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Wyświetlanie 1-20 z 227
Tytuł:
Compensatory Protection of Thioredoxin-Deficient Cells from Etoposide-Induced Cell Death by Selenoprotein W via Interaction with 14-3-3.
Autorzy:
Kang H; Laboratory of Cellular and Molecular Biochemistry, Department of Life Sciences, Korea University, Seoul 02841, Korea.
Jeon YH; Laboratory of Cellular and Molecular Biochemistry, Department of Life Sciences, Korea University, Seoul 02841, Korea.
Ham M; Laboratory of Cellular and Molecular Biochemistry, Department of Life Sciences, Korea University, Seoul 02841, Korea.
Ko K; Laboratory of Cellular and Molecular Biochemistry, Department of Life Sciences, Korea University, Seoul 02841, Korea.
Kim IY; Laboratory of Cellular and Molecular Biochemistry, Department of Life Sciences, Korea University, Seoul 02841, Korea.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Sep 25; Vol. 22 (19). Date of Electronic Publication: 2021 Sep 25.
Typ publikacji:
Journal Article
MeSH Terms:
14-3-3 Proteins/*metabolism
Cell Death/*drug effects
Etoposide/*pharmacology
Selenoprotein W/*pharmacology
Thioredoxins/*metabolism
Animals ; Cell Line ; Cell Line, Tumor ; HEK293 Cells ; Humans ; MCF-7 Cells ; Mice ; Oxidation-Reduction/drug effects ; Phosphorylation/drug effects ; Poly (ADP-Ribose) Polymerase-1/metabolism ; Protein Binding/drug effects ; Signal Transduction/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Role of Selenoprotein W in participating in the progression of non-alcoholic fatty liver disease
Autorzy:
Zhiruo Miao
Wei Wang
Zhiying Miao
Qiyuan Cao
Shiwen Xu
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Temat:
Selenoprotein W
Non-alcoholic fatty liver disease
Metabolism
Glycolysis
Pyruvate kinase M2
Pyroptosis
Medicine (General)
R5-920
Biology (General)
QH301-705.5
Źródło:
Redox Biology, Vol 71, Iss , Pp 103114- (2024)
Opis pliku:
electronic resource
Relacje:
http://www.sciencedirect.com/science/article/pii/S2213231724000909; https://doaj.org/toc/2213-2317
Dostęp URL:
https://doaj.org/article/60c17e58aa8b4d82bdb930ea18c0947e  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Tytuł:
Lesion of striatal patches disrupts habitual behaviors and increases behavioral variability.
Autorzy:
Nadel JA; Neuroscience Department, Oberlin College, Oberlin, OH, United States of America.; Laboratory for Integrative Neuroscience, National Institute on Alcohol Abuse and Alcoholism, US National Institutes of Health, Rockville, Maryland, United States of America.
Pawelko SS; Neuroscience Department, Oberlin College, Oberlin, OH, United States of America.
Copes-Finke D; Neuroscience Department, Oberlin College, Oberlin, OH, United States of America.
Neidhart M; Neuroscience Department, Oberlin College, Oberlin, OH, United States of America.
Howard CD; Neuroscience Department, Oberlin College, Oberlin, OH, United States of America.
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Źródło:
PloS one [PLoS One] 2020 Jan 08; Vol. 15 (1), pp. e0224715. Date of Electronic Publication: 2020 Jan 08 (Print Publication: 2020).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Behavior, Animal/*physiology
Corpus Striatum/*physiology
Neostriatum/*physiology
Neurons/*physiology
Animals ; Caspase 3/genetics ; Central Nervous System Stimulants/pharmacology ; Dopamine/metabolism ; Habits ; Integrases/genetics ; Locomotion/physiology ; Male ; Mice ; Mice, Transgenic ; Motivation/genetics ; Motivation/physiology ; Selenoprotein W
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Selenoprotein W depletion in vitro might indicate that its main function is not as an antioxidative enzyme.
Autorzy:
Xiao-Long W; Biomedicine Division, Wildlife Resource College, Northeast Forestry University, Ministry of Education, P. R. China.
Chuan-Ping Y
Kai X
Ou-Jv Q
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Źródło:
Biochemistry. Biokhimiia [Biochemistry (Mosc)] 2010 Feb; Vol. 75 (2), pp. 201-7.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antioxidants/*metabolism
Selenoprotein W/*deficiency
Selenoprotein W/*metabolism
Animals ; Apoptosis ; Base Sequence ; Cell Line ; Cell Survival/genetics ; Gene Knockdown Techniques ; Glutathione/metabolism ; Mice ; Muscle, Skeletal/cytology ; Muscle, Skeletal/enzymology ; Muscle, Skeletal/metabolism ; RNA Interference ; RNA, Small Interfering/genetics ; Selenoprotein W/genetics ; Transfection
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
PIWI-interacting RNA-36712 restrains breast cancer progression and chemoresistance by interaction with SEPW1 pseudogene SEPW1P RNA.
Autorzy:
Tan L; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Mai D; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Zhang B; Department of Breast Surgery, National Cancer Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Jiang X; Department of Surgical Oncology, Sun Yat-sen University Cancer Center, Guangzhou, China.
Zhang J; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Bai R; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Ye Y; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Li M; Department of Pathology, Sun Yat-sen University Cancer Center, Guangzhou, China.
Pan L; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Su J; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Zheng Y; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Liu Z; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Zuo Z; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Zhao Q; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Li X; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Huang X; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Yang J; Department of Etiology and Carcinogenesis, National Cancer Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Tan W; Department of Etiology and Carcinogenesis, National Cancer Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
Zheng J; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China. .
Lin D; State Key Laboratory of Oncology in South China and Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.; Department of Etiology and Carcinogenesis, National Cancer Center/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.
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Źródło:
Molecular cancer [Mol Cancer] 2019 Jan 12; Vol. 18 (1), pp. 9. Date of Electronic Publication: 2019 Jan 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Breast Neoplasms/*drug therapy
Breast Neoplasms/*genetics
RNA, Small Interfering/*genetics
Selenoprotein W/*genetics
Animals ; Breast/drug effects ; Breast/pathology ; Breast Neoplasms/metabolism ; Breast Neoplasms/pathology ; Cell Proliferation/drug effects ; Cell Proliferation/genetics ; Disease Progression ; Down-Regulation ; Doxorubicin/pharmacology ; Drug Resistance, Neoplasm ; Female ; Humans ; MicroRNAs/genetics ; Paclitaxel/pharmacology ; Prognosis ; Pseudogenes ; RNA, Messenger/genetics ; RNA, Small Interfering/biosynthesis ; Up-Regulation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Selenoprotein T deficiency alters cell adhesion and elevates selenoprotein W expression in murine fibroblast cells.
Autorzy:
Sengupta A; Molecular Biology of Selenium Section, Laboratory of Cancer Prevention, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.
Carlson BA
Labunskyy VM
Gladyshev VN
Hatfield DL
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Źródło:
Biochemistry and cell biology = Biochimie et biologie cellulaire [Biochem Cell Biol] 2009 Dec; Vol. 87 (6), pp. 953-61.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural
MeSH Terms:
Cell Adhesion/*physiology
Fibroblasts/*metabolism
Selenoprotein W/*metabolism
Selenoproteins/*deficiency
Animals ; Fibroblasts/cytology ; Gene Expression Profiling ; Gene Knockdown Techniques ; Mice ; Microarray Analysis ; Molecular Sequence Data ; NIH 3T3 Cells ; Oxidative Stress ; Selenoprotein W/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Antioxidative role of selenoprotein W in oxidant-induced mouse embryonic neuronal cell death.
Autorzy:
Chung YW; Laboratory of Cellular and Molecular Biochemistry, School of Life Sciences and Biotechnology, Korea University, Seoul 136-701, Korea.
Jeong D
Noh OJ
Park YH
Kang SI
Lee MG
Lee TH
Yim MB
Kim IY
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Źródło:
Molecules and cells [Mol Cells] 2009 May 31; Vol. 27 (5), pp. 609-13. Date of Electronic Publication: 2009 May 15.
Typ publikacji:
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antioxidants/*metabolism
Nervous System/*embryology
Nervous System/*pathology
Neurons/*metabolism
Selenoprotein W/*metabolism
Animals ; Apoptosis/genetics ; Cells, Cultured ; Cerebral Cortex/pathology ; Embryo, Mammalian ; Gene Expression Profiling ; Gene Expression Regulation, Developmental ; Hydrogen Peroxide/metabolism ; Mice ; Nervous System/growth & development ; Neurons/pathology ; Oxidative Stress ; RNA, Small Interfering/genetics ; Selenoprotein W/genetics ; Superoxide Dismutase/genetics ; Superoxide Dismutase/metabolism ; Superoxide Dismutase-1
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Comparative Proteomic Analysis Reveals the Effect of Selenoprotein W Deficiency on Oligodendrogenesis in Fear Memory
Autorzy:
Jiaxin Situ
Xuelian Huang
Mingyang Zuo
Yingying Huang
Bingyu Ren
Qiong Liu
Pokaż więcej
Temat:
selenoprotein W
contextual fear conditioning
tandem mass tag (TMT)-based quantitative proteomics
oligodendrogenesis
Therapeutics. Pharmacology
RM1-950
Źródło:
Antioxidants, Vol 11, Iss 5, p 999 (2022)
Opis pliku:
electronic resource
Relacje:
https://www.mdpi.com/2076-3921/11/5/999; https://doaj.org/toc/2076-3921
Dostęp URL:
https://doaj.org/article/5b8cccd55175460ab0dd724ebf0541ea  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Tytuł:
Different distributions of selenoprotein W and thioredoxin during postnatal brain development and embryogenesis.
Autorzy:
Jeong DW; Laboratory of Cellular and Molecular Biochemistry, School of Life Sciences and Biotechnology, Korea University, Seoul 136-701, Korea.
Kim EH
Kim TS
Chung YW
Kim H
Kim IY
Pokaż więcej
Źródło:
Molecules and cells [Mol Cells] 2004 Feb 29; Vol. 17 (1), pp. 156-9.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Protein Biosynthesis*
Proteins*
Brain/*metabolism
Thioredoxins/*biosynthesis
Amino Acid Sequence ; Animals ; Antioxidants/metabolism ; Antioxidants/pharmacology ; Binding Sites ; Brain/embryology ; Cell Differentiation ; Embryo, Mammalian/metabolism ; Glutathione/metabolism ; Immunoblotting ; In Situ Hybridization ; Liver/metabolism ; Molecular Sequence Data ; Neurons/metabolism ; Phylogeny ; RNA, Messenger/metabolism ; Rats ; Selenium/metabolism ; Selenoprotein W ; Selenoproteins ; Sequence Homology, Amino Acid ; Thioredoxins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Mammalian selenium-containing proteins.
Autorzy:
Behne D; Department Molecular Trace Element Research in the Life Sciences, Hahn-Meitner-Institut Berlin, Glienicker Strasse 100, D-14109 Berlin, Germany. />Kyriakopoulos A
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Źródło:
Annual review of nutrition [Annu Rev Nutr] 2001; Vol. 21, pp. 453-73.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Proteins/*chemistry
Selenium/*analysis
Animals ; Carrier Proteins ; Glutathione Peroxidase/physiology ; Humans ; Iodide Peroxidase/physiology ; Proteins/physiology ; Selenium-Binding Proteins ; Selenoprotein P ; Selenoprotein W ; Selenoproteins ; Thioredoxin-Disulfide Reductase/physiology
Czasopismo naukowe
Szczegóły
Tytuł:
Selenium metabolism in zebrafish: multiplicity of selenoprotein genes and expression of a protein containing 17 selenocysteine residues.
Autorzy:
Kryukov GV; Department of Biochemistry, University of Nebraska, Lincoln, NE 68588-0664, USA.
Gladyshev VN
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Źródło:
Genes to cells : devoted to molecular & cellular mechanisms [Genes Cells] 2000 Dec; Vol. 5 (12), pp. 1049-60.
Typ publikacji:
Journal Article; Research Support, U.S. Gov't, P.H.S.
MeSH Terms:
Multigene Family*
Protein Biosynthesis*
Proteins/*genetics
Selenium/*metabolism
Selenocysteine/*metabolism
Amino Acid Sequence ; Animals ; Base Sequence ; Cell Line ; Chlorocebus aethiops ; DNA, Complementary/chemistry ; Gene Duplication ; Humans ; Molecular Sequence Data ; Phylogeny ; Proteins/isolation & purification ; Proteins/metabolism ; Selenoprotein P ; Selenoprotein W ; Selenoproteins ; Sequence Alignment ; Sequence Homology, Amino Acid ; Zebrafish ; Zebrafish Proteins
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Responsiveness of selenoproteins to dietary selenium.
Autorzy:
Allan CB; NHLBI, National Institutes of Health, Bethesda, Maryland 20892, USA. />Lacourciere GM
Stadtman TC
Pokaż więcej
Źródło:
Annual review of nutrition [Annu Rev Nutr] 1999; Vol. 19, pp. 1-16.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Diet*
Proteins/*metabolism
Selenium/*administration & dosage
Animals ; Glutathione Peroxidase/metabolism ; Humans ; Iodide Peroxidase/metabolism ; Selenoprotein P ; Selenoprotein W ; Selenoproteins ; Thioredoxin-Disulfide Reductase/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
Compensatory Protection of Thioredoxin-Deficient Cells from Etoposide-Induced Cell Death by Selenoprotein W via Interaction with 14-3-3
Autorzy:
Hyunwoo Kang
Yeong Ha Jeon
Minju Ham
Kwanyoung Ko
Ick Young Kim
Pokaż więcej
Temat:
selenoprotein W
thioredoxin
14-3-3
Akt
cell death
Biology (General)
QH301-705.5
Chemistry
QD1-999
Źródło:
International Journal of Molecular Sciences, Vol 22, Iss 19, p 10338 (2021)
Opis pliku:
electronic resource
Relacje:
https://www.mdpi.com/1422-0067/22/19/10338; https://doaj.org/toc/1661-6596; https://doaj.org/toc/1422-0067
Dostęp URL:
https://doaj.org/article/3d4d46f2f44a4f2fb7b720a5f412259a  Link otwiera się w nowym oknie
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Identification, characterization of selenoprotein W and its mRNA expression patterns in response to somatostatin 14, cysteamine hydrochloride, 17β-estradiol and a binary mixture of 17β-estradiol and cysteamine hydrochloride in topmouth culter (Erythroculter ilishaeformis)
Autorzy:
Dong, Haiyan
Chen, Wenbo
Sun, Chao
Sun, Jianwei
Wang, Yanlin
Xie, Chao
Fu, Qianwen
Zhu, Junjie
Ye, Jinyun
Pokaż więcej
Źródło:
Fish Physiology and Biochemistry. February 2017 43(1):115-126
Czasopismo naukowe
Szczegóły
Wyświetlanie 1-20 z 227

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