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Wyszukujesz frazę ""Qian, Youcun"" wg kryterium: Autor

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    Wyświetlanie 1-6 z 6
    Tytuł:
    The impact of lung microbiota dysbiosis on inflammation.
    Autorzy:
    Yang D; CAS Key Laboratory of Tissue Microenvironment and Tumor, CAS Center for Excellence in Molecular Cell Science, Shanghai Institute of Nutrition and Health, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.
    Xing Y; CAS Key Laboratory of Tissue Microenvironment and Tumor, CAS Center for Excellence in Molecular Cell Science, Shanghai Institute of Nutrition and Health, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.
    Song X; Department of Immunology, Harvard Medical School, Boston, MA, USA.
    Qian Y; CAS Key Laboratory of Tissue Microenvironment and Tumor, CAS Center for Excellence in Molecular Cell Science, Shanghai Institute of Nutrition and Health, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.; School of Life Science and Technology, ShanghaiTech University, Shanghai, China.
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    Źródło:
    Immunology [Immunology] 2020 Feb; Vol. 159 (2), pp. 156-166. Date of Electronic Publication: 2019 Nov 11.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't; Review
    MeSH Terms:
    Dysbiosis*
    Microbiota*
    Lung/*microbiology
    Pneumonia/*microbiology
    Adaptive Immunity ; Animals ; Host-Pathogen Interactions ; Humans ; Immunity, Innate ; Lung/immunology ; Lung/metabolism ; Pneumonia/immunology ; Pneumonia/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Tumor necrosis factor-α blockade ameliorates diabetic nephropathy in rats.
    Autorzy:
    Cheng D; Department of Nephrology, Shanghai Eighth People's Hospital, Shanghai, China.
    Liang R; Department of Nephrology, Shanghai Eighth People's Hospital, Shanghai, China.
    Huang B; Department of Emergency, Xiangya Hospital, Central South University, Changsha, China.
    Hou J; Department of Nephrology, Shanghai Eighth People's Hospital, Shanghai, China.
    Yin J; Department of Nephrology, Shanghai Eighth People's Hospital, Shanghai, China.
    Zhao T; Department of Nephrology, Shanghai Eighth People's Hospital, Shanghai, China.
    Zhou L; Department of Nephrology, Shanghai Eighth People's Hospital, Shanghai, China.
    Wu R; Department of Nephrology, Shanghai Eighth People's Hospital, Shanghai, China.
    Qian Y; Department of Nephrology, Shanghai Eighth People's Hospital, Shanghai, China.; Key Laboratory of Stem Cell Biology, CAS Center for Excellence in Molecular Cell Science, Institute of Health Sciences, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences & Shanghai Jiao Tong University School of Medicine, Shanghai, China.
    Wang F; Department of Nephrology, Shanghai Eighth People's Hospital, Shanghai, China.
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    Źródło:
    Clinical kidney journal [Clin Kidney J] 2019 Nov 07; Vol. 14 (1), pp. 301-308. Date of Electronic Publication: 2019 Nov 07 (Print Publication: 2021).
    Typ publikacji:
    Journal Article
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    The microRNA miR-23b suppresses IL-17-associated autoimmune inflammation by targeting TAB2, TAB3 and IKK-α.
    Autorzy:
    Zhu S; The Key Laboratory of Stem Cell Biology, Institute of Health Sciences, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences and Shanghai Jiao Tong University School of Medicine, Shanghai, China.
    Pan W
    Song X
    Liu Y
    Shao X
    Tang Y
    Liang D
    He D
    Wang H
    Liu W
    Shi Y
    Harley JB
    Shen N
    Qian Y
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    Źródło:
    Nature medicine [Nat Med] 2012 Jul; Vol. 18 (7), pp. 1077-86.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Autoimmune Diseases/*genetics
    Carrier Proteins/*metabolism
    I-kappa B Kinase/*metabolism
    Inflammation/*genetics
    Interleukin-17/*metabolism
    MicroRNAs/*metabolism
    Animals ; Autoimmune Diseases/complications ; Autoimmune Diseases/pathology ; Cytokines/metabolism ; Disease Models, Animal ; Down-Regulation/genetics ; Gene Expression Profiling ; Humans ; Immunoblotting ; Inflammation/complications ; Inflammation/pathology ; Inflammation Mediators/metabolism ; Mice ; MicroRNAs/genetics ; Signal Transduction/genetics
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    IL-17RE is the functional receptor for IL-17C and mediates mucosal immunity to infection with intestinal pathogens.
    Autorzy:
    Song X; The Key Laboratory of Stem Cell Biology, Institute of Health Sciences, Chinese Academy of Sciences & Shanghai Jiao Tong University School of Medicine, Shanghai, China.
    Zhu S
    Shi P
    Liu Y
    Shi Y
    Levin SD
    Qian Y
    Pokaż więcej
    Źródło:
    Nature immunology [Nat Immunol] 2011 Oct 12; Vol. 12 (12), pp. 1151-8. Date of Electronic Publication: 2011 Oct 12.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Enterobacteriaceae Infections/*immunology
    Immunity, Mucosal/*immunology
    Interleukin-17/*metabolism
    Intestinal Mucosa/*immunology
    Receptors, Interleukin-17/*metabolism
    Animals ; Antimicrobial Cationic Peptides/genetics ; Antimicrobial Cationic Peptides/immunology ; Cell Line ; Citrobacter rodentium ; Colon/immunology ; Colon/metabolism ; Enterobacteriaceae Infections/genetics ; Enterobacteriaceae Infections/microbiology ; Gene Expression Regulation ; HEK293 Cells ; HT29 Cells ; Humans ; Immunity, Mucosal/genetics ; Interleukins/metabolism ; Intestinal Mucosa/microbiology ; Intestinal Mucosa/pathology ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Receptors, Interleukin-17/genetics ; Signal Transduction ; Interleukin-22
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    The adaptor Act1 is required for interleukin 17-dependent signaling associated with autoimmune and inflammatory disease.
    Autorzy:
    Qian Y; Department of Immunology, Cleveland Clinic, Cleveland, Ohio 44195, USA.
    Liu C
    Hartupee J
    Altuntas CZ
    Gulen MF
    Jane-Wit D
    Xiao J
    Lu Y
    Giltiay N
    Liu J
    Kordula T
    Zhang QW
    Vallance B
    Swaidani S
    Aronica M
    Tuohy VK
    Hamilton T
    Li X
    Pokaż więcej
    Źródło:
    Nature immunology [Nat Immunol] 2007 Mar; Vol. 8 (3), pp. 247-56. Date of Electronic Publication: 2007 Feb 04.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural
    MeSH Terms:
    Autoimmune Diseases*
    Adaptor Proteins, Signal Transducing/*immunology
    Inflammation/*immunology
    Interleukin-17/*immunology
    Signal Transduction/*immunology
    Adaptor Proteins, Signal Transducing/metabolism ; Adoptive Transfer ; Animals ; Autoimmunity ; B-Cell Activation Factor Receptor/immunology ; B-Cell Activation Factor Receptor/metabolism ; CD40 Antigens/immunology ; CD40 Antigens/metabolism ; Colitis/immunology ; Colitis/metabolism ; Colitis/pathology ; Encephalomyelitis, Autoimmune, Experimental/immunology ; Encephalomyelitis, Autoimmune, Experimental/metabolism ; Encephalomyelitis, Autoimmune, Experimental/pathology ; Enzyme-Linked Immunosorbent Assay ; Female ; Fluorescent Antibody Technique ; Gene Expression ; Gene Expression Regulation/immunology ; HeLa Cells ; Humans ; Interleukin-17/metabolism ; Mice ; Receptors, Interleukin-17/immunology ; Receptors, Interleukin-17/metabolism ; Reverse Transcriptase Polymerase Chain Reaction ; T-Lymphocytes/immunology ; Transfection ; Tumor Necrosis Factor Receptor-Associated Peptides and Proteins
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    SIGIRR, a negative regulator of Toll-like receptor-interleukin 1 receptor signaling.
    Autorzy:
    Wald D; Cleveland Clinic Foundation, Department of Immunology, Cleveland, Ohio 44195, USA.
    Qin J
    Zhao Z
    Qian Y
    Naramura M
    Tian L
    Towne J
    Sims JE
    Stark GR
    Li X
    Pokaż więcej
    Źródło:
    Nature immunology [Nat Immunol] 2003 Sep; Vol. 4 (9), pp. 920-7. Date of Electronic Publication: 2003 Aug 17.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
    MeSH Terms:
    Membrane Glycoproteins/*immunology
    Receptors, Cell Surface/*immunology
    Receptors, Interleukin-1/*immunology
    Animals ; Chemokines/immunology ; Chemokines/metabolism ; Gene Expression Regulation ; Humans ; Immunoblotting ; Interleukin-1/immunology ; Interleukin-18/immunology ; Jurkat Cells ; Lipopolysaccharides/immunology ; Membrane Glycoproteins/antagonists & inhibitors ; Mice ; Mice, Inbred BALB C ; Mice, Inbred C57BL ; Mice, Knockout ; NF-kappa B/immunology ; RNA/chemistry ; RNA/genetics ; Receptors, Cell Surface/antagonists & inhibitors ; Signal Transduction/immunology ; Toll-Like Receptors ; Transcription Factor AP-1/immunology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
      Wyświetlanie 1-6 z 6

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