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Tytuł:
Intermedin protects thapsigargin‑induced endoplasmic reticulum stress in cardiomyocytes by modulating protein kinase A and sarco/endoplasmic reticulum Ca ‑ATPase.
Autorzy:
Li Z; Department of Pharmacology, Shanxi Medical University, Taiyuan, Shanxi 030001, P.R. China.
Guo J; Department of Cardiology, Shanxi Medical University First Hospital, Taiyuan, Shanxi 030001, P.R. China.
Bian Y; Department of Cardiology, Shanxi Medical University Second Hospital, Taiyuan, Shanxi 030001, P.R. China.
Zhang M; Department of Pharmacology, Shanxi Medical University, Taiyuan, Shanxi 030001, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2021 Feb; Vol. 23 (2). Date of Electronic Publication: 2020 Dec 10.
Typ publikacji:
Journal Article
MeSH Terms:
Adrenomedullin/*metabolism
Calcium Signaling/*drug effects
Cyclic AMP-Dependent Protein Kinases/*metabolism
Endoplasmic Reticulum Stress/*drug effects
Myocytes, Cardiac/*metabolism
Neuropeptides/*metabolism
Sarcoplasmic Reticulum Calcium-Transporting ATPases/*metabolism
Thapsigargin/*pharmacology
Adrenomedullin/genetics ; Animals ; Calcium Signaling/genetics ; Cyclic AMP-Dependent Protein Kinases/genetics ; Endoplasmic Reticulum Stress/genetics ; Female ; Male ; Neuropeptides/genetics ; Rats ; Rats, Sprague-Dawley ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
SMN-deficiency disrupts SERCA2 expression and intracellular Ca signaling in cardiomyocytes from SMA mice and patient-derived iPSCs.
Autorzy:
Khayrullina G; Department of Anatomy, Physiology and Genetics, F. Edward Hebert School of Medicine, Bethesda, MD, USA.
Moritz KE; Department of Anatomy, Physiology and Genetics, F. Edward Hebert School of Medicine, Bethesda, MD, USA.
Schooley JF
Fatima N; Department of Anatomy, Physiology and Genetics, F. Edward Hebert School of Medicine, Bethesda, MD, USA.
Viollet C; Department of Anatomy, Physiology and Genetics, F. Edward Hebert School of Medicine, Bethesda, MD, USA.; The American Genome Center, Uniformed Services University of the Health Sciences, Bethesda, MD, USA.
McCormack NM; Department of Anatomy, Physiology and Genetics, F. Edward Hebert School of Medicine, Bethesda, MD, USA.
Smyth JT; Department of Anatomy, Physiology and Genetics, F. Edward Hebert School of Medicine, Bethesda, MD, USA.
Doughty ML; Department of Anatomy, Physiology and Genetics, F. Edward Hebert School of Medicine, Bethesda, MD, USA.
Dalgard CL; Department of Anatomy, Physiology and Genetics, F. Edward Hebert School of Medicine, Bethesda, MD, USA.; The American Genome Center, Uniformed Services University of the Health Sciences, Bethesda, MD, USA.
Flagg TP; Department of Anatomy, Physiology and Genetics, F. Edward Hebert School of Medicine, Bethesda, MD, USA. .
Burnett BG; Department of Anatomy, Physiology and Genetics, F. Edward Hebert School of Medicine, Bethesda, MD, USA. .
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Źródło:
Skeletal muscle [Skelet Muscle] 2020 May 08; Vol. 10 (1), pp. 16. Date of Electronic Publication: 2020 May 08.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms:
Calcium Signaling*
Induced Pluripotent Stem Cells/*metabolism
Muscular Atrophy, Spinal/*metabolism
Myocytes, Cardiac/*metabolism
Sarcoplasmic Reticulum Calcium-Transporting ATPases/*metabolism
Survival of Motor Neuron 1 Protein/*genetics
Animals ; Cell Line ; Cells, Cultured ; Humans ; Mice ; Muscular Atrophy, Spinal/genetics ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Characterization of the PLN p.Arg14del Mutation in Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes.
Autorzy:
Badone B; Laboratory of Cardiac Cellular Physiology, Department of Biotechnology and Bioscience, University of Milano-Bicocca, 20126 Milan, Italy.
Ronchi C; Laboratory of Cardiac Cellular Physiology, Department of Biotechnology and Bioscience, University of Milano-Bicocca, 20126 Milan, Italy.
Lodola F; Laboratory of Cardiac Cellular Physiology, Department of Biotechnology and Bioscience, University of Milano-Bicocca, 20126 Milan, Italy.
Knaust AE; Department of Experimental Pharmacology and Toxicology, Cardiovascular Research Center, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany.; German Centre for Cardiovascular Research (DZHK), Partner Site Hamburg/Kiel/Lübeck, 20249 Hamburg, Germany.
Hansen A; Department of Experimental Pharmacology and Toxicology, Cardiovascular Research Center, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany.; German Centre for Cardiovascular Research (DZHK), Partner Site Hamburg/Kiel/Lübeck, 20249 Hamburg, Germany.
Eschenhagen T; Department of Experimental Pharmacology and Toxicology, Cardiovascular Research Center, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany.; German Centre for Cardiovascular Research (DZHK), Partner Site Hamburg/Kiel/Lübeck, 20249 Hamburg, Germany.
Zaza A; Laboratory of Cardiac Cellular Physiology, Department of Biotechnology and Bioscience, University of Milano-Bicocca, 20126 Milan, Italy.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Dec 16; Vol. 22 (24). Date of Electronic Publication: 2021 Dec 16.
Typ publikacji:
Journal Article
MeSH Terms:
Calcium-Binding Proteins/*genetics
Induced Pluripotent Stem Cells/*metabolism
Mutation/*genetics
Myocytes, Cardiac/*metabolism
Adult ; Animals ; Calcium/metabolism ; Cardiomyopathy, Dilated/genetics ; Cardiomyopathy, Dilated/metabolism ; Cattle ; Cells, Cultured ; Endoplasmic Reticulum/genetics ; Endoplasmic Reticulum/metabolism ; Female ; Heterozygote ; Humans ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Muscle-specific sirtuin 3 overexpression does not attenuate the pathological effects of high-fat/high-sucrose feeding but does enhance cardiac SERCA2a activity.
Autorzy:
Oldfield CJ; Faculty of Kinesiology and Recreation Management, University of Manitoba, Winnipeg, MB, Canada.; Institute of Cardiovascular Sciences, St. Boniface Hospital Albrechtsen Research Centre, Winnipeg, MB, Canada.
Moffatt TL; Faculty of Kinesiology and Recreation Management, University of Manitoba, Winnipeg, MB, Canada.; Institute of Cardiovascular Sciences, St. Boniface Hospital Albrechtsen Research Centre, Winnipeg, MB, Canada.
O'Hara KA; Institute of Cardiovascular Sciences, St. Boniface Hospital Albrechtsen Research Centre, Winnipeg, MB, Canada.
Xiang B; Department of Pharmacology and Therapeutics, Max Rady College of Medicine, Rady Faculty of Health Sciences, University of Manitoba, Winnipeg, MB, Canada.; Diabetes Research Envisioned and Accomplished in Manitoba (DREAM) Theme of the Children's Hospital Research Institute of Manitoba, Winnipeg, MB, Canada.
Dolinsky VW; Department of Pharmacology and Therapeutics, Max Rady College of Medicine, Rady Faculty of Health Sciences, University of Manitoba, Winnipeg, MB, Canada.; Diabetes Research Envisioned and Accomplished in Manitoba (DREAM) Theme of the Children's Hospital Research Institute of Manitoba, Winnipeg, MB, Canada.
Duhamel TA; Faculty of Kinesiology and Recreation Management, University of Manitoba, Winnipeg, MB, Canada.; Institute of Cardiovascular Sciences, St. Boniface Hospital Albrechtsen Research Centre, Winnipeg, MB, Canada.
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Źródło:
Physiological reports [Physiol Rep] 2021 Aug; Vol. 9 (16), pp. e14961.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Diabetic Cardiomyopathies/*metabolism
Myocytes, Cardiac/*metabolism
Sarcoplasmic Reticulum Calcium-Transporting ATPases/*metabolism
Sirtuin 3/*metabolism
Acetylation ; Animals ; Calcium Signaling ; Diabetic Cardiomyopathies/etiology ; Diet, Carbohydrate Loading/adverse effects ; Diet, High-Fat/adverse effects ; Male ; Mice ; Mice, Inbred C57BL ; Myocytes, Cardiac/physiology ; Sirtuin 3/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
SERCA is critical to control the Bowditch effect in the heart.
Autorzy:
Balcazar D; Centro de Investigaciones Cardiovasculares - CONICET/Universidad Nacional de la Plata, La Plata, Argentina.
Regge V; Centro de Investigaciones Cardiovasculares - CONICET/Universidad Nacional de la Plata and Departamento de Ciencias Básicas y Experimentales -UNNOBA, La Plata, Argentina.
Santalla M; Centro de Investigaciones Cardiovasculares - CONICET/Universidad Nacional de la Plata and Departamento de Ciencias Básicas y Experimentales -UNNOBA, La Plata, Argentina.
Meyer H; University of Osnabrück, Biology, Department of Zoology and Developmental Biology, Barbarastraße 11, 49076, Osnabrück, Germany.
Paululat A; University of Osnabrück, Biology, Department of Zoology and Developmental Biology, Barbarastraße 11, 49076, Osnabrück, Germany.
Mattiazzi A; Centro de Investigaciones Cardiovasculares - CONICET/Universidad Nacional de la Plata, La Plata, Argentina.
Ferrero P; Centro de Investigaciones Cardiovasculares - CONICET/Universidad Nacional de la Plata and Departamento de Ciencias Básicas y Experimentales -UNNOBA, La Plata, Argentina. .
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Źródło:
Scientific reports [Sci Rep] 2018 Aug 20; Vol. 8 (1), pp. 12447. Date of Electronic Publication: 2018 Aug 20.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Heart/*physiology
Myocytes, Cardiac/*metabolism
Sarcoplasmic Reticulum Calcium-Transporting ATPases/*metabolism
Animals ; Arrhythmias, Cardiac/genetics ; Arrhythmias, Cardiac/metabolism ; Calcium/metabolism ; Drosophila melanogaster/genetics ; Drosophila melanogaster/metabolism ; Heart Rate/physiology ; Humans ; Mutation/genetics ; Myocardial Contraction/genetics ; Myocardial Contraction/physiology ; Sarcoplasmic Reticulum/genetics ; Sarcoplasmic Reticulum/metabolism ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Automatic Quantification of Cardiomyocyte Dimensions and Connexin 43 Lateralization in Fluorescence Images.
Autorzy:
Oliver-Gelabert A; Biomedical Signal Interpretation and Computational Simulation (BSICoS), Institute of Engineering Research (I3A), University of Zaragoza & Instituto de Investigación Sanitaria (IIS), 50018 Zaragoza, Spain.
García-Mendívil L; Biomedical Signal Interpretation and Computational Simulation (BSICoS), Institute of Engineering Research (I3A), University of Zaragoza & Instituto de Investigación Sanitaria (IIS), 50018 Zaragoza, Spain.
Vallejo-Gil JM; Department of Cardiovascular Surgery, University Hospital Miguel Servet, 50018 Zaragoza, Spain.
Fresneda-Roldán PC; Department of Cardiovascular Surgery, University Hospital Miguel Servet, 50018 Zaragoza, Spain.
Andelová K; Centre of Experimental Medicine, SAS, 84104 Bratislava, Slovakia.
Fañanás-Mastral J; Department of Cardiovascular Surgery, University Hospital Miguel Servet, 50018 Zaragoza, Spain.
Vázquez-Sancho M; Department of Cardiovascular Surgery, University Hospital Miguel Servet, 50018 Zaragoza, Spain.
Matamala-Adell M; Department of Cardiovascular Surgery, University Hospital Miguel Servet, 50018 Zaragoza, Spain.
Sorribas-Berjón F; Department of Cardiovascular Surgery, University Hospital Miguel Servet, 50018 Zaragoza, Spain.
Ballester-Cuenca C; Department of Cardiovascular Surgery, University Hospital Miguel Servet, 50018 Zaragoza, Spain.
Tribulova N; Centre of Experimental Medicine, SAS, 84104 Bratislava, Slovakia.
Ordovás L; Biomedical Signal Interpretation and Computational Simulation (BSICoS), Institute of Engineering Research (I3A), University of Zaragoza & Instituto de Investigación Sanitaria (IIS), 50018 Zaragoza, Spain.; Aragon Agency for Research and Development (ARAID), 50018 Zaragoza, Spain.
Raúl Diez E; Institute of Experimental Medicine and Biology of Cuyo (IMBECU), CONICET, 855 5500 Mendoza, Argentina.
Pueyo E; Biomedical Signal Interpretation and Computational Simulation (BSICoS), Institute of Engineering Research (I3A), University of Zaragoza & Instituto de Investigación Sanitaria (IIS), 50018 Zaragoza, Spain.; Biomedical Research Networking Center in Bioengineering, Biomaterials and Nanomedicine (CIBER-BBN), 50018 Zaragoza, Spain.
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Źródło:
Biomolecules [Biomolecules] 2020 Sep 17; Vol. 10 (9). Date of Electronic Publication: 2020 Sep 17.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Connexin 43/*metabolism
Microscopy, Fluorescence/*methods
Myocardium/*metabolism
Myocytes, Cardiac/*metabolism
Algorithms ; Animals ; Humans ; Male ; Myocardium/cytology ; Myocytes, Cardiac/cytology ; Rats, Wistar ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Sensitivity analysis revealing the effect of modulating ionic mechanisms on calcium dynamics in simulated human heart failure.
Autorzy:
Mora MT; Centro de Investigación e Innovación en Bioingeniería, Universitat Politècnica de València, Valencia, Spain.
Ferrero JM; Centro de Investigación e Innovación en Bioingeniería, Universitat Politècnica de València, Valencia, Spain.
Romero L; Centro de Investigación e Innovación en Bioingeniería, Universitat Politècnica de València, Valencia, Spain.
Trenor B; Centro de Investigación e Innovación en Bioingeniería, Universitat Politècnica de València, Valencia, Spain.
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Źródło:
PloS one [PLoS One] 2017 Nov 08; Vol. 12 (11), pp. e0187739. Date of Electronic Publication: 2017 Nov 08 (Print Publication: 2017).
Typ publikacji:
Journal Article
MeSH Terms:
Models, Cardiovascular*
Calcium/*metabolism
Heart Failure/*metabolism
Myocytes, Cardiac/*metabolism
Sarcoplasmic Reticulum Calcium-Transporting ATPases/*metabolism
Sodium-Calcium Exchanger/*metabolism
Action Potentials/physiology ; Electric Stimulation ; Excitation Contraction Coupling/physiology ; Gene Expression ; Heart Failure/pathology ; Heart Failure/physiopathology ; Humans ; Ion Transport ; Kinetics ; Myocardial Contraction/physiology ; Myocardium/metabolism ; Myocardium/pathology ; Myocytes, Cardiac/pathology ; Sarcoplasmic Reticulum/metabolism ; Sarcoplasmic Reticulum/pathology ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/genetics ; Sodium-Calcium Exchanger/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
The effects of macrophages on cardiomyocyte calcium-handling function using in vitro culture models.
Autorzy:
Hitscherich PG; Department of Biomedical Engineering, New Jersey Institute of Technology, Newark, New Jersey.
Xie LH; Department of Cell Biology and Molecular Medicine, Rutgers New Jersey Medical School, Newark, New Jersey.
Del Re D; Department of Cell Biology and Molecular Medicine, Rutgers New Jersey Medical School, Newark, New Jersey.
Lee EJ; Department of Biomedical Engineering, New Jersey Institute of Technology, Newark, New Jersey.
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Źródło:
Physiological reports [Physiol Rep] 2019 Jul; Vol. 7 (13), pp. e14137.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms:
Calcium Signaling*
Myocytes, Cardiac/*metabolism
Animals ; Cellular Microenvironment ; Coculture Techniques ; Culture Media, Conditioned/pharmacology ; Mice ; Myocytes, Cardiac/drug effects ; Osteopontin/genetics ; Osteopontin/metabolism ; RAW 264.7 Cells ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/genetics ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/metabolism ; Troponin I/genetics ; Troponin I/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Resveratrol prevents palmitic-acid-induced cardiomyocyte contractile impairment.
Autorzy:
Lieben Louis X; Institute of Cardiovascular Sciences, St. Boniface Hospital Albrechtsen Research Centre, Winnipeg, MB R2H 2A6, Canada.; Department of Physiology and Pathophysiology, University of Manitoba, Winnipeg, MB R3E 0J9, Canada.; Canadian Centre for Agri-Food Research in Health and Medicine, St. Boniface Hospital Albrechtsen, Research Centre, Winnipeg, MB R2H 2A6, Canada.; Department of Biochemistry and Medical Genetics, University of Manitoba, Winnipeg, MB R3E 0J9, Canada.
Raj P; Department of Physiology and Pathophysiology, University of Manitoba, Winnipeg, MB R3E 0J9, Canada.; Canadian Centre for Agri-Food Research in Health and Medicine, St. Boniface Hospital Albrechtsen, Research Centre, Winnipeg, MB R2H 2A6, Canada.
Meikle Z; Institute of Cardiovascular Sciences, St. Boniface Hospital Albrechtsen Research Centre, Winnipeg, MB R2H 2A6, Canada.; Department of Biochemistry and Medical Genetics, University of Manitoba, Winnipeg, MB R3E 0J9, Canada.
Yu L; Canadian Centre for Agri-Food Research in Health and Medicine, St. Boniface Hospital Albrechtsen, Research Centre, Winnipeg, MB R2H 2A6, Canada.; Agriculture and Agri-Food Canada, Winnipeg, MB R2H 2A6, Canada.
Susser SE; Institute of Cardiovascular Sciences, St. Boniface Hospital Albrechtsen Research Centre, Winnipeg, MB R2H 2A6, Canada.; Department of Physiology and Pathophysiology, University of Manitoba, Winnipeg, MB R3E 0J9, Canada.
MacInnis S; Institute of Cardiovascular Sciences, St. Boniface Hospital Albrechtsen Research Centre, Winnipeg, MB R2H 2A6, Canada.
Duhamel TA; Institute of Cardiovascular Sciences, St. Boniface Hospital Albrechtsen Research Centre, Winnipeg, MB R2H 2A6, Canada.; Health, Leisure & Human Performance Research Institute, Faculty of Kinesiology & Recreation Management, University of Manitoba, MB R3E 0J9, Canada.
Wigle JT; Institute of Cardiovascular Sciences, St. Boniface Hospital Albrechtsen Research Centre, Winnipeg, MB R2H 2A6, Canada.; Department of Biochemistry and Medical Genetics, University of Manitoba, Winnipeg, MB R3E 0J9, Canada.
Netticadan T; Department of Physiology and Pathophysiology, University of Manitoba, Winnipeg, MB R3E 0J9, Canada.; Canadian Centre for Agri-Food Research in Health and Medicine, St. Boniface Hospital Albrechtsen, Research Centre, Winnipeg, MB R2H 2A6, Canada.; Agriculture and Agri-Food Canada, Winnipeg, MB R2H 2A6, Canada.
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Źródło:
Canadian journal of physiology and pharmacology [Can J Physiol Pharmacol] 2019 Dec; Vol. 97 (12), pp. 1132-1140. Date of Electronic Publication: 2019 Aug 02.
Typ publikacji:
Journal Article
MeSH Terms:
Myocardial Contraction/*drug effects
Myocytes, Cardiac/*drug effects
Myocytes, Cardiac/*physiology
Palmitic Acid/*adverse effects
Resveratrol/*pharmacology
Animals ; Apoptosis/drug effects ; Gene Expression Regulation/drug effects ; Male ; Myocytes, Cardiac/cytology ; Myocytes, Cardiac/metabolism ; Rats ; Rats, Sprague-Dawley ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/metabolism ; Troponin I/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
AKAP6 and phospholamban colocalize and interact in HEK-293T cells and primary murine cardiomyocytes.
Autorzy:
Hakem Zadeh F; Department of Physiology, Faculty of Medicine, University of Toronto, Toronto, Ontario.; Translational Biology and Engineering Program (TBEP), Ted Rogers Centre for Heart Research, Toronto, Ontario.
Teng ACT; Department of Physiology, Faculty of Medicine, University of Toronto, Toronto, Ontario.; Translational Biology and Engineering Program (TBEP), Ted Rogers Centre for Heart Research, Toronto, Ontario.
Kuzmanov U; Translational Biology and Engineering Program (TBEP), Ted Rogers Centre for Heart Research, Toronto, Ontario.
Chambers PJ; Department of Kinesiology, University of Waterloo, Waterloo, Ontario.
Tupling AR; Department of Kinesiology, University of Waterloo, Waterloo, Ontario.
Gramolini AO; Department of Physiology, Faculty of Medicine, University of Toronto, Toronto, Ontario.; Translational Biology and Engineering Program (TBEP), Ted Rogers Centre for Heart Research, Toronto, Ontario.
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Źródło:
Physiological reports [Physiol Rep] 2019 Jul; Vol. 7 (14), pp. e14144.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
A Kinase Anchor Proteins/*metabolism
Calcium-Binding Proteins/*metabolism
Myocytes, Cardiac/*metabolism
Animals ; Calcium Signaling ; Cells, Cultured ; HEK293 Cells ; Humans ; Mice ; Protein Binding ; Rats ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Nanoscale reorganization of sarcoplasmic reticulum in pressure-overload cardiac hypertrophy visualized by dSTORM.
Autorzy:
Hadipour-Lakmehsari S; Translational Biology and Engineering Program, Ted Rogers Centre for Heart Research, Toronto, Ontario, M5G 1M1, Canada.; Department of Physiology, Faculty of Medicine, University of Toronto, Toronto, Ontario, M5S 1M8, Canada.
Driouchi A; Department of Biochemistry, Faculty of Medicine, University of Toronto, Toronto, Ontario, M5S 1A8, Canada.; Institute of Biomaterials and Biomedical Engineering, University of Toronto, Toronto, Ontario, M5S 3G9, Canada.; Terrence Donnelly Centre for Cellular and Biomolecular Research, University of Toronto, Toronto, Ontario, M5S 3E1, Canada.
Lee SH; Translational Biology and Engineering Program, Ted Rogers Centre for Heart Research, Toronto, Ontario, M5G 1M1, Canada.; Department of Physiology, Faculty of Medicine, University of Toronto, Toronto, Ontario, M5S 1M8, Canada.
Kuzmanov U; Translational Biology and Engineering Program, Ted Rogers Centre for Heart Research, Toronto, Ontario, M5G 1M1, Canada.; Terrence Donnelly Centre for Cellular and Biomolecular Research, University of Toronto, Toronto, Ontario, M5S 3E1, Canada.
Callaghan NI; Translational Biology and Engineering Program, Ted Rogers Centre for Heart Research, Toronto, Ontario, M5G 1M1, Canada.; Institute of Biomaterials and Biomedical Engineering, University of Toronto, Toronto, Ontario, M5S 3G9, Canada.
Heximer SP; Translational Biology and Engineering Program, Ted Rogers Centre for Heart Research, Toronto, Ontario, M5G 1M1, Canada.; Department of Physiology, Faculty of Medicine, University of Toronto, Toronto, Ontario, M5S 1M8, Canada.
Simmons CA; Translational Biology and Engineering Program, Ted Rogers Centre for Heart Research, Toronto, Ontario, M5G 1M1, Canada.; Institute of Biomaterials and Biomedical Engineering, University of Toronto, Toronto, Ontario, M5S 3G9, Canada.
Yip CM; Department of Biochemistry, Faculty of Medicine, University of Toronto, Toronto, Ontario, M5S 1A8, Canada. .; Institute of Biomaterials and Biomedical Engineering, University of Toronto, Toronto, Ontario, M5S 3G9, Canada. .; Terrence Donnelly Centre for Cellular and Biomolecular Research, University of Toronto, Toronto, Ontario, M5S 3E1, Canada. .
Gramolini AO; Translational Biology and Engineering Program, Ted Rogers Centre for Heart Research, Toronto, Ontario, M5G 1M1, Canada. .; Department of Physiology, Faculty of Medicine, University of Toronto, Toronto, Ontario, M5S 1M8, Canada. .
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Źródło:
Scientific reports [Sci Rep] 2019 May 27; Vol. 9 (1), pp. 7867. Date of Electronic Publication: 2019 May 27.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cardiomegaly/*pathology
Myocytes, Cardiac/*pathology
Sarcoplasmic Reticulum/*pathology
Animals ; Calcium Channels, L-Type/analysis ; Calcium-Binding Proteins/analysis ; Cardiomegaly/diagnostic imaging ; Cells, Cultured ; Fourier Analysis ; Mice ; Microscopy ; Optical Imaging ; Pressure ; Ryanodine Receptor Calcium Release Channel/analysis ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/analysis
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
TGR5 activation ameliorates hyperglycemia-induced cardiac hypertrophy in H9c2 cells.
Autorzy:
Cheng KC; Department of Psychosomatic Internal Medicine, Kagoshima University Graduate School of Medical and Dental Sciences, Kagoshima, 890, Japan.
Chang WT; Department of Cardiology, Chi-Mei Medical Center, Yong Kang, Tainan City, 71003, Taiwan.; Department of Medical Research, Chi-Mei Medical Center, Yong Kang, Tainan City, 71003, Taiwan.
Kuo FY; Cardiovascular Center, Veterans General Hospital, Kaohsiung City, 81362, Taiwan.
Chen ZC; Department of Cardiology, Chi-Mei Medical Center, Yong Kang, Tainan City, 71003, Taiwan.; Department of Pharmacy, Chia Nan University of Pharmacy & Science, Jean-Tae City, Tainan County, 71701, Taiwan.
Li Y; Department of Psychosomatic Internal Medicine, Kagoshima University Graduate School of Medical and Dental Sciences, Kagoshima, 890, Japan. .; Department of Medical Research, Chi-Mei Medical Center, Yong Kang, Tainan City, 71003, Taiwan. .
Cheng JT; Department of Medical Research, Chi-Mei Medical Center, Yong Kang, Tainan City, 71003, Taiwan. .
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Źródło:
Scientific reports [Sci Rep] 2019 Mar 06; Vol. 9 (1), pp. 3633. Date of Electronic Publication: 2019 Mar 06.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Oxidative Stress*
Cardiomegaly/*prevention & control
Glucose/*pharmacology
Hyperglycemia/*complications
Myocytes, Cardiac/*pathology
Receptors, G-Protein-Coupled/*metabolism
Cardiomegaly/etiology ; Cardiomegaly/metabolism ; Cardiomegaly/pathology ; Cells, Cultured ; Humans ; Myocytes, Cardiac/drug effects ; Myocytes, Cardiac/metabolism ; Phosphorylation ; Receptors, G-Protein-Coupled/genetics ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/metabolism ; Sweetening Agents/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Cardiac-directed expression of a catalytically inactive adenylyl cyclase 6 protects the heart from sustained β-adrenergic stimulation.
Autorzy:
Gao MH; VA San Diego Healthcare System, Department of Medicine, Division of Cardiology, San Diego CA, United States of America.; University of California, San Diego, Department of Medicine, Division of Cardiology, La Jolla CA, United States of America.
Lai NC; VA San Diego Healthcare System, Department of Medicine, Division of Cardiology, San Diego CA, United States of America.; University of California, San Diego, Department of Medicine, Division of Cardiology, La Jolla CA, United States of America.
Giamouridis D; VA San Diego Healthcare System, Department of Medicine, Division of Cardiology, San Diego CA, United States of America.; University of California, San Diego, Department of Medicine, Division of Cardiology, La Jolla CA, United States of America.
Kim YC; VA San Diego Healthcare System, Department of Medicine, Division of Cardiology, San Diego CA, United States of America.; University of California, San Diego, Department of Medicine, Division of Cardiology, La Jolla CA, United States of America.
Guo T; VA San Diego Healthcare System, Department of Medicine, Division of Cardiology, San Diego CA, United States of America.; University of California, San Diego, Department of Medicine, Division of Cardiology, La Jolla CA, United States of America.
Hammond HK; VA San Diego Healthcare System, Department of Medicine, Division of Cardiology, San Diego CA, United States of America.; University of California, San Diego, Department of Medicine, Division of Cardiology, La Jolla CA, United States of America.
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Źródło:
PloS one [PLoS One] 2017 Aug 02; Vol. 12 (8), pp. e0181282. Date of Electronic Publication: 2017 Aug 02 (Print Publication: 2017).
Typ publikacji:
Journal Article
MeSH Terms:
Mutation*
Adenylyl Cyclases/*genetics
Adrenergic beta-Agonists/*administration & dosage
Isoproterenol/*administration & dosage
Myocytes, Cardiac/*cytology
Ventricular Function, Left/*drug effects
Adenylyl Cyclases/metabolism ; Adrenergic beta-Agonists/pharmacology ; Animals ; Apoptosis ; Caspases/genetics ; Caspases/metabolism ; Gene Expression Regulation/drug effects ; Isoproterenol/pharmacology ; Male ; Mice ; Mice, Transgenic ; Myocytes, Cardiac/drug effects ; Myocytes, Cardiac/metabolism ; Proto-Oncogene Proteins c-bcl-2/genetics ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/genetics ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/metabolism ; Stroke Volume/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Mechanisms of TFAM-mediated cardiomyocyte protection.
Autorzy:
Kunkel GH; Department of Physiology and Biophysics, University of Louisville, KY, USA.; Department of Physiology and Biophysics, University of Louisville, KY, USA.
Chaturvedi P; Department of Physiology and Biophysics, University of Louisville, KY, USA.; Department of Physiology and Biophysics, University of Louisville, KY, USA.
Thelian N; Department of Physiology and Biophysics, University of Louisville, KY, USA.; Department of Physiology and Biophysics, University of Louisville, KY, USA.
Nair R; Department of Physiology and Biophysics, University of Louisville, KY, USA.; Department of Physiology and Biophysics, University of Louisville, KY, USA.
Tyagi SC; Department of Physiology and Biophysics, University of Louisville, KY, USA.; Department of Physiology and Biophysics, University of Louisville, KY, USA.
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Źródło:
Canadian journal of physiology and pharmacology [Can J Physiol Pharmacol] 2018 Feb; Vol. 96 (2), pp. 173-181. Date of Electronic Publication: 2017 Aug 11.
Typ publikacji:
Journal Article
MeSH Terms:
DNA-Binding Proteins/*metabolism
Mitochondrial Proteins/*metabolism
Myocytes, Cardiac/*metabolism
Transcription Factors/*metabolism
Animals ; CRISPR-Cas Systems/genetics ; Calpain/metabolism ; Cell Line ; Hydrogen Peroxide/pharmacology ; Matrix Metalloproteinase 9/metabolism ; Mice ; Myocytes, Cardiac/drug effects ; NFATC Transcription Factors/metabolism ; Peptide Hydrolases/metabolism ; Sarcoplasmic Reticulum Calcium-Transporting ATPases
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
HDAC Inhibition Improves the Sarcoendoplasmic Reticulum Ca -ATPase Activity in Cardiac Myocytes.
Autorzy:
Meraviglia V; Institute for Biomedicine, Eurac Research, 39100 Bolzano, Italy (affiliated institute of the University of Lübeck, 23562 Lübeck, Germany). .
Bocchi L; Department of Chemistry, Life Sciences and Environmental Sustainability, University of Parma, 43124 Parma, Italy. .
Sacchetto R; Department of Comparative Biomedicine and Food Science, University of Padova, 35020 Legnaro (Padova), Italy. .
Florio MC; Institute for Biomedicine, Eurac Research, 39100 Bolzano, Italy (affiliated institute of the University of Lübeck, 23562 Lübeck, Germany). .
Motta BM; Institute for Biomedicine, Eurac Research, 39100 Bolzano, Italy (affiliated institute of the University of Lübeck, 23562 Lübeck, Germany). .
Corti C; Institute for Biomedicine, Eurac Research, 39100 Bolzano, Italy (affiliated institute of the University of Lübeck, 23562 Lübeck, Germany). .
Weichenberger CX; Institute for Biomedicine, Eurac Research, 39100 Bolzano, Italy (affiliated institute of the University of Lübeck, 23562 Lübeck, Germany). .
Savi M; Department of Chemistry, Life Sciences and Environmental Sustainability, University of Parma, 43124 Parma, Italy. .
D'Elia Y; Institute for Biomedicine, Eurac Research, 39100 Bolzano, Italy (affiliated institute of the University of Lübeck, 23562 Lübeck, Germany). .
Rosato-Siri MD; Institute for Biomedicine, Eurac Research, 39100 Bolzano, Italy (affiliated institute of the University of Lübeck, 23562 Lübeck, Germany). .
Suffredini S; Institute for Biomedicine, Eurac Research, 39100 Bolzano, Italy (affiliated institute of the University of Lübeck, 23562 Lübeck, Germany). .
Piubelli C; Institute for Biomedicine, Eurac Research, 39100 Bolzano, Italy (affiliated institute of the University of Lübeck, 23562 Lübeck, Germany). .
Pompilio G; Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino, IRCCS, 20138 Milano, Italy. .; Dipartimento di Scienze Cliniche e di Comunità, Università degli Studi di Milano, 20122 Milano, Italy. .
Pramstaller PP; Institute for Biomedicine, Eurac Research, 39100 Bolzano, Italy (affiliated institute of the University of Lübeck, 23562 Lübeck, Germany). .
Domingues FS; Institute for Biomedicine, Eurac Research, 39100 Bolzano, Italy (affiliated institute of the University of Lübeck, 23562 Lübeck, Germany). .
Stilli D; Department of Chemistry, Life Sciences and Environmental Sustainability, University of Parma, 43124 Parma, Italy. .
Rossini A; Institute for Biomedicine, Eurac Research, 39100 Bolzano, Italy (affiliated institute of the University of Lübeck, 23562 Lübeck, Germany). .
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2018 Jan 31; Vol. 19 (2). Date of Electronic Publication: 2018 Jan 31.
Typ publikacji:
Journal Article
MeSH Terms:
Protein Processing, Post-Translational*
Hydroxamic Acids/*pharmacology
Myocytes, Cardiac/*drug effects
Sarcoplasmic Reticulum Calcium-Transporting ATPases/*metabolism
Acetylation ; Animals ; Histone Deacetylase Inhibitors/pharmacology ; Humans ; Male ; Myocytes, Cardiac/enzymology ; Myocytes, Cardiac/metabolism ; Rats ; Rats, Wistar ; Vorinostat
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Cardiotoxic Effects of Short-Term Doxorubicin Administration: Involvement of Connexin 43 in Calcium Impairment.
Autorzy:
Pecoraro M; Department of Pharmacy, University of Salerno, Fisciano (SA) 84084, Italy. .
Rodríguez-Sinovas A; Cardiovascular Diseases Research Group, Department of Cardiology, Vall d'Hebron University Hospital and Research Institute, Universitat Autònoma de Barcelona (UAB), Barcelona 08035, Spain. .
Marzocco S; Department of Pharmacy, University of Salerno, Fisciano (SA) 84084, Italy. .
Ciccarelli M; Department of Medicine and Surgery, University of Salerno, Baronissi (SA) 84084, Italy. .
Iaccarino G; Department of Medicine and Surgery, University of Salerno, Baronissi (SA) 84084, Italy. .
Pinto A; Department of Pharmacy, University of Salerno, Fisciano (SA) 84084, Italy. .
Popolo A; Department of Pharmacy, University of Salerno, Fisciano (SA) 84084, Italy. .
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2017 Oct 11; Vol. 18 (10). Date of Electronic Publication: 2017 Oct 11.
Typ publikacji:
Journal Article
MeSH Terms:
Antibiotics, Antineoplastic/*adverse effects
Calcium/*metabolism
Cardiomyopathies/*chemically induced
Connexin 43/*metabolism
Doxorubicin/*adverse effects
Myocytes, Cardiac/*drug effects
Animals ; Antibiotics, Antineoplastic/administration & dosage ; Calcium/analysis ; Calcium-Binding Proteins/metabolism ; Cardiomyopathies/metabolism ; Cardiotoxicity/etiology ; Disease Models, Animal ; Doxorubicin/administration & dosage ; Female ; Mice ; Mice, Inbred C57BL ; Myocytes, Cardiac/metabolism ; Primary Cell Culture ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/metabolism ; Time Factors
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Inhibition of mevalonate pathway prevents ischemia-induced cardiac dysfunction in rats via RhoA-independent signaling pathway.
Autorzy:
Yang Y; Department of Cardiology, Sir Run Run Shaw Hospital, College of Medicine, Zhejiang University, Hangzhou, China.
Rong X; Department of Cardiology, Sir Run Run Shaw Hospital, College of Medicine, Zhejiang University, Hangzhou, China.
Lv X; Department of Cardiology, Sir Run Run Shaw Hospital, College of Medicine, Zhejiang University, Hangzhou, China.
Jiang W; Department of Cardiology, Wenzhou People's Hospital, Wenzhou, China.
Yang Y; Department of Cardiology, Sir Run Run Shaw Hospital, College of Medicine, Zhejiang University, Hangzhou, China.
Lai D; Department of Cardiology, Sir Run Run Shaw Hospital, College of Medicine, Zhejiang University, Hangzhou, China.
Xu S; Department of Cardiology, Sir Run Run Shaw Hospital, College of Medicine, Zhejiang University, Hangzhou, China.; Institute of Translational Medicine, College of Medicine, Zhejiang University, Hangzhou, China.
Fu G; Department of Cardiology, Sir Run Run Shaw Hospital, College of Medicine, Zhejiang University, Hangzhou, China.
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Źródło:
Cardiovascular therapeutics [Cardiovasc Ther] 2017 Oct; Vol. 35 (5).
Typ publikacji:
Journal Article
MeSH Terms:
Alendronate/*pharmacology
Cardiomyopathy, Dilated/*prevention & control
Hydroxymethylglutaryl-CoA Reductase Inhibitors/*pharmacology
Mevalonic Acid/*metabolism
Myocardial Infarction/*drug therapy
Myocytes, Cardiac/*drug effects
Rosuvastatin Calcium/*pharmacology
Ventricular Function, Left/*drug effects
Ventricular Remodeling/*drug effects
rho GTP-Binding Proteins/*metabolism
Animals ; Calcium Signaling/drug effects ; Calcium-Binding Proteins/metabolism ; Cardiomyopathy, Dilated/enzymology ; Cardiomyopathy, Dilated/pathology ; Cardiomyopathy, Dilated/physiopathology ; Cell Line ; Disease Models, Animal ; Geranyltranstransferase/antagonists & inhibitors ; Geranyltranstransferase/metabolism ; Male ; Myocardial Infarction/enzymology ; Myocardial Infarction/pathology ; Myocardial Infarction/physiopathology ; Myocytes, Cardiac/enzymology ; Myocytes, Cardiac/pathology ; Rats, Sprague-Dawley ; Ryanodine Receptor Calcium Release Channel/genetics ; Ryanodine Receptor Calcium Release Channel/metabolism ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/metabolism ; Tacrolimus Binding Proteins/genetics ; Tacrolimus Binding Proteins/metabolism ; rho GTP-Binding Proteins/antagonists & inhibitors
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Increase in Cardiac Ischemia-Reperfusion Injuries in Opa1+/- Mouse Model.
Autorzy:
Le Page S; Institut MITOVASC, Université Angers, CHU Angers, Angers, France.; Laboratoire Cardioprotection Remodelage et Thrombose, Angers, France.; Service de Cardiologie, CHU Angers, Angers, France.
Niro M; Institut MITOVASC, Université Angers, CHU Angers, Angers, France.; Laboratoire Cardioprotection Remodelage et Thrombose, Angers, France.; Service de Cardiologie, CHU Angers, Angers, France.
Fauconnier J; INSERM U1046, Université Montpellier I et II, Montpellier, France.
Cellier L; Institut MITOVASC, Université Angers, CHU Angers, Angers, France.; Laboratoire Cardioprotection Remodelage et Thrombose, Angers, France.
Tamareille S; Institut MITOVASC, Université Angers, CHU Angers, Angers, France.; Laboratoire Cardioprotection Remodelage et Thrombose, Angers, France.
Gharib A; INSERM UMR 1060, CarMeN, Lyon, France.
Chevrollier A; Institut MITOVASC, Université Angers, CHU Angers, Angers, France.; INSERM UMR_S1083, CNRS UMR_C6214, BNMI, Angers, France.
Loufrani L; Institut MITOVASC, Université Angers, CHU Angers, Angers, France.; INSERM UMR_S1083, CNRS UMR_C6214, BNMI, Angers, France.
Grenier C; Institut MITOVASC, Université Angers, CHU Angers, Angers, France.; INSERM UMR_S1083, CNRS UMR_C6214, BNMI, Angers, France.
Kamel R; Institut MITOVASC, Université Angers, CHU Angers, Angers, France.; Laboratoire Cardioprotection Remodelage et Thrombose, Angers, France.
Sarzi E; Institut des Neurosciences de Montpellier, INSERM U1051, Université Montpellier I et II, Montpellier, France.
Lacampagne A; INSERM U1046, Université Montpellier I et II, Montpellier, France.
Ovize M; INSERM UMR 1060, CarMeN, Lyon, France.
Henrion D; Institut MITOVASC, Université Angers, CHU Angers, Angers, France.; INSERM UMR_S1083, CNRS UMR_C6214, BNMI, Angers, France.
Reynier P; Institut MITOVASC, Université Angers, CHU Angers, Angers, France.; INSERM UMR_S1083, CNRS UMR_C6214, BNMI, Angers, France.
Lenaers G; Institut MITOVASC, Université Angers, CHU Angers, Angers, France.; INSERM UMR_S1083, CNRS UMR_C6214, BNMI, Angers, France.
Mirebeau-Prunier D; Institut MITOVASC, Université Angers, CHU Angers, Angers, France.; INSERM UMR_S1083, CNRS UMR_C6214, BNMI, Angers, France.
Prunier F; Institut MITOVASC, Université Angers, CHU Angers, Angers, France.; Laboratoire Cardioprotection Remodelage et Thrombose, Angers, France.; Service de Cardiologie, CHU Angers, Angers, France.
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Źródło:
PloS one [PLoS One] 2016 Oct 10; Vol. 11 (10), pp. e0164066. Date of Electronic Publication: 2016 Oct 10 (Print Publication: 2016).
Typ publikacji:
Journal Article
MeSH Terms:
Mitochondrial Dynamics*
Calcium/*metabolism
GTP Phosphohydrolases/*metabolism
Mitochondria, Heart/*metabolism
Myocardial Reperfusion Injury/*metabolism
Myocytes, Cardiac/*metabolism
Animals ; Disease Models, Animal ; GTP Phosphohydrolases/genetics ; Homeodomain Proteins/genetics ; Homeodomain Proteins/metabolism ; Mice ; Mice, Mutant Strains ; Mitochondria, Heart/genetics ; Mitochondrial Membrane Transport Proteins/genetics ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; Myocardial Reperfusion Injury/genetics ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/genetics ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Cardiomyocyte-Specific Ablation of Med1 Subunit of the Mediator Complex Causes Lethal Dilated Cardiomyopathy in Mice.
Autorzy:
Jia Y; Department of Pathology, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, United States of America.
Chang HC; Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, United States of America.
Schipma MJ; Next Generation Sequencing Core Facility, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, United States of America.
Liu J; Department of Pathology, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, United States of America.
Shete V; Department of Pathology, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, United States of America.
Liu N; Department of Pathology, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, United States of America.
Sato T; Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, United States of America.
Thorp EB; Department of Pathology, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, United States of America.
Barger PM; Center for Cardiovascular Research, Washington University School of Medicine, St. Louis, Missouri, United States of America.
Zhu YJ; Department of Pathology, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, United States of America.
Viswakarma N; Department of Surgery, University of Illinois at Chicago, Chicago, Illinois, United States of America.
Kanwar YS; Department of Pathology, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, United States of America.
Ardehali H; Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, United States of America.
Thimmapaya B; Department of Microbiology and Immunology, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, United States of America.
Reddy JK; Department of Pathology, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, United States of America.
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Źródło:
PloS one [PLoS One] 2016 Aug 22; Vol. 11 (8), pp. e0160755. Date of Electronic Publication: 2016 Aug 22 (Print Publication: 2016).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Genes, Lethal*
Cardiomyopathy, Dilated/*genetics
Heart Failure/*genetics
Mediator Complex Subunit 1/*genetics
Mitochondria/*metabolism
Myocytes, Cardiac/*metabolism
Animals ; Apoptosis ; Cadherins/genetics ; Cadherins/metabolism ; Calcium Channels, L-Type/genetics ; Calcium Channels, L-Type/metabolism ; Calcium Signaling ; Cardiomyopathy, Dilated/metabolism ; Cardiomyopathy, Dilated/pathology ; Cyclic Nucleotide Phosphodiesterases, Type 1/genetics ; Cyclic Nucleotide Phosphodiesterases, Type 1/metabolism ; Embryo, Mammalian ; Energy Metabolism ; Female ; Gene Deletion ; Gene Expression Profiling ; Gene Expression Regulation ; Gestational Age ; Heart Failure/metabolism ; Heart Failure/pathology ; Mediator Complex Subunit 1/deficiency ; Mice ; Mice, Knockout ; Mitochondria/pathology ; Myocardial Contraction ; Myocytes, Cardiac/pathology ; Peroxisome Proliferator-Activated Receptors/genetics ; Peroxisome Proliferator-Activated Receptors/metabolism ; Pregnancy ; Ryanodine Receptor Calcium Release Channel/genetics ; Ryanodine Receptor Calcium Release Channel/metabolism ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/genetics ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
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