Temat: "STAT5 Transcription Factor" - OpacWWW

Skocz do pozycji: 1.

Tytuł:: Myeloid-CITED2 Deficiency Exacerbates Diet-Induced Obesity and Pro-Inflammatory Macrophage Response.
Autorzy:: Zafar A; Department of Pathology, Case Western Reserve University School of Medicine, Cleveland, OH 44106, USA.
Ng HP; Department of Pathology, Case Western Reserve University School of Medicine, Cleveland, OH 44106, USA.
Chan ER; Cleveland Institute for Computational Biology, Case Western Reserve University School of Medicine, Cleveland, OH 44106, USA.
Dunwoodie SL; Victor Chang Cardiac Research Institute, Sydney, NSW 2010, Australia.; School of Clinical Medicine, Faculty of Medicine and Health, UNSW, Sydney, NSW 2052, Australia.
Mahabeleshwar GH; Department of Pathology, Case Western Reserve University School of Medicine, Cleveland, OH 44106, USA.; Pokaż więcej
Źródło:: Cells [Cells] 2023 Aug 24; Vol. 12 (17). Date of Electronic Publication: 2023 Aug 24.
Typ publikacji:: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:: Insulin Resistance*
Obesity*
Repressor Proteins*/genetics
STAT5 Transcription Factor*
Trans-Activators*/genetics
Diet, High-Fat/adverse effects ; Inflammation ; Macrophages ; Animals

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 2.

Tytuł:: JAK3 Y841 Autophosphorylation Is Critical for STAT5B Activation, Kinase Domain Stability and Dimer Formation.
Autorzy:: Rodriguez G; Department of Biological Sciences, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.; Border Biomedical Research Center, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.
Martinez GS; Department of Biological Sciences, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.; Border Biomedical Research Center, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.
Negrete OD; Department of Biological Sciences, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.; Border Biomedical Research Center, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.
Sun S; Department of Physics, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.; Computational Science Program, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.
Guo W; Department of Physics, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.; Computational Science Program, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.
Xie Y; Department of Physics, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.; Computational Science Program, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.
Li L; Department of Physics, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.; Computational Science Program, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.
Xiao C; Computational Science Program, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.; Department of Biochemistry, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.
Ross JA; Department of Biological Sciences, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.; Border Biomedical Research Center, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.
Kirken RA; Department of Biological Sciences, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.; Border Biomedical Research Center, The University of Texas at El Paso, 500 W University Ave., El Paso, TX 79968, USA.; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2023 Jul 25; Vol. 24 (15). Date of Electronic Publication: 2023 Jul 25.
Typ publikacji:: Journal Article
MeSH Terms:: STAT5 Transcription Factor*/genetics
Signal Transduction*
Phosphorylation ; Janus Kinase 1/genetics ; Protein Processing, Post-Translational ; Tyrosine/metabolism

Czasopismo naukowe

na półce

Pełny tekst Szczegóły

Skocz do pozycji: 3.

Tytuł:: Spermatogenesis associated serine rich 2 like plays a prognostic factor and therapeutic target in acute myeloid leukemia by regulating the JAK2/STAT3/STAT5 axis.
Autorzy:: Li F; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China.; Department of Hematology, The Affiliated People's Hospital of Ningbo University, Baizhang road 251#, Ningbo, China.
Ye W; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China.
Yao Y; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China.
Wei W; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China.
Lin X; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China.
Zhuang H; Department of Hematology, The Affiliated People's Hospital of Ningbo University, Baizhang road 251#, Ningbo, China.
Li C; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China.; Zhejiang Provincial Key Lab of Hematopoietic Malignancy, Zhejiang University, Hangzhou, China.
Li X; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China.; Zhejiang Provincial Key Lab of Hematopoietic Malignancy, Zhejiang University, Hangzhou, China.
Ling Q; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China.
Hu C; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China.; Zhejiang Provincial Key Lab of Hematopoietic Malignancy, Zhejiang University, Hangzhou, China.
Huang X; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China.; Zhejiang Provincial Key Lab of Hematopoietic Malignancy, Zhejiang University, Hangzhou, China.
Qian Y; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China.
Mao S; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China.
Huang J; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China.; Zhejiang Provincial Key Lab of Hematopoietic Malignancy, Zhejiang University, Hangzhou, China.
Lu Y; Department of Hematology, The Affiliated People's Hospital of Ningbo University, Baizhang road 251#, Ningbo, China. .
Jin J; Department of Hematology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Qingchun road 79#, Hangzhou, China. .; Zhejiang Provincial Key Lab of Hematopoietic Malignancy, Zhejiang University, Hangzhou, China. .; Zhejiang University Cancer Center, Hangzhou, China. .; Pokaż więcej
Źródło:: Journal of translational medicine [J Transl Med] 2023 Feb 11; Vol. 21 (1), pp. 115. Date of Electronic Publication: 2023 Feb 11.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Leukemia, Myeloid, Acute*/drug therapy
STAT5 Transcription Factor*/genetics
STAT5 Transcription Factor*/metabolism
STAT5 Transcription Factor*/pharmacology
Animals ; Mice ; Homoharringtonine/pharmacology ; Janus Kinases/metabolism ; Janus Kinases/pharmacology ; Janus Kinases/therapeutic use ; Prognosis ; Signal Transduction ; STAT Transcription Factors/metabolism ; STAT Transcription Factors/pharmacology ; STAT Transcription Factors/therapeutic use ; Humans

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 4.

Tytuł:: IDH2/R140Q mutation confers cytokine-independent proliferation of TF-1 cells by activating constitutive STAT3/5 phosphorylation.
Autorzy:: Yang J; Jiangsu Provincial Medical Innovation Center, Affiliated Hospital of Integrated Traditional Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, 210028, China.
Chen J; Jiangsu Provincial Medical Innovation Center, Affiliated Hospital of Integrated Traditional Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, 210028, China.
Chang J; Jiangsu Provincial Medical Innovation Center, Affiliated Hospital of Integrated Traditional Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, 210028, China.
Sun X; Jiangsu Provincial Medical Innovation Center, Affiliated Hospital of Integrated Traditional Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, 210028, China.
Wei Q; Jiangsu Provincial Medical Innovation Center, Affiliated Hospital of Integrated Traditional Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, 210028, China.
Cai X; Jiangsu Provincial Medical Innovation Center, Affiliated Hospital of Integrated Traditional Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, 210028, China.
Cao P; Jiangsu Provincial Medical Innovation Center, Affiliated Hospital of Integrated Traditional Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, 210028, China. cao_.; The Quzhou Affiliated Hospital of Wenzhou Medical University, Quzhou People's Hospital, Quzhou, 324000, China. cao_.; Zhenjiang Hospital of Chinese Traditional and Western Medicine, Zhenjiang, 212002, China. cao_.; Pokaż więcej
Źródło:: Cell communication and signaling : CCS [Cell Commun Signal] 2024 Feb 12; Vol. 22 (1), pp. 116. Date of Electronic Publication: 2024 Feb 12.
Typ publikacji:: Video-Audio Media; Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Granulocyte-Macrophage Colony-Stimulating Factor*/genetics
Granulocyte-Macrophage Colony-Stimulating Factor*/metabolism
Leukemia, Myeloid, Acute*/genetics
Humans ; STAT5 Transcription Factor/metabolism ; Phosphorylation ; Mutation ; Cell Proliferation ; STAT3 Transcription Factor/metabolism

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 5.

Tytuł:: Phospho-Tudor-SN coordinates with STAT5 to regulate prolactin-stimulated milk protein synthesis and proliferation of bovine mammary epithelial cells.
Autorzy:: Ao J; College of Life Science, Northeast Agricultural University, Heilongjiang, Harbin, China.
Ma Z; College of Life Science, Northeast Agricultural University, Heilongjiang, Harbin, China.
Li R; College of Life Science, Northeast Agricultural University, Heilongjiang, Harbin, China.
Zhang S; College of Life Science, Northeast Agricultural University, Heilongjiang, Harbin, China.
Gao X; College of Animal Science, Yangtze University, Jingzhou, Hubei, China.
Zhang M; College of Life Science, Northeast Agricultural University, Heilongjiang, Harbin, China.; College of Animal Science, Yangtze University, Jingzhou, Hubei, China.; Pokaż więcej
Źródło:: Animal biotechnology [Anim Biotechnol] 2022 Nov; Vol. 33 (6), pp. 1161-1169. Date of Electronic Publication: 2021 Apr 13.
Typ publikacji:: Journal Article
MeSH Terms:: Milk Proteins*
STAT5 Transcription Factor*/genetics
STAT5 Transcription Factor*/metabolism
Cattle ; Animals ; Prolactin/pharmacology ; Prolactin/metabolism ; Micrococcal Nuclease/metabolism ; Mammary Glands, Animal/metabolism ; Signal Transduction/physiology ; Epithelial Cells/metabolism ; TOR Serine-Threonine Kinases/genetics ; TOR Serine-Threonine Kinases/metabolism ; Cell Proliferation

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 6.

Tytuł:: Notch3 restricts metastasis of breast cancers through regulation of the JAK/STAT5A signaling pathway.
Autorzy:: Chen MN; Department of Medical Oncology, Cancer Hospital of Shantou University Medical College, Shantou, China.
Fang ZX; The Breast Center, Cancer Hospital of Shantou University Medical College, Shantou, China.
Wu Z; The Breast Center, Cancer Hospital of Shantou University Medical College, Shantou, China.
Bai JW; Department of Medical Oncology/Xiamen Key Laboratory for Endocrine-Related Cancer Precision Medicine, Xiamen University Medical School, Xiang'an Hospital of Xiamen University, Xiamen, China.
Li RH; Department of Medical Oncology/Xiamen Key Laboratory for Endocrine-Related Cancer Precision Medicine, Xiamen University Medical School, Xiang'an Hospital of Xiamen University, Xiamen, China.
Wen XF; Department of Medical Oncology, Cancer Hospital of Shantou University Medical College, Shantou, China.
Zhang GJ; Xiamen Key Laboratory for Endocrine-Related Cancer Precision Medicine/Department of Breast and Thyroid Surgery, Xiamen University Medical School, Xiang'an Hospital of Xiamen University, Xiamen, China. .
Liu J; The Breast Center, Cancer Hospital of Shantou University Medical College, Shantou, China. .; Pokaż więcej
Źródło:: BMC cancer [BMC Cancer] 2023 Dec 20; Vol. 23 (1), pp. 1257. Date of Electronic Publication: 2023 Dec 20.
Typ publikacji:: Journal Article
MeSH Terms:: Breast Neoplasms*/pathology
Humans ; Female ; STAT5 Transcription Factor/genetics ; STAT5 Transcription Factor/metabolism ; Signal Transduction/genetics ; Chromatin Immunoprecipitation ; Receptor, Notch3/genetics ; Tumor Suppressor Proteins/genetics

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 7.

Tytuł:: SOCS2 inhibits hepatoblastoma metastasis via downregulation of the JAK2/STAT5 signal pathway.
Autorzy:: Lv Y; Department of Pediatric Surgery and Laboratory of Pediatric Surgery, West China Hospital, Sichuan University, Chengdu, 610041, China.
Xie X; Department of Pediatric Surgery and Laboratory of Pediatric Surgery, West China Hospital, Sichuan University, Chengdu, 610041, China.
Zou G; Department of General Surgery, People's Hospital of Tibet Autonomous Region, Tibet, 850000, China.
Kong M; Department of Pediatric Surgery, Children's Hospital Affiliated to Shandong University, Jinan, 250022, China.
Yang J; Liver Transplantation Center, Department of General Surgery, West China Hospital, Sichuan University, Chengdu, 610041, China.
Chen J; Department of Pediatric Surgery and Laboratory of Pediatric Surgery, West China Hospital, Sichuan University, Chengdu, 610041, China.
Xiang B; Department of Pediatric Surgery and Laboratory of Pediatric Surgery, West China Hospital, Sichuan University, Chengdu, 610041, China. xb_.; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2023 Dec 09; Vol. 13 (1), pp. 21814. Date of Electronic Publication: 2023 Dec 09.
Typ publikacji:: Journal Article
MeSH Terms:: Hepatoblastoma*/genetics
Liver Neoplasms*/pathology
Child ; Humans ; Down-Regulation ; STAT5 Transcription Factor/genetics ; STAT5 Transcription Factor/metabolism ; Signal Transduction ; Suppressor of Cytokine Signaling Proteins/genetics ; Suppressor of Cytokine Signaling Proteins/metabolism ; Cytokines/metabolism ; Cell Line, Tumor ; Gene Expression Regulation, Neoplastic ; Janus Kinase 2/genetics ; Janus Kinase 2/metabolism

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 8.

Tytuł:: Dual inhibition of STAT3 and STAT5 may overcome imatinib resistance in chronic myeloid leukemia.
Autorzy:: Yin L; Department of Hematology, The Affiliated Hospital of Xuzhou Medical University, Xuzhou, People's Republic of China.; Blood Diseases Institute, Xuzhou Medical University, Xuzhou, People's Republic of China.; Key Laboratory of Bone Marrow Stem Cells, Xuzhou, People's Republic of China.
Xu J; Blood Diseases Institute, Xuzhou Medical University, Xuzhou, People's Republic of China.; Key Laboratory of Bone Marrow Stem Cells, Xuzhou, People's Republic of China.
Wu W; Medical Record and Statistical Office, The Affiliated Hospital of Xuzhou Medical University, Xuzhou, People's Republic of China.
Niu M; Department of Hematology, The Affiliated Hospital of Xuzhou Medical University, Xuzhou, People's Republic of China.; Blood Diseases Institute, Xuzhou Medical University, Xuzhou, People's Republic of China.; Key Laboratory of Bone Marrow Stem Cells, Xuzhou, People's Republic of China.
Li Z; Department of Hematology, The Affiliated Hospital of Xuzhou Medical University, Xuzhou, People's Republic of China.; Blood Diseases Institute, Xuzhou Medical University, Xuzhou, People's Republic of China.; Key Laboratory of Bone Marrow Stem Cells, Xuzhou, People's Republic of China.
Zhu F; Department of Hematology, The Affiliated Hospital of Xuzhou Medical University, Xuzhou, People's Republic of China.; Blood Diseases Institute, Xuzhou Medical University, Xuzhou, People's Republic of China.; Key Laboratory of Bone Marrow Stem Cells, Xuzhou, People's Republic of China.
Xu K; Department of Hematology, The Affiliated Hospital of Xuzhou Medical University, Xuzhou, People's Republic of China.; Blood Diseases Institute, Xuzhou Medical University, Xuzhou, People's Republic of China.; Key Laboratory of Bone Marrow Stem Cells, Xuzhou, People's Republic of China.; Pokaż więcej
Źródło:: Hematology (Amsterdam, Netherlands) [Hematology] 2023 Dec; Vol. 28 (1), pp. 2224625.
Typ publikacji:: Journal Article
MeSH Terms:: Antineoplastic Agents*/pharmacology
Antineoplastic Agents*/therapeutic use
Leukemia, Myelogenous, Chronic, BCR-ABL Positive*/drug therapy
Leukemia, Myelogenous, Chronic, BCR-ABL Positive*/genetics
Leukemia, Myelogenous, Chronic, BCR-ABL Positive*/pathology
Humans ; Imatinib Mesylate/pharmacology ; Imatinib Mesylate/therapeutic use ; STAT5 Transcription Factor/genetics ; STAT5 Transcription Factor/metabolism ; Drug Resistance, Neoplasm/genetics ; Apoptosis ; K562 Cells ; Fusion Proteins, bcr-abl/genetics ; Fusion Proteins, bcr-abl/metabolism ; Protein Kinase Inhibitors/pharmacology ; Protein Kinase Inhibitors/therapeutic use ; STAT3 Transcription Factor/genetics ; STAT3 Transcription Factor/metabolism

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 9.

Tytuł:: Cadmium Activates EGFR/STAT5 Signaling to Overcome Calcium Chelation and Promote Epithelial to Mesenchymal Transition.
Autorzy:: Stavrou A; Department of Medicine, Division of Environmental Medicine, New York University Grossman School of Medicine, New York, NY 10010, USA.
Ortiz A; Department of Medicine, Division of Environmental Medicine, New York University Grossman School of Medicine, New York, NY 10010, USA.
Costa M; Department of Medicine, Division of Environmental Medicine, New York University Grossman School of Medicine, New York, NY 10010, USA.; Pokaż więcej
Źródło:: Biomolecules [Biomolecules] 2023 Jan 06; Vol. 13 (1). Date of Electronic Publication: 2023 Jan 06.
Typ publikacji:: Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:: Cadmium*/toxicity
Cadmium*/metabolism
STAT5 Transcription Factor*/metabolism
Humans ; Animals ; Calcium/metabolism ; Epithelial-Mesenchymal Transition/physiology ; HEK293 Cells ; ErbB Receptors/genetics ; ErbB Receptors/metabolism ; Cadherins/genetics ; Cadherins/metabolism

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 10.

Tytuł:: AMPK Amplifies IL2-STAT5 Signaling to Maintain Stability of Regulatory T Cells in Aged Mice.
Autorzy:: Pokhrel RH; College of Pharmacy, Yeungnam University, Gyeongsan 38541, Korea.
Kang B; Department of Pediatrics, School of Medicine, Kyungpook National University, Daegu 41944, Korea.
Timilshina M; College of Pharmacy, Yeungnam University, Gyeongsan 38541, Korea.
Chang JH; College of Pharmacy, Yeungnam University, Gyeongsan 38541, Korea.; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2022 Oct 16; Vol. 23 (20). Date of Electronic Publication: 2022 Oct 16.
Typ publikacji:: Journal Article
MeSH Terms:: T-Lymphocytes, Regulatory*
STAT5 Transcription Factor*/genetics
STAT5 Transcription Factor*/metabolism
Mice ; Animals ; Interleukin-2/genetics ; Interleukin-2/metabolism ; AMP-Activated Protein Kinases/genetics ; AMP-Activated Protein Kinases/metabolism ; Mice, Knockout ; Forkhead Transcription Factors/metabolism

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 11.

Tytuł:: PRL/PRLR Can Promote Insulin Resistance by Activating the JAK2/STAT5 Signaling Pathway.
Autorzy:: Wang PY; Reproductive Medicine Center, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang 325000, China.
Jin CC; Reproductive Medicine Center, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang 325000, China.
Liu C; Reproductive Medicine Center, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang 325000, China.
Zhao ZJ; Zhuji Yaojiang Town Central Health Center, Shaoxing, Zhejiang 311822, China.
Yang HY; Reproductive Medicine Center, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang 325000, China.; Pokaż więcej
Źródło:: Computational and mathematical methods in medicine [Comput Math Methods Med] 2022 Oct 04; Vol. 2022, pp. 1456187. Date of Electronic Publication: 2022 Oct 04 (Print Publication: 2022).
Typ publikacji:: Journal Article; Retracted Publication
MeSH Terms:: Insulin Resistance*
STAT5 Transcription Factor*/genetics
STAT5 Transcription Factor*/metabolism
Humans ; Janus Kinase 2/metabolism ; Oleic Acid/pharmacology ; Prolactin/metabolism ; Receptors, Prolactin/genetics ; Receptors, Prolactin/metabolism ; Signal Transduction ; Triglycerides

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 12.

Tytuł:: Oncostatin M and STAT3 Signaling Pathways Support Human Trophoblast Differentiation by Inhibiting Inflammatory Stress in Response to IFNγ and GM-CSF.
Autorzy:: Ravelojaona M; Groupe de Recherche en Signalisation Cellulaire (GRSC), Département de Biologie Médicale, Université du Québec à Trois-Rivières, 3351 Boul. des Forges, Trois-Rivières, QC G8Z 4M3, Canada.; Centre de Recherche Interuniversitaire en Reproduction et Développement-Réseau Québécois en Reproduction (CIRD-RQR), Université de Montréal, St-Hyacinthe, QC J2S 2M2, Canada.; Regroupement Intersectoriel de Recherche en Santé de l'Université du Québec (RISUQ), Université du Québec, Québec, QC G1K 9H7, Canada.
Girouard J; Groupe de Recherche en Signalisation Cellulaire (GRSC), Département de Biologie Médicale, Université du Québec à Trois-Rivières, 3351 Boul. des Forges, Trois-Rivières, QC G8Z 4M3, Canada.; Centre de Recherche Interuniversitaire en Reproduction et Développement-Réseau Québécois en Reproduction (CIRD-RQR), Université de Montréal, St-Hyacinthe, QC J2S 2M2, Canada.; Regroupement Intersectoriel de Recherche en Santé de l'Université du Québec (RISUQ), Université du Québec, Québec, QC G1K 9H7, Canada.
Kana Tsapi ES; Groupe de Recherche en Signalisation Cellulaire (GRSC), Département de Biologie Médicale, Université du Québec à Trois-Rivières, 3351 Boul. des Forges, Trois-Rivières, QC G8Z 4M3, Canada.
Chambers M; Medical School, Swansea University, Swansea SA2 8PP, UK.
Vaillancourt C; Centre de Recherche Interuniversitaire en Reproduction et Développement-Réseau Québécois en Reproduction (CIRD-RQR), Université de Montréal, St-Hyacinthe, QC J2S 2M2, Canada.; Regroupement Intersectoriel de Recherche en Santé de l'Université du Québec (RISUQ), Université du Québec, Québec, QC G1K 9H7, Canada.; Centre Armand Frappier Santé Biotechnologie, Institut National de la Recherche Scientifique (INRS), Laval, QC H7V 1B7, Canada.
Van Themsche C; Groupe de Recherche en Signalisation Cellulaire (GRSC), Département de Biologie Médicale, Université du Québec à Trois-Rivières, 3351 Boul. des Forges, Trois-Rivières, QC G8Z 4M3, Canada.; Centre de Recherche Interuniversitaire en Reproduction et Développement-Réseau Québécois en Reproduction (CIRD-RQR), Université de Montréal, St-Hyacinthe, QC J2S 2M2, Canada.; Regroupement Intersectoriel de Recherche en Santé de l'Université du Québec (RISUQ), Université du Québec, Québec, QC G1K 9H7, Canada.
Thornton CA; Medical School, Swansea University, Swansea SA2 8PP, UK.
Reyes-Moreno C; Groupe de Recherche en Signalisation Cellulaire (GRSC), Département de Biologie Médicale, Université du Québec à Trois-Rivières, 3351 Boul. des Forges, Trois-Rivières, QC G8Z 4M3, Canada.; Centre de Recherche Interuniversitaire en Reproduction et Développement-Réseau Québécois en Reproduction (CIRD-RQR), Université de Montréal, St-Hyacinthe, QC J2S 2M2, Canada.; Regroupement Intersectoriel de Recherche en Santé de l'Université du Québec (RISUQ), Université du Québec, Québec, QC G1K 9H7, Canada.; Pokaż więcej
Źródło:: Cells [Cells] 2024 Jan 25; Vol. 13 (3). Date of Electronic Publication: 2024 Jan 25.
Typ publikacji:: Journal Article
MeSH Terms:: Granulocyte-Macrophage Colony-Stimulating Factor*/pharmacology
Granulocyte-Macrophage Colony-Stimulating Factor*/metabolism
Interferon-gamma*/pharmacology
Interferon-gamma*/metabolism
Pregnancy ; Female ; Humans ; Oncostatin M/pharmacology ; Oncostatin M/metabolism ; STAT5 Transcription Factor/metabolism ; Interleukin-6/metabolism ; Signal Transduction ; Trophoblasts/metabolism ; STAT3 Transcription Factor/metabolism

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 13.

Tytuł:: Leptin-Mediated Induction of IL-6 Expression in Hofbauer Cells Contributes to Preeclampsia Pathogenesis.
Autorzy:: Ozmen A; Department of Obstetrics & Gynecology, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA.
Nwabuobi C; Department of Obstetrics & Gynecology, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA.
Tang Z; Department of Obstetrics, Gynecology and Reproductive Sciences, Yale University School of Medicine, New Haven, CT 06520, USA.
Guo X; Department of Obstetrics & Gynecology, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA.
Larsen K; Department of Obstetrics & Gynecology, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA.
Guller S; Department of Obstetrics, Gynecology and Reproductive Sciences, Yale University School of Medicine, New Haven, CT 06520, USA.
Blas J; Department of Obstetrics & Gynecology, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA.
Moore M; Department of Obstetrics & Gynecology, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA.
Kayisli UA; Department of Obstetrics & Gynecology, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA.
Lockwood CJ; Department of Obstetrics & Gynecology, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA.
Guzeloglu-Kayisli O; Department of Obstetrics & Gynecology, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA.; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2023 Dec 21; Vol. 25 (1). Date of Electronic Publication: 2023 Dec 21.
Typ publikacji:: Journal Article
MeSH Terms:: Leptin*/pharmacology
Pre-Eclampsia*
Pregnancy ; Humans ; Female ; Interleukin-6 ; STAT5 Transcription Factor ; NF-kappa B ; Placenta

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 14.

Tytuł:: Astragalus polysaccharide ameliorates insulin resistance in HepG2 cells through activating the STAT5/IGF-1 pathway.
Autorzy:: Yue X; Department of Clinical College, HE University, Shenyang, Liaoning, China.
Hao W; Department of Clinical College, HE University, Shenyang, Liaoning, China.
Wang M; Department of Clinical College, HE University, Shenyang, Liaoning, China.
Fu Y; Department of Burn and Plastic Surgery, General Hospital of Northern Theater Command, Shenyang, Liaoning, China.; Pokaż więcej
Źródło:: Immunity, inflammation and disease [Immun Inflamm Dis] 2023 Nov; Vol. 11 (11), pp. e1071.
Typ publikacji:: Journal Article
MeSH Terms:: Insulin Resistance*
Diabetes Mellitus, Type 2*
Humans ; Insulin-Like Growth Factor I/metabolism ; Proto-Oncogene Proteins c-akt ; Hep G2 Cells ; STAT5 Transcription Factor/metabolism ; Glucose/pharmacology ; Polysaccharides/pharmacology

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 15.

Tytuł:: PDGFRβ promotes oncogenic progression via STAT3/STAT5 hyperactivation in anaplastic large cell lymphoma.
Autorzy:: Garces de Los Fayos Alonso I; Department of Pathology, Medical University of Vienna, 1090, Vienna, Austria.; Unit of Laboratory Animal Pathology, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.
Zujo L; Department of Pathology, Medical University of Vienna, 1090, Vienna, Austria.; Unit of Laboratory Animal Pathology, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.; Division of Nuclear Medicine, Medical University of Vienna, 1090, Vienna, Austria.
Wiest I; Department of Pathology, Medical University of Vienna, 1090, Vienna, Austria.; Unit of Laboratory Animal Pathology, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.; Division of Nuclear Medicine, Medical University of Vienna, 1090, Vienna, Austria.
Kodajova P; Unit of Laboratory Animal Pathology, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.
Timelthaler G; Center for Cancer Research, Medical University of Vienna, 1090, Vienna, Austria.
Edtmayer S; Division Pharmacology, Department of Pharmacology, Physiology and Microbiology, Karl Landsteiner University of Health Sciences, 3500, Krems, Austria.
Zrimšek M; Department of Pathology, Medical University of Vienna, 1090, Vienna, Austria.
Kollmann S; Institute of Pharmacology and Toxicology, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.
Giordano C; Department of Pathology, Medical University of Vienna, 1090, Vienna, Austria.
Kothmayer M; Department of Pathology, Medical University of Vienna, 1090, Vienna, Austria.; Unit of Laboratory Animal Pathology, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.; Centre for Anatomy and Cell Biology, Medical University of Vienna, 1090, Vienna, Austria.
Neubauer HA; Institute of Animal Breeding and Genetics, Unit of Functional Cancer Genomics, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.
Dey S; Department of Dermatology, Medical University of Graz, 8036, Graz, Austria.; Center for Medical Research (ZMF), Medical University of Graz, 8010, Graz, Austria.
Schlederer M; Department of Pathology, Medical University of Vienna, 1090, Vienna, Austria.
Schmalzbauer BS; Unit of Laboratory Animal Pathology, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.; Institute of Pharmacology and Toxicology, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.
Limberger T; Department of Pathology, Medical University of Vienna, 1090, Vienna, Austria.; Division of Nuclear Medicine, Medical University of Vienna, 1090, Vienna, Austria.; CBMed Core Lab, Medical University of Vienna, 1090, Vienna, Austria.
Probst C; Department of Pathology, Medical University of Vienna, 1090, Vienna, Austria.; Unit of Laboratory Animal Pathology, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.; Division of Nuclear Medicine, Medical University of Vienna, 1090, Vienna, Austria.
Pusch O; Centre for Anatomy and Cell Biology, Medical University of Vienna, 1090, Vienna, Austria.
Högler S; Unit of Laboratory Animal Pathology, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.
Tangermann S; Unit of Laboratory Animal Pathology, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.
Merkel O; Department of Pathology, Medical University of Vienna, 1090, Vienna, Austria.
Schiefer AI; Department of Pathology, Medical University of Vienna, 1090, Vienna, Austria.
Kornauth C; Department of Medicine I, Division of Hematology and Hemostaseology, Medical University of Vienna, 1090, Vienna, Austria.; Comprehensive Cancer Center Vienna, Vienna General Hospital, Medical University of Vienna, 1090, Vienna, Austria.
Prutsch N; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, MA, USA.
Zimmerman M; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, MA, USA.
Abraham B; Department of Computational Biology, St. Jude Children's Research Hospital, Memphis, TN, USA.
Anagnostopoulos J; Institute of Pathology, University of Wuerzburg, 97080, Würzburg, Germany.; Institute of Pathology, Charité-Medical University of Berlin, 10117, Berlin, Germany.
Quintanilla-Martinez L; Institute of Pathology and Neuropathology and Cluster of excellence iFIT, 'Image-Guided and Functionally Instructed Tumor Therapy', University of Tübingen, 72076, Tübingen, Germany.
Mathas S; Department of Hematology, Oncology, and Cancer Immunology, Charité-Medical University of Berlin, 12200, Berlin, Germany.; German Cancer Consortium (DKTK) German Cancer Research Center (DKFZ), 69120, Heidelberg, Germany.; Max-Delbrück-Center (MDC) for Molecular Medicine, 13125, Berlin, Germany.; Experimental and Clinical Research Center, a joint cooperation between the Charité and the MDC, 13125, Berlin, Germany.
Wolf P; Department of Dermatology, Medical University of Graz, 8036, Graz, Austria.
Stoiber D; Division Pharmacology, Department of Pharmacology, Physiology and Microbiology, Karl Landsteiner University of Health Sciences, 3500, Krems, Austria.
Staber PB; Department of Medicine I, Division of Hematology and Hemostaseology, Medical University of Vienna, 1090, Vienna, Austria.; Comprehensive Cancer Center Vienna, Vienna General Hospital, Medical University of Vienna, 1090, Vienna, Austria.
Egger G; Department of Pathology, Medical University of Vienna, 1090, Vienna, Austria.; Comprehensive Cancer Center Vienna, Vienna General Hospital, Medical University of Vienna, 1090, Vienna, Austria.; Boltzmann Institute Applied Diagnostics, 1090, Vienna, Austria.
Klapper W; Department of Pathology, Hematopathology Section and Lymph Node Registry, University of Kiel/University Hospital Schleswig-Holstein, 24105, Kiel, Germany.
Woessmann W; Pediatric Hematology and Oncology, University Hospital Hamburg-Eppendorf, Hamburg, Germany.
Look TA; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, MA, USA.
Gunning P; Department of Chemical and Physical Sciences, University of Toronto Mississauga, Mississauga, ON, L5L 1C6, Canada.; Department of Chemistry, University of Toronto, Toronto, ON, M5S 3H6, Canada.
Turner SD; Division of Cellular and Molecular Pathology, Department of Pathology, University of Cambridge, Cambridge, CB20QQ, UK.; Central European Institute of Technology (CEITEC), Masaryk University, Brno, Czech Republic.
Moriggl R; Institute of Animal Breeding and Genetics, Unit of Functional Cancer Genomics, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.
Lagger S; Unit of Laboratory Animal Pathology, University of Veterinary Medicine Vienna, 1210, Vienna, Austria.
Kenner L; Department of Pathology, Medical University of Vienna, 1090, Vienna, Austria. .; Unit of Laboratory Animal Pathology, University of Veterinary Medicine Vienna, 1210, Vienna, Austria. .; Division of Nuclear Medicine, Medical University of Vienna, 1090, Vienna, Austria. .; Center for Medical Research (ZMF), Medical University of Graz, 8010, Graz, Austria. .; CBMed Core Lab, Medical University of Vienna, 1090, Vienna, Austria. .; Christian Doppler Laboratory of Applied Metabolomics, Department of Biomedical Imaging and Image-guided Therapy, Medical University of Vienna, 1090, Vienna, Austria. .; Pokaż więcej
Źródło:: Molecular cancer [Mol Cancer] 2022 Aug 31; Vol. 21 (1), pp. 172. Date of Electronic Publication: 2022 Aug 31.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Lymphoma, Large-Cell, Anaplastic*/genetics
Lymphoma, Large-Cell, Anaplastic*/pathology
Receptor, Platelet-Derived Growth Factor beta*/metabolism
Receptor, Platelet-Derived Growth Factor beta*/pharmacology
STAT3 Transcription Factor*/metabolism
STAT5 Transcription Factor*/genetics
Anaplastic Lymphoma Kinase ; Animals ; Carcinogenesis/metabolism ; Cell Line, Tumor ; Humans ; Mice ; Phosphorylation ; Signal Transduction

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 16.

Tytuł:: Erythropoietin mitigated thioacetamide-induced renal injury via JAK2/STAT5 and AMPK pathway.
Autorzy:: Elbaset MA; Pharmacology Department, Medical Research and Clinical Studies Institute, National Research Centre, 33 El-Bohouth St., Dokki, P.O. 12622, Cairo, Egypt. .
Mohamed BMSA; Pharmacology Department, Medical Research and Clinical Studies Institute, National Research Centre, 33 El-Bohouth St., Dokki, P.O. 12622, Cairo, Egypt.
Gad SA; Pharmacology Department, Medical Research and Clinical Studies Institute, National Research Centre, 33 El-Bohouth St., Dokki, P.O. 12622, Cairo, Egypt.
Afifi SM; Pharmacognosy Department, Faculty of Pharmacy, University of Sadat City, Sadat City, 32897, Egypt.
Esatbeyoglu T; Department of Food Development and Food Quality, Institute of Food Science and Human Nutrition, Gottfried Wilhelm Leibniz University Hannover, Am Kleinen Felde 30, 30167, Hannover, Germany. .
Abdelrahman SS; Department of Pathology, Faculty of Veterinary Medicine, Cairo University, Giza, Egypt.
Fayed HM; Pharmacology Department, Medical Research and Clinical Studies Institute, National Research Centre, 33 El-Bohouth St., Dokki, P.O. 12622, Cairo, Egypt.; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2023 Sep 11; Vol. 13 (1), pp. 14929. Date of Electronic Publication: 2023 Sep 11.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: AMP-Activated Protein Kinases*
Erythropoietin*/pharmacology
Animals ; Rats ; Thioacetamide/toxicity ; STAT5 Transcription Factor ; Kidney

Czasopismo naukowe

na półce

Pełny tekst Pełny tekst Szczegóły

Skocz do pozycji: 17.

Tytuł:: 22-kD growth hormone-induced nuclear GHR/STAT5/CyclinD1 signaling pathway plays an important role in promoting mesenchymal stem cell proliferation.
Autorzy:: Zheng WV; Intervention and Cell Therapy Center, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.
Li Y; Department of Infectious Disease, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.
Xu Y; Intervention and Cell Therapy Center, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.
Zhou T; Intervention and Cell Therapy Center, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.
Li D; Intervention and Cell Therapy Center, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.
Cheng X; Intervention and Cell Therapy Center, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.; Department of Minimally Invasion Intervention, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.
Xiong Y; Intervention and Cell Therapy Center, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.; Department of Minimally Invasion Intervention, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.
Wang S; Health Management Center, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.
Chen Z; Intervention and Cell Therapy Center, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.; Department of Minimally Invasion Intervention, Peking University Shenzhen Hospital, Shenzhen, Guangdong, China.; Pokaż więcej
Źródło:: BioFactors (Oxford, England) [Biofactors] 2022 Jan; Vol. 48 (1), pp. 67-85. Date of Electronic Publication: 2021 Dec 05.
Typ publikacji:: Journal Article
MeSH Terms:: Mesenchymal Stem Cells*/metabolism
STAT5 Transcription Factor*/genetics
STAT5 Transcription Factor*/metabolism
STAT5 Transcription Factor*/pharmacology
Cell Nucleus/genetics ; Cell Nucleus/metabolism ; Cell Proliferation ; Growth Hormone/genetics ; Growth Hormone/metabolism ; Growth Hormone/pharmacology ; Receptors, Somatotropin/genetics ; Receptors, Somatotropin/metabolism ; Signal Transduction

Czasopismo naukowe

na półce

Pełny tekst Szczegóły

Skocz do pozycji: 18.

Tytuł:: Isoflavones and their metabolites influence the milk production ability of bovine mammary epithelial cells in a type-specific manner.
Autorzy:: Tsugami Y; Laboratory of Animal Histophysiology, Graduate School of Integrated Sciences for Life, Hiroshima University, Hiroshima, Japan.
Wakasa H; Laboratory of Cell and Tissue Biology, Research Faculty of Agriculture, Hokkaido University, Sapporo, Japan.
Kawahara M; Laboratory of Animal Genetics and Reproduction, Research Faculty of Agriculture, Hokkaido University, Sapporo, Japan.
Nishimura T; Laboratory of Cell and Tissue Biology, Research Faculty of Agriculture, Hokkaido University, Sapporo, Japan.
Kobayashi K; Laboratory of Cell and Tissue Biology, Research Faculty of Agriculture, Hokkaido University, Sapporo, Japan.; Pokaż więcej
Źródło:: Animal science journal = Nihon chikusan Gakkaiho [Anim Sci J] 2022 Jan-Dec; Vol. 93 (1), pp. e13720.
Typ publikacji:: Journal Article
MeSH Terms:: Isoflavones*/pharmacology
STAT5 Transcription Factor*/genetics
Animals ; Caseins/metabolism ; Cattle ; Claudin-3/metabolism ; Epithelial Cells/metabolism ; Equol/metabolism ; Female ; Lactoferrin/metabolism ; Mammary Glands, Animal ; Milk/metabolism ; RNA, Messenger/metabolism

Czasopismo naukowe

na półce

Pełny tekst Szczegóły

Skocz do pozycji: 19.

Tytuł:: Nuclear PLD1 combined with NPM1 induces gemcitabine resistance through tumorigenic IL7R in pancreatic adenocarcinoma.
Autorzy:: Fu D; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Yan J; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Zhang Z; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Liu Y; Department of General Surgery, Changzheng Hospital, Naval Medical University (Second Military Medical University), Shanghai 200003, China.
Ma X; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Ding J; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Yang S; Department of Cellular and Molecular Physiology, Penn State College of Medicine, Hershey, Pennsylvania 17033, USA.
Zhao R; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Chang A; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Gao C; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Liu J; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Zhao T; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Wang X; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Huang C; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Gao S; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Ma Y; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Tang B; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Feng Y; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Wang H; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.
Hao J; Department of Pancreatic Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Centre for Cancer, Key Laboratory of Cancer Prevention and Therapy, Tianjin 300060, China.; Pokaż więcej
Źródło:: Cancer biology & medicine [Cancer Biol Med] 2023 Jun 27; Vol. 20 (8).
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Adenocarcinoma*/drug therapy
Adenocarcinoma*/genetics
Carcinoma, Pancreatic Ductal*/drug therapy
Carcinoma, Pancreatic Ductal*/genetics
Carcinoma, Pancreatic Ductal*/pathology
Pancreatic Neoplasms*/drug therapy
Pancreatic Neoplasms*/genetics
Humans ; Antimetabolites, Antineoplastic/pharmacology ; Antimetabolites, Antineoplastic/therapeutic use ; Deoxycytidine/pharmacology ; Deoxycytidine/therapeutic use ; Drug Resistance, Neoplasm/genetics ; Gemcitabine ; Nuclear Proteins/genetics ; Nuclear Proteins/metabolism ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Receptors, Interleukin-7/metabolism ; RNA, Guide, CRISPR-Cas Systems ; STAT5 Transcription Factor/metabolism ; STAT5 Transcription Factor/pharmacology ; Pancreatic Neoplasms

Czasopismo naukowe

na półce

Pełny tekst Szczegóły

Skocz do pozycji: 20.

Tytuł:: Adipocyte STAT5 deficiency does not affect blood glucose homeostasis in obese mice.
Autorzy:: Beghini M; Division of Endocrinology & Metabolism, Department of Medicine III, Medical University of Vienna, Vienna, Austria.
Wagner T; Division of Endocrinology & Metabolism, Department of Medicine III, Medical University of Vienna, Vienna, Austria.
Luca AC; Division of Endocrinology & Metabolism, Department of Medicine III, Medical University of Vienna, Vienna, Austria.
Metz M; Division of Endocrinology & Metabolism, Department of Medicine III, Medical University of Vienna, Vienna, Austria.
Kaltenecker D; Institute of Animal Breeding and Genetics, University of Veterinary Medicine Vienna, Vienna, Austria.
Spirk K; Division of Endocrinology & Metabolism, Department of Medicine III, Medical University of Vienna, Vienna, Austria.; Institute of Animal Breeding and Genetics, University of Veterinary Medicine Vienna, Vienna, Austria.
Hackl MT; Division of Endocrinology & Metabolism, Department of Medicine III, Medical University of Vienna, Vienna, Austria.
Haybaeck J; Institute of Pathology, Neuropathology and Molecular Pathology, Medical University of Innsbruck, Innsbruck, Austria.; Diagnostic & Research Center for Molecular Biomedicine, Institute of Pathology, Medical University of Graz, Graz, Austria.
Moriggl R; Institute of Animal Breeding and Genetics, University of Veterinary Medicine Vienna, Vienna, Austria.
Kautzky-Willer A; Division of Endocrinology & Metabolism, Department of Medicine III, Medical University of Vienna, Vienna, Austria.
Scherer T; Division of Endocrinology & Metabolism, Department of Medicine III, Medical University of Vienna, Vienna, Austria.
Fürnsinn C; Division of Endocrinology & Metabolism, Department of Medicine III, Medical University of Vienna, Vienna, Austria.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2021 Nov 24; Vol. 16 (11), pp. e0260501. Date of Electronic Publication: 2021 Nov 24 (Print Publication: 2021).
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Adipocytes/*metabolism
Blood Glucose/*metabolism
Obesity/*genetics
STAT5 Transcription Factor/*genetics
Adiposity ; Animals ; Blood Glucose/genetics ; Gene Deletion ; Lipid Metabolism ; Male ; Mice, Inbred C57BL ; Mice, Obese ; Obesity/metabolism ; STAT5 Transcription Factor/metabolism ; Mice

Czasopismo naukowe

na półce

Pełny tekst Szczegóły

Informacja

Wyszukujesz frazę ""STAT5 Transcription Factor"" wg kryterium: Temat

Źródło danych

Ogranicz do:

Ogranicz do bazy

Typ dokumentu

Temat

Wydawca

Publikacja

Język