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Wyszukujesz frazę ""Receptors, IgE"" wg kryterium: Temat


Tytuł:
IRF3 Activation in Mast Cells Promotes FcεRI-Mediated Allergic Inflammation.
Autorzy:
Choi YA; Cell & Matrix Research Institute, Department of Pharmacology, School of Medicine, Kyungpook National University, Daegu 41944, Republic of Korea.
Dhakal H; Cell & Matrix Research Institute, Department of Pharmacology, School of Medicine, Kyungpook National University, Daegu 41944, Republic of Korea.
Lee S; Immunoregulatory Materials Research Center, Korea Research Institute of Bioscience and Biotechnology, Jeongeup 56212, Republic of Korea.
Kim N; Cell & Matrix Research Institute, Department of Pharmacology, School of Medicine, Kyungpook National University, Daegu 41944, Republic of Korea.
Lee B; Department of Biochemistry and Cell Biology, School of Medicine, Kyungpook National University, Daegu 41944, Republic of Korea.
Kwon TK; Department of Immunology, School of Medicine, Keimyung University, Daegu 42601, Republic of Korea.
Khang D; Department of Physiology, School of Medicine, Gachon University, Incheon 21999, Republic of Korea.
Kim SH; Cell & Matrix Research Institute, Department of Pharmacology, School of Medicine, Kyungpook National University, Daegu 41944, Republic of Korea.
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Źródło:
Cells [Cells] 2023 May 28; Vol. 12 (11). Date of Electronic Publication: 2023 May 28.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Receptors, IgE*/metabolism
Anaphylaxis*
Rats ; Mice ; Animals ; Mast Cells/metabolism ; Cell Line ; Inflammation/metabolism
Czasopismo naukowe
Tytuł:
Kaempferol Suppresses the Activation of Mast Cells by Modulating the Expression of FcεRI and SHIP1.
Autorzy:
Nagata K; Department of Biological Science and Technology, Faculty of Advanced Engineering, Tokyo University of Science, 6-3-1 Niijuku, Katsushika-ku, Tokyo 125-8585, Japan.
Araumi S; Department of Biological Science and Technology, Faculty of Advanced Engineering, Tokyo University of Science, 6-3-1 Niijuku, Katsushika-ku, Tokyo 125-8585, Japan.
Ando D; Department of Biological Science and Technology, Faculty of Advanced Engineering, Tokyo University of Science, 6-3-1 Niijuku, Katsushika-ku, Tokyo 125-8585, Japan.
Ito N; Department of Biological Science and Technology, Faculty of Advanced Engineering, Tokyo University of Science, 6-3-1 Niijuku, Katsushika-ku, Tokyo 125-8585, Japan.
Ando M; Department of Biological Science and Technology, Faculty of Advanced Engineering, Tokyo University of Science, 6-3-1 Niijuku, Katsushika-ku, Tokyo 125-8585, Japan.
Ikeda Y; Department of Biological Science and Technology, Faculty of Advanced Engineering, Tokyo University of Science, 6-3-1 Niijuku, Katsushika-ku, Tokyo 125-8585, Japan.
Takahashi M; Department of Biological Science and Technology, Faculty of Advanced Engineering, Tokyo University of Science, 6-3-1 Niijuku, Katsushika-ku, Tokyo 125-8585, Japan.
Noguchi S; Department of Biological Science and Technology, Faculty of Advanced Engineering, Tokyo University of Science, 6-3-1 Niijuku, Katsushika-ku, Tokyo 125-8585, Japan.
Yasuda Y; Department of Biological Science and Technology, Faculty of Advanced Engineering, Tokyo University of Science, 6-3-1 Niijuku, Katsushika-ku, Tokyo 125-8585, Japan.
Nakano N; Atopy (Allergy) Research Center, Juntendo University Graduate School of Medicine, 2-1-1 Hongo Bunkyo-ku, Tokyo 113-8421, Japan.
Ando T; Atopy (Allergy) Research Center, Juntendo University Graduate School of Medicine, 2-1-1 Hongo Bunkyo-ku, Tokyo 113-8421, Japan.
Hara M; Atopy (Allergy) Research Center, Juntendo University Graduate School of Medicine, 2-1-1 Hongo Bunkyo-ku, Tokyo 113-8421, Japan.
Yashiro T; Department of Biological Science and Technology, Faculty of Advanced Engineering, Tokyo University of Science, 6-3-1 Niijuku, Katsushika-ku, Tokyo 125-8585, Japan.
Hachisu M; Department of Biological Science and Technology, Faculty of Advanced Engineering, Tokyo University of Science, 6-3-1 Niijuku, Katsushika-ku, Tokyo 125-8585, Japan.
Nishiyama C; Department of Biological Science and Technology, Faculty of Advanced Engineering, Tokyo University of Science, 6-3-1 Niijuku, Katsushika-ku, Tokyo 125-8585, Japan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Mar 22; Vol. 24 (6). Date of Electronic Publication: 2023 Mar 22.
Typ publikacji:
Journal Article
MeSH Terms:
Mast Cells*/metabolism
Receptors, IgE*/genetics
Receptors, IgE*/metabolism
Cell Degranulation ; Immunoglobulin E/metabolism ; Interleukin-1 Receptor-Like 1 Protein/metabolism ; Kaempferols/pharmacology ; Kaempferols/metabolism ; NF-E2-Related Factor 2/genetics ; NF-E2-Related Factor 2/metabolism ; RNA, Messenger/metabolism ; Toll-Like Receptor 4/genetics ; Toll-Like Receptor 4/metabolism
Czasopismo naukowe
Tytuł:
Signals from the TAFA4-PTEN-PU.1 axis alleviate nasal allergy by modulating the expression of FcεRI in mast cells.
Autorzy:
Zhou C; Longgang Traditional Chinese Medicine Hospital and Beijing University of Chinese Medicine Shenzhen Hospital, Shenzhen, China.
Li M; Longgang Traditional Chinese Medicine Hospital and Beijing University of Chinese Medicine Shenzhen Hospital, Shenzhen, China.
Liu Y; Department of Respirology & Allergy, Third Affiliated Hospital of Shenzhen University, Shenzhen, China.
Wang X; Guangdong Provincial Regional Disease Key Laboratory, Shenzhen, China.; Institute of Allergy & Immunology of Shenzhen University, State Key Laboratory of Respiratory Diseases Allergy Division at Shenzhen University, Shenzhen, China.
Zhang S; Guangdong Provincial Regional Disease Key Laboratory, Shenzhen, China.; Institute of Allergy & Immunology of Shenzhen University, State Key Laboratory of Respiratory Diseases Allergy Division at Shenzhen University, Shenzhen, China.
Guan L; Department of Respirology & Allergy, Third Affiliated Hospital of Shenzhen University, Shenzhen, China.
Hong J; Guangdong Provincial Regional Disease Key Laboratory, Shenzhen, China.; Institute of Allergy & Immunology of Shenzhen University, State Key Laboratory of Respiratory Diseases Allergy Division at Shenzhen University, Shenzhen, China.
Zhou W; Guangdong Provincial Regional Disease Key Laboratory, Shenzhen, China.; Institute of Allergy & Immunology of Shenzhen University, State Key Laboratory of Respiratory Diseases Allergy Division at Shenzhen University, Shenzhen, China.
Wu G; Department of Respirology & Allergy, Third Affiliated Hospital of Shenzhen University, Shenzhen, China.
Diao W; Longgang Traditional Chinese Medicine Hospital and Beijing University of Chinese Medicine Shenzhen Hospital, Shenzhen, China.
Huang Q; Department of Respirology & Allergy, Third Affiliated Hospital of Shenzhen University, Shenzhen, China.
Yang P; Guangdong Provincial Regional Disease Key Laboratory, Shenzhen, China.; Institute of Allergy & Immunology of Shenzhen University, State Key Laboratory of Respiratory Diseases Allergy Division at Shenzhen University, Shenzhen, China.
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Źródło:
Clinical and experimental immunology [Clin Exp Immunol] 2023 Mar 08; Vol. 211 (1), pp. 15-22.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Receptors, IgE*
Rhinitis, Allergic*
Animals ; Humans ; Mice ; Antigens ; Cytokines/metabolism ; Immunoglobulin E ; Mast Cells
Czasopismo naukowe
Tytuł:
Fc Fragment of IgE Receptor Ig (FCER1G) acts as a key gene involved in cancer immune infiltration and tumour microenvironment.
Autorzy:
Yang R; Department of Urology and Guangdong Key Laboratory of Urology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou Medical University, Guangzhou, China.
Chen Z; Department of Urology and Guangdong Key Laboratory of Urology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou Medical University, Guangzhou, China.
Liang L; Department of Urology and Guangdong Key Laboratory of Urology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou Medical University, Guangzhou, China.
Ao S; Department of Urology and Guangdong Key Laboratory of Urology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou Medical University, Guangzhou, China.
Zhang J; Department of Urology and Guangdong Key Laboratory of Urology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou Medical University, Guangzhou, China.
Chang Z; Department of Urology and Guangdong Key Laboratory of Urology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou Medical University, Guangzhou, China.
Wang Z; Department of Urology and Guangdong Key Laboratory of Urology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou Medical University, Guangzhou, China.
Zhou Y; Department of Urology and Guangdong Key Laboratory of Urology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou Medical University, Guangzhou, China.
Duan X; Department of Urology and Guangdong Key Laboratory of Urology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou Medical University, Guangzhou, China.
Deng T; Department of Urology and Guangdong Key Laboratory of Urology, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou Medical University, Guangzhou, China.
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Źródło:
Immunology [Immunology] 2023 Feb; Vol. 168 (2), pp. 302-319. Date of Electronic Publication: 2022 Aug 22.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Receptors, IgE*
Neoplasms*/genetics
Humans ; Immunoglobulin Fc Fragments ; Tumor Microenvironment/genetics ; Carcinogenesis ; Prognosis ; Biomarkers, Tumor
Czasopismo naukowe
Tytuł:
Quercetin and Resveratrol Differentially Decrease Expression of the High-Affinity IgE Receptor (FcεRI) by Human and Mouse Mast Cells.
Autorzy:
Alam SB; Nanotechnology Research Center, National Research Council, Edmonton, AB T6G 2M9, Canada.
Wagner A; Nanotechnology Research Center, National Research Council, Edmonton, AB T6G 2M9, Canada.
Willows S; Nanotechnology Research Center, National Research Council, Edmonton, AB T6G 2M9, Canada.
Kulka M; Nanotechnology Research Center, National Research Council, Edmonton, AB T6G 2M9, Canada.; Medical Microbiology and Immunology, University of Alberta, Edmonton, AB T6G 2E1, Canada.
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2022 Oct 08; Vol. 27 (19). Date of Electronic Publication: 2022 Oct 08.
Typ publikacji:
Journal Article
MeSH Terms:
Mast Cells*/metabolism
Receptors, IgE*/metabolism
Animals ; Antigens/metabolism ; Cell Degranulation ; Congenital Disorders of Glycosylation ; Humans ; Mice ; Mitogen-Activated Protein Kinase 3/metabolism ; Quercetin/metabolism ; Quercetin/pharmacology ; Resveratrol/metabolism ; Resveratrol/pharmacology ; Syk Kinase/metabolism
SCR Disease Name:
Congenital disorder of glycosylation, type 2C
Czasopismo naukowe
Tytuł:
Cyclic Hypoxia Induces Transcriptomic Changes in Mast Cells Leading to a Hyperresponsive Phenotype after FcεRI Cross-Linking.
Autorzy:
Segura-Villalobos D; Departamento de Farmacobiología, Centro de Investigación y de Estudios Avanzados (Cinvestav), Unidad Sede Sur. Calzada de los Tenorios No. 235, Col. Granjas Coapa, Tlalpan, Mexico City 14330, Mexico.
Lamas M; Departamento de Farmacobiología, Centro de Investigación y de Estudios Avanzados (Cinvestav), Unidad Sede Sur. Calzada de los Tenorios No. 235, Col. Granjas Coapa, Tlalpan, Mexico City 14330, Mexico.
González-Espinosa C; Departamento de Farmacobiología, Centro de Investigación y de Estudios Avanzados (Cinvestav), Unidad Sede Sur. Calzada de los Tenorios No. 235, Col. Granjas Coapa, Tlalpan, Mexico City 14330, Mexico.
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Źródło:
Cells [Cells] 2022 Jul 19; Vol. 11 (14). Date of Electronic Publication: 2022 Jul 19.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Mast Cells*/metabolism
Receptors, IgE*/genetics
Receptors, IgE*/metabolism
Animals ; Hypoxia/genetics ; Hypoxia/metabolism ; Mice ; Phenotype ; Transcriptome/genetics
Czasopismo naukowe
Tytuł:
A novel test for type-I allergy based on crosslink formation of immunoglobulin-E receptors by allergen-specific immunoglobulin-E antibodies and an allergen.
Autorzy:
Koga Y; Department of Pharmaceutical Services, Graduate School of Biomedical and Health Sciences, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima, 734-8553, Japan.
Ishii S; Department of Pharmaceutical Services, Graduate School of Biomedical and Health Sciences, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima, 734-8553, Japan.
Yokooji T; Department of Pharmaceutical Services, Graduate School of Biomedical and Health Sciences, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima, 734-8553, Japan.; Department of Frontier Science for Pharmacotherapy, Graduate School of Biomedical and Health Sciences, Hiroshima University, Hiroshima, Japan.
Yamamoto K; Department of Frontier Science for Pharmacotherapy, Graduate School of Biomedical and Health Sciences, Hiroshima University, Hiroshima, Japan.
Ogino R; Department of Frontier Science for Pharmacotherapy, Graduate School of Biomedical and Health Sciences, Hiroshima University, Hiroshima, Japan.
Taogoshi T; Department of Pharmaceutical Services, Graduate School of Biomedical and Health Sciences, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima, 734-8553, Japan.
Matsuo H; Department of Pharmaceutical Services, Graduate School of Biomedical and Health Sciences, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima, 734-8553, Japan. .
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Źródło:
Scientific reports [Sci Rep] 2023 Nov 11; Vol. 13 (1), pp. 19676. Date of Electronic Publication: 2023 Nov 11.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Hypersensitivity, Immediate*
Hypersensitivity*
Humans ; Allergens ; Receptors, IgE ; Immunoglobulin E
Czasopismo naukowe
Tytuł:
Regulation of Trafficking and Signaling of the High Affinity IgE Receptor by FcεRIβ and the Potential Impact of FcεRIβ Splicing in Allergic Inflammation.
Autorzy:
Arthur GK; Department of Population Health and Pathobiology, College of Veterinary Medicine, NC State University, Raleigh, NC 27607, USA.
Cruse G; Department of Molecular Biomedical Sciences, College of Veterinary Medicine, NC State University, Raleigh, NC 27607, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Jan 12; Vol. 23 (2). Date of Electronic Publication: 2022 Jan 12.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Gene Expression Regulation*
RNA Splicing*
Signal Transduction*
Hypersensitivity/*etiology
Hypersensitivity/*metabolism
Receptors, IgE/*genetics
Receptors, IgE/*metabolism
Alternative Splicing ; Animals ; Biomarkers ; Cell Degranulation/genetics ; Cell Degranulation/immunology ; Disease Susceptibility ; Humans ; Hypersensitivity/diagnosis ; Hypersensitivity/therapy ; Mast Cells/immunology ; Mast Cells/metabolism ; Receptors, IgE/chemistry ; Structure-Activity Relationship
Czasopismo naukowe
Tytuł:
Fc Epsilon RI-Neuroimmune Interplay in Pruritus Triggered by Particulate Matter in Atopic Dermatitis Patients.
Autorzy:
Isaifan D; Department of Biological and Environmental Sciences, College of Arts and Sciences, Qatar University, Doha P.O. Box 2713, Qatar.
Crovella S; Laboratory of Animal Research Center (LARC), Qatar University, Doha P.O. Box 2713, Qatar.
Soubra L; Department of Biological and Environmental Sciences, College of Arts and Sciences, Qatar University, Doha P.O. Box 2713, Qatar.
Al-Nesf M; Allergy and Immunology Division, Hamad Medical Corporation, Doha P.O. Box 3050, Qatar.
Steinhoff M; Department of Dermatology & Venereology, Weill Cornell Medicine, New York, NY 10065, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Jul 24; Vol. 24 (14). Date of Electronic Publication: 2023 Jul 24.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Dermatitis, Atopic*/metabolism
Air Pollutants*/adverse effects
Humans ; Receptors, IgE/metabolism ; Particulate Matter/adverse effects ; Pruritus/metabolism
Czasopismo naukowe
Tytuł:
Plant-Derived Molecule 4-Methylumbelliferone Suppresses FcεRI-Mediated Mast Cell Activation and Allergic Inflammation.
Autorzy:
Wang HN; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Health Science Center, Shenzhen University, Shenzhen 518060, China.
Xiang QA; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Health Science Center, Shenzhen University, Shenzhen 518060, China.
Lin HH; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Health Science Center, Shenzhen University, Shenzhen 518060, China.
Chen JN; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Health Science Center, Shenzhen University, Shenzhen 518060, China.
Guo WJ; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Health Science Center, Shenzhen University, Shenzhen 518060, China.
Guo WM; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Health Science Center, Shenzhen University, Shenzhen 518060, China.
Yue XN; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Health Science Center, Shenzhen University, Shenzhen 518060, China.
Zhao ZF; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Health Science Center, Shenzhen University, Shenzhen 518060, China.
Ji K; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Health Science Center, Shenzhen University, Shenzhen 518060, China.
Chen JJ; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Health Science Center, Shenzhen University, Shenzhen 518060, China.
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2022 Feb 27; Vol. 27 (5). Date of Electronic Publication: 2022 Feb 27.
Typ publikacji:
Journal Article
MeSH Terms:
Receptors, IgE*
Czasopismo naukowe
Tytuł:
FcεRI: A Master Regulator of Mast Cell Functions.
Autorzy:
Nagata Y; Faculty of Pharmaceutical Science, Institute of Medical, Pharmaceutical and Health Sciences, Kanazawa University, Kakuma-machi, Kanazawa 920-1192, Japan.
Suzuki R; Faculty of Pharmaceutical Science, Institute of Medical, Pharmaceutical and Health Sciences, Kanazawa University, Kakuma-machi, Kanazawa 920-1192, Japan.
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Źródło:
Cells [Cells] 2022 Feb 11; Vol. 11 (4). Date of Electronic Publication: 2022 Feb 11.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Hypersensitivity*
Receptors, IgE*/metabolism
Desensitization, Immunologic ; Humans ; Immunoglobulin E/metabolism ; Mast Cells/metabolism
Czasopismo naukowe
Tytuł:
ORMDL3 overexpression facilitates FcεRI-mediated transcription of proinflammatory cytokines and thapsigargin-mediated PERK phosphorylation in RBL-2H3 cells.
Autorzy:
Ogi K; Division of Otorhinolaryngology Head and Neck Surgery, Department of Sensory and Locomotor Medicine, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.
Takabayashi T; Division of Otorhinolaryngology Head and Neck Surgery, Department of Sensory and Locomotor Medicine, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.
Tomita K; Division of Otorhinolaryngology Head and Neck Surgery, Department of Sensory and Locomotor Medicine, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.
Sakashita M; Division of Otorhinolaryngology Head and Neck Surgery, Department of Sensory and Locomotor Medicine, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.
Morikawa T; Division of Otorhinolaryngology Head and Neck Surgery, Department of Sensory and Locomotor Medicine, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.
Ninomiya T; Division of Otorhinolaryngology Head and Neck Surgery, Department of Sensory and Locomotor Medicine, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.
Okamoto M; Division of Otorhinolaryngology Head and Neck Surgery, Department of Sensory and Locomotor Medicine, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.
Narita N; Division of Otorhinolaryngology Head and Neck Surgery, Department of Sensory and Locomotor Medicine, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.
Fujieda S; Division of Otorhinolaryngology Head and Neck Surgery, Department of Sensory and Locomotor Medicine, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.
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Źródło:
Immunity, inflammation and disease [Immun Inflamm Dis] 2021 Dec; Vol. 9 (4), pp. 1394-1405. Date of Electronic Publication: 2021 Jul 19.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cytokines*/metabolism
Receptors, IgE*/genetics
Cell Degranulation ; Endoplasmic Reticulum/metabolism ; Humans ; Membrane Proteins/genetics ; Membrane Proteins/metabolism ; Orosomucoid ; Phosphorylation ; Thapsigargin/pharmacology
Czasopismo naukowe
Tytuł:
Basophil Characteristics as a Marker of the Pathogenesis of Chronic Spontaneous Urticaria in Relation to the Coagulation and Complement Systems.
Autorzy:
Yanase Y; Department of Pharmacotherapy, Institute of Biomedical and Health Sciences, Hiroshima University, Hiroshima 739-0046, Japan.
Matsubara D; Department of Dermatology, Institute of Biomedical and Health Sciences, Hiroshima University, Hiroshima 739-0046, Japan.
Takahagi S; Department of Dermatology, Institute of Biomedical and Health Sciences, Hiroshima University, Hiroshima 739-0046, Japan.
Tanaka A; Department of Dermatology, Institute of Biomedical and Health Sciences, Hiroshima University, Hiroshima 739-0046, Japan.
Ozawa K; Department of Pharmacotherapy, Institute of Biomedical and Health Sciences, Hiroshima University, Hiroshima 739-0046, Japan.
Hide M; Department of Dermatology, Institute of Biomedical and Health Sciences, Hiroshima University, Hiroshima 739-0046, Japan.; Hiroshima City Hiroshima Citizens Hospital, Hiroshima 730-8518, Japan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Jun 19; Vol. 24 (12). Date of Electronic Publication: 2023 Jun 19.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Chronic Urticaria*
Urticaria*/etiology
Humans ; Basophils ; Skin ; Receptors, IgE ; Complement System Proteins ; Chronic Disease
Czasopismo naukowe
Tytuł:
Degranulation of Mast Cells as a Target for Drug Development.
Autorzy:
Yang BG; Research Institute, GI Biome Inc., Seongnam 13201, Republic of Korea.
Kim AR; Research Institute, GI Biome Inc., Seongnam 13201, Republic of Korea.
Lee D; Research Institute, GI Biome Inc., Seongnam 13201, Republic of Korea.
An SB; Research Institute, GI Biome Inc., Seongnam 13201, Republic of Korea.
Shim YA; Research Institute, GI Innovation Inc., Songpa-gu, Seoul 05855, Republic of Korea.
Jang MH; Research Institute, GI Innovation Inc., Songpa-gu, Seoul 05855, Republic of Korea.
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Źródło:
Cells [Cells] 2023 May 29; Vol. 12 (11). Date of Electronic Publication: 2023 May 29.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Mast Cells*/metabolism
Hypersensitivity*/metabolism
Humans ; Receptors, IgE/metabolism ; Drug Development ; Nerve Tissue Proteins/metabolism ; Receptors, Neuropeptide/metabolism ; Receptors, G-Protein-Coupled/metabolism
Czasopismo naukowe
Tytuł:
Interleukin-3-dependent potentiation of IgE responsiveness in mouse basophils.
Autorzy:
Kitano T; Department of Molecular and Cellular Health Sciences, Graduate School of Pharmaceutical Sciences, Nagoya City University, Nagoya, Japan.
Togawa K; Department of Molecular and Cellular Health Sciences, Graduate School of Pharmaceutical Sciences, Nagoya City University, Nagoya, Japan.
Takemori J; Department of Molecular and Cellular Health Sciences, Graduate School of Pharmaceutical Sciences, Nagoya City University, Nagoya, Japan.
Motoki Y; Department of Molecular and Cellular Health Sciences, Graduate School of Pharmaceutical Sciences, Nagoya City University, Nagoya, Japan.
Kishida K; Department of Molecular and Cellular Health Sciences, Graduate School of Pharmaceutical Sciences, Nagoya City University, Nagoya, Japan.
Itoh S; Department of Molecular and Cellular Health Sciences, Graduate School of Pharmaceutical Sciences, Nagoya City University, Nagoya, Japan.
Takamoto M; Department of Infection and Host Defense, Shinshu University School of Medicine, Matsumoto, Japan.
Taki S; Department of Molecular and Cellular Immunology, Shinshu University School of Medicine, Matsumoto, Japan.
Hida S; Department of Molecular and Cellular Health Sciences, Graduate School of Pharmaceutical Sciences, Nagoya City University, Nagoya, Japan.
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Źródło:
Genes to cells : devoted to molecular & cellular mechanisms [Genes Cells] 2023 Mar; Vol. 28 (3), pp. 226-236. Date of Electronic Publication: 2023 Jan 27.
Typ publikacji:
Journal Article
MeSH Terms:
Interleukin-3*/metabolism
Interleukin-3*/pharmacology
Basophils*/metabolism
Mice ; Animals ; Interleukin-4/metabolism ; Receptors, IgE/metabolism ; Immunoglobulin E/metabolism
Czasopismo naukowe
Tytuł:
Thalidomide Attenuates Mast Cell Activation by Upregulating SHP-1 Signaling and Interfering with the Action of CRBN.
Autorzy:
Chang HW; College of Pharmacy, Yeungnam University, Gyeongsan 38541, Republic of Korea.
Sim KH; Department of Pharmacology, School of Medicine, Daegu Catholic University, Daegu 42472, Republic of Korea.
Lee YJ; Department of Pharmacology, School of Medicine, Daegu Catholic University, Daegu 42472, Republic of Korea.
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Źródło:
Cells [Cells] 2023 Feb 01; Vol. 12 (3). Date of Electronic Publication: 2023 Feb 01.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
AMP-Activated Protein Kinases*/metabolism
Hypersensitivity*/metabolism
Animals ; Mice ; Adaptor Proteins, Signal Transducing/metabolism ; Mast Cells/metabolism ; Receptors, IgE/metabolism ; Signal Transduction ; Thalidomide/pharmacology ; Thalidomide/therapeutic use
Czasopismo naukowe
Tytuł:
FcεRI Signaling in the Modulation of Allergic Response: Role of Mast Cell-Derived Exosomes.
Autorzy:
Lecce M; Department of Molecular Medicine, 'Sapienza' University of Rome, Laboratory Affiliated to Istituto Pasteur Italia-Fondazione Cenci Bolognetti, Viale Regina Elena 291, 00161 Rome, Italy.
Molfetta R; Department of Molecular Medicine, 'Sapienza' University of Rome, Laboratory Affiliated to Istituto Pasteur Italia-Fondazione Cenci Bolognetti, Viale Regina Elena 291, 00161 Rome, Italy.
Milito ND; Department of Molecular Medicine, 'Sapienza' University of Rome, Laboratory Affiliated to Istituto Pasteur Italia-Fondazione Cenci Bolognetti, Viale Regina Elena 291, 00161 Rome, Italy.
Santoni A; Department of Molecular Medicine, 'Sapienza' University of Rome, Laboratory Affiliated to Istituto Pasteur Italia-Fondazione Cenci Bolognetti, Viale Regina Elena 291, 00161 Rome, Italy.; Istituto di Ricovero e Cura a Carattere Scientifico IRCCS Neuromed, 86077 Pozzilli, IS, Italy.
Paolini R; Department of Molecular Medicine, 'Sapienza' University of Rome, Laboratory Affiliated to Istituto Pasteur Italia-Fondazione Cenci Bolognetti, Viale Regina Elena 291, 00161 Rome, Italy.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Jul 30; Vol. 21 (15). Date of Electronic Publication: 2020 Jul 30.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Hypersensitivity/*genetics
Mast Cells/*immunology
Receptors, IgE/*genetics
Animals ; Antigens/genetics ; Antigens/immunology ; Cell Degranulation/genetics ; Exosomes/genetics ; Exosomes/immunology ; Humans ; Hypersensitivity/immunology ; Hypersensitivity/pathology ; Immunoglobulin E/genetics ; Immunoglobulin E/immunology ; Mast Cells/pathology ; Receptors, IgE/immunology ; Signal Transduction/genetics
Czasopismo naukowe
Tytuł:
Arf1 facilitates mast cell proliferation via the mTORC1 pathway.
Autorzy:
Kotani Y; Department of Cell Signaling, Institute of Biomedical Science, Kansai Medical University, Hirakata, Osaka, 573-1010, Japan.; Department of Biological Science, Graduate School of Humanities and Sciences, Nara Women's University, Nara, 630-8506, Japan.
Sumiyoshi M; Department of Cell Signaling, Institute of Biomedical Science, Kansai Medical University, Hirakata, Osaka, 573-1010, Japan.
Sasada M; Department of Biological Science, Graduate School of Humanities and Sciences, Nara Women's University, Nara, 630-8506, Japan.
Watanabe T; Department of Biological Science, Graduate School of Humanities and Sciences, Nara Women's University, Nara, 630-8506, Japan. .
Matsuda S; Department of Cell Signaling, Institute of Biomedical Science, Kansai Medical University, Hirakata, Osaka, 573-1010, Japan. .
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Źródło:
Scientific reports [Sci Rep] 2022 Dec 24; Vol. 12 (1), pp. 22297. Date of Electronic Publication: 2022 Dec 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Degranulation*/genetics
ADP-Ribosylation Factor 1*/genetics
ADP-Ribosylation Factor 1*/metabolism
Mechanistic Target of Rapamycin Complex 1/metabolism ; Receptors, IgE/metabolism ; Cytokines/metabolism ; Cell Proliferation ; Mast Cells/metabolism
Czasopismo naukowe
Tytuł:
Identification of CD203c as a New Basophil-Specific Flow-Marker in Ph Chronic Myeloid Leukemia.
Autorzy:
Sadovnik I; Department of Internal Medicine I, Division of Hematology and Hemostaseology, Medical University of Vienna, 1090 Vienna, Austria.; Ludwig Boltzmann Institute for Hematology and Oncology, Medical University of Vienna, 1090 Vienna, Austria.
Ivanov D; Department of Internal Medicine I, Division of Hematology and Hemostaseology, Medical University of Vienna, 1090 Vienna, Austria.
Smiljkovic D; Department of Internal Medicine I, Division of Hematology and Hemostaseology, Medical University of Vienna, 1090 Vienna, Austria.; Ludwig Boltzmann Institute for Hematology and Oncology, Medical University of Vienna, 1090 Vienna, Austria.
Stefanzl G; Department of Internal Medicine I, Division of Hematology and Hemostaseology, Medical University of Vienna, 1090 Vienna, Austria.; Ludwig Boltzmann Institute for Hematology and Oncology, Medical University of Vienna, 1090 Vienna, Austria.
Degenfeld-Schonburg L; Department of Internal Medicine I, Division of Hematology and Hemostaseology, Medical University of Vienna, 1090 Vienna, Austria.
Herndlhofer S; Department of Internal Medicine I, Division of Hematology and Hemostaseology, Medical University of Vienna, 1090 Vienna, Austria.; Ludwig Boltzmann Institute for Hematology and Oncology, Medical University of Vienna, 1090 Vienna, Austria.
Eisenwort G; Department of Internal Medicine I, Division of Hematology and Hemostaseology, Medical University of Vienna, 1090 Vienna, Austria.; Ludwig Boltzmann Institute for Hematology and Oncology, Medical University of Vienna, 1090 Vienna, Austria.; Third Medical Department for Hematology and Oncology, Hanusch Hospital Vienna, 1140 Vienna, Austria.
Hauswirth AW; Department of Internal Medicine I, Division of Hematology and Hemostaseology, Medical University of Vienna, 1090 Vienna, Austria.; Ludwig Boltzmann Institute for Hematology and Oncology, Medical University of Vienna, 1090 Vienna, Austria.
Sliwa T; Third Medical Department for Hematology and Oncology, Hanusch Hospital Vienna, 1140 Vienna, Austria.
Keil F; Ludwig Boltzmann Institute for Hematology and Oncology, Medical University of Vienna, 1090 Vienna, Austria.; Third Medical Department for Hematology and Oncology, Hanusch Hospital Vienna, 1140 Vienna, Austria.
Sperr WR; Department of Internal Medicine I, Division of Hematology and Hemostaseology, Medical University of Vienna, 1090 Vienna, Austria.; Ludwig Boltzmann Institute for Hematology and Oncology, Medical University of Vienna, 1090 Vienna, Austria.
Valent P; Department of Internal Medicine I, Division of Hematology and Hemostaseology, Medical University of Vienna, 1090 Vienna, Austria.; Ludwig Boltzmann Institute for Hematology and Oncology, Medical University of Vienna, 1090 Vienna, Austria.
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Źródło:
Cells [Cells] 2022 Dec 20; Vol. 12 (1). Date of Electronic Publication: 2022 Dec 20.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Basophils*
Leukemia, Myelogenous, Chronic, BCR-ABL Positive*/metabolism
Humans ; Chronic Disease ; Receptors, IgE/metabolism ; Up-Regulation
Czasopismo naukowe
Tytuł:
The Crosstalk between FcεRI and Sphingosine Signaling in Allergic Inflammation.
Autorzy:
Jo H; Department of Biochemistry, College of Natural Sciences, Kangwon National University, Chuncheon 24341, Korea.
Shim K; Department of Biochemistry, College of Natural Sciences, Kangwon National University, Chuncheon 24341, Korea.
Jeoung D; Department of Biochemistry, College of Natural Sciences, Kangwon National University, Chuncheon 24341, Korea.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Nov 11; Vol. 23 (22). Date of Electronic Publication: 2022 Nov 11.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Anaphylaxis*
Asthma*
Humans ; Sphingosine/metabolism ; Receptors, IgE/metabolism ; Inflammation
Czasopismo naukowe

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