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Wyszukujesz frazę ""STAT3"" wg kryterium: Temat


Tytuł :
Amelioration of TPA-induced skin inflammation by the leaf extract of Vernonia amygdalina involves ERK/STAT3 (Ser727) signaling inhibition.
Autorzy :
Wu JY; Research and Development Centre for Natural Health Products, HKBU Institute for Research and Continuing Education, Shenzhen, China; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Hong Kong, China.
Xie JH; Department of Food and Biological Engineering, Zhangzhou Institute of Technology, China.
Chen YJ; Research and Development Centre for Natural Health Products, HKBU Institute for Research and Continuing Education, Shenzhen, China; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Hong Kong, China.
Fu XQ; Research and Development Centre for Natural Health Products, HKBU Institute for Research and Continuing Education, Shenzhen, China; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Hong Kong, China.
Wang RJ; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Hong Kong, China.
Deng YY; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Hong Kong, China.
Wang S; Dalian Fusheng Natural Medicine Research Institute, China.
Yu HX; Jilin Yatai Traditional Chinese Medicine Innovation Research Institute, China.
Liang C; Division of Life Science and State Key Lab of Molecular Neuroscience, Hong Kong University of Science and Technology, China.
Yu ZL; Research and Development Centre for Natural Health Products, HKBU Institute for Research and Continuing Education, Shenzhen, China; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Hong Kong, China. Electronic address: .
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Źródło :
Phytomedicine : international journal of phytotherapy and phytopharmacology [Phytomedicine] 2022 Jul 20; Vol. 102, pp. 154194. Date of Electronic Publication: 2022 May 23.
Typ publikacji :
Journal Article
MeSH Terms :
STAT3 Transcription Factor*/metabolism
Vernonia*
Animals ; Anti-Inflammatory Agents/pharmacology ; Anti-Inflammatory Agents/therapeutic use ; Edema/drug therapy ; Ethanol ; Extracellular Signal-Regulated MAP Kinases/metabolism ; Inflammation/chemically induced ; Inflammation/drug therapy ; Interleukin-6/metabolism ; Lipopolysaccharides/pharmacology ; Mice ; NF-kappa B/metabolism ; Nitric Oxide/metabolism ; Nitric Oxide Synthase Type II/metabolism ; Plant Extracts/therapeutic use ; RNA, Messenger ; Rats ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Tytuł :
Silencing GOLGA8B inhibits cell invasion and metastasis by suppressing STAT3 signaling pathway in lung squamous cell carcinoma.
Autorzy :
Li Z; Department of Oncology, Xiangya Hospital, Central South University, Changsha, Hunan Province 410008, China.
Li Y; Department of Nursing, Xiangya Hospital, Central South University, Changsha, Hunan Province 410008, China.
Li N; Department of Oncology, Xiangya Hospital, Central South University, Changsha, Hunan Province 410008, China.
Shen L; Department of Oncology, Xiangya Hospital, Central South University, Changsha, Hunan Province 410008, China.
Liu A; Department of Geriatrics, Xiangya Hospital, Central South University, Changsha, Hunan Province 410008, China.; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha 410008, China.
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Źródło :
Clinical science (London, England : 1979) [Clin Sci (Lond)] 2022 Jun 17; Vol. 136 (11), pp. 895-909.
Typ publikacji :
Journal Article
MeSH Terms :
Carcinoma, Non-Small-Cell Lung*/metabolism
Carcinoma, Squamous Cell*/genetics
Carcinoma, Squamous Cell*/pathology
Golgi Matrix Proteins*/metabolism
Lung Neoplasms*/metabolism
STAT3 Transcription Factor*/genetics
STAT3 Transcription Factor*/metabolism
Animals ; Gene Expression Regulation, Neoplastic ; Humans ; Lung/metabolism ; Mice ; Neoplasm Invasiveness/genetics ; Neoplasm Metastasis/genetics ; Signal Transduction
Czasopismo naukowe
Tytuł :
T cell clonal expansion and STAT3 mutations: a characteristic feature of acquired chronic T cell-mediated pure red cell aplasia.
Autorzy :
Kawakami F; Division of Hematology, Department of Internal Medicine, Shinshu University School of Medicine, 3-1-1 Asahi, Matsumoto, Nagano, 3908621, Japan.
Kawakami T; Division of Hematology, Department of Internal Medicine, Shinshu University School of Medicine, 3-1-1 Asahi, Matsumoto, Nagano, 3908621, Japan.
Yamane T; Department of Clinical Laboratory Investigation, Graduate School of Medicine, Shinshu University, Matsumoto, Japan.; Department of Laboratory Medicine, Shinshu University Hospital, Matsumoto, Japan.
Maruyama M; Department of Clinical Laboratory Investigation, Graduate School of Medicine, Shinshu University, Matsumoto, Japan.
Kobayashi J; Department of Laboratory Medicine, Nagano Children's Hospital, Azumino, Japan.
Nishina S; Division of Hematology, Department of Internal Medicine, Shinshu University School of Medicine, 3-1-1 Asahi, Matsumoto, Nagano, 3908621, Japan.
Sakai H; Division of Hematology, Department of Internal Medicine, Shinshu University School of Medicine, 3-1-1 Asahi, Matsumoto, Nagano, 3908621, Japan.
Higuchi Y; Department of Clinical Laboratory Investigation, Graduate School of Medicine, Shinshu University, Matsumoto, Japan.; Department of Biomedical Laboratory Sciences, Shinshu University School of Medicine, 3-1-1 Asahi, Matsumoto, Nagano, 3908621, Japan.
Hamanaka K; Department of Thoracic Surgery, Shinshu University School of Medicine, Matsumoto, Japan.
Hirokawa M; Department of General Internal Medicine and Clinical Laboratory Medicine Graduate School of Medicine, Akita University, Akita, Japan.
Nakao S; Department of Hematology, Faculty of Medicine Science, Institute of Medical, Pharmaceutical and Health Sciences, Kanazawa University, Kanazawa, Japan.
Nakazawa H; Division of Hematology, Department of Internal Medicine, Shinshu University School of Medicine, 3-1-1 Asahi, Matsumoto, Nagano, 3908621, Japan. .
Ishida F; Division of Hematology, Department of Internal Medicine, Shinshu University School of Medicine, 3-1-1 Asahi, Matsumoto, Nagano, 3908621, Japan. .; Department of Clinical Laboratory Investigation, Graduate School of Medicine, Shinshu University, Matsumoto, Japan. .; Department of Biomedical Laboratory Sciences, Shinshu University School of Medicine, 3-1-1 Asahi, Matsumoto, Nagano, 3908621, Japan. .
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Źródło :
International journal of hematology [Int J Hematol] 2022 Jun; Vol. 115 (6), pp. 816-825. Date of Electronic Publication: 2022 Mar 11.
Typ publikacji :
Journal Article
MeSH Terms :
Leukemia, Large Granular Lymphocytic*/genetics
Leukemia, Large Granular Lymphocytic*/immunology
Leukemia, Large Granular Lymphocytic*/pathology
Red-Cell Aplasia, Pure*/genetics
Red-Cell Aplasia, Pure*/immunology
Red-Cell Aplasia, Pure*/pathology
STAT3 Transcription Factor*/genetics
STAT3 Transcription Factor*/immunology
Thymoma*/genetics
Thymoma*/immunology
Thymus Neoplasms*/immunology
CD8-Positive T-Lymphocytes/pathology ; Humans ; Mutation ; Receptors, Antigen, T-Cell/genetics ; Receptors, Antigen, T-Cell/immunology
Czasopismo naukowe
Tytuł :
Luteolin binds Src, promotes STAT3 protein ubiquitination and exerts anti-melanoma effects in cell and mouse models.
Autorzy :
Li T; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Kowloon Tong, Hong Kong, China.
Fu X; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Kowloon Tong, Hong Kong, China.
Liu B; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Kowloon Tong, Hong Kong, China.
Wang X; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Kowloon Tong, Hong Kong, China.
Li J; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Kowloon Tong, Hong Kong, China.
Zhu P; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Kowloon Tong, Hong Kong, China.
Niu X; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Kowloon Tong, Hong Kong, China.
Bai J; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Kowloon Tong, Hong Kong, China.
Liu Y; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Kowloon Tong, Hong Kong, China.
Lu X; Shaanxi Engineering Laboratory for Food Green Processing and Safety Control, College of Food Engineering and Nutritional Science, Shaanxi Normal University, Xi'an, China.
Yu ZL; Consun Chinese Medicines Research Centre for Renal Diseases, School of Chinese Medicine, Hong Kong Baptist University, Kowloon Tong, Hong Kong, China; Research and Development Centre for Natural Health Products, HKBU Institute for Research and Continuing Education, Shenzhen, China. Electronic address: .
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Źródło :
Biochemical pharmacology [Biochem Pharmacol] 2022 Jun; Vol. 200, pp. 115044. Date of Electronic Publication: 2022 Apr 20.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Luteolin*/pharmacology
Melanoma*/drug therapy
STAT3 Transcription Factor*/metabolism
Animals ; Apoptosis ; Cell Line, Tumor ; Disease Models, Animal ; Humans ; Mice ; Proto-Oncogene Proteins pp60(c-src)/metabolism ; Ubiquitination
Czasopismo naukowe
Tytuł :
Cryptotanshinone ameliorates MPP -induced oxidative stress and apoptosis of SH-SY5Y neuroblastoma cells: the role of STAT3 in Parkinson's disease.
Autorzy :
Wang Q; Department of Pharmacy, The Third Affiliated Hospital of Baotou Medical College, No.16 Tuanjie Street, Qingshan District, Baotou City, 014030, Inner Mongolia, China.
Liu Y; Department of Pharmacy, The Third Affiliated Hospital of Baotou Medical College, No.16 Tuanjie Street, Qingshan District, Baotou City, 014030, Inner Mongolia, China. .
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Źródło :
Metabolic brain disease [Metab Brain Dis] 2022 Jun; Vol. 37 (5), pp. 1477-1485. Date of Electronic Publication: 2022 Apr 09.
Typ publikacji :
Journal Article
MeSH Terms :
Neuroblastoma*
Parkinson Disease*/drug therapy
Parkinson Disease*/metabolism
STAT3 Transcription Factor/*metabolism
1-Methyl-4-phenylpyridinium/toxicity ; Animals ; Apoptosis ; Cell Line, Tumor ; Cell Survival ; Humans ; Mice ; Oxidative Stress ; Phenanthrenes ; STAT3 Transcription Factor/pharmacology ; STAT3 Transcription Factor/therapeutic use
Czasopismo naukowe
Tytuł :
Downregulation of STK25 promotes autophagy via the Janus kinase 2/signal transducer and activator of transcription 3 pathway in colorectal cancer.
Autorzy :
Chen J; Department of Gastrointestinal Surgery IV, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Peking University Cancer Hospital & Institute, Beijing, China.
Gao P; Department of Gastrointestinal Surgery IV, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Peking University Cancer Hospital & Institute, Beijing, China.
Peng L; Department of Gastrointestinal Surgery IV, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Peking University Cancer Hospital & Institute, Beijing, China.
Liu T; Department of Gastrointestinal Surgery IV, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Peking University Cancer Hospital & Institute, Beijing, China.
Wu F; Inner Mongolia People's Hospital, Hohhot, China.
Xu K; Department of Gastrointestinal Surgery IV, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Peking University Cancer Hospital & Institute, Beijing, China.
Chen L; Department of Gastrointestinal Surgery IV, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Peking University Cancer Hospital & Institute, Beijing, China.
Tan F; Department of Gastrointestinal Surgery IV, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Peking University Cancer Hospital & Institute, Beijing, China.
Xing P; Department of Gastrointestinal Surgery IV, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Peking University Cancer Hospital & Institute, Beijing, China.
Wang Z; Department of Gastrointestinal Surgery IV, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Peking University Cancer Hospital & Institute, Beijing, China.
Di J; Department of Gastrointestinal Surgery IV, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Peking University Cancer Hospital & Institute, Beijing, China.
Jiang B; Department of Gastrointestinal Surgery IV, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Peking University Cancer Hospital & Institute, Beijing, China.
Su X; Department of Gastrointestinal Surgery IV, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education), Peking University Cancer Hospital & Institute, Beijing, China.
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Źródło :
Molecular carcinogenesis [Mol Carcinog] 2022 Jun; Vol. 61 (6), pp. 572-586. Date of Electronic Publication: 2022 Mar 29.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Colorectal Neoplasms*/pathology
STAT3 Transcription Factor*/genetics
STAT3 Transcription Factor*/metabolism
Autophagy/genetics ; Cell Line, Tumor ; Down-Regulation ; Humans ; Intracellular Signaling Peptides and Proteins/metabolism ; Janus Kinase 2/genetics ; Janus Kinase 2/metabolism ; Protein Serine-Threonine Kinases/genetics ; Signal Transduction
Czasopismo naukowe
Tytuł :
A Novel STAT3 Gain-of-Function Mutation in Fatal Infancy-Onset Interstitial Lung Disease.
Autorzy :
Deng M; Department of Pediatric Research Institute, Children's Hospital of Chongqing Medical Univeristy, National Clinical Research Center for Child Health and Disorders, Ministry of Education Key Laboratory of Child Development and Disorders, Chongqing Key Laboratory of Child Infection and Immunity, Chongqing, China.
Li Y; Department of Pediatric Research Institute, Children's Hospital of Chongqing Medical Univeristy, National Clinical Research Center for Child Health and Disorders, Ministry of Education Key Laboratory of Child Development and Disorders, Chongqing Key Laboratory of Child Infection and Immunity, Chongqing, China.
Li Y; Department of Pediatric Research Institute, Children's Hospital of Chongqing Medical Univeristy, National Clinical Research Center for Child Health and Disorders, Ministry of Education Key Laboratory of Child Development and Disorders, Chongqing Key Laboratory of Child Infection and Immunity, Chongqing, China.
Mao X; Department of Pediatric Research Institute, Children's Hospital of Chongqing Medical Univeristy, National Clinical Research Center for Child Health and Disorders, Ministry of Education Key Laboratory of Child Development and Disorders, Chongqing Key Laboratory of Child Infection and Immunity, Chongqing, China.
Ke H; Beijing National Laboratory for Molecular Sciences, Key Laboratory of Bioorganic Chemistry and Molecular Engineering of Ministry of Education, Department of Chemical Biology, College of Chemistry and Molecular Engineering, Synthetic and Functional Biomolecules Center, and Peking-Tsinghua Center for Life Sciences, Peking University, Beijing, China.
Liang W; Department of Pediatrics, The University of Hong Kong-Shenzhen Hospital, Shenzhen, China.
Lei X; Beijing National Laboratory for Molecular Sciences, Key Laboratory of Bioorganic Chemistry and Molecular Engineering of Ministry of Education, Department of Chemical Biology, College of Chemistry and Molecular Engineering, Synthetic and Functional Biomolecules Center, and Peking-Tsinghua Center for Life Sciences, Peking University, Beijing, China.
Lau YL; Department of Pediatrics, The University of Hong Kong-Shenzhen Hospital, Shenzhen, China.; Department of Paediatrics and Adolescent Medicine, The University of Hong Kong, Hong Kong, Hong Kong SAR, China.
Mao H; Department of Pediatric Research Institute, Children's Hospital of Chongqing Medical Univeristy, National Clinical Research Center for Child Health and Disorders, Ministry of Education Key Laboratory of Child Development and Disorders, Chongqing Key Laboratory of Child Infection and Immunity, Chongqing, China.
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Źródło :
Frontiers in immunology [Front Immunol] 2022 May 23; Vol. 13, pp. 866638. Date of Electronic Publication: 2022 May 23 (Print Publication: 2022).
Typ publikacji :
Case Reports; Journal Article
MeSH Terms :
Gain of Function Mutation*
Lung Diseases, Interstitial*/diagnosis
Lung Diseases, Interstitial*/genetics
STAT3 Transcription Factor*/genetics
Autoimmunity ; Humans ; Infant ; Mutation
Czasopismo naukowe
Tytuł :
β-Elemene Attenuates Renal Fibrosis in the Unilateral Ureteral Obstruction Model by Inhibition of STAT3 and Smad3 Signaling via Suppressing MyD88 Expression.
Autorzy :
Sun W; Department of Internal Medicine, Chonnam National University Medical School, 42 Jebongro, Gwangju 61469, Korea.
Kim DH; Department of Internal Medicine, Chonnam National University Medical School, 42 Jebongro, Gwangju 61469, Korea.
Byon CH; Department of Internal Medicine, Chonnam National University Medical School, 42 Jebongro, Gwangju 61469, Korea.
Choi HI; Department of Internal Medicine, Chonnam National University Medical School, 42 Jebongro, Gwangju 61469, Korea.
Park JS; Department of Internal Medicine, Chonnam National University Medical School, 42 Jebongro, Gwangju 61469, Korea.
Bae EH; Department of Internal Medicine, Chonnam National University Medical School, 42 Jebongro, Gwangju 61469, Korea.
Ma SK; Department of Internal Medicine, Chonnam National University Medical School, 42 Jebongro, Gwangju 61469, Korea.
Kim SW; Department of Internal Medicine, Chonnam National University Medical School, 42 Jebongro, Gwangju 61469, Korea.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2022 May 16; Vol. 23 (10). Date of Electronic Publication: 2022 May 16.
Typ publikacji :
Journal Article
MeSH Terms :
Myeloid Differentiation Factor 88*/metabolism
Renal Insufficiency, Chronic*/metabolism
STAT3 Transcription Factor*/antagonists & inhibitors
STAT3 Transcription Factor*/metabolism
Sesquiterpenes*/pharmacology
Smad3 Protein*/antagonists & inhibitors
Smad3 Protein*/metabolism
Ureteral Obstruction*/complications
Ureteral Obstruction*/drug therapy
Ureteral Obstruction*/metabolism
Adaptor Proteins, Signal Transducing/metabolism ; Animals ; Cell Line ; Fibrosis ; Mice ; Rats ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Tytuł :
MS: Wip1 suppresses angiogenesis through the STAT3-VEGF signalling pathway in serous ovarian cancer.
Autorzy :
Yin S; Department of Gynaecologic Oncology, Ovarian Cancer Program, Zhongshan Hospital, Fudan University, Shanghai, China.; Department of Obstetrics and Gynecology, Zhongshan Hospital, Fudan University, Shanghai, China.
Yang L; Department of Obstetrics and Gynecology, Shanghai Fifth People's Hospital, Fudan University, Shanghai, China.
Zheng Y; Department of Gynaecologic Oncology, Ovarian Cancer Program, Zhongshan Hospital, Fudan University, Shanghai, China.
Zang R; Department of Gynaecologic Oncology, Ovarian Cancer Program, Zhongshan Hospital, Fudan University, Shanghai, China. .
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Źródło :
Journal of ovarian research [J Ovarian Res] 2022 May 10; Vol. 15 (1), pp. 56. Date of Electronic Publication: 2022 May 10.
Typ publikacji :
Journal Article
MeSH Terms :
Cystadenocarcinoma, Serous*/blood supply
Cystadenocarcinoma, Serous*/genetics
Cystadenocarcinoma, Serous*/metabolism
Ovarian Neoplasms*/blood supply
Ovarian Neoplasms*/pathology
Protein Phosphatase 2C*/genetics
Protein Phosphatase 2C*/metabolism
STAT3 Transcription Factor*/genetics
STAT3 Transcription Factor*/metabolism
Vascular Endothelial Growth Factor A*/genetics
Vascular Endothelial Growth Factor A*/metabolism
Carcinoma, Ovarian Epithelial/blood supply ; Carcinoma, Ovarian Epithelial/genetics ; Carcinoma, Ovarian Epithelial/metabolism ; Cell Line, Tumor ; Female ; Humans ; Neovascularization, Pathologic/genetics ; Signal Transduction
Czasopismo naukowe
Tytuł :
Renal cell carcinoma-derived exosomes deliver lncARSR to induce macrophage polarization and promote tumor progression via STAT3 pathway.
Autorzy :
Zhang W; Department of Urology, Tongde Hospital of Zhejiang Province, Hangzhou, Zhejiang 310012, China.
Zheng X; Cancer Institute of Integrated Traditional Chinese and Western Medicine, Key laboratory of cancer prevention and therapy combining traditional Chinese and Western Medicine of Zhejiang Province, Zhejiang Academy of Traditional Chinese Medicine, Tongde Hospital of Zhejiang Province, Hangzhou, Zhejiang 310012, China.
Yu Y; College of Life Sciences, Zhejiang Chinese Medical University, Hangzhou, 310053, China.
Zheng L; Cancer Institute of Integrated Traditional Chinese and Western Medicine, Key laboratory of cancer prevention and therapy combining traditional Chinese and Western Medicine of Zhejiang Province, Zhejiang Academy of Traditional Chinese Medicine, Tongde Hospital of Zhejiang Province, Hangzhou, Zhejiang 310012, China.
Lan J; Cancer Institute of Integrated Traditional Chinese and Western Medicine, Key laboratory of cancer prevention and therapy combining traditional Chinese and Western Medicine of Zhejiang Province, Zhejiang Academy of Traditional Chinese Medicine, Tongde Hospital of Zhejiang Province, Hangzhou, Zhejiang 310012, China.
Wu Y; Cancer Institute of Integrated Traditional Chinese and Western Medicine, Key laboratory of cancer prevention and therapy combining traditional Chinese and Western Medicine of Zhejiang Province, Zhejiang Academy of Traditional Chinese Medicine, Tongde Hospital of Zhejiang Province, Hangzhou, Zhejiang 310012, China.
Liu H; Cancer Institute of Integrated Traditional Chinese and Western Medicine, Key laboratory of cancer prevention and therapy combining traditional Chinese and Western Medicine of Zhejiang Province, Zhejiang Academy of Traditional Chinese Medicine, Tongde Hospital of Zhejiang Province, Hangzhou, Zhejiang 310012, China.
Zhao A; Department of Tumor Research Institute, Cancer Hospital of the University of Chinese Academy of Sciences, Zhejiang Cancer Hospital, Hangzhou, China.
Huang H; Department of Urology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou 325000, China.
Chen W; Cancer Institute of Integrated Traditional Chinese and Western Medicine, Key laboratory of cancer prevention and therapy combining traditional Chinese and Western Medicine of Zhejiang Province, Zhejiang Academy of Traditional Chinese Medicine, Tongde Hospital of Zhejiang Province, Hangzhou, Zhejiang 310012, China.
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Źródło :
International journal of biological sciences [Int J Biol Sci] 2022 May 01; Vol. 18 (8), pp. 3209-3222. Date of Electronic Publication: 2022 May 01 (Print Publication: 2022).
Typ publikacji :
Journal Article
MeSH Terms :
Carcinoma, Renal Cell*/metabolism
Exosomes*/genetics
Exosomes*/metabolism
Kidney Neoplasms*/metabolism
RNA, Long Noncoding*/genetics
STAT3 Transcription Factor*/genetics
STAT3 Transcription Factor*/metabolism
Humans ; Macrophages/metabolism ; MicroRNAs/genetics ; MicroRNAs/metabolism ; Tumor Microenvironment
Czasopismo naukowe
Tytuł :
CircZNF532 knockdown protects retinal pigment epithelial cells against high glucose-induced apoptosis and pyroptosis by regulating the miR-20b-5p/STAT3 axis.
Autorzy :
Liang GH; Department of Ophthalmology, The Affiliated Hospital of Youjiang Medical University for Nationalities, Baise, China.
Luo YN; Department of Ophthalmology, The Affiliated Hospital of Youjiang Medical University for Nationalities, Baise, China.
Wei RZ; Department of Ophthalmology, The Affiliated Hospital of Youjiang Medical University for Nationalities, Baise, China.
Yin JY; Department of Ophthalmology, The First Affiliated Hospital of Jinan University, Guangzhou, China.
Qin ZL; Department of Ophthalmology, The Affiliated Hospital of Youjiang Medical University for Nationalities, Baise, China.
Lu LL; Department of Ophthalmology, The Affiliated Hospital of Youjiang Medical University for Nationalities, Baise, China.
Ma WH; Department of Ophthalmology, The Affiliated Hospital of Youjiang Medical University for Nationalities, Baise, China.
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Źródło :
Journal of diabetes investigation [J Diabetes Investig] 2022 May; Vol. 13 (5), pp. 781-795. Date of Electronic Publication: 2022 Feb 11.
Typ publikacji :
Journal Article
MeSH Terms :
Diabetic Retinopathy*/genetics
Diabetic Retinopathy*/metabolism
MicroRNAs*/genetics
MicroRNAs*/metabolism
RNA, Circular*/genetics
RNA, Circular*/metabolism
Retinal Pigment Epithelium*/cytology
STAT3 Transcription Factor*/genetics
STAT3 Transcription Factor*/metabolism
Animals ; Apoptosis/genetics ; Apoptosis/physiology ; Epithelial Cells/metabolism ; Glucose/metabolism ; Humans ; Mice ; Pyroptosis/genetics ; Pyroptosis/physiology
Czasopismo naukowe
Tytuł :
Dapagliflozin protects against doxorubicin-induced cardiotoxicity by restoring STAT3.
Autorzy :
Chang WT; Institute of Clinical Medicine, College of Medicine, National Cheng Kung University, Tainan, Taiwan.; Division of Cardiology, Department of Internal Medicine, Chi-Mei Medical Center, Tainan, Taiwan.; Department of Biotechnology, Southern Taiwan University of Science and Technology, Tainan, Taiwan.
Shih JY; Division of Cardiology, Department of Internal Medicine, Chi-Mei Medical Center, Tainan, Taiwan.; Department of Health and Nutrition, Chia Nan University of Pharmacy and Science, Tainan, Taiwan.
Lin YW; Division of Cardiology, Department of Internal Medicine, Chi-Mei Medical Center, Tainan, Taiwan.
Chen ZC; Division of Cardiology, Department of Internal Medicine, Chi-Mei Medical Center, Tainan, Taiwan.
Kan WC; Division of Nephrology, Department of Internal Medicine, Chi Mei Medical Center, 901, Zhonghua Road, Yongkang District, Tainan, Taiwan ROC. .; Department of Biological Science and Technology, Chung Hwa University of Medical Technology, Tainan, Taiwan. .
Lin TH; Faculty of Medicine, College of Medicine, Kaohsiung Medical University, Kaohsiung, Taiwan.; Division of Cardiology, Department of Internal Medicine, Kaohsiung Medical University Hospital, Kaohsiung, Taiwan.
Hong CS; Division of Cardiology, Department of Internal Medicine, Chi-Mei Medical Center, Tainan, Taiwan.
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Źródło :
Archives of toxicology [Arch Toxicol] 2022 Jul; Vol. 96 (7), pp. 2021-2032. Date of Electronic Publication: 2022 Apr 19.
Typ publikacji :
Journal Article
MeSH Terms :
Cardiotoxicity*/metabolism
STAT3 Transcription Factor*/metabolism
Animals ; Apoptosis ; Apoptosis Regulatory Proteins/metabolism ; Benzhydryl Compounds ; Doxorubicin/toxicity ; Glucosides ; Humans ; Myocytes, Cardiac ; Oxidative Stress ; Rats ; Rats, Sprague-Dawley ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Tytuł :
ALKBH5 regulates STAT3 activity to affect the proliferation and tumorigenicity of osteosarcoma via an m6A-YTHDF2-dependent manner.
Autorzy :
Yang Z; Department of Orthopedics, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, 1095#, Jiefang Ave, Wuhan 430030, China; Hubei Key Laboratory of Orthopedics, Wuhan 430030, China.
Cai Z; Department of Orthopedics, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, 1095#, Jiefang Ave, Wuhan 430030, China; Hubei Key Laboratory of Orthopedics, Wuhan 430030, China.
Yang C; Department of Orthopedics, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, 1095#, Jiefang Ave, Wuhan 430030, China; Hubei Key Laboratory of Orthopedics, Wuhan 430030, China.
Luo Z; Department of Orthopedics, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, 1095#, Jiefang Ave, Wuhan 430030, China; Hubei Key Laboratory of Orthopedics, Wuhan 430030, China. Electronic address: .
Bao X; Department of Orthopedics, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, 1095#, Jiefang Ave, Wuhan 430030, China; Hubei Key Laboratory of Orthopedics, Wuhan 430030, China. Electronic address: .
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Źródło :
EBioMedicine [EBioMedicine] 2022 Jun; Vol. 80, pp. 104019. Date of Electronic Publication: 2022 Apr 28.
Typ publikacji :
Journal Article
MeSH Terms :
AlkB Homolog 5, RNA Demethylase*/genetics
Bone Neoplasms*/genetics
Osteosarcoma*/genetics
RNA-Binding Proteins*/genetics
STAT3 Transcription Factor*/metabolism
Adenosine/*analogs & derivatives
Adenosine/genetics ; Cell Proliferation/genetics ; Humans ; RNA/genetics ; RNA, Messenger/genetics
Czasopismo naukowe
Tytuł :
Blocking STAT3 signaling augments MEK/ERK inhibitor efficacy in esophageal squamous cell carcinoma.
Autorzy :
Zheng ZY; The Key Laboratory of Molecular Biology for High Cancer Incidence Coastal Chaoshan Area, Department of Biochemistry and Molecular Biology, Shantou University Medical College, Shantou, 515041, Guangdong, China.; Guangdong Provincial Key Laboratory of Infectious Diseases and Molecular Immunopathology, Institute of Oncologic Pathology, Shantou University Medical College, Shantou University Medical College, Shantou, 515041, Guangdong, China.; Guangdong Esophageal Cancer Research Institute, Shantou Sub-center, Cancer Research Center, Shantou University Medical College, Shantou, 515041, Guangdong, China.
Chu MY; The Key Laboratory of Molecular Biology for High Cancer Incidence Coastal Chaoshan Area, Department of Biochemistry and Molecular Biology, Shantou University Medical College, Shantou, 515041, Guangdong, China.; Guangdong Provincial Key Laboratory of Infectious Diseases and Molecular Immunopathology, Institute of Oncologic Pathology, Shantou University Medical College, Shantou University Medical College, Shantou, 515041, Guangdong, China.
Lin W; The Key Laboratory of Molecular Biology for High Cancer Incidence Coastal Chaoshan Area, Department of Biochemistry and Molecular Biology, Shantou University Medical College, Shantou, 515041, Guangdong, China.; Guangdong Provincial Key Laboratory of Infectious Diseases and Molecular Immunopathology, Institute of Oncologic Pathology, Shantou University Medical College, Shantou University Medical College, Shantou, 515041, Guangdong, China.
Zheng YQ; The Key Laboratory of Molecular Biology for High Cancer Incidence Coastal Chaoshan Area, Department of Biochemistry and Molecular Biology, Shantou University Medical College, Shantou, 515041, Guangdong, China.; Guangdong Provincial Key Laboratory of Infectious Diseases and Molecular Immunopathology, Institute of Oncologic Pathology, Shantou University Medical College, Shantou University Medical College, Shantou, 515041, Guangdong, China.
Xu XE; The Key Laboratory of Molecular Biology for High Cancer Incidence Coastal Chaoshan Area, Department of Biochemistry and Molecular Biology, Shantou University Medical College, Shantou, 515041, Guangdong, China.; Guangdong Provincial Key Laboratory of Infectious Diseases and Molecular Immunopathology, Institute of Oncologic Pathology, Shantou University Medical College, Shantou University Medical College, Shantou, 515041, Guangdong, China.
Chen Y; The Key Laboratory of Molecular Biology for High Cancer Incidence Coastal Chaoshan Area, Department of Biochemistry and Molecular Biology, Shantou University Medical College, Shantou, 515041, Guangdong, China.; Guangdong Provincial Key Laboratory of Infectious Diseases and Molecular Immunopathology, Institute of Oncologic Pathology, Shantou University Medical College, Shantou University Medical College, Shantou, 515041, Guangdong, China.
Liao LD; The Key Laboratory of Molecular Biology for High Cancer Incidence Coastal Chaoshan Area, Department of Biochemistry and Molecular Biology, Shantou University Medical College, Shantou, 515041, Guangdong, China.; Guangdong Provincial Key Laboratory of Infectious Diseases and Molecular Immunopathology, Institute of Oncologic Pathology, Shantou University Medical College, Shantou University Medical College, Shantou, 515041, Guangdong, China.
Wu ZY; Shantou Central Hospital, Affiliated Shantou Hospital of Sun Yat-Sen University, Shantou, 515041, Guangdong, China.
Wang SH; Shantou Central Hospital, Affiliated Shantou Hospital of Sun Yat-Sen University, Shantou, 515041, Guangdong, China.
Li EM; The Key Laboratory of Molecular Biology for High Cancer Incidence Coastal Chaoshan Area, Department of Biochemistry and Molecular Biology, Shantou University Medical College, Shantou, 515041, Guangdong, China. .; Guangdong Esophageal Cancer Research Institute, Shantou Sub-center, Cancer Research Center, Shantou University Medical College, Shantou, 515041, Guangdong, China. .
Xu LY; The Key Laboratory of Molecular Biology for High Cancer Incidence Coastal Chaoshan Area, Department of Biochemistry and Molecular Biology, Shantou University Medical College, Shantou, 515041, Guangdong, China. .; Guangdong Provincial Key Laboratory of Infectious Diseases and Molecular Immunopathology, Institute of Oncologic Pathology, Shantou University Medical College, Shantou University Medical College, Shantou, 515041, Guangdong, China. .; Guangdong Esophageal Cancer Research Institute, Shantou Sub-center, Cancer Research Center, Shantou University Medical College, Shantou, 515041, Guangdong, China. .
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Źródło :
Cell death & disease [Cell Death Dis] 2022 May 25; Vol. 13 (5), pp. 496. Date of Electronic Publication: 2022 May 25.
Typ publikacji :
Journal Article
MeSH Terms :
Esophageal Neoplasms*/drug therapy
Esophageal Neoplasms*/genetics
Esophageal Neoplasms*/metabolism
Esophageal Squamous Cell Carcinoma*/drug therapy
Esophageal Squamous Cell Carcinoma*/genetics
Esophageal Squamous Cell Carcinoma*/metabolism
MAP Kinase Signaling System*/drug effects
Mitogen-Activated Protein Kinase Kinases*/antagonists & inhibitors
Mitogen-Activated Protein Kinase Kinases*/metabolism
Protein Kinase Inhibitors*/pharmacology
STAT3 Transcription Factor*/antagonists & inhibitors
STAT3 Transcription Factor*/genetics
STAT3 Transcription Factor*/metabolism
Cell Line, Tumor ; Humans ; Signal Transduction/drug effects ; Suppressor of Cytokine Signaling Proteins/metabolism
Czasopismo naukowe
Tytuł :
Interleukin 13 promotes long-term recovery after ischemic stroke by inhibiting the activation of STAT3.
Autorzy :
Chen D; State Key Laboratory of Medical Neurobiology, MOE Frontiers Center for Brain Science, and Institutes of Brain Science, Fudan University, 138 Yixueyuan Road, 200032, Shanghai, China.
Li J; State Key Laboratory of Medical Neurobiology, MOE Frontiers Center for Brain Science, and Institutes of Brain Science, Fudan University, 138 Yixueyuan Road, 200032, Shanghai, China.
Huang Y; State Key Laboratory of Medical Neurobiology, MOE Frontiers Center for Brain Science, and Institutes of Brain Science, Fudan University, 138 Yixueyuan Road, 200032, Shanghai, China.
Wei P; State Key Laboratory of Medical Neurobiology, MOE Frontiers Center for Brain Science, and Institutes of Brain Science, Fudan University, 138 Yixueyuan Road, 200032, Shanghai, China.
Miao W; State Key Laboratory of Medical Neurobiology, MOE Frontiers Center for Brain Science, and Institutes of Brain Science, Fudan University, 138 Yixueyuan Road, 200032, Shanghai, China.
Yang Y; State Key Laboratory of Medical Neurobiology, MOE Frontiers Center for Brain Science, and Institutes of Brain Science, Fudan University, 138 Yixueyuan Road, 200032, Shanghai, China.
Gao Y; State Key Laboratory of Medical Neurobiology, MOE Frontiers Center for Brain Science, and Institutes of Brain Science, Fudan University, 138 Yixueyuan Road, 200032, Shanghai, China. .
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Źródło :
Journal of neuroinflammation [J Neuroinflammation] 2022 May 16; Vol. 19 (1), pp. 112. Date of Electronic Publication: 2022 May 16.
Typ publikacji :
Journal Article
MeSH Terms :
Brain Ischemia*/drug therapy
Interleukin-13*/pharmacology
Ischemic Stroke*/drug therapy
STAT3 Transcription Factor*/metabolism
Animals ; Anti-Inflammatory Agents/therapeutic use ; Infarction, Middle Cerebral Artery/complications ; Infarction, Middle Cerebral Artery/drug therapy ; Infarction, Middle Cerebral Artery/metabolism ; Male ; Mice ; Mice, Inbred C57BL ; Microglia/metabolism
Czasopismo naukowe
Tytuł :
Activating IL-6/STAT3 Enhances Protein Stability of Proteasome 20S α + β in Colorectal Cancer by miR-1254.
Autorzy :
Ren W; Department of Gastroenterology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450003, China.
Zhang X; Department of Gastroenterology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450003, China.
Li Q; Oncology Department, Shenzhen Hospital, Southern Medical University, Shenzhen, Guangdong 518101, China.
Pu C; Department of Gastroenterology, Zhongda Hospital, Southeast University, Nanjing, Jiangsu 210009, China.
Zhang D; Department of Gastroenterology, The Third Xiangya Hospital of Central South University, Changsha, Hunan 410000, China.
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Źródło :
BioMed research international [Biomed Res Int] 2022 May 14; Vol. 2022, pp. 4250013. Date of Electronic Publication: 2022 May 14 (Print Publication: 2022).
Typ publikacji :
Journal Article
MeSH Terms :
Colorectal Neoplasms*/metabolism
Colorectal Neoplasms*/pathology
Interleukin-6*/genetics
Interleukin-6*/metabolism
MicroRNAs*/metabolism
Proteasome Endopeptidase Complex*/genetics
Proteasome Endopeptidase Complex*/metabolism
STAT3 Transcription Factor*/genetics
STAT3 Transcription Factor*/metabolism
Animals ; Cell Line, Tumor ; Cell Proliferation ; Gene Expression Regulation, Neoplastic ; Humans ; Mice ; Prospective Studies ; Protein Stability ; Proto-Oncogene Proteins/genetics
Czasopismo naukowe
Tytuł :
Growth differentiation factor 11 induces skeletal muscle atrophy via a STAT3-dependent mechanism in pulmonary arterial hypertension.
Autorzy :
Xiang G; Department of Pulmonary and Critical Care Medicine, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, 200032, China.
Ying K; Department of Pulmonary and Critical Care Medicine, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, 200032, China.
Jiang P; Department of Pulmonary and Critical Care Medicine, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, 200032, China.
Jia M; Department of Pulmonary and Critical Care Medicine, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, 200032, China.
Sun Y; Department of Pulmonary and Critical Care Medicine, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, 200032, China.
Li S; Department of Pulmonary and Critical Care Medicine, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, 200032, China. .
Wu X; Department of Pulmonary and Critical Care Medicine, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, 200032, China. follow_the_.
Hao S; Department of Pulmonary and Critical Care Medicine, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, 200032, China. .
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Źródło :
Skeletal muscle [Skelet Muscle] 2022 May 06; Vol. 12 (1), pp. 10. Date of Electronic Publication: 2022 May 06.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Growth Differentiation Factors*/genetics
Growth Differentiation Factors*/metabolism
Muscular Atrophy*/metabolism
Pulmonary Arterial Hypertension*
STAT3 Transcription Factor*/metabolism
Bone Morphogenetic Proteins/metabolism ; Humans ; Muscle, Skeletal/metabolism ; Muscle, Skeletal/pathology
Czasopismo naukowe
Tytuł :
MicroRNA-382 Promotes M2-Like Macrophage via the SIRP-α/STAT3 Signaling Pathway in Aristolochic Acid-Induced Renal Fibrosis.
Autorzy :
Wang X; Department of Nephrology, Zhongshan Hospital, Fudan University, Shanghai, China.; Shanghai Medical Center of Kidney Disease, Shanghai, China.; Shanghai Institute of Kidney and Dialysis, Shanghai, China.
Jia P; Department of Nephrology, Zhongshan Hospital, Fudan University, Shanghai, China.; Shanghai Medical Center of Kidney Disease, Shanghai, China.; Shanghai Institute of Kidney and Dialysis, Shanghai, China.; Shanghai Key Laboratory of Kidney and Blood Purification, Shanghai, China.
Ren T; Department of Nephrology, Zhongshan Hospital, Fudan University, Shanghai, China.; Shanghai Medical Center of Kidney Disease, Shanghai, China.; Shanghai Institute of Kidney and Dialysis, Shanghai, China.
Zou Z; Department of Nephrology, Zhongshan Hospital, Fudan University, Shanghai, China.; Shanghai Medical Center of Kidney Disease, Shanghai, China.; Shanghai Institute of Kidney and Dialysis, Shanghai, China.
Xu S; Department of Nephrology, Zhongshan Hospital, Fudan University, Shanghai, China.; Shanghai Medical Center of Kidney Disease, Shanghai, China.; Shanghai Institute of Kidney and Dialysis, Shanghai, China.
Zhang Y; Department of Nephrology, Zhongshan Hospital, Fudan University, Shanghai, China.; Shanghai Medical Center of Kidney Disease, Shanghai, China.; Shanghai Institute of Kidney and Dialysis, Shanghai, China.
Shi Y; Department of Nephrology, Zhongshan Hospital, Fudan University, Shanghai, China.; Shanghai Medical Center of Kidney Disease, Shanghai, China.; Shanghai Institute of Kidney and Dialysis, Shanghai, China.; Shanghai Key Laboratory of Kidney and Blood Purification, Shanghai, China.
Bao S; Department of Nephrology, Zhongshan Hospital, Fudan University, Shanghai, China.; Shanghai Medical Center of Kidney Disease, Shanghai, China.; Shanghai Institute of Kidney and Dialysis, Shanghai, China.
Li Y; Department of Nephrology, Zhongshan Hospital, Fudan University, Shanghai, China.; Shanghai Medical Center of Kidney Disease, Shanghai, China.; Shanghai Institute of Kidney and Dialysis, Shanghai, China.
Fang Y; Department of Nephrology, Zhongshan Hospital, Fudan University, Shanghai, China.; Shanghai Medical Center of Kidney Disease, Shanghai, China.; Shanghai Institute of Kidney and Dialysis, Shanghai, China.; Shanghai Key Laboratory of Kidney and Blood Purification, Shanghai, China.
Ding X; Department of Nephrology, Zhongshan Hospital, Fudan University, Shanghai, China.; Shanghai Medical Center of Kidney Disease, Shanghai, China.; Shanghai Institute of Kidney and Dialysis, Shanghai, China.; Shanghai Key Laboratory of Kidney and Blood Purification, Shanghai, China.
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Źródło :
Frontiers in immunology [Front Immunol] 2022 May 02; Vol. 13, pp. 864984. Date of Electronic Publication: 2022 May 02 (Print Publication: 2022).
Typ publikacji :
Journal Article
MeSH Terms :
Kidney Diseases*/chemically induced
Kidney Diseases*/metabolism
Macrophages*/metabolism
MicroRNAs*/genetics
MicroRNAs*/metabolism
Receptors, Immunologic*
STAT3 Transcription Factor*/metabolism
Animals ; Aristolochic Acids ; Fibrosis ; Macrophage Activation ; Mice ; Mice, Knockout ; Signal Transduction
Czasopismo naukowe
Tytuł :
Dynamic regulation of O-GlcNAcylation and phosphorylation on STAT3 under hypoxia-induced EMT.
Autorzy :
Fu Y; Institute of Hematology, School of Medicine, Northwest University, Xi'an, China.
Ning L; Shaanxi Provincial Key Laboratory of Papermaking Technology and Specialty Paper Development, College of Bioresources Chemical and Materials Engineering, Shaanxi University of Science & Technology, Xi'an, China.
Feng J; Institute of Hematology, School of Medicine, Northwest University, Xi'an, China.
Yu X; Institute of Hematology, School of Medicine, Northwest University, Xi'an, China.
Guan F; Key Laboratory of Resource Biology and Biotechnology in Western China, Ministry of Education, School of Life Science, Northwest University, Xi'an, China.
Li X; Institute of Hematology, School of Medicine, Northwest University, Xi'an, China. Electronic address: .
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Źródło :
Cellular signalling [Cell Signal] 2022 May; Vol. 93, pp. 110277. Date of Electronic Publication: 2022 Feb 04.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Epithelial-Mesenchymal Transition*/genetics
STAT3 Transcription Factor*/metabolism
Humans ; Hypoxia ; Phosphorylation ; Ubiquitination
Czasopismo naukowe
Tytuł :
Maresin 1 alleviates sevoflurane-induced neuroinflammation in neonatal rats via JAK2/STAT3/IL-6 pathways.
Autorzy :
Wu Y; Department of Anesthesia, Wuhan Children's Hospital (Wuhan Maternal and Child Healthcare Hospital), Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430077, China.
Sun Z; Department of Anesthesia, Wuhan Children's Hospital (Wuhan Maternal and Child Healthcare Hospital), Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430077, China.
Wang T; Department of Anesthesia, Wuhan Children's Hospital (Wuhan Maternal and Child Healthcare Hospital), Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430077, China.
Wang Y; Department of Anesthesia, Wuhan Children's Hospital (Wuhan Maternal and Child Healthcare Hospital), Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430077, China.
Zhong L; Department of Anesthesia, Wuhan Children's Hospital (Wuhan Maternal and Child Healthcare Hospital), Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430077, China.
Kong F; Department of Anesthesia, Wuhan Children's Hospital (Wuhan Maternal and Child Healthcare Hospital), Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430077, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2022 Jul; Vol. 108, pp. 108912. Date of Electronic Publication: 2022 Jun 04.
Typ publikacji :
Journal Article
MeSH Terms :
Janus Kinase 2*/metabolism
STAT3 Transcription Factor*/metabolism
Animals ; Animals, Newborn ; Docosahexaenoic Acids ; Interleukin-6/metabolism ; Neuroinflammatory Diseases ; Rats ; Sevoflurane
Czasopismo naukowe

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