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Tytuł:
Clinicopathological Significance of AKT1 and PLK1 Expression in Oral Squamous Cell Carcinoma.
Autorzy:
Sun EC; Department of Stomatology, Shihezi University School of Medicine & the First Affiliated Hospital to Shihezi University School of Medicine, Shihezi, 832002 Xinjiang, China.
Dong SS; Department of Pathology, Northern Jiangsu People's Hospital Affiliated to Yangzhou University/Clinical Medical College, Yangzhou University, Yangzhou, Jiangsu 225000, China.
Li ZJ; Department of Stomatology, Shihezi University School of Medicine & the First Affiliated Hospital to Shihezi University School of Medicine, Shihezi, 832002 Xinjiang, China.
Li CX; Department of Stomatology, Shihezi University School of Medicine & the First Affiliated Hospital to Shihezi University School of Medicine, Shihezi, 832002 Xinjiang, China.
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Źródło:
Disease markers [Dis Markers] 2022 Jun 17; Vol. 2022, pp. 7300593. Date of Electronic Publication: 2022 Jun 17 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Cell Cycle Proteins*/biosynthesis
Mouth Neoplasms*/metabolism
Protein Serine-Threonine Kinases*/biosynthesis
Proto-Oncogene Proteins*/biosynthesis
Proto-Oncogene Proteins c-akt*
Squamous Cell Carcinoma of Head and Neck*/metabolism
Biomarkers, Tumor/metabolism ; Humans ; Prognosis ; Polo-Like Kinase 1
Czasopismo naukowe
Tytuł:
AXL targeting by a specific small molecule or monoclonal antibody inhibits renal cell carcinoma progression in an orthotopic mice model.
Autorzy:
Chen TJ; Department of Clinical Medicine, University of Bergen, Bergen, Norway.
Mydel P; Department of Clinical Science, University of Bergen, Bergen, Norway.; Department of Microbiology, Jagiellonian University, Krakow, Poland.
Benedyk-Machaczka M; Department of Microbiology, Jagiellonian University, Krakow, Poland.
Kamińska M; Department of Microbiology, Jagiellonian University, Krakow, Poland.
Kalucka U; Department of Microbiology, Jagiellonian University, Krakow, Poland.
Blø M; BerGenBio ASA, Bergen, Norway.
Furriol J; Department of Clinical Medicine, University of Bergen, Bergen, Norway.
Gausdal G; BerGenBio ASA, Bergen, Norway.
Lorens J; Department of Biomedicine, Centre for Cancer Biomarkers, Norwegian Centre of Excellence, University of Bergen, Bergen, Norway.
Osman T; Department of Clinical Medicine, University of Bergen, Bergen, Norway.
Marti HP; Department of Clinical Medicine, University of Bergen, Bergen, Norway.; Department of Medicine, Haukeland University Hospital, Bergen, Norway.
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Źródło:
Physiological reports [Physiol Rep] 2021 Dec; Vol. 9 (23), pp. e15140.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Carcinoma, Renal Cell/*metabolism
Kidney Neoplasms/*metabolism
Proto-Oncogene Proteins/*metabolism
Receptor Protein-Tyrosine Kinases/*metabolism
Animals ; Benzocycloheptenes/pharmacology ; Carcinoma, Renal Cell/genetics ; Carcinoma, Renal Cell/pathology ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Disease Models, Animal ; Disease Progression ; Humans ; Kidney Neoplasms/genetics ; Kidney Neoplasms/pathology ; Mice ; Phosphorylation/drug effects ; Protein Kinase Inhibitors/pharmacology ; Proto-Oncogene Proteins/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; Receptor Protein-Tyrosine Kinases/genetics ; Signal Transduction/drug effects ; Triazoles/pharmacology ; Axl Receptor Tyrosine Kinase
Czasopismo naukowe
Tytuł:
TCL1A, B Cell Regulation and Tolerance in Renal Transplantation.
Autorzy:
Brinas F; Université de Nantes, CHU Nantes, Inserm, Centre de Recherche en Transplantation et Immunologie, UMR 1064, ITUN, F-44000 Nantes, France.; Labex IGO, F-44000 Nantes, France.
Danger R; Université de Nantes, CHU Nantes, Inserm, Centre de Recherche en Transplantation et Immunologie, UMR 1064, ITUN, F-44000 Nantes, France.; Labex IGO, F-44000 Nantes, France.
Brouard S; Université de Nantes, CHU Nantes, Inserm, Centre de Recherche en Transplantation et Immunologie, UMR 1064, ITUN, F-44000 Nantes, France.; Labex IGO, F-44000 Nantes, France.; Centre d'Investigation Clinique en Biothérapie, Centre de Ressources Biologiques (CRB), Labex IGO, F-44000 Nantes, France.
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Źródło:
Cells [Cells] 2021 Jun 01; Vol. 10 (6). Date of Electronic Publication: 2021 Jun 01.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Kidney Transplantation*
Transplantation Tolerance*
B-Lymphocytes/*immunology
Cell Differentiation/*immunology
Proto-Oncogene Proteins/*immunology
Humans ; Interleukin-10/immunology ; Proto-Oncogene Proteins c-akt/immunology ; T-Lymphocytes/immunology
Czasopismo naukowe
Tytuł:
In oxygen-deprived tumor cells ERp57 provides radioprotection and ensures proliferation via c-Myc, PLK1 and the AKT pathway.
Autorzy:
Ocklenburg T; Institut Für Physiologie, Universität Duisburg-Essen, Duisburg, Germany.
Neumann F; Institut Für Physiologie, Universität Duisburg-Essen, Duisburg, Germany.
Wolf A; Institut Für Physiologie, Universität Duisburg-Essen, Duisburg, Germany.
Vogel J; Institut Für Physiologie, Universität Duisburg-Essen, Duisburg, Germany.
Göpelt K; Institut Für Physiologie, Universität Duisburg-Essen, Duisburg, Germany.
Baumann M; Institut Für Physiologie, Universität Duisburg-Essen, Duisburg, Germany.
Baumann J; Institut Für Physiologie, Universität Duisburg-Essen, Duisburg, Germany.
Kranz P; Institut Für Physiologie, Universität Duisburg-Essen, Duisburg, Germany.
Metzen E; Institut Für Physiologie, Universität Duisburg-Essen, Duisburg, Germany.
Brockmeier U; Department of Neurology, University Hospital Essen, Essen, Germany. .
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Źródło:
Scientific reports [Sci Rep] 2021 Mar 30; Vol. 11 (1), pp. 7199. Date of Electronic Publication: 2021 Mar 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Tumor Hypoxia*
Cell Cycle Proteins/*metabolism
Colonic Neoplasms/*metabolism
Protein Disulfide-Isomerases/*metabolism
Protein Serine-Threonine Kinases/*metabolism
Proto-Oncogene Proteins/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Proto-Oncogene Proteins c-myc/*metabolism
Apoptosis ; Cell Proliferation ; Colonic Neoplasms/genetics ; Colonic Neoplasms/radiotherapy ; Gene Deletion ; Gene Expression Regulation, Neoplastic ; Gene Knockdown Techniques ; HCT116 Cells ; Humans ; Oxygen/metabolism ; Protein Disulfide-Isomerases/genetics ; Signal Transduction ; Polo-Like Kinase 1
Czasopismo naukowe
Tytuł:
Celastrol mediates autophagy and apoptosis via the ROS/JNK and Akt/mTOR signaling pathways in glioma cells.
Autorzy:
Liu X; School of Traditional Chinese Medicine, Capital Medical University, Beijing, China.; Beijing Key Lab of TCM Collateral Disease Theory Research, Capital Medical University, Beijing, China.
Zhao P; Basic Discipline of Integrated Chinese and Western Medicine, Henan University of Chinese Medicine, Zhengzhou, Henan, China.
Wang X; School of Traditional Chinese Medicine, Capital Medical University, Beijing, China. .; Beijing Key Lab of TCM Collateral Disease Theory Research, Capital Medical University, Beijing, China. .
Wang L; School of Traditional Chinese Medicine, Capital Medical University, Beijing, China.; Beijing Key Lab of TCM Collateral Disease Theory Research, Capital Medical University, Beijing, China.
Zhu Y; School of Traditional Chinese Medicine, Capital Medical University, Beijing, China.; Beijing Key Lab of TCM Collateral Disease Theory Research, Capital Medical University, Beijing, China.
Song Y; School of Traditional Chinese Medicine, Capital Medical University, Beijing, China.; Beijing Key Lab of TCM Collateral Disease Theory Research, Capital Medical University, Beijing, China.
Gao W; School of Traditional Chinese Medicine, Capital Medical University, Beijing, China. .; Beijing Key Lab of TCM Collateral Disease Theory Research, Capital Medical University, Beijing, China. .; Advanced Innovation Center for Human Brain Protection, Capital Medical University, Beijing, China. .
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Źródło:
Journal of experimental & clinical cancer research : CR [J Exp Clin Cancer Res] 2019 May 03; Vol. 38 (1), pp. 184. Date of Electronic Publication: 2019 May 03.
Typ publikacji:
Journal Article
MeSH Terms:
Glioma/*drug therapy
Protein-Tyrosine Kinases/*genetics
Proto-Oncogene Proteins/*genetics
TOR Serine-Threonine Kinases/*genetics
Triterpenes/*pharmacology
Apoptosis/drug effects ; Autophagosomes/drug effects ; Autophagy/drug effects ; Cell Cycle Checkpoints/drug effects ; Cell Proliferation/drug effects ; Glioma/genetics ; Glioma/pathology ; Humans ; MAP Kinase Kinase 4/genetics ; Microtubule-Associated Proteins/genetics ; Pentacyclic Triterpenes ; Proto-Oncogene Proteins c-akt/genetics ; Proto-Oncogene Proteins c-myc/genetics ; Reactive Oxygen Species/metabolism ; Signal Transduction/drug effects
Czasopismo naukowe
Tytuł:
Reduced menin expression impairs rapamycin effects as evidenced by an increase in mTORC2 signaling and cell migration.
Autorzy:
Razmara M; Department of medical sciences, Science for Life Laboratory, Uppsala University, Uppsala, Sweden. .
Monazzam A; Department of medical sciences, Science for Life Laboratory, Uppsala University, Uppsala, Sweden.
Skogseid B; Department of medical sciences, Science for Life Laboratory, Uppsala University, Uppsala, Sweden.
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Źródło:
Cell communication and signaling : CCS [Cell Commun Signal] 2018 Oct 01; Vol. 16 (1), pp. 64. Date of Electronic Publication: 2018 Oct 01.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression Regulation/*drug effects
Mechanistic Target of Rapamycin Complex 2/*metabolism
Proto-Oncogene Proteins/*metabolism
Signal Transduction/*drug effects
Sirolimus/*pharmacology
Animals ; Cell Line ; Gene Knockdown Techniques ; Humans ; Mice ; Phosphorylation/drug effects ; Proto-Oncogene Proteins/deficiency ; Proto-Oncogene Proteins/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; RNA, Small Interfering/genetics
Czasopismo naukowe
Tytuł:
AKT regulation of mesothelial-to-mesenchymal transition in peritoneal dialysis is modulated by Smurf2 and deubiquitinating enzyme USP4.
Autorzy:
Xiao L; Department of Nephrology, Second Xiangya Hospital, Central South University, Changsha, Hunan, 410011, China. .
Peng X; Department of Nephrology, Second Xiangya Hospital, Central South University, Changsha, Hunan, 410011, China. .
Liu F; Department of Nephrology, Second Xiangya Hospital, Central South University, Changsha, Hunan, 410011, China. .
Tang C; Department of Nephrology, Second Xiangya Hospital, Central South University, Changsha, Hunan, 410011, China. .
Hu C; Department of Nephrology, Second Xiangya Hospital, Central South University, Changsha, Hunan, 410011, China. .
Xu X; Department of Nephrology, Second Xiangya Hospital, Central South University, Changsha, Hunan, 410011, China. .
Wang M; Department of Nephrology, Second Xiangya Hospital, Central South University, Changsha, Hunan, 410011, China. .
Luo Y; Department of Nephrology, Second Xiangya Hospital, Central South University, Changsha, Hunan, 410011, China. .
Yang S; Department of Nephrology, Second Xiangya Hospital, Central South University, Changsha, Hunan, 410011, China. ysk_.
Song P; Department of Nephrology, Second Xiangya Hospital, Central South University, Changsha, Hunan, 410011, China. .
Xiao P; Department of Nephrology, Second Xiangya Hospital, Central South University, Changsha, Hunan, 410011, China. .
Kanwar YS; Departments of Pathology & Medicine, Northwestern University, Chicago, USA. .
Sun L; Department of Nephrology, Second Xiangya Hospital, Central South University, Changsha, Hunan, 410011, China. .
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Źródło:
BMC cell biology [BMC Cell Biol] 2015 Mar 06; Vol. 16, pp. 7. Date of Electronic Publication: 2015 Mar 06.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Proto-Oncogene Proteins/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Ubiquitin Thiolesterase/*metabolism
Ubiquitin-Protein Ligases/*metabolism
Animals ; Cell Line ; Chromones/pharmacology ; Down-Regulation/drug effects ; Epithelial Cells/cytology ; Epithelial Cells/drug effects ; Epithelial Cells/metabolism ; Epithelial-Mesenchymal Transition/drug effects ; Female ; Fibronectins/genetics ; Fibronectins/metabolism ; Humans ; Mice ; Morpholines/pharmacology ; Peritoneal Dialysis ; Peritoneum/metabolism ; Phosphatidylinositol 3-Kinases/metabolism ; Phosphoinositide-3 Kinase Inhibitors ; Proto-Oncogene Proteins/antagonists & inhibitors ; Proto-Oncogene Proteins/genetics ; Proto-Oncogene Proteins c-akt/antagonists & inhibitors ; Proto-Oncogene Proteins c-akt/genetics ; Smad7 Protein/genetics ; Smad7 Protein/metabolism ; Transforming Growth Factor beta1/metabolism ; Transforming Growth Factor beta1/pharmacology ; Ubiquitin Thiolesterase/antagonists & inhibitors ; Ubiquitin Thiolesterase/genetics ; Ubiquitin-Protein Ligases/genetics ; Ubiquitin-Specific Proteases ; Up-Regulation/drug effects ; Vimentin/genetics ; Vimentin/metabolism ; Zonula Occludens-1 Protein/genetics ; Zonula Occludens-1 Protein/metabolism
Czasopismo naukowe
Tytuł:
Embelin suppresses growth of human pancreatic cancer xenografts, and pancreatic cancer cells isolated from KrasG12D mice by inhibiting Akt and Sonic hedgehog pathways.
Autorzy:
Huang M; Department of Pharmacology, Toxicology and Therapeutics, and Medicine, The University of Kansas Cancer Center, The University of Kansas Medical Center, Kansas City, Kansas, United States of America.
Tang SN; Department of Pharmacology, Toxicology and Therapeutics, and Medicine, The University of Kansas Cancer Center, The University of Kansas Medical Center, Kansas City, Kansas, United States of America.
Upadhyay G; Department of Pharmacology, Toxicology and Therapeutics, and Medicine, The University of Kansas Cancer Center, The University of Kansas Medical Center, Kansas City, Kansas, United States of America.
Marsh JL; Department of Biochemistry, University of Texas Health Science Center at Tyler, Tyler, Texas, United States of America.
Jackman CP; Department of Biochemistry, University of Texas Health Science Center at Tyler, Tyler, Texas, United States of America.
Shankar S; Department of Pathology and Laboratory Medicine, The University of Kansas Cancer Center, The University of Kansas Medical Center, Kansas City, Kansas, United States of America.
Srivastava RK; Department of Pharmacology, Toxicology and Therapeutics, and Medicine, The University of Kansas Cancer Center, The University of Kansas Medical Center, Kansas City, Kansas, United States of America.
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Źródło:
PloS one [PLoS One] 2014 Apr 02; Vol. 9 (4), pp. e92161. Date of Electronic Publication: 2014 Apr 02 (Print Publication: 2014).
Typ publikacji:
Journal Article
MeSH Terms:
Benzoquinones/*pharmacology
Hedgehog Proteins/*metabolism
Pancreatic Neoplasms/*drug therapy
Proto-Oncogene Proteins/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Proto-Oncogene Proteins p21(ras)/*metabolism
Signal Transduction/*drug effects
ras Proteins/*metabolism
Animals ; Cell Line, Tumor ; Hedgehog Proteins/genetics ; Heterografts ; Humans ; Mice ; Mice, Inbred BALB C ; Mice, Mutant Strains ; Mice, Nude ; Neoplasm Metastasis ; Neoplasm Transplantation ; Neovascularization, Pathologic/drug therapy ; Neovascularization, Pathologic/genetics ; Neovascularization, Pathologic/metabolism ; Neovascularization, Pathologic/pathology ; Pancreatic Neoplasms/genetics ; Pancreatic Neoplasms/metabolism ; Pancreatic Neoplasms/pathology ; Proto-Oncogene Proteins/genetics ; Proto-Oncogene Proteins c-akt/genetics ; Proto-Oncogene Proteins p21(ras)/genetics ; Signal Transduction/genetics ; Xenograft Model Antitumor Assays ; ras Proteins/genetics
Czasopismo naukowe
Tytuł:
AF1q Mediates Tumor Progression in Colorectal Cancer by Regulating AKT Signaling.
Autorzy:
Hu J; Cancer Research Institute, Tongji Hospital, Huazhong University of Science and Technology, 1095 Jiefang Ave., Wuhan 430030, China. .
Li G; Cancer Research Institute, Tongji Hospital, Huazhong University of Science and Technology, 1095 Jiefang Ave., Wuhan 430030, China. .
Liu L; Cancer Research Institute, Tongji Hospital, Huazhong University of Science and Technology, 1095 Jiefang Ave., Wuhan 430030, China. .
Wang Y; Cancer Research Institute, Tongji Hospital, Huazhong University of Science and Technology, 1095 Jiefang Ave., Wuhan 430030, China. .
Li X; Cancer Research Institute, Tongji Hospital, Huazhong University of Science and Technology, 1095 Jiefang Ave., Wuhan 430030, China. .
Gong J; Cancer Research Institute, Tongji Hospital, Huazhong University of Science and Technology, 1095 Jiefang Ave., Wuhan 430030, China. .
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2017 May 05; Vol. 18 (5). Date of Electronic Publication: 2017 May 05.
Typ publikacji:
Journal Article
MeSH Terms:
Signal Transduction*
Biomarkers, Tumor/*metabolism
Colorectal Neoplasms/*metabolism
Neoplasm Proteins/*metabolism
Proto-Oncogene Proteins/*metabolism
Animals ; Biomarkers, Tumor/genetics ; Cell Line, Tumor ; Cell Movement ; Cell Proliferation ; Colorectal Neoplasms/genetics ; Colorectal Neoplasms/pathology ; Epithelial-Mesenchymal Transition ; Humans ; Lymphatic Metastasis ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Neoplasm Proteins/genetics ; Proto-Oncogene Proteins/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; Up-Regulation
Czasopismo naukowe
Tytuł:
CHIP regulates AKT/FoxO/Bim signaling in MCF7 and MCF10A cells.
Autorzy:
Lv Y; Department of Breast Surgery, QiLu Hospital of Shandong University, Jinan, Shandong, China.
Song S; Department of Molecular Microbiology and Immunology, University of Southern California, Los Angeles, California, United States of America.
Zhang K; Department of Breast Surgery, QiLu Hospital of Shandong University, Jinan, Shandong, China.
Gao H; Department of Breast Surgery, QiLu Hospital of Shandong University, Jinan, Shandong, China.
Ma R; Department of Breast Surgery, QiLu Hospital of Shandong University, Jinan, Shandong, China.
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Źródło:
PloS one [PLoS One] 2013 Dec 20; Vol. 8 (12), pp. e83312. Date of Electronic Publication: 2013 Dec 20 (Print Publication: 2013).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression Regulation, Neoplastic*
Apoptosis Regulatory Proteins/*genetics
Breast Neoplasms/*genetics
Forkhead Transcription Factors/*genetics
Membrane Proteins/*genetics
Proto-Oncogene Proteins/*genetics
Proto-Oncogene Proteins c-akt/*genetics
Ubiquitin-Protein Ligases/*genetics
Apoptosis ; Apoptosis Regulatory Proteins/metabolism ; Bcl-2-Like Protein 11 ; Breast Neoplasms/metabolism ; Breast Neoplasms/pathology ; Cell Line, Tumor ; Cell Survival ; Female ; Forkhead Box Protein O1 ; Forkhead Transcription Factors/metabolism ; Humans ; Membrane Proteins/metabolism ; PTEN Phosphohydrolase/genetics ; PTEN Phosphohydrolase/metabolism ; Phosphatidylinositol 3-Kinases/genetics ; Phosphatidylinositol 3-Kinases/metabolism ; Protein Isoforms/genetics ; Protein Isoforms/metabolism ; Proto-Oncogene Proteins/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Signal Transduction ; Transcription, Genetic ; Ubiquitin-Protein Ligases/metabolism
Czasopismo naukowe
Tytuł:
Antitumor activity of miR-34a in peritoneal mesothelioma relies on c-MET and AXL inhibition: persistent activation of ERK and AKT signaling as a possible cytoprotective mechanism.
Autorzy:
El Bezawy R; Molecular Pharmacology Unit, Department of Experimental Oncology and Molecular Medicine, Fondazione IRCCS Istituto Nazionale dei Tumori, 20133, Milan, Italy.
De Cesare M; Molecular Pharmacology Unit, Department of Experimental Oncology and Molecular Medicine, Fondazione IRCCS Istituto Nazionale dei Tumori, 20133, Milan, Italy.
Pennati M; Molecular Pharmacology Unit, Department of Experimental Oncology and Molecular Medicine, Fondazione IRCCS Istituto Nazionale dei Tumori, 20133, Milan, Italy.
Deraco M; Colon-Rectal Cancer Surgery Unit, Department of Surgery, Fondazione IRCCS Istituto Nazionale dei Tumori, 20133, Milan, Italy.
Gandellini P; Molecular Pharmacology Unit, Department of Experimental Oncology and Molecular Medicine, Fondazione IRCCS Istituto Nazionale dei Tumori, 20133, Milan, Italy.
Zuco V; Molecular Pharmacology Unit, Department of Experimental Oncology and Molecular Medicine, Fondazione IRCCS Istituto Nazionale dei Tumori, 20133, Milan, Italy. .
Zaffaroni N; Molecular Pharmacology Unit, Department of Experimental Oncology and Molecular Medicine, Fondazione IRCCS Istituto Nazionale dei Tumori, 20133, Milan, Italy. .
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Źródło:
Journal of hematology & oncology [J Hematol Oncol] 2017 Jan 18; Vol. 10 (1), pp. 19. Date of Electronic Publication: 2017 Jan 18.
Typ publikacji:
Journal Article
MeSH Terms:
Lung Neoplasms/*pathology
Mesothelioma/*pathology
MicroRNAs/*physiology
Proto-Oncogene Proteins/*antagonists & inhibitors
Proto-Oncogene Proteins c-met/*antagonists & inhibitors
Receptor Protein-Tyrosine Kinases/*antagonists & inhibitors
Animals ; Cell Proliferation ; Down-Regulation ; Heterografts ; Humans ; Lung Neoplasms/drug therapy ; Lung Neoplasms/metabolism ; MAP Kinase Signaling System ; Mesothelioma/drug therapy ; Mesothelioma/metabolism ; Mesothelioma, Malignant ; Mice ; MicroRNAs/genetics ; MicroRNAs/therapeutic use ; Peritoneal Neoplasms/pathology ; Proto-Oncogene Proteins c-akt/metabolism ; Signal Transduction ; Axl Receptor Tyrosine Kinase
Czasopismo naukowe
Tytuł:
miR-613 suppresses ischemia-reperfusion-induced cardiomyocyte apoptosis by targeting the programmed cell death 10 gene.
Autorzy:
Wu Z; ICU, Departmentof Cardiac Surgery, Tianjin Chest Hospital.
Qi Y
Guo Z
Li P
Zhou D
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Źródło:
Bioscience trends [Biosci Trends] 2016 Sep 05; Vol. 10 (4), pp. 251-7. Date of Electronic Publication: 2016 Aug 17.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis Regulatory Proteins/*physiology
Membrane Proteins/*physiology
MicroRNAs/*physiology
Proto-Oncogene Proteins/*physiology
Reperfusion Injury/*genetics
Apoptosis/genetics ; Apoptosis Regulatory Proteins/genetics ; Apoptosis Regulatory Proteins/metabolism ; Cell Line ; Gene Expression Regulation ; Humans ; Membrane Proteins/genetics ; Membrane Proteins/metabolism ; Myocytes, Cardiac/metabolism ; Myocytes, Cardiac/pathology ; Proto-Oncogene Proteins/genetics ; Proto-Oncogene Proteins/metabolism ; Proto-Oncogene Proteins c-akt ; Signal Transduction
Czasopismo naukowe
Tytuł:
Epidermal growth factor induces HCCR expression via PI3K/Akt/mTOR signaling in PANC-1 pancreatic cancer cells.
Autorzy:
Xu Z; Department of Gastroenterology, the First Affiliated Hospital of Nanjing Medical University, Nanjing 210029, China.
Zhang Y
Jiang J
Yang Y
Shi R
Hao B
Zhang Z
Huang Z
Kim JW
Zhang G
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Źródło:
BMC cancer [BMC Cancer] 2010 Apr 27; Vol. 10, pp. 161. Date of Electronic Publication: 2010 Apr 27.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Signal Transduction*/drug effects
Epidermal Growth Factor/*metabolism
Intracellular Signaling Peptides and Proteins/*metabolism
Pancreatic Neoplasms/*enzymology
Phosphatidylinositol 3-Kinases/*metabolism
Protein Serine-Threonine Kinases/*metabolism
Proto-Oncogene Proteins/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Animals ; Antibodies ; Blotting, Western ; Cell Line, Tumor ; Cell Movement ; Cell Proliferation ; Chromones/pharmacology ; Humans ; Immunohistochemistry ; Mice ; Mice, Inbred BALB C ; Morpholines/pharmacology ; Neoplasm Invasiveness ; Pancreatic Neoplasms/genetics ; Pancreatic Neoplasms/pathology ; Phosphoinositide-3 Kinase Inhibitors ; Promoter Regions, Genetic ; Protein Kinase Inhibitors/pharmacology ; Proto-Oncogene Proteins/genetics ; Proto-Oncogene Proteins/immunology ; Proto-Oncogene Proteins c-akt/antagonists & inhibitors ; Proto-Oncogene Proteins c-akt/genetics ; RNA Interference ; Recombinant Proteins/metabolism ; Sirolimus/pharmacology ; TOR Serine-Threonine Kinases ; Time Factors ; Tissue Array Analysis ; Transfection ; Up-Regulation
Czasopismo naukowe
Tytuł:
BIM-mediated AKT phosphorylation is a key modulator of arsenic trioxide-induced apoptosis in cisplatin-sensitive and -resistant ovarian cancer cells.
Autorzy:
Yuan Z; State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University, Chengdu, China.
Wang F
Zhao Z
Zhao X
Qiu J
Nie C
Wei Y
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Źródło:
PloS one [PLoS One] 2011; Vol. 6 (5), pp. e20586. Date of Electronic Publication: 2011 May 31.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Apoptosis Regulatory Proteins/*metabolism
Arsenicals/*pharmacology
Cell Survival/*drug effects
Cisplatin/*pharmacology
Membrane Proteins/*metabolism
Ovarian Neoplasms/*metabolism
Oxides/*pharmacology
Proto-Oncogene Proteins/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Apoptosis Regulatory Proteins/genetics ; Arsenic Trioxide ; Bcl-2-Like Protein 11 ; Cell Line, Tumor ; Electrophoresis, Polyacrylamide Gel ; Female ; Forkhead Box Protein O3 ; Forkhead Transcription Factors/genetics ; Forkhead Transcription Factors/metabolism ; Gene Silencing ; Humans ; Immunoblotting ; Membrane Proteins/genetics ; Phosphorylation/drug effects ; Proto-Oncogene Proteins/genetics ; Proto-Oncogene Proteins c-akt/genetics ; RNA, Small Interfering
Czasopismo naukowe
Tytuł:
Sphingosine kinase 1 regulates the Akt/FOXO3a/Bim pathway and contributes to apoptosis resistance in glioma cells.
Autorzy:
Guan H; Department of Endocrinology and Diabetes Center, The First Affiliated Hospital of Sun Yat-sen University, Guangzhou, Guangdong, China.
Song L
Cai J
Huang Y
Wu J
Yuan J
Li J
Li M
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Źródło:
PloS one [PLoS One] 2011; Vol. 6 (5), pp. e19946. Date of Electronic Publication: 2011 May 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Apoptosis Regulatory Proteins/*metabolism
Forkhead Transcription Factors/*metabolism
Glioma/*pathology
Membrane Proteins/*metabolism
Phosphotransferases (Alcohol Group Acceptor)/*metabolism
Proto-Oncogene Proteins/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Animals ; Apoptosis Regulatory Proteins/genetics ; Bcl-2-Like Protein 11 ; Blotting, Western ; Brain Neoplasms/genetics ; Brain Neoplasms/metabolism ; Brain Neoplasms/pathology ; Forkhead Box Protein O3 ; Forkhead Transcription Factors/antagonists & inhibitors ; Forkhead Transcription Factors/genetics ; Glioma/genetics ; Glioma/metabolism ; Humans ; Immunoenzyme Techniques ; Luciferases/metabolism ; Membrane Proteins/genetics ; Mice ; Mice, Nude ; Phosphatidylinositol 3-Kinase ; Phosphorylation ; Phosphotransferases (Alcohol Group Acceptor)/genetics ; Proto-Oncogene Proteins/genetics ; Proto-Oncogene Proteins c-akt/genetics ; RNA, Messenger/genetics ; RNA, Small Interfering/genetics ; Reverse Transcriptase Polymerase Chain Reaction ; Signal Transduction ; Tumor Cells, Cultured
Czasopismo naukowe
Tytuł:
p53/PUMA expression in human pulmonary fibroblasts mediates cell activation and migration in silicosis.
Autorzy:
Wang W; Department of Respiratory Medicine, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu 210029, China.; Department of Physiology, Medical School of Southeast University, Nanjing, Jiangsu 210009, China.; Nine Department of Respiratory Medicine, Nanjing Chest Hospital, Nanjing, Jiangsu 210029, China.
Liu H; Department of Physiology, Medical School of Southeast University, Nanjing, Jiangsu 210009, China.; Neurobiology Laboratory, New Drug Screening Centre, China Pharmaceutical University, Nanjing, Jiangsu 210009, China.
Dai X; Department of Physiology, Medical School of Southeast University, Nanjing, Jiangsu 210009, China.
Fang S; Nine Department of Respiratory Medicine, Nanjing Chest Hospital, Nanjing, Jiangsu 210029, China.
Wang X; Department of Physiology, Medical School of Southeast University, Nanjing, Jiangsu 210009, China.
Zhang Y; Nine Department of Respiratory Medicine, Nanjing Chest Hospital, Nanjing, Jiangsu 210029, China.
Yao H; Department of Pharmacology, Medical School of Southeast University, Nanjing, Jiangsu 210009, China.
Zhang X; Department of Respiratory Medicine, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu 210029, China.
Chao J; Department of Physiology, Medical School of Southeast University, Nanjing, Jiangsu 210009, China.; Key Laboratory of Developmental Genes and Human Disease, Southeast University, Nanjing, 210096, China.
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Źródło:
Scientific reports [Sci Rep] 2015 Nov 18; Vol. 5, pp. 16900. Date of Electronic Publication: 2015 Nov 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression*
Apoptosis Regulatory Proteins/*genetics
Fibroblasts/*metabolism
Proto-Oncogene Proteins/*genetics
Silicosis/*etiology
Tumor Suppressor Protein p53/*genetics
Apoptosis Regulatory Proteins/metabolism ; Cell Movement/genetics ; Cell Survival/drug effects ; Cell Survival/genetics ; Collagen/metabolism ; Extracellular Matrix/metabolism ; Gene Expression Regulation/drug effects ; Humans ; Mitogen-Activated Protein Kinases/antagonists & inhibitors ; Mitogen-Activated Protein Kinases/metabolism ; Phosphatidylinositol 3-Kinases/metabolism ; Proto-Oncogene Proteins/metabolism ; Proto-Oncogene Proteins c-akt/antagonists & inhibitors ; Proto-Oncogene Proteins c-akt/metabolism ; RNA, Small Interfering/genetics ; Signal Transduction/drug effects ; Silicon Dioxide/adverse effects ; Silicosis/diagnosis ; Silicosis/metabolism ; Tomography, X-Ray Computed ; Tumor Suppressor Protein p53/metabolism
Czasopismo naukowe
Tytuł:
Resveratrol induces apoptosis through modulation of the Akt/FoxO3a/Bim pathway in HepG2 cells.
Autorzy:
Liu MH; Department of Clinical Laboratory, Affiliated Nanhua Hospital, University of South China, Hengyang, Hunan 421001, P.R. China.
Lin XL; Department of Pathology, The Third People's Hospital of Huizhou Affiliated to Guangzhou Medical University, Huizhou, Guangdong 516002, P.R. China.
Li J; Department of Ultrasonic Diagnosis, BoAi Hospital of Zhongshan, Zhongshan, Guangdong 528403, P.R. China.
He J; Department of Clinical Laboratory, Affiliated Nanhua Hospital, University of South China, Hengyang, Hunan 421001, P.R. China.
Tan TP; Department of Clinical Laboratory, Affiliated Nanhua Hospital, University of South China, Hengyang, Hunan 421001, P.R. China.
Wu SJ; Department of Clinical Laboratory, Affiliated Nanhua Hospital, University of South China, Hengyang, Hunan 421001, P.R. China.
Yu S; Department of Clinical Laboratory, Affiliated Nanhua Hospital, University of South China, Hengyang, Hunan 421001, P.R. China.
Chen L; Department of Clinical Laboratory, Affiliated Nanhua Hospital, University of South China, Hengyang, Hunan 421001, P.R. China.
Liu J; Department of Clinical Laboratory, Affiliated Nanhua Hospital, University of South China, Hengyang, Hunan 421001, P.R. China.
Tian W; Department of Clinical Laboratory, Affiliated Nanhua Hospital, University of South China, Hengyang, Hunan 421001, P.R. China.
Chen YD; Department of Clinical Laboratory, Affiliated Nanhua Hospital, University of South China, Hengyang, Hunan 421001, P.R. China.
Fu HY; Department of Clinical Laboratory, Affiliated Nanhua Hospital, University of South China, Hengyang, Hunan 421001, P.R. China.
Yuan C; Department of Cardiology, The First Hospital of Changsha, Changsha, Hunan 410005, P.R. China.
Zhang Y; Department of Pathology, Mawangdui Hospital, Changsha, Hunan 410016, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2016 Feb; Vol. 13 (2), pp. 1689-94. Date of Electronic Publication: 2015 Dec 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Apoptosis Regulatory Proteins/*metabolism
Forkhead Transcription Factors/*metabolism
Membrane Proteins/*metabolism
Proto-Oncogene Proteins/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Signal Transduction/*drug effects
Stilbenes/*pharmacology
Bcl-2-Like Protein 11 ; Cell Nucleus/drug effects ; Cell Nucleus/metabolism ; Cell Survival/drug effects ; Forkhead Box Protein O3 ; Hep G2 Cells ; Humans ; Phosphorylation/drug effects ; Protein Transport/drug effects ; Resveratrol
Czasopismo naukowe
Tytuł:
Axl receptor tyrosine kinase is a potential therapeutic target in renal cell carcinoma.
Autorzy:
Yu H; 1] Xiangya Hospital, Central South University, Changsha, Hunan 410008, People's Republic of China [2] Department of Medicine, University of Southern California, Norris Hospital, NOR 6332, 1441 Eastlake Avenue, Los Angeles, CA 90033, USA.
Liu R; Department of Medicine, University of Southern California, Norris Hospital, NOR 6332, 1441 Eastlake Avenue, Los Angeles, CA 90033, USA.
Ma B; Department of Medicine, University of Southern California, Norris Hospital, NOR 6332, 1441 Eastlake Avenue, Los Angeles, CA 90033, USA.
Li X; Department of Medicine, University of Southern California, Norris Hospital, NOR 6332, 1441 Eastlake Avenue, Los Angeles, CA 90033, USA.
Yen HY; Department of Medicine, University of Southern California, Norris Hospital, NOR 6332, 1441 Eastlake Avenue, Los Angeles, CA 90033, USA.
Zhou Y; Department of Medicine, University of Southern California, Norris Hospital, NOR 6332, 1441 Eastlake Avenue, Los Angeles, CA 90033, USA.
Krasnoperov V; Vasgene Therapeutics Inc., Los Angeles, CA 90033, USA.
Xia Z; The State Key Laboratory of Medical Genetics and School of Life Sciences, Central South University, Changsha, Hunan 410078, People's Republic of China.
Zhang X; 1] Xiangya Hospital, Central South University, Changsha, Hunan 410008, People's Republic of China [2] Department of Medicine, University of Southern California, Norris Hospital, NOR 6332, 1441 Eastlake Avenue, Los Angeles, CA 90033, USA.
Bove AM; Department of Urology, Campus Bio-Medico University, Rome 00128, Italy.
Buscarini M; Department of Urology, Campus Bio-Medico University, Rome 00128, Italy.
Parekh D; Division of Surgery, University of Southern California, Los Angeles, CA 90033, USA.
Gill IS; Department of Urology, University of Southern California, Los Angeles, CA 90033, USA.
Liao Q; Department of Orthopedic Surgery, Xiangya Hospital, Central South University, Changsha, Hunan 41008, People's Republic of China.
Tretiakova M; Department of Pathology, University of Washington, Seattle, WA 98195, USA.
Quinn D; Department of Medicine, University of Southern California, Norris Hospital, NOR 6332, 1441 Eastlake Avenue, Los Angeles, CA 90033, USA.
Zhao J; Xiangya Hospital, Central South University, Changsha, Hunan 410008, People's Republic of China.
Gill PS; Department of Medicine, University of Southern California, Norris Hospital, NOR 6332, 1441 Eastlake Avenue, Los Angeles, CA 90033, USA.
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Źródło:
British journal of cancer [Br J Cancer] 2015 Aug 11; Vol. 113 (4), pp. 616-25. Date of Electronic Publication: 2015 Jul 16.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Carcinoma, Renal Cell/*metabolism
Kidney Neoplasms/*metabolism
Proto-Oncogene Proteins/*metabolism
Receptor Protein-Tyrosine Kinases/*metabolism
Animals ; Apoptosis/physiology ; Cell Line ; Cell Line, Tumor ; Cell Movement/physiology ; Cell Proliferation/physiology ; Cell Survival/physiology ; HEK293 Cells ; HT29 Cells ; Humans ; Male ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Phosphatidylinositol 3-Kinases/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Signal Transduction/physiology ; Axl Receptor Tyrosine Kinase
Czasopismo naukowe
Tytuł:
Transforming growth factor type-β inhibits Mas receptor expression in fibroblasts but not in myoblasts or differentiated myotubes; Relevance to fibrosis associated to muscular dystrophies.
Autorzy:
Cofre C; Center for Aging and Regeneration, CARE Chile-UC and Department of Cell and Molecular Biology, Faculty of Biological Sciences, Pontificia Universidad Católica de Chile, Santiago, Chile.
Acuña MJ
Contreras O
Morales MG
Riquelme C
Cabello-Verrugio C
Brandan E
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Źródło:
BioFactors (Oxford, England) [Biofactors] 2015 Mar-Apr; Vol. 41 (2), pp. 111-20. Date of Electronic Publication: 2015 Mar 23.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Fibroblasts/*drug effects
Muscle Fibers, Skeletal/*drug effects
Myoblasts/*drug effects
Proto-Oncogene Proteins/*antagonists & inhibitors
Receptors, G-Protein-Coupled/*antagonists & inhibitors
Transforming Growth Factor beta/*pharmacology
Angiotensin I/pharmacology ; Angiotensin II/pharmacology ; Animals ; Cell Line ; Disease Models, Animal ; Fibroblasts/metabolism ; Fibroblasts/pathology ; Fibrosis ; Gene Expression Regulation ; MAP Kinase Kinase 1/genetics ; MAP Kinase Kinase 1/metabolism ; MAP Kinase Kinase 2/genetics ; MAP Kinase Kinase 2/metabolism ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Inbred mdx ; Mice, Knockout ; Muscle Fibers, Skeletal/metabolism ; Muscle Fibers, Skeletal/pathology ; Muscle, Skeletal/drug effects ; Muscle, Skeletal/metabolism ; Muscle, Skeletal/pathology ; Muscular Dystrophy, Duchenne/genetics ; Muscular Dystrophy, Duchenne/metabolism ; Muscular Dystrophy, Duchenne/pathology ; Myoblasts/metabolism ; Myoblasts/pathology ; Organ Specificity ; Peptide Fragments/pharmacology ; Phosphatidylinositol 3-Kinases/genetics ; Phosphatidylinositol 3-Kinases/metabolism ; Phosphorylation/drug effects ; Primary Cell Culture ; Proto-Oncogene Mas ; Proto-Oncogene Proteins/genetics ; Proto-Oncogene Proteins/metabolism ; Proto-Oncogene Proteins c-akt/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; Receptors, G-Protein-Coupled/genetics ; Receptors, G-Protein-Coupled/metabolism ; Signal Transduction
Czasopismo naukowe

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