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Tytuł:
Increased Type I interferon signaling and brain endothelial barrier dysfunction in an experimental model of Alzheimer's disease.
Autorzy:
Jana A; Division of Cardiology, Department of Medicine, College of Medicine, University of Illinois, Chicago, IL, 60612, USA.
Wang X; Department of Biomedical Engineering, University of Illinois at Chicago, Chicago, IL, 60612, USA.; Department of Biochemistry and Molecular Genetics, University of Illinois, College of Medicine, Chicago, IL, 60607, USA.
Leasure JW; Department of Biochemistry and Molecular Genetics, University of Illinois, College of Medicine, Chicago, IL, 60607, USA.
Magana L; Department of Biochemistry and Molecular Genetics, University of Illinois, College of Medicine, Chicago, IL, 60607, USA.
Wang L; Division of Cardiology, Department of Medicine, College of Medicine, University of Illinois, Chicago, IL, 60612, USA.; Department of Biochemistry and Molecular Genetics, University of Illinois, College of Medicine, Chicago, IL, 60607, USA.
Kim YM; Division of Cardiology, Department of Medicine, College of Medicine, University of Illinois, Chicago, IL, 60612, USA.; Department of Biochemistry and Molecular Genetics, University of Illinois, College of Medicine, Chicago, IL, 60607, USA.
Dodiya H; Department of Neurobiology, University of Chicago, Chicago, IL, 60637, USA.; The Microbiome Center, University of Chicago, Chicago, IL, 60637, USA.
Toth PT; Research Resources Center, University of Chicago, Chicago, IL, 60612, USA.; Department of Pharmacology and Regenerative Medicine, University of Chicago, Chicago, IL, 60612, USA.
Sisodia SS; Department of Neurobiology, University of Chicago, Chicago, IL, 60637, USA.; The Microbiome Center, University of Chicago, Chicago, IL, 60637, USA.
Rehman J; Division of Cardiology, Department of Medicine, College of Medicine, University of Illinois, Chicago, IL, 60612, USA. .; Department of Biomedical Engineering, University of Illinois at Chicago, Chicago, IL, 60612, USA. .; Department of Biochemistry and Molecular Genetics, University of Illinois, College of Medicine, Chicago, IL, 60607, USA. .; Department of Pharmacology and Regenerative Medicine, University of Chicago, Chicago, IL, 60612, USA. .
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Źródło:
Scientific reports [Sci Rep] 2022 Oct 01; Vol. 12 (1), pp. 16488. Date of Electronic Publication: 2022 Oct 01.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Alzheimer Disease*/metabolism
Interferon Type I*/metabolism
Amyloid beta-Peptides/metabolism ; Animals ; Blood-Brain Barrier/metabolism ; Brain/metabolism ; Claudin-5/metabolism ; Disease Models, Animal ; Endothelial Cells/metabolism ; Endothelium/metabolism ; Female ; Humans ; Interferon-beta/metabolism ; Mice ; Mice, Transgenic ; Occludin/metabolism ; Plaque, Amyloid/pathology ; RNA/metabolism ; Receptor, Interferon alpha-beta/metabolism ; Tight Junction Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Virological, immunological and pathological findings of transplacentally transmitted bluetongue virus serotype 1 in IFNAR1-blocked mice during early and mid gestation.
Autorzy:
Saminathan M; Division of Pathology, ICAR-Indian Veterinary Research Institute (ICAR-IVRI), Izatnagar, Bareilly, 243122, Uttar Pradesh, India.
Singh KP; Centre for Animal Disease Research and Diagnosis (CADRAD), ICAR-IVRI, Izatnagar, Bareilly, 243122, Uttar Pradesh, India. .
Vineetha S; Division of Pathology, ICAR-Indian Veterinary Research Institute (ICAR-IVRI), Izatnagar, Bareilly, 243122, Uttar Pradesh, India.
Maity M; Division of Pathology, ICAR-Indian Veterinary Research Institute (ICAR-IVRI), Izatnagar, Bareilly, 243122, Uttar Pradesh, India.
Biswas SK; Division of Virology, ICAR-IVRI, Mukteswar Campus, Nainital, 263138, Uttarakhand, India.
Manjunathareddy GB; ICAR-National Institute of Veterinary Epidemiology and Disease Informatics, Bengaluru, 560064, Karnataka, India.
Chauhan HC; Department of Veterinary Microbiology, College of Veterinary Science and Animal Husbandry, Sardarkrushinagar Dantiwada Agricultural University, Sardarkrushinagar, 385506, Gujarat, India.
Milton AAP; Division of Animal Health, ICAR-RC for NEH Region, Umiam, Barapani, 793103, Meghalaya, India.
Ramakrishnan MA; Division of Virology, ICAR-IVRI, Mukteswar Campus, Nainital, 263138, Uttarakhand, India.
Maan S; College of Veterinary Sciences, LLR University of Veterinary and Animal Sciences, Hisar, 125 004, Haryana, India.
Maan NS; College of Veterinary Sciences, LLR University of Veterinary and Animal Sciences, Hisar, 125 004, Haryana, India.
Hemadri D; ICAR-National Institute of Veterinary Epidemiology and Disease Informatics, Bengaluru, 560064, Karnataka, India.
Chandel BS; Department of Veterinary Microbiology, College of Veterinary Science and Animal Husbandry, Sardarkrushinagar Dantiwada Agricultural University, Sardarkrushinagar, 385506, Gujarat, India.
Gupta VK; Centre for Animal Disease Research and Diagnosis (CADRAD), ICAR-IVRI, Izatnagar, Bareilly, 243122, Uttar Pradesh, India.
Mertens PPC; School of Veterinary Medicine and Science, University of Nottingham, Sutton Bonnington, Leicestershire, UK.
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Źródło:
Scientific reports [Sci Rep] 2020 Feb 07; Vol. 10 (1), pp. 2164. Date of Electronic Publication: 2020 Feb 07.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Infectious Disease Transmission, Vertical*
Bluetongue/*pathology
Fetal Diseases/*immunology
Fetal Diseases/*pathology
Pregnancy Complications, Infectious/*pathology
Receptor, Interferon alpha-beta/*deficiency
Animals ; Antigens, Viral/immunology ; Bluetongue/immunology ; Bluetongue/transmission ; Bluetongue/virology ; Bluetongue virus/immunology ; Bluetongue virus/pathogenicity ; Bone and Bones/abnormalities ; Brain/abnormalities ; Female ; Fetal Diseases/virology ; Mice ; Pregnancy ; Pregnancy Complications, Infectious/immunology ; Pregnancy Complications, Infectious/virology ; Receptor, Interferon alpha-beta/genetics ; Spleen/immunology ; T-Lymphocytes/immunology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
A novel Schmallenberg virus subunit vaccine candidate protects IFNAR mice against virulent SBV challenge.
Autorzy:
Boshra H; Center for Cooperative Research in Biosciences (CIC bioGUNE), Basque Research and Technology Alliance (BRTA), Bizkaia Technology Park, 48160, Derio, Spain. .; Department of Pathology, Fundamental and Applied Research for Animals & Health (FARAH), Faculty of Veterinary Medicine, University of Liège, Bât B43, avenue de Cureghem 6, 4000, Liège, Belgium. .
Lorenzo G; Animal Health Research Center (INIA-CISA), 28130, Valdeolmos, Madrid, Spain.
Charro D; Center for Cooperative Research in Biosciences (CIC bioGUNE), Basque Research and Technology Alliance (BRTA), Bizkaia Technology Park, 48160, Derio, Spain.
Moreno S; Animal Health Research Center (INIA-CISA), 28130, Valdeolmos, Madrid, Spain.
Guerra GS; Center for Cooperative Research in Biosciences (CIC bioGUNE), Basque Research and Technology Alliance (BRTA), Bizkaia Technology Park, 48160, Derio, Spain.
Sanchez I; Vacunek SL, Bizkaia Technology Park, 48160, Derio, Spain.
Garrido JM; Animal Health Department, NEIKER-Basque Institute for Agricultural Research and Development, Derio, Bizkaia, Spain.
Geijo M; Animal Health Department, NEIKER-Basque Institute for Agricultural Research and Development, Derio, Bizkaia, Spain.
Brun A; Animal Health Research Center (INIA-CISA), 28130, Valdeolmos, Madrid, Spain.
Abrescia NGA; Center for Cooperative Research in Biosciences (CIC bioGUNE), Basque Research and Technology Alliance (BRTA), Bizkaia Technology Park, 48160, Derio, Spain. .; Basque Foundation for Science, IKERBASQUE, 48013, Bilbao, Spain. .; Centro de Investigación Biomédica en Red de Enfermedades Hepáticas y Digestivas (CIBERehd), Instituto de Salud Carlos III, Madrid, Spain. .
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Źródło:
Scientific reports [Sci Rep] 2020 Nov 23; Vol. 10 (1), pp. 18725. Date of Electronic Publication: 2020 Nov 23.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Orthobunyavirus/*immunology
Orthobunyavirus/*pathogenicity
Viral Vaccines/*immunology
Adjuvants, Immunologic/administration & dosage ; Animals ; Antibodies, Viral/biosynthesis ; Antibody Specificity ; Broadly Neutralizing Antibodies/biosynthesis ; Bunyaviridae Infections/immunology ; Bunyaviridae Infections/prevention & control ; Bunyaviridae Infections/veterinary ; In Vitro Techniques ; Interferon-gamma/metabolism ; Lymphocyte Activation ; Mice ; Mice, Knockout ; Orthobunyavirus/genetics ; Receptor, Interferon alpha-beta/deficiency ; Receptor, Interferon alpha-beta/genetics ; Ruminants ; Saponins/administration & dosage ; Vaccines, Subunit/administration & dosage ; Vaccines, Subunit/genetics ; Vaccines, Subunit/immunology ; Viral Vaccines/administration & dosage ; Viral Vaccines/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
A functional polymorphism in IFNAR1 gene is associated with susceptibility and severity of HFMD with EV71 infection.
Autorzy:
Zou R; Department of Infectious Diseases, Shenzhen Third People's Hospital, University of South China, Shenzhen, 518112, China.; Guangdong Key Laboratory of Infectious Diseases, Shenzhen Third People's Hospital, Guangdong Medical College, Shenzhen, 518112, China.
Zhang G; Guangdong Key Laboratory of Infectious Diseases, Shenzhen Third People's Hospital, Guangdong Medical College, Shenzhen, 518112, China.; Shenzhen Key Laboratory of Pathogen and Immunity, Shenzhen Third People's Hospital, Shenzhen, 518112, China.
Li S; Guangdong Key Laboratory of Infectious Diseases, Shenzhen Third People's Hospital, Guangdong Medical College, Shenzhen, 518112, China.
Wang W; Guangdong Key Laboratory of Infectious Diseases, Shenzhen Third People's Hospital, Guangdong Medical College, Shenzhen, 518112, China.
Yuan J; Guangdong Key Laboratory of Infectious Diseases, Shenzhen Third People's Hospital, Guangdong Medical College, Shenzhen, 518112, China.
Li J; Guangdong Key Laboratory of Infectious Diseases, Shenzhen Third People's Hospital, Guangdong Medical College, Shenzhen, 518112, China.
Wang Y; Guangdong Key Laboratory of Infectious Diseases, Shenzhen Third People's Hospital, Guangdong Medical College, Shenzhen, 518112, China.
Lin Y; Guangdong Key Laboratory of Infectious Diseases, Shenzhen Third People's Hospital, Guangdong Medical College, Shenzhen, 518112, China.
Deng Y; Department of Infectious Diseases, Shenzhen Third People's Hospital, University of South China, Shenzhen, 518112, China.; Guangdong Key Laboratory of Infectious Diseases, Shenzhen Third People's Hospital, Guangdong Medical College, Shenzhen, 518112, China.
Zhou B; Guangdong Key Laboratory of Infectious Diseases, Shenzhen Third People's Hospital, Guangdong Medical College, Shenzhen, 518112, China.
Gao GF; Shenzhen Key Laboratory of Pathogen and Immunity, Shenzhen Third People's Hospital, Shenzhen, 518112, China.; Center for Molecular Virology, CAS Key Laboratory of Pathogenic Microbiology and Immunology, Institute of Microbiology, Chinese Academy of Sciences, 100101, Beijing.
Liu Y; Department of Infectious Diseases, Shenzhen Third People's Hospital, University of South China, Shenzhen, 518112, China.; Guangdong Key Laboratory of Infectious Diseases, Shenzhen Third People's Hospital, Guangdong Medical College, Shenzhen, 518112, China.; Shenzhen Key Laboratory of Pathogen and Immunity, Shenzhen Third People's Hospital, Shenzhen, 518112, China.
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Źródło:
Scientific reports [Sci Rep] 2015 Dec 18; Vol. 5, pp. 18541. Date of Electronic Publication: 2015 Dec 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Genetic Predisposition to Disease*
Polymorphism, Single Nucleotide*
Enterovirus A, Human/*physiology
Hand, Foot and Mouth Disease/*genetics
Hand, Foot and Mouth Disease/*pathology
Receptor, Interferon alpha-beta/*genetics
2',5'-Oligoadenylate Synthetase/genetics ; 2',5'-Oligoadenylate Synthetase/metabolism ; Alleles ; Child, Preschool ; China ; Female ; Genotype ; Hand, Foot and Mouth Disease/virology ; Humans ; Infant ; Leukocytes, Mononuclear/metabolism ; Leukocytes, Mononuclear/virology ; Male ; Myxovirus Resistance Proteins/genetics ; Myxovirus Resistance Proteins/metabolism ; Promoter Regions, Genetic ; Receptor, Interferon alpha-beta/metabolism ; Severity of Illness Index ; Transcriptome
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Zika virus infection causes temporary paralysis in adult mice with motor neuron synaptic retraction and evidence for proximal peripheral neuropathy.
Autorzy:
Morrey JD; Institute for Antiviral Research, Department of Animal, Dairy, and Veterinary Sciences, 5600 Old Main Hill, Utah State University, Logan, Utah, 84322-5600, United States of America. .
Oliveira ALR; Institute of Biology, University of Campinas, Campinas, SP, Brazil.
Wang H; Institute for Antiviral Research, Department of Animal, Dairy, and Veterinary Sciences, 5600 Old Main Hill, Utah State University, Logan, Utah, 84322-5600, United States of America.
Zukor K; Institute for Antiviral Research, Department of Animal, Dairy, and Veterinary Sciences, 5600 Old Main Hill, Utah State University, Logan, Utah, 84322-5600, United States of America.
de Castro MV; Institute of Biology, University of Campinas, Campinas, SP, Brazil.
Siddharthan V; Institute for Antiviral Research, Department of Animal, Dairy, and Veterinary Sciences, 5600 Old Main Hill, Utah State University, Logan, Utah, 84322-5600, United States of America.
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Źródło:
Scientific reports [Sci Rep] 2019 Dec 20; Vol. 9 (1), pp. 19531. Date of Electronic Publication: 2019 Dec 20.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Motor Neurons/*physiology
Paralysis/*etiology
Zika Virus/*pathogenicity
Zika Virus Infection/*complications
Animals ; Electrophysiology ; Female ; Guillain-Barre Syndrome/virology ; Male ; Mice ; Paralysis/virology ; Peripheral Nervous System Diseases/etiology ; Peripheral Nervous System Diseases/virology ; RNA, Viral/genetics ; Receptor, Interferon alpha-beta/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Self-DNA release and STING-dependent sensing drives inflammation to cigarette smoke in mice.
Autorzy:
Nascimento M; University of Orleans and CNRS, INEM-UMR7355, Orleans, France.
Gombault A; University of Orleans and CNRS, INEM-UMR7355, Orleans, France.
Lacerda-Queiroz N; University of Orleans and CNRS, INEM-UMR7355, Orleans, France.
Panek C; University of Orleans and CNRS, INEM-UMR7355, Orleans, France.
Savigny F; University of Orleans and CNRS, INEM-UMR7355, Orleans, France.
Sbeity M; University of Orleans and CNRS, INEM-UMR7355, Orleans, France.
Bourinet M; University of Orleans and CNRS, INEM-UMR7355, Orleans, France.
Le Bert M; University of Orleans and CNRS, INEM-UMR7355, Orleans, France.
Riteau N; University of Orleans and CNRS, INEM-UMR7355, Orleans, France.
Ryffel B; University of Orleans and CNRS, INEM-UMR7355, Orleans, France.
Quesniaux VFJ; University of Orleans and CNRS, INEM-UMR7355, Orleans, France.
Couillin I; University of Orleans and CNRS, INEM-UMR7355, Orleans, France. .
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Źródło:
Scientific reports [Sci Rep] 2019 Oct 16; Vol. 9 (1), pp. 14848. Date of Electronic Publication: 2019 Oct 16.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
DNA/*metabolism
Membrane Proteins/*metabolism
Nucleotidyltransferases/*metabolism
Pneumonia/*metabolism
Pulmonary Emphysema/*metabolism
Receptor, Interferon alpha-beta/*metabolism
Tobacco Smoke Pollution/*adverse effects
Animals ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Repetitive Sequences, Nucleic Acid
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
A VSV-based Zika virus vaccine protects mice from lethal challenge.
Autorzy:
Emanuel J; Laboratory of Virology, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, MT, USA.
Callison J; Laboratory of Virology, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, MT, USA.
Dowd KA; Laboratory of Viral Diseases, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, USA.
Pierson TC; Laboratory of Viral Diseases, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, USA.
Feldmann H; Laboratory of Virology, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, MT, USA.
Marzi A; Laboratory of Virology, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, MT, USA. .
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Źródło:
Scientific reports [Sci Rep] 2018 Jul 23; Vol. 8 (1), pp. 11043. Date of Electronic Publication: 2018 Jul 23.
Typ publikacji:
Journal Article; Research Support, N.I.H., Intramural
MeSH Terms:
Zika Virus/*immunology
Zika Virus/*pathogenicity
Animals ; Ebola Vaccines/therapeutic use ; Ebolavirus/immunology ; Ebolavirus/pathogenicity ; Female ; Glycoproteins/immunology ; Glycoproteins/metabolism ; Hemorrhagic Fever, Ebola/immunology ; Hemorrhagic Fever, Ebola/prevention & control ; Hemorrhagic Fever, Ebola/virology ; Male ; Mice ; Receptor, Interferon alpha-beta/genetics ; Receptor, Interferon alpha-beta/metabolism ; Vesiculovirus/immunology ; Vesiculovirus/pathogenicity ; Viral Vaccines/therapeutic use
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Efficacy of a T Cell-Biased Adenovirus Vector as a Zika Virus Vaccine.
Autorzy:
Bullard BL; School of Biological Sciences, Nebraska Center for Virology, University of Nebraska, Lincoln, USA.
Corder BN; School of Biological Sciences, Nebraska Center for Virology, University of Nebraska, Lincoln, USA.
Gorman MJ; Departments of Medicine, Molecular Microbiology, Pathology & Immunology, Washington University School of Medicine, Saint Louis, Missouri, 63110, USA.
Diamond MS; Departments of Medicine, Molecular Microbiology, Pathology & Immunology, Washington University School of Medicine, Saint Louis, Missouri, 63110, USA.
Weaver EA; School of Biological Sciences, Nebraska Center for Virology, University of Nebraska, Lincoln, USA. .
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Źródło:
Scientific reports [Sci Rep] 2018 Dec 20; Vol. 8 (1), pp. 18017. Date of Electronic Publication: 2018 Dec 20.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Adenoviridae/*immunology
Genetic Vectors/*immunology
T-Lymphocytes/*immunology
Zika Virus/*immunology
Zika Virus Infection/*prevention & control
Adenoviridae/genetics ; Animals ; Cells, Cultured ; Disease Models, Animal ; Female ; HEK293 Cells ; Humans ; Immunity, Cellular ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Receptor, Interferon alpha-beta/genetics ; T-Lymphocytes/metabolism ; Treatment Outcome ; Viral Vaccines/immunology ; Viral Vaccines/therapeutic use ; Zika Virus Infection/immunology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
IFNβ Protects Neurons from Damage in a Murine Model of HIV-1 Associated Brain Injury.
Autorzy:
Thaney VE; Infectious and Inflammatory Disease Center, Sanford Burnham Prebys Medical Discovery Institute, 10901 North Torrey Pines Road, La Jolla, CA 92037, USA.; Graduate School of Biomedical Sciences, Sanford-Burnham Prebys Medical Discovery Institute, 10901 North Torrey Pines Road, La Jolla, CA 92037, USA.
O'Neill AM; Infectious and Inflammatory Disease Center, Sanford Burnham Prebys Medical Discovery Institute, 10901 North Torrey Pines Road, La Jolla, CA 92037, USA.
Hoefer MM; Infectious and Inflammatory Disease Center, Sanford Burnham Prebys Medical Discovery Institute, 10901 North Torrey Pines Road, La Jolla, CA 92037, USA.
Maung R; Infectious and Inflammatory Disease Center, Sanford Burnham Prebys Medical Discovery Institute, 10901 North Torrey Pines Road, La Jolla, CA 92037, USA.
Sanchez AB; Infectious and Inflammatory Disease Center, Sanford Burnham Prebys Medical Discovery Institute, 10901 North Torrey Pines Road, La Jolla, CA 92037, USA.
Kaul M; Infectious and Inflammatory Disease Center, Sanford Burnham Prebys Medical Discovery Institute, 10901 North Torrey Pines Road, La Jolla, CA 92037, USA.; Department of Psychiatry, University of California, San Diego, 9500 Gilman Drive, San Diego, CA 92093, USA.
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Źródło:
Scientific reports [Sci Rep] 2017 Apr 20; Vol. 7, pp. 46514. Date of Electronic Publication: 2017 Apr 20.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Disease Models, Animal*
Brain Injuries/*prevention & control
Interferon-beta/*pharmacology
Neurons/*drug effects
Neuroprotective Agents/*pharmacology
Animals ; Brain Injuries/virology ; Cells, Cultured ; Chemokine CCL4/genetics ; Chemokine CCL4/metabolism ; Gene Expression/drug effects ; HIV Envelope Protein gp120/antagonists & inhibitors ; HIV Envelope Protein gp120/genetics ; HIV Envelope Protein gp120/metabolism ; HIV-1/physiology ; Humans ; Interferon-beta/genetics ; Interferon-beta/metabolism ; Mice, Transgenic ; Neurons/pathology ; Neurons/virology ; Rats ; Receptor, Interferon alpha-beta/genetics ; Receptor, Interferon alpha-beta/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Soybean-derived Bowman-Birk Inhibitor (BBI) Inhibits HIV Replication in Macrophages.
Autorzy:
Ma TC; Wuhan University School of Basic Medical Sciences, Wuhan, Hubei, 430071, P.R. China.
Zhou RH; Wuhan University School of Basic Medical Sciences, Wuhan, Hubei, 430071, P.R. China.
Wang X; Department of Pathology and Laboratory Medicine, Temple University Lewis Katz School of Medicine, Philadelphia, PA 19140, USA.
Li JL; Department of Pathology and Laboratory Medicine, Temple University Lewis Katz School of Medicine, Philadelphia, PA 19140, USA.
Sang M; Wuhan University School of Basic Medical Sciences, Wuhan, Hubei, 430071, P.R. China.
Zhou L; Wuhan University School of Basic Medical Sciences, Wuhan, Hubei, 430071, P.R. China.
Zhuang K; Wuhan University School of Basic Medical Sciences, Wuhan, Hubei, 430071, P.R. China.; State Key Laboratory of Virology, Wuhan University, Wuhan, Hubei, 430071, P.R. China.
Hou W; Wuhan University School of Basic Medical Sciences, Wuhan, Hubei, 430071, P.R. China.; State Key Laboratory of Virology, Wuhan University, Wuhan, Hubei, 430071, P.R. China.
Guo DY; Wuhan University School of Basic Medical Sciences, Wuhan, Hubei, 430071, P.R. China.; State Key Laboratory of Virology, Wuhan University, Wuhan, Hubei, 430071, P.R. China.
Ho WZ; Wuhan University School of Basic Medical Sciences, Wuhan, Hubei, 430071, P.R. China.; State Key Laboratory of Virology, Wuhan University, Wuhan, Hubei, 430071, P.R. China.; Department of Pathology and Laboratory Medicine, Temple University Lewis Katz School of Medicine, Philadelphia, PA 19140, USA.
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Źródło:
Scientific reports [Sci Rep] 2016 Oct 13; Vol. 6, pp. 34752. Date of Electronic Publication: 2016 Oct 13.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Anti-HIV Agents/*pharmacology
HIV-1/*drug effects
Host-Pathogen Interactions/*immunology
Interferon-beta/*antagonists & inhibitors
Macrophages/*immunology
Trypsin Inhibitor, Bowman-Birk Soybean/*pharmacology
Virus Replication/*drug effects
2',5'-Oligoadenylate Synthetase/genetics ; 2',5'-Oligoadenylate Synthetase/immunology ; APOBEC-3G Deaminase/genetics ; APOBEC-3G Deaminase/immunology ; Adaptor Proteins, Signal Transducing ; Antibodies, Neutralizing/pharmacology ; Cells, Cultured ; Cytokines/genetics ; Cytokines/immunology ; Cytosine Deaminase/genetics ; Cytosine Deaminase/immunology ; Gene Expression Regulation ; HIV-1/genetics ; HIV-1/growth & development ; Host-Pathogen Interactions/genetics ; Humans ; Interferon Regulatory Factor-3/genetics ; Interferon Regulatory Factor-3/immunology ; Interferon-beta/genetics ; Interferon-beta/immunology ; Macrophages/metabolism ; Macrophages/virology ; Myxovirus Resistance Proteins/genetics ; Myxovirus Resistance Proteins/immunology ; Oxidoreductases Acting on CH-CH Group Donors ; Proteins/genetics ; Proteins/immunology ; RNA-Binding Proteins ; Receptor, Interferon alpha-beta/antagonists & inhibitors ; Receptor, Interferon alpha-beta/genetics ; Receptor, Interferon alpha-beta/immunology ; Signal Transduction ; Transcription Factors/genetics ; Transcription Factors/immunology ; Ubiquitins/genetics ; Ubiquitins/immunology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Rotavirus acceleration of type 1 diabetes in non-obese diabetic mice depends on type I interferon signalling.
Autorzy:
Pane JA; Department of Microbiology and Immunology, The University of Melbourne at the Peter Doherty Institute for Infection and Immunity, Melbourne, Victoria, Australia.
Fleming FE; Department of Microbiology and Immunology, The University of Melbourne at the Peter Doherty Institute for Infection and Immunity, Melbourne, Victoria, Australia.
Graham KL; St Vincent's Institute, Fitzroy, Victoria, Australia.; Department of Medicine, The University of Melbourne, St. Vincent's Hospital, Fitzroy, Victoria, Australia.
Thomas HE; St Vincent's Institute, Fitzroy, Victoria, Australia.; Department of Medicine, The University of Melbourne, St. Vincent's Hospital, Fitzroy, Victoria, Australia.
Kay TW; St Vincent's Institute, Fitzroy, Victoria, Australia.; Department of Medicine, The University of Melbourne, St. Vincent's Hospital, Fitzroy, Victoria, Australia.
Coulson BS; Department of Microbiology and Immunology, The University of Melbourne at the Peter Doherty Institute for Infection and Immunity, Melbourne, Victoria, Australia.
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Źródło:
Scientific reports [Sci Rep] 2016 Jul 13; Vol. 6, pp. 29697. Date of Electronic Publication: 2016 Jul 13.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Diabetes Mellitus, Experimental/*immunology
Diabetes Mellitus, Type 1/*immunology
Interferon Type I/*immunology
Rotavirus/*immunology
Rotavirus Infections/*immunology
Signal Transduction/*immunology
Animals ; Diabetes Mellitus, Experimental/genetics ; Diabetes Mellitus, Experimental/pathology ; Diabetes Mellitus, Experimental/virology ; Diabetes Mellitus, Type 1/genetics ; Diabetes Mellitus, Type 1/pathology ; Diabetes Mellitus, Type 1/virology ; Interferon Type I/genetics ; Mice ; Mice, Inbred NOD ; Mice, Knockout ; Receptor, Interferon alpha-beta/genetics ; Receptor, Interferon alpha-beta/immunology ; Rotavirus Infections/genetics ; Rotavirus Infections/pathology ; Signal Transduction/genetics ; Th1 Cells/immunology ; Th1 Cells/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Tofla virus: A newly identified Nairovirus of the Crimean-Congo hemorrhagic fever group isolated from ticks in Japan.
Autorzy:
Shimada S; Department of Virology, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.; Leading graduate school program, Nagasaki University, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.
Aoki K; Department of Virology, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.
Nabeshima T; Department of Virology, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.
Fuxun Y; Department of Virology, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.
Kurosaki Y; Department of Emerging Infectious Diseases, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.
Shiogama K; Department of Pathology, Fujita Health University School of Medicine, 1-98 Dengakugakubo, Kutsukake-cho, Toyoake, Aichi, 470-1192, Japan.
Onouchi T; Department of Pathology, Fujita Health University School of Medicine, 1-98 Dengakugakubo, Kutsukake-cho, Toyoake, Aichi, 470-1192, Japan.
Sakaguchi M; Central Laboratory, Institute of Tropical Medicine, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.
Fuchigami T; Department of Hygienic Chemistry, Graduate School of Biomedical Sciences, Nagasaki University, 1-14 Bunkyo-machi, Nagasaki, 852-8521, Japan.
Ono H; Department of Hygienic Chemistry, Graduate School of Biomedical Sciences, Nagasaki University, 1-14 Bunkyo-machi, Nagasaki, 852-8521, Japan.
Nishi K; Department of Radioisotope Medicine, Atomic Bomb Diseases Institute, Nagasaki University, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.
Posadas-Herrera G; Department of Virology, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.
Uchida L; Department of Virology, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.
Takamatsu Y; Department of Virology, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.
Yasuda J; Department of Emerging Infectious Diseases, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.; Leading graduate school program, Nagasaki University, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.
Tsutsumi Y; Department of Pathology, Fujita Health University School of Medicine, 1-98 Dengakugakubo, Kutsukake-cho, Toyoake, Aichi, 470-1192, Japan.
Fujita H; Mahara Institute of Medical Acarology, 56-3 korekuni Aratano-cho, Anan, Tokushima 779-1510, Japan.
Morita K; Department of Virology, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.; Leading graduate school program, Nagasaki University, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.
Hayasaka D; Department of Virology, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.; Leading graduate school program, Nagasaki University, 1-12-4 Sakamoto, Nagasaki, 852-8523, Japan.
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Źródło:
Scientific reports [Sci Rep] 2016 Feb 11; Vol. 6, pp. 20213. Date of Electronic Publication: 2016 Feb 11.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Genome, Viral*
Phylogeny*
Arboviruses/*genetics
Bunyaviridae Infections/*virology
Nairovirus/*genetics
Ticks/*virology
Animals ; Arboviruses/classification ; Arboviruses/pathogenicity ; Bunyaviridae Infections/mortality ; Bunyaviridae Infections/pathology ; Cell Line, Tumor ; Chlorocebus aethiops ; Epidemiological Monitoring ; Fluorodeoxyglucose F18/metabolism ; Gastrointestinal Tract/pathology ; Gastrointestinal Tract/virology ; HEK293 Cells ; Humans ; Japan ; Mice ; Mice, Knockout ; Nairovirus/classification ; Nairovirus/pathogenicity ; Neuroglia/pathology ; Neuroglia/virology ; Neurons/pathology ; Neurons/virology ; Neutralization Tests ; Positron Emission Tomography Computed Tomography ; Receptor, Interferon alpha-beta/deficiency ; Receptor, Interferon alpha-beta/genetics ; Sequence Analysis, RNA ; Survival Analysis ; Vero Cells
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Discovery of an antibody for pan-ebolavirus therapy.
Autorzy:
Furuyama W; Division of Global Epidemiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.
Marzi A; Laboratory of Virology, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Rocky Mountain Laboratories, Hamilton, Montana, USA.
Nanbo A; Department of Cell Physiology, Graduate School of Medicine, Hokkaido University, Sapporo, Japan.
Haddock E; Laboratory of Virology, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Rocky Mountain Laboratories, Hamilton, Montana, USA.
Maruyama J; Division of Global Epidemiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.
Miyamoto H; Division of Global Epidemiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.
Igarashi M; Division of Global Epidemiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.
Yoshida R; Division of Global Epidemiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.
Noyori O; Division of Global Epidemiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.
Feldmann H; Laboratory of Virology, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Rocky Mountain Laboratories, Hamilton, Montana, USA.
Takada A; Division of Global Epidemiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.
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Źródło:
Scientific reports [Sci Rep] 2016 Feb 10; Vol. 6, pp. 20514. Date of Electronic Publication: 2016 Feb 10.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Antibodies, Monoclonal/*therapeutic use
Antibodies, Neutralizing/*therapeutic use
Hemorrhagic Fever, Ebola/*drug therapy
Amino Acid Sequence ; Animals ; Antibodies, Monoclonal/immunology ; Antibodies, Monoclonal/pharmacology ; Antibodies, Neutralizing/immunology ; Antibodies, Neutralizing/pharmacology ; Chlorocebus aethiops ; Disease Models, Animal ; Ebolavirus/metabolism ; Epitopes/chemistry ; Epitopes/immunology ; Female ; Hemorrhagic Fever, Ebola/mortality ; Hemorrhagic Fever, Ebola/veterinary ; Humans ; Mice ; Mice, Inbred BALB C ; Mice, Inbred C57BL ; Mice, Knockout ; Molecular Sequence Data ; Protein Structure, Tertiary ; Receptor, Interferon alpha-beta/deficiency ; Receptor, Interferon alpha-beta/genetics ; Sequence Alignment ; Survival Rate ; Vero Cells ; Virus Internalization/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Anti-tumorigenic effects of Type 1 interferon are subdued by integrated stress responses.
Autorzy:
Bhattacharya S; Department of Animal Biology and Mari Lowe Center for Comparative Oncology, School of Veterinary Medicine, University of Pennsylvania, Philadelphia, PA 19104-4539, USA.
HuangFu WC
Dong G
Qian J
Baker DP
Karar J
Koumenis C
Diehl JA
Fuchs SY
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Źródło:
Oncogene [Oncogene] 2013 Sep 05; Vol. 32 (36), pp. 4214-21. Date of Electronic Publication: 2012 Oct 08.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Stress, Physiological*
Interferon Type I/*metabolism
Animals ; Cell Line ; Cricetinae ; Eukaryotic Initiation Factor-2/metabolism ; Humans ; Mice ; Neoplasms/metabolism ; Phosphorylation ; Protein Serine-Threonine Kinases/metabolism ; Receptor, Interferon alpha-beta/metabolism ; Signal Transduction ; eIF-2 Kinase/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
2'-O methylation of the viral mRNA cap evades host restriction by IFIT family members.
Autorzy:
Daffis S; Department of Medicine, Washington University School of Medicine, St Louis, Missouri 63110, USA.
Szretter KJ
Schriewer J
Li J
Youn S
Errett J
Lin TY
Schneller S
Zust R
Dong H
Thiel V
Sen GC
Fensterl V
Klimstra WB
Pierson TC
Buller RM
Gale M Jr
Shi PY
Diamond MS
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Źródło:
Nature [Nature] 2010 Nov 18; Vol. 468 (7322), pp. 452-6.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Carrier Proteins/*metabolism
Gene Expression Regulation/*immunology
Immunity, Innate/*immunology
Interferons/*immunology
Proteins/*metabolism
RNA Caps/*metabolism
RNA, Viral/*metabolism
3T3 Cells ; Adaptor Proteins, Signal Transducing ; Animals ; Apoptosis Regulatory Proteins ; Carrier Proteins/genetics ; Cells, Cultured ; Coronavirus/enzymology ; Coronavirus/genetics ; Coronavirus/immunology ; Coronavirus/physiology ; Fibroblasts ; Gene Expression Regulation/genetics ; Humans ; Immunity, Innate/genetics ; Interferons/deficiency ; Interferons/genetics ; Methylation ; Methyltransferases/metabolism ; Mice ; Mice, Inbred C57BL ; Models, Genetic ; Models, Immunological ; Neoplasm Proteins/genetics ; Neoplasm Proteins/metabolism ; Poxviridae/enzymology ; Poxviridae/genetics ; Poxviridae/immunology ; Poxviridae/physiology ; Protein Biosynthesis/immunology ; Proteins/genetics ; RNA Caps/genetics ; RNA Caps/immunology ; RNA, Viral/genetics ; RNA, Viral/immunology ; RNA-Binding Proteins ; Receptor, Interferon alpha-beta/deficiency ; Receptor, Interferon alpha-beta/genetics ; Survival Rate ; Virus Replication ; West Nile virus/enzymology ; West Nile virus/genetics ; West Nile virus/immunology ; West Nile virus/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
IFNalpha activates dormant haematopoietic stem cells in vivo.
Autorzy:
Essers MA; Division of Stem Cells and Cancer, Deutsches Krebsforschungszentrum (DKFZ), DKFZ-ZMBH Alliance, Im Neuenheimer Feld 280, D-69120 Heidelberg, Germany.
Offner S
Blanco-Bose WE
Waibler Z
Kalinke U
Duchosal MA
Trumpp A
Pokaż więcej
Źródło:
Nature [Nature] 2009 Apr 16; Vol. 458 (7240), pp. 904-8. Date of Electronic Publication: 2009 Feb 11.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Cycle/*drug effects
Hematopoietic Stem Cells/*cytology
Hematopoietic Stem Cells/*drug effects
Interferon-alpha/*pharmacology
Animals ; Antigens, Ly/metabolism ; Cell Count ; Cell Proliferation/drug effects ; Fluorouracil/pharmacology ; Membrane Proteins/deficiency ; Membrane Proteins/metabolism ; Mice ; Mice, Inbred C57BL ; Phosphorylation/drug effects ; Receptor, Interferon alpha-beta/deficiency ; Receptor, Interferon alpha-beta/metabolism ; STAT1 Transcription Factor/deficiency ; STAT1 Transcription Factor/metabolism ; Signal Transduction/drug effects ; Up-Regulation/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Subcapsular sinus macrophages prevent CNS invasion on peripheral infection with a neurotropic virus.
Autorzy:
Iannacone M; Immune Disease Institute and Department of Pathology, Harvard Medical School, 77 Avenue Louis Pasteur, Boston, Massachusetts 02115, USA. Matteo_ />Moseman EA
Tonti E
Bosurgi L
Junt T
Henrickson SE
Whelan SP
Guidotti LG
von Andrian UH
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Źródło:
Nature [Nature] 2010 Jun 24; Vol. 465 (7301), pp. 1079-83.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Central Nervous System/*immunology
Central Nervous System/*virology
Lymph Nodes/*immunology
Lymph Nodes/*virology
Macrophages/*immunology
Rhabdoviridae Infections/*immunology
Vesiculovirus/*immunology
Animals ; Central Nervous System/cytology ; Dendritic Cells/immunology ; Injections ; Interferon Type I/immunology ; Lymph Nodes/cytology ; Lymph Nodes/innervation ; Macrophages/virology ; Mice ; Mice, Inbred BALB C ; Mice, Inbred C57BL ; Paralysis/complications ; Paralysis/virology ; Peripheral Nerves/virology ; Receptor, Interferon alpha-beta/deficiency ; Rhabdoviridae Infections/complications ; Rhabdoviridae Infections/virology ; Survival Rate ; Vesicular stomatitis Indiana virus/immunology ; Vesicular stomatitis Indiana virus/pathogenicity ; Vesicular stomatitis Indiana virus/physiology ; Vesicular stomatitis New Jersey virus/immunology ; Vesicular stomatitis New Jersey virus/pathogenicity ; Vesicular stomatitis New Jersey virus/physiology ; Vesiculovirus/pathogenicity ; Vesiculovirus/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
CD69 acts downstream of interferon-alpha/beta to inhibit S1P1 and lymphocyte egress from lymphoid organs.
Autorzy:
Shiow LR; Howard Hughes Medical Institute, University of California, San Francisco, San Francisco, California 94143, USA.
Rosen DB
Brdicková N
Xu Y
An J
Lanier LL
Cyster JG
Matloubian M
Pokaż więcej
Źródło:
Nature [Nature] 2006 Mar 23; Vol. 440 (7083), pp. 540-4. Date of Electronic Publication: 2006 Mar 08.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Antigens, CD/*physiology
Antigens, Differentiation, T-Lymphocyte/*physiology
Lymphocytes/*physiology
Receptors, Lysosphingolipid/*physiology
Animals ; Cell Line ; Cell Movement ; Down-Regulation ; Humans ; Interferon-alpha/physiology ; Interferon-beta/physiology ; Jurkat Cells ; Lectins, C-Type ; Lymph/cytology ; Lymphocyte Count ; Lymphoid Tissue/cytology ; Membrane Proteins/metabolism ; Mice ; Mice, Inbred C57BL ; Poly I-C/pharmacology ; Receptor, Interferon alpha-beta ; Receptors, Interferon/metabolism ; Receptors, Lysosphingolipid/antagonists & inhibitors ; Signal Transduction ; Transfection
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Wyświetlanie 1-20 z 24

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