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Tytuł:
Cross talk of tumor protein D52 (TPD52) with KLF9, PKCε, and MicroRNA 223 in ovarian cancer.
Autorzy:
Khan K; Atta-ur-Rahman School of Applied Biosciences (ASAB), National University of Sciences and Technology (NUST), Islamabad, 44000, Pakistan.
Zafar S; Atta-ur-Rahman School of Applied Biosciences (ASAB), National University of Sciences and Technology (NUST), Islamabad, 44000, Pakistan.
Badshah Y; Atta-ur-Rahman School of Applied Biosciences (ASAB), National University of Sciences and Technology (NUST), Islamabad, 44000, Pakistan.
Ashraf NM; School of Biochemistry & Biotechnology, University of the Punjab, Lahore, Pakistan.
Rafiq M; School of Interdisciplinary Engineering & Sciences (SINES), National University of Sciences and Technology, Islamabad, 44000, Pakistan.
Danish L; Agricultural Research Institute, Tarnab, Peshawar, Pakistan.
Shabbir M; Atta-ur-Rahman School of Applied Biosciences (ASAB), National University of Sciences and Technology (NUST), Islamabad, 44000, Pakistan. .; Department of Healthcare Biotechnology, Atta-ur-Rahman School of Applied Biosciences, National University of Sciences and Technology, Islamabad, Pakistan. .
Trembley JH; Research Service, Minneapolis VA Health Care System, Minneapolis, MN, USA.; Department of Laboratory Medicine and Pathology, University of Minnesota, Minneapolis, MN, USA.; Masonic Cancer Center, University of Minnesota, Minneapolis, MN, USA.
Afsar T; Department of Community Health Sciences, College of Applied Medical Sciences, King Saud University, Riyadh, Saudi Arabia.
Almajwal A; Department of Community Health Sciences, College of Applied Medical Sciences, King Saud University, Riyadh, Saudi Arabia.
Razak S; Department of Community Health Sciences, College of Applied Medical Sciences, King Saud University, Riyadh, Saudi Arabia. .
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Źródło:
Journal of ovarian research [J Ovarian Res] 2023 Oct 13; Vol. 16 (1), pp. 202. Date of Electronic Publication: 2023 Oct 13.
Typ publikacji:
Journal Article
MeSH Terms:
Kruppel-Like Transcription Factors*/genetics
MicroRNAs*/genetics
Neoplasm Proteins*/genetics
Ovarian Neoplasms*/genetics
Ovarian Neoplasms*/pathology
Protein Kinase C-epsilon*/genetics
Female ; Humans ; Phylogeny
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interplay between Protein Kinase C Epsilon and Reactive Oxygen Species during Myogenic Differentiation.
Autorzy:
Pozzi G; Department of Medicine and Surgery (DiMeC), University of Parma, Via Gramsci, 14, 43126 Parma, Italy.
Presta V; Department of Medicine and Surgery (DiMeC), University of Parma, Via Gramsci, 14, 43126 Parma, Italy.
Masselli E; Department of Medicine and Surgery (DiMeC), University of Parma, Via Gramsci, 14, 43126 Parma, Italy.
Condello G; Department of Medicine and Surgery (DiMeC), University of Parma, Via Gramsci, 14, 43126 Parma, Italy.
Cortellazzi S; Department of Medicine and Surgery (DiMeC), University of Parma, Via Gramsci, 14, 43126 Parma, Italy.
Arcari ML; Department of Medicine and Surgery (DiMeC), University of Parma, Via Gramsci, 14, 43126 Parma, Italy.
Micheloni C; Department of Medicine and Surgery (DiMeC), University of Parma, Via Gramsci, 14, 43126 Parma, Italy.
Vitale M; Department of Medicine and Surgery (DiMeC), University of Parma, Via Gramsci, 14, 43126 Parma, Italy.; Italian Foundation for Research in Balneotherapy (FoRST), 00198 Rome, Italy.
Gobbi G; Department of Medicine and Surgery (DiMeC), University of Parma, Via Gramsci, 14, 43126 Parma, Italy.
Mirandola P; Department of Medicine and Surgery (DiMeC), University of Parma, Via Gramsci, 14, 43126 Parma, Italy.
Carubbi C; Department of Medicine and Surgery (DiMeC), University of Parma, Via Gramsci, 14, 43126 Parma, Italy.
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Źródło:
Cells [Cells] 2023 Jul 05; Vol. 12 (13). Date of Electronic Publication: 2023 Jul 05.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Protein Kinase C-epsilon*
Antioxidants*
Animals ; Mice ; Reactive Oxygen Species/metabolism ; Cell Differentiation/physiology ; Cell Line
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Elucidating the role of missense SNP of protein kinase C epsilon in HCV-induced hepatocellular carcinoma.
Autorzy:
Rehman A; Atta-ur-Rahman School of Applied Biosciences (ASAB), National University of Sciences and Technology (NUST), Islamabad, Pakistan.
Shabbir M; Atta-ur-Rahman School of Applied Biosciences (ASAB), National University of Sciences and Technology (NUST), Islamabad, Pakistan. .
Badshah Y; Atta-ur-Rahman School of Applied Biosciences (ASAB), National University of Sciences and Technology (NUST), Islamabad, Pakistan.
Khan K; Atta-ur-Rahman School of Applied Biosciences (ASAB), National University of Sciences and Technology (NUST), Islamabad, Pakistan.
Trembley JH; Minneapolis VA Health Care System Research Service, Minneapolis, MN, USA.; Department of Laboratory Medicine and Pathology, University of Minnesota, Minneapolis, MN, USA.; Masonic Cancer Center, University of Minnesota, Minneapolis, MN, USA.
Ashraf NM; School of Biochemistry & Biotechnology, University of the Punjab, Lahore, Pakistan.
Afsar T; Department of Community Health Sciences, College of Applied Medical Sciences, King Saud University, Riyadh, Saudi Arabia.
Almajwal A; Department of Community Health Sciences, College of Applied Medical Sciences, King Saud University, Riyadh, Saudi Arabia.
Alruwaili NW; Department of Community Health Sciences, College of Applied Medical Sciences, King Saud University, Riyadh, Saudi Arabia.
Alshamari A; Department of Community Health Sciences, College of Applied Medical Sciences, King Saud University, Riyadh, Saudi Arabia.
Alanezi TN; College of Medicine, King Saud University, 11481, Riyadh, Saudi Arabia.
Razak S; Department of Community Health Sciences, College of Applied Medical Sciences, King Saud University, Riyadh, Saudi Arabia. .
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Źródło:
BMC cancer [BMC Cancer] 2023 Feb 13; Vol. 23 (1), pp. 147. Date of Electronic Publication: 2023 Feb 13.
Typ publikacji:
Journal Article
MeSH Terms:
Carcinoma, Hepatocellular*/pathology
Carcinoma, Hepatocellular*/virology
Hepatitis C*/complications
Hepatitis C*/genetics
Liver Neoplasms*/pathology
Liver Neoplasms*/virology
Protein Kinase C-epsilon*/genetics
Humans ; Case-Control Studies ; Genetic Predisposition to Disease ; Genotype ; Hepacivirus ; Polymorphism, Single Nucleotide ; Mutation, Missense
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Exploring the prognostic significance of PKCε variants in cervical cancer.
Autorzy:
Zafar S; Department of Healthcare Biotechnology, Atta-Ur-Rahman School of Applied Biosciences, National University of Sciences and Technology, Islamabad, Pakistan.
Khan K; Department of Healthcare Biotechnology, Atta-Ur-Rahman School of Applied Biosciences, National University of Sciences and Technology, Islamabad, Pakistan.
Badshah Y; Department of Healthcare Biotechnology, Atta-Ur-Rahman School of Applied Biosciences, National University of Sciences and Technology, Islamabad, Pakistan. yasmeenb_.
Shahid K; Department of Healthcare Biotechnology, Atta-Ur-Rahman School of Applied Biosciences, National University of Sciences and Technology, Islamabad, Pakistan.
Trembley JH; Department of Laboratory Medicine and Pathology, University of Minnesota, Minneapolis, MN, USA.; Masonic Cancer Center, University of Minnesota, Minneapolis, MN, USA.; Minneapolis VA Health Care System Research Service, Minneapolis, MN, USA.
Hafeez A; Department of Healthcare Biotechnology, Atta-Ur-Rahman School of Applied Biosciences, National University of Sciences and Technology, Islamabad, Pakistan.
Ashraf NM; School of Biochemistry and Biotechnology, University of the Punjab, Lahore, Pakistan.
Arslan H; University of Bonn, LIMES Institute (AG-Netea), Carl-Troll-Str. 31, 53115, Bonn, Germany.
Shabbir M; Department of Healthcare Biotechnology, Atta-Ur-Rahman School of Applied Biosciences, National University of Sciences and Technology, Islamabad, Pakistan.
Afsar T; Department of Community Health Sciences, College of Applied Medical Sciences, King Saud University, Riyadh, Saudi Arabia.
Almajwal A; Department of Community Health Sciences, College of Applied Medical Sciences, King Saud University, Riyadh, Saudi Arabia.
Razak S; Department of Community Health Sciences, College of Applied Medical Sciences, King Saud University, Riyadh, Saudi Arabia. .
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Źródło:
BMC cancer [BMC Cancer] 2023 Sep 04; Vol. 23 (1), pp. 819. Date of Electronic Publication: 2023 Sep 04.
Typ publikacji:
Journal Article
MeSH Terms:
Uterine Cervical Neoplasms*/genetics
Humans ; Female ; Protein Kinase C-epsilon/genetics ; Prognosis ; Computational Biology ; Molecular Dynamics Simulation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The ε-Isozyme of Protein Kinase C (PKCε) Is Impaired in ALS Motor Cortex and Its Pulse Activation by Bryostatin-1 Produces Long Term Survival in Degenerating SOD1-G93A Motor Neuron-like Cells.
Autorzy:
La Cognata V; Institute for Biomedical Research and Innovation, National Research Council, 95126 Catania, Italy.
D'Amico AG; Section of Human Anatomy and Histology, Department of Biomedical and Biotechnological Sciences, University of Catania, 95123 Catania, Italy.
Maugeri G; Section of Human Anatomy and Histology, Department of Biomedical and Biotechnological Sciences, University of Catania, 95123 Catania, Italy.
Morello G; Institute for Biomedical Research and Innovation, National Research Council, 95126 Catania, Italy.
Guarnaccia M; Institute for Biomedical Research and Innovation, National Research Council, 95126 Catania, Italy.
Magrì B; Section of Human Anatomy and Histology, Department of Biomedical and Biotechnological Sciences, University of Catania, 95123 Catania, Italy.
Aronica E; Department of (Neuro) Pathology, Amsterdam UMC, University of Amsterdam, Amsterdam Neuroscience, Meibergdreef 9, 1105 Amsterdam, The Netherlands.
Alkon DL; Synaptogenix, Inc., New York, NY 10036, USA.
D'Agata V; Section of Human Anatomy and Histology, Department of Biomedical and Biotechnological Sciences, University of Catania, 95123 Catania, Italy.
Cavallaro S; Institute for Biomedical Research and Innovation, National Research Council, 95126 Catania, Italy.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Aug 15; Vol. 24 (16). Date of Electronic Publication: 2023 Aug 15.
Typ publikacji:
Journal Article
MeSH Terms:
Amyotrophic Lateral Sclerosis*/genetics
Motor Cortex*
Neurodegenerative Diseases*
Humans ; Protein Kinase C-epsilon/genetics ; Isoenzymes/genetics ; Superoxide Dismutase-1/genetics ; Bryostatins/pharmacology ; Motor Neurons
SCR Disease Name:
Amyotrophic lateral sclerosis 1
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Bidirectional sex-dependent regulation of α6 and β3 nicotinic acetylcholine receptors by protein kinase Cε.
Autorzy:
Moen JK; Graduate Program in Neuroscience, University of Minnesota, Minneapolis, Minnesota, USA.
DeBaker MC; Graduate Program in Neuroscience, University of Minnesota, Minneapolis, Minnesota, USA.
Myjak JE; Department of Pharmacology, University of Minnesota, Minneapolis, Minnesota, USA.
Wickman K; Graduate Program in Neuroscience, University of Minnesota, Minneapolis, Minnesota, USA.; Department of Pharmacology, University of Minnesota, Minneapolis, Minnesota, USA.
Lee AM; Graduate Program in Neuroscience, University of Minnesota, Minneapolis, Minnesota, USA.; Department of Pharmacology, University of Minnesota, Minneapolis, Minnesota, USA.
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Źródło:
Addiction biology [Addict Biol] 2021 May; Vol. 26 (3), pp. e12954. Date of Electronic Publication: 2020 Aug 10.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Ethanol/*pharmacology
Protein Kinase C-epsilon/*metabolism
Receptors, Nicotinic/*metabolism
Varenicline/*pharmacology
Animals ; Female ; Male ; Mesencephalon/drug effects ; Mesencephalon/metabolism ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Nicotinic Agonists/pharmacology ; Protein Kinase C-epsilon/genetics ; Protein Subunits/metabolism ; Receptors, Nicotinic/genetics ; Reflex, Righting/drug effects ; Reward ; Sex Factors ; Ventral Tegmental Area/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Berberine ameliorates neuronal AD-like change via activating Pi3k/PGCε pathway.
Autorzy:
Wu N; Hubei Key Laboratory of Diabetes and Angiopathy, Hubei University of Science and Technology, Xianning, China.; Basic Medical College, Hubei University of Science and Technology, Xianning, China.
Liu W; Hubei Key Laboratory of Diabetes and Angiopathy, Hubei University of Science and Technology, Xianning, China.
Wang J; Hubei Key Laboratory of Diabetes and Angiopathy, Hubei University of Science and Technology, Xianning, China.
Han Y; Hubei Key Laboratory of Diabetes and Angiopathy, Hubei University of Science and Technology, Xianning, China.
Ye Y; Hubei Key Laboratory of Diabetes and Angiopathy, Hubei University of Science and Technology, Xianning, China.
Liu X; Hubei Key Laboratory of Diabetes and Angiopathy, Hubei University of Science and Technology, Xianning, China.
Yu Y; Department of Oncology, Renmin Hospital, Hubei University of Medicine, Shiyan, China.
Chen Q; Hubei Key Laboratory of Diabetes and Angiopathy, Hubei University of Science and Technology, Xianning, China.
Bao Y; Basic Medical College, Hubei University of Science and Technology, Xianning, China.
Liu C; Hubei Key Laboratory of Diabetes and Angiopathy, Hubei University of Science and Technology, Xianning, China.
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Źródło:
BioFactors (Oxford, England) [Biofactors] 2021 Jul; Vol. 47 (4), pp. 587-599. Date of Electronic Publication: 2021 Mar 19.
Typ publikacji:
Journal Article
MeSH Terms:
Berberine/*pharmacology
Cognitive Dysfunction/*prevention & control
Diabetes Mellitus, Experimental/*drug therapy
Hypoglycemic Agents/*pharmacology
Phosphatidylinositol 3-Kinases/*genetics
Protein Kinase C-epsilon/*genetics
Amyloid beta-Peptides/genetics ; Amyloid beta-Peptides/metabolism ; Animals ; Cell Line, Tumor ; Cognitive Dysfunction/genetics ; Cognitive Dysfunction/metabolism ; Cognitive Dysfunction/physiopathology ; Diabetes Mellitus, Experimental/chemically induced ; Diabetes Mellitus, Experimental/genetics ; Diabetes Mellitus, Experimental/pathology ; Gene Expression Regulation ; Glucose/antagonists & inhibitors ; Glucose/pharmacology ; Glucose Transporter Type 3/genetics ; Glucose Transporter Type 3/metabolism ; Glycogen Synthase Kinase 3 beta/genetics ; Glycogen Synthase Kinase 3 beta/metabolism ; Humans ; Insulin/pharmacology ; Insulin Resistance ; Male ; Maze Learning/drug effects ; Metformin/pharmacology ; Neurons/cytology ; Neurons/drug effects ; Neurons/metabolism ; Peptide Fragments/genetics ; Peptide Fragments/metabolism ; Phosphatidylinositol 3-Kinases/metabolism ; Protein Kinase C-epsilon/metabolism ; Rats ; Rats, Wistar ; Signal Transduction ; Streptozocin/administration & dosage
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Silencing P2X7R Alleviates Diabetic Neuropathic Pain Involving TRPV1 via PKCε/P38MAPK/NF-κB Signaling Pathway in Rats.
Autorzy:
Chen L; Department of Physiology, Basic Medical College of Nanchang University, Nanchang 330006, China.
Wang H; Department of Physiology, Basic Medical College of Nanchang University, Nanchang 330006, China.
Xing J; Department of Physiology, Basic Medical College of Nanchang University, Nanchang 330006, China.
Shi X; Department of Physiology, Basic Medical College of Nanchang University, Nanchang 330006, China.
Huang H; Department of Physiology, Basic Medical College of Nanchang University, Nanchang 330006, China.
Huang J; Department of Physiology, Basic Medical College of Nanchang University, Nanchang 330006, China.
Xu C; Department of Physiology, Basic Medical College of Nanchang University, Nanchang 330006, China.; Jiangxi Provincial Key Laboratory of Autonomic Nervous Function and Disease, Nanchang 330006, China.; The Clinical Medical School, Jiangxi Medical College, Shangrao 334000, China.; The First Affiliated Hospital, Jiangxi Medical College, Shangrao 334000, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Nov 16; Vol. 23 (22). Date of Electronic Publication: 2022 Nov 16.
Typ publikacji:
Journal Article
MeSH Terms:
Diabetes Mellitus, Experimental*/complications
Diabetic Neuropathies*/genetics
Neuralgia*/genetics
Neuralgia*/metabolism
Receptors, Purinergic P2X7*/genetics
Receptors, Purinergic P2X7*/metabolism
Animals ; Male ; Rats ; Hyperalgesia/metabolism ; NF-kappa B/metabolism ; p38 Mitogen-Activated Protein Kinases/genetics ; p38 Mitogen-Activated Protein Kinases/metabolism ; Protein Kinase C-epsilon/genetics ; Protein Kinase C-epsilon/metabolism ; Rats, Sprague-Dawley ; RNA, Small Interfering/genetics ; Signal Transduction/genetics ; TRPV Cation Channels/genetics ; TRPV Cation Channels/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Regulation of Autophagy by Protein Kinase C-ε in Breast Cancer Cells.
Autorzy:
Basu A; Department of Microbiology, Immunology and Genetics, University of North Texas Health Science Center, Fort Worth, TX 76107, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Jun 15; Vol. 21 (12). Date of Electronic Publication: 2020 Jun 15.
Typ publikacji:
Journal Article
MeSH Terms:
Autophagy*/drug effects
Breast Neoplasms/*genetics
Protein Kinase C-epsilon/*genetics
RNA, Small Interfering/*pharmacology
Breast Neoplasms/metabolism ; Cell Line, Tumor ; Female ; Gene Expression Regulation, Neoplastic/drug effects ; Gene Silencing ; Humans ; MCF-7 Cells ; Macrolides/pharmacology ; Mechanistic Target of Rapamycin Complex 1/metabolism ; Microtubule-Associated Proteins/metabolism ; Protein Kinase C-epsilon/metabolism ; Sequestosome-1 Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
CR4056, a powerful analgesic imidazoline-2 receptor ligand, inhibits the inflammation-induced PKCε phosphorylation and membrane translocation in sensory neurons.
Autorzy:
Vellani V; Dipartimento di Scienze Biomediche, Metaboliche e Neuroscienze, Università di Modena e Reggio Emilia, Modena, Italy.
Sabatini C; Rottapharm Biotech, Monza, Italy.; PhD Program in Neuroscience, Dipartimento di Medicina e chirurgia, Università degli Studi di Milano-Bicocca, Monza, Italy.
Milia C; PhD Program in Neuroscience, Dipartimento di Medicina e chirurgia, Università degli Studi di Milano-Bicocca, Monza, Italy.
Caselli G; Rottapharm Biotech, Monza, Italy.
Lanza M; Rottapharm Biotech, Monza, Italy.
Letari O; Rottapharm Biotech, Monza, Italy.
Rovati LC; Rottapharm Biotech, Monza, Italy.
Giacomoni C; Dipartimento di Economia, Scienze e Diritto, Università degli Studi della Repubblica di San Marino, San Marino.
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Źródło:
British journal of pharmacology [Br J Pharmacol] 2020 Jan; Vol. 177 (1), pp. 48-64. Date of Electronic Publication: 2019 Nov 07.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Analgesics/*metabolism
Cell Membrane/*metabolism
Imidazoles/*metabolism
Imidazoline Receptors/*metabolism
Protein Kinase C-epsilon/*antagonists & inhibitors
Quinazolines/*metabolism
Sensory Receptor Cells/*metabolism
Amino Acid Sequence ; Analgesics/pharmacology ; Analgesics/therapeutic use ; Animals ; Cell Membrane/drug effects ; Cells, Cultured ; Dose-Response Relationship, Drug ; Freund's Adjuvant/toxicity ; Humans ; Imidazoles/pharmacology ; Imidazoles/therapeutic use ; Inflammation/chemically induced ; Inflammation/drug therapy ; Inflammation/metabolism ; Male ; Pain/chemically induced ; Pain/drug therapy ; Pain/metabolism ; Phosphorylation/drug effects ; Phosphorylation/physiology ; Protein Kinase C-epsilon/metabolism ; Quinazolines/pharmacology ; Quinazolines/therapeutic use ; Rats ; Rats, Wistar ; Sensory Receptor Cells/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Cardioprotective Signaling Pathways in Obese Mice Submitted to Regular Exercise: Effect on Oxysterols.
Autorzy:
Barau C; Inserm U955-IMRB, UPEC, Ecole Nationale Vétérinaire d'Alfort, F-94010 Créteil, France.; Plateforme de Ressources Biologiques, APHP, Hôpitaux Universitaires Henri Mondor, F-94000 Créteil, France.
Leick S; Inserm U955-IMRB, UPEC, Ecole Nationale Vétérinaire d'Alfort, F-94010 Créteil, France.
Caccia C; Fondazione IRCCS-Istituto Neurologico Carlo Besta, 20131 Milano, Italy.
Portal L; Inserm U955-IMRB, UPEC, Ecole Nationale Vétérinaire d'Alfort, F-94010 Créteil, France.
Leoni V; Laboratory of Clinical Biochemistry, Hospital Pius XI of Desio, ASST-Brianza, University of Milano-Bicocca, 20126 Monza, Italy.; Department of Medicine and Surgery, University of Milano-Bicocca, 20126 Monza, Italy.
Le Corvoisier P; Inserm U955-IMRB, UPEC, Ecole Nationale Vétérinaire d'Alfort, F-94010 Créteil, France.; INSERM CIC 1430, APHP, Hôpitaux Universitaires Henri Mondor, F-94000 Créteil, France.
Morin D; Inserm U955-IMRB, UPEC, Ecole Nationale Vétérinaire d'Alfort, F-94010 Créteil, France.
Ghaleh B; Inserm U955-IMRB, UPEC, Ecole Nationale Vétérinaire d'Alfort, F-94010 Créteil, France.; Plateforme de Ressources Biologiques, APHP, Hôpitaux Universitaires Henri Mondor, F-94000 Créteil, France.
Pons S; Inserm U955-IMRB, UPEC, Ecole Nationale Vétérinaire d'Alfort, F-94010 Créteil, France.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Sep 16; Vol. 23 (18). Date of Electronic Publication: 2022 Sep 16.
Typ publikacji:
Journal Article
MeSH Terms:
Myocardial Reperfusion Injury*/metabolism
Oxysterols*/metabolism
Animals ; Humans ; Mice ; Calcium/metabolism ; Mice, Obese ; Mitochondria, Heart/metabolism ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; Obesity/metabolism ; Protein Kinase C-epsilon/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Signal Transduction/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Impact of Zinc on Oxidative Signaling Pathways in the Development of Pulmonary Vasoconstriction Induced by Hypobaric Hypoxia.
Autorzy:
Arriaza K; Institute of Health Studies, Arturo Prat University, Iquique 1100000, Chile.
Cuevas C; Institute of Health Studies, Arturo Prat University, Iquique 1100000, Chile.
Pena E; Institute of Health Studies, Arturo Prat University, Iquique 1100000, Chile.
Siques P; Institute of Health Studies, Arturo Prat University, Iquique 1100000, Chile.
Brito J; Institute of Health Studies, Arturo Prat University, Iquique 1100000, Chile.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Jun 23; Vol. 23 (13). Date of Electronic Publication: 2022 Jun 23.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Papillomavirus Infections*/metabolism
Vasoconstriction*/physiology
Altitude Sickness ; Humans ; Hypertension, Pulmonary ; Hypoxia/metabolism ; NADPH Oxidases/metabolism ; Oxidative Stress ; Protein Kinase C-epsilon/metabolism ; Pulmonary Artery/metabolism ; Reactive Oxygen Species/metabolism ; Signal Transduction ; Zinc/metabolism
SCR Disease Name:
Pulmonary edema of mountaineers
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Maternal Prkce expression in mature oocytes is critical for the first cleavage facilitating maternal-to-zygotic transition in mouse early embryos.
Autorzy:
Zhang S; School of Life Sciences and Biotechnology & Shanghai Children's Hospital, Shanghai Jiao Tong University, Shanghai, China.
Gong X; School of Life Sciences and Biotechnology & Shanghai Children's Hospital, Shanghai Jiao Tong University, Shanghai, China.
Zhou Y; School of Life Sciences and Biotechnology & Shanghai Children's Hospital, Shanghai Jiao Tong University, Shanghai, China.; Department of Histo-Embryology, Genetics and Developmental Biology, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; NHC Key Laboratory of Medical Embryogenesis and Developmental Molecular Biology & Shanghai Key Laboratory of Embryo and Reproduction Engineering, Shanghai, China.
Ma Q; School of Life Sciences and Biotechnology & Shanghai Children's Hospital, Shanghai Jiao Tong University, Shanghai, China.; NHC Key Laboratory of Medical Embryogenesis and Developmental Molecular Biology & Shanghai Key Laboratory of Embryo and Reproduction Engineering, Shanghai, China.
Cai Q; School of Life Sciences and Biotechnology & Shanghai Children's Hospital, Shanghai Jiao Tong University, Shanghai, China.; NHC Key Laboratory of Medical Embryogenesis and Developmental Molecular Biology & Shanghai Key Laboratory of Embryo and Reproduction Engineering, Shanghai, China.
Yang G; School of Life Sciences and Biotechnology & Shanghai Children's Hospital, Shanghai Jiao Tong University, Shanghai, China.; NHC Key Laboratory of Medical Embryogenesis and Developmental Molecular Biology & Shanghai Key Laboratory of Embryo and Reproduction Engineering, Shanghai, China.
Guo X; School of Life Sciences and Biotechnology & Shanghai Children's Hospital, Shanghai Jiao Tong University, Shanghai, China.; NHC Key Laboratory of Medical Embryogenesis and Developmental Molecular Biology & Shanghai Key Laboratory of Embryo and Reproduction Engineering, Shanghai, China.
Chen Y; School of Life Sciences and Biotechnology & Shanghai Children's Hospital, Shanghai Jiao Tong University, Shanghai, China.; NHC Key Laboratory of Medical Embryogenesis and Developmental Molecular Biology & Shanghai Key Laboratory of Embryo and Reproduction Engineering, Shanghai, China.
Xu M; School of Life Sciences and Biotechnology & Shanghai Children's Hospital, Shanghai Jiao Tong University, Shanghai, China.; NHC Key Laboratory of Medical Embryogenesis and Developmental Molecular Biology & Shanghai Key Laboratory of Embryo and Reproduction Engineering, Shanghai, China.
Zhu Y; School of Life Sciences and Biotechnology & Shanghai Children's Hospital, Shanghai Jiao Tong University, Shanghai, China.; NHC Key Laboratory of Medical Embryogenesis and Developmental Molecular Biology & Shanghai Key Laboratory of Embryo and Reproduction Engineering, Shanghai, China.
Zeng Y; School of Life Sciences and Biotechnology & Shanghai Children's Hospital, Shanghai Jiao Tong University, Shanghai, China.; NHC Key Laboratory of Medical Embryogenesis and Developmental Molecular Biology & Shanghai Key Laboratory of Embryo and Reproduction Engineering, Shanghai, China.
Zeng F; School of Life Sciences and Biotechnology & Shanghai Children's Hospital, Shanghai Jiao Tong University, Shanghai, China.; Department of Histo-Embryology, Genetics and Developmental Biology, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; NHC Key Laboratory of Medical Embryogenesis and Developmental Molecular Biology & Shanghai Key Laboratory of Embryo and Reproduction Engineering, Shanghai, China.; School of Pharmacy, Macau University of Science and Technonlogy, Taipa, Macau, China.
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Źródło:
Cell proliferation [Cell Prolif] 2022 Jun; Vol. 55 (6), pp. e13231. Date of Electronic Publication: 2022 May 18.
Typ publikacji:
Journal Article
MeSH Terms:
Oocytes*/metabolism
Zygote*/metabolism
Embryo, Mammalian/*metabolism
Protein Kinase C-epsilon/*metabolism
Animals ; Embryo, Mammalian/cytology ; Female ; Gene Expression Regulation, Developmental ; Mice ; Mice, Inbred DBA ; Mice, Inbred MRL lpr ; Mice, Knockout ; Pregnancy ; RNA, Complementary/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Therapeutic hypercapnia reduces blood-brain barrier damage possibly via protein kinase Cε in rats with lateral fluid percussion injury.
Autorzy:
Yang WC; Department of Anesthesiology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
Wang Q; Department of Anesthesiology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
Chi LT; Department of Anesthesiology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
Wang YZ; Department of Anesthesiology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
Cao HL; Department of Anesthesiology, Second Affiliated Hospital of Harbin Medical University, Harbin, China.
Li WZ; Department of Anesthesiology, Second Affiliated Hospital of Harbin Medical University, Harbin, China. .; Anesthesiology Key Laboratory, Education Department, Harbin Medical University, No. 246 Xuefu Road, Harbin, 150086, China. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2019 Feb 13; Vol. 16 (1), pp. 36. Date of Electronic Publication: 2019 Feb 13.
Typ publikacji:
Journal Article
MeSH Terms:
Hypercapnia*
Blood-Brain Barrier/*physiopathology
Brain Injuries, Traumatic/*pathology
Brain Injuries, Traumatic/*therapy
Carbon Dioxide/*therapeutic use
Protein Kinase C-epsilon/*metabolism
Animals ; Blood Pressure/drug effects ; Brain Edema/etiology ; Brain Edema/therapy ; Brain Injuries, Traumatic/diagnostic imaging ; Carbazoles/therapeutic use ; Disease Models, Animal ; Heart Rate/drug effects ; Male ; Neurologic Examination ; Oxygen/metabolism ; Protein Kinase C-epsilon/genetics ; Protein Kinase Inhibitors/therapeutic use ; Rats ; Rats, Sprague-Dawley ; Staurosporine/therapeutic use ; Time Factors ; Tomography Scanners, X-Ray Computed
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
miR-205 enhances radiation sensitivity of prostate cancer cells by impairing DNA damage repair through PKCε and ZEB1 inhibition.
Autorzy:
El Bezawy R; Molecular Pharmacology Unit, Department of Applied Research and Technological Development, Fondazione IRCCS Istituto Nazionale dei Tumori di Milano, Via Amadeo 42, 20133, Milan, Italy.
Tinelli S; Molecular Pharmacology Unit, Department of Applied Research and Technological Development, Fondazione IRCCS Istituto Nazionale dei Tumori di Milano, Via Amadeo 42, 20133, Milan, Italy.
Tortoreto M; Molecular Pharmacology Unit, Department of Applied Research and Technological Development, Fondazione IRCCS Istituto Nazionale dei Tumori di Milano, Via Amadeo 42, 20133, Milan, Italy.
Doldi V; Molecular Pharmacology Unit, Department of Applied Research and Technological Development, Fondazione IRCCS Istituto Nazionale dei Tumori di Milano, Via Amadeo 42, 20133, Milan, Italy.
Zuco V; Molecular Pharmacology Unit, Department of Applied Research and Technological Development, Fondazione IRCCS Istituto Nazionale dei Tumori di Milano, Via Amadeo 42, 20133, Milan, Italy.
Folini M; Molecular Pharmacology Unit, Department of Applied Research and Technological Development, Fondazione IRCCS Istituto Nazionale dei Tumori di Milano, Via Amadeo 42, 20133, Milan, Italy.
Stucchi C; Medical Physics Unit, Fondazione IRCCS Istituto Nazionale dei Tumori di Milano, 20133, Milan, Italy.
Rancati T; Prostate Cancer Program, Fondazione IRCCS Istituto Nazionale dei Tumori di Milano, 20133, Milan, Italy.
Valdagni R; Prostate Cancer Program, Fondazione IRCCS Istituto Nazionale dei Tumori di Milano, 20133, Milan, Italy.; Radiation Oncology 1 Unit, Fondazione IRCCS Istituto Nazionale dei Tumori di Milano, 20133, Milan, Italy.
Gandellini P; Molecular Pharmacology Unit, Department of Applied Research and Technological Development, Fondazione IRCCS Istituto Nazionale dei Tumori di Milano, Via Amadeo 42, 20133, Milan, Italy.
Zaffaroni N; Molecular Pharmacology Unit, Department of Applied Research and Technological Development, Fondazione IRCCS Istituto Nazionale dei Tumori di Milano, Via Amadeo 42, 20133, Milan, Italy. .
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Źródło:
Journal of experimental & clinical cancer research : CR [J Exp Clin Cancer Res] 2019 Feb 04; Vol. 38 (1), pp. 51. Date of Electronic Publication: 2019 Feb 04.
Typ publikacji:
Journal Article
MeSH Terms:
MicroRNAs/*genetics
Prostatic Neoplasms/*radiotherapy
Protein Kinase C-epsilon/*antagonists & inhibitors
Radiation Tolerance/*genetics
Zinc Finger E-box-Binding Homeobox 1/*antagonists & inhibitors
Animals ; Cell Line, Tumor ; DNA Repair/genetics ; Humans ; Male ; Mice ; Mice, SCID ; Molecular Mimicry ; PC-3 Cells ; Protein Kinase C-epsilon/genetics ; Transfection ; Xenograft Model Antitumor Assays ; Zinc Finger E-box-Binding Homeobox 1/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Diverse dynamics features of novel protein kinase C (PKC) isozymes determine the selectivity of a fluorinated balanol analogue for PKCε.
Autorzy:
Hardianto A; Department of Molecular Sciences, Macquarie University, Sydney, NSW, 2109, Australia.; Department of Chemistry, Universitas Padjadjaran, Jatinangor, West Java, 45363, Indonesia.
Khanna V; Department of Molecular Sciences, Macquarie University, Sydney, NSW, 2109, Australia.; School of Medicine, Faculty of Medicine, Nursing and Health Sciences, Flinders University, Adelaide, SA, 5042, Australia.
Liu F; Department of Molecular Sciences, Macquarie University, Sydney, NSW, 2109, Australia.
Ranganathan S; Department of Molecular Sciences, Macquarie University, Sydney, NSW, 2109, Australia. .
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Źródło:
BMC bioinformatics [BMC Bioinformatics] 2019 Feb 04; Vol. 19 (Suppl 13), pp. 342. Date of Electronic Publication: 2019 Feb 04.
Typ publikacji:
Journal Article
MeSH Terms:
Halogenation*
Azepines/*metabolism
Hydroxybenzoates/*metabolism
Protein Kinase C-epsilon/*metabolism
Adenosine Triphosphate/metabolism ; Azepines/chemistry ; Cluster Analysis ; Humans ; Hydroxybenzoates/chemistry ; Isoenzymes/metabolism ; Molecular Dynamics Simulation ; Phosphorylation ; Protein Conformation ; Protein Kinase C-epsilon/chemistry ; Ribose/chemistry ; Sequence Alignment ; Static Electricity ; Substrate Specificity
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
PKCε phosphorylation regulates the mitochondrial translocation of ATF2 in ischemia-induced neurodegeneration.
Autorzy:
Kumar V; Department of Biological Sciences, School of Biomedical Sciences, Kent State University, Kent, OH, 44242, USA.
Weng YC; Center for Neuropsychiatric Research, National Health Research Institutes, Miaoli, 35053, Taiwan, ROC.
Wu YC; Center for Neuropsychiatric Research, National Health Research Institutes, Miaoli, 35053, Taiwan, ROC.
Huang YT; Center for Neuropsychiatric Research, National Health Research Institutes, Miaoli, 35053, Taiwan, ROC.
Chou WH; Department of Biological Sciences, School of Biomedical Sciences, Kent State University, Kent, OH, 44242, USA. .; Center for Neuropsychiatric Research, National Health Research Institutes, Miaoli, 35053, Taiwan, ROC. .
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Źródło:
BMC neuroscience [BMC Neurosci] 2018 Nov 29; Vol. 19 (1), pp. 76. Date of Electronic Publication: 2018 Nov 29.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Brain Ischemia/*metabolism
Mitochondria/*metabolism
Neurodegenerative Diseases/*metabolism
Protein Kinase C-epsilon/*metabolism
Animals ; Biological Transport/physiology ; Brain Ischemia/pathology ; Cytoplasm/metabolism ; Cytoplasm/pathology ; Mice, Knockout ; Nerve Degeneration/metabolism ; Nerve Degeneration/pathology ; Neurodegenerative Diseases/pathology ; Neurons/metabolism ; Neurons/pathology ; Phosphorylation ; Protein Kinase C-epsilon/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Phosphorylation at Ser 727 Increases STAT3 Interaction with PKC ε Regulating Neuron-Glia Crosstalk via IL-6-Mediated Hyperalgesia In Vivo and In Vitro.
Autorzy:
Li X; Department of Anesthesiology, Shenzhen Second People's Hospital, The First Affiliated Hospital of Shenzhen University, Health Science Center, Shenzhen 518035, China.
Zhou B; Department of Geriatric & Spinal Pain Multi-Department Treatment, Shenzhen Second People's Hospital, The First Affiliated Hospital of Shenzhen University, Health Science Center, Shenzhen 518035, China.
Yang H; Department of Anesthesiology, Shenzhen Second People's Hospital, The First Affiliated Hospital of Shenzhen University, Health Science Center, Shenzhen 518035, China.
Yang X; Department of Anesthesiology, Shenzhen Second People's Hospital, The First Affiliated Hospital of Shenzhen University, Health Science Center, Shenzhen 518035, China.
Zhao Z; Department of Anesthesiology, Shenzhen Second People's Hospital, The First Affiliated Hospital of Shenzhen University, Health Science Center, Shenzhen 518035, China.
Pan Z; Department of Anesthesiology, Shenzhen Second People's Hospital, The First Affiliated Hospital of Shenzhen University, Health Science Center, Shenzhen 518035, China.
Liao X; Department of Anesthesiology, Shenzhen Second People's Hospital, The First Affiliated Hospital of Shenzhen University, Health Science Center, Shenzhen 518035, China.
Jian W; Department of Anesthesiology, Shenzhen Second People's Hospital, The First Affiliated Hospital of Shenzhen University, Health Science Center, Shenzhen 518035, China.
Liu Y; Department of Anesthesiology, Shenzhen Second People's Hospital, The First Affiliated Hospital of Shenzhen University, Health Science Center, Shenzhen 518035, China.
Lu H; Department of Anesthesiology, Ruijin Hospital Affiliated to Shanghai Jiaotong University, Shanghai 200025, China.
Xue Q; Department of Anesthesiology, Ruijin Hospital Affiliated to Shanghai Jiaotong University, Shanghai 200025, China.
Luo Y; Department of Anesthesiology, Ruijin Hospital Affiliated to Shanghai Jiaotong University, Shanghai 200025, China.
Yu B; Department of Anesthesiology, Ruijin Hospital Affiliated to Shanghai Jiaotong University, Shanghai 200025, China.
Huang H; Department of Anesthesiology, Second Affiliated Hospital of Guangzhou Medical University, No. 250 Changgangxi Road, Guangzhou 510260, China.
Ma D; Division of Anaesthetics, Pain Medicine and Intensive Care, Department of Surgery and Cancer, Faculty of Medicine, Imperial College London, Chelsea & Westminster Hospital, London, UK.
Liu Z; Department of Anesthesiology, Shenzhen Second People's Hospital, The First Affiliated Hospital of Shenzhen University, Health Science Center, Shenzhen 518035, China.
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Źródło:
Mediators of inflammation [Mediators Inflamm] 2022 Jan 28; Vol. 2022, pp. 2782080. Date of Electronic Publication: 2022 Jan 28 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Hyperalgesia*/metabolism
Interleukin-6*/metabolism
Animals ; Neuroglia/metabolism ; Neurons/metabolism ; Phosphorylation ; Protein Kinase C-epsilon/metabolism ; Protein Kinase C-epsilon/pharmacology ; Rats ; Spinal Cord/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Aldehyde Trapping by ADX-102 Is Protective against Cigarette Smoke and Alcohol Mediated Lung Cell Injury.
Autorzy:
Ochoa CA; Department of Internal Medicine, College of Medicine, University of Nebraska Medical Center, Omaha, NE 68198-5910, USA.
Nissen CG; Department of Environmental, Agricultural and Occupational Health, College of Public Health, University of Nebraska Medical Center, Omaha, NE 68198-5910, USA.
Mosley DD; Department of Internal Medicine, College of Medicine, University of Nebraska Medical Center, Omaha, NE 68198-5910, USA.
Bauer CD; Department of Internal Medicine, College of Medicine, University of Nebraska Medical Center, Omaha, NE 68198-5910, USA.
Jordan DL; Department of Internal Medicine, College of Medicine, University of Nebraska Medical Center, Omaha, NE 68198-5910, USA.
Bailey KL; Department of Internal Medicine, College of Medicine, University of Nebraska Medical Center, Omaha, NE 68198-5910, USA.; Veterans Affairs Nebraska-Western Iowa Health Care System, Omaha, NE 68105, USA.
Wyatt TA; Department of Internal Medicine, College of Medicine, University of Nebraska Medical Center, Omaha, NE 68198-5910, USA.; Department of Environmental, Agricultural and Occupational Health, College of Public Health, University of Nebraska Medical Center, Omaha, NE 68198-5910, USA.; Veterans Affairs Nebraska-Western Iowa Health Care System, Omaha, NE 68105, USA.
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Źródło:
Biomolecules [Biomolecules] 2022 Mar 02; Vol. 12 (3). Date of Electronic Publication: 2022 Mar 02.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Alcoholism*/metabolism
Cigarette Smoking*
Lung Injury*
Aldehydes/metabolism ; Aminoquinolines ; Animals ; Ethanol/toxicity ; Lung/metabolism ; Mice ; Protein Kinase C-epsilon/metabolism ; Nicotiana
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The G Protein-Coupled Serotonin 1A Receptor Augments Protein Kinase Cε-Mediated Neurogenesis in Neonatal Mouse Hippocampus-PKCε-Mediated Signaling in the Early Hippocampus.
Autorzy:
Samaddar S; Department of Physical Therapy, The College of Staten Island, City University of New York, Staten Island, NY 10314, USA.
Purkayastha S; Global Health and Wellbeing, Unilever, 18 Nepal Park, Singapore 139407, Singapore.
Diallo S; Weiss Memorial Hospital, 4646 Marine Drive, Chicago, IL 60640, USA.
Tantry SJ; Medicinal Chemistry at Jubilant Biosys, Bangalore 560022, India.
Schroder R; Eurofins Lancaster PSS, Merck Sharp and Dohme, Rahway, NJ 07065, USA.
Chanthrakumar P; Warren Alpert Medical School, Brown University, Providence, RI 02903, USA.
Flory MJ; Research Design and Analysis Service, New York State Institute for Developmental Disabilities, Staten Island, NY 10314, USA.
Banerjee P; Department of Chemistry, Center for Developmental Neuroscience, The College of Staten Island, City University of New York, Staten Island, NY 10314, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Feb 10; Vol. 23 (4). Date of Electronic Publication: 2022 Feb 10.
Typ publikacji:
Journal Article
MeSH Terms:
Hippocampus/*metabolism
Neurogenesis/*physiology
Protein Kinase C-epsilon/*metabolism
Receptor, Serotonin, 5-HT1A/*metabolism
Receptors, G-Protein-Coupled/*metabolism
Serotonin/*metabolism
Signal Transduction/*physiology
8-Hydroxy-2-(di-n-propylamino)tetralin/pharmacology ; Animals ; Animals, Newborn ; Apoptosis/drug effects ; Bromodeoxyuridine/pharmacology ; Dentate Gyrus/drug effects ; Dentate Gyrus/metabolism ; Dentate Gyrus/physiology ; Female ; Hippocampus/drug effects ; Hippocampus/physiology ; Male ; Mice ; Mice, Inbred C57BL ; Neurogenesis/drug effects ; Neurons/drug effects ; Neurons/metabolism ; Neurons/physiology ; Serotonin Receptor Agonists/pharmacology ; Signal Transduction/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
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