The E3 ubiquitin ligase Cul4b promotes CD4+ T cell expansion by aiding the repair of damaged DNA.
Autorzy:
Dar AA; Division of Protective Immunity, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, United States of America. Sawada K; Division of Protective Immunity, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, United States of America. Dybas JM; Division of Protective Immunity, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, United States of America.; Department of Biomedical Health and Informatics, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, United States of America. Moser EK; Division of Protective Immunity, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, United States of America. Lewis EL; Medical Scientist Training Program, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America. Park E; Center for Computational and Genomic Medicine, The Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, United States of America. Fazelinia H; Division of Cell Pathology, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, United States of America. Spruce LA; Division of Cell Pathology, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, United States of America. Ding H; Division of Cell Pathology, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, United States of America. SeeholzerSH; Division of Cell Pathology, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, United States of America. Oliver PM; Division of Protective Immunity, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, United States of America.; Department of Pathology, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
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Źródło:
PLoS biology [PLoS Biol] 2021 Feb 01; Vol. 19 (2), pp. e3001041. Date of Electronic Publication: 2021 Feb 01 (Print Publication: 2021).
Regulation of Janus Kinase 2 by an Inflammatory Bowel Disease Causal Non-coding Single Nucleotide Polymorphism.
Autorzy:
Cardinale CJ; Center for Applied Genomics, Children's Hospital of Philadelphia, Philadelphia, PA, USA. March ME; Center for Applied Genomics, Children's Hospital of Philadelphia, Philadelphia, PA, USA. Lin X; Department of Computer Science, New Jersey Institute of Technology, Newark, NJ, USA. Liu Y; Center for Applied Genomics, Children's Hospital of Philadelphia, Philadelphia, PA, USA. Spruce LA; Proteomics Core Facility, Children's Hospital of Philadelphia, Philadelphia, PA, USA. Bradfield JP; Quantinuum Research LLC, Philadelphia, PA, USA. Wei Z; Department of Computer Science, New Jersey Institute of Technology, Newark, NJ, USA. SeeholzerSH; Proteomics Core Facility, Children's Hospital of Philadelphia, Philadelphia, PA, USA. Grant SFA; Center for Applied Genomics, Children's Hospital of Philadelphia, Philadelphia, PA, USA.; Center for Spatial and Functional Genomics, Children's Hospital of Philadelphia, Philadelphia, PA, USA.; Department of Pediatrics, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, PA, USA. Hakonarson H; Center for Applied Genomics, Children's Hospital of Philadelphia, Philadelphia, PA, USA.; Department of Pediatrics, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, PA, USA.
Quantitative phosphoproteomics of CXCL12 (SDF-1) signaling.
Autorzy:
Wojcechowskyj JA; Department of Microbiology, University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania, United States of America. Lee JY SeeholzerSH Doms RW
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Źródło:
PloS one [PLoS One] 2011; Vol. 6 (9), pp. e24918. Date of Electronic Publication: 2011 Sep 20.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
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