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    Wyświetlanie 1-7 z 7
    Tytuł:
    Reduced sister chromatid cohesion acts as a tumor penetrance modifier.
    Autorzy:
    Wang J; Department of Pharmacology and Toxicology, University of Alabama at Birmingham Heersink School of Medicine, Birmingham, Alabama, United States of America.
    Thomas HR; Department of Pharmacology and Toxicology, University of Alabama at Birmingham Heersink School of Medicine, Birmingham, Alabama, United States of America.
    Chen Y; Department of Genetics, University of Alabama at Birmingham Heersink School of Medicine, Birmingham, Alabama, United States of America.; Informatics Institute, University of Alabama at Birmingham Heersink School of Medicine, Alabama, United States of America.
    Percival SM; Department of Pharmacology and Toxicology, University of Alabama at Birmingham Heersink School of Medicine, Birmingham, Alabama, United States of America.
    Waldrep SC; Department of Pharmacology and Toxicology, University of Alabama at Birmingham Heersink School of Medicine, Birmingham, Alabama, United States of America.
    Ramaker RC; Hudson Alpha Institute for Biotechnology, Huntsville, Alabama, United States of America.
    Thompson RG; Department of Pharmacology and Toxicology, University of Alabama at Birmingham Heersink School of Medicine, Birmingham, Alabama, United States of America.
    Cooper SJ; Hudson Alpha Institute for Biotechnology, Huntsville, Alabama, United States of America.
    Chong Z; Department of Genetics, University of Alabama at Birmingham Heersink School of Medicine, Birmingham, Alabama, United States of America.; Informatics Institute, University of Alabama at Birmingham Heersink School of Medicine, Alabama, United States of America.
    Parant JM; Department of Pharmacology and Toxicology, University of Alabama at Birmingham Heersink School of Medicine, Birmingham, Alabama, United States of America.
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    Źródło:
    PLoS genetics [PLoS Genet] 2022 Aug 22; Vol. 18 (8), pp. e1010341. Date of Electronic Publication: 2022 Aug 22 (Print Publication: 2022).
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
    MeSH Terms:
    Chromosome Segregation*/genetics
    Neoplasms*/genetics
    Acetyltransferases/genetics ; Animals ; Cell Cycle Proteins/genetics ; Cell Cycle Proteins/metabolism ; Chromatids/genetics ; Chromatids/metabolism ; Chromosomal Proteins, Non-Histone/genetics ; Chromosomal Proteins, Non-Histone/metabolism ; Humans ; Mice ; Penetrance ; Tumor Suppressor Protein p53/genetics ; Zebrafish/genetics
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Pooled CRISPR screening in pancreatic cancer cells implicates co-repressor complexes as a cause of multiple drug resistance via regulation of epithelial-to-mesenchymal transition.
    Autorzy:
    Ramaker RC; University of Alabama-Birmingham, Birmingham, AL, 35294, USA.; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA.
    Hardigan AA; University of Alabama-Birmingham, Birmingham, AL, 35294, USA.; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA.
    Gordon ER; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA.
    Wright CA; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA.; University of Alabama - Huntsville, Huntsville, AL, 35899, USA.
    Myers RM; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA.
    Cooper SJ; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA. .
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    Źródło:
    BMC cancer [BMC Cancer] 2021 May 29; Vol. 21 (1), pp. 632. Date of Electronic Publication: 2021 May 29.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Antineoplastic Agents/*pharmacology
    Carcinoma, Pancreatic Ductal/*drug therapy
    Drug Resistance, Multiple/*genetics
    Drug Resistance, Neoplasm/*genetics
    Pancreatic Neoplasms/*drug therapy
    Antineoplastic Agents/therapeutic use ; CRISPR-Cas Systems/genetics ; Carcinoma, Pancreatic Ductal/genetics ; Carcinoma, Pancreatic Ductal/mortality ; Carcinoma, Pancreatic Ductal/pathology ; Cell Line, Tumor ; Cell Movement/genetics ; Chromatin Immunoprecipitation Sequencing ; Co-Repressor Proteins/genetics ; Co-Repressor Proteins/metabolism ; Epithelial-Mesenchymal Transition/genetics ; Gene Expression Regulation, Neoplastic ; Gene Knockdown Techniques ; Histone Deacetylase 1/genetics ; Histone Deacetylase 1/metabolism ; Humans ; Pancreatic Neoplasms/genetics ; Pancreatic Neoplasms/mortality ; Pancreatic Neoplasms/pathology ; RNA-Seq
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    De novo mutations in the GTP/GDP-binding region of RALA, a RAS-like small GTPase, cause intellectual disability and developmental delay.
    Autorzy:
    Hiatt SM; HudsonAlpha Institute for Biotechnology, Huntsville, AL, United States of America.
    Neu MB; HudsonAlpha Institute for Biotechnology, Huntsville, AL, United States of America.; Department of Genetics, University of Alabama at Birmingham, Birmingham, AL, United States of America.
    Ramaker RC; HudsonAlpha Institute for Biotechnology, Huntsville, AL, United States of America.; Department of Genetics, University of Alabama at Birmingham, Birmingham, AL, United States of America.
    Hardigan AA; HudsonAlpha Institute for Biotechnology, Huntsville, AL, United States of America.; Department of Genetics, University of Alabama at Birmingham, Birmingham, AL, United States of America.
    Prokop JW; Department of Pediatrics and Human Development, Michigan State University, East Lansing, MI, United States of America.
    Hancarova M; Department of Biology and Medical Genetics, Charles University 2nd Faculty of Medicine and University Hospital Motol, Prague, Czech Republic.
    Prchalova D; Department of Biology and Medical Genetics, Charles University 2nd Faculty of Medicine and University Hospital Motol, Prague, Czech Republic.
    Havlovicova M; Department of Biology and Medical Genetics, Charles University 2nd Faculty of Medicine and University Hospital Motol, Prague, Czech Republic.
    Prchal J; Laboratory of NMR Spectroscopy, University of Chemistry and Technology, Prague, Czech Republic.
    Stranecky V; Department of Pediatrics and Adolescent Medicine, Diagnostic and Research Unit for Rare Diseases, Charles University 1st Faculty of Medicine and General University Hospital, Prague, Czech Republic.
    Yim DKC; Kaiser Permanente-Hawaii, Honolulu, HI, United States of America.
    Powis Z; Department of Emerging Genetic Medicine, Ambry Genetics, Aliso Viejo, CA, United States of America.
    Keren B; Department of Genetics, La Pitié-Salpêtrière Hospital, Assistance Publique-Hôpitaux de Paris, Paris, France.
    Nava C; Department of Genetics, La Pitié-Salpêtrière Hospital, Assistance Publique-Hôpitaux de Paris, Paris, France.
    Mignot C; Department of Genetics, La Pitié-Salpêtrière Hospital, Assistance Publique-Hôpitaux de Paris, Paris, France.; Centre de Référence Déficiences Intellectuelles de Causes Rares, Paris, France.; Groupe de Recherche Clinique UPMC 'Déficience Intellectuelle et Autisme', Paris, France.
    Rio M; Centre de Référence Déficiences Intellectuelles de Causes Rares, Paris, France.; Assistance Publique-Hôpitaux de Paris, service de Génétique, Hôpital Necker-Enfants-Malades, Paris, France.
    Revah-Politi A; Institute for Genomic Medicine, Columbia University Medical Center, New York, NY, United States of America.
    Hemati P; Institute for Genomic Medicine, Columbia University Medical Center, New York, NY, United States of America.
    Stong N; Institute for Genomic Medicine, Columbia University Medical Center, New York, NY, United States of America.
    Iglesias AD; Division of Clinical Genetics, Department of Pediatrics, Columbia University Medical Center, New York, NY, United States of America.
    Suchy SF; GeneDx, Gaithersburg, MD, United States of America.
    Willaert R; GeneDx, Gaithersburg, MD, United States of America.
    Wentzensen IM; GeneDx, Gaithersburg, MD, United States of America.
    Wheeler PG; Arnold Palmer Hospital, Division of Genetics, Orlando, FL, United States of America.
    Brick L; Department of Genetics, McMaster Children's Hospital, Hamilton, Ontario, Canada.
    Kozenko M; Department of Genetics, McMaster Children's Hospital, Hamilton, Ontario, Canada.
    Hurst ACE; Department of Genetics, University of Alabama at Birmingham, Birmingham, AL, United States of America.
    Wheless JW; Division of Pediatric Neurology, University of Tennessee Health Science Center, Neuroscience Institute & Le Bonheur Comprehensive Epilepsy Program, Memphis, TN, United States of America.; Le Bonheur Children's Hospital, Memphis, TN, United States of America.
    Lacassie Y; Division of Clinical Genetics, Louisiana State University Health Sciences Center, New Orleans, LA, United States of America.; Department of Genetics, Children's Hospital, New Orleans, LA, United States of America.
    Myers RM; HudsonAlpha Institute for Biotechnology, Huntsville, AL, United States of America.
    Barsh GS; HudsonAlpha Institute for Biotechnology, Huntsville, AL, United States of America.
    Sedlacek Z; Department of Biology and Medical Genetics, Charles University 2nd Faculty of Medicine and University Hospital Motol, Prague, Czech Republic.
    Cooper GM; HudsonAlpha Institute for Biotechnology, Huntsville, AL, United States of America.
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    Źródło:
    PLoS genetics [PLoS Genet] 2018 Nov 30; Vol. 14 (11), pp. e1007671. Date of Electronic Publication: 2018 Nov 30 (Print Publication: 2018).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Mutation*
    Developmental Disabilities/*genetics
    Intellectual Disability/*genetics
    Mitochondrial Proteins/*genetics
    Protein Interaction Domains and Motifs/*genetics
    ral GTP-Binding Proteins/*genetics
    ras Proteins/*genetics
    Facies ; Genotype ; Guanosine Diphosphate/metabolism ; Guanosine Triphosphate/metabolism ; Humans ; Mitochondrial Proteins/chemistry ; Models, Molecular ; Mutation, Missense ; Phenotype ; Protein Conformation ; ral GTP-Binding Proteins/chemistry ; ras Proteins/chemistry
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Post-mortem molecular profiling of three psychiatric disorders.
    Autorzy:
    Ramaker RC; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.; Department of Genetics, The University of Alabama at Birmingham, Birmingham, AL, USA.
    Bowling KM; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.
    Lasseigne BN; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.
    Hagenauer MH; Mental Health Research Institute, University of Michigan, Ann Arbor, MI, USA.
    Hardigan AA; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.; Department of Genetics, The University of Alabama at Birmingham, Birmingham, AL, USA.
    Davis NS; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.; Present address: Duke University, Durham, NC, USA.
    Gertz J; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.; Present address: University of Utah School of Medicine, Salt Lake City, UT, USA.
    Cartagena PM; Department of Psychiatry and Human Behavior, College of Medicine, University of California, Irvine, CA, USA.
    Walsh DM; Department of Psychiatry and Human Behavior, College of Medicine, University of California, Irvine, CA, USA.
    Vawter MP; Department of Psychiatry and Human Behavior, College of Medicine, University of California, Irvine, CA, USA.
    Jones EG
    Schatzberg AF; Department of Psychiatry, Stanford University School of Medicine, Stanford, CA, USA.
    Barchas JD; Psychiatry, Weill Cornell Medical College, New York, NY, USA.
    Watson SJ; Mental Health Research Institute, University of Michigan, Ann Arbor, MI, USA.
    Bunney BG; Department of Psychiatry and Human Behavior, College of Medicine, University of California, Irvine, CA, USA.
    Akil H; Mental Health Research Institute, University of Michigan, Ann Arbor, MI, USA.
    Bunney WE; Department of Psychiatry and Human Behavior, College of Medicine, University of California, Irvine, CA, USA.
    Li JZ; Department of Human Genetics, University of Michigan, Ann Arbor, MI, USA.
    Cooper SJ; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.
    Myers RM; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA. .
    Pokaż więcej
    Źródło:
    Genome medicine [Genome Med] 2017 Jul 28; Vol. 9 (1), pp. 72. Date of Electronic Publication: 2017 Jul 28.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
    MeSH Terms:
    Transcriptome*
    Bipolar Disorder/*genetics
    Brain/*metabolism
    Depressive Disorder, Major/*genetics
    Schizophrenia/*genetics
    Autopsy ; Bipolar Disorder/metabolism ; Chromatin Immunoprecipitation ; Depressive Disorder, Major/metabolism ; Early Growth Response Protein 1/metabolism ; Female ; Humans ; Male ; Metabolomics ; Schizophrenia/metabolism ; Sequence Analysis, RNA
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Genome-wide DNA methylation measurements in prostate tissues uncovers novel prostate cancer diagnostic biomarkers and transcription factor binding patterns.
    Autorzy:
    Kirby MK; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.; Present Address: TRM Oncology, 5901-C Peachtree Dunwoody Rd, Suite 200, Atlanta, GA, 30328, USA.
    Ramaker RC; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.; Department of Genetics, Kaul Human Genetics Building, Suite 230, 720 20th Street South, Birmingham, AL, 35294, USA.
    Roberts BS; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.
    Lasseigne BN; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.
    Gunther DS; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.; Present Address: University of Southern California, University Park, Los Angeles, CA, 90089, USA.
    Burwell TC; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.; Present Address: Boeing Co., 499 Boeing Blvd, SW, Huntsville, AL, 35824, USA.
    Davis NS; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.; Present Address: Duke University, 101 Science Drive, Durham, NC, 27708, USA.
    Gulzar ZG; Department of Urology, Stanford University Medical Center, Room S287, 300 Pasteur Drive, Stanford, CA, 94305-5118, USA.; Present Address: NuGEN technologies, 201 Industrial Rd #310, San Carlos, CA, 94070, USA.
    Absher DM; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.
    Cooper SJ; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA.
    Brooks JD; Department of Urology, Stanford University Medical Center, Room S287, 300 Pasteur Drive, Stanford, CA, 94305-5118, USA. .
    Myers RM; HudsonAlpha Institute for Biotechnology, 601 Genome Way, Huntsville, AL, 35806, USA. .
    Pokaż więcej
    Źródło:
    BMC cancer [BMC Cancer] 2017 Apr 17; Vol. 17 (1), pp. 273. Date of Electronic Publication: 2017 Apr 17.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    DNA Methylation*
    Biomarkers, Tumor/*genetics
    Prostatic Neoplasms/*genetics
    Transcription Factors/*genetics
    Adult ; Aged ; Biomarkers, Tumor/metabolism ; Cytosine/metabolism ; Enhancer of Zeste Homolog 2 Protein/genetics ; Enhancer of Zeste Homolog 2 Protein/metabolism ; Humans ; Male ; Middle Aged ; Neoplasm Staging ; Prostatic Neoplasms/metabolism ; Prostatic Neoplasms/pathology ; Prostatic Neoplasms, Castration-Resistant/genetics ; Prostatic Neoplasms, Castration-Resistant/metabolism ; Transcription Factors/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Kinase Gene Expression Profiling of Metastatic Clear Cell Renal Cell Carcinoma Tissue Identifies Potential New Therapeutic Targets.
    Autorzy:
    Ghatalia P; Department of Internal Medicine, University of Alabama at Birmingham (UAB), Birmingham, AL, United States of America.
    Yang ES; Department of Radiation Oncology, UAB, Birmingham, AL, United States of America.
    Lasseigne BN; HudsonAlpha Institute for Biotechnology, Huntsville, AL, United States of America.
    Ramaker RC; HudsonAlpha Institute for Biotechnology, Huntsville, AL, United States of America.; Department of Genetics, UAB, Birmingham, AL, United States of America.
    Cooper SJ; HudsonAlpha Institute for Biotechnology, Huntsville, AL, United States of America.
    Chen D; UAB Department of Preventive Medicine, Birmingham, AL, United States of America.
    Sudarshan S; UAB Department of Urology, Birmingham, AL, United States of America.
    Wei S; UAB Department of Urologic Pathology, Birmingham, AL, United States of America.
    Guru AS; Department of Radiation Oncology, UAB, Birmingham, AL, United States of America.
    Zhao A; Department of Radiation Oncology, UAB, Birmingham, AL, United States of America.
    Cooper T; Department of Radiation Oncology, UAB, Birmingham, AL, United States of America.
    Della Manna DL; Department of Radiation Oncology, UAB, Birmingham, AL, United States of America.
    Naik G; UAB Department of Medicine, Section of Hematology-Oncology and the UAB Comprehensive Cancer Center, Birmingham, AL, United States of America.
    Myers RM; HudsonAlpha Institute for Biotechnology, Huntsville, AL, United States of America.
    Sonpavde G; UAB Department of Medicine, Section of Hematology-Oncology and the UAB Comprehensive Cancer Center, Birmingham, AL, United States of America.
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    Źródło:
    PloS one [PLoS One] 2016 Aug 30; Vol. 11 (8), pp. e0160924. Date of Electronic Publication: 2016 Aug 30 (Print Publication: 2016).
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Carcinoma, Renal Cell/*genetics
    Gene Expression Profiling/*methods
    Kidney Neoplasms/*genetics
    Protein Kinases/*genetics
    Sequence Analysis, RNA/*methods
    Adult ; Aged ; Female ; Gene Expression Regulation, Enzymologic ; Gene Expression Regulation, Neoplastic ; Humans ; Male ; Middle Aged ; Neoplasm Metastasis ; Signal Transduction ; Up-Regulation
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Distinct gene regulatory programs define the inhibitory effects of liver X receptors and PPARG on cancer cell proliferation.
    Autorzy:
    Savic D; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA.
    Ramaker RC; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA.; Department of Genetics, University of Alabama at Birmingham, Birmingham, AL, 35294, USA.
    Roberts BS; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA.
    Dean EC; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA.
    Burwell TC; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA.
    Meadows SK; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA.
    Cooper SJ; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA.
    Garabedian MJ; Departments of Microbiology and Urology, New York University, New York, NY, 10016, USA.
    Gertz J; Department of Oncological Sciences, Huntsman Cancer Institute, University of Utah, Salt Lake City, UT, 84112, USA.
    Myers RM; HudsonAlpha Institute for Biotechnology, Huntsville, AL, 35806, USA. .
    Pokaż więcej
    Źródło:
    Genome medicine [Genome Med] 2016 Jul 11; Vol. 8 (1), pp. 74. Date of Electronic Publication: 2016 Jul 11.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
    MeSH Terms:
    Gene Expression Regulation, Neoplastic*
    Gene Regulatory Networks*
    Liver X Receptors/*genetics
    PPAR gamma/*genetics
    Cell Proliferation ; Chromatin/chemistry ; Chromatin/metabolism ; Chromatin Assembly and Disassembly ; Energy Metabolism/genetics ; Glutathione/metabolism ; HT29 Cells ; Humans ; Liver X Receptors/metabolism ; Oxidative Stress/genetics ; PPAR gamma/metabolism ; Signal Transduction
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
      Wyświetlanie 1-7 z 7

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