cornell Medical College, Cornell University, New York City, New York, USA.
Yu P; Cardiovascular Biology Research Program, Oklahoma Medical Research Foundation, Oklahoma City, Oklahoma, USA; Department of Cell Biology, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA. Electronic address: .
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Źródło :
The Journal of biological chemistry [J Biol Chem] 2021 Oct; Vol. 297 (4), pp. 101149. Date of Electronic Publication: 2021 Aug 30.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Proliferation*
Glycolysis*
Signal Transduction*
Endothelial Cells/*metabolism
Receptor, Fibroblast Growth Factor, Type 1/*metabolism
Carnitine O-Palmitoyltransferase/genetics ; Carnitine O-Palmitoyltransferase/metabolism ; Gene Knockdown Techniques ; Humans ; PPAR alpha/genetics ; PPAR alpha/metabolism ; Receptor, Fibroblast Growth Factor, Type 1/genetics
Czasopismo naukowe
Tytuł :
MUC1 Promotes Mesangial Cell Proliferation and Kidney Fibrosis in Diabetic Nephropathy Through Activating STAT and β-Catenin Signal Pathway.
Autorzy :
Tao Y; Department of Nephrology, Qinghai Provincial People's Hospital, Xining City, China.
Han J; Department of Nephrology, Qinghai Provincial People's Hospital, Xining City, China.
Liu W; Department of Nephrology, Qinghai Provincial People's Hospital, Xining City, China.
An L; Department of Nephrology, Qinghai Provincial People's Hospital, Xining City, China.
Hu W; Department of Nephrology, Qinghai Provincial People's Hospital, Xining City, China.
Wang N; Department of Nephrology, Qinghai Provincial People's Hospital, Xining City, China.
Yu Y; Department of Nephrology, Wuhan Third Hospital, Tongren Hospital of Wuhan University, Wuhan City, China.
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Źródło :
DNA and cell biology [DNA Cell Biol] 2021 Oct; Vol. 40 (10), pp. 1308-1316. Date of Electronic Publication: 2021 Sep 14.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Proliferation*
Signal Transduction*
Diabetic Nephropathies/*metabolism
Mesangial Cells/*metabolism
Mucin-1/*metabolism
Animals ; Cells, Cultured ; Fibronectins/metabolism ; Fibrosis ; Kidney/metabolism ; Kidney/pathology ; Male ; Mesangial Cells/physiology ; Mice ; Mice, Inbred C57BL ; Mucin-1/genetics ; STAT Transcription Factors/antagonists & inhibitors ; STAT Transcription Factors/metabolism ; Sulfonamides/pharmacology ; Tyrphostins/pharmacology ; beta Catenin/antagonists & inhibitors ; beta Catenin/metabolism
Czasopismo naukowe
Tytuł :
Loss of haspin suppresses cancer cell proliferation by interfering with cell cycle progression at multiple stages.
Autorzy :
Wang P; School of Pharmaceutical Science and Technology, Tianjin University, Tianjin, P. R. China.; Division of Pharmaceutical Sciences, School of Pharmacy, The University of Wisconsin, Madison, Wisconsin, USA.
Hua X; Division of Pharmaceutical Sciences, School of Pharmacy, The University of Wisconsin, Madison, Wisconsin, USA.
Sun Y; Division of Pharmaceutical Sciences, School of Pharmacy, The University of Wisconsin, Madison, Wisconsin, USA.
Li H; Division of Pharmaceutical Sciences, School of Pharmacy, The University of Wisconsin, Madison, Wisconsin, USA.
Bryner YH; Division of Pharmaceutical Sciences, School of Pharmacy, The University of Wisconsin, Madison, Wisconsin, USA.
Hsung RP; Division of Pharmaceutical Sciences, School of Pharmacy, The University of Wisconsin, Madison, Wisconsin, USA.
Dai J; Division of Pharmaceutical Sciences, School of Pharmacy, The University of Wisconsin, Madison, Wisconsin, USA.
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Źródło :
FASEB journal : official publication of the Federation of American Societies for Experimental Biology [FASEB J] 2021 Oct; Vol. 35 (10), pp. e21923.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Cycle*/drug effects
Cell Cycle*/genetics
Cell Proliferation*/drug effects
Cell Proliferation*/genetics
Intracellular Signaling Peptides and Proteins/*deficiency
Neoplasms/*pathology
Protein-Serine-Threonine Kinases/*deficiency
Cell Line, Tumor ; Fluorescent Dyes ; G1 Phase Cell Cycle Checkpoints/drug effects ; G2 Phase/drug effects ; Humans ; Interphase/drug effects ; Intracellular Signaling Peptides and Proteins/antagonists & inhibitors ; Intracellular Signaling Peptides and Proteins/genetics ; Mitosis/drug effects ; Neoplasms/genetics ; Protein-Serine-Threonine Kinases/antagonists & inhibitors ; Protein-Serine-Threonine Kinases/genetics ; S Phase/drug effects ; Tubercidin/analogs & derivatives ; Tubercidin/pharmacology ; Tumor Suppressor Protein p53/genetics ; Ubiquitination ; Up-Regulation/drug effects
Czasopismo naukowe
Tytuł :
The transcribed ultraconserved element uc.51 promotes the proliferation and metastasis of breast cancer by stabilizing NONO.
Autorzy :
Shi X; Department of Breast Disease, The First Affiliated Hospital of Nanjing Medical University, No. 300 Guangzhou Road, Nanjing, 210000, China.
Huang X; Department of Breast Disease, The First Affiliated Hospital of Nanjing Medical University, No. 300 Guangzhou Road, Nanjing, 210000, China.
Chen R; Department of Breast Disease, The First Affiliated Hospital of Nanjing Medical University, No. 300 Guangzhou Road, Nanjing, 210000, China.
Li Y; Department of Breast Disease, The First Affiliated Hospital of Nanjing Medical University, No. 300 Guangzhou Road, Nanjing, 210000, China.
Xu Y; Department of Breast Disease, The First Affiliated Hospital of Nanjing Medical University, No. 300 Guangzhou Road, Nanjing, 210000, China.
Zhang W; Department of Breast Disease, The First Affiliated Hospital of Nanjing Medical University, No. 300 Guangzhou Road, Nanjing, 210000, China.
Zhu Q; Department of Breast Disease, The First Affiliated Hospital of Nanjing Medical University, No. 300 Guangzhou Road, Nanjing, 210000, China.
Zha X; Department of Breast Disease, The First Affiliated Hospital of Nanjing Medical University, No. 300 Guangzhou Road, Nanjing, 210000, China. .; Collaborative Innovation Center for Cancer Personalized Medicine, Nanjing Medical University, Nanjing, 210000, China. .
Wang J; Department of Breast Disease, The First Affiliated Hospital of Nanjing Medical University, No. 300 Guangzhou Road, Nanjing, 210000, China. .; Collaborative Innovation Center for Cancer Personalized Medicine, Nanjing Medical University, Nanjing, 210000, China. .
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Źródło :
Clinical & experimental metastasis [Clin Exp Metastasis] 2021 Dec; Vol. 38 (6), pp. 551-571. Date of Electronic Publication: 2021 Oct 29.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*
Cell Proliferation*
Breast Neoplasms/*metabolism
DNA-Binding Proteins/*metabolism
Lung Neoplasms/*metabolism
RNA, Long Noncoding/*metabolism
RNA-Binding Proteins/*metabolism
Adult ; Animals ; Breast Neoplasms/genetics ; Breast Neoplasms/pathology ; Cyclic AMP Response Element-Binding Protein ; DNA-Binding Proteins/genetics ; Female ; Gene Expression Regulation, Neoplastic ; Humans ; Ki-67 Antigen/genetics ; Ki-67 Antigen/metabolism ; Lung Neoplasms/genetics ; Lung Neoplasms/secondary ; MCF-7 Cells ; Mice, Inbred BALB C ; Mice, Nude ; Neoplasm Invasiveness ; Phosphorylation ; RNA, Long Noncoding/genetics ; RNA-Binding Proteins/genetics ; Signal Transduction ; Tumor Burden
Czasopismo naukowe
Tytuł :
Dichotomous metabolic networks govern human ILC2 proliferation and function.
Autorzy :
Surace L; Innate Immunity Unit, Institut Pasteur, Inserm U1223, Paris, France.
Doisne JM; Innate Immunity Unit, Institut Pasteur, Inserm U1223, Paris, France.
Croft CA; Innate Immunity Unit, Institut Pasteur, Inserm U1223, Paris, France.; Université de Paris, Sorbonne Paris Cité, Paris, France.
Thaller A; Innate Immunity Unit, Institut Pasteur, Inserm U1223, Paris, France.; Université de Paris, Sorbonne Paris Cité, Paris, France.
Escoll P; Biology of Intracellular Bacteria Unit, Institut Pasteur, CNRS UMR 3525, Paris, France.
Marie S; Innate Immunity Unit, Institut Pasteur, Inserm U1223, Paris, France.
Petrosemoli N; Bioinformatics and Biostatistics Hub, Center of Bioinformatics, Biostatistics, and Integrative Biology, Institut Pasteur, Paris, France.
Guillemot V; Bioinformatics and Biostatistics Hub, Center of Bioinformatics, Biostatistics, and Integrative Biology, Institut Pasteur, Paris, France.
Dardalhon V; Institut de Génétique Moléculaire de Montpellier, University of Montpellier, CNRS, Montpellier, France.
Topazio D; Department of Otolaryngology, Hospital 'Mazzini', Teramo, Italy.
Cama A; Department of Maxillofacial and Otolaryngology, Hospital 'F. Renzetti', Lanciano, Italy.
Buchrieser C; Biology of Intracellular Bacteria Unit, Institut Pasteur, CNRS UMR 3525, Paris, France.
Taylor N; Institut de Génétique Moléculaire de Montpellier, University of Montpellier, CNRS, Montpellier, France.
Amit I; Department of Immunology, Weizmann Institute of Science, Rehovot, Israel.
Musumeci O; Unit of Neurology and Neuromuscular Disorders, Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy.
Di Santo JP; Innate Immunity Unit, Institut Pasteur, Inserm U1223, Paris, France. .
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Źródło :
Nature immunology [Nat Immunol] 2021 Nov; Vol. 22 (11), pp. 1367-1374. Date of Electronic Publication: 2021 Oct 22.
Typ publikacji :
Letter; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Proliferation*/drug effects
Energy Metabolism*/drug effects
Immunity, Innate*/drug effects
Lymphocyte Activation*/drug effects
Cytokines/*metabolism
Mitochondrial Diseases/*metabolism
Th2 Cells/*metabolism
Amino Acids, Branched-Chain/metabolism ; Arginine/metabolism ; Case-Control Studies ; Cells, Cultured ; Humans ; Interleukin-33/pharmacology ; Mitochondria/metabolism ; Mitochondrial Diseases/diagnosis ; Mitochondrial Diseases/immunology ; Phenotype ; Th2 Cells/drug effects ; Th2 Cells/immunology
Raport
Tytuł :
MicroRNA-211 attenuates cell proliferation in T-cell lymphoblastic lymphoma through targeting TCF12.
Autorzy :
An L; Department of Hematology, Yantai Yuhuangding Hospital, Yantai City, Shandong Province, 264001, China.
Li X; Department of Pathology, Yantaishan Hospital, Yantai City, Shandong Province, 264003, China.
Yang J; Department of Hematology, Shandong Zaozhuang Municipal Hospital, Zaozhuang City, Shandong Province, 277100, China. Electronic address: .
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Źródło :
Leukemia research [Leuk Res] 2021 Nov; Vol. 110, pp. 106653. Date of Electronic Publication: 2021 Jun 29.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Proliferation*
Gene Expression Regulation, Neoplastic*
Basic Helix-Loop-Helix Transcription Factors/*metabolism
Biomarkers, Tumor/*metabolism
MicroRNAs/*genetics
Precursor T-Cell Lymphoblastic Leukemia-Lymphoma/*pathology
Adult ; Animals ; Apoptosis ; Basic Helix-Loop-Helix Transcription Factors/genetics ; Biomarkers, Tumor/genetics ; Female ; Humans ; Male ; Mice ; Precursor T-Cell Lymphoblastic Leukemia-Lymphoma/genetics ; Precursor T-Cell Lymphoblastic Leukemia-Lymphoma/metabolism ; Prognosis ; Survival Rate ; Tumor Cells, Cultured ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
Proliferation of endothelial cells (HUVEC) on specific-modified collagen sponges loaded with different growth factors.
Autorzy :
Groger A; Department of Plastic Surgery and Hand Surgery, Burn Center, Medical Faculty, RWTH Aachen University, Aachen, Germany.
Megas IF; Department of Plastic Surgery and Hand Surgery, Gemeinschaftskrankenhaus Havelhöhe, Berlin, Germany.
Noah EM; Department of Plastic Surgery and Hand Surgery, Burn Center, Medical Faculty, RWTH Aachen University, Aachen, Germany.; Department of Plastic, Reconstructive, Aesthetic and Hand Surgery, Rotes Kreuz Krankenhaus Kassel, Kassel, Germany.
Pallua N; Department of Plastic Surgery and Hand Surgery, Burn Center, Medical Faculty, RWTH Aachen University, Aachen, Germany.
Grieb G; Department of Plastic Surgery and Hand Surgery, Burn Center, Medical Faculty, RWTH Aachen University, Aachen, Germany.; Department of Plastic Surgery and Hand Surgery, Gemeinschaftskrankenhaus Havelhöhe, Berlin, Germany.
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Źródło :
The International journal of artificial organs [Int J Artif Organs] 2021 Nov; Vol. 44 (11), pp. 880-886. Date of Electronic Publication: 2021 Sep 08.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Proliferation*
Collagen*
Human Umbilical Vein Endothelial Cells/*cytology
Biocompatible Materials ; Fibroblast Growth Factor 2 ; Humans ; Neovascularization, Physiologic ; Vascular Endothelial Growth Factor A
Czasopismo naukowe
Tytuł :
CD146 immunohistochemical staining for the separation of benign from malignant mesothelial proliferations.
Autorzy :
Salisbury T; Department of Pathology, Vancouver General Hospital, JPPN 1401, 910 West 10th Ave, Vancouver, BC, V5Z 1M9, Canada.; Department of Pathology, University of British Columbia, Vancouver, BC, Canada.
Churg A; Department of Pathology, Vancouver General Hospital, JPPN 1401, 910 West 10th Ave, Vancouver, BC, V5Z 1M9, Canada. .; Department of Pathology, University of British Columbia, Vancouver, BC, Canada. .
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Źródło :
Virchows Archiv : an international journal of pathology [Virchows Arch] 2021 Nov; Vol. 479 (5), pp. 1047-1050. Date of Electronic Publication: 2021 Mar 15.
Typ publikacji :
Comparative Study; Journal Article
MeSH Terms :
Cell Proliferation*
Immunohistochemistry*
Biomarkers, Tumor/*analysis
Mesothelioma, Malignant/*immunology
CD146 Antigen/analysis ; Diagnosis, Differential ; Humans ; Mesothelioma, Malignant/pathology ; Predictive Value of Tests ; Tissue Array Analysis
Czasopismo naukowe
Tytuł :
Asymmetric crypt fission in sessile serrated lesions.
Autorzy :
Rubio CA; Department of Pathology, Karolinska Institute, Stockholm, Sweden .
Schmidt PT; Medicine (Solna), Karolinska Institute and Ersta Hospital, Stockholm, Sweden.
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Źródło :
Journal of clinical pathology [J Clin Pathol] 2021 Nov; Vol. 74 (11), pp. 712-717. Date of Electronic Publication: 2020 Oct 12.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Proliferation*
Colon/*pathology
Intestinal Mucosa/*pathology
Cohort Studies ; Colonic Neoplasms/etiology ; Humans ; Ki-67 Antigen ; Serpins/analysis
Czasopismo naukowe
Tytuł :
Overexpression of ARHGAP30 suppresses growth of cervical cancer cells by downregulating ribosome biogenesis.
Autorzy :
Wu A; Department of Obstetrics and Gynecology, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
Lin L; Department of Obstetrics and Gynecology, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
Li X; Department of Obstetrics and Gynecology, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
Xu Q; Department of Obstetrics and Gynecology, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
Xu W; Department of Obstetrics and Gynecology, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
Zhu X; Department of Obstetrics and Gynecology, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
Teng Y; Department of Obstetrics and Gynecology, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
Yang XM; State Key Laboratory of Oncogenes and Related Genes, Shanghai Cancer Institute, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China.
Ai Z; Department of Obstetrics and Gynecology, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
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Źródło :
Cancer science [Cancer Sci] 2021 Nov; Vol. 112 (11), pp. 4515-4525. Date of Electronic Publication: 2021 Sep 21.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Proliferation*
GTPase-Activating Proteins/*metabolism
Ribosomes/*metabolism
Uterine Cervical Neoplasms/*metabolism
Animals ; Cell Line, Tumor ; Cell Nucleolus/metabolism ; Down-Regulation ; Female ; HeLa Cells ; Humans ; Immunoprecipitation ; Mass Spectrometry ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Neoplasm Proteins/metabolism ; Phosphoproteins/metabolism ; Protein Biosynthesis ; RNA, Ribosomal/biosynthesis ; RNA-Binding Proteins/metabolism ; Tumor Stem Cell Assay ; Ubiquitination ; Uterine Cervical Neoplasms/mortality ; Uterine Cervical Neoplasms/pathology
Czasopismo naukowe
Tytuł :
Collagen polarization promotes epithelial elongation by stimulating locoregional cell proliferation.
Autorzy :
Katsuno-Kambe H; Division of Cell and Developmental Biology, Institute for Molecular Bioscience, The University of Queensland, Brisbane, Australia.
Teo JL; Division of Cell and Developmental Biology, Institute for Molecular Bioscience, The University of Queensland, Brisbane, Australia.
Ju RJ; Division of Cell and Developmental Biology, Institute for Molecular Bioscience, The University of Queensland, Brisbane, Australia.
Hudson J; QIMR Berghofer Medical Research Institute, Brisbane, Australia.
Stehbens SJ; Division of Cell and Developmental Biology, Institute for Molecular Bioscience, The University of Queensland, Brisbane, Australia.
Yap AS; Division of Cell and Developmental Biology, Institute for Molecular Bioscience, The University of Queensland, Brisbane, Australia.
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Źródło :
ELife [Elife] 2021 Oct 18; Vol. 10. Date of Electronic Publication: 2021 Oct 18.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Proliferation*
Collagen/*physiology
Epithelial Cells/*cytology
Humans
Czasopismo naukowe
Tytuł :
MicroRNA-7 overexpression positively regulates the CD8 SP cell development via targeting PIK3R1.
Autorzy :
Hu L; Special Key Laboratory of Gene Detection & Therapy of Guizhou Provincial Education Department, Guizhou, 563000, China; Department of Immunology & Talent Base of Biological Therapy of Guizhou Province, Zunyi Medical University, Guizhou, 563000, China.
Zhou Y; Special Key Laboratory of Gene Detection & Therapy of Guizhou Provincial Education Department, Guizhou, 563000, China; Department of Medical Physics, Zunyi Medical University, Zunyi, Guizhou, 563003, China.
Yang J; Special Key Laboratory of Gene Detection & Therapy of Guizhou Provincial Education Department, Guizhou, 563000, China; Department of Immunology & Talent Base of Biological Therapy of Guizhou Province, Zunyi Medical University, Guizhou, 563000, China.
Zhao X; Special Key Laboratory of Gene Detection & Therapy of Guizhou Provincial Education Department, Guizhou, 563000, China; Department of Immunology & Talent Base of Biological Therapy of Guizhou Province, Zunyi Medical University, Guizhou, 563000, China.
Mao L; Special Key Laboratory of Gene Detection & Therapy of Guizhou Provincial Education Department, Guizhou, 563000, China; Department of Immunology & Talent Base of Biological Therapy of Guizhou Province, Zunyi Medical University, Guizhou, 563000, China.
Zheng W; Department of Laboratory Medicine, Qiannan Medical University for Nationalities, Guizhou 558000, China.
Zhao J; Special Key Laboratory of Gene Detection & Therapy of Guizhou Provincial Education Department, Guizhou, 563000, China; Department of Immunology & Talent Base of Biological Therapy of Guizhou Province, Zunyi Medical University, Guizhou, 563000, China.
Guo M; Special Key Laboratory of Gene Detection & Therapy of Guizhou Provincial Education Department, Guizhou, 563000, China; Department of Immunology & Talent Base of Biological Therapy of Guizhou Province, Zunyi Medical University, Guizhou, 563000, China.
Chen C; Special Key Laboratory of Gene Detection & Therapy of Guizhou Provincial Education Department, Guizhou, 563000, China; Department of Immunology & Talent Base of Biological Therapy of Guizhou Province, Zunyi Medical University, Guizhou, 563000, China.
He Z; Department of Paediatrics, Affiliated Hospital of Zunyi Medical University, Guizhou, 563000, China; Key Laboratory of Adult Stem Cell Transformation Research, Chinese Academy of Medical Sciences, Guizhou, 563000, China.
Xu L; Special Key Laboratory of Gene Detection & Therapy of Guizhou Provincial Education Department, Guizhou, 563000, China; Department of Immunology & Talent Base of Biological Therapy of Guizhou Province, Zunyi Medical University, Guizhou, 563000, China. Electronic address: .
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Źródło :
Experimental cell research [Exp Cell Res] 2021 Oct 15; Vol. 407 (2), pp. 112824. Date of Electronic Publication: 2021 Sep 10.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Differentiation*
Cell Proliferation*
Gene Expression Regulation*
CD8-Positive T-Lymphocytes/*cytology
Class Ia Phosphatidylinositol 3-Kinase/*chemistry
MicroRNAs/*metabolism
Animals ; CD8-Positive T-Lymphocytes/immunology ; CD8-Positive T-Lymphocytes/metabolism ; Class Ia Phosphatidylinositol 3-Kinase/genetics ; Class Ia Phosphatidylinositol 3-Kinase/metabolism ; Mice ; Mice, Inbred C57BL ; MicroRNAs/genetics ; Phosphatidylinositol 3-Kinases/genetics ; Phosphatidylinositol 3-Kinases/metabolism ; Proto-Oncogene Proteins c-akt/genetics ; Proto-Oncogene Proteins c-akt/metabolism
Czasopismo naukowe
Tytuł :
Expression of cell adhesion molecule 1 in human and murine endometrial glandular cells and its increase during the proliferative phase by estrogen and cell density.
Autorzy :
Kimura R; Department of Pathology, Kindai University Faculty of Medicine, 377-2 Ohno-higashi, Osaka-sayama, Osaka 589-8511, Japan.
Otani T; Department of Pathology, Kindai University Faculty of Medicine, 377-2 Ohno-higashi, Osaka-sayama, Osaka 589-8511, Japan.
Shiraishi N; Genome Medical Center, Kindai University Hospital, Osaka, Japan.
Hagiyama M; Department of Pathology, Kindai University Faculty of Medicine, 377-2 Ohno-higashi, Osaka-sayama, Osaka 589-8511, Japan.
Yoneshige A; Department of Pathology, Kindai University Faculty of Medicine, 377-2 Ohno-higashi, Osaka-sayama, Osaka 589-8511, Japan.
Wada A; Department of Pathology, Kindai University Faculty of Medicine, 377-2 Ohno-higashi, Osaka-sayama, Osaka 589-8511, Japan.
Kajiyama H; Department of Pathology, Kindai University Faculty of Medicine, 377-2 Ohno-higashi, Osaka-sayama, Osaka 589-8511, Japan.
Takeuchi F; Department of Pathology, Kindai University Faculty of Medicine, 377-2 Ohno-higashi, Osaka-sayama, Osaka 589-8511, Japan.
Mizuguchi N; Kindai University Life Science Research Institute, Osaka, Japan.
Morishita K; Division of Tumor and Cellular Biochemistry, Department of Medical Sciences, Faculty of Medicine, University of Miyazaki, Miyazaki, Japan.
Ito A; Department of Pathology, Kindai University Faculty of Medicine, 377-2 Ohno-higashi, Osaka-sayama, Osaka 589-8511, Japan. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2021 Oct 15; Vol. 283, pp. 119854. Date of Electronic Publication: 2021 Jul 29.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Proliferation*
Gene Expression Regulation*
Cell Adhesion Molecule-1/*biosynthesis
Endometrium/*metabolism
Epithelial Cells/*metabolism
Adult ; Animals ; Cell Adhesion Molecule-1/genetics ; Cell Line, Tumor ; Estrogens/pharmacology ; Female ; Humans ; Mice
Czasopismo naukowe

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