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Wyszukujesz frazę ""STAT1 Transcription Factor"" wg kryterium: Temat


Tytuł:
Regulation of STAT1 and STAT4 Expression by Growth Factor and Interferon Supplementation in Sjögren's Syndrome Cell Culture Models.
Autorzy:
Mougeot JC; Translational Research Laboratories, Oral Medicine, Oral & Maxillofacial Surgery, Atrium Health-Carolinas Medical Center, Charlotte, NC 28203, USA.; Department of Otolaryngology, Head & Neck Surgery, Wake Forest University School of Medicine, Winston-Salem, NC 27157, USA.
Thornburg TE; Translational Research Laboratories, Oral Medicine, Oral & Maxillofacial Surgery, Atrium Health-Carolinas Medical Center, Charlotte, NC 28203, USA.; Department of Otolaryngology, Head & Neck Surgery, Wake Forest University School of Medicine, Winston-Salem, NC 27157, USA.
Noll BD; Translational Research Laboratories, Oral Medicine, Oral & Maxillofacial Surgery, Atrium Health-Carolinas Medical Center, Charlotte, NC 28203, USA.; Department of Otolaryngology, Head & Neck Surgery, Wake Forest University School of Medicine, Winston-Salem, NC 27157, USA.
Brennan MT; Translational Research Laboratories, Oral Medicine, Oral & Maxillofacial Surgery, Atrium Health-Carolinas Medical Center, Charlotte, NC 28203, USA.; Department of Otolaryngology, Head & Neck Surgery, Wake Forest University School of Medicine, Winston-Salem, NC 27157, USA.
Mougeot FB; Translational Research Laboratories, Oral Medicine, Oral & Maxillofacial Surgery, Atrium Health-Carolinas Medical Center, Charlotte, NC 28203, USA.; Department of Otolaryngology, Head & Neck Surgery, Wake Forest University School of Medicine, Winston-Salem, NC 27157, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2024 Mar 09; Vol. 25 (6). Date of Electronic Publication: 2024 Mar 09.
Typ publikacji:
Journal Article
MeSH Terms:
Epidermal Growth Factor*/pharmacology
Epidermal Growth Factor*/metabolism
Sjogren's Syndrome*/drug therapy
Sjogren's Syndrome*/genetics
Humans ; Interferon-alpha/pharmacology ; Immunologic Factors ; Cell Culture Techniques ; RNA, Messenger/metabolism ; Dietary Supplements ; STAT1 Transcription Factor/genetics ; STAT1 Transcription Factor/metabolism ; Phosphorylation ; STAT4 Transcription Factor/genetics ; STAT4 Transcription Factor/metabolism
Czasopismo naukowe
Tytuł:
P4HA1, transcriptionally activated by STAT1, promotes esophageal cancer progression.
Autorzy:
Gou W; Department of Pathology, The First Affiliated Hospital, Xinjiang Medical University, Urumqi, Xinjiang Province, China.; Department of Pathology, People's Hospital of Wanning, Wanning, Hainan Province, China.
Yang Y; Department of Endoscopy, People's Hospital of Wanning, Wanning, Hainan Province, China.
Shan Q; Department of Pathology, People's Hospital of Wanning, Wanning, Hainan Province, China.
Xia S; Department of Pathology, People's Hospital of Wanning, Wanning, Hainan Province, China.
Ma Y; Department of Pathology, The First Affiliated Hospital, Xinjiang Medical University, Urumqi, Xinjiang Province, China.
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Źródło:
Pathology international [Pathol Int] 2023 Apr; Vol. 73 (4), pp. 147-158. Date of Electronic Publication: 2023 Feb 03.
Typ publikacji:
Journal Article
MeSH Terms:
Esophageal Neoplasms*/pathology
MicroRNAs*/genetics
Procollagen-Proline Dioxygenase*/genetics
Procollagen-Proline Dioxygenase*/metabolism
STAT1 Transcription Factor*/genetics
STAT1 Transcription Factor*/metabolism
Humans ; Apoptosis ; Cell Line, Tumor ; Cell Proliferation ; Collagen/metabolism ; Gene Expression Regulation, Neoplastic
Czasopismo naukowe
Tytuł:
Inhibition of complement C3 prevents osteoarthritis progression in guinea pigs by blocking STAT1 activation.
Autorzy:
Xu JX; Department of Drug Discovery and Biomedical Sciences, Medical University of South Carolina, 70 President Street, Charleston, SC, 29425, USA. .
Xu FZ; Department of Drug Discovery and Biomedical Sciences, Medical University of South Carolina, 70 President Street, Charleston, SC, 29425, USA.; UAB Heersink School of Medicine, Alabama, AL, 35233, USA.
Furbish A; Department of Drug Discovery and Biomedical Sciences, Medical University of South Carolina, 70 President Street, Charleston, SC, 29425, USA.
Braxton AM; Department of Comparative Medicine, Medical University of South Carolina, 114 Doughty Street, Charleston, SC, 29425, USA.
Brumfield B; Department of Drug Discovery and Biomedical Sciences, Medical University of South Carolina, 70 President Street, Charleston, SC, 29425, USA.
Helke KL; Department of Comparative Medicine, Medical University of South Carolina, 114 Doughty Street, Charleston, SC, 29425, USA.
Peterson YK; Department of Drug Discovery and Biomedical Sciences, Medical University of South Carolina, 70 President Street, Charleston, SC, 29425, USA. .
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Źródło:
Communications biology [Commun Biol] 2024 Mar 27; Vol. 7 (1), pp. 370. Date of Electronic Publication: 2024 Mar 27.
Typ publikacji:
Journal Article
MeSH Terms:
Complement C3*/metabolism
Complement C3*/therapeutic use
Osteoarthritis*/prevention & control
Osteoarthritis*/drug therapy
Osteoarthritis*/metabolism
Adult ; Humans ; Guinea Pigs ; Animals ; Quality of Life ; Interleukin-1beta/metabolism ; Synovial Fluid ; STAT1 Transcription Factor/metabolism
Czasopismo naukowe
Tytuł:
Cytokine Signaling in Pediatric Kidney Tumor Cell Lines WT-CLS1, WT-3ab and G-401.
Autorzy:
Fasler-Kan E; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Milošević M; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Ruggiero S; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Aliu N; Department of Human Genetics, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Cholewa D; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Häcker FM; Department of Pediatric Surgery, Children's Hospital of Eastern Switzerland, CH-9000 St. Gallen, Switzerland.; Faculty of Medicine, University of Basel, CH-4031 Basel, Switzerland.
Dekany G; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Bartenstein A; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
Berger SM; Department of Pediatric Surgery, Children's Hospital, Inselspital Bern, University of Bern, CH-3010 Bern, Switzerland.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2024 Feb 14; Vol. 25 (4). Date of Electronic Publication: 2024 Feb 14.
Typ publikacji:
Journal Article
MeSH Terms:
Tumor Necrosis Factor-alpha*/metabolism
Kidney Neoplasms*/pathology
Child ; Humans ; Cytokines/metabolism ; Interferon-alpha/metabolism ; Histocompatibility Antigens Class I/metabolism ; HLA Antigens ; Cell Line, Tumor ; STAT1 Transcription Factor/metabolism ; Kidney/metabolism
Czasopismo naukowe
Tytuł:
Trophoblast-derived miR-410-5p induces M2 macrophage polarization and mediates immunotolerance at the fetal-maternal interface by targeting the STAT1 signaling pathway.
Autorzy:
Yang J; Reproductive Medical Center, Renmin Hospital of Wuhan University & Hubei Clinic Research Center for Assisted Reproductive Technology and Embryonic Development, Wuhan, 430060, Hubei, People's Republic of China.; Department of Gynecology, Affiliated Cancer Hospital of Xinjiang Medical University, Urumqi, 830000, Xinjiang, People's Republic of China.
Li L; Shenzhen Key Laboratory of Reproductive Immunology for Peri-Implantation, Shenzhen Zhongshan Institute for Reproductive Medicine and Genetics, Guangdong Engineering Technology Research Center of Reproductive Immunology for Peri-Implantation, Shenzhen Zhongshan Obstetrics & Gynecology Hospital (Formerly Shenzhen Zhongshan Urology Hospital), Shenzhen, 518045, Guangdong, People's Republic of China. .
Wang L; Shenzhen Key Laboratory of Reproductive Immunology for Peri-Implantation, Shenzhen Zhongshan Institute for Reproductive Medicine and Genetics, Guangdong Engineering Technology Research Center of Reproductive Immunology for Peri-Implantation, Shenzhen Zhongshan Obstetrics & Gynecology Hospital (Formerly Shenzhen Zhongshan Urology Hospital), Shenzhen, 518045, Guangdong, People's Republic of China.
Chen R; Shenzhen Key Laboratory of Reproductive Immunology for Peri-Implantation, Shenzhen Zhongshan Institute for Reproductive Medicine and Genetics, Guangdong Engineering Technology Research Center of Reproductive Immunology for Peri-Implantation, Shenzhen Zhongshan Obstetrics & Gynecology Hospital (Formerly Shenzhen Zhongshan Urology Hospital), Shenzhen, 518045, Guangdong, People's Republic of China.
Yang X; Department of Clinical Laboratory, Shenzhen Zhongshan Obstetrics & Gynecology Hospital (Formerly Shenzhen Zhongshan Urology Hospital), Shenzhen, 518045, Guangdong, People's Republic of China.
Wu J; Department of Gynecology, Shenzhen Zhongshan Obstetrics & Gynecology Hospital (Formerly Shenzhen Zhongshan Urology Hospital), Shenzhen, 518045, Guangdong, People's Republic of China.
Feng G; Shenzhen Key Laboratory of Reproductive Immunology for Peri-Implantation, Shenzhen Zhongshan Institute for Reproductive Medicine and Genetics, Guangdong Engineering Technology Research Center of Reproductive Immunology for Peri-Implantation, Shenzhen Zhongshan Obstetrics & Gynecology Hospital (Formerly Shenzhen Zhongshan Urology Hospital), Shenzhen, 518045, Guangdong, People's Republic of China.
Ding J; Reproductive Medical Center, Renmin Hospital of Wuhan University & Hubei Clinic Research Center for Assisted Reproductive Technology and Embryonic Development, Wuhan, 430060, Hubei, People's Republic of China.
Diao L; Shenzhen Key Laboratory of Reproductive Immunology for Peri-Implantation, Shenzhen Zhongshan Institute for Reproductive Medicine and Genetics, Guangdong Engineering Technology Research Center of Reproductive Immunology for Peri-Implantation, Shenzhen Zhongshan Obstetrics & Gynecology Hospital (Formerly Shenzhen Zhongshan Urology Hospital), Shenzhen, 518045, Guangdong, People's Republic of China.
Chen J; Reproductive Medical Center, Renmin Hospital of Wuhan University & Hubei Clinic Research Center for Assisted Reproductive Technology and Embryonic Development, Wuhan, 430060, Hubei, People's Republic of China. .
Yang J; Reproductive Medical Center, Renmin Hospital of Wuhan University & Hubei Clinic Research Center for Assisted Reproductive Technology and Embryonic Development, Wuhan, 430060, Hubei, People's Republic of China. .
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Źródło:
Journal of translational medicine [J Transl Med] 2024 Jan 04; Vol. 22 (1), pp. 19. Date of Electronic Publication: 2024 Jan 04.
Typ publikacji:
Journal Article
MeSH Terms:
Abortion, Spontaneous*/genetics
Abortion, Spontaneous*/metabolism
MicroRNAs*/genetics
MicroRNAs*/metabolism
Humans ; Female ; Pregnancy ; Mice ; Animals ; Placenta/metabolism ; STAT1 Transcription Factor/genetics ; STAT1 Transcription Factor/metabolism ; Lipopolysaccharides/metabolism ; Trophoblasts/metabolism ; Signal Transduction/genetics ; Macrophages/metabolism
Czasopismo naukowe
Tytuł:
AIM2 promotes irradiation resistance, migration ability and PD-L1 expression through STAT1/NF-κB activation in oral squamous cell carcinoma.
Autorzy:
Chiu HW; Graduate Institute of Clinical Medicine, College of Medicine, Taipei Medical University, Taipei, 11031, Taiwan.; Department of Medical Research, Shuang Ho Hospital, Taipei Medical University, New Taipei City, 23561, Taiwan.; TMU Research Center of Urology and Kidney, Taipei Medical University, Taipei, 11031, Taiwan.
Lee HL; Department of Radiology, School of Medicine, College of Medicine, Taipei Medical University, Taipei, 11031, Taiwan.; Department of Radiation Oncology, Taipei Medical University Hospital, Taipei, 11031, Taiwan.
Lee HH; Department of Neurology, Taipei Medical University Hospital, Taipei Medical University, Taipei, 11031, Taiwan.; Department of Neurology, School of Medicine, College of Medicine, Taipei Medical University, Taipei, 11031, Taiwan.; Department of Neurology, Vertigo and Balance Impairment Center, Shuang Ho Hospital, Taipei Medical University, New Taipei City, 23561, Taiwan.
Lu HW; Graduate Institute of Clinical Medicine, College of Medicine, Taipei Medical University, Taipei, 11031, Taiwan.; Department of Otolaryngology Head and Neck Surgery, Shuang Ho Hospital, Taipei Medical University, New Taipei City, 23561, Taiwan.; Department of Otolaryngology, Taipei Medical University Hospital, Taipei Medical University, Taipei, 11031, Taiwan.
Lin KY; Department of Obstetrics and Gynaecology, North Shore Private Hospital, Sydney, NSW, Australia.; Department of Gynecology, Ryde Hospital, Northern Sydney Local Health District, Sydney, NSW, Australia.; Northern Clinical School, Faculty of Medicine and Health, The University of Sydney, Sydney, NSW, Australia.
Lin YF; Graduate Institute of Clinical Medicine, College of Medicine, Taipei Medical University, Taipei, 11031, Taiwan.; Cell Physiology and Molecular Image Research Center, Wan Fang Hospital, Taipei Medical University, Taipei, 11696, Taiwan.
Lin CH; Department of Otolaryngology, Taipei Medical University Hospital, Taipei Medical University, Taipei, 11031, Taiwan. .; Department of Otolaryngology, School of Medicine, College of Medicine, Taipei Medical University, 250 Wu-Hsing Street, Taipei, 11031, Taiwan. .
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Źródło:
Journal of translational medicine [J Transl Med] 2024 Jan 03; Vol. 22 (1), pp. 13. Date of Electronic Publication: 2024 Jan 03.
Typ publikacji:
Journal Article
MeSH Terms:
Carcinoma, Squamous Cell*/pathology
Head and Neck Neoplasms*/genetics
Head and Neck Neoplasms*/pathology
Mouth Neoplasms*/genetics
Mouth Neoplasms*/pathology
Humans ; B7-H1 Antigen/genetics ; B7-H1 Antigen/metabolism ; DNA-Binding Proteins/genetics ; DNA-Binding Proteins/metabolism ; NF-kappa B/metabolism ; Squamous Cell Carcinoma of Head and Neck ; STAT1 Transcription Factor/genetics ; STAT1 Transcription Factor/metabolism
Czasopismo naukowe
Tytuł:
PVT1 promotes proliferation and macrophage immunosuppressive polarization through STAT1 and CX3CL1 regulation in glioblastoma multiforme.
Autorzy:
Huang L; Department of Pathophysiology, Beijing Neurosurgical Institute, Capital Medical University, China.
Wang Z; Department of Neurosurgery, Beijing Tiantan Hospital, Capital Medical University, Beijing, China.
Liao C; Department of Molecular Neuropathology, Beijing Neurosurgical Institute, Capital Medical University, Beijing, China.
Zhao Z; Department of Molecular Neuropathology, Beijing Neurosurgical Institute, Capital Medical University, Beijing, China.
Gao H; Department of Cell Biology, Beijing Neurosurgical Institute, Capital Medical University, Beijing, China.
Huang R; Department of Neurosurgery, Beijing Tiantan Hospital, Capital Medical University, Beijing, China.
Chen J; Department of Molecular Neuropathology, Beijing Neurosurgical Institute, Capital Medical University, Beijing, China.
Wu F; Department of Molecular Neuropathology, Beijing Neurosurgical Institute, Capital Medical University, Beijing, China.
Zeng F; Department of Molecular Neuropathology, Beijing Neurosurgical Institute, Capital Medical University, Beijing, China.
Zhang Y; Department of Molecular Neuropathology, Beijing Neurosurgical Institute, Capital Medical University, Beijing, China.
Jiang T; Department of Neurosurgery, Beijing Tiantan Hospital, Capital Medical University, Beijing, China.; Department of Molecular Neuropathology, Beijing Neurosurgical Institute, Capital Medical University, Beijing, China.; Center of Brain Tumor, Beijing Institute for Brain Disorders, Beijing, China.; China National Clinical Research Center for Neurological Diseases, Beijing, China.; Chinese Glioma Genome Atlas Network and Asian Glioma Genome Atlas Network, Beijing, China.
Hu H; Department of Molecular Neuropathology, Beijing Neurosurgical Institute, Capital Medical University, Beijing, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Jan; Vol. 30 (1), pp. e14566.
Typ publikacji:
Journal Article
MeSH Terms:
Glioblastoma*/pathology
Glioma*/genetics
RNA, Long Noncoding*/genetics
RNA, Long Noncoding*/metabolism
MicroRNAs*/metabolism
Humans ; Cell Line, Tumor ; Macrophages/pathology ; Cell Proliferation/genetics ; Gene Expression Regulation, Neoplastic ; Tumor Microenvironment ; STAT1 Transcription Factor/genetics ; STAT1 Transcription Factor/metabolism ; Chemokine CX3CL1/genetics ; Chemokine CX3CL1/metabolism
Czasopismo naukowe
Tytuł:
Ultrasound reduces inflammation by modulating M1/M2 polarization of microglia through STAT1/STAT6/PPARγ signaling pathways.
Autorzy:
Hsu CH; Department of Biomedical Imaging and Radiological Sciences, School of Biomedical Science and Engineering, National Yang Ming Chiao Tung University, Taipei, Taiwan.
Pan YJ; Department of Psychiatry, Far Eastern Memorial Hospital, New Taipei, Taiwan.; Department of Chemical Engineering and Materials Science, Yuan Ze University, Taoyuan, Taiwan.
Zheng YT; Department of Biomedical Imaging and Radiological Sciences, School of Biomedical Science and Engineering, National Yang Ming Chiao Tung University, Taipei, Taiwan.
Lo RY; Department of Neurology, Buddhist Tzu Chi General Hospital and Tzu Chi University, Hualien, Taiwan.
Yang FY; Department of Biomedical Imaging and Radiological Sciences, School of Biomedical Science and Engineering, National Yang Ming Chiao Tung University, Taipei, Taiwan.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2023 Dec; Vol. 29 (12), pp. 4113-4123. Date of Electronic Publication: 2023 Jul 03.
Typ publikacji:
Journal Article
MeSH Terms:
Microglia*
PPAR gamma*
Humans ; Lipopolysaccharides/pharmacology ; STAT1 Transcription Factor/metabolism ; STAT1 Transcription Factor/pharmacology ; Signal Transduction ; Inflammation/metabolism ; STAT6 Transcription Factor
Czasopismo naukowe
Tytuł:
ESCO2 promotes hypopharyngeal carcinoma progression in a STAT1-dependent manner.
Autorzy:
Hu J; Department of Otorhinolaryngology, Head and Neck Surgery, the Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Yan J; Department of Otorhinolaryngology, Head and Neck Surgery, the Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Chen Y; Department of Otorhinolaryngology, Head and Neck Surgery, the Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Li X; Department of Otorhinolaryngology, Head and Neck Surgery, the Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Yang L; Department of Otorhinolaryngology, Head and Neck Surgery, the Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Di H; Department of Otorhinolaryngology, Head and Neck Surgery, the Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Zhang H; Department of Otorhinolaryngology, Head and Neck Surgery, the Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Shi Y; Department of Otorhinolaryngology, Head and Neck Surgery, the Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Zhao J; Department of Otorhinolaryngology, Head and Neck Surgery, the Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Shi Y; Department of Otorhinolaryngology, Head and Neck Surgery, the Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Xu Y; Department of Otorhinolaryngology, Head and Neck Surgery, the Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Ren X; Department of Otorhinolaryngology, Head and Neck Surgery, the Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Wang Z; Department of Otorhinolaryngology, Head and Neck Surgery, the Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China. .; Department of Maxillofacial Surgery, Affiliated Stomatological Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China. .
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Źródło:
BMC cancer [BMC Cancer] 2023 Nov 15; Vol. 23 (1), pp. 1114. Date of Electronic Publication: 2023 Nov 15.
Typ publikacji:
Journal Article
MeSH Terms:
Chromosome Segregation*
Head and Neck Neoplasms*
Animals ; Mice ; Humans ; Mice, Nude ; Cell Proliferation ; Squamous Cell Carcinoma of Head and Neck/genetics ; Cell Line, Tumor ; STAT1 Transcription Factor/genetics ; STAT1 Transcription Factor/metabolism ; Acetyltransferases/genetics ; Chromosomal Proteins, Non-Histone/genetics
Czasopismo naukowe
Tytuł:
Microglial STAT1-sufficiency is required for resistance to toxoplasmic encephalitis.
Autorzy:
Cowan MN; Center for Brain Immunology and Glia, Department of Neuroscience, University of Virginia, Charlottesville, Virginia, United States of America.
Kovacs MA; Center for Brain Immunology and Glia, Department of Neuroscience, University of Virginia, Charlottesville, Virginia, United States of America.
Sethi I; Center for Brain Immunology and Glia, Department of Neuroscience, University of Virginia, Charlottesville, Virginia, United States of America.
Babcock IW; Center for Brain Immunology and Glia, Department of Neuroscience, University of Virginia, Charlottesville, Virginia, United States of America.
Still K; Center for Brain Immunology and Glia, Department of Neuroscience, University of Virginia, Charlottesville, Virginia, United States of America.
Batista SJ; Center for Brain Immunology and Glia, Department of Neuroscience, University of Virginia, Charlottesville, Virginia, United States of America.
O'Brien CA; Center for Brain Immunology and Glia, Department of Neuroscience, University of Virginia, Charlottesville, Virginia, United States of America.
Thompson JA; Center for Brain Immunology and Glia, Department of Neuroscience, University of Virginia, Charlottesville, Virginia, United States of America.
Sibley LA; Center for Brain Immunology and Glia, Department of Neuroscience, University of Virginia, Charlottesville, Virginia, United States of America.
Labuzan SA; Center for Brain Immunology and Glia, Department of Neuroscience, University of Virginia, Charlottesville, Virginia, United States of America.
Harris TH; Center for Brain Immunology and Glia, Department of Neuroscience, University of Virginia, Charlottesville, Virginia, United States of America.
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Źródło:
PLoS pathogens [PLoS Pathog] 2022 Sep 06; Vol. 18 (9), pp. e1010637. Date of Electronic Publication: 2022 Sep 06 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Encephalitis*/metabolism
Encephalitis*/pathology
STAT1 Transcription Factor*/genetics
STAT1 Transcription Factor*/metabolism
Toxoplasma*/metabolism
Toxoplasmosis, Cerebral*/metabolism
Animals ; Brain/pathology ; Mice ; Microglia/metabolism
Czasopismo naukowe
Tytuł:
RanBP2/Nup358 Mediates Sumoylation of STAT1 and Antagonizes Interferon-α-Mediated Antiviral Innate Immunity.
Autorzy:
Li J; Department of Immunology, School of Basic Medical Sciences, Fujian Medical University, Fuzhou 350108, China.
Su L; Department of Immunology, School of Basic Medical Sciences, Fujian Medical University, Fuzhou 350108, China.
Jiang J; Department of Immunology, School of Basic Medical Sciences, Fujian Medical University, Fuzhou 350108, China.
Wang YE; Department of Biochemistry, University of Toronto, Toronto, ON M5G 1M1, Canada.
Ling Y; Department of Immunology, School of Basic Medical Sciences, Fujian Medical University, Fuzhou 350108, China.
Qiu Y; Department of Biochemistry, University of Toronto, Toronto, ON M5G 1M1, Canada.
Yu H; Department of Immunology, School of Basic Medical Sciences, Fujian Medical University, Fuzhou 350108, China.
Huang Y; Department of Immunology, School of Basic Medical Sciences, Fujian Medical University, Fuzhou 350108, China.
Wu J; Department of Immunology, School of Basic Medical Sciences, Fujian Medical University, Fuzhou 350108, China.
Jiang S; Department of Immunology, School of Basic Medical Sciences, Fujian Medical University, Fuzhou 350108, China.
Zhang T; Department of Immunology, School of Basic Medical Sciences, Fujian Medical University, Fuzhou 350108, China.
Palazzo AF; Department of Biochemistry, University of Toronto, Toronto, ON M5G 1M1, Canada.
Shen Q; Department of Immunology, School of Basic Medical Sciences, Fujian Medical University, Fuzhou 350108, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Dec 25; Vol. 25 (1). Date of Electronic Publication: 2023 Dec 25.
Typ publikacji:
Journal Article
MeSH Terms:
Interferon-alpha*/pharmacology
Virus Diseases*
Humans ; Nuclear Pore Complex Proteins ; Sumoylation ; Immunity, Innate ; Antiviral Agents ; STAT1 Transcription Factor
Czasopismo naukowe
Tytuł:
ISGF3 and STAT2/IRF9 Control Basal and IFN-Induced Transcription through Genome-Wide Binding of Phosphorylated and Unphosphorylated Complexes to Common ISRE-Containing ISGs.
Autorzy:
Nowicka H; Human Molecular Genetics Research Unit, Faculty of Biology, Institute of Molecular Biology and Biotechnology, Adam Mickiewicz University, 61-614 Poznan, Poland.
Sekrecka A; Human Molecular Genetics Research Unit, Faculty of Biology, Institute of Molecular Biology and Biotechnology, Adam Mickiewicz University, 61-614 Poznan, Poland.
Blaszczyk K; Human Molecular Genetics Research Unit, Faculty of Biology, Institute of Molecular Biology and Biotechnology, Adam Mickiewicz University, 61-614 Poznan, Poland.
Kluzek K; Human Molecular Genetics Research Unit, Faculty of Biology, Institute of Molecular Biology and Biotechnology, Adam Mickiewicz University, 61-614 Poznan, Poland.
Chang CY; Graduate Institute of Immunology, National Taiwan University College of Medicine, Taipei 100, Taiwan.
Wesoly J; Laboratory of High Throughput Technologies, Faculty of Biology, Adam Mickiewicz University, 61-614 Poznan, Poland.
Lee CK; Graduate Institute of Immunology, National Taiwan University College of Medicine, Taipei 100, Taiwan.
Bluyssen HAR; Human Molecular Genetics Research Unit, Faculty of Biology, Institute of Molecular Biology and Biotechnology, Adam Mickiewicz University, 61-614 Poznan, Poland.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Dec 18; Vol. 24 (24). Date of Electronic Publication: 2023 Dec 18.
Typ publikacji:
Journal Article
MeSH Terms:
Interferon Type I*/metabolism
Interleukin-1 Receptor-Like 1 Protein*/metabolism
Interferon-Stimulated Gene Factor 3*/metabolism
STAT2 Transcription Factor*/metabolism
Antiviral Agents/pharmacology ; DNA/pharmacology ; Immunoglobulins/metabolism ; Signal Transduction ; STAT1 Transcription Factor/metabolism ; Humans
Czasopismo naukowe
Tytuł:
GADD45A regulates subcutaneous fat deposition and lipid metabolism by interacting with Stat1.
Autorzy:
You W; College of Animal Sciences, Zhejiang University, Hangzhou, China.; The Key Laboratory of Molecular Animal Nutrition, Ministry of Education, Hangzhou, China.; Zhejiang Provincial Laboratory of Feed and Animal Nutrition, No. 866 Yuhangtang Road, Hangzhou, 310058, Zhejiang, China.
Liu S; College of Animal Sciences, Zhejiang University, Hangzhou, China.; The Key Laboratory of Molecular Animal Nutrition, Ministry of Education, Hangzhou, China.; Zhejiang Provincial Laboratory of Feed and Animal Nutrition, No. 866 Yuhangtang Road, Hangzhou, 310058, Zhejiang, China.
Li J; College of Animal Sciences, Zhejiang University, Hangzhou, China.; The Key Laboratory of Molecular Animal Nutrition, Ministry of Education, Hangzhou, China.; Zhejiang Provincial Laboratory of Feed and Animal Nutrition, No. 866 Yuhangtang Road, Hangzhou, 310058, Zhejiang, China.
Tu Y; College of Animal Sciences, Zhejiang University, Hangzhou, China.; The Key Laboratory of Molecular Animal Nutrition, Ministry of Education, Hangzhou, China.; Zhejiang Provincial Laboratory of Feed and Animal Nutrition, No. 866 Yuhangtang Road, Hangzhou, 310058, Zhejiang, China.
Shan T; College of Animal Sciences, Zhejiang University, Hangzhou, China. .; The Key Laboratory of Molecular Animal Nutrition, Ministry of Education, Hangzhou, China. .; Zhejiang Provincial Laboratory of Feed and Animal Nutrition, No. 866 Yuhangtang Road, Hangzhou, 310058, Zhejiang, China. .
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Źródło:
BMC biology [BMC Biol] 2023 Oct 09; Vol. 21 (1), pp. 212. Date of Electronic Publication: 2023 Oct 09.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Lipid Metabolism*/genetics
Obesity*/genetics
Animals ; Humans ; Mice ; Adipogenesis/genetics ; Adipose Tissue, White/metabolism ; Cell Cycle Proteins/metabolism ; Mice, Inbred C57BL ; Mice, Knockout ; STAT1 Transcription Factor/metabolism ; STAT1 Transcription Factor/pharmacology ; Subcutaneous Fat/metabolism
Czasopismo naukowe
Tytuł:
CYLD stimulates macrophage phagocytosis of leukemic cells through STAT1 signalling in acute myeloid leukemia.
Autorzy:
Huyen NT; Graduate University of Science and Technology, Vietnam Academy of Science and Technology, Cau Giay, Ha Noi, Vietnam.; Faculty of Biotechnology, Vietnam National University of Agriculture, Gia Lam, Hanoi, Vietnam.
Ngoc NT; University of Science and Technology of Hanoi, Vietnam Academy of Science and Technology, Cau Giay, Ha Noi, Vietnam.
Giang NH; Institute of Genome Research, Vietnam Academy of Science and Technology, Cau Giay, Hanoi, Vietnam.
Trang DT; Institute of Genome Research, Vietnam Academy of Science and Technology, Cau Giay, Hanoi, Vietnam.
Hanh HH; Institute of Genome Research, Vietnam Academy of Science and Technology, Cau Giay, Hanoi, Vietnam.
Binh VD; National Institute of Hematology and Blood Transfusion, Pham Van Bach, Ha Noi, Vietnam.
Giang NV; Faculty of Biotechnology, Vietnam National University of Agriculture, Gia Lam, Hanoi, Vietnam.
Canh NX; Faculty of Biotechnology, Vietnam National University of Agriculture, Gia Lam, Hanoi, Vietnam.
Xuan NT; Graduate University of Science and Technology, Vietnam Academy of Science and Technology, Cau Giay, Ha Noi, Vietnam.; Institute of Genome Research, Vietnam Academy of Science and Technology, Cau Giay, Hanoi, Vietnam.
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Źródło:
PloS one [PLoS One] 2023 Aug 07; Vol. 18 (8), pp. e0283586. Date of Electronic Publication: 2023 Aug 07 (Print Publication: 2023).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Leukemia, Myeloid, Acute*/genetics
Humans ; Macrophages/metabolism ; Cytokines/metabolism ; Phagocytosis ; RNA, Small Interfering ; Deubiquitinating Enzyme CYLD/metabolism ; STAT1 Transcription Factor/genetics ; STAT1 Transcription Factor/metabolism
Czasopismo naukowe
Tytuł:
The Cell-Autonomous Pro-Metastatic Activities of PD-L1 in Breast Cancer Are Regulated by N-Linked Glycosylation-Dependent Activation of STAT3 and STAT1.
Autorzy:
Erlichman N; The Shmunis School of Biomedicine and Cancer Research, George S. Wise Faculty of Life Sciences, Tel Aviv University, Tel Aviv 6997801, Israel.
Meshel T; The Shmunis School of Biomedicine and Cancer Research, George S. Wise Faculty of Life Sciences, Tel Aviv University, Tel Aviv 6997801, Israel.
Baram T; The Shmunis School of Biomedicine and Cancer Research, George S. Wise Faculty of Life Sciences, Tel Aviv University, Tel Aviv 6997801, Israel.
Abu Raiya A; The Shmunis School of Biomedicine and Cancer Research, George S. Wise Faculty of Life Sciences, Tel Aviv University, Tel Aviv 6997801, Israel.
Horvitz T; The Shmunis School of Biomedicine and Cancer Research, George S. Wise Faculty of Life Sciences, Tel Aviv University, Tel Aviv 6997801, Israel.
Ben-Yaakov H; The Shmunis School of Biomedicine and Cancer Research, George S. Wise Faculty of Life Sciences, Tel Aviv University, Tel Aviv 6997801, Israel.
Ben-Baruch A; The Shmunis School of Biomedicine and Cancer Research, George S. Wise Faculty of Life Sciences, Tel Aviv University, Tel Aviv 6997801, Israel.
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Źródło:
Cells [Cells] 2023 Sep 23; Vol. 12 (19). Date of Electronic Publication: 2023 Sep 23.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
B7-H1 Antigen*/metabolism
Triple Negative Breast Neoplasms*/pathology
Mice ; Animals ; Humans ; Glycosylation ; Cell Line, Tumor ; T-Lymphocytes ; STAT1 Transcription Factor/metabolism ; STAT3 Transcription Factor/metabolism
Czasopismo naukowe
Tytuł:
Zika virus NS2A inhibits interferon signaling by degradation of STAT1 and STAT2.
Autorzy:
Fanunza E; Department of Life and Environmental Sciences, University of Cagliari, Monserrato, Italy.
Carletti F; Laboratory of Virology, National Institute for Infectious Diseases, L.Spallanzani͟ IRCCS, Rome, Italy.
Quartu M; Department of Biomedical Sciences, University of Cagliari, Monserrato, Italy.
Grandi N; Department of Life and Environmental Sciences, University of Cagliari, Monserrato, Italy.
Ermellino L; Department of Life and Environmental Sciences, University of Cagliari, Monserrato, Italy.
Milia J; Department of Life and Environmental Sciences, University of Cagliari, Monserrato, Italy.
Corona A; Department of Life and Environmental Sciences, University of Cagliari, Monserrato, Italy.
Capobianchi MR; Laboratory of Virology, National Institute for Infectious Diseases, L.Spallanzani͟ IRCCS, Rome, Italy.
Ippolito G; Laboratory of Virology, National Institute for Infectious Diseases, L.Spallanzani͟ IRCCS, Rome, Italy.
Tramontano E; Department of Life and Environmental Sciences, University of Cagliari, Monserrato, Italy.
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Źródło:
Virulence [Virulence] 2021 Dec; Vol. 12 (1), pp. 1580-1596.
Typ publikacji:
Journal Article
MeSH Terms:
Immune Evasion*
STAT1 Transcription Factor*/genetics
STAT1 Transcription Factor*/metabolism
STAT2 Transcription Factor*/genetics
Viral Nonstructural Proteins*/genetics
Viral Nonstructural Proteins*/metabolism
Zika Virus Infection*/immunology
Interferons/*immunology
Humans ; Immunity, Innate ; Zika Virus
Czasopismo naukowe
Tytuł:
PRL-3 induces a positive signaling circuit between glycolysis and activation of STAT1/2.
Autorzy:
Vandsemb EN; Department of Clinical and Molecular Medicine, Faculty of Medicine and Health Sciences, Norwegian University of Science and Technology (NTNU), Trondheim, Norway.
Rye MB; Department of Clinical and Molecular Medicine, Faculty of Medicine and Health Sciences, Norwegian University of Science and Technology (NTNU), Trondheim, Norway.; Clinic of Surgery, St. Olavs University Hospital, Trondheim, Norway.; Clinic of Laboratory Medicine, St. Olavs University Hospital, Trondheim, Norway.; Biocore - Bioinformatics Core Facility, Norwegian University of Science and Technology (NTNU), Trondheim, Norway.
Steiro IJ; Department of Clinical and Molecular Medicine, Faculty of Medicine and Health Sciences, Norwegian University of Science and Technology (NTNU), Trondheim, Norway.
Elsaadi S; Department of Clinical and Molecular Medicine, Faculty of Medicine and Health Sciences, Norwegian University of Science and Technology (NTNU), Trondheim, Norway.; Clinic of Laboratory Medicine, St. Olavs University Hospital, Trondheim, Norway.
Rø TB; Department of Clinical and Molecular Medicine, Faculty of Medicine and Health Sciences, Norwegian University of Science and Technology (NTNU), Trondheim, Norway.; Children's Clinic, St. Olavs University Hospital, Trondheim, Norway.
Slørdahl TS; Department of Clinical and Molecular Medicine, Faculty of Medicine and Health Sciences, Norwegian University of Science and Technology (NTNU), Trondheim, Norway.; Clinic of Medicine, St. Olavs University Hospital, Trondheim, Norway.
Sponaas AM; Department of Clinical and Molecular Medicine, Faculty of Medicine and Health Sciences, Norwegian University of Science and Technology (NTNU), Trondheim, Norway.
Børset M; Department of Clinical and Molecular Medicine, Faculty of Medicine and Health Sciences, Norwegian University of Science and Technology (NTNU), Trondheim, Norway.; Department of Immunology and Transfusion Medicine, St. Olavs University Hospital, Norway.
Abdollahi P; Department of Clinical and Molecular Medicine, Faculty of Medicine and Health Sciences, Norwegian University of Science and Technology (NTNU), Trondheim, Norway.; Clinic of Laboratory Medicine, St. Olavs University Hospital, Trondheim, Norway.; Clinic of Medicine, St. Olavs University Hospital, Trondheim, Norway.
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Źródło:
The FEBS journal [FEBS J] 2021 Dec; Vol. 288 (23), pp. 6700-6715. Date of Electronic Publication: 2021 Jun 20.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Transcriptional Activation*
Glycolysis/*genetics
Neoplasm Proteins/*genetics
Protein Tyrosine Phosphatases/*genetics
STAT1 Transcription Factor/*genetics
STAT2 Transcription Factor/*genetics
Signal Transduction/*genetics
Cell Line, Tumor ; Cell Survival/genetics ; Cytokines/genetics ; Cytokines/metabolism ; Exoribonucleases/genetics ; Exoribonucleases/metabolism ; Gene Expression Profiling/methods ; Gene Expression Regulation, Neoplastic ; Humans ; Interferon-Stimulated Gene Factor 3, gamma Subunit/genetics ; Interferon-Stimulated Gene Factor 3, gamma Subunit/metabolism ; Neoplasm Proteins/metabolism ; Protein Tyrosine Phosphatases/metabolism ; RNA-Seq/methods ; STAT1 Transcription Factor/metabolism ; STAT2 Transcription Factor/metabolism ; Ubiquitins/genetics ; Ubiquitins/metabolism
Czasopismo naukowe
Tytuł:
Proteasome inhibitors restore the STAT1 pathway and enhance the expression of MHC class I on human colon cancer cells.
Autorzy:
Liang YH; Graduate Institutes of Oncology, National Taiwan University College of Medicine, No 7, Chung-Shan South Rd, Taipei, 10002, Taiwan, R.O.C.; Graduate Institutes of Centers of Genomic and Precision Medicine, National Taiwan University College of Medicine, Taipei, Taiwan, R.O.C.; Departments of Oncology, National Taiwan University Hospital, Taipei, Taiwan, R.O.C.; National Taiwan University Cancer Center, Taipei, Taiwan, R.O.C.
Chen KH; Graduate Institutes of Oncology, National Taiwan University College of Medicine, No 7, Chung-Shan South Rd, Taipei, 10002, Taiwan, R.O.C.; Departments of Oncology, National Taiwan University Hospital, Taipei, Taiwan, R.O.C.; National Taiwan University Cancer Center, Taipei, Taiwan, R.O.C.
Tsai JH; Departments of Pathology, National Taiwan University Hospital, Taipei, Taiwan, R.O.C.
Cheng YM; Departments of Pathology, National Taiwan University Hospital, Taipei, Taiwan, R.O.C.
Lee CC; Graduate Institutes of Oncology, National Taiwan University College of Medicine, No 7, Chung-Shan South Rd, Taipei, 10002, Taiwan, R.O.C.; Graduate Institutes of Clinical Medicine, National Taiwan University College of Medicine, Taipei, Taiwan, R.O.C.
Kao CH; Graduate Institutes of Oncology, National Taiwan University College of Medicine, No 7, Chung-Shan South Rd, Taipei, 10002, Taiwan, R.O.C.; Graduate Institutes of Clinical Medicine, National Taiwan University College of Medicine, Taipei, Taiwan, R.O.C.
Chan KY; Graduate Institutes of Oncology, National Taiwan University College of Medicine, No 7, Chung-Shan South Rd, Taipei, 10002, Taiwan, R.O.C.
Chen YT; Graduate Institutes of Centers of Genomic and Precision Medicine, National Taiwan University College of Medicine, Taipei, Taiwan, R.O.C.
Hsu WL; Graduate Institutes of Centers of Genomic and Precision Medicine, National Taiwan University College of Medicine, Taipei, Taiwan, R.O.C.
Yeh KH; Graduate Institutes of Oncology, National Taiwan University College of Medicine, No 7, Chung-Shan South Rd, Taipei, 10002, Taiwan, R.O.C.. .; Graduate Institutes of Clinical Medicine, National Taiwan University College of Medicine, Taipei, Taiwan, R.O.C.. .; Departments of Oncology, National Taiwan University Hospital, Taipei, Taiwan, R.O.C.. .; National Taiwan University Cancer Center, Taipei, Taiwan, R.O.C.. .
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Źródło:
Journal of biomedical science [J Biomed Sci] 2021 Nov 10; Vol. 28 (1), pp. 75. Date of Electronic Publication: 2021 Nov 10.
Typ publikacji:
Journal Article
MeSH Terms:
Colonic Neoplasms/*physiopathology
Gene Expression/*drug effects
Genes, MHC Class I/*drug effects
Histocompatibility Antigens Class I/*genetics
Proteasome Inhibitors/*pharmacology
STAT1 Transcription Factor/*genetics
Signal Transduction/*drug effects
Cell Line, Tumor ; Humans ; STAT1 Transcription Factor/metabolism
Czasopismo naukowe
Tytuł:
STAT1-Induced Upregulation lncRNA LINC00958 Accelerates the Epithelial Ovarian Cancer Tumorigenesis by Regulating Wnt/ β -Catenin Signaling.
Autorzy:
Xie M; Department of Gynecology and Obstetrics, The Second Affiliated Hospital of Harbin Medical University, Harbin, Heilongjiang, China.
Fu Q; Department of Gynecology and Obstetrics, The Second Affiliated Hospital of Harbin Medical University, Harbin, Heilongjiang, China.
Wang PP; Department of Gynecology and Obstetrics, The Second Affiliated Hospital of Harbin Medical University, Harbin, Heilongjiang, China.
Cui YL; Department of Gynecology and Obstetrics, The Second Affiliated Hospital of Harbin Medical University, Harbin, Heilongjiang, China.
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Źródło:
Disease markers [Dis Markers] 2021 Nov 08; Vol. 2021, pp. 1405045. Date of Electronic Publication: 2021 Nov 08 (Print Publication: 2021).
Typ publikacji:
Journal Article
MeSH Terms:
Gene Expression Regulation, Neoplastic*
Biomarkers, Tumor/*metabolism
Carcinoma, Ovarian Epithelial/*pathology
Ovarian Neoplasms/*pathology
RNA, Long Noncoding/*genetics
STAT1 Transcription Factor/*metabolism
Wnt1 Protein/*metabolism
beta Catenin/*metabolism
Apoptosis ; Biomarkers, Tumor/genetics ; Carcinoma, Ovarian Epithelial/genetics ; Carcinoma, Ovarian Epithelial/metabolism ; Cell Proliferation ; Female ; Humans ; Ovarian Neoplasms/genetics ; Ovarian Neoplasms/metabolism ; Prognosis ; STAT1 Transcription Factor/genetics ; Tumor Cells, Cultured ; Wnt1 Protein/genetics ; beta Catenin/genetics
Czasopismo naukowe
Tytuł:
Chronic demodicosis in patients with immune dysregulation: An unexpected infectious manifestation of Signal transducer and activator of transcription (STAT)1 gain-of-function.
Autorzy:
Shamriz O; Allergy and Clinical Immunology Unit, Department of Medicine, Hadassah Medical Organization, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.; The Lautenberg Center for Immunology and Cancer Research, Institute of Medical Research Israel-Canada, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.
Lev A; Pediatric Department A and Immunology Service, Jeffrey Modell Foundation Center, Edmond and Lily Safra Children's Hospital, Sheba Medical Center, Tel Hashomer, Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.; Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Simon AJ; Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.; Sheba Cancer Research Center and Institute of Hematology, Sheba Medical Center, Tel Hashomer, Ramat Gan, Israel.
Barel O; Sheba Cancer Research Center and Institute of Hematology, Sheba Medical Center, Tel Hashomer, Ramat Gan, Israel.; The Genomic Unit, Sheba Cancer Research Center, Sheba Medical Center, Ramat Gan, Israel.; Sheba Medical Center, Wohl Institute of Translational Medicine, Ramat Gan, Israel.
Javasky E; Sheba Cancer Research Center and Institute of Hematology, Sheba Medical Center, Tel Hashomer, Ramat Gan, Israel.; The Genomic Unit, Sheba Cancer Research Center, Sheba Medical Center, Ramat Gan, Israel.; Sheba Medical Center, Wohl Institute of Translational Medicine, Ramat Gan, Israel.
Matza-Porges S; Department of Human Genetics, Institute for Medical Research the Hebrew University of Jerusalem,, Jerusalem, Israel.; Department of Biotechnology, Hadassah Academic College, Jerusalem, Israel.
Shaulov A; Department of Hematology, Hadassah Medical Organization, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.
Davidovics Z; Gastroenterology Unit, Department of Pediatrics, Hadassah Medical Organization, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.
Toker O; Allergy and Clinical Immunology Unit, Shaare Zedek Medical Center, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.
Somech R; Pediatric Department A and Immunology Service, Jeffrey Modell Foundation Center, Edmond and Lily Safra Children's Hospital, Sheba Medical Center, Tel Hashomer, Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.; Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Zlotogorski A; Pediatric Dermatology Service, Department of Dermatology, Hadassah Medical Organization, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.
Molho-Pessach V; Pediatric Dermatology Service, Department of Dermatology, Hadassah Medical Organization, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.
Tal Y; Allergy and Clinical Immunology Unit, Department of Medicine, Hadassah Medical Organization, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.
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Źródło:
Clinical and experimental immunology [Clin Exp Immunol] 2021 Oct; Vol. 206 (1), pp. 56-67. Date of Electronic Publication: 2021 Jul 12.
Typ publikacji:
Journal Article
MeSH Terms:
Gain of Function Mutation*
Genetic Diseases, Inborn*/genetics
Genetic Diseases, Inborn*/immunology
Genetic Diseases, Inborn*/parasitology
Immune System Diseases*/genetics
Immune System Diseases*/immunology
Immune System Diseases*/parasitology
Mite Infestations*/genetics
Mite Infestations*/immunology
STAT1 Transcription Factor*/genetics
STAT1 Transcription Factor*/immunology
Skin Diseases, Parasitic*/genetics
Skin Diseases, Parasitic*/immunology
Mites/*immunology
Adolescent ; Adult ; Animals ; Child ; Chronic Disease ; Female ; Humans ; Infant ; Male ; Middle Aged ; Retrospective Studies
Czasopismo naukowe

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