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Tytuł:
Altered Circulating Follicular T Helper Cell Subsets and Follicular T Regulatory Cells Are Indicators of a Derailed B Cell Response in Lupus, Which Could Be Modified by Targeting IL-21R.
Autorzy:
Szabó K; Division of Clinical Immunology, Institute of Internal Medicine, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
Jámbor I; Division of Clinical Immunology, Institute of Internal Medicine, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
Pázmándi K; Department of Immunology, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
Nagy N; Division of Clinical Immunology, Institute of Internal Medicine, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
Papp G; Division of Clinical Immunology, Institute of Internal Medicine, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
Tarr T; Division of Clinical Immunology, Institute of Internal Medicine, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Oct 13; Vol. 23 (20). Date of Electronic Publication: 2022 Oct 13.
Typ publikacji:
Journal Article
MeSH Terms:
Receptors, Interleukin-21*/metabolism
Lupus Erythematosus, Systemic*
Humans ; T-Lymphocytes, Helper-Inducer ; T-Lymphocytes, Regulatory ; Autoantibodies/metabolism ; Antibodies, Monoclonal/metabolism ; Complement C4/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interleukin 21 Receptor/Ligand Interaction Is Linked to Disease Progression in Pancreatic Cancer.
Autorzy:
Linnebacher A; Institute of Pathology, University Hospital Heidelberg, 69120 Heidelberg, Germany.
Mayer P; Clinic for Diagnostic and Interventional Radiology, University Hospital Heidelberg, 69120 Heidelberg, Germany.
Marnet N; Institute of Pathology, University Hospital Heidelberg, 69120 Heidelberg, Germany.
Bergmann F; Institute of Pathology, University Hospital Heidelberg, 69120 Heidelberg, Germany.; Clinical Pathology, Klinikum Darmstadt GmbH, 64283 Darmstadt, Germany.
Herpel E; Institute of Pathology, University Hospital Heidelberg, 69120 Heidelberg, Germany.
Revia S; Helmholtz-University Group 'Cell Plasticity and Epigenetic Remodeling', German Cancer Research Center (DKFZ) and Institute of Pathology, University Hospital, 69120 Heidelberg, Germany.
Yin L; Molecular OncoSurgery Group, Department of General, Visceral, and Transplantation Surgery, University of Heidelberg and German Cancer Research Center, 69120 Heidelberg, Germany.
Liu L; Molecular OncoSurgery Group, Department of General, Visceral, and Transplantation Surgery, University of Heidelberg and German Cancer Research Center, 69120 Heidelberg, Germany.
Hackert T; Department of General, Visceral, and Transplantation Surgery, University Hospital Heidelberg, 69120 Heidelberg, Germany.
Giese T; Institute of Immunology, University Hospital Heidelberg, 69120 Heidelberg, Germany.
Herr I; Molecular OncoSurgery Group, Department of General, Visceral, and Transplantation Surgery, University of Heidelberg and German Cancer Research Center, 69120 Heidelberg, Germany.
Gaida MM; Institute of Pathology, University Hospital Heidelberg, 69120 Heidelberg, Germany. .; Institute of Pathology, University Medical Center Mainz, 55131 Mainz, Germany. .
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Źródło:
Cells [Cells] 2019 Sep 18; Vol. 8 (9). Date of Electronic Publication: 2019 Sep 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Disease Progression*
Carcinoma, Pancreatic Ductal/*metabolism
Pancreatic Neoplasms/*metabolism
Receptors, Interleukin-21/*metabolism
Animals ; Carcinoma, Pancreatic Ductal/pathology ; Cell Proliferation ; Chickens ; Humans ; Interleukins/metabolism ; Ligands ; Pancreatic Neoplasms/pathology ; Receptors, Interleukin-21/genetics ; Tumor Cells, Cultured
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Expression of Interleukin-21 and Interleukin-21 receptor in lymphocytes derived from tumor-draining lymph nodes of breast cancer.
Autorzy:
Balouchi-Anaraki S; Shiraz Institute for Cancer Research, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.
Mohammadsadeghi S; Shiraz Institute for Cancer Research, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.
Norouzian M; Shiraz Institute for Cancer Research, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.; Department of Laboratory Sciences, School of Allied Medical Sciences, Hormozgan University of Medical Sciences, Bandar Abbas, Iran.
Rasolmali R; Department of Pathology, Shiraz Central Hospital, Shiraz, Iran.
Talei AR; Breast Diseases Research Center, Shiraz University of Medical Sciences, Shiraz, Iran.
Mehdipour F; Shiraz Institute for Cancer Research, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.
Ghaderi A; Shiraz Institute for Cancer Research, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.; Department of Immunology, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.
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Źródło:
Breast disease [Breast Dis] 2022; Vol. 41 (1), pp. 373-382.
Typ publikacji:
Journal Article
MeSH Terms:
Breast Neoplasms*/pathology
Interleukin-21 Receptor alpha Subunit/*immunology
CD4-Positive T-Lymphocytes ; Female ; Humans ; Interleukins ; Lymph Nodes/pathology ; Receptors, Interleukin-21/genetics ; Receptors, Interleukin-21/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interleukin-21 receptor signalling is not critically required for imiquimod-induced psoriasiform dermatitis in mice.
Autorzy:
Kim HJ; Department of dermatology, Gachon University Gil Medical Center, Incheon, Korea.
Kim SH; Department of dermatology, Severance hospital, Cutaneous Biology Research Institute and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul, Korea.
Kim TG; Department of dermatology, Severance hospital, Cutaneous Biology Research Institute and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul, Korea.
Park JY; Department of dermatology, Severance hospital, Cutaneous Biology Research Institute and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul, Korea.
Lee M; Department of dermatology, Severance hospital, Cutaneous Biology Research Institute and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul, Korea.
Kim DS; Department of dermatology, Severance hospital, Cutaneous Biology Research Institute and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul, Korea.
Lee MG; Department of dermatology, Severance hospital, Cutaneous Biology Research Institute and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul, Korea.
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Źródło:
Experimental dermatology [Exp Dermatol] 2018 Feb; Vol. 27 (2), pp. 191-195. Date of Electronic Publication: 2018 Jan 05.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Dermatitis/*genetics
Interleukin-21 Receptor alpha Subunit/*metabolism
Psoriasis/*genetics
Receptors, Interleukin-21/*metabolism
Animals ; CD4-Positive T-Lymphocytes/cytology ; Dermatitis/metabolism ; Imiquimod ; Inflammation ; Interferon Inducers/pharmacology ; Interleukin-21 Receptor alpha Subunit/genetics ; Interleukin-23 Subunit p19/metabolism ; Intraepithelial Lymphocytes/cytology ; Mice ; Mice, Knockout ; Mice, Transgenic ; Psoriasis/chemically induced ; Psoriasis/metabolism ; Receptors, Interleukin-21/genetics ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
MicroRNA-30b Is Both Necessary and Sufficient for Interleukin-21 Receptor-Mediated Angiogenesis in Experimental Peripheral Arterial Disease.
Autorzy:
Wang T; State Key Laboratory of Respiratory Diseases, Guangzhou Institute of Respiratory Health, Guangzhou Medical University, Guangzhou 510120, China.; Robert M Berne Cardiovascular Research Center, University of Virginia, Charlottesville, VA 22908, USA.
Yang L; Robert M Berne Cardiovascular Research Center, University of Virginia, Charlottesville, VA 22908, USA.; Department of Pharmacology, Nankai University, Tianjing 300071, China.
Yuan M; Robert M Berne Cardiovascular Research Center, University of Virginia, Charlottesville, VA 22908, USA.; Department of Cardiology, Renmin Hospital of Wuhan University, Wuhan 430070, China.
Farber CR; Center for Public Health Genomics, University of Virginia, Charlottesville, VA 22908, USA.
Spolski R; Laboratory of Molecular Immunology and the Immunology Center, National Heart Lung and Blood Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Leonard WJ; Laboratory of Molecular Immunology and the Immunology Center, National Heart Lung and Blood Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Ganta VC; Robert M Berne Cardiovascular Research Center, University of Virginia, Charlottesville, VA 22908, USA.; Vascular Biology Center and Department of Medicine, Augusta University, Augusta, GA 30912, USA.
Annex BH; Robert M Berne Cardiovascular Research Center, University of Virginia, Charlottesville, VA 22908, USA.; Vascular Biology Center and Department of Medicine, Augusta University, Augusta, GA 30912, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Dec 27; Vol. 23 (1). Date of Electronic Publication: 2021 Dec 27.
Typ publikacji:
Journal Article
MeSH Terms:
MicroRNAs/*metabolism
Neovascularization, Physiologic/*genetics
Peripheral Arterial Disease/*genetics
Receptors, Interleukin-21/*metabolism
Animals ; Cell Survival/genetics ; Hindlimb/pathology ; Human Umbilical Vein Endothelial Cells/metabolism ; Humans ; Ischemia/genetics ; Ischemia/pathology ; Mice, Transgenic ; MicroRNAs/genetics ; Models, Biological ; Multigene Family ; Myoglobin/metabolism ; Perfusion ; Peripheral Arterial Disease/pathology ; Phosphorylation ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; STAT3 Transcription Factor/metabolism ; Suppressor of Cytokine Signaling 3 Protein/metabolism ; Up-Regulation/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Combined immunodeficiency with marginal zone lymphoma due to a novel homozygous mutation in IL-21R gene and successful treatment with hematopoietic stem cell transplantation.
Autorzy:
Edeer Karaca N; Faculty of Medicine, Department of Pediatric Immunology and Allergy, Ege University, İzmir, Turkey.
Özek G; Faculty of Medicine, Department of Pediatric Hematology and Oncology, Ege University, İzmir, Turkey.
Ataseven E; Faculty of Medicine, Department of Pediatric Hematology and Oncology, Ege University, İzmir, Turkey.
Tökmeci N; Faculty of Medicine, Department of Pediatric Immunology and Allergy, Ege University, İzmir, Turkey.
Şenol HD; Faculty of Medicine, Department of Pediatric Immunology and Allergy, Ege University, İzmir, Turkey.
Kıran E; Faculty of Medicine, Department of Pediatric Gastroenterology Gastroenterology, Hepatology and Nutrition, Ege University, İzmir, Turkey.
Aykut A; Faculty of Medicine, Department of Medical Genetics, Ege University, İzmir, Turkey.
Durmaz A; Faculty of Medicine, Department of Medical Genetics, Ege University, İzmir, Turkey.
Aksu G; Faculty of Medicine, Department of Pediatric Immunology and Allergy, Ege University, İzmir, Turkey.
Aksoylar S; Faculty of Medicine, Department of Pediatric Hematology and Oncology, Ege University, İzmir, Turkey.
Aydoğdu S; Faculty of Medicine, Department of Pediatric Gastroenterology Gastroenterology, Hepatology and Nutrition, Ege University, İzmir, Turkey.
Çetingül N; Faculty of Medicine, Department of Pediatric Hematology and Oncology, Ege University, İzmir, Turkey.
Kütükçüler N; Faculty of Medicine, Department of Pediatric Immunology and Allergy, Ege University, İzmir, Turkey.
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Źródło:
Pediatric hematology and oncology [Pediatr Hematol Oncol] 2021 Nov; Vol. 38 (8), pp. 745-752. Date of Electronic Publication: 2021 May 08.
Typ publikacji:
Case Reports; Journal Article
MeSH Terms:
Hematopoietic Stem Cell Transplantation*
Lymphoma*/genetics
Lymphoma*/therapy
Primary Immunodeficiency Diseases*/complications
Primary Immunodeficiency Diseases*/genetics
Primary Immunodeficiency Diseases*/therapy
Child ; Cryptosporidiosis ; Cytomegalovirus Infections ; Diarrhea/etiology ; Diarrhea/therapy ; Exanthema/etiology ; Exanthema/therapy ; Female ; Humans ; Mutation ; Persistent Infection ; Pneumonia, Viral ; Receptors, Interleukin-21/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
IL-21 and IL-21 receptor in the immunopathogenesis of multiple sclerosis.
Autorzy:
Ghalamfarsa G; a Cellular and Molecular Research Center, Yasuj University of Medical Sciences , Yasuj , Iran ;
Mahmoudi M; b Immunology Research Center, Department of Immunology and Allergy , School of Medicine, Mashhad University of Medical Sciences , Mashhad , Iran ;
Mohammadnia-Afrouzi M; c Department of Immunology and Microbiology , School of Medicine, Babol University of Medical Sciences , Babol , Iran ;
Yazdani Y; d Infectious Diseases Research Center and Laboratory Science Research Center, Golestan University of Medical Sciences , Gorgan , Iran ;
Anvari E; e Department of Physiology , Faculty of Medicine, Ilam University of Medical Sciences , Ilam , Iran ;
Hadinia A; a Cellular and Molecular Research Center, Yasuj University of Medical Sciences , Yasuj , Iran ;
Ghanbari A; a Cellular and Molecular Research Center, Yasuj University of Medical Sciences , Yasuj , Iran ;
Setayesh M; f Biology Department , School of Sciences, Shiraz University , Shiraz , Iran ;
Yousefi M; g Immunology Research Center, Tabriz University of Medical Sciences , Tabriz , Iran ;; h Department of Immunology , Faculty of Medicine, Tabriz University of Medical Sciences , Tabriz , Iran ;
Jadidi-Niaragh F; i Department of Immunology , School of Public Health, Tehran University of Medical Sciences , Tehran , Iran.
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Źródło:
Journal of immunotoxicology [J Immunotoxicol] 2016 May; Vol. 13 (3), pp. 274-85. Date of Electronic Publication: 2015 Oct 28.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Encephalomyelitis, Autoimmune, Experimental/*immunology
Interleukins/*metabolism
Lymphocyte Subsets/*immunology
Multiple Sclerosis/*immunology
Receptors, Interleukin-21/*metabolism
Animals ; Autoimmunity ; Cell Differentiation ; Disease Models, Animal ; Humans ; Interleukins/immunology ; Lymphocyte Activation ; Mice ; Receptors, Interleukin-21/immunology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
A conserved sugar bridge connected to the WSXWS motif has an important role for transport of IL-21R to the plasma membrane.
Autorzy:
Siupka P; Department of Molecular Biology and Genetics, University of Aarhus, Aarhus, Denmark.
Hamming OT; Department of Molecular Biology and Genetics, University of Aarhus, Aarhus, Denmark.
Kang L; Novo Nordisk R&D Center China, Beijing, China.
Gad HH; Department of Molecular Biology and Genetics, University of Aarhus, Aarhus, Denmark.
Hartmann R; Department of Molecular Biology and Genetics, University of Aarhus, Aarhus, Denmark.
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Źródło:
Genes and immunity [Genes Immun] 2015 Sep; Vol. 16 (6), pp. 405-13. Date of Electronic Publication: 2015 Jun 04.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Receptors, Interleukin-21/*metabolism
Amino Acid Motifs ; Cell Membrane/metabolism ; Crystallography, X-Ray ; Fibronectins/chemistry ; Fibronectins/metabolism ; HEK293 Cells ; Humans ; Interleukins/chemistry ; Interleukins/metabolism ; Models, Molecular ; Protein Structure, Tertiary ; Protein Transport ; Receptors, Interleukin-21/chemistry ; Structure-Activity Relationship ; Subcellular Fractions/metabolism ; Tryptophan/chemistry ; Tryptophan/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Targeting Myeloid-Derived Suppressor Cells Is a Novel Strategy for Anti-Psoriasis Therapy.
Autorzy:
Chen C; Department of Dermatology, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Key Laboratory of Skin Cancer and Psoriasis, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Engineering Research Centre of Skin Health and Disease, Xiangya Hospital, Central South University, Changsha, Hunan, China.; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, Hunan 410008, China.
Tan L; Department of Dermatology, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Key Laboratory of Skin Cancer and Psoriasis, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Engineering Research Centre of Skin Health and Disease, Xiangya Hospital, Central South University, Changsha, Hunan, China.; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, Hunan 410008, China.
Zhu W; Department of Dermatology, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Key Laboratory of Skin Cancer and Psoriasis, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Engineering Research Centre of Skin Health and Disease, Xiangya Hospital, Central South University, Changsha, Hunan, China.; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, Hunan 410008, China.
Lei L; Department of Dermatology, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Key Laboratory of Skin Cancer and Psoriasis, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Engineering Research Centre of Skin Health and Disease, Xiangya Hospital, Central South University, Changsha, Hunan, China.; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, Hunan 410008, China.
Kuang Y; Department of Dermatology, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Key Laboratory of Skin Cancer and Psoriasis, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Engineering Research Centre of Skin Health and Disease, Xiangya Hospital, Central South University, Changsha, Hunan, China.; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, Hunan 410008, China.
Liu P; Department of Dermatology, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Key Laboratory of Skin Cancer and Psoriasis, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Engineering Research Centre of Skin Health and Disease, Xiangya Hospital, Central South University, Changsha, Hunan, China.; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, Hunan 410008, China.
Li J; Department of Dermatology, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Key Laboratory of Skin Cancer and Psoriasis, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Engineering Research Centre of Skin Health and Disease, Xiangya Hospital, Central South University, Changsha, Hunan, China.; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, Hunan 410008, China.
Chen X; Department of Dermatology, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Key Laboratory of Skin Cancer and Psoriasis, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Engineering Research Centre of Skin Health and Disease, Xiangya Hospital, Central South University, Changsha, Hunan, China.; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, Hunan 410008, China.
Peng C; Department of Dermatology, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Key Laboratory of Skin Cancer and Psoriasis, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Engineering Research Centre of Skin Health and Disease, Xiangya Hospital, Central South University, Changsha, Hunan, China.; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, Hunan 410008, China.
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Źródło:
Mediators of inflammation [Mediators Inflamm] 2020 Jun 28; Vol. 2020, pp. 8567320. Date of Electronic Publication: 2020 Jun 28 (Print Publication: 2020).
Typ publikacji:
Journal Article
MeSH Terms:
Myeloid-Derived Suppressor Cells/*drug effects
Myeloid-Derived Suppressor Cells/*metabolism
Psoriasis/*drug therapy
Psoriasis/*metabolism
Adult ; Animals ; CD4-Positive T-Lymphocytes/metabolism ; Cell Differentiation/drug effects ; Cells, Cultured ; Deoxycytidine/analogs & derivatives ; Deoxycytidine/pharmacology ; Female ; Flow Cytometry ; Humans ; Male ; Membrane Proteins/metabolism ; Mice ; Mice, Inbred BALB C ; Middle Aged ; Psoriasis/immunology ; Receptors, Interleukin-21/metabolism ; T-Lymphocytes, Regulatory/drug effects ; T-Lymphocytes, Regulatory/metabolism ; Th17 Cells/drug effects ; Th17 Cells/metabolism ; Young Adult ; Gemcitabine
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interleukin 21 (IL-21) regulates chronic allograft vasculopathy (CAV) in murine heart allograft rejection.
Autorzy:
Khattar M; Department of Medical Microbiology and Immunology, The University of Toledo College of Medicine and Life Sciences, Toledo, OH, United States of America.
Baum CE; Department of Medical Microbiology and Immunology, The University of Toledo College of Medicine and Life Sciences, Toledo, OH, United States of America.
Schroder P; Department of Surgery, Duke University Medical Center, Durham, NC, United States of America.
Breidenbach JD; Department of Medical Microbiology and Immunology, The University of Toledo College of Medicine and Life Sciences, Toledo, OH, United States of America.
Haller ST; Department of Medicine, The University of Toledo College of Medicine and Life Sciences, Toledo, OH, United States of America.
Chen W; Department of Medical Microbiology and Immunology, The University of Toledo College of Medicine and Life Sciences, Toledo, OH, United States of America.
Stepkowski S; Department of Medical Microbiology and Immunology, The University of Toledo College of Medicine and Life Sciences, Toledo, OH, United States of America.
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Źródło:
PloS one [PLoS One] 2019 Nov 22; Vol. 14 (11), pp. e0225624. Date of Electronic Publication: 2019 Nov 22 (Print Publication: 2019).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Graft Rejection/*etiology
Heart Transplantation/*adverse effects
Interleukins/*genetics
Transplantation, Homologous/*adverse effects
Vascular Diseases/*etiology
Animals ; Basic-Leucine Zipper Transcription Factors/deficiency ; Basic-Leucine Zipper Transcription Factors/genetics ; Graft Rejection/immunology ; Histocompatibility Antigens Class II/genetics ; Histocompatibility Antigens Class II/immunology ; Interferon-gamma/metabolism ; Interleukins/deficiency ; Mice ; Mice, Inbred BALB C ; Mice, Inbred C57BL ; Mice, Knockout ; Receptors, Interleukin-21/genetics ; Recombinant Fusion Proteins/administration & dosage ; Recombinant Fusion Proteins/biosynthesis ; Skin Transplantation/adverse effects ; T-Lymphocytes/cytology ; T-Lymphocytes/immunology ; T-Lymphocytes/metabolism ; Vascular Diseases/prevention & control
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interleukin-21 enhances Toll-like receptor 2/4-mediated cytokine production via phosphorylation in the STAT3, Akt and p38 MAPK signalling pathways in human monocytic THP-1 cells.
Autorzy:
Jian L; Department of Rheumatology and Immunology, Peking University Third Hospital, Beijing, China.
Sun L; Department of Rheumatology and Immunology, Peking University Third Hospital, Beijing, China.
Li C; Department of Rheumatology and Immunology, Peking University Third Hospital, Beijing, China.
Yu R; Department of Rheumatology and Immunology, Peking University Third Hospital, Beijing, China.
Ma Z; Department of Rheumatology and Immunology, Peking University Third Hospital, Beijing, China.
Wang X; Department of Rheumatology and Immunology, Peking University Third Hospital, Beijing, China.
Zhao J; Department of Rheumatology and Immunology, Peking University Third Hospital, Beijing, China.
Liu X; Department of Rheumatology and Immunology, Peking University Third Hospital, Beijing, China.
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Źródło:
Scandinavian journal of immunology [Scand J Immunol] 2019 Jun; Vol. 89 (6), pp. e12761. Date of Electronic Publication: 2019 Apr 11.
Typ publikacji:
Journal Article
MeSH Terms:
Cytokines/*biosynthesis
Interleukins/*immunology
Monocytes/*metabolism
Toll-Like Receptor 2/*metabolism
Toll-Like Receptor 4/*metabolism
Cell Line, Tumor ; Humans ; Phosphorylation ; Proto-Oncogene Proteins c-akt/metabolism ; Receptors, Interleukin-21/biosynthesis ; STAT3 Transcription Factor/metabolism ; Signal Transduction ; THP-1 Cells ; p38 Mitogen-Activated Protein Kinases/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interleukin-21 receptor signalling is important for innate immune protection against HSV-2 infections.
Autorzy:
Kratholm SK; Department of Biomedicine, Aarhus University, Aarhus, Denmark.
Iversen MB; Department of Biomedicine, Aarhus University, Aarhus, Denmark.
Reinert L; Department of Biomedicine, Aarhus University, Aarhus, Denmark.
Jensen SK; Department of Biomedicine, Aarhus University, Aarhus, Denmark.
Hokland M; Department of Biomedicine, Aarhus University, Aarhus, Denmark.
Andersen T; Department of Biomedicine, Aarhus University, Aarhus, Denmark.
Rankin A; Immunology and Autoimmunity, Pfizer Inc. Cambridge, Massachusetts, United States of America.
Young D; Immunology and Autoimmunity, Pfizer Inc. Cambridge, Massachusetts, United States of America.
Frische S; The Water and Salt Research Centre, Institute of Anatomy, Aarhus University, Aarhus, Denmark.
Paludan SR; Department of Biomedicine, Aarhus University, Aarhus, Denmark.
Holm CK; Department of Biomedicine, Aarhus University, Aarhus, Denmark.
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Źródło:
PloS one [PLoS One] 2013 Dec 16; Vol. 8 (12), pp. e81790. Date of Electronic Publication: 2013 Dec 16 (Print Publication: 2013).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Herpes Genitalis/*metabolism
Herpesvirus 2, Human/*immunology
Immunity, Innate/*physiology
Receptors, Interleukin-21/*metabolism
Animals ; Female ; Herpes Genitalis/immunology ; Herpes Genitalis/virology ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Receptors, Interleukin-21/genetics ; Vagina/metabolism ; Vagina/virology ; Viral Load
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Abundant expression of interleukin-21 receptor in follicular lymphoma cells is associated with more aggressive disease.
Autorzy:
Wood B; Department of Pathology and Molecular Medicine, Queen's University, Kingston, Ontario, Canada.
Sikdar S
Choi SJ
Virk S
Alhejaily A
Baetz T
LeBrun DP
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Źródło:
Leukemia & lymphoma [Leuk Lymphoma] 2013 Jun; Vol. 54 (6), pp. 1212-20. Date of Electronic Publication: 2012 Nov 23.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Lymphoma, Follicular/*metabolism
Lymphoma, Follicular/*pathology
Receptors, Interleukin-21/*metabolism
Adult ; Aged ; Aged, 80 and over ; Disease Progression ; Female ; Gene Expression ; Humans ; Immunohistochemistry ; Lymphoid Tissue/metabolism ; Lymphoid Tissue/pathology ; Lymphoma, Follicular/genetics ; Lymphoma, Follicular/mortality ; Lymphoma, Follicular/therapy ; Male ; Middle Aged ; Neoplasm Staging ; Prognosis ; Receptors, Interleukin-21/genetics
Czasopismo naukowe
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Tytuł:
Interleukin 21 collaborates with interferon-γ for the optimal expression of interferon-stimulated genes and enhances protection against enteric microbial infection.
Autorzy:
Solaymani-Mohammadi S; Vaccine Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD, United States of America.
Berzofsky JA; Vaccine Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD, United States of America.
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Źródło:
PLoS pathogens [PLoS Pathog] 2019 Feb 28; Vol. 15 (2), pp. e1007614. Date of Electronic Publication: 2019 Feb 28 (Print Publication: 2019).
Typ publikacji:
Journal Article; Research Support, N.I.H., Intramural
MeSH Terms:
Enterobacteriaceae Infections/*immunology
Interferon-gamma/*metabolism
Interleukins/*metabolism
Animals ; Antiviral Agents ; CD4-Positive T-Lymphocytes ; Citrobacter rodentium/immunology ; Citrobacter rodentium/pathogenicity ; Colitis/immunology ; Female ; Gastrointestinal Microbiome/immunology ; Interferon-gamma/physiology ; Interleukins/physiology ; Intestinal Mucosa ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Phosphorylation ; Receptors, Interleukin-21/immunology ; Receptors, Interleukin-21/metabolism ; STAT1 Transcription Factor/metabolism ; STAT3 Transcription Factor/metabolism ; Signal Transduction
Czasopismo naukowe
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Tytuł:
Association analysis of SNPs in the IL21R gene with recurrent airway obstruction (RAO) in Swiss Warmblood horses.
Autorzy:
Klukowska-Rötzler J; Institute of Genetics, Vetsuisse Faculty, University of Bern, Switzerland. />Gerber V
Leeb T
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Źródło:
Animal genetics [Anim Genet] 2012 Aug; Vol. 43 (4), pp. 475-6. Date of Electronic Publication: 2011 Nov 07.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Polymorphism, Single Nucleotide*
Airway Obstruction/*prevention & control
Airway Obstruction/*veterinary
Horse Diseases/*prevention & control
Horses/*genetics
Receptors, Interleukin-21/*genetics
Airway Obstruction/genetics ; Animals ; Chromosomes, Mammalian/genetics ; Genome ; Haplotypes ; Horse Diseases/genetics ; Phenotype ; Receptors, Interleukin-21/metabolism ; Recurrence
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
IL-21 promotes myocardial ischaemia/reperfusion injury through the modulation of neutrophil infiltration.
Autorzy:
Wang K; Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Key Laboratory of Biological Targeted Therapy of Education Ministry and Hubei Province, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Wen S; Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Key Laboratory of Biological Targeted Therapy of Education Ministry and Hubei Province, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Jiao J; Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Key Laboratory of Biological Targeted Therapy of Education Ministry and Hubei Province, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Tang T; Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Key Laboratory of Biological Targeted Therapy of Education Ministry and Hubei Province, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zhao X; Department of Cardiology, Beijing Anzhen Hospital, Capital Medical University, Beijing, China.
Zhang M; Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Key Laboratory of Biological Targeted Therapy of Education Ministry and Hubei Province, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Lv B; Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Key Laboratory of Biological Targeted Therapy of Education Ministry and Hubei Province, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Lu Y; Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Key Laboratory of Biological Targeted Therapy of Education Ministry and Hubei Province, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zhou X; Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Key Laboratory of Biological Targeted Therapy of Education Ministry and Hubei Province, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Li J; Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Key Laboratory of Biological Targeted Therapy of Education Ministry and Hubei Province, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Nie S; Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Key Laboratory of Biological Targeted Therapy of Education Ministry and Hubei Province, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Liao Y; Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Key Laboratory of Biological Targeted Therapy of Education Ministry and Hubei Province, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Wang Q; Key Laboratory of Molecular Biophysics of the Ministry of Education, Cardio-X Institute, College of Life Science and Technology and Center of Human Genome Research, Huazhong University of Science and Technology, Wuhan, China.
Tu X; Key Laboratory of Molecular Biophysics of the Ministry of Education, Cardio-X Institute, College of Life Science and Technology and Center of Human Genome Research, Huazhong University of Science and Technology, Wuhan, China.
Mallat Z; Division of Cardiovascular Medicine, Department of Medicine, University of Cambridge, Cambridge, UK.
Xia N; Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Key Laboratory of Biological Targeted Therapy of Education Ministry and Hubei Province, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Cheng X; Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Key Laboratory of Biological Targeted Therapy of Education Ministry and Hubei Province, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
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Źródło:
British journal of pharmacology [Br J Pharmacol] 2018 Apr; Vol. 175 (8), pp. 1329-1343. Date of Electronic Publication: 2017 Apr 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Interleukins/*physiology
Myocardial Reperfusion Injury/*metabolism
Neutrophils/*physiology
Animals ; Cell Movement ; Cells, Cultured ; Chemokine CXCL1/physiology ; Chemokine CXCL2/physiology ; Fibroblasts/physiology ; Male ; Mice, Inbred C57BL ; Myocytes, Cardiac/physiology ; Receptors, Interleukin-21/physiology ; Troponin T/blood
Czasopismo naukowe
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Tytuł:
Distinct T helper cell dependence of memory B-cell proliferation versus plasma cell differentiation.
Autorzy:
Zabel F; Department of Dermatology, Zurich University Hospital, Schlieren/Zurich, Switzerland.
Fettelschoss A; Department of Dermatology, Zurich University Hospital, Schlieren/Zurich, Switzerland.
Vogel M; Inselspital, Department of Immunology, University of Bern, Bern, Switzerland.
Johansen P; Department of Dermatology, Zurich University Hospital, Zurich, Switzerland.
Kündig TM; Department of Dermatology, Zurich University Hospital, Zurich, Switzerland.
Bachmann MF; Department of Dermatology, Zurich University Hospital, Schlieren/Zurich, Switzerland.; Inselspital, Department of Immunology, University of Bern, Bern, Switzerland.
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Źródło:
Immunology [Immunology] 2017 Mar; Vol. 150 (3), pp. 329-342. Date of Electronic Publication: 2017 Jan 05.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Immunologic Memory*/genetics
B-Lymphocyte Subsets/*immunology
B-Lymphocytes/*immunology
Plasma Cells/*immunology
T-Lymphocytes, Helper-Inducer/*immunology
Animals ; Antibody Formation ; CD40 Ligand/genetics ; Cell Differentiation ; Cell Proliferation ; Cytokines/metabolism ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Receptors, Interleukin-21/genetics
Czasopismo naukowe
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Tytuł:
Mechanism involved in interleukin-21-induced phagocytosis in human monocytes and macrophages.
Autorzy:
Vallières F; Laboratoire de recherche en inflammation et physiologie des granulocytes, Université du Québec, INRS-Institut Armand-Frappier, Laval, QC, Canada.
Girard D; Laboratoire de recherche en inflammation et physiologie des granulocytes, Université du Québec, INRS-Institut Armand-Frappier, Laval, QC, Canada.
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Źródło:
Clinical and experimental immunology [Clin Exp Immunol] 2017 Feb; Vol. 187 (2), pp. 294-303. Date of Electronic Publication: 2016 Nov 22.
Typ publikacji:
Journal Article
MeSH Terms:
Phagocytosis*
Escherichia coli/*immunology
Interleukins/*metabolism
Macrophages/*immunology
Receptors, Interleukin-21/*metabolism
Animals ; Cattle ; Cells, Cultured ; Erythrocytes/immunology ; Humans ; Interleukins/immunology ; STAT1 Transcription Factor/metabolism ; STAT3 Transcription Factor/metabolism ; Signal Transduction ; Zymosan/metabolism ; p38 Mitogen-Activated Protein Kinases/metabolism
Czasopismo naukowe
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Tytuł:
Interleukin 21 - its potential role in the therapy of B-cell lymphomas.
Autorzy:
Bhatt S; a Dana-Farber Cancer Institute/Harvard Medical School , Boston , MA , USA.
Sarosiek KA; a Dana-Farber Cancer Institute/Harvard Medical School , Boston , MA , USA.
Lossos IS; b Department of Molecular and Cellular Pharmacology , University of Miami Miller School of Medicine , Miami , FL , USA.; c Department of Medicine, Division of Hematology-Oncology , Sylvester Comprehensive Cancer Center, University of Miami Miller School of Medicine , Miami , FL , USA.
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Źródło:
Leukemia & lymphoma [Leuk Lymphoma] 2017 Jan; Vol. 58 (1), pp. 17-29. Date of Electronic Publication: 2016 Jul 13.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Interleukins/*genetics
Interleukins/*metabolism
Lymphoma, B-Cell/*etiology
Lymphoma, B-Cell/*metabolism
Animals ; Clinical Trials as Topic ; Cytotoxicity, Immunologic/drug effects ; Drug Evaluation, Preclinical ; Humans ; Immune System/cytology ; Immune System/immunology ; Immune System/metabolism ; Immunologic Factors/pharmacology ; Immunologic Factors/therapeutic use ; Immunomodulation/drug effects ; Interleukins/pharmacology ; Interleukins/therapeutic use ; Lymphoma, B-Cell/drug therapy ; Receptors, Interleukin-21/metabolism ; Signal Transduction/drug effects ; Treatment Outcome
Czasopismo naukowe
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